A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Nathanne S. et al. NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. 2016, Anais.. Orlando: AHA, 2016. . Acesso em: 10 nov. 2024.
APA
Ferreira, N. S., Bruder-Nascimento, T., Pereira, C. A., Zanotto, C. Z., Prado, D. S., Alves-Filho, J. C., et al. (2016). NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. In Final Program. Orlando: AHA.
NLM
Ferreira NS, Bruder-Nascimento T, Pereira CA, Zanotto CZ, Prado DS, Alves-Filho JC, Carlos D, Tostes R de CA. NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. Final Program. 2016 ;[citado 2024 nov. 10 ]
Vancouver
Ferreira NS, Bruder-Nascimento T, Pereira CA, Zanotto CZ, Prado DS, Alves-Filho JC, Carlos D, Tostes R de CA. NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. Final Program. 2016 ;[citado 2024 nov. 10 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Camila A. et al. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. 2016, Anais.. Orlando: AHA, 2016. . Acesso em: 10 nov. 2024.
APA
Pereira, C. A., Ferreira, N. S., Zanotto, C. Z., Carlos, D., & Tostes, R. de C. A. (2016). NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. In Final Program. Orlando: AHA.
NLM
Pereira CA, Ferreira NS, Zanotto CZ, Carlos D, Tostes R de CA. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. Final Program. 2016 ;[citado 2024 nov. 10 ]
Vancouver
Pereira CA, Ferreira NS, Zanotto CZ, Carlos D, Tostes R de CA. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. Final Program. 2016 ;[citado 2024 nov. 10 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NEVES, Karla B. et al. Chemerin decreases vascular insulin signaling. 2014, Anais.. San Francisco: AHA, 2014. Disponível em: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf. Acesso em: 10 nov. 2024.
APA
Neves, K. B., Lobato, N. de S., Lopes, R. A., Mestriner, F. L. A. C., Oliveira, A. M. de, & Passaglia, R. de C. A. T. (2014). Chemerin decreases vascular insulin signaling. In Final Program. San Francisco: AHA. Recuperado de http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
NLM
Neves KB, Lobato N de S, Lopes RA, Mestriner FLAC, Oliveira AM de, Passaglia R de CAT. Chemerin decreases vascular insulin signaling [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Vancouver
Neves KB, Lobato N de S, Lopes RA, Mestriner FLAC, Oliveira AM de, Passaglia R de CAT. Chemerin decreases vascular insulin signaling [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
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ABNT
LOPES, Rheure A. et al. Downregulation of vascular factor‐erythroid 2‐related factor‐2 and associated antioxidant enzymes in hypertension. 2014, Anais.. San Francisco: AHA, 2014. Disponível em: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf. Acesso em: 10 nov. 2024.
APA
Lopes, R. A., Neves, K. B., Montezano, A., Passaglia, R. de C. A. T., & Touyz, R. (2014). Downregulation of vascular factor‐erythroid 2‐related factor‐2 and associated antioxidant enzymes in hypertension. In Final Program. San Francisco: AHA. Recuperado de http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
NLM
Lopes RA, Neves KB, Montezano A, Passaglia R de CAT, Touyz R. Downregulation of vascular factor‐erythroid 2‐related factor‐2 and associated antioxidant enzymes in hypertension [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Vancouver
Lopes RA, Neves KB, Montezano A, Passaglia R de CAT, Touyz R. Downregulation of vascular factor‐erythroid 2‐related factor‐2 and associated antioxidant enzymes in hypertension [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Camila A. et al. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. 2014, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2014. . Acesso em: 10 nov. 2024.
APA
Pereira, C. A., Ferreira, N. S., Mestriner, F. L. A. C., Resstel, L. B. M., Carneiro, F. S., & Tostes, R. de C. A. (2014). Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. In Final Program. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Pereira CA, Ferreira NS, Mestriner FLAC, Resstel LBM, Carneiro FS, Tostes R de CA. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Final Program. 2014 ;[citado 2024 nov. 10 ]
Vancouver
Pereira CA, Ferreira NS, Mestriner FLAC, Resstel LBM, Carneiro FS, Tostes R de CA. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Final Program. 2014 ;[citado 2024 nov. 10 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NEVES, Karla B. et al. Chemerin, a novel adipokine, regulates human vascular cell function through redox‐sensitive processes involving NADPH oxidase 1/4 and endothelial nitric oxide sinthase. 2014, Anais.. San Francisco: AHA, 2014. Disponível em: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf. Acesso em: 10 nov. 2024.
