Source: Cytokine. Unidades: FMRP, EERP
Subjects: ESTRESSE OXIDATIVO, ETANOL, CITOTOXINAS, SEPSE, INTERLEUCINAS
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NAKASHIMA, Marcelo A. et al. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, v. 121, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2019.154734. Acesso em: 04 out. 2024.APA
Nakashima, M. A., Silva, C. B. P. da, Gonzaga, N. de A., Simplicio, J. A., Omoto, A. C. M., Tirapelli, L. F., et al. (2019). Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, 121. doi:10.1016/j.cyto.2019.154734NLM
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734Vancouver
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734