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Artigo de periodico
Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis (2022)
Marinovic, Marcelo Paradiso; Sousa Filho, Celso Pereira Batista de ; Batista, F. A. H; Avelino, T. M; Cogliati, Bruno ; Figueira, A. C. M; Otton, Rosemari ; Rodrigues, Alice Cristina
Source: Journal of Nutritional Biochemistry. Unidades: FMVZ, ICB
Subjects: CHÁ, ADIPONECTINA, FÍGADO GORDUROSO, OBESIDADE, ANTIOXIDANTES, INSULINA, EXTRATOS (FORMAS FARMACÊUTICAS), FARMACOLOGIA
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ABNT
MARINOVIC, Marcelo Paradiso et al. Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis. Journal of Nutritional Biochemistry, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2022.108957. Acesso em: 10 nov. 2024.
APA
Marinovic, M. P., Sousa Filho, C. P. B. de, Batista, F. A. H., Avelino, T. M., Cogliati, B., Figueira, A. C. M., et al. (2022). Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis. Journal of Nutritional Biochemistry. doi:10.1016/j.jnutbio.2022.108957
NLM
Marinovic MP, Sousa Filho CPB de, Batista FAH, Avelino TM, Cogliati B, Figueira ACM, Otton R, Rodrigues AC. Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis [Internet]. Journal of Nutritional Biochemistry. 2022 ;[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.jnutbio.2022.108957
Vancouver
Marinovic MP, Sousa Filho CPB de, Batista FAH, Avelino TM, Cogliati B, Figueira ACM, Otton R, Rodrigues AC. Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis [Internet]. Journal of Nutritional Biochemistry. 2022 ;[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.jnutbio.2022.108957
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Source: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Subjects: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
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COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 10 nov. 2024.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Artigo de periodico
Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization (2019)
Santos, Nilton Barreto dos ; Franco, Roseane Durante; Camarini, Rosana ; Munhoz, Carolina Demarchi ; Eichler, Rosangela Aparecida dos Santos ; Gewehr, Mayara Calegaro Ferrari ; Reckziegel, Patricia ; Dale, Camila Squarzoni ; Silva, Victoria R. O. da; Borges, Vanessa de Fátima; Lima, Braulio Henrique Freire ; Cunha, Fernando de Queiroz ; Visniauskas, Bruna ; Chagas, Jair Ribeiro ; Tufik, Sergio; Peres, Fernanda Fiel; Abilio, Vanessa Costhek ; Florio, Jorge Camilo ; Iwai, Leo Kei ; Rioli, Vanessa ; Presoto, Benedito C.; Guimarães, Alessander O. ; Pesquero, João Bosco ; Bader, Michael ; Castro, Leandro Mantovani de ; Ferro, Emer Suavinho
Source: Biomolecules. Unidades: ICB, FMVZ, FMRP
Subjects: ENZIMAS, NEUROPEPTÍDEOS, DEGENERAÇÃO NEURAL, INFLAMAÇÃO, ANATOMIA, FARMACOLOGIA, OLIGOPEPTÍDEOS, CAMUNDONGOS, FENÓTIPOS
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SANTOS, Nilton Barreto dos et al. Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization. Biomolecules, v. 9, n. 8, p. 1-38, 2019Tradução . . Disponível em: https://doi.org/10.3390/biom9080382. Acesso em: 10 nov. 2024.
