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PAULINI, Janaina et al. Blood Pressure Variability: An Independent Marker to Morbimortality. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 17 nov. 2024. , 2013
APA
Paulini, J., Nishi, F., Jorge, L., Pires, R., Krieger, E. M., & Irigoyen, M. C. (2013). Blood Pressure Variability: An Independent Marker to Morbimortality. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo.
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ALVES, M. J. N. N. et al. Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients. Hypertension, v. 60, n. 3, p. 669-676, 2012Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.112.195776. Acesso em: 17 nov. 2024.
APA
Alves, M. J. N. N., Santos, M. R. dos, Nobre, T. S., Martinez, D. G., Pereira Barretto, A. C., Brum, P. C., et al. (2012). Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients. Hypertension, 60( 3), 669-676. doi:10.1161/HYPERTENSIONAHA.112.195776
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FERNANDES, Tiago et al. Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7). Hypertension, v. 58, n. 2, p. 182-189, 2011Tradução . . Disponível em: http://hyper.ahajournals.org/content/early/2011/06/27/HYPERTENSIONAHA.110.168252.full.pdf+html. Acesso em: 17 nov. 2024.
APA
Fernandes, T., Hashimoto, N. Y., Magalhaes, F. C., Fernandes, F. B., Casarini, D. E., Carmona, A. K., et al. (2011). Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7). Hypertension, 58( 2), 182-189. doi:10.1161/HYPERTENSIONAHA.110.168252
NLM
Fernandes T, Hashimoto NY, Magalhaes FC, Fernandes FB, Casarini DE, Carmona AK, Krieger JE, Phillips MI, Oliveira EM. Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7) [Internet]. Hypertension. 2011 ; 58( 2): 182-189.[citado 2024 nov. 17 ] Available from: http://hyper.ahajournals.org/content/early/2011/06/27/HYPERTENSIONAHA.110.168252.full.pdf+html
Vancouver
Fernandes T, Hashimoto NY, Magalhaes FC, Fernandes FB, Casarini DE, Carmona AK, Krieger JE, Phillips MI, Oliveira EM. Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7) [Internet]. Hypertension. 2011 ; 58( 2): 182-189.[citado 2024 nov. 17 ] Available from: http://hyper.ahajournals.org/content/early/2011/06/27/HYPERTENSIONAHA.110.168252.full.pdf+html
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MARTINEZ, Daniel G. et al. Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients. Hypertension, v. 58, n. 6, p. 1049-1056, 2011Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.111.176644. Acesso em: 17 nov. 2024.
APA
Martinez, D. G., Nicolau, J. C., Lage, R. L., Toschi-Dias, E., de Matos, L. D. N. J., Alves, M. J. N. N., et al. (2011). Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients. Hypertension, 58( 6), 1049-1056. doi:10.1161/HYPERTENSIONAHA.111.176644
NLM
Martinez DG, Nicolau JC, Lage RL, Toschi-Dias E, de Matos LDNJ, Alves MJNN, Trombetta IC, Dias da Silva VJ, Middlekauff HR, Negrão CE, Rondon MU. Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients [Internet]. Hypertension. 2011 ; 58( 6): 1049-1056.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.176644
Vancouver
Martinez DG, Nicolau JC, Lage RL, Toschi-Dias E, de Matos LDNJ, Alves MJNN, Trombetta IC, Dias da Silva VJ, Middlekauff HR, Negrão CE, Rondon MU. Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients [Internet]. Hypertension. 2011 ; 58( 6): 1049-1056.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.176644
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FANELLI, Camilla e ZATZ, Roberto. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html. Acesso em: 17 nov. 2024. , 2011
APA
Fanelli, C., & Zatz, R. (2011). Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. doi:10.1161/HYPERTENSIONAHA.110.167775
NLM
Fanelli C, Zatz R. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] [Internet]. Hypertension. 2011 ; 57( 3): 373-374.[citado 2024 nov. 17 ] Available from: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html
Vancouver
Fanelli C, Zatz R. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] [Internet]. Hypertension. 2011 ; 57( 3): 373-374.[citado 2024 nov. 17 ] Available from: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html
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GIACHINI, Fernanda R. et al. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, v. 57, n. 3, p. 655-663, 2011Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327. Acesso em: 17 nov. 2024.
