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Artigo de periodico
The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior (2018)
Teixeira, Lívia Clemente Motta ; Nils, Aline Vilar Machado; Battagello, Daniella Sabino ; Diniz, Giovanne Baroni ; Silva, Jéssica Andrade da ; Silva Junior, José Sinésio; Matos, Raphael Afonso ; Amaral, Fernanda Gaspar do; Xavier, Gilberto Fernando ; Bittencourt, Jackson Cioni ; Reiter, Russel J. ; Lucassen, Paul John ; Korosi, Aniko ; Cipolla Neto, José
Source: Hormones and Behavior. Unidades: IB, ICB
Subjects: MELATONINA, GLÂNDULA PINEAL, GRAVIDEZ, FOTOPERIODISMO, FETO, PARÂMETROS DE RESPOSTAS, LACTAÇÃO
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TEIXEIRA, Lívia Clemente Motta et al. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior. Hormones and Behavior, v. 105, p. 146-156, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2018.08.006. Acesso em: 02 nov. 2024.
APA
Teixeira, L. C. M., Nils, A. V. M., Battagello, D. S., Diniz, G. B., Silva, J. A. da, Silva Junior, J. S., et al. (2018). The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior. Hormones and Behavior, 105, 146-156. doi:10.1016/j.yhbeh.2018.08.006
NLM
Teixeira LCM, Nils AVM, Battagello DS, Diniz GB, Silva JA da, Silva Junior JS, Matos RA, Amaral FG do, Xavier GF, Bittencourt JC, Reiter RJ, Lucassen PJ, Korosi A, Cipolla Neto J. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior [Internet]. Hormones and Behavior. 2018 ; 105 146-156.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.yhbeh.2018.08.006
Vancouver
Teixeira LCM, Nils AVM, Battagello DS, Diniz GB, Silva JA da, Silva Junior JS, Matos RA, Amaral FG do, Xavier GF, Bittencourt JC, Reiter RJ, Lucassen PJ, Korosi A, Cipolla Neto J. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior [Internet]. Hormones and Behavior. 2018 ; 105 146-156.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.yhbeh.2018.08.006
Artigo de periodico
Tn6350, a novel transposon carrying pyocin S8 genes encoding a bacteriocin with activity against carbapenemase-producing Pseudomonas aeruginosa (2017)
Turano, Helena; Gomes, Fernando ; Carvalho, Gesiele Almeida Barros; Lopes, Ralf Bruno Moura; Cerdeira, Louise Teixeira; Netto, Luis Eduardo Soares ; Gales, Ana Cristina; Lincopan, Nilton ; Netto, Luis Eduardo Soares
Source: Antimicrobial Agents and Chemotherapy. Unidades: ICB, IB
Subjects: MICROBIOLOGIA, PSEUDOMONAS, GENÉTICA BACTERIANA, ATIVAÇÃO ENZIMÁTICA, RESISTÊNCIA MICROBIANA ÀS DROGAS, SEQUENCIAMENTO GENÉTICO, AGENTES ANTIMICROBIANOS
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TURANO, Helena et al. Tn6350, a novel transposon carrying pyocin S8 genes encoding a bacteriocin with activity against carbapenemase-producing Pseudomonas aeruginosa. Antimicrobial Agents and Chemotherapy, v. 61, n. 5, 2017Tradução . . Disponível em: https://doi.org/10.1128/AAC.00100-17. Acesso em: 02 nov. 2024.
APA
Turano, H., Gomes, F., Carvalho, G. A. B., Lopes, R. B. M., Cerdeira, L. T., Netto, L. E. S., et al. (2017). Tn6350, a novel transposon carrying pyocin S8 genes encoding a bacteriocin with activity against carbapenemase-producing Pseudomonas aeruginosa. Antimicrobial Agents and Chemotherapy, 61( 5). doi:10.1128/AAC.00100-17
NLM
Turano H, Gomes F, Carvalho GAB, Lopes RBM, Cerdeira LT, Netto LES, Gales AC, Lincopan N, Netto LES. Tn6350, a novel transposon carrying pyocin S8 genes encoding a bacteriocin with activity against carbapenemase-producing Pseudomonas aeruginosa [Internet]. Antimicrobial Agents and Chemotherapy. 2017 ; 61( 5):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1128/AAC.00100-17
Vancouver
Turano H, Gomes F, Carvalho GAB, Lopes RBM, Cerdeira LT, Netto LES, Gales AC, Lincopan N, Netto LES. Tn6350, a novel transposon carrying pyocin S8 genes encoding a bacteriocin with activity against carbapenemase-producing Pseudomonas aeruginosa [Internet]. Antimicrobial Agents and Chemotherapy. 2017 ; 61( 5):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1128/AAC.00100-17
Artigo de periodico
Spatial learning and neurogenesis: effects of cessation of wheel running and survival of novel neurons by engagement in cognitive tasks (2016)
Motta-Teixeira, Lívia Clemente; Takada, Silvia Honda; Machado-Nils, Aline Vilar; Nogueira, Maria Inês; Xavier, Gilberto Fernando
Source: Hippocampus. Unidades: ICB, IB
Assunto: ANATOMIA
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MOTTA-TEIXEIRA, Lívia Clemente et al. Spatial learning and neurogenesis: effects of cessation of wheel running and survival of novel neurons by engagement in cognitive tasks. Hippocampus, v. 26, n. 6, p. 794-803, 2016Tradução . . Disponível em: https://doi.org/10.1002/hipo.225606. Acesso em: 02 nov. 2024.