APA
Santos, N. B. dos, Franco, R. D., Camarini, R., Munhoz, C. D., Eichler, R. A. dos S., Gewehr, M. C. F., et al. (2019). Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization. Biomolecules, 9( 8), 1-38. doi:10.3390/biom9080382
NLM
Santos NB dos, Franco RD, Camarini R, Munhoz CD, Eichler RA dos S, Gewehr MCF, Reckziegel P, Dale CS, Silva VRO da, Borges V de F, Lima BHF, Cunha F de Q, Visniauskas B, Chagas JR, Tufik S, Peres FF, Abilio VC, Florio JC, Iwai LK, Rioli V, Presoto BC, Guimarães AO, Pesquero JB, Bader M, Castro LM de, Ferro ES. Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization [Internet]. Biomolecules. 2019 ; 9( 8): 1-38.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/biom9080382
Vancouver
Santos NB dos, Franco RD, Camarini R, Munhoz CD, Eichler RA dos S, Gewehr MCF, Reckziegel P, Dale CS, Silva VRO da, Borges V de F, Lima BHF, Cunha F de Q, Visniauskas B, Chagas JR, Tufik S, Peres FF, Abilio VC, Florio JC, Iwai LK, Rioli V, Presoto BC, Guimarães AO, Pesquero JB, Bader M, Castro LM de, Ferro ES. Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization [Internet]. Biomolecules. 2019 ; 9( 8): 1-38.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/biom9080382
Artigo de periodico-carta/editorial
Stathmin 1 expression in plasma cell neoplasms. [Carta] (2017)
Machado Neto, João Agostinho ; Pericole, Fernando Vieira; Costa, Fernando Ferreira; Traina, Fabiola; Saad, Sara Teresinha Olalla
Source: Revista Brasileira de Hematologia e Hemoterapia. Unidades: ICB, FMRP
Subjects: EXPRESSÃO GÊNICA, NEOPLASIAS, HEMATOLOGIA
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MACHADO NETO, João Agostinho et al. Stathmin 1 expression in plasma cell neoplasms. [Carta]. Revista Brasileira de Hematologia e Hemoterapia. Rio de Janeiro: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.bjhh.2017.02.005. Acesso em: 10 nov. 2024. , 2017
APA
Machado Neto, J. A., Pericole, F. V., Costa, F. F., Traina, F., & Saad, S. T. O. (2017). Stathmin 1 expression in plasma cell neoplasms. [Carta]. Revista Brasileira de Hematologia e Hemoterapia. Rio de Janeiro: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.bjhh.2017.02.005
NLM
Machado Neto JA, Pericole FV, Costa FF, Traina F, Saad STO. Stathmin 1 expression in plasma cell neoplasms. [Carta] [Internet]. Revista Brasileira de Hematologia e Hemoterapia. 2017 ; 39( 2): 183-185.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bjhh.2017.02.005
Vancouver
Machado Neto JA, Pericole FV, Costa FF, Traina F, Saad STO. Stathmin 1 expression in plasma cell neoplasms. [Carta] [Internet]. Revista Brasileira de Hematologia e Hemoterapia. 2017 ; 39( 2): 183-185.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bjhh.2017.02.005
Artigo de periodico
Paclitaxel induces Stathmin 1 phosphorylation, microtubule stability and apoptosis in acute lymphoblastic leukemia cells (2017)
Machado Neto, João Agostinho ; Alves, Ana Paula Nunes Rodrigues; Fernandes, Jaqueline Cristina; Silva, Juan Luiz Coelho; Ribeiro, Renata Scopim; Fenerich, Bruna Alves; Silva, Fernanda Borges da; Scheucher, Priscila Santos; Simões, Belinda Pinto; Rego, Eduardo Magalhães; Traina, Fabiola
Source: Heliyon. Unidades: ICB, FMRP
Subjects: LEUCEMIA, BIOLOGIA CELULAR, ONCOLOGIA
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MACHADO NETO, João Agostinho et al. Paclitaxel induces Stathmin 1 phosphorylation, microtubule stability and apoptosis in acute lymphoblastic leukemia cells. Heliyon, v. 3, n. 9, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.heliyon.2017.e00405. Acesso em: 10 nov. 2024.