APA
Giachini, F. R., Lima, V. V., Carneiro, F. S., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, 57( 3), 655-663. doi:10.1161/HYPERTENSIONAHA.110.164327
NLM
Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
Vancouver
Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
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PEDROSA, Rodrigo P. et al. Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension. Hypertension, v. 58, n. 5, p. 811-817, 2011Tradução . . Disponível em: https://doi.org/10.1161/\200BHYPERTENSIONAHA.111.179788. Acesso em: 17 nov. 2024.
APA
Pedrosa, R. P., Drager, L. F., Gonzaga, C. C., Sousa, M. G., Paula, L. K. G. de, Amaro, A. C. S., et al. (2011). Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension. Hypertension, 58( 5), 811-817. doi:10.1161/\200BHYPERTENSIONAHA.111.179788
NLM
Pedrosa RP, Drager LF, Gonzaga CC, Sousa MG, Paula LKG de, Amaro ACS, Amodeo C, Bortolotto LA, Krieger EM, Bradley TD, Lorenzi-Filho G. Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension [Internet]. Hypertension. 2011 ; 58( 5): 811-817.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/\200BHYPERTENSIONAHA.111.179788
Vancouver
Pedrosa RP, Drager LF, Gonzaga CC, Sousa MG, Paula LKG de, Amaro ACS, Amodeo C, Bortolotto LA, Krieger EM, Bradley TD, Lorenzi-Filho G. Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension [Internet]. Hypertension. 2011 ; 58( 5): 811-817.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/\200BHYPERTENSIONAHA.111.179788
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SANDRIM, Valéria C. et al. Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension, v. 52, n. 2, p. 402-407, 2008Tradução . . Acesso em: 17 nov. 2024.
APA
Sandrim, V. C., Palei, A. C. T., Metzger, I. F., Gomes, V. A., Cavalli, R. C., & Tanus-Santos, J. E. (2008). Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension, 52( 2), 402-407.
NLM
Sandrim VC, Palei ACT, Metzger IF, Gomes VA, Cavalli RC, Tanus-Santos JE. Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension. 2008 ; 52( 2): 402-407.[citado 2024 nov. 17 ]
Vancouver
Sandrim VC, Palei ACT, Metzger IF, Gomes VA, Cavalli RC, Tanus-Santos JE. Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension. 2008 ; 52( 2): 402-407.[citado 2024 nov. 17 ]
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PEREIRA, Tiago Veiga et al. Meta-Analysis of the Association of 4 Angiotensinogen Polymorphisms With Essential Hypertension: A Role Beyond M235T?. Hypertension, v. 51, n. 3, p. 778-783, 2008Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.107.100370. Acesso em: 17 nov. 2024.
APA
Pereira, T. V., Nunes, A. C. F., Rudnicki, M., Krieger, J. E., Yamada, Y., & Pereira, A. C. (2008). Meta-Analysis of the Association of 4 Angiotensinogen Polymorphisms With Essential Hypertension: A Role Beyond M235T? Hypertension, 51( 3), 778-783. doi:10.1161/HYPERTENSIONAHA.107.100370
NLM
Pereira TV, Nunes ACF, Rudnicki M, Krieger JE, Yamada Y, Pereira AC. Meta-Analysis of the Association of 4 Angiotensinogen Polymorphisms With Essential Hypertension: A Role Beyond M235T? [Internet]. Hypertension. 2008 ; 51( 3): 778-783.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.107.100370
Vancouver
Pereira TV, Nunes ACF, Rudnicki M, Krieger JE, Yamada Y, Pereira AC. Meta-Analysis of the Association of 4 Angiotensinogen Polymorphisms With Essential Hypertension: A Role Beyond M235T? [Internet]. Hypertension. 2008 ; 51( 3): 778-783.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.107.100370
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ABNT
SIVIERI JÚNIOR, Disney Oliver et al. Potentiation of bradykinin effect by angiotensin-converting enzyme inhibition does not correlate with angiotensin-converting enzyme activity in the rat mesenteric arteries. Hypertension, v. 50, p. 110-115, 2007Tradução . . Acesso em: 17 nov. 2024.
