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Artigo de periodico
Methylation status of LDLR, PCSK9 and LDLRAP1 is associated with cardiovascular events in familial hypercholesterolemia (2024)
Marçal, Elisangela da Silva Rodrigues; Borges, Jéssica Bassani ; Bastos, Gisele Medeiros ; Hirata, Thiago Dominguez Crespo ; Oliveira, Victor Fernandes de ; Gonçalves, Rodrigo M ; Faludi, André Arpad; França, Joao Italo Dias ; Silva, Daiana Vitor de Oliveira; Malaquias, Vanessa Barbosa ; Luchessi, Andre Ducati ; Silbiger, Vivian Nogueira ; Nakazone, Marcelo Arruda ; Carmo, Tayanne Silva do ; Souza, Dorotéia Rossi Silva; Sampaio, Marcelo Ferraz; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Epigenomics. Unidades: FCF, ICB
Subjects: HIPERCOLESTEROLEMIA, METILAÇÃO
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ABNT
MARÇAL, Elisangela da Silva Rodrigues et al. Methylation status of LDLR, PCSK9 and LDLRAP1 is associated with cardiovascular events in familial hypercholesterolemia. Epigenomics, 2024Tradução . . Disponível em: https://dx.doi.org/10.1080/17501911.2024.2351792. Acesso em: 09 nov. 2024.
APA
Marçal, E. da S. R., Borges, J. B., Bastos, G. M., Hirata, T. D. C., Oliveira, V. F. de, Gonçalves, R. M., et al. (2024). Methylation status of LDLR, PCSK9 and LDLRAP1 is associated with cardiovascular events in familial hypercholesterolemia. Epigenomics. doi:10.1080/17501911.2024.2351792
NLM
Marçal E da SR, Borges JB, Bastos GM, Hirata TDC, Oliveira VF de, Gonçalves RM, Faludi AA, França JID, Silva DV de O, Malaquias VB, Luchessi AD, Silbiger VN, Nakazone MA, Carmo TS do, Souza DRS, Sampaio MF, Hirata RDC, Hirata MH. Methylation status of LDLR, PCSK9 and LDLRAP1 is associated with cardiovascular events in familial hypercholesterolemia [Internet]. Epigenomics. 2024 ;[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.1080/17501911.2024.2351792
Vancouver
Marçal E da SR, Borges JB, Bastos GM, Hirata TDC, Oliveira VF de, Gonçalves RM, Faludi AA, França JID, Silva DV de O, Malaquias VB, Luchessi AD, Silbiger VN, Nakazone MA, Carmo TS do, Souza DRS, Sampaio MF, Hirata RDC, Hirata MH. Methylation status of LDLR, PCSK9 and LDLRAP1 is associated with cardiovascular events in familial hypercholesterolemia [Internet]. Epigenomics. 2024 ;[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.1080/17501911.2024.2351792
Artigo de periodico
Effects of LDLR variants rs5928, rs750518671 and rs879254797 on protein structure and functional activity in HepG2 cells transfected with CRISPR/ Cas9 constructs (2024)
Mori, Augusto Akira ; Malaquias, Vanessa Barbosa ; Bonjour, Kennedy ; Ferreira, Glaucio Monteiro ; Bortolin, Raul Hernandes ; Borges, Jéssica Bassani ; Oliveira, Victor Fernandes de ; Gonçalves, Rodrigo Marques; Faludi, Andre Arpad; Bastos, Gisele Monteiro; Thurow, Helena; Sampaio, Marcelo Ferraz; Ciconelli, Rozana Mesquita; Cury, Adriano Namo; Fajardo, Cristina Moreno ; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Gene. Unidades: FCF, ICB
Subjects: HIPERCOLESTEROLEMIA, RECEPTORES DE LDL
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MORI, Augusto Akira et al. Effects of LDLR variants rs5928, rs750518671 and rs879254797 on protein structure and functional activity in HepG2 cells transfected with CRISPR/ Cas9 constructs. Gene, v. 890, p. 1-12 art. 147821, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.gene.2023.147821. Acesso em: 09 nov. 2024.
APA
Mori, A. A., Malaquias, V. B., Bonjour, K., Ferreira, G. M., Bortolin, R. H., Borges, J. B., et al. (2024). Effects of LDLR variants rs5928, rs750518671 and rs879254797 on protein structure and functional activity in HepG2 cells transfected with CRISPR/ Cas9 constructs. Gene, 890, 1-12 art. 147821. doi:10.1016/j.gene.2023.147821
NLM
Mori AA, Malaquias VB, Bonjour K, Ferreira GM, Bortolin RH, Borges JB, Oliveira VF de, Gonçalves RM, Faludi AA, Bastos GM, Thurow H, Sampaio MF, Ciconelli RM, Cury AN, Fajardo CM, Hirata RDC, Hirata MH. Effects of LDLR variants rs5928, rs750518671 and rs879254797 on protein structure and functional activity in HepG2 cells transfected with CRISPR/ Cas9 constructs [Internet]. Gene. 2024 ; 890 1-12 art. 147821.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.gene.2023.147821
Vancouver
Mori AA, Malaquias VB, Bonjour K, Ferreira GM, Bortolin RH, Borges JB, Oliveira VF de, Gonçalves RM, Faludi AA, Bastos GM, Thurow H, Sampaio MF, Ciconelli RM, Cury AN, Fajardo CM, Hirata RDC, Hirata MH. Effects of LDLR variants rs5928, rs750518671 and rs879254797 on protein structure and functional activity in HepG2 cells transfected with CRISPR/ Cas9 constructs [Internet]. Gene. 2024 ; 890 1-12 art. 147821.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.gene.2023.147821
Artigo de periodico
The impact of miR-155-5p on myotube differentiation: elucidating molecular targets in skeletal muscle disorders (2024)
Lopes, Letícia Oliveira; Cury, Sarah Santiloni ; Moraes, Diogo de ; Oliveira, Jakeline Santos; Oliveira, Grasieli de; Marques, Otavio Cabral ; Fernandez, Geysson Javier ; Hirata, Mario Hiroyuki ; Wang, Da Zhi ; Dal-Pai-Silva, Maeli ; Carvalho, Robson Francisco ; Freire, Paula Paccielli
Source: International Journal of Molecular Sciences. Unidades: FM, FCF, ICB
Subjects: MICRORNAS, DISTROFIA MUSCULAR
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ABNT
LOPES, Letícia Oliveira et al. The impact of miR-155-5p on myotube differentiation: elucidating molecular targets in skeletal muscle disorders. International Journal of Molecular Sciences, v. 25, p. 1-18, 2024Tradução . . Disponível em: https://dx.doi.org/10.3390/ijms25031777. Acesso em: 09 nov. 2024.