APA
Motta-Teixeira, L. C., Takada, S. H., Machado-Nils, A. V., Nogueira, M. I., & Xavier, G. F. (2016). Spatial learning and neurogenesis: effects of cessation of wheel running and survival of novel neurons by engagement in cognitive tasks. Hippocampus, 26( 6), 794-803. doi:10.1002/hipo.225606
NLM
Motta-Teixeira LC, Takada SH, Machado-Nils AV, Nogueira MI, Xavier GF. Spatial learning and neurogenesis: effects of cessation of wheel running and survival of novel neurons by engagement in cognitive tasks [Internet]. Hippocampus. 2016 ; 26( 6): 794-803.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1002/hipo.225606
Vancouver
Motta-Teixeira LC, Takada SH, Machado-Nils AV, Nogueira MI, Xavier GF. Spatial learning and neurogenesis: effects of cessation of wheel running and survival of novel neurons by engagement in cognitive tasks [Internet]. Hippocampus. 2016 ; 26( 6): 794-803.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1002/hipo.225606
Artigo de periodico
Binding Affinity, Specificity and Comparative Biodistribution of the Parental Murine Monoclonal Antibody MX35 (Anti-NaPi2b) and Its Humanized Version Rebmab200 (2015)
Lindegren, Sture; Andrade, Luciana N. S.; Bäck, Tom; Machado, Camila Maria L.; Horta, Bruno Brasil; Buchpiguel, Carlos; Moro, Ana Maria; Okamoto, Oswaldo Keith ; Jacobsson, Lars; Cederkrantz, Elin; Washiyama, Kohshin; Aneheim, Emma; Palm, Stig; Jensen, Holger; Tuma, Maria Carolina B.; Chammas, Roger; Hultborn, Ragnar; Albertsson, Per
Source: PLOS ONE. Unidades: FM, IB
Subjects: PROTEÍNAS, RATOS, NEOPLASIAS CRANIANAS, TOMOGRAFIA COMPUTADORIZADA POR EMISSÃO DE FÓTON ÚNICO, ANTICORPOS MONOCLONAIS
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LINDEGREN, Sture et al. Binding Affinity, Specificity and Comparative Biodistribution of the Parental Murine Monoclonal Antibody MX35 (Anti-NaPi2b) and Its Humanized Version Rebmab200. PLOS ONE, v. 10, n. 5, p. 16 ; art. e0126298, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0126298. Acesso em: 02 nov. 2024.