APA
Sivieri Júnior, D. O., Bispo-da-Silva, L. B., Oliveira, E. B. de, Resende, A. C., & Salgado, M. C. de O. (2007). Potentiation of bradykinin effect by angiotensin-converting enzyme inhibition does not correlate with angiotensin-converting enzyme activity in the rat mesenteric arteries. Hypertension, 50, 110-115.
NLM
Sivieri Júnior DO, Bispo-da-Silva LB, Oliveira EB de, Resende AC, Salgado MC de O. Potentiation of bradykinin effect by angiotensin-converting enzyme inhibition does not correlate with angiotensin-converting enzyme activity in the rat mesenteric arteries. Hypertension. 2007 ; 50 110-115.[citado 2024 nov. 17 ]
Vancouver
Sivieri Júnior DO, Bispo-da-Silva LB, Oliveira EB de, Resende AC, Salgado MC de O. Potentiation of bradykinin effect by angiotensin-converting enzyme inhibition does not correlate with angiotensin-converting enzyme activity in the rat mesenteric arteries. Hypertension. 2007 ; 50 110-115.[citado 2024 nov. 17 ]
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CALLERA, Glaucia E. et al. Aldosterone activates vascular p38MAP kinase and NADPH oxidase Via c-Src. Hypertension, v. 45, n. 4, p. 773-779, 2005Tradução . . Acesso em: 17 nov. 2024.
APA
Callera, G. E., Touyz, R. M., Passaglia, R. de C. A. T., Yogi, A., He, Y., Malkinson, S., & Schiffrin, E. L. (2005). Aldosterone activates vascular p38MAP kinase and NADPH oxidase Via c-Src. Hypertension, 45( 4), 773-779.
NLM
Callera GE, Touyz RM, Passaglia R de CAT, Yogi A, He Y, Malkinson S, Schiffrin EL. Aldosterone activates vascular p38MAP kinase and NADPH oxidase Via c-Src. Hypertension. 2005 ; 45( 4): 773-779.[citado 2024 nov. 17 ]
Vancouver
Callera GE, Touyz RM, Passaglia R de CAT, Yogi A, He Y, Malkinson S, Schiffrin EL. Aldosterone activates vascular p38MAP kinase and NADPH oxidase Via c-Src. Hypertension. 2005 ; 45( 4): 773-779.[citado 2024 nov. 17 ]
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ABNT
CALLERA, Glaucia E. et al. c-Src-Dependent nongenomic signaling responses to aldosterone are increased in vascular myocytes from spontaneously hypertensive rats. Hypertension, v. 46, n. 4, p. 1032-1038, 2005Tradução . . Acesso em: 17 nov. 2024.
APA
Callera, G. E., Montezano, A. C. I., Yogi, A., Passaglia, R. de C. A. T., He, Y., Schiffrin, E. L., & Touyz, R. M. (2005). c-Src-Dependent nongenomic signaling responses to aldosterone are increased in vascular myocytes from spontaneously hypertensive rats. Hypertension, 46( 4), 1032-1038.
NLM
Callera GE, Montezano ACI, Yogi A, Passaglia R de CAT, He Y, Schiffrin EL, Touyz RM. c-Src-Dependent nongenomic signaling responses to aldosterone are increased in vascular myocytes from spontaneously hypertensive rats. Hypertension. 2005 ; 46( 4): 1032-1038.[citado 2024 nov. 17 ]
Vancouver
Callera GE, Montezano ACI, Yogi A, Passaglia R de CAT, He Y, Schiffrin EL, Touyz RM. c-Src-Dependent nongenomic signaling responses to aldosterone are increased in vascular myocytes from spontaneously hypertensive rats. Hypertension. 2005 ; 46( 4): 1032-1038.[citado 2024 nov. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Alexandre C. et al. Beta2 adrenoceptor functional gene variants, obesity, and blood pressure level interactions in the general population. Hypertension, v. 42, p. 685-692, 2003Tradução . . Acesso em: 17 nov. 2024.
APA
Pereira, A. C., Floriano, M. S., Mota, G. F. A., Cunha, R. S., Herkenhoff, F. L., Mill, J. G., & Krieger, J. E. (2003). Beta2 adrenoceptor functional gene variants, obesity, and blood pressure level interactions in the general population. Hypertension, 42, 685-692.