APA
Lopes, L. O., Cury, S. S., Moraes, D. de, Oliveira, J. S., Oliveira, G. de, Marques, O. C., et al. (2024). The impact of miR-155-5p on myotube differentiation: elucidating molecular targets in skeletal muscle disorders. International Journal of Molecular Sciences, 25, 1-18. doi:10.3390/ijms25031777
NLM
Lopes LO, Cury SS, Moraes D de, Oliveira JS, Oliveira G de, Marques OC, Fernandez GJ, Hirata MH, Wang DZ, Dal-Pai-Silva M, Carvalho RF, Freire PP. The impact of miR-155-5p on myotube differentiation: elucidating molecular targets in skeletal muscle disorders [Internet]. International Journal of Molecular Sciences. 2024 ; 25 1-18.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.3390/ijms25031777
Vancouver
Lopes LO, Cury SS, Moraes D de, Oliveira JS, Oliveira G de, Marques OC, Fernandez GJ, Hirata MH, Wang DZ, Dal-Pai-Silva M, Carvalho RF, Freire PP. The impact of miR-155-5p on myotube differentiation: elucidating molecular targets in skeletal muscle disorders [Internet]. International Journal of Molecular Sciences. 2024 ; 25 1-18.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.3390/ijms25031777
Artigo de periodico
Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions (2023)
Hamasaki, Mike Yoshio ; Mendes, Caroline; Batagello, Daniella Sabino ; Hirata, Mario Hiroyuki ; Britto, Luiz Roberto Giorgetti de ; Nogueira, Maria Inês
Source: NeuroReport. Unidades: FCF, ICB
Subjects: ANÓXIA, CÓRTEX CEREBRAL
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ABNT
HAMASAKI, Mike Yoshio et al. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, v. 34, n. 11, p. 575-582, 2023Tradução . . Disponível em: https://doi.org/10.1097/WNR.0000000000001927. Acesso em: 09 nov. 2024.
APA
Hamasaki, M. Y., Mendes, C., Batagello, D. S., Hirata, M. H., Britto, L. R. G. de, & Nogueira, M. I. (2023). Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, 34( 11), 575-582. doi:10.1097/WNR.0000000000001927
NLM
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Vancouver
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Artigo de periodico
The let-7b-5p, miR-326, and miR-125a-3p are associated with left ventricular systolic dysfunction in post-myocardial infarction (2023)
Komatsu, Raquel Costa Silva Dantas ; Cruz, Marina Sampaio; Freire, Paula Paccielli ; Diniz, Rosiane Viana Zuza; Bortolin, Raul Hernandes ; Marques, Otavio Cabral ; Souza, Kamilla Batista da Silva; Hirata, Mario Hiroyuki ; Hirata, Rosario Dominguez Crespo ; Reis, Bruna Zavarize ; Jurisica, Igor ; Silbiger, Vivian Nogueira ; Luchessi, Andre Ducati
Source: Frontiers in Cardiovascular Medicine. Unidades: FM, FCF, ICB
Subjects: INFARTO DO MIOCÁRDIO, MICRORNAS
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ABNT
KOMATSU, Raquel Costa Silva Dantas et al. The let-7b-5p, miR-326, and miR-125a-3p are associated with left ventricular systolic dysfunction in post-myocardial infarction. Frontiers in Cardiovascular Medicine, v. 10, p. 1-11 art. 1151855, 2023Tradução . . Disponível em: https://dx.doi.org/10.3389/fcvm.2023.1151855. Acesso em: 09 nov. 2024.
APA
Komatsu, R. C. S. D., Cruz, M. S., Freire, P. P., Diniz, R. V. Z., Bortolin, R. H., Marques, O. C., et al. (2023). The let-7b-5p, miR-326, and miR-125a-3p are associated with left ventricular systolic dysfunction in post-myocardial infarction. Frontiers in Cardiovascular Medicine, 10, 1-11 art. 1151855. doi:10.3389/fcvm.2023.1151855
NLM
Komatsu RCSD, Cruz MS, Freire PP, Diniz RVZ, Bortolin RH, Marques OC, Souza KB da S, Hirata MH, Hirata RDC, Reis BZ, Jurisica I, Silbiger VN, Luchessi AD. The let-7b-5p, miR-326, and miR-125a-3p are associated with left ventricular systolic dysfunction in post-myocardial infarction [Internet]. Frontiers in Cardiovascular Medicine. 2023 ; 10 1-11 art. 1151855.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.3389/fcvm.2023.1151855
Vancouver
Komatsu RCSD, Cruz MS, Freire PP, Diniz RVZ, Bortolin RH, Marques OC, Souza KB da S, Hirata MH, Hirata RDC, Reis BZ, Jurisica I, Silbiger VN, Luchessi AD. The let-7b-5p, miR-326, and miR-125a-3p are associated with left ventricular systolic dysfunction in post-myocardial infarction [Internet]. Frontiers in Cardiovascular Medicine. 2023 ; 10 1-11 art. 1151855.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.3389/fcvm.2023.1151855
Artigo de periodico
T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections (2023)
Santiago-Carvalho, Igor ; Almeida-Santos, Gislane ; Macêdo, Bruna de Gois ; Barbosa-Bomfim, Caio César ; Almeida, Fabricio Moreira ; Cione, Marcos Vinícios Pinheiro ; Vardam-Kaur, Trupti ; Masuda, Mia ; Dijk, Sarah Van ; Melo, Bruno Marcel Silva de ; Nascimento, Rogério Silva do; Souza, Rebeka da Conceição ; Peixoto-Rangel, Alba Lucínia ; Coutinho-Silva, Robson ; Lassounskaia, Elena ; Silva, Henrique Borges da ; Hirata, Mario Hiroyuki ; Alves Filho, José Carlos ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Source: Cell Reports. Unidades: FCF, FMRP, ICB
Subjects: IMUNOLOGIA, PNEUMOPATIAS, TUBERCULOSE PULMONAR, INFLUENZA, MODELOS ANIMAIS DE DOENÇAS, RATOS, QUIMIOCINAS, PULMÃO
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ABNT
SANTIAGO-CARVALHO, Igor et al. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Cell Reports, v. 42, n. 11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2023.113448. Acesso em: 09 nov. 2024.