APA
Lindegren, S., Andrade, L. N. S., Bäck, T., Machado, C. M. L., Horta, B. B., Buchpiguel, C., et al. (2015). Binding Affinity, Specificity and Comparative Biodistribution of the Parental Murine Monoclonal Antibody MX35 (Anti-NaPi2b) and Its Humanized Version Rebmab200. PLOS ONE, 10( 5), 16 ; art. e0126298. doi:10.1371/journal.pone.0126298
NLM
Lindegren S, Andrade LNS, Bäck T, Machado CML, Horta BB, Buchpiguel C, Moro AM, Okamoto OK, Jacobsson L, Cederkrantz E, Washiyama K, Aneheim E, Palm S, Jensen H, Tuma MCB, Chammas R, Hultborn R, Albertsson P. Binding Affinity, Specificity and Comparative Biodistribution of the Parental Murine Monoclonal Antibody MX35 (Anti-NaPi2b) and Its Humanized Version Rebmab200 [Internet]. PLOS ONE. 2015 ; 10( 5): 16 ; art. e0126298.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0126298
Vancouver
Lindegren S, Andrade LNS, Bäck T, Machado CML, Horta BB, Buchpiguel C, Moro AM, Okamoto OK, Jacobsson L, Cederkrantz E, Washiyama K, Aneheim E, Palm S, Jensen H, Tuma MCB, Chammas R, Hultborn R, Albertsson P. Binding Affinity, Specificity and Comparative Biodistribution of the Parental Murine Monoclonal Antibody MX35 (Anti-NaPi2b) and Its Humanized Version Rebmab200 [Internet]. PLOS ONE. 2015 ; 10( 5): 16 ; art. e0126298.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0126298
Artigo de periodico
p27 variant and corticotropinoma susceptibility: a genetic and in vitro study (2014)
Sekiya, Tomoko; Bronstein, Marcello Delano ; Benfini, Katiuscia; Longuini, Viviane Cristina; Jallad, Raquel Soares; Machado, Marcio Cardoso; Gonçalves, Tatiana Denk; Osaki, Luciana Harumi; Higashi, Leonardo; Viana Junior, Jose; Kater, Claudio; Lee, Misu; Molatore, Sara; Francisco, Guilherme; Chammas, Roger ; Naslavsky, Michel ; Schlesinger, David; Gama, Patrícia ; Duarte, Yeda Aparecida de Oliveira ; Lebrão, Maria Lúcia ; Zatz, Mayana ; Meirelles, Osorio; Liberman, Bernardo; Fragoso, Maria Candida Barisson Villares ; Toledo, Sergio Pereira de Almeida; Pellegata, Natalia S.; Toledo, Rodrigo de Almeida; Zatz, Mayana
Source: Endocrine-Related Cancer. Unidades: ICB, FM, EE, IB, FSP
Subjects: BIOLOGIA CELULAR, GENÉTICA, MUTAÇÃO GENÉTICA, RATOS, POLIMORFISMO, GENÓTIPOS, NEOPLASIAS DA TIREOIDE, ADENOMA
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SEKIYA, Tomoko et al. p27 variant and corticotropinoma susceptibility: a genetic and in vitro study. Endocrine-Related Cancer, v. 21, n. 3, p. 395-404, 2014Tradução . . Disponível em: https://doi.org/10.1530/ERC-13-0486. Acesso em: 02 nov. 2024.
APA
Sekiya, T., Bronstein, M. D., Benfini, K., Longuini, V. C., Jallad, R. S., Machado, M. C., et al. (2014). p27 variant and corticotropinoma susceptibility: a genetic and in vitro study. Endocrine-Related Cancer, 21( 3), 395-404. doi:10.1530/ERC-13-0486
NLM
Sekiya T, Bronstein MD, Benfini K, Longuini VC, Jallad RS, Machado MC, Gonçalves TD, Osaki LH, Higashi L, Viana Junior J, Kater C, Lee M, Molatore S, Francisco G, Chammas R, Naslavsky M, Schlesinger D, Gama P, Duarte YA de O, Lebrão ML, Zatz M, Meirelles O, Liberman B, Fragoso MCBV, Toledo SP de A, Pellegata NS, Toledo R de A, Zatz M. p27 variant and corticotropinoma susceptibility: a genetic and in vitro study [Internet]. Endocrine-Related Cancer. 2014 ; 21( 3): 395-404.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1530/ERC-13-0486
Vancouver
Sekiya T, Bronstein MD, Benfini K, Longuini VC, Jallad RS, Machado MC, Gonçalves TD, Osaki LH, Higashi L, Viana Junior J, Kater C, Lee M, Molatore S, Francisco G, Chammas R, Naslavsky M, Schlesinger D, Gama P, Duarte YA de O, Lebrão ML, Zatz M, Meirelles O, Liberman B, Fragoso MCBV, Toledo SP de A, Pellegata NS, Toledo R de A, Zatz M. p27 variant and corticotropinoma susceptibility: a genetic and in vitro study [Internet]. Endocrine-Related Cancer. 2014 ; 21( 3): 395-404.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1530/ERC-13-0486
Artigo de periodico
Rebmab200, a Humanized Monoclonal Antibody Targeting the Sodium Phosphate Transporter NaPi2b Displays Strong Immune Mediated Cytotoxicity against Cancer: A Novel Reagent for Targeted Antibody Therapy of Cancer (2013)
Santos, Mariana Lopes dos; Yeda, Fernanda Perez; Tsuruta, Lilian Rumi; Horta, Bruno Brasil; Pimenta, Alécio A.; Degaki, Theri Leica; Soares, Ibere C.; Tuma, Maria Carolina; Okamoto, Oswaldo Keith ; Alves, Venancio A. F.; Old, Lloyd J.; Ritter, Gerd; Moro, Ana Maria
Source: PLOS ONE. Unidades: IB, FM
Subjects: REAÇÃO EM CADEIA POR POLIMERASE, SISTEMA IMUNE, SÓDIO, NEOPLASIAS (IMUNOLOGIA;TOXICIDADE), FOSFATOS, ANTICORPOS MONOCLONAIS
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SANTOS, Mariana Lopes dos et al. Rebmab200, a Humanized Monoclonal Antibody Targeting the Sodium Phosphate Transporter NaPi2b Displays Strong Immune Mediated Cytotoxicity against Cancer: A Novel Reagent for Targeted Antibody Therapy of Cancer. PLOS ONE, v. 8, n. 7, p. 10 , 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0070332. Acesso em: 02 nov. 2024.
