Filtros : "Indexado na Base de Dados PuBmed" "FISIOLOGIA" Removidos: "FCF" "Folador, A." "Lopes, R A" Limpar

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  • Source: Journal of Cellular Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      GRACIANO, Maria Fernanda R. et al. NAD(P)H oxidase participates in the palmitate-induced superoxide production and insulin secretion by rat pancreatic islets. Journal of Cellular Physiology, v. 226, n. 4, p. 1110-1117, 2011Tradução . . Disponível em: https://doi.org/10.1002/jcp.22432. Acesso em: 01 out. 2024.
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      Graciano, M. F. R., Santos, L. R. B. dos, Curi, R., & Carpinelli, A. R. (2011). NAD(P)H oxidase participates in the palmitate-induced superoxide production and insulin secretion by rat pancreatic islets. Journal of Cellular Physiology, 226( 4), 1110-1117. doi:10.1002/jcp.22432
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      Graciano MFR, Santos LRB dos, Curi R, Carpinelli AR. NAD(P)H oxidase participates in the palmitate-induced superoxide production and insulin secretion by rat pancreatic islets [Internet]. Journal of Cellular Physiology. 2011 ; 226( 4): 1110-1117.[citado 2024 out. 01 ] Available from: https://doi.org/10.1002/jcp.22432
    • Vancouver

      Graciano MFR, Santos LRB dos, Curi R, Carpinelli AR. NAD(P)H oxidase participates in the palmitate-induced superoxide production and insulin secretion by rat pancreatic islets [Internet]. Journal of Cellular Physiology. 2011 ; 226( 4): 1110-1117.[citado 2024 out. 01 ] Available from: https://doi.org/10.1002/jcp.22432
  • Source: European Journal of Pain. Unidade: ICB

    Assunto: FISIOLOGIA

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      PAGANO, Rosana L. et al. Transdural motor cortex stimulation reverses neuropathic pain in rats: a profile of neuronal activation. European Journal of Pain, v. 15, p. 268.e1-268.e14, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.ejpain.2010.08.003. Acesso em: 01 out. 2024.
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      Pagano, R. L., Assis, D. V., Clara, J. A., Alves, A. da S., Dale, C. S., Teixeira, M. J., et al. (2011). Transdural motor cortex stimulation reverses neuropathic pain in rats: a profile of neuronal activation. European Journal of Pain, 15, 268.e1-268.e14. doi:10.1016/j.ejpain.2010.08.003
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      Pagano RL, Assis DV, Clara JA, Alves A da S, Dale CS, Teixeira MJ, Fonoff ET, Britto LRG de. Transdural motor cortex stimulation reverses neuropathic pain in rats: a profile of neuronal activation [Internet]. European Journal of Pain. 2011 ;15 268.e1-268.e14.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.ejpain.2010.08.003
    • Vancouver

      Pagano RL, Assis DV, Clara JA, Alves A da S, Dale CS, Teixeira MJ, Fonoff ET, Britto LRG de. Transdural motor cortex stimulation reverses neuropathic pain in rats: a profile of neuronal activation [Internet]. European Journal of Pain. 2011 ;15 268.e1-268.e14.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.ejpain.2010.08.003
  • Source: Clinical and Experimental Immunology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BELOTTO, M. F. et al. Moderate exercise improves leucocyte function and decreases inflammation in diabetes. Clinical and Experimental Immunology, v. 162, n. 2, p. 237-243, 2010Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2249.2010.04240.x. Acesso em: 01 out. 2024.
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      Belotto, M. F., Magdalon, J., Rodrigues, H. G., Vinolo, M. A. R., Curi, R., Pithon-Curi, T. C., & Hatanaka, E. (2010). Moderate exercise improves leucocyte function and decreases inflammation in diabetes. Clinical and Experimental Immunology, 162( 2), 237-243. doi:10.1111/j.1365-2249.2010.04240.x
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      Belotto MF, Magdalon J, Rodrigues HG, Vinolo MAR, Curi R, Pithon-Curi TC, Hatanaka E. Moderate exercise improves leucocyte function and decreases inflammation in diabetes [Internet]. Clinical and Experimental Immunology. 2010 ; 162( 2): 237-243.[citado 2024 out. 01 ] Available from: https://doi.org/10.1111/j.1365-2249.2010.04240.x
    • Vancouver