APA
Santiago-Carvalho, I., Almeida-Santos, G., Macêdo, B. de G., Barbosa-Bomfim, C. C., Almeida, F. M., Cione, M. V. P., et al. (2023). T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Cell Reports, 42( 11). doi:10.1016/j.celrep.2023.113448
NLM
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Vardam-Kaur T, Masuda M, Dijk SV, Melo BMS de, Nascimento RS do, Souza R da C, Peixoto-Rangel AL, Coutinho-Silva R, Lassounskaia E, Silva HB da, Hirata MH, Alves Filho JC, Alvarez JM, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Cell Reports. 2023 ; 42( 11):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.celrep.2023.113448
Vancouver
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Vardam-Kaur T, Masuda M, Dijk SV, Melo BMS de, Nascimento RS do, Souza R da C, Peixoto-Rangel AL, Coutinho-Silva R, Lassounskaia E, Silva HB da, Hirata MH, Alves Filho JC, Alvarez JM, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Cell Reports. 2023 ; 42( 11):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.celrep.2023.113448
Artigo de periodico
Lipidomic analysis identified potential predictive biomarkers of statin response in subjects with Familial hypercholesterolemia (2023)
Cerda, Alvaro; Bortolin, Raul Hernandes ; Yoshinaga, Marcos Yukio ; Freitas, Renata Caroline Costa de ; Hernandez, Carolina Dagli; Borges, Jessica Bassani; Oliveira, Victor Fernandes de ; Gonçalves, Rodrigo Marques; Faludi, André Arpad; Bastos, Gisele Medeiros ; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Chemistry and Physics of Lipids. Unidades: FCF, ICB
Subjects: BIOMARCADORES, ESTATINAS, HIPERCOLESTEROLEMIA
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ABNT
CERDA, Alvaro et al. Lipidomic analysis identified potential predictive biomarkers of statin response in subjects with Familial hypercholesterolemia. Chemistry and Physics of Lipids, v. 257, p. 1-9 art. 105348, 2023Tradução . . Disponível em: https://dx.doi.org/10.1016/j.chemphyslip.2023.105348. Acesso em: 09 nov. 2024.
APA
Cerda, A., Bortolin, R. H., Yoshinaga, M. Y., Freitas, R. C. C. de, Hernandez, C. D., Borges, J. B., et al. (2023). Lipidomic analysis identified potential predictive biomarkers of statin response in subjects with Familial hypercholesterolemia. Chemistry and Physics of Lipids, 257, 1-9 art. 105348. doi:10.1016/j.chemphyslip.2023.105348
NLM
Cerda A, Bortolin RH, Yoshinaga MY, Freitas RCC de, Hernandez CD, Borges JB, Oliveira VF de, Gonçalves RM, Faludi AA, Bastos GM, Hirata RDC, Hirata MH. Lipidomic analysis identified potential predictive biomarkers of statin response in subjects with Familial hypercholesterolemia [Internet]. Chemistry and Physics of Lipids. 2023 ; 257 1-9 art. 105348.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.1016/j.chemphyslip.2023.105348
Vancouver
Cerda A, Bortolin RH, Yoshinaga MY, Freitas RCC de, Hernandez CD, Borges JB, Oliveira VF de, Gonçalves RM, Faludi AA, Bastos GM, Hirata RDC, Hirata MH. Lipidomic analysis identified potential predictive biomarkers of statin response in subjects with Familial hypercholesterolemia [Internet]. Chemistry and Physics of Lipids. 2023 ; 257 1-9 art. 105348.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.1016/j.chemphyslip.2023.105348
Artigo de periodico
Intranasal liposomal formulation of spike protein adjuvanted with CpG protects and boosts heterologous immunity of hACE2 transgenic mice to SARS-CoV-2 infection (2023)
Russo, Momtchilo ; Corrêa, Maria Cássia Mendes ; Lins, Bruna Bezerra ; Kersten, Victor; Pernambuco Filho, Paulo Castanho de Almeida ; Martins, Toni Ricardo ; Mendoza, Tânia Regina Tozetto ; Boas, Lucy Santos Vilas ; Gomes, Brisa Moreira; Dati, Lívia Mendonça Munhoz ; Duarte Neto, Amaro Nunes ; Reigado, Gustavo Roncoli ; Frederico, Ana Beatriz T. ; Cunha, Danielle R. de A. de Brito e ; Paula, Anderson Vicente de ; Silva, José Igor Gomes da ; Vasconcelos, Carlos Frederico Alves de ; Alcalde, Felipe Santiago Chambergo ; Dantas, Viviane Abreu Nunes Cerqueira ; Bom, Ana Paula Dinis Ano; Castilho, Leda R. ; Martins, Rodrigo A. P.; Hirata, Mario Hiroyuki ; Mirotti, Luciana
Source: Vaccines. Unidades: ICB, FM, EACH, FCF, BIOTECNOLOGIA
Subjects: IMUNIDADE, CAMUNDONGOS, LIPOSSOMOS, VACINAS VIRAIS, INFECÇÕES POR CORONAVIRUS, COVID-19, ADENOVÍRUS, PROTEÍNAS RECOMBINANTES, GLICOPROTEÍNAS, CAMUNDONGOS, VACINAÇÃO
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ABNT
RUSSO, Momtchilo et al. Intranasal liposomal formulation of spike protein adjuvanted with CpG protects and boosts heterologous immunity of hACE2 transgenic mice to SARS-CoV-2 infection. Vaccines, v. No 2023, n. 11, p. 01-17, 2023Tradução . . Disponível em: http://dx.doi.org/10.3390/vaccines11111732. Acesso em: 09 nov. 2024.