APA
Machado Neto, J. A., Alves, A. P. N. R., Fernandes, J. C., Silva, J. L. C., Ribeiro, R. S., Fenerich, B. A., et al. (2017). Paclitaxel induces Stathmin 1 phosphorylation, microtubule stability and apoptosis in acute lymphoblastic leukemia cells. Heliyon, 3( 9). doi:10.1016/j.heliyon.2017.e00405
NLM
Machado Neto JA, Alves APNR, Fernandes JC, Silva JLC, Ribeiro RS, Fenerich BA, Silva FB da, Scheucher PS, Simões BP, Rego EM, Traina F. Paclitaxel induces Stathmin 1 phosphorylation, microtubule stability and apoptosis in acute lymphoblastic leukemia cells [Internet]. Heliyon. 2017 ; 3( 9):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.heliyon.2017.e00405
Vancouver
Machado Neto JA, Alves APNR, Fernandes JC, Silva JLC, Ribeiro RS, Fenerich BA, Silva FB da, Scheucher PS, Simões BP, Rego EM, Traina F. Paclitaxel induces Stathmin 1 phosphorylation, microtubule stability and apoptosis in acute lymphoblastic leukemia cells [Internet]. Heliyon. 2017 ; 3( 9):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.heliyon.2017.e00405
Artigo de periodico
MyomiRs as markers of insulin resistance and decreased myogenesis in skeletal muscle of diet-induced obese mice (2016)
Frias, Flávia de Toledo; Mendonça, Mariana de; Martins, Amanda Roque; Gindro, Ana Flávia; Cogliati, Bruno ; Curi, Rui; Rodrigues, Alice Cristina
Source: Frontiers in Endocrinology. Unidades: FMVZ, ICB
Subjects: DIABETES MELLITUS, INSULINA, METABOLISMO, MÚSCULO ESQUELÉTICO, OBESIDADE, PROTEÍNA 1 DE LIGAÇÃO AOS FATORES DE CRESCIMENTO INSULIN-LIKE
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FRIAS, Flávia de Toledo et al. MyomiRs as markers of insulin resistance and decreased myogenesis in skeletal muscle of diet-induced obese mice. Frontiers in Endocrinology, v. 7, n. Ju 2016, p. 12 , 2016Tradução . . Disponível em: https://doi.org/10.3389/fendo.2016.00076. Acesso em: 10 nov. 2024.
APA
Frias, F. de T., Mendonça, M. de, Martins, A. R., Gindro, A. F., Cogliati, B., Curi, R., & Rodrigues, A. C. (2016). MyomiRs as markers of insulin resistance and decreased myogenesis in skeletal muscle of diet-induced obese mice. Frontiers in Endocrinology, 7( Ju 2016), 12 . doi:10.3389/fendo.2016.00076
NLM
Frias F de T, Mendonça M de, Martins AR, Gindro AF, Cogliati B, Curi R, Rodrigues AC. MyomiRs as markers of insulin resistance and decreased myogenesis in skeletal muscle of diet-induced obese mice [Internet]. Frontiers in Endocrinology. 2016 ; 7( Ju 2016): 12 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2016.00076
Vancouver
Frias F de T, Mendonça M de, Martins AR, Gindro AF, Cogliati B, Curi R, Rodrigues AC. MyomiRs as markers of insulin resistance and decreased myogenesis in skeletal muscle of diet-induced obese mice [Internet]. Frontiers in Endocrinology. 2016 ; 7( Ju 2016): 12 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2016.00076
Artigo de periodico
Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring (2015)
Kirsten, Thiago Berti; Chaves-Kirsten, Gabriela Pena; Bernardes, Suene; Scavone, Cristoforo ; Sarkis, Jorge Eduardo de Souza; Bernardi, Maria Martha; Felicio, Luciano Freitas
Source: Plos One. Unidades: ICB, FMVZ
Subjects: AUTISMO, GRAVIDEZ, LIPOPOLISSACARÍDEOS, RATOS
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KIRSTEN, Thiago Berti et al. Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring. Plos One, v. 10, n. 7, p. 1-18, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0134565. Acesso em: 10 nov. 2024.