APA
Santos, M. L. dos, Yeda, F. P., Tsuruta, L. R., Horta, B. B., Pimenta, A. A., Degaki, T. L., et al. (2013). Rebmab200, a Humanized Monoclonal Antibody Targeting the Sodium Phosphate Transporter NaPi2b Displays Strong Immune Mediated Cytotoxicity against Cancer: A Novel Reagent for Targeted Antibody Therapy of Cancer. PLOS ONE, 8( 7), 10 . doi:10.1371/journal.pone.0070332
NLM
Santos ML dos, Yeda FP, Tsuruta LR, Horta BB, Pimenta AA, Degaki TL, Soares IC, Tuma MC, Okamoto OK, Alves VAF, Old LJ, Ritter G, Moro AM. Rebmab200, a Humanized Monoclonal Antibody Targeting the Sodium Phosphate Transporter NaPi2b Displays Strong Immune Mediated Cytotoxicity against Cancer: A Novel Reagent for Targeted Antibody Therapy of Cancer [Internet]. PLOS ONE. 2013 ; 8( 7): 10 .[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0070332
Vancouver
Santos ML dos, Yeda FP, Tsuruta LR, Horta BB, Pimenta AA, Degaki TL, Soares IC, Tuma MC, Okamoto OK, Alves VAF, Old LJ, Ritter G, Moro AM. Rebmab200, a Humanized Monoclonal Antibody Targeting the Sodium Phosphate Transporter NaPi2b Displays Strong Immune Mediated Cytotoxicity against Cancer: A Novel Reagent for Targeted Antibody Therapy of Cancer [Internet]. PLOS ONE. 2013 ; 8( 7): 10 .[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0070332
Artigo de periodico
Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate (2013)
Kobayashi, Gerson Shigeru; Alvizi, Lucas; Sunaga, Daniele Yumi; Francis-West, Philippa; Kuta, Anna; Almada, Bruno Vinícius Pimenta; Ferreira, Simone Gomes; Andrade-Lima, Leonardo Carmo de; Bueno, Daniela Franco; Raposo-Amaral, Cássio Eduardo; Menck, Carlos Frederico Martins ; Passos-Bueno, Maria Rita
Source: PLOS ONE. Unidades: ICB, IB
Assunto: MICROBIOLOGIA
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KOBAYASHI, Gerson Shigeru et al. Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate. PLOS ONE, v. 8, n. 6, p. 1-11, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0065677. Acesso em: 02 nov. 2024.
APA
Kobayashi, G. S., Alvizi, L., Sunaga, D. Y., Francis-West, P., Kuta, A., Almada, B. V. P., et al. (2013). Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate. PLOS ONE, 8( 6), 1-11. doi:10.1371/journal.pone.0065677
NLM
Kobayashi GS, Alvizi L, Sunaga DY, Francis-West P, Kuta A, Almada BVP, Ferreira SG, Andrade-Lima LC de, Bueno DF, Raposo-Amaral CE, Menck CFM, Passos-Bueno MR. Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate [Internet]. PLOS ONE. 2013 ; 8( 6): 1-11.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0065677
Vancouver
Kobayashi GS, Alvizi L, Sunaga DY, Francis-West P, Kuta A, Almada BVP, Ferreira SG, Andrade-Lima LC de, Bueno DF, Raposo-Amaral CE, Menck CFM, Passos-Bueno MR. Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate [Internet]. PLOS ONE. 2013 ; 8( 6): 1-11.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0065677
Artigo de periodico
Cardiac hypertrophy and structural and metabolic remodeling related to seasonal dormancy in the first annual cycle in tegu lizards (2013)
Silveira, Lilian Cristina da; Nascimento, Lucas Francisco R, do; Colquhoun, Alison ; Abe, Augusto S.; Souza, Silvia Cristina Ribeiro de
Source: Comparative Bichemistry and Physiology, Part A, Molecular and Integrative Physiology. Unidades: ICB, IB
Assunto: HISTOLOGIA
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ABNT
SILVEIRA, Lilian Cristina da et al. Cardiac hypertrophy and structural and metabolic remodeling related to seasonal dormancy in the first annual cycle in tegu lizards. Comparative Bichemistry and Physiology, Part A, Molecular and Integrative Physiology, v. 165, n. 3, p. 371-380, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.cbpa.2013.04.006. Acesso em: 02 nov. 2024.