      Belotto MF, Magdalon J, Rodrigues HG, Vinolo MAR, Curi R, Pithon-Curi TC, Hatanaka E. Moderate exercise improves leucocyte function and decreases inflammation in diabetes [Internet]. Clinical and Experimental Immunology. 2010 ; 162( 2): 237-243.[citado 2024 out. 01 ] Available from: https://doi.org/10.1111/j.1365-2249.2010.04240.x
  • Source: Journal of Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      REN, Xueying et al. Nutrient selection in the absence of taste receptor signaling. Journal of Neuroscience, v. 30, n. 23, p. 8012-8023, 2010Tradução . . Disponível em: https://doi.org/10.1523/JNEUROSCI.5749-09.2010. Acesso em: 01 out. 2024.
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      Ren, X., Ferreira, J. G., Zhou, L., Lagnado, S. J. S., Yeckel, C. W., & Araújo, I. E. de. (2010). Nutrient selection in the absence of taste receptor signaling. Journal of Neuroscience, 30( 23), 8012-8023. doi:10.1523/JNEUROSCI.5749-09.2010
    • NLM

      Ren X, Ferreira JG, Zhou L, Lagnado SJS, Yeckel CW, Araújo IE de. Nutrient selection in the absence of taste receptor signaling [Internet]. Journal of Neuroscience. 2010 ; 30( 23): 8012-8023.[citado 2024 out. 01 ] Available from: https://doi.org/10.1523/JNEUROSCI.5749-09.2010
    • Vancouver

      Ren X, Ferreira JG, Zhou L, Lagnado SJS, Yeckel CW, Araújo IE de. Nutrient selection in the absence of taste receptor signaling [Internet]. Journal of Neuroscience. 2010 ; 30( 23): 8012-8023.[citado 2024 out. 01 ] Available from: https://doi.org/10.1523/JNEUROSCI.5749-09.2010
  • Source: American Journal of Physiology: Endocrinology and Metabolism. Unidade: ICB

    Assunto: FISIOLOGIA

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      SILVA, Robinson Sabino da et al. SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats. American Journal of Physiology: Endocrinology and Metabolism, v. 299, n. 6, p. E1028-E1037, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00395.2010. Acesso em: 01 out. 2024.
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      Silva, R. S. da, Wagner, A. B. T., Burgi, K., Okamoto, M. M., Alves, A. da S., Lima, G. A. de, et al. (2010). SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats. American Journal of Physiology: Endocrinology and Metabolism, 299( 6), E1028-E1037. doi:10.1152/ajpendo.00395.2010
    • NLM

      Silva RS da, Wagner ABT, Burgi K, Okamoto MM, Alves A da S, Lima GA de, Freitas HS, Antunes VR, Machado UF. SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2010 ; 299( 6): E1028-E1037.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/ajpendo.00395.2010
    • Vancouver

      Silva RS da, Wagner ABT, Burgi K, Okamoto MM, Alves A da S, Lima GA de, Freitas HS, Antunes VR, Machado UF. SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2010 ; 299( 6): E1028-E1037.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/ajpendo.00395.2010
  • Source: Advances in Physiology Education. Unidade: ICB

    Assunto: FISIOLOGIA

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      HADDAD JUNIOR, Hamilton e BALDO, Marcus Vinícius Chrysóstomo. Teaching diffusion with a coin. Advances in Physiology Education. Bethesda: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1152/advan.00009.2010. Acesso em: 01 out. 2024. , 2010
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      Haddad Junior, H., & Baldo, M. V. C. (2010). Teaching diffusion with a coin. Advances in Physiology Education. Bethesda: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1152/advan.00009.2010
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      Haddad Junior H, Baldo MVC. Teaching diffusion with a coin [Internet]. Advances in Physiology Education. 2010 ; 34( 3): 156-157.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/advan.00009.2010
    • Vancouver

      Haddad Junior H, Baldo MVC. Teaching diffusion with a coin [Internet]. Advances in Physiology Education. 2010 ; 34( 3): 156-157.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/advan.00009.2010
  • Source: Cell Metabolism. Unidade: ICB