APA
Russo, M., Corrêa, M. C. M., Lins, B. B., Kersten, V., Pernambuco Filho, P. C. de A., Martins, T. R., et al. (2023). Intranasal liposomal formulation of spike protein adjuvanted with CpG protects and boosts heterologous immunity of hACE2 transgenic mice to SARS-CoV-2 infection. Vaccines, No 2023( 11), 01-17. doi:10.3390/vaccines11111732
NLM
Russo M, Corrêa MCM, Lins BB, Kersten V, Pernambuco Filho PC de A, Martins TR, Mendoza TRT, Boas LSV, Gomes BM, Dati LMM, Duarte Neto AN, Reigado GR, Frederico ABT, Cunha DR de A de B e, Paula AV de, Silva JIG da, Vasconcelos CFA de, Alcalde FSC, Dantas VANC, Bom APDA, Castilho LR, Martins RAP, Hirata MH, Mirotti L. Intranasal liposomal formulation of spike protein adjuvanted with CpG protects and boosts heterologous immunity of hACE2 transgenic mice to SARS-CoV-2 infection [Internet]. Vaccines. 2023 ; No 2023( 11): 01-17.[citado 2024 nov. 09 ] Available from: http://dx.doi.org/10.3390/vaccines11111732
Vancouver
Russo M, Corrêa MCM, Lins BB, Kersten V, Pernambuco Filho PC de A, Martins TR, Mendoza TRT, Boas LSV, Gomes BM, Dati LMM, Duarte Neto AN, Reigado GR, Frederico ABT, Cunha DR de A de B e, Paula AV de, Silva JIG da, Vasconcelos CFA de, Alcalde FSC, Dantas VANC, Bom APDA, Castilho LR, Martins RAP, Hirata MH, Mirotti L. Intranasal liposomal formulation of spike protein adjuvanted with CpG protects and boosts heterologous immunity of hACE2 transgenic mice to SARS-CoV-2 infection [Internet]. Vaccines. 2023 ; No 2023( 11): 01-17.[citado 2024 nov. 09 ] Available from: http://dx.doi.org/10.3390/vaccines11111732
Artigo de periodico
Interferome signature dynamics during the anti-dengue immune response: a systems biology characterization (2023)
Usuda, Julia N ; Plaça, Desireé Rodrigues ; Fonseca, Dennyson Leandro Mathias da ; Marques, Alexandre H. C; Filgueiras, Igor Salerno ; Chaves, Victor Gabriel Bastos ; Adri, Anny Silva; Carvalho, Amanda Torrentes de ; Hirata, Mario Hiroyuki ; Freire, Paula Paccielli ; Catar, Rusan ; Miranda, Gustavo Cabral de ; Schimke, Lena F; Moll, Guido ; Marques, Otavio Cabral
Source: Frontiers in Immunology. Unidades: FCF, ICB, BIOTECNOLOGIA
Subjects: DENGUE, ARBOVÍRUS
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ABNT
USUDA, Julia N et al. Interferome signature dynamics during the anti-dengue immune response: a systems biology characterization. Frontiers in Immunology, v. 14, p. 1-16 art. 1243516, 2023Tradução . . Disponível em: https://dx.doi.org/10.3389/fimmu.2023.1243516. Acesso em: 09 nov. 2024.
APA
Usuda, J. N., Plaça, D. R., Fonseca, D. L. M. da, Marques, A. H. C., Filgueiras, I. S., Chaves, V. G. B., et al. (2023). Interferome signature dynamics during the anti-dengue immune response: a systems biology characterization. Frontiers in Immunology, 14, 1-16 art. 1243516. doi:10.3389/fimmu.2023.1243516
NLM
Usuda JN, Plaça DR, Fonseca DLM da, Marques AHC, Filgueiras IS, Chaves VGB, Adri AS, Carvalho AT de, Hirata MH, Freire PP, Catar R, Miranda GC de, Schimke LF, Moll G, Marques OC. Interferome signature dynamics during the anti-dengue immune response: a systems biology characterization [Internet]. Frontiers in Immunology. 2023 ; 14 1-16 art. 1243516.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.3389/fimmu.2023.1243516
Vancouver
Usuda JN, Plaça DR, Fonseca DLM da, Marques AHC, Filgueiras IS, Chaves VGB, Adri AS, Carvalho AT de, Hirata MH, Freire PP, Catar R, Miranda GC de, Schimke LF, Moll G, Marques OC. Interferome signature dynamics during the anti-dengue immune response: a systems biology characterization [Internet]. Frontiers in Immunology. 2023 ; 14 1-16 art. 1243516.[citado 2024 nov. 09 ] Available from: https://dx.doi.org/10.3389/fimmu.2023.1243516
Artigo de periodico
Functional analysis of PCSK9 3'UTR variants and mRNA-miRNA interactions in patients with familial hypercholesterolemia (2021)
Los, Bruna ; Borges, Jéssica Bassani ; Oliveira, Victor Fernandes de ; Freitas, Renata Caroline Costa de ; Hernandez, Carolina Dagli ; Bortolin, Raul Hernandes ; Gonçalves, Rodrigo M ; Faludi, André Arpad ; Rodrigues, Alice Cristina ; Bastos, Gisele Medeiros ; Jannes, Cinthia Elim ; Pereira , Alexandre C ; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Epigenomics. Unidades: IDPC, ICB, FCF
Subjects: HIPERCOLESTEROLEMIA, MICRORNAS
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ABNT
LOS, Bruna et al. Functional analysis of PCSK9 3'UTR variants and mRNA-miRNA interactions in patients with familial hypercholesterolemia. Epigenomics, v. 13, n. 10, p. 779–791, 2021Tradução . . Disponível em: https://doi.org/10.2217/epi-2020-0462. Acesso em: 09 nov. 2024.