APA
Kirsten, T. B., Chaves-Kirsten, G. P., Bernardes, S., Scavone, C., Sarkis, J. E. de S., Bernardi, M. M., & Felicio, L. F. (2015). Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring. Plos One, 10( 7), 1-18. doi:10.1371/journal.pone.0134565
NLM
Kirsten TB, Chaves-Kirsten GP, Bernardes S, Scavone C, Sarkis JE de S, Bernardi MM, Felicio LF. Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring [Internet]. Plos One. 2015 ; 10( 7): 1-18.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0134565
Vancouver
Kirsten TB, Chaves-Kirsten GP, Bernardes S, Scavone C, Sarkis JE de S, Bernardi MM, Felicio LF. Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring [Internet]. Plos One. 2015 ; 10( 7): 1-18.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0134565
Artigo de periodico
Exposure of Neonatal mice to Tobacco Smoke Disturbs Synaptic Proteins and spatial learning and memory from late infancy to early adulthood (2015)
Torres, Larissa Helena; Garcia, Raphael C. T; Blois, Anne M. M.; Dati, Lívia M. M; Durão, Ana Carolina; Alves, Adilson Silva; Pacheco Neto, Maurílio; Mauad, Thais; Britto, Luiz Roberto Giorgetti de ; Xavier, Gilberto Fernando ; Camarini, Rosana ; Marcourakis, Tania
Source: PLOS ONE. Unidades: ICB, FM, IB, FCF
Subjects: POLUIÇÃO AMBIENTAL, FUMO (EXPOSIÇÕES), DOENÇAS, MEMÓRIA ANIMAL (DESEMPENHO), RATOS, MODELOS ANIMAIS, NEUROFISIOLOGIA, SISTEMA NERVOSO CENTRAL, FARMACOLOGIA, TOXICOLOGIA
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TORRES, Larissa Helena et al. Exposure of Neonatal mice to Tobacco Smoke Disturbs Synaptic Proteins and spatial learning and memory from late infancy to early adulthood. PLOS ONE, v. 10, n. 8, p. e0136399, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0136399. Acesso em: 10 nov. 2024.
APA
Torres, L. H., Garcia, R. C. T., Blois, A. M. M., Dati, L. M. M., Durão, A. C., Alves, A. S., et al. (2015). Exposure of Neonatal mice to Tobacco Smoke Disturbs Synaptic Proteins and spatial learning and memory from late infancy to early adulthood. PLOS ONE, 10( 8), e0136399. doi:10.1371/journal.pone.0136399
NLM
Torres LH, Garcia RCT, Blois AMM, Dati LMM, Durão AC, Alves AS, Pacheco Neto M, Mauad T, Britto LRG de, Xavier GF, Camarini R, Marcourakis T. Exposure of Neonatal mice to Tobacco Smoke Disturbs Synaptic Proteins and spatial learning and memory from late infancy to early adulthood [Internet]. PLOS ONE. 2015 ; 10( 8): e0136399.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0136399
Vancouver
Torres LH, Garcia RCT, Blois AMM, Dati LMM, Durão AC, Alves AS, Pacheco Neto M, Mauad T, Britto LRG de, Xavier GF, Camarini R, Marcourakis T. Exposure of Neonatal mice to Tobacco Smoke Disturbs Synaptic Proteins and spatial learning and memory from late infancy to early adulthood [Internet]. PLOS ONE. 2015 ; 10( 8): e0136399.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0136399
Artigo de periodico
Role of steroid hormones and morphine treatment in the modulation of opioid receptor gene expression in brain structures in the female rat (2015)
Cruz, Wesley Soares; Pereira, Lucas Assis; Cezar, Luana Carvalho; Camarini, Rosana ; Felicio, Luciano Freitas ; Bernardi, Maria Martha; Teodorov, Elizabeth
Source: SpringerPlus. Unidades: ICB, FMVZ
Subjects: HORMÔNIOS (EFEITOS), MORFINA (EFEITOS), RECEPTORES (GENÉTICA), GENÉTICA BIOQUÍMICA, CÉREBRO (ALTERAÇÃO)
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CRUZ, Wesley Soares et al. Role of steroid hormones and morphine treatment in the modulation of opioid receptor gene expression in brain structures in the female rat. SpringerPlus, v. 4, p. 1-6, 2015Tradução . . Disponível em: https://doi.org/10.1186/s40064-015-1021-8. Acesso em: 10 nov. 2024.