APA
Silveira, L. C. da, Nascimento, L. F. R., Colquhoun, A., Abe, A. S., & Souza, S. C. R. de. (2013). Cardiac hypertrophy and structural and metabolic remodeling related to seasonal dormancy in the first annual cycle in tegu lizards. Comparative Bichemistry and Physiology, Part A, Molecular and Integrative Physiology, 165( 3), 371-380. doi:10.1016/j.cbpa.2013.04.006
NLM
Silveira LC da, Nascimento LFR, Colquhoun A, Abe AS, Souza SCR de. Cardiac hypertrophy and structural and metabolic remodeling related to seasonal dormancy in the first annual cycle in tegu lizards [Internet]. Comparative Bichemistry and Physiology, Part A, Molecular and Integrative Physiology. 2013 ; 165( 3): 371-380.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cbpa.2013.04.006
Vancouver
Silveira LC da, Nascimento LFR, Colquhoun A, Abe AS, Souza SCR de. Cardiac hypertrophy and structural and metabolic remodeling related to seasonal dormancy in the first annual cycle in tegu lizards [Internet]. Comparative Bichemistry and Physiology, Part A, Molecular and Integrative Physiology. 2013 ; 165( 3): 371-380.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cbpa.2013.04.006
Artigo de periodico
Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome (2013)
Yeh, Erika; Fanganiello, Roberto D.; Sunaga, Daniele Y.; Zhou, Xueyan; Holmes, Gregory; Rocha, Katia M.; Alonso, Nivaldo; Matushita, Hamilton; Wang, Yingli; Jabs, Ethylin W.; Passos-Bueno, Maria Rita
Source: PLOS ONE. Unidades: FM, IB
Subjects: FATORES DE CRESCIMENTO (DEFICIÊNCIA), MUTAÇÃO GENÉTICA, FENÓTIPOS, GENÓTIPOS, BIOLOGIA MOLECULAR, CRANIOSSINOSTOSE (GENÉTICA)
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YEH, Erika et al. Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome. PLOS ONE, v. 8, n. 4, p. 7 , 2013Tradução . . Disponível em: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0060439. Acesso em: 02 nov. 2024.
APA
Yeh, E., Fanganiello, R. D., Sunaga, D. Y., Zhou, X., Holmes, G., Rocha, K. M., et al. (2013). Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome. PLOS ONE, 8( 4), 7 . doi:10.1371/journal.pone.0060439
NLM
Yeh E, Fanganiello RD, Sunaga DY, Zhou X, Holmes G, Rocha KM, Alonso N, Matushita H, Wang Y, Jabs EW, Passos-Bueno MR. Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome [Internet]. PLOS ONE. 2013 ; 8( 4): 7 .[citado 2024 nov. 02 ] Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0060439
Vancouver
Yeh E, Fanganiello RD, Sunaga DY, Zhou X, Holmes G, Rocha KM, Alonso N, Matushita H, Wang Y, Jabs EW, Passos-Bueno MR. Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome [Internet]. PLOS ONE. 2013 ; 8( 4): 7 .[citado 2024 nov. 02 ] Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0060439
Artigo de periodico
Bias and learning in temporal binding: intervals between actions and outcomes are compressed by prior bias (2013)
Cravo, Andre M.; Haddad Junior, Hamilton; Claessens, Peter M.E.; Baldo, Marcus Vinícius Chrysóstomo
Source: Consciousness and Cognition. Unidades: IB, ICB
Assunto: FISIOLOGIA
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ABNT
CRAVO, Andre M. et al. Bias and learning in temporal binding: intervals between actions and outcomes are compressed by prior bias. Consciousness and Cognition, v. 22, n. 4, p. 1174-1180, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.concog.2013.08.001. Acesso em: 02 nov. 2024.