APA
Neves, K. B., Lopes, R. A., Nguyen, A. D., Lobato, N. S., Oliveira, A. M. de, Montezano, A. C., et al. (2014). Chemerin, a novel adipokine, regulates human vascular cell function through redox‐sensitive processes involving NADPH oxidase 1/4 and endothelial nitric oxide sinthase. In Final Program. San Francisco: AHA. Recuperado de http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
NLM
Neves KB, Lopes RA, Nguyen AD, Lobato NS, Oliveira AM de, Montezano AC, Passaglia R de CAT, Touyz RM. Chemerin, a novel adipokine, regulates human vascular cell function through redox‐sensitive processes involving NADPH oxidase 1/4 and endothelial nitric oxide sinthase [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Vancouver
Neves KB, Lopes RA, Nguyen AD, Lobato NS, Oliveira AM de, Montezano AC, Passaglia R de CAT, Touyz RM. Chemerin, a novel adipokine, regulates human vascular cell function through redox‐sensitive processes involving NADPH oxidase 1/4 and endothelial nitric oxide sinthase [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVON, Vânia Cláudia et al. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis. 2014, Anais.. San Francisco: AHA, 2014. Disponível em: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf. Acesso em: 10 nov. 2024.
APA
Olivon, V. C., Ferreira, R. G., Mestriner, F. L. A. C., Alves Filho, J. C. F., Cunha, F. de Q., & Passaglia, R. de C. A. T. (2014). Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis. In Final Program. San Francisco: AHA. Recuperado de http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
NLM
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Vancouver
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Rheure A. et al. Castration impairs rat internal pudendal artery reactivity and structure. 2014, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2014. . Acesso em: 10 nov. 2024.
APA
Lopes, R. A., Neves, K. B., Silva, M., Olivon, V. C., Ruginsk, S., Ramalho, L. N. Z., et al. (2014). Castration impairs rat internal pudendal artery reactivity and structure. In Final Program. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Lopes RA, Neves KB, Silva M, Olivon VC, Ruginsk S, Ramalho LNZ, Rodrigues JA, Tostes R de CA, Carneiro FS. Castration impairs rat internal pudendal artery reactivity and structure. Final Program. 2014 ;[citado 2024 nov. 10 ]
Vancouver
Lopes RA, Neves KB, Silva M, Olivon VC, Ruginsk S, Ramalho LNZ, Rodrigues JA, Tostes R de CA, Carneiro FS. Castration impairs rat internal pudendal artery reactivity and structure. Final Program. 2014 ;[citado 2024 nov. 10 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Nathanne et al. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. 2014, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2014. . Acesso em: 10 nov. 2024.
APA
Ferreira, N., Cau, S. B., Manzato, C. P., Silva, M. A., Carneiro, F. S., & Tostes, R. de C. A. (2014). G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. In Final Program. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Ferreira N, Cau SB, Manzato CP, Silva MA, Carneiro FS, Tostes R de CA. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Final Program. 2014 ;[citado 2024 nov. 10 ]
Vancouver
Ferreira N, Cau SB, Manzato CP, Silva MA, Carneiro FS, Tostes R de CA. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Final Program. 2014 ;[citado 2024 nov. 10 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAU, Stefany B. et al. Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. 2012, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 10 nov. 2024.
APA
Cau, S. B., Silva, M. A., Coimbra, T. M., Alves Filho, J. C., Zamboni, D. S., & Tostes, R. de C. A. (2012). Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. In Final Program. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Cau SB, Silva MA, Coimbra TM, Alves Filho JC, Zamboni DS, Tostes R de CA. Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. Final Program. 2012 ;[citado 2024 nov. 10 ]
Vancouver
Cau SB, Silva MA, Coimbra TM, Alves Filho JC, Zamboni DS, Tostes R de CA. Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. Final Program. 2012 ;[citado 2024 nov. 10 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRUDER-NASCIMENTO, Thiago et al. Angiotensin II induces Fat1 overexpression and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation. 2012, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 10 nov. 2024.
APA
Bruder-Nascimento, T., Chinnasamy, P., Riascos-Bernal, D., Tostes, R. de C. A., & Sibinga, N. E. (2012). Angiotensin II induces Fat1 overexpression and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation. In Final Program. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Bruder-Nascimento T, Chinnasamy P, Riascos-Bernal D, Tostes R de CA, Sibinga NE. Angiotensin II induces Fat1 overexpression and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation. Final Program. 2012 ;[citado 2024 nov. 10 ]
Vancouver
Bruder-Nascimento T, Chinnasamy P, Riascos-Bernal D, Tostes R de CA, Sibinga NE. Angiotensin II induces Fat1 overexpression and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation. Final Program. 2012 ;[citado 2024 nov. 10 ]