APA
Cruz, W. S., Pereira, L. A., Cezar, L. C., Camarini, R., Felicio, L. F., Bernardi, M. M., & Teodorov, E. (2015). Role of steroid hormones and morphine treatment in the modulation of opioid receptor gene expression in brain structures in the female rat. SpringerPlus, 4, 1-6. doi:10.1186/s40064-015-1021-8
NLM
Cruz WS, Pereira LA, Cezar LC, Camarini R, Felicio LF, Bernardi MM, Teodorov E. Role of steroid hormones and morphine treatment in the modulation of opioid receptor gene expression in brain structures in the female rat [Internet]. SpringerPlus. 2015 ; 4 1-6.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/s40064-015-1021-8
Vancouver
Cruz WS, Pereira LA, Cezar LC, Camarini R, Felicio LF, Bernardi MM, Teodorov E. Role of steroid hormones and morphine treatment in the modulation of opioid receptor gene expression in brain structures in the female rat [Internet]. SpringerPlus. 2015 ; 4 1-6.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/s40064-015-1021-8
Artigo de periodico
Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs (2013)
Araújo, Karla Patrícia Cardoso; Bonuccelli, Gloria; Duarte, Caio Nogueira; Gaiad, Thaís Peixoto; Moreira, Dayson Friaça; Feder, David; Belizário, José Ernesto; Miglino, Maria Angélica; Lisanti, Michael P; Ambrósio, Carlos Eduardo
Source: Plos One. Unidades: FMVZ, FZEA, ICB
Subjects: ATROFIA MUSCULAR, CÃES, DISTROFIA MUSCULAR ANIMAL, GLICOPROTEÍNAS
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ABNT
ARAÚJO, Karla Patrícia Cardoso et al. Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs. Plos One, v. 8, n. 4, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0061367. Acesso em: 10 nov. 2024.
APA
Araújo, K. P. C., Bonuccelli, G., Duarte, C. N., Gaiad, T. P., Moreira, D. F., Feder, D., et al. (2013). Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs. Plos One, 8( 4). doi:10.1371/journal.pone.0061367
NLM
Araújo KPC, Bonuccelli G, Duarte CN, Gaiad TP, Moreira DF, Feder D, Belizário JE, Miglino MA, Lisanti MP, Ambrósio CE. Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs [Internet]. Plos One. 2013 ; 8( 4):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0061367
Vancouver
Araújo KPC, Bonuccelli G, Duarte CN, Gaiad TP, Moreira DF, Feder D, Belizário JE, Miglino MA, Lisanti MP, Ambrósio CE. Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs [Internet]. Plos One. 2013 ; 8( 4):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0061367
Trabalho de evento-resumo periodico
Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle (2009)
Araújo, Karla Patrícia Cardoso; Moreira, Dayson Friaça; Gaiad, Thaís Peixoto; Miglino, Maria Angélica; Górniak, Silvana Lima ; Feder, D; Belizário, José Ernesto; Ambrósio, Carlos Eduardo
Source: Neuromuscular Disorders. Conference titles: International Congress of the World Muscle Society. Unidades: FMVZ, ICB, FZEA
Subjects: FARMACOLOGIA, CÃO GOLDEN RETRIEVER, DISTROFIA MUSCULAR ANIMAL, NEUROLOGIA VETERINÁRIA
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ARAÚJO, Karla Patrícia Cardoso et al. Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle. Neuromuscular Disorders. Oxford: Elsevier Science. Disponível em: https://doi.org/10.1016/j.nmd.2009.06.218. Acesso em: 10 nov. 2024. , 2009
APA
Araújo, K. P. C., Moreira, D. F., Gaiad, T. P., Miglino, M. A., Górniak, S. L., Feder, D., et al. (2009). Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle. Neuromuscular Disorders. Oxford: Elsevier Science. doi:10.1016/j.nmd.2009.06.218