    Assunto: FISIOLOGIA

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      DE MARINIS, Yang Z. et al. GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis. Cell Metabolism, v. 11, n. 6, p. 543-553, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.cmet.2010.04.007. Acesso em: 01 out. 2024.
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      De Marinis, Y. Z., Salehi, A., Ward, C. E., Zhang, Q., Abdulkader, F. R. de M., Bengtsson, M., et al. (2010). GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis. Cell Metabolism, 11( 6), 543-553. doi:10.1016/j.cmet.2010.04.007
    • NLM

      De Marinis YZ, Salehi A, Ward CE, Zhang Q, Abdulkader FR de M, Bengtsson M, Braha O, Braun M, Ramracheya R, Amisten S, Habib AM, Moritoh Y, Zhang E, Reimann F, Rosengren AH, Shibasaki T, Gribble F, Renström E, Seino S, Eliasson L, Rorsman P. GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis [Internet]. Cell Metabolism. 2010 ; 11( 6): 543-553.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.cmet.2010.04.007
    • Vancouver

      De Marinis YZ, Salehi A, Ward CE, Zhang Q, Abdulkader FR de M, Bengtsson M, Braha O, Braun M, Ramracheya R, Amisten S, Habib AM, Moritoh Y, Zhang E, Reimann F, Rosengren AH, Shibasaki T, Gribble F, Renström E, Seino S, Eliasson L, Rorsman P. GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis [Internet]. Cell Metabolism. 2010 ; 11( 6): 543-553.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.cmet.2010.04.007
  • Source: European Journal of Heart Failure. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      MALFITANO, Christiane et al. Hyperglycaemia protects the heart after myocardial infarction: aspects of programmed cell survival and cell death. European Journal of Heart Failure, v. 12, n. 7, p. 659-667, 2010Tradução . . Disponível em: https://doi.org/10.1093/eurjhf/hfq053. Acesso em: 01 out. 2024.
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      Malfitano, C., Loureiro, T. C. A., Rodrigues, B., Sirvente, R., Salemi, V. M. C., Rabechi, N. B., et al. (2010). Hyperglycaemia protects the heart after myocardial infarction: aspects of programmed cell survival and cell death. European Journal of Heart Failure, 12( 7), 659-667. doi:10.1093/eurjhf/hfq053
    • NLM

      Malfitano C, Loureiro TCA, Rodrigues B, Sirvente R, Salemi VMC, Rabechi NB, Lacchini S, Curi R, Irigoyen CC. Hyperglycaemia protects the heart after myocardial infarction: aspects of programmed cell survival and cell death [Internet]. European Journal of Heart Failure. 2010 ; 12( 7): 659-667.[citado 2024 out. 01 ] Available from: https://doi.org/10.1093/eurjhf/hfq053
    • Vancouver

      Malfitano C, Loureiro TCA, Rodrigues B, Sirvente R, Salemi VMC, Rabechi NB, Lacchini S, Curi R, Irigoyen CC. Hyperglycaemia protects the heart after myocardial infarction: aspects of programmed cell survival and cell death [Internet]. European Journal of Heart Failure. 2010 ; 12( 7): 659-667.[citado 2024 out. 01 ] Available from: https://doi.org/10.1093/eurjhf/hfq053
  • Source: British Journal of Nutrition. Unidades: EEFERP, ICB

    Assunto: FISIOLOGIA

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      ZOPPI, Claudio C. et al. Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis. British Journal of Nutrition, v. 103, n. 9, p. 1237-1250, 2010Tradução . . Disponível em: https://doi.org/10.1017/S0007114509993060. Acesso em: 01 out. 2024.
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      Zoppi, C. C., Silveira, L. dos R., Oliveira, C. A. M., Boschero, A. C., Curi, R., & Carneiro, E. M. (2010). Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis. British Journal of Nutrition, 103( 9), 1237-1250. doi:10.1017/S0007114509993060
    • NLM

      Zoppi CC, Silveira L dos R, Oliveira CAM, Boschero AC, Curi R, Carneiro EM. Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis [Internet]. British Journal of Nutrition. 2010 ; 103( 9): 1237-1250.[citado 2024 out. 01 ] Available from: https://doi.org/10.1017/S0007114509993060
    • Vancouver