APA
Cravo, A. M., Haddad Junior, H., Claessens, P. M. E., & Baldo, M. V. C. (2013). Bias and learning in temporal binding: intervals between actions and outcomes are compressed by prior bias. Consciousness and Cognition, 22( 4), 1174-1180. doi:10.1016/j.concog.2013.08.001
NLM
Cravo AM, Haddad Junior H, Claessens PME, Baldo MVC. Bias and learning in temporal binding: intervals between actions and outcomes are compressed by prior bias [Internet]. Consciousness and Cognition. 2013 ; 22( 4): 1174-1180.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.concog.2013.08.001
Vancouver
Cravo AM, Haddad Junior H, Claessens PME, Baldo MVC. Bias and learning in temporal binding: intervals between actions and outcomes are compressed by prior bias [Internet]. Consciousness and Cognition. 2013 ; 22( 4): 1174-1180.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.concog.2013.08.001
Artigo de periodico
Changes in CREB activation in the prefrontal cortex and hippocampus blunt ethanol-induced behavioral sensitization in adolescent mice (2013)
Soares-Simi, Sabrina L.; Pastrello, Daniel M.; Ferreira, Zulma S ; Yonamine, Maurício ; Marcourakis, Tania ; Scavone, Cristoforo ; Camarini, Rosana
Source: Frontiers in Integrative Neuroscience. Unidades: ICB, IB, FCF
Assunto: FARMACOLOGIA
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ABNT
SOARES-SIMI, Sabrina L. et al. Changes in CREB activation in the prefrontal cortex and hippocampus blunt ethanol-induced behavioral sensitization in adolescent mice. Frontiers in Integrative Neuroscience, v. 7, p. 1-12, 2013Tradução . . Disponível em: https://doi.org/10.3389/fnint.2013.00094. Acesso em: 02 nov. 2024.
APA
Soares-Simi, S. L., Pastrello, D. M., Ferreira, Z. S., Yonamine, M., Marcourakis, T., Scavone, C., & Camarini, R. (2013). Changes in CREB activation in the prefrontal cortex and hippocampus blunt ethanol-induced behavioral sensitization in adolescent mice. Frontiers in Integrative Neuroscience, 7, 1-12. doi:10.3389/fnint.2013.00094
NLM
Soares-Simi SL, Pastrello DM, Ferreira ZS, Yonamine M, Marcourakis T, Scavone C, Camarini R. Changes in CREB activation in the prefrontal cortex and hippocampus blunt ethanol-induced behavioral sensitization in adolescent mice [Internet]. Frontiers in Integrative Neuroscience. 2013 ; 7 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3389/fnint.2013.00094
Vancouver
Soares-Simi SL, Pastrello DM, Ferreira ZS, Yonamine M, Marcourakis T, Scavone C, Camarini R. Changes in CREB activation in the prefrontal cortex and hippocampus blunt ethanol-induced behavioral sensitization in adolescent mice [Internet]. Frontiers in Integrative Neuroscience. 2013 ; 7 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3389/fnint.2013.00094
Artigo de periodico
Intracellular mechanisms of hydroquinone toxicity on endotoxin-activated neutrophils (2012)
Hebeda, Cristina Bichels; Pinedo, Fernanda Júdice; Bolonheis, Simone Marques; Ferreira, Zulma S ; Muscará, Marcelo Nicolás ; Teixeira, Simone Aparecida; Farsky, Sandra Helena Poliselli
Source: Archives of Toxicology. Unidades: ICB, IB, FCF
Assunto: FARMACOLOGIA
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ABNT
HEBEDA, Cristina Bichels et al. Intracellular mechanisms of hydroquinone toxicity on endotoxin-activated neutrophils. Archives of Toxicology, v. 86, n. 11, p. 1773-1781, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00204-012-0886-3. Acesso em: 02 nov. 2024.
APA
Hebeda, C. B., Pinedo, F. J., Bolonheis, S. M., Ferreira, Z. S., Muscará, M. N., Teixeira, S. A., & Farsky, S. H. P. (2012). Intracellular mechanisms of hydroquinone toxicity on endotoxin-activated neutrophils. Archives of Toxicology, 86( 11), 1773-1781. doi:10.1007/s00204-012-0886-3
NLM
Hebeda CB, Pinedo FJ, Bolonheis SM, Ferreira ZS, Muscará MN, Teixeira SA, Farsky SHP. Intracellular mechanisms of hydroquinone toxicity on endotoxin-activated neutrophils [Internet]. Archives of Toxicology. 2012 ; 86( 11): 1773-1781.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00204-012-0886-3
Vancouver
Hebeda CB, Pinedo FJ, Bolonheis SM, Ferreira ZS, Muscará MN, Teixeira SA, Farsky SHP. Intracellular mechanisms of hydroquinone toxicity on endotoxin-activated neutrophils [Internet]. Archives of Toxicology. 2012 ; 86( 11): 1773-1781.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00204-012-0886-3
Artigo de periodico
Behavioral meaningful opioidergic stimulation activates kappa receptor gene expression (2012)
Teodorov, Elizabeth; Ferrari, Merari de Fátima Ramires ; Fior-Chadi, Débora Rejane ; Camarini, Rosana ; Felício, Luciano Freitas
Source: Brazilian Journal of Medical and Biological Research. Unidades: IB, ICB, FMVZ
Subjects: REAÇÃO EM CADEIA POR POLIMERASE, EXPRESSÃO GÊNICA, FARMACOECONOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TEODOROV, Elizabeth et al. Behavioral meaningful opioidergic stimulation activates kappa receptor gene expression. Brazilian Journal of Medical and Biological Research, v. 45, n. 10, p. 982-987, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500091. Acesso em: 02 nov. 2024.