NLM
Araújo KPC, Moreira DF, Gaiad TP, Miglino MA, Górniak SL, Feder D, Belizário JE, Ambrósio CE. Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle [Internet]. Neuromuscular Disorders. 2009 ; 19( 8-9): 613.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.nmd.2009.06.218
Vancouver
Araújo KPC, Moreira DF, Gaiad TP, Miglino MA, Górniak SL, Feder D, Belizário JE, Ambrósio CE. Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle [Internet]. Neuromuscular Disorders. 2009 ; 19( 8-9): 613.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.nmd.2009.06.218
Artigo de periodico
Effect of chronic ethanol consumption on endothelin-1 generation and conversion of exogenous big-endothelin-1 by the rat carotid artery (2007)
Tirapelli, Carlos Renato ; Casolari, Débora A.; Yogi, Álvaro; Passaglia, Rita de Cássia Aleixo Tostes; Legros, Eurode; Lanchote, Vera Lúcia; Uyemura, Sérgio Akira; Oliveira, Ana Maria de
Source: Alcohol. Unidades: ICB, FCFRP
Subjects: ÁLCOOL, ARTÉRIAS CARÓTIDAS
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ABNT
TIRAPELLI, Carlos Renato et al. Effect of chronic ethanol consumption on endothelin-1 generation and conversion of exogenous big-endothelin-1 by the rat carotid artery. Alcohol, v. 41, n. 2, p. 77-85, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.alcohol.2007.02.004. Acesso em: 10 nov. 2024.
APA
Tirapelli, C. R., Casolari, D. A., Yogi, Á., Passaglia, R. de C. A. T., Legros, E., Lanchote, V. L., et al. (2007). Effect of chronic ethanol consumption on endothelin-1 generation and conversion of exogenous big-endothelin-1 by the rat carotid artery. Alcohol, 41( 2), 77-85. doi:10.1016/j.alcohol.2007.02.004
NLM
Tirapelli CR, Casolari DA, Yogi Á, Passaglia R de CAT, Legros E, Lanchote VL, Uyemura SA, Oliveira AM de. Effect of chronic ethanol consumption on endothelin-1 generation and conversion of exogenous big-endothelin-1 by the rat carotid artery [Internet]. Alcohol. 2007 ; 41( 2): 77-85.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.alcohol.2007.02.004
Vancouver
Tirapelli CR, Casolari DA, Yogi Á, Passaglia R de CAT, Legros E, Lanchote VL, Uyemura SA, Oliveira AM de. Effect of chronic ethanol consumption on endothelin-1 generation and conversion of exogenous big-endothelin-1 by the rat carotid artery [Internet]. Alcohol. 2007 ; 41( 2): 77-85.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.alcohol.2007.02.004
Artigo de periodico
Functional characterization and expression of endothelin receptors in rat carotid artery: involvement of nitric oxide, a vasodilator prostanoid and the opening of 'K POT.+' channels in 'ET IND.B'-induced relaxation (2005)
Tirapelli, Carlos Renato ; Casolari, Debora A.; Yogi, Alvaro; Montezano, Augusto C.; Tostes, Rita C.; Legros, Eurode; D'Orléans-Juste, Pedro; Oliveira, Ana M. de
Source: British Journal of Pharmacology. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TIRAPELLI, Carlos Renato et al. Functional characterization and expression of endothelin receptors in rat carotid artery: involvement of nitric oxide, a vasodilator prostanoid and the opening of 'K POT.+' channels in 'ET IND.B'-induced relaxation. British Journal of Pharmacology, v. 146, n. 6, p. 903-912, 2005Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0706388. Acesso em: 10 nov. 2024.