APA
Los, B., Borges, J. B., Oliveira, V. F. de, Freitas, R. C. C. de, Hernandez, C. D., Bortolin, R. H., et al. (2021). Functional analysis of PCSK9 3'UTR variants and mRNA-miRNA interactions in patients with familial hypercholesterolemia. Epigenomics, 13( 10), 779–791. doi:10.2217/epi-2020-0462
NLM
Los B, Borges JB, Oliveira VF de, Freitas RCC de, Hernandez CD, Bortolin RH, Gonçalves RM, Faludi AA, Rodrigues AC, Bastos GM, Jannes CE, Pereira AC, Hirata RDC, Hirata MH. Functional analysis of PCSK9 3'UTR variants and mRNA-miRNA interactions in patients with familial hypercholesterolemia [Internet]. Epigenomics. 2021 ; 13( 10): 779–791.[citado 2024 nov. 09 ] Available from: https://doi.org/10.2217/epi-2020-0462
Vancouver
Los B, Borges JB, Oliveira VF de, Freitas RCC de, Hernandez CD, Bortolin RH, Gonçalves RM, Faludi AA, Rodrigues AC, Bastos GM, Jannes CE, Pereira AC, Hirata RDC, Hirata MH. Functional analysis of PCSK9 3'UTR variants and mRNA-miRNA interactions in patients with familial hypercholesterolemia [Internet]. Epigenomics. 2021 ; 13( 10): 779–791.[citado 2024 nov. 09 ] Available from: https://doi.org/10.2217/epi-2020-0462
Artigo de periodico
Functional analysis of PCSK9 3′UTR variants and mRNA–miRNA interactions in patients with familial hypercholesterolemia (2021)
Los, Bruna ; Borges, Jéssica Bassani ; Oliveira, Victor Fernandes de ; Freitas, Renata Carolina Costa de ; Hernandez, Carolina Dagli ; Bortolin, Raul Hernandes ; Gonçalves, Rodrigo M. ; Faludi, André A. ; Bastos, Gisele M. ; Jannes, Cinthia E. ; Pereira, Alexandre C. ; Rodrigues, Alice Cristina ; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Epigenomics. Unidades: ICB, FCF
Subjects: FARMACOLOGIA, HIPERCOLESTEROLEMIA, VARIAÇÃO GENÉTICA, MICRORNAS, ENVELHECIMENTO, RNA MENSAGEIRO, EPIGÊNESE GENÉTICA
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ABNT
LOS, Bruna et al. Functional analysis of PCSK9 3′UTR variants and mRNA–miRNA interactions in patients with familial hypercholesterolemia. Epigenomics, v. 13, n. 10, p. 779–791, 2021Tradução . . Disponível em: https://doi.org/10.2217/epi-2020-0462. Acesso em: 09 nov. 2024.
APA
Los, B., Borges, J. B., Oliveira, V. F. de, Freitas, R. C. C. de, Hernandez, C. D., Bortolin, R. H., et al. (2021). Functional analysis of PCSK9 3′UTR variants and mRNA–miRNA interactions in patients with familial hypercholesterolemia. Epigenomics, 13( 10), 779–791. doi:10.2217/epi-2020-0462
NLM
Los B, Borges JB, Oliveira VF de, Freitas RCC de, Hernandez CD, Bortolin RH, Gonçalves RM, Faludi AA, Bastos GM, Jannes CE, Pereira AC, Rodrigues AC, Hirata RDC, Hirata MH. Functional analysis of PCSK9 3′UTR variants and mRNA–miRNA interactions in patients with familial hypercholesterolemia [Internet]. Epigenomics. 2021 ; 13( 10): 779–791.[citado 2024 nov. 09 ] Available from: https://doi.org/10.2217/epi-2020-0462
Vancouver
Los B, Borges JB, Oliveira VF de, Freitas RCC de, Hernandez CD, Bortolin RH, Gonçalves RM, Faludi AA, Bastos GM, Jannes CE, Pereira AC, Rodrigues AC, Hirata RDC, Hirata MH. Functional analysis of PCSK9 3′UTR variants and mRNA–miRNA interactions in patients with familial hypercholesterolemia [Internet]. Epigenomics. 2021 ; 13( 10): 779–791.[citado 2024 nov. 09 ] Available from: https://doi.org/10.2217/epi-2020-0462
Artigo de periodico
P2x7 receptor signaling blockade reduces lung inflammation and necrosis during severe experimental tuberculosis (2021)
Carvalho, Igor Santiago ; Santos, Gislane Almeida ; Bomfim, Caio César Barbosa ; Souza, Paula Carolina de; Silva, Juan Carlo Santos e ; Melo, Bruno Marcel Silva de ; Amaral, Eduardo Pinheiro ; Cione, Marcos Vinícios Pinheiro; Lasunskaia, Elena ; Hirata, Mario Hiroyuki ; Alves Filho, José Carlos Farias ; Nakaya, Helder Takashi Imoto ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Source: Frontiers in Cellular and Infection Microbiology. Unidades: FCF, FMRP, ICB
Subjects: TUBERCULOSE PULMONAR, RECEPTORES, PULMÃO, EXPRESSÃO GÊNICA, NECROSE, SÍNDROME DE IMUNODEFICIÊNCIA ADQUIRIDA, CAMUNDONGOS, CÉLULAS DENDRÍTICAS, CÉLULAS MIELOIDES, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Igor Santiago et al. P2x7 receptor signaling blockade reduces lung inflammation and necrosis during severe experimental tuberculosis. Frontiers in Cellular and Infection Microbiology, v. 11, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2021.672472. Acesso em: 09 nov. 2024.