APA
Teodorov, E., Ferrari, M. de F. R., Fior-Chadi, D. R., Camarini, R., & Felício, L. F. (2012). Behavioral meaningful opioidergic stimulation activates kappa receptor gene expression. Brazilian Journal of Medical and Biological Research, 45( 10), 982-987. doi:10.1590/S0100-879X2012007500091
NLM
Teodorov E, Ferrari M de FR, Fior-Chadi DR, Camarini R, Felício LF. Behavioral meaningful opioidergic stimulation activates kappa receptor gene expression [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 10): 982-987.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/S0100-879X2012007500091
Vancouver
Teodorov E, Ferrari M de FR, Fior-Chadi DR, Camarini R, Felício LF. Behavioral meaningful opioidergic stimulation activates kappa receptor gene expression [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 10): 982-987.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/S0100-879X2012007500091
Artigo de periodico
Early and late neurodegeneration and memory disruption after intracerebroventricular streptozotocin (2012)
Santos, Taisa de Oliveira; Mazucanti, Caio Henrique Yokoyama; Xavier, Gilberto Fernando ; Torrão, Andréa da Silva
Source: Physiology and Behavior. Unidades: ICB, IB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Taisa de Oliveira et al. Early and late neurodegeneration and memory disruption after intracerebroventricular streptozotocin. Physiology and Behavior, v. 107, n. 3, p. 401-413, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.physbeh.2012.06.019. Acesso em: 02 nov. 2024.
APA
Santos, T. de O., Mazucanti, C. H. Y., Xavier, G. F., & Torrão, A. da S. (2012). Early and late neurodegeneration and memory disruption after intracerebroventricular streptozotocin. Physiology and Behavior, 107( 3), 401-413. doi:10.1016/j.physbeh.2012.06.019
NLM
Santos T de O, Mazucanti CHY, Xavier GF, Torrão A da S. Early and late neurodegeneration and memory disruption after intracerebroventricular streptozotocin [Internet]. Physiology and Behavior. 2012 ; 107( 3): 401-413.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.physbeh.2012.06.019
Vancouver
Santos T de O, Mazucanti CHY, Xavier GF, Torrão A da S. Early and late neurodegeneration and memory disruption after intracerebroventricular streptozotocin [Internet]. Physiology and Behavior. 2012 ; 107( 3): 401-413.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.physbeh.2012.06.019
Artigo de periodico
Transforming growth factor-alpha and nonsyndromic cleft lip with or without palate in brazilian patients: results of a large case-control study (2004)
Passos-Bueno, Maria Rita ; Gaspar, Dinamar A.; Kamiya, Tânia Yoshico; Tescarollo, Graziela; Rabanéa, Daniel Saraiva; Richieri-Costa, Antonio; Alonso, Nivaldo; Araújo, Belmino
Source: The Cleft Palate-Craniofacial Journal. Unidades: IB, HRAC
Subjects: GENES, FISSURA LÁBIOPALATINA, GENÓTIPOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PASSOS-BUENO, Maria Rita et al. Transforming growth factor-alpha and nonsyndromic cleft lip with or without palate in brazilian patients: results of a large case-control study. The Cleft Palate-Craniofacial Journal, v. 41, n. 4, p. 387-391, 2004Tradução . . Acesso em: 02 nov. 2024.
APA
Passos-Bueno, M. R., Gaspar, D. A., Kamiya, T. Y., Tescarollo, G., Rabanéa, D. S., Richieri-Costa, A., et al. (2004). Transforming growth factor-alpha and nonsyndromic cleft lip with or without palate in brazilian patients: results of a large case-control study. The Cleft Palate-Craniofacial Journal, 41( 4), 387-391.