APA
Tirapelli, C. R., Casolari, D. A., Yogi, A., Montezano, A. C., Tostes, R. C., Legros, E., et al. (2005). Functional characterization and expression of endothelin receptors in rat carotid artery: involvement of nitric oxide, a vasodilator prostanoid and the opening of 'K POT.+' channels in 'ET IND.B'-induced relaxation. British Journal of Pharmacology, 146( 6), 903-912. doi:10.1038/sj.bjp.0706388
NLM
Tirapelli CR, Casolari DA, Yogi A, Montezano AC, Tostes RC, Legros E, D'Orléans-Juste P, Oliveira AM de. Functional characterization and expression of endothelin receptors in rat carotid artery: involvement of nitric oxide, a vasodilator prostanoid and the opening of 'K POT.+' channels in 'ET IND.B'-induced relaxation [Internet]. British Journal of Pharmacology. 2005 ; 146( 6): 903-912.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/sj.bjp.0706388
Vancouver
Tirapelli CR, Casolari DA, Yogi A, Montezano AC, Tostes RC, Legros E, D'Orléans-Juste P, Oliveira AM de. Functional characterization and expression of endothelin receptors in rat carotid artery: involvement of nitric oxide, a vasodilator prostanoid and the opening of 'K POT.+' channels in 'ET IND.B'-induced relaxation [Internet]. British Journal of Pharmacology. 2005 ; 146( 6): 903-912.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/sj.bjp.0706388
Artigo de periodico
Endothelin-1 contributes to the sexual differences in renal damage in DOCA-salt rats (2005)
Montezano, A. C. I.; Callera, G. E.; Mota, A. L.; Fortes, Zuleica Bruno; Nigro, Dorothy; Carvalho, Maria Helena Catelli de; Zorn, Telma Maria Tenório; Tostes, Rita de Cássia Aleixo
Source: Peptides. Unidade: ICB
Subjects: FARMACOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MONTEZANO, A. C. I. et al. Endothelin-1 contributes to the sexual differences in renal damage in DOCA-salt rats. Peptides, v. 26, n. 8, p. 1454-1462, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.peptides.2005.03.038. Acesso em: 10 nov. 2024.
APA
Montezano, A. C. I., Callera, G. E., Mota, A. L., Fortes, Z. B., Nigro, D., Carvalho, M. H. C. de, et al. (2005). Endothelin-1 contributes to the sexual differences in renal damage in DOCA-salt rats. Peptides, 26( 8), 1454-1462. doi:10.1016/j.peptides.2005.03.038
NLM
Montezano ACI, Callera GE, Mota AL, Fortes ZB, Nigro D, Carvalho MHC de, Zorn TMT, Tostes R de CA. Endothelin-1 contributes to the sexual differences in renal damage in DOCA-salt rats [Internet]. Peptides. 2005 ; 26( 8): 1454-1462.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.peptides.2005.03.038
Vancouver
Montezano ACI, Callera GE, Mota AL, Fortes ZB, Nigro D, Carvalho MHC de, Zorn TMT, Tostes R de CA. Endothelin-1 contributes to the sexual differences in renal damage in DOCA-salt rats [Internet]. Peptides. 2005 ; 26( 8): 1454-1462.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.peptides.2005.03.038
Artigo de periodico
Inflammatory mechanisms underlying the rat pulmonary neutrophil influx induced by airway exposure to staphylococcal enterotoxin type A (2005)
Desouza, Ivani A.; Franco-Penteado, Carla F.; Camargo, Enilton A.; Lima, Carmen S. P.; Teixeira, Simone A.; Muscará, Marcelo Nicolás ; De Nucci, Gilberto ; Antunes, Edson
Source: British Journal of Pharmacology. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DESOUZA, Ivani A. et al. Inflammatory mechanisms underlying the rat pulmonary neutrophil influx induced by airway exposure to staphylococcal enterotoxin type A. British Journal of Pharmacology, v. 146, n. 6, p. 781-791, 2005Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0706393. Acesso em: 10 nov. 2024.