NLM
Pereira AC, Floriano MS, Mota GFA, Cunha RS, Herkenhoff FL, Mill JG, Krieger JE. Beta2 adrenoceptor functional gene variants, obesity, and blood pressure level interactions in the general population. Hypertension. 2003 ; 42 685-692.[citado 2024 nov. 17 ]
Vancouver
Pereira AC, Floriano MS, Mota GFA, Cunha RS, Herkenhoff FL, Mill JG, Krieger JE. Beta2 adrenoceptor functional gene variants, obesity, and blood pressure level interactions in the general population. Hypertension. 2003 ; 42 685-692.[citado 2024 nov. 17 ]
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ABNT
SOUZA, Hugo Celso Dutra de et al. SR141716A-sensitive enhancement of ET-1 hypotensive effect by chronic NOS inhibition. Hypertension, v. 42, p. 802-805, 2003Tradução . . Acesso em: 17 nov. 2024.
APA
Souza, H. C. D. de, Salgado, H. C., Ballejo, G., & Salgado, M. C. de O. (2003). SR141716A-sensitive enhancement of ET-1 hypotensive effect by chronic NOS inhibition. Hypertension, 42, 802-805.
NLM
Souza HCD de, Salgado HC, Ballejo G, Salgado MC de O. SR141716A-sensitive enhancement of ET-1 hypotensive effect by chronic NOS inhibition. Hypertension. 2003 ; 42 802-805.[citado 2024 nov. 17 ]
Vancouver
Souza HCD de, Salgado HC, Ballejo G, Salgado MC de O. SR141716A-sensitive enhancement of ET-1 hypotensive effect by chronic NOS inhibition. Hypertension. 2003 ; 42 802-805.[citado 2024 nov. 17 ]
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ABNT
MARQUES, Georgia D. M. et al. N-domain angiotensin I-converting enzyme with 80 kDa as a possible genetic marker of hypertension. Hypertension, v. 42, p. 693-701, 2003Tradução . . Acesso em: 17 nov. 2024.
APA
Marques, G. D. M., Quinto, B. M. R., Plavinik, F. L., Krieger, J. E., Marson, O., & Casarini, D. E. (2003). N-domain angiotensin I-converting enzyme with 80 kDa as a possible genetic marker of hypertension. Hypertension, 42, 693-701.
NLM
Marques GDM, Quinto BMR, Plavinik FL, Krieger JE, Marson O, Casarini DE. N-domain angiotensin I-converting enzyme with 80 kDa as a possible genetic marker of hypertension. Hypertension. 2003 ; 42 693-701.[citado 2024 nov. 17 ]
Vancouver
Marques GDM, Quinto BMR, Plavinik FL, Krieger JE, Marson O, Casarini DE. N-domain angiotensin I-converting enzyme with 80 kDa as a possible genetic marker of hypertension. Hypertension. 2003 ; 42 693-701.[citado 2024 nov. 17 ]
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ABNT
CALLERA, Gláucia Elena e VARANDA, Wamberto Antônio e BENDHACK, Lusiane Maria. 'Ca POT.2+' influx is increased in 2-kidney, 1-clip hypertensive rat aorta. Hypertension, v. 38, p. 592-596, 2001Tradução . . Acesso em: 17 nov. 2024.
APA
Callera, G. E., Varanda, W. A., & Bendhack, L. M. (2001). 'Ca POT.2+' influx is increased in 2-kidney, 1-clip hypertensive rat aorta. Hypertension, 38, 592-596.
NLM
Callera GE, Varanda WA, Bendhack LM. 'Ca POT.2+' influx is increased in 2-kidney, 1-clip hypertensive rat aorta. Hypertension. 2001 ; 38 592-596.[citado 2024 nov. 17 ]
Vancouver
Callera GE, Varanda WA, Bendhack LM. 'Ca POT.2+' influx is increased in 2-kidney, 1-clip hypertensive rat aorta. Hypertension. 2001 ; 38 592-596.[citado 2024 nov. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Valdo José Dias et al. Antihypertensive action of amiodarone in spontaneously hypertensive rats. Hypertension, v. 38, n. part. 2, p. 597-601, 2001Tradução . . Acesso em: 17 nov. 2024.
APA
Silva, V. J. D., Viana, P. C. C., Alves, R. de M., Salgado, H. C., Montano, N., & Fazan Junior, R. (2001). Antihypertensive action of amiodarone in spontaneously hypertensive rats. Hypertension, 38( part. 2), 597-601.