NLM
Passos-Bueno MR, Gaspar DA, Kamiya TY, Tescarollo G, Rabanéa DS, Richieri-Costa A, Alonso N, Araújo B. Transforming growth factor-alpha and nonsyndromic cleft lip with or without palate in brazilian patients: results of a large case-control study. The Cleft Palate-Craniofacial Journal. 2004 ; 41( 4): 387-391.[citado 2024 nov. 02 ]
Vancouver
Passos-Bueno MR, Gaspar DA, Kamiya TY, Tescarollo G, Rabanéa DS, Richieri-Costa A, Alonso N, Araújo B. Transforming growth factor-alpha and nonsyndromic cleft lip with or without palate in brazilian patients: results of a large case-control study. The Cleft Palate-Craniofacial Journal. 2004 ; 41( 4): 387-391.[citado 2024 nov. 02 ]
Artigo de periodico
Waardenburg syndrome: clinical differentiation between types I and II (2003)
Pardono, Eliete; Bever, Yolande van; Ende, Jenneke van den; Havrenne, Poti Chimetta; Iughetti, Paula; Maestrelli, Sylvia Regina Pedrosa; Costa Filho, Orozimbo Alves; Richieri-Costa, Antonio; Frota-Pessoa, Oswaldo; Otto, Paulo A
Source: American Journal of Medical Genetics. Unidades: FOB, HRAC, IB
Assunto: ANORMALIDADES CRANIOFACIAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PARDONO, Eliete et al. Waardenburg syndrome: clinical differentiation between types I and II. American Journal of Medical Genetics, v. 117A, n. 3, p. 223-235, 2003Tradução . . Disponível em: https://doi.org/10.1002/ajmg.a.10193. Acesso em: 02 nov. 2024.
APA
Pardono, E., Bever, Y. van, Ende, J. van den, Havrenne, P. C., Iughetti, P., Maestrelli, S. R. P., et al. (2003). Waardenburg syndrome: clinical differentiation between types I and II. American Journal of Medical Genetics, 117A( 3), 223-235. doi:10.1002/ajmg.a.10193
NLM
Pardono E, Bever Y van, Ende J van den, Havrenne PC, Iughetti P, Maestrelli SRP, Costa Filho OA, Richieri-Costa A, Frota-Pessoa O, Otto PA. Waardenburg syndrome: clinical differentiation between types I and II [Internet]. American Journal of Medical Genetics. 2003 ; 117A( 3): 223-235.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1002/ajmg.a.10193
Vancouver
Pardono E, Bever Y van, Ende J van den, Havrenne PC, Iughetti P, Maestrelli SRP, Costa Filho OA, Richieri-Costa A, Frota-Pessoa O, Otto PA. Waardenburg syndrome: clinical differentiation between types I and II [Internet]. American Journal of Medical Genetics. 2003 ; 117A( 3): 223-235.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1002/ajmg.a.10193
Artigo de periodico
Pfeiffer mutation in an Apert patient: how wide is the spectrum of variability due to mutations in the FGFR2 gene? (1997)
Passos-Bueno, Maria Rita ; Sertié, Andréa Laurato; Richieri-Costa, Antonio
Source: American Journal of Medical Genetics. Unidades: IB, HRAC
Subjects: SÍNDROMES OROFACIODIGITAIS, MUTAÇÃO, POLIMORFISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PASSOS-BUENO, Maria Rita e SERTIÉ, Andréa Laurato e RICHIERI-COSTA, Antonio. Pfeiffer mutation in an Apert patient: how wide is the spectrum of variability due to mutations in the FGFR2 gene?. American Journal of Medical Genetics, v. 71, n. 2, p. 243-245, 1997Tradução . . Disponível em: https://doi.org/10.1002/(sici)1096-8628(19970808)71:2%3C243::aid-ajmg27%3E3.0.co;2-d. Acesso em: 02 nov. 2024.
APA
Passos-Bueno, M. R., Sertié, A. L., & Richieri-Costa, A. (1997). Pfeiffer mutation in an Apert patient: how wide is the spectrum of variability due to mutations in the FGFR2 gene? American Journal of Medical Genetics, 71( 2), 243-245. doi:10.1002/(sici)1096-8628(19970808)71:2%3C243::aid-ajmg27%3E3.0.co;2-d
NLM
Passos-Bueno MR, Sertié AL, Richieri-Costa A. Pfeiffer mutation in an Apert patient: how wide is the spectrum of variability due to mutations in the FGFR2 gene? [Internet]. American Journal of Medical Genetics. 1997 ; 71( 2): 243-245.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1002/(sici)1096-8628(19970808)71:2%3C243::aid-ajmg27%3E3.0.co;2-d
Vancouver
Passos-Bueno MR, Sertié AL, Richieri-Costa A. Pfeiffer mutation in an Apert patient: how wide is the spectrum of variability due to mutations in the FGFR2 gene? [Internet]. American Journal of Medical Genetics. 1997 ; 71( 2): 243-245.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1002/(sici)1096-8628(19970808)71:2%3C243::aid-ajmg27%3E3.0.co;2-d

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