APA
Desouza, I. A., Franco-Penteado, C. F., Camargo, E. A., Lima, C. S. P., Teixeira, S. A., Muscará, M. N., et al. (2005). Inflammatory mechanisms underlying the rat pulmonary neutrophil influx induced by airway exposure to staphylococcal enterotoxin type A. British Journal of Pharmacology, 146( 6), 781-791. doi:10.1038/sj.bjp.0706393
NLM
Desouza IA, Franco-Penteado CF, Camargo EA, Lima CSP, Teixeira SA, Muscará MN, De Nucci G, Antunes E. Inflammatory mechanisms underlying the rat pulmonary neutrophil influx induced by airway exposure to staphylococcal enterotoxin type A [Internet]. British Journal of Pharmacology. 2005 ; 146( 6): 781-791.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/sj.bjp.0706393
Vancouver
Desouza IA, Franco-Penteado CF, Camargo EA, Lima CSP, Teixeira SA, Muscará MN, De Nucci G, Antunes E. Inflammatory mechanisms underlying the rat pulmonary neutrophil influx induced by airway exposure to staphylococcal enterotoxin type A [Internet]. British Journal of Pharmacology. 2005 ; 146( 6): 781-791.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/sj.bjp.0706393
Artigo de periodico
Endothelin receptor antagonists: another potential alternative for cardiovascular diseases (2005)
Tostes, Rita C. A.; Muscará, Marcelo Nicolás
Source: Current Drug Targets. Cardiovascular and Hematological Disorders. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TOSTES, Rita C. A. e MUSCARÁ, Marcelo Nicolás. Endothelin receptor antagonists: another potential alternative for cardiovascular diseases. Current Drug Targets. Cardiovascular and Hematological Disorders, v. 5, n. 4, p. 287-301, 2005Tradução . . Acesso em: 10 nov. 2024.
APA
Tostes, R. C. A., & Muscará, M. N. (2005). Endothelin receptor antagonists: another potential alternative for cardiovascular diseases. Current Drug Targets. Cardiovascular and Hematological Disorders, 5( 4), 287-301.
NLM
Tostes RCA, Muscará MN. Endothelin receptor antagonists: another potential alternative for cardiovascular diseases. Current Drug Targets. Cardiovascular and Hematological Disorders. 2005 ; 5( 4): 287-301.[citado 2024 nov. 10 ]
Vancouver
Tostes RCA, Muscará MN. Endothelin receptor antagonists: another potential alternative for cardiovascular diseases. Current Drug Targets. Cardiovascular and Hematological Disorders. 2005 ; 5( 4): 287-301.[citado 2024 nov. 10 ]
Artigo de periodico
Regulation of allergic lung inflammation in rats: interaction between estradiol and corticosterone (2004)
Oliveira, Ana Paula Ligeiro de; Oliveira-Filho, Ricardo Martins; Silva, Zilma Lúcia da; Borelli, Primavera ; Lima, Wothan Tavares de
Source: Neuroimmunomodulation. Unidades: FCF, ICB
Subjects: INFLAMAÇÃO, ASMA, PULMÃO, BRONCOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Ana Paula Ligeiro de et al. Regulation of allergic lung inflammation in rats: interaction between estradiol and corticosterone. Neuroimmunomodulation, v. 11, n. 1, p. 20-27, 2004Tradução . . Acesso em: 10 nov. 2024.
APA
Oliveira, A. P. L. de, Oliveira-Filho, R. M., Silva, Z. L. da, Borelli, P., & Lima, W. T. de. (2004). Regulation of allergic lung inflammation in rats: interaction between estradiol and corticosterone. Neuroimmunomodulation, 11( 1), 20-27.
NLM
Oliveira APL de, Oliveira-Filho RM, Silva ZL da, Borelli P, Lima WT de. Regulation of allergic lung inflammation in rats: interaction between estradiol and corticosterone. Neuroimmunomodulation. 2004 ; 11( 1): 20-27.[citado 2024 nov. 10 ]
Vancouver
Oliveira APL de, Oliveira-Filho RM, Silva ZL da, Borelli P, Lima WT de. Regulation of allergic lung inflammation in rats: interaction between estradiol and corticosterone. Neuroimmunomodulation. 2004 ; 11( 1): 20-27.[citado 2024 nov. 10 ]

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