NLM
Silva VJD, Viana PCC, Alves R de M, Salgado HC, Montano N, Fazan Junior R. Antihypertensive action of amiodarone in spontaneously hypertensive rats. Hypertension. 2001 ; 38( part. 2): 597-601.[citado 2024 nov. 17 ]
Vancouver
Silva VJD, Viana PCC, Alves R de M, Salgado HC, Montano N, Fazan Junior R. Antihypertensive action of amiodarone in spontaneously hypertensive rats. Hypertension. 2001 ; 38( part. 2): 597-601.[citado 2024 nov. 17 ]
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ABNT
RABELO, Fábio A. W. et al. Nonendothelial NO blunts sympathetic response of normothensive rats but not of SHR. Hypertension, v. 38, p. part. II 565-568, 2001Tradução . . Acesso em: 17 nov. 2024.
APA
Rabelo, F. A. W., Russo, E. M., Salgado, M. C. de O., & Coelho, E. B. (2001). Nonendothelial NO blunts sympathetic response of normothensive rats but not of SHR. Hypertension, 38, part. II 565-568.
NLM
Rabelo FAW, Russo EM, Salgado MC de O, Coelho EB. Nonendothelial NO blunts sympathetic response of normothensive rats but not of SHR. Hypertension. 2001 ; 38 part. II 565-568.[citado 2024 nov. 17 ]
Vancouver
Rabelo FAW, Russo EM, Salgado MC de O, Coelho EB. Nonendothelial NO blunts sympathetic response of normothensive rats but not of SHR. Hypertension. 2001 ; 38 part. II 565-568.[citado 2024 nov. 17 ]
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ABNT
SILVA, Alexandre Alves da e HEIMANN, Joel Cláudio. Dietary salt overload during pregnancy and lactation increases blood pressure, and decreases salt sensitivity and the modulation of the renin-angiotensin system in response to salt in the adult offspring. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 17 nov. 2024. , 1999
APA
Silva, A. A. da, & Heimann, J. C. (1999). Dietary salt overload during pregnancy and lactation increases blood pressure, and decreases salt sensitivity and the modulation of the renin-angiotensin system in response to salt in the adult offspring. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo.
NLM
Silva AA da, Heimann JC. Dietary salt overload during pregnancy and lactation increases blood pressure, and decreases salt sensitivity and the modulation of the renin-angiotensin system in response to salt in the adult offspring. Hypertension. 1999 ; 34( 2): 365 res.P-140.[citado 2024 nov. 17 ]
Vancouver
Silva AA da, Heimann JC. Dietary salt overload during pregnancy and lactation increases blood pressure, and decreases salt sensitivity and the modulation of the renin-angiotensin system in response to salt in the adult offspring. Hypertension. 1999 ; 34( 2): 365 res.P-140.[citado 2024 nov. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HILBERS, Urte et al. Local renin-angiotensin system is involved in 'K POT. +'-induced aldosterone secretion from human adrenocortical NCI-H295 cells. Hypertension, v. 33, p. 1025-1030, 1999Tradução . . Acesso em: 17 nov. 2024.
APA
Hilbers, U., Peters, J., Bornstein, S. R., Corrêa, F. M. de A., Jöhren, O., Saavedra, J. M., & Ehrhart-Bornstein, M. (1999). Local renin-angiotensin system is involved in 'K POT. +'-induced aldosterone secretion from human adrenocortical NCI-H295 cells. Hypertension, 33, 1025-1030.
NLM
Hilbers U, Peters J, Bornstein SR, Corrêa FM de A, Jöhren O, Saavedra JM, Ehrhart-Bornstein M. Local renin-angiotensin system is involved in 'K POT. +'-induced aldosterone secretion from human adrenocortical NCI-H295 cells. Hypertension. 1999 ; 33 1025-1030.[citado 2024 nov. 17 ]
Vancouver
Hilbers U, Peters J, Bornstein SR, Corrêa FM de A, Jöhren O, Saavedra JM, Ehrhart-Bornstein M. Local renin-angiotensin system is involved in 'K POT. +'-induced aldosterone secretion from human adrenocortical NCI-H295 cells. Hypertension. 1999 ; 33 1025-1030.[citado 2024 nov. 17 ]