      Zoppi CC, Silveira L dos R, Oliveira CAM, Boschero AC, Curi R, Carneiro EM. Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis [Internet]. British Journal of Nutrition. 2010 ; 103( 9): 1237-1250.[citado 2024 out. 01 ] Available from: https://doi.org/10.1017/S0007114509993060
  • Source: Cellular Physiology and Biochemistry. Unidade: ICB

    Assunto: FISIOLOGIA

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      LACROIX, Luciene Regina Carraro et al. Role of CFTR and ClC-5 in modulating vacuolar H+-ATPase activity in kidney proximal tubule. Cellular Physiology and Biochemistry, v. 26, n. 4-5, p. 563-576, 2010Tradução . . Disponível em: https://doi.org/10.1159/000322324. Acesso em: 01 out. 2024.
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      Lacroix, L. R. C., Lessa, L. M. A., Bezerra, C. N. A., Pessoa, T. D., Souza-Menezes, J., Morales, M. M., et al. (2010). Role of CFTR and ClC-5 in modulating vacuolar H+-ATPase activity in kidney proximal tubule. Cellular Physiology and Biochemistry, 26( 4-5), 563-576. doi:10.1159/000322324
    • NLM

      Lacroix LRC, Lessa LMA, Bezerra CNA, Pessoa TD, Souza-Menezes J, Morales MM, Girardi ACC, Malnic G. Role of CFTR and ClC-5 in modulating vacuolar H+-ATPase activity in kidney proximal tubule [Internet]. Cellular Physiology and Biochemistry. 2010 ; 26( 4-5): 563-576.[citado 2024 out. 01 ] Available from: https://doi.org/10.1159/000322324
    • Vancouver

      Lacroix LRC, Lessa LMA, Bezerra CNA, Pessoa TD, Souza-Menezes J, Morales MM, Girardi ACC, Malnic G. Role of CFTR and ClC-5 in modulating vacuolar H+-ATPase activity in kidney proximal tubule [Internet]. Cellular Physiology and Biochemistry. 2010 ; 26( 4-5): 563-576.[citado 2024 out. 01 ] Available from: https://doi.org/10.1159/000322324
  • Source: Brain Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      FERREIRA, Ana Francisca Barros et al. Moderate exercise changes synaptic and cytoskeletal proteins in motor regions of the rat brain. Brain Research, v. 1361, p. 31-42, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2010.09.045. Acesso em: 01 out. 2024.
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      Ferreira, A. F. B., Real, C. C., Rodrigues, A. C., Alves, A. da S., & Britto, L. R. G. de. (2010). Moderate exercise changes synaptic and cytoskeletal proteins in motor regions of the rat brain. Brain Research, 1361, 31-42. doi:10.1016/j.brainres.2010.09.045
    • NLM

      Ferreira AFB, Real CC, Rodrigues AC, Alves A da S, Britto LRG de. Moderate exercise changes synaptic and cytoskeletal proteins in motor regions of the rat brain [Internet]. Brain Research. 2010 ; 1361 31-42.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.brainres.2010.09.045
    • Vancouver

      Ferreira AFB, Real CC, Rodrigues AC, Alves A da S, Britto LRG de. Moderate exercise changes synaptic and cytoskeletal proteins in motor regions of the rat brain [Internet]. Brain Research. 2010 ; 1361 31-42.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.brainres.2010.09.045
  • Source: Advances in Physiology Education. Unidade: ICB

    Assunto: FISIOLOGIA

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      MONTAGNA, Erik et al. What is transmitted in “synaptic transmission”?. Advances in Physiology Education. Bethesda: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1152/advan.00006.2010. Acesso em: 01 out. 2024. , 2010
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      Montagna, E., Azevedo, A. M. S. de, Romano, C., & Ranvaud, R. D. P. K. C. (2010). What is transmitted in “synaptic transmission”? Advances in Physiology Education. Bethesda: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1152/advan.00006.2010
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      Montagna E, Azevedo AMS de, Romano C, Ranvaud RDPKC. What is transmitted in “synaptic transmission”? [Internet]. Advances in Physiology Education. 2010 ; 34( 2): 115-116.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/advan.00006.2010
    • Vancouver