APA
Carvalho, I. S., Santos, G. A., Bomfim, C. C. B., Souza, P. C. de, Silva, J. C. S. e, Melo, B. M. S. de, et al. (2021). P2x7 receptor signaling blockade reduces lung inflammation and necrosis during severe experimental tuberculosis. Frontiers in Cellular and Infection Microbiology, 11, 1-11. doi:10.3389/fcimb.2021.672472
NLM
Carvalho IS, Santos GA, Bomfim CCB, Souza PC de, Silva JCS e, Melo BMS de, Amaral EP, Cione MVP, Lasunskaia E, Hirata MH, Alves Filho JCF, Nakaya HTI, Alvarez JM, Lima MRD'I. P2x7 receptor signaling blockade reduces lung inflammation and necrosis during severe experimental tuberculosis [Internet]. Frontiers in Cellular and Infection Microbiology. 2021 ; 11 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fcimb.2021.672472
Vancouver
Carvalho IS, Santos GA, Bomfim CCB, Souza PC de, Silva JCS e, Melo BMS de, Amaral EP, Cione MVP, Lasunskaia E, Hirata MH, Alves Filho JCF, Nakaya HTI, Alvarez JM, Lima MRD'I. P2x7 receptor signaling blockade reduces lung inflammation and necrosis during severe experimental tuberculosis [Internet]. Frontiers in Cellular and Infection Microbiology. 2021 ; 11 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fcimb.2021.672472
Artigo de periodico
Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria (2021)
Bomfim, Caio César Barbosa ; Amaral, Eduardo Pinheiro ; Carvalho, Igor Santiago ; Santos, Gislane Almeida ; Salles, Érika Machado de ; Hastreiter, Araceli Aparecida ; Nascimento, Rogério Silva do; Almeida, Fabricio M.; Simão, Thatiana Lopes Biá Ventura ; Rezende, Andreza Linhares; Hirata, Mario Hiroyuki ; Fock, Ricardo Ambrósio ; Alvarez, José Maria ; Lasunskaia, Elena B ; Lima, Maria Regina D'Império
Source: Journal of Infectious Diseases. Unidades: ICB, FCF
Subjects: TUBERCULOSE PULMONAR, CÉLULAS SANGUINEAS, GRANULOCITOS CITOPLASMATICOS, MEDULA ÓSSEA DE ANIMAL, VIRULÊNCIA, NEUTRÓFILOS, PROLIFERAÇÃO CELULAR, MYCOBACTERIUM BOVIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOMFIM, Caio César Barbosa et al. Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria. Journal of Infectious Diseases, v. 223, n. 3, p. 494–507, 2021Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiaa708. Acesso em: 09 nov. 2024.
APA
Bomfim, C. C. B., Amaral, E. P., Carvalho, I. S., Santos, G. A., Salles, É. M. de, Hastreiter, A. A., et al. (2021). Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria. Journal of Infectious Diseases, 223( 3), 494–507. doi:10.1093/infdis/jiaa708
NLM
Bomfim CCB, Amaral EP, Carvalho IS, Santos GA, Salles ÉM de, Hastreiter AA, Nascimento RS do, Almeida FM, Simão TLBV, Rezende AL, Hirata MH, Fock RA, Alvarez JM, Lasunskaia EB, Lima MRD'I. Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria [Internet]. Journal of Infectious Diseases. 2021 ; 223( 3): 494–507.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1093/infdis/jiaa708
Vancouver
Bomfim CCB, Amaral EP, Carvalho IS, Santos GA, Salles ÉM de, Hastreiter AA, Nascimento RS do, Almeida FM, Simão TLBV, Rezende AL, Hirata MH, Fock RA, Alvarez JM, Lasunskaia EB, Lima MRD'I. Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria [Internet]. Journal of Infectious Diseases. 2021 ; 223( 3): 494–507.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1093/infdis/jiaa708
Artigo de periodico
Profiling plasma-extracellular vesicle proteins and microRNAs in diabetes onset in middle-aged male participants in the ELSA-Brasil study (2021)
Masi, Laureane Nunes ; Lotufo, Paulo Andrade ; Ferreira, Frederico Moraes ; Rodrigues, Alice Cristina ; Serdan, Tamires Duarte Afonso ; Siqueira, Talita Souza ; Braga, Aécio Assunção ; Saldarriaga, Magda Elizabeth Graciano ; Loureiro, Tatiana Carolina Alba ; Borges, Fernanda Teixeira ; Cury, Diego Pulzatto ; Hirata, Mario Hiroyuki ; Gorjão, Renata ; Curi, Tania Cristina Phiton ; Lottenberg, Simão Augusto ; Fedeli, Ligia Maria Giongo ; Nakaya, Helder Takashi Imoto ; Bensenor, Isabela Judith Martins ; Curi, Rui ; Hirabara, Sandro Massao
Source: Physiological Reports. Unidades: FM, ICB, FCF, HU
Subjects: MICRORNAS, HIPERGLICEMIA, PROTEÔMICA, GLICOSE, FARMACOLOGIA, PLASMA
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ABNT
MASI, Laureane Nunes et al. Profiling plasma-extracellular vesicle proteins and microRNAs in diabetes onset in middle-aged male participants in the ELSA-Brasil study. Physiological Reports, v. 9, p. 1-13 art. e14731, 2021Tradução . . Disponível em: https://doi.org/10.14814/phy2.14731. Acesso em: 09 nov. 2024.
APA
Masi, L. N., Lotufo, P. A., Ferreira, F. M., Rodrigues, A. C., Serdan, T. D. A., Siqueira, T. S., et al. (2021). Profiling plasma-extracellular vesicle proteins and microRNAs in diabetes onset in middle-aged male participants in the ELSA-Brasil study. Physiological Reports, 9, 1-13 art. e14731. doi:10.14814/phy2.14731
NLM
Masi LN, Lotufo PA, Ferreira FM, Rodrigues AC, Serdan TDA, Siqueira TS, Braga AA, Saldarriaga MEG, Loureiro TCA, Borges FT, Cury DP, Hirata MH, Gorjão R, Curi TCP, Lottenberg SA, Fedeli LMG, Nakaya HTI, Bensenor IJM, Curi R, Hirabara SM. Profiling plasma-extracellular vesicle proteins and microRNAs in diabetes onset in middle-aged male participants in the ELSA-Brasil study [Internet]. Physiological Reports. 2021 ; 9 1-13 art. e14731.[citado 2024 nov. 09 ] Available from: https://doi.org/10.14814/phy2.14731
Vancouver
Masi LN, Lotufo PA, Ferreira FM, Rodrigues AC, Serdan TDA, Siqueira TS, Braga AA, Saldarriaga MEG, Loureiro TCA, Borges FT, Cury DP, Hirata MH, Gorjão R, Curi TCP, Lottenberg SA, Fedeli LMG, Nakaya HTI, Bensenor IJM, Curi R, Hirabara SM. Profiling plasma-extracellular vesicle proteins and microRNAs in diabetes onset in middle-aged male participants in the ELSA-Brasil study [Internet]. Physiological Reports. 2021 ; 9 1-13 art. e14731.[citado 2024 nov. 09 ] Available from: https://doi.org/10.14814/phy2.14731
Artigo de periodico
Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells (2020)
Souza, Camila O. de ; Teixeira, Alexandre Abilio de Souza ; Biondo, Luana Amorim ; Silveira, Loreana Sanches ; Breda, Cristiane Naffah de Souza ; Braga, Tarcio Teodoro ; Câmara, Niels Olsen Saraiva ; Belchior, Thiago; Festuccia, William Tadeu Lara ; Diniz, Tiego Aparecido ; Ferreira, Glaucio Monteiro ; Hirata, Mario Hiroyuki ; Chaves Filho, Adriano de Britto ; Yoshinaga, Marcos Yukio ; Miyamoto, Sayuri ; Calder, Phillip C. ; Sethif, Jaswinder K.; Rosa Neto, José Cesar
Source: Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. Unidades: ICB, FCF, IQ
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, IMUNOLOGIA, OBESIDADE, FÍGADO, MACRÓFAGOS, INFLAMAÇÃO, CITOCINAS, ÁCIDOS GRAXOS, CÉLULAS MIELOIDES, CAMUNDONGOS
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ABNT
SOUZA, Camila O. de et al. Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids, v. 1865, n. 10, p. 12 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbalip.2020.158776. Acesso em: 09 nov. 2024.
