Filtros : "Indexado na Base de Dados PubMed" "MUNHOZ, CAROLINA DEMARCHI" Removidos: "- Engenharia Química" "Financiado pela Cystic Fibrosis Canada" "IFUSP" Limpar

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  • Source: Alcohol. Unidades: ICB, FCF

    Subjects: ÓXIDO NÍTRICO, ETANOL, ESTRESSE, ADOLESCÊNCIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SANTOS, Jaqueline Rocha Borges dos et al. Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice. Alcohol, v. 68, p. 71-79, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.alcohol.2017.10.004. Acesso em: 02 nov. 2024.
    • APA

      Santos, J. R. B. dos, Rae, M., Teixeira, A. M. A., Teixeira, S. A., Munhoz, C. D., Marcourakis, T., et al. (2018). Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice. Alcohol, 68, 71-79. doi:10.1016/j.alcohol.2017.10.004
    • NLM

      Santos JRB dos, Rae M, Teixeira AMA, Teixeira SA, Munhoz CD, Marcourakis T, Szumlinski KK, Camarini R. Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice [Internet]. Alcohol. 2018 ; 68 71-79.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.alcohol.2017.10.004
    • Vancouver

      Santos JRB dos, Rae M, Teixeira AMA, Teixeira SA, Munhoz CD, Marcourakis T, Szumlinski KK, Camarini R. Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice [Internet]. Alcohol. 2018 ; 68 71-79.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.alcohol.2017.10.004
  • Source: Journal of Molecular Medicine. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      TAKANO, Ana Paula Cremasco et al. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, v. 95, n. 6, p. 671-682, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1511-y. Acesso em: 02 nov. 2024.
    • APA

      Takano, A. P. C., Munhoz, C. D., Moriscot, A. S., Gupta, S., & Barreto-Chaves, M. L. M. (2017). S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, 95( 6), 671-682. doi:10.1007/s00109-017-1511-y
    • NLM

      Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
    • Vancouver

      Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
  • Source: Neuropharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      NOVAES, Leonardo S. et al. Environmental enrichment protects against stress-inducedanxiety: Role of glucocorticoid receptor, ERK, and CREB signaling in the basolateral amygdala. Neuropharmacology, v. 113, p. 547-466, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2016.10.026. Acesso em: 02 nov. 2024.
    • APA

      Novaes, L. S., Santos, N. B. dos, Batalhote, R. F. P., Malta, M. B., Camarini, R., Scavone, C., & Munhoz, C. D. (2017). Environmental enrichment protects against stress-inducedanxiety: Role of glucocorticoid receptor, ERK, and CREB signaling in the basolateral amygdala. Neuropharmacology, 113, 547-466. doi:10.1016/j.neuropharm.2016.10.026
    • NLM

      Novaes LS, Santos NB dos, Batalhote RFP, Malta MB, Camarini R, Scavone C, Munhoz CD. Environmental enrichment protects against stress-inducedanxiety: Role of glucocorticoid receptor, ERK, and CREB signaling in the basolateral amygdala [Internet]. Neuropharmacology. 2017 ; 113 547-466.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuropharm.2016.10.026
    • Vancouver

      Novaes LS, Santos NB dos, Batalhote RFP, Malta MB, Camarini R, Scavone C, Munhoz CD. Environmental enrichment protects against stress-inducedanxiety: Role of glucocorticoid receptor, ERK, and CREB signaling in the basolateral amygdala [Internet]. Neuropharmacology. 2017 ; 113 547-466.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuropharm.2016.10.026
  • Source: Aging. Unidade: ICB

    Subjects: FARMACOLOGIA, RATOS, HIPOCAMPU DE ANIMAL

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    • ABNT

      ORELLANA, Ana Maria Marques et al. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus. Aging, v. 7, n. 12, p. 1094-1111, 2015Tradução . . Disponível em: https://doi.org/10.18632/aging.100853. Acesso em: 02 nov. 2024.
    • APA

      Orellana, A. M. M., Vasconcelos, A. R., Leite, J. A., Lima, L. de S., Andreotti, D. Z., Munhoz, C. D., et al. (2015). Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus. Aging, 7( 12), 1094-1111. doi:10.18632/aging.100853
    • NLM

      Orellana AMM, Vasconcelos AR, Leite JA, Lima L de S, Andreotti DZ, Munhoz CD, Kawamoto EM, Scavone C. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus [Internet]. Aging. 2015 ; 7( 12): 1094-1111.[citado 2024 nov. 02 ] Available from: https://doi.org/10.18632/aging.100853
    • Vancouver

      Orellana AMM, Vasconcelos AR, Leite JA, Lima L de S, Andreotti DZ, Munhoz CD, Kawamoto EM, Scavone C. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus [Internet]. Aging. 2015 ; 7( 12): 1094-1111.[citado 2024 nov. 02 ] Available from: https://doi.org/10.18632/aging.100853
  • Source: Neuroendocrinology. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      SORRELLS, Shawn F. et al. Glucocorticoids increase excitotoxic injury and inflammation in the hippocampus of adult male rats. Neuroendocrinology, v. 100, n. 2-3, p. 129-140, 2014Tradução . . Disponível em: https://doi.org/10.1159/000367849. Acesso em: 02 nov. 2024.
    • APA