      Montagna E, Azevedo AMS de, Romano C, Ranvaud RDPKC. What is transmitted in “synaptic transmission”? [Internet]. Advances in Physiology Education. 2010 ; 34( 2): 115-116.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/advan.00006.2010
  • Source: Journal of Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      LAZARENKO, Roman M. et al. Anesthetic activation of central respiratory chemoreceptor neurons involves inhibition of a THIK-1-like background K + current. Journal of Neuroscience, v. 30, n. 27, p. 9324-9334, 2010Tradução . . Disponível em: https://doi.org/10.1523/JNEUROSCI.1956-10.2010. Acesso em: 01 out. 2024.
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      Lazarenko, R. M., Fortuna, M. G., Shi, Y., Mulkey, D. K., Takakura, A. C., Moreira, T. dos S., et al. (2010). Anesthetic activation of central respiratory chemoreceptor neurons involves inhibition of a THIK-1-like background K + current. Journal of Neuroscience, 30( 27), 9324-9334. doi:10.1523/JNEUROSCI.1956-10.2010
    • NLM

      Lazarenko RM, Fortuna MG, Shi Y, Mulkey DK, Takakura AC, Moreira T dos S, Guyenet PG, Bayliss DA. Anesthetic activation of central respiratory chemoreceptor neurons involves inhibition of a THIK-1-like background K + current [Internet]. Journal of Neuroscience. 2010 ; 30( 27): 9324-9334.[citado 2024 out. 01 ] Available from: https://doi.org/10.1523/JNEUROSCI.1956-10.2010
    • Vancouver

      Lazarenko RM, Fortuna MG, Shi Y, Mulkey DK, Takakura AC, Moreira T dos S, Guyenet PG, Bayliss DA. Anesthetic activation of central respiratory chemoreceptor neurons involves inhibition of a THIK-1-like background K + current [Internet]. Journal of Neuroscience. 2010 ; 30( 27): 9324-9334.[citado 2024 out. 01 ] Available from: https://doi.org/10.1523/JNEUROSCI.1956-10.2010
  • Source: Journal of Applied Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      MOREIRA, Thiago dos Santos e TAKAKURA, Ana Carolina e ANTUNES, Vagner Roberto. Systhemic hypertension - involvement of the CNS to this complex puzzle [Carta]. Journal of Applied Physiology. Washington: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 01 out. 2024. , 2010
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      Moreira, T. dos S., Takakura, A. C., & Antunes, V. R. (2010). Systhemic hypertension - involvement of the CNS to this complex puzzle [Carta]. Journal of Applied Physiology. Washington: Instituto de Ciências Biomédicas, Universidade de São Paulo.
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      Moreira T dos S, Takakura AC, Antunes VR. Systhemic hypertension - involvement of the CNS to this complex puzzle [Carta]. Journal of Applied Physiology. 2010 ; 109( 6): 2004.[citado 2024 out. 01 ]
    • Vancouver

      Moreira T dos S, Takakura AC, Antunes VR. Systhemic hypertension - involvement of the CNS to this complex puzzle [Carta]. Journal of Applied Physiology. 2010 ; 109( 6): 2004.[citado 2024 out. 01 ]
  • Source: Amino Acids. Unidades: FCF, FSP, ICB

    Assunto: FISIOLOGIA

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      ROGERO, Marcelo Macedo et al. Effects of glutamine on the nuclear factor-kappaB signaling pathway of murine peritoneal macrophages. Amino Acids, v. 39, n. 2, p. 435-441, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00726-009-0459-9. Acesso em: 01 out. 2024.
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      Rogero, M. M., Borelli, P., Borges, M. C., Vinolo, M. A. R., Nakajima, K., Crisma, A. R., et al. (2010). Effects of glutamine on the nuclear factor-kappaB signaling pathway of murine peritoneal macrophages. Amino Acids, 39( 2), 435-441. doi:10.1007/s00726-009-0459-9
    • NLM

      Rogero MM, Borelli P, Borges MC, Vinolo MAR, Nakajima K, Crisma AR, Ramos AD, Tirapegui J, Fock RA, Curi R. Effects of glutamine on the nuclear factor-kappaB signaling pathway of murine peritoneal macrophages [Internet]. Amino Acids. 2010 ; 39( 2): 435-441.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s00726-009-0459-9
    • Vancouver