APA
Souza, C. O. de, Teixeira, A. A. de S., Biondo, L. A., Silveira, L. S., Breda, C. N. de S., Braga, T. T., et al. (2020). Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids, 1865( 10), 12 . doi:10.1016/j.bbalip.2020.158776
NLM
Souza CO de, Teixeira AA de S, Biondo LA, Silveira LS, Breda CN de S, Braga TT, Câmara NOS, Belchior T, Festuccia WTL, Diniz TA, Ferreira GM, Hirata MH, Chaves Filho A de B, Yoshinaga MY, Miyamoto S, Calder PC, Sethif JK, Rosa Neto JC. Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 2020 ; 1865( 10): 12 .[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbalip.2020.158776
Vancouver
Souza CO de, Teixeira AA de S, Biondo LA, Silveira LS, Breda CN de S, Braga TT, Câmara NOS, Belchior T, Festuccia WTL, Diniz TA, Ferreira GM, Hirata MH, Chaves Filho A de B, Yoshinaga MY, Miyamoto S, Calder PC, Sethif JK, Rosa Neto JC. Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 2020 ; 1865( 10): 12 .[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbalip.2020.158776
Trabalho de evento-resumo
Circulating miR-421 expression is associated with insulin resistance in metabolic syndrome patients (2019)
Bortolin, Raul Hernandes ; Braga, Aécio Assunção; Saldarriaga, Magda Elizabeth Graciano; Hirata, Thiago Dominguez Crespo ; Cerda, Alvaro; Freitas, Renata Caroline Costa de ; Wang, H. L; Borges, J. B; França, J. I. D; Masi, Laureane Nunes; Curi, Rui; Curi, Tania Cristina Pithon; Sampaio, M. F; Castro, L. R; Bastos, G. M; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Abstracts. Conference titles: American Association for Clinical Chemistry/AACC. Unidades: ICB, FCF
Subjects: SÍNDROME X METABÓLICA, RESISTÊNCIA À INSULINA
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ABNT
BORTOLIN, Raul Hernandes et al. Circulating miR-421 expression is associated with insulin resistance in metabolic syndrome patients. 2019, Anais.. Washington: AACC, 2019. . Acesso em: 09 nov. 2024.
APA
Bortolin, R. H., Braga, A. A., Saldarriaga, M. E. G., Hirata, T. D. C., Cerda, A., Freitas, R. C. C. de, et al. (2019). Circulating miR-421 expression is associated with insulin resistance in metabolic syndrome patients. In Abstracts. Washington: AACC.
NLM
Bortolin RH, Braga AA, Saldarriaga MEG, Hirata TDC, Cerda A, Freitas RCC de, Wang HL, Borges JB, França JID, Masi LN, Curi R, Curi TCP, Sampaio MF, Castro LR, Bastos GM, Hirata RDC, Hirata MH. Circulating miR-421 expression is associated with insulin resistance in metabolic syndrome patients. Abstracts. 2019 ;[citado 2024 nov. 09 ]
Vancouver
Bortolin RH, Braga AA, Saldarriaga MEG, Hirata TDC, Cerda A, Freitas RCC de, Wang HL, Borges JB, França JID, Masi LN, Curi R, Curi TCP, Sampaio MF, Castro LR, Bastos GM, Hirata RDC, Hirata MH. Circulating miR-421 expression is associated with insulin resistance in metabolic syndrome patients. Abstracts. 2019 ;[citado 2024 nov. 09 ]
Trabalho de evento-resumo
Studies of LDL receptor activity in patients with familial hypercholesterolemia (2019)
Afonso, Thais Kristini Almendros; Oliveira, Victor Fernandes de ; Ferreira, Glaucio Monteiro ; Borges, Jéssica Bassani ; Bastos, Gisele Medeiros ; Pithon-Curi, Tania Cristina; Gorjão, Renata; Curi, Rui ; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki
Source: Proceedings. Conference titles: International Conference of the Brazilian Association of Bioinformatics and Computational Biology/AB3C. Unidades: ICB, FCF
Assunto: HIPERCOLESTEROLEMIA
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ABNT
AFONSO, Thais Kristini Almendros et al. Studies of LDL receptor activity in patients with familial hypercholesterolemia. 2019, Anais.. Campos do Jordão: Associação Brasileira de Bioinformática e Biologia Computacional/AB3C, 2019. . Acesso em: 09 nov. 2024.
APA
Afonso, T. K. A., Oliveira, V. F. de, Ferreira, G. M., Borges, J. B., Bastos, G. M., Pithon-Curi, T. C., et al. (2019). Studies of LDL receptor activity in patients with familial hypercholesterolemia. In Proceedings. Campos do Jordão: Associação Brasileira de Bioinformática e Biologia Computacional/AB3C.