      Sorrells, S. F., Munhoz, C. D., Manley, N. C., Yen, S., & Sapolsky, R. M. (2014). Glucocorticoids increase excitotoxic injury and inflammation in the hippocampus of adult male rats. Neuroendocrinology, 100( 2-3), 129-140. doi:10.1159/000367849
    • NLM

      Sorrells SF, Munhoz CD, Manley NC, Yen S, Sapolsky RM. Glucocorticoids increase excitotoxic injury and inflammation in the hippocampus of adult male rats [Internet]. Neuroendocrinology. 2014 ; 100( 2-3): 129-140.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1159/000367849
    • Vancouver

      Sorrells SF, Munhoz CD, Manley NC, Yen S, Sapolsky RM. Glucocorticoids increase excitotoxic injury and inflammation in the hippocampus of adult male rats [Internet]. Neuroendocrinology. 2014 ; 100( 2-3): 129-140.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1159/000367849
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      MANLEY, Nathan C. et al. Derivation of injury-responsive dendritic cells for acute brain targeting and therapeutic protein delivery in the stroke-injured rat. PLOS ONE, v. 8, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0061789. Acesso em: 02 nov. 2024.
    • APA

      Manley, N. C., Caso, J. R., Works, M. G., Cutler, A. B., Zemlyak, I., Sun, G., et al. (2013). Derivation of injury-responsive dendritic cells for acute brain targeting and therapeutic protein delivery in the stroke-injured rat. PLOS ONE, 8, 1-10. doi:10.1371/journal.pone.0061789
    • NLM

      Manley NC, Caso JR, Works MG, Cutler AB, Zemlyak I, Sun G, Munhoz CD, Chang S, Sorrells SF, Ermini FV, Decker JH, Bertrand AA, Dinkel KM, Steinberg GK, Sapolsky RM. Derivation of injury-responsive dendritic cells for acute brain targeting and therapeutic protein delivery in the stroke-injured rat [Internet]. PLOS ONE. 2013 ; 8 1-10.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0061789
    • Vancouver

      Manley NC, Caso JR, Works MG, Cutler AB, Zemlyak I, Sun G, Munhoz CD, Chang S, Sorrells SF, Ermini FV, Decker JH, Bertrand AA, Dinkel KM, Steinberg GK, Sapolsky RM. Derivation of injury-responsive dendritic cells for acute brain targeting and therapeutic protein delivery in the stroke-injured rat [Internet]. PLOS ONE. 2013 ; 8 1-10.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0061789
  • Source: Neuropharmacology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 02 nov. 2024.
    • APA

      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
  • Source: Journal of Neuroscience. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      SORRELLS, Shawn F. et al. Glucocorticoid signaling in myeloid cells worsens acute CNS injury and inflammation. Journal of Neuroscience, v. 33, n. 18, p. 7877-7889, 2013Tradução . . Disponível em: https://doi.org/10.1523/JNEUROSCI.4705-12.2013. Acesso em: 02 nov. 2024.
    • APA

      Sorrells, S. F., Caso, J. R., Munhoz, C. D., Hu, C. K., Tran, K. V., Miguel, Z. D., et al. (2013). Glucocorticoid signaling in myeloid cells worsens acute CNS injury and inflammation. Journal of Neuroscience, 33( 18), 7877-7889. doi:10.1523/JNEUROSCI.4705-12.2013
    • NLM

      Sorrells SF, Caso JR, Munhoz CD, Hu CK, Tran KV, Miguel ZD, Chien BY, Sapolsky RM. Glucocorticoid signaling in myeloid cells worsens acute CNS injury and inflammation [Internet]. Journal of Neuroscience. 2013 ; 33( 18): 7877-7889.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1523/JNEUROSCI.4705-12.2013
    • Vancouver

      Sorrells SF, Caso JR, Munhoz CD, Hu CK, Tran KV, Miguel ZD, Chien BY, Sapolsky RM. Glucocorticoid signaling in myeloid cells worsens acute CNS injury and inflammation [Internet]. Journal of Neuroscience. 2013 ; 33( 18): 7877-7889.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1523/JNEUROSCI.4705-12.2013
  • Source: PLOS ONE. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      HERNANDES, Marina S. et al. Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration. PLOS ONE, v. 8, n. 9, p. 1-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0075532. Acesso em: 02 nov. 2024.
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      Hernandes, M. S., Santos, G. D. R., Café-Mendes, C. C., Lima, L. de S., Scavone, C., Munhoz, C. D., & Britto, L. R. G. de. (2013). Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration. PLOS ONE, 8( 9), 1-13. doi:10.1371/journal.pone.0075532
    • NLM

      Hernandes MS, Santos GDR, Café-Mendes CC, Lima L de S, Scavone C, Munhoz CD, Britto LRG de. Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration [Internet]. PLOS ONE. 2013 ; 8( 9): 1-13.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0075532
    • Vancouver

      Hernandes MS, Santos GDR, Café-Mendes CC, Lima L de S, Scavone C, Munhoz CD, Britto LRG de. Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration [Internet]. PLOS ONE. 2013 ; 8( 9): 1-13.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pone.0075532

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