      Rogero MM, Borelli P, Borges MC, Vinolo MAR, Nakajima K, Crisma AR, Ramos AD, Tirapegui J, Fock RA, Curi R. Effects of glutamine on the nuclear factor-kappaB signaling pathway of murine peritoneal macrophages [Internet]. Amino Acids. 2010 ; 39( 2): 435-441.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s00726-009-0459-9
  • Source: Experimental Eye Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEONELLI, Mauro e MARTINS, Daniel O. e BRITTO, Luiz Roberto Giorgetti de. TRPV1 receptors are involved in protein nitration and Müller cell reaction in the acutely axotomized rat retina. Experimental Eye Research, v. 91, p. 755-768, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.exer.2010.08.026. Acesso em: 01 out. 2024.
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      Leonelli, M., Martins, D. O., & Britto, L. R. G. de. (2010). TRPV1 receptors are involved in protein nitration and Müller cell reaction in the acutely axotomized rat retina. Experimental Eye Research, 91, 755-768. doi:10.1016/j.exer.2010.08.026
    • NLM

      Leonelli M, Martins DO, Britto LRG de. TRPV1 receptors are involved in protein nitration and Müller cell reaction in the acutely axotomized rat retina [Internet]. Experimental Eye Research. 2010 ; 91 755-768.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.exer.2010.08.026
    • Vancouver

      Leonelli M, Martins DO, Britto LRG de. TRPV1 receptors are involved in protein nitration and Müller cell reaction in the acutely axotomized rat retina [Internet]. Experimental Eye Research. 2010 ; 91 755-768.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.exer.2010.08.026
  • Source: Cellular Physiology and Biochemistry. Unidade: ICB

    Assunto: FISIOLOGIA

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      EGUTI, Débora Mai N. et al. Regulation of Na+/H+ exchanger isoform 1 (NHE1) by calmodulin-binding sites: role of angiotensin II. Cellular Physiology and Biochemistry, v. 26, n. 4-5, p. 541-552, 2010Tradução . . Disponível em: https://doi.org/10.1159/000322322. Acesso em: 01 out. 2024.
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      Eguti, D. M. N., Thieme, K., Leung, G., Souza, M. O. de, & Aires, M. de M. (2010). Regulation of Na+/H+ exchanger isoform 1 (NHE1) by calmodulin-binding sites: role of angiotensin II. Cellular Physiology and Biochemistry, 26( 4-5), 541-552. doi:10.1159/000322322
    • NLM

      Eguti DMN, Thieme K, Leung G, Souza MO de, Aires M de M. Regulation of Na+/H+ exchanger isoform 1 (NHE1) by calmodulin-binding sites: role of angiotensin II [Internet]. Cellular Physiology and Biochemistry. 2010 ; 26( 4-5): 541-552.[citado 2024 out. 01 ] Available from: https://doi.org/10.1159/000322322
    • Vancouver

      Eguti DMN, Thieme K, Leung G, Souza MO de, Aires M de M. Regulation of Na+/H+ exchanger isoform 1 (NHE1) by calmodulin-binding sites: role of angiotensin II [Internet]. Cellular Physiology and Biochemistry. 2010 ; 26( 4-5): 541-552.[citado 2024 out. 01 ] Available from: https://doi.org/10.1159/000322322
  • Source: Lipids. Unidades: FCF, ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      RODRIGUES, Hosana Gomes et al. Dietary free oleic and linoleic acid enhances neutrophil function and modulates the inflammatory response in rats. Lipids, v. 45, n. 9, p. 809-819, 2010Tradução . . Disponível em: https://doi.org/10.1007/s11745-010-3461-9. Acesso em: 01 out. 2024.
    • APA

      Rodrigues, H. G., Vinolo, M. A. R., Magdalon, J., Fujiwara, H., Cavalcanti, D. M. H., Farsky, S. H. P., et al. (2010). Dietary free oleic and linoleic acid enhances neutrophil function and modulates the inflammatory response in rats. Lipids, 45( 9), 809-819. doi:10.1007/s11745-010-3461-9
    • NLM