NLM
Afonso TKA, Oliveira VF de, Ferreira GM, Borges JB, Bastos GM, Pithon-Curi TC, Gorjão R, Curi R, Hirata RDC, Hirata MH. Studies of LDL receptor activity in patients with familial hypercholesterolemia. Proceedings. 2019 ;[citado 2024 nov. 09 ]
Vancouver
Afonso TKA, Oliveira VF de, Ferreira GM, Borges JB, Bastos GM, Pithon-Curi TC, Gorjão R, Curi R, Hirata RDC, Hirata MH. Studies of LDL receptor activity in patients with familial hypercholesterolemia. Proceedings. 2019 ;[citado 2024 nov. 09 ]
Artigo de periodico
Inhibiting adenosine receptor signaling promotes accumulation of effector CD4+ T cells in the lung parenchyma during severe tuberculosis (2019)
Amaral, Eduardo Pinheiro ; Salles, Érika Machado de ; Bomfim, Caio César Barbosa ; Salgado, Rafael Moysés; Almeida, Fabricio Moreira; Souza, Paula Carolina de; Alvarez, José Maria ; Hirata, Mario Hiroyuki ; Lasunskaia, Elena B ; Lima, Maria Regina D'Império
Source: Journal of Infectious Diseases. Unidades: ICB, FCF
Subjects: ADENOSINA, CAFEÍNA, TUBERCULOSE, MYCOBACTERIUM TUBERCULOSIS, CAMUNDONGOS, PULMÃO, INTERFERONS
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ABNT
AMARAL, Eduardo Pinheiro et al. Inhibiting adenosine receptor signaling promotes accumulation of effector CD4+ T cells in the lung parenchyma during severe tuberculosis. Journal of Infectious Diseases, v. 219, n. 6, p. 964-974, 2019Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiy586. Acesso em: 09 nov. 2024.
APA
Amaral, E. P., Salles, É. M. de, Bomfim, C. C. B., Salgado, R. M., Almeida, F. M., Souza, P. C. de, et al. (2019). Inhibiting adenosine receptor signaling promotes accumulation of effector CD4+ T cells in the lung parenchyma during severe tuberculosis. Journal of Infectious Diseases, 219( 6), 964-974. doi:10.1093/infdis/jiy586
NLM
Amaral EP, Salles ÉM de, Bomfim CCB, Salgado RM, Almeida FM, Souza PC de, Alvarez JM, Hirata MH, Lasunskaia EB, Lima MRD'I. Inhibiting adenosine receptor signaling promotes accumulation of effector CD4+ T cells in the lung parenchyma during severe tuberculosis [Internet]. Journal of Infectious Diseases. 2019 ; 219( 6): 964-974.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1093/infdis/jiy586
Vancouver
Amaral EP, Salles ÉM de, Bomfim CCB, Salgado RM, Almeida FM, Souza PC de, Alvarez JM, Hirata MH, Lasunskaia EB, Lima MRD'I. Inhibiting adenosine receptor signaling promotes accumulation of effector CD4+ T cells in the lung parenchyma during severe tuberculosis [Internet]. Journal of Infectious Diseases. 2019 ; 219( 6): 964-974.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1093/infdis/jiy586
Artigo de periodico
Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis (2017)
Nascimento, Ivan Pereira; Rodriguez, Dúnia; Santos, Carina Carvalho dos; Amaral, Eduardo Pinheiro; Rofatto, Henrique Krambeck; Junqueira-Kipnis, Ana Paula; Gonçalves, Eduardo Dantas Casillo; Lima, Maria Regina D'Império ; Hirata, Mario Hiroyuki ; Silva, Célio Lopes; Winter, Nathalie; Gicquel, Brigitte; Mills, Kingston H G; Pizza, Mariagrazia; Rappuoli, Rino; Leite, Luciana C. C
Source: Scientific Reports. Unidades: ICB, FCF, FMRP
Subjects: IMUNOLOGIA, TUBERCULOSE, MYCOBACTERIUM TUBERCULOSIS, ADJUVANTES IMUNOLÓGICOS, PROTEÍNAS RECOMBINANTES, VACINAS, CITOCININAS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Ivan Pereira et al. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis. Scientific Reports, v. 7, n. 1, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-02003-9. Acesso em: 09 nov. 2024.
APA
Nascimento, I. P., Rodriguez, D., Santos, C. C. dos, Amaral, E. P., Rofatto, H. K., Junqueira-Kipnis, A. P., et al. (2017). Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis. Scientific Reports, 7( 1), 1-11. doi:10.1038/s41598-017-02003-9
NLM
Nascimento IP, Rodriguez D, Santos CC dos, Amaral EP, Rofatto HK, Junqueira-Kipnis AP, Gonçalves EDC, Lima MRD'I, Hirata MH, Silva CL, Winter N, Gicquel B, Mills KHG, Pizza M, Rappuoli R, Leite LCC. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/s41598-017-02003-9
Vancouver
Nascimento IP, Rodriguez D, Santos CC dos, Amaral EP, Rofatto HK, Junqueira-Kipnis AP, Gonçalves EDC, Lima MRD'I, Hirata MH, Silva CL, Winter N, Gicquel B, Mills KHG, Pizza M, Rappuoli R, Leite LCC. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/s41598-017-02003-9
Artigo de periodico
P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis (2017)
Bomfim, Caio César Barbosa; Amaral, Eduardo Pinheiro; Cassado, Alexandra dos Anjos; Salles, Érika Machado; Nascimento, Rogério Silva do; Lasunskaia, Elena; Hirata, Mario Hiroyuki ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Source: Frontiers in Immunology. Unidades: ICB, FCF
Assunto: IMUNOLOGIA
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ABNT
BOMFIM, Caio César Barbosa et al. P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis. Frontiers in Immunology, v. 8, n. 435, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00435. Acesso em: 09 nov. 2024.
APA
Bomfim, C. C. B., Amaral, E. P., Cassado, A. dos A., Salles, É. M., Nascimento, R. S. do, Lasunskaia, E., et al. (2017). P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis. Frontiers in Immunology, 8( 435), 1-10. doi:10.3389/fimmu.2017.00435
NLM
Bomfim CCB, Amaral EP, Cassado A dos A, Salles ÉM, Nascimento RS do, Lasunskaia E, Hirata MH, Alvarez JM, Lima MRD'I. P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis [Internet]. Frontiers in Immunology. 2017 ; 8( 435): 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00435
Vancouver
Bomfim CCB, Amaral EP, Cassado A dos A, Salles ÉM, Nascimento RS do, Lasunskaia E, Hirata MH, Alvarez JM, Lima MRD'I. P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis [Internet]. Frontiers in Immunology. 2017 ; 8( 435): 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00435

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