      Rodrigues HG, Vinolo MAR, Magdalon J, Fujiwara H, Cavalcanti DMH, Farsky SHP, Calder PC, Hatanaka E, Curi R. Dietary free oleic and linoleic acid enhances neutrophil function and modulates the inflammatory response in rats [Internet]. Lipids. 2010 ; 45( 9): 809-819.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s11745-010-3461-9
    • Vancouver

      Rodrigues HG, Vinolo MAR, Magdalon J, Fujiwara H, Cavalcanti DMH, Farsky SHP, Calder PC, Hatanaka E, Curi R. Dietary free oleic and linoleic acid enhances neutrophil function and modulates the inflammatory response in rats [Internet]. Lipids. 2010 ; 45( 9): 809-819.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s11745-010-3461-9
  • Source: Journal of Cellular Physiology. Unidades: FMRP, ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SILVA, Lucas T. Parreiras e et al. Evidences of a role for eukaryotic translation initiation factor 5A (eIF5A) in mouse embryogenesis and cell differentiation. Journal of Cellular Physiology, v. 225, n. 2, p. 50-505, 2010Tradução . . Disponível em: https://doi.org/10.1002/jcp.22229. Acesso em: 01 out. 2024.
    • APA

      Silva, L. T. P. e, Luchessi, A. D., Reis, R. I., Oliver, C., Jamur, M. C., Ramos, R. G. P., et al. (2010). Evidences of a role for eukaryotic translation initiation factor 5A (eIF5A) in mouse embryogenesis and cell differentiation. Journal of Cellular Physiology, 225( 2), 50-505. doi:10.1002/jcp.22229
    • NLM

      Silva LTP e, Luchessi AD, Reis RI, Oliver C, Jamur MC, Ramos RGP, Oliveira EB de, Curi R, Costa Neto CM da. Evidences of a role for eukaryotic translation initiation factor 5A (eIF5A) in mouse embryogenesis and cell differentiation [Internet]. Journal of Cellular Physiology. 2010 ; 225( 2): 50-505.[citado 2024 out. 01 ] Available from: https://doi.org/10.1002/jcp.22229
    • Vancouver

      Silva LTP e, Luchessi AD, Reis RI, Oliver C, Jamur MC, Ramos RGP, Oliveira EB de, Curi R, Costa Neto CM da. Evidences of a role for eukaryotic translation initiation factor 5A (eIF5A) in mouse embryogenesis and cell differentiation [Internet]. Journal of Cellular Physiology. 2010 ; 225( 2): 50-505.[citado 2024 out. 01 ] Available from: https://doi.org/10.1002/jcp.22229
  • Source: Cardiovascular Diabetology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      LEHNEN, Alexandre M. et al. The beneficial effects of exercise in rodents are preserved after detraining: a phenomenon unrelated to GLUT4 expression. Cardiovascular Diabetology, v. 9, n. 67, 2010Tradução . . Disponível em: https://doi.org/10.1186/1475-2840-9-67. Acesso em: 01 out. 2024.
    • APA

      Lehnen, A. M., Leguisamo, N. M., Pinto, G. H., Markoski, M. M., De Angelis, K., Machado, U. F., & Schaan, B. (2010). The beneficial effects of exercise in rodents are preserved after detraining: a phenomenon unrelated to GLUT4 expression. Cardiovascular Diabetology, 9( 67). doi:10.1186/1475-2840-9-67
    • NLM

      Lehnen AM, Leguisamo NM, Pinto GH, Markoski MM, De Angelis K, Machado UF, Schaan B. The beneficial effects of exercise in rodents are preserved after detraining: a phenomenon unrelated to GLUT4 expression [Internet]. Cardiovascular Diabetology. 2010 ; 9( 67):[citado 2024 out. 01 ] Available from: https://doi.org/10.1186/1475-2840-9-67
    • Vancouver

      Lehnen AM, Leguisamo NM, Pinto GH, Markoski MM, De Angelis K, Machado UF, Schaan B. The beneficial effects of exercise in rodents are preserved after detraining: a phenomenon unrelated to GLUT4 expression [Internet]. Cardiovascular Diabetology. 2010 ; 9( 67):[citado 2024 out. 01 ] Available from: https://doi.org/10.1186/1475-2840-9-67

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