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LARA, Priscila Guirão et al. AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis. Frontiers in Immunology, v. 12, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.681671. Acesso em: 09 out. 2024.
APA
Lara, P. G., Esteves, E. V. da S., Campos, H. S. de, Assis, J. B. de, Henrique, M. de O., Barros, M. S. de, et al. (2021). AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis. Frontiers in Immunology, 12, 1-15. doi:10.3389/fimmu.2021.681671
NLM
Lara PG, Esteves EV da S, Campos HS de, Assis JB de, Henrique M de O, Barros MS de, Santos Neto L, Silva Junior PI, Martins J de O, Cardoso CR de B, Ribeiro JMC, Nunes A de S. AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis [Internet]. Frontiers in Immunology. 2021 ; 12 1-15.[citado 2024 out. 09 ] Available from: https://doi.org/10.3389/fimmu.2021.681671
Vancouver
Lara PG, Esteves EV da S, Campos HS de, Assis JB de, Henrique M de O, Barros MS de, Santos Neto L, Silva Junior PI, Martins J de O, Cardoso CR de B, Ribeiro JMC, Nunes A de S. AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis [Internet]. Frontiers in Immunology. 2021 ; 12 1-15.[citado 2024 out. 09 ] Available from: https://doi.org/10.3389/fimmu.2021.681671
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KLIONSKY, Daniel J et al. Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy, v. 17, n. 1, p. 1-382, 2021Tradução . . Disponível em: https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280. Acesso em: 09 out. 2024.
APA
Klionsky, D. J., Baptista, M. da S., Martins, J. de O., Brum, P. C., Ferreira, J. C. B., Palmisano, G., et al. (2021). Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy, 17( 1), 1-382. Recuperado de https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280
NLM
Klionsky DJ, Baptista M da S, Martins J de O, Brum PC, Ferreira JCB, Palmisano G, Câmara NOS, Leopoldino AM, Araújo TF. Guidelines for the use and interpretation of assays for monitoring autophagy [Internet]. Autophagy. 2021 ; 17( 1): 1-382.[citado 2024 out. 09 ] Available from: https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280
Vancouver
Klionsky DJ, Baptista M da S, Martins J de O, Brum PC, Ferreira JCB, Palmisano G, Câmara NOS, Leopoldino AM, Araújo TF. Guidelines for the use and interpretation of assays for monitoring autophagy [Internet]. Autophagy. 2021 ; 17( 1): 1-382.[citado 2024 out. 09 ] Available from: https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280
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MINI, Camila Alessandra et al. Immortalized keratinocytes cells generates an effective model of Epidermal Human Equivalent for irritation and corrosion tests. Toxicology in Vitro, v. 71, p. 1-9, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2020.105069. Acesso em: 09 out. 2024.
APA
Mini, C. A., Dreossi, S. A. C., Abe, F. R., Maria-Engler, S. S., & Oliveira, D. P. de. (2021). Immortalized keratinocytes cells generates an effective model of Epidermal Human Equivalent for irritation and corrosion tests. Toxicology in Vitro, 71, 1-9. doi:10.1016/j.tiv.2020.105069
NLM
Mini CA, Dreossi SAC, Abe FR, Maria-Engler SS, Oliveira DP de. Immortalized keratinocytes cells generates an effective model of Epidermal Human Equivalent for irritation and corrosion tests [Internet]. Toxicology in Vitro. 2021 ; 71 1-9.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.tiv.2020.105069
Vancouver
Mini CA, Dreossi SAC, Abe FR, Maria-Engler SS, Oliveira DP de. Immortalized keratinocytes cells generates an effective model of Epidermal Human Equivalent for irritation and corrosion tests [Internet]. Toxicology in Vitro. 2021 ; 71 1-9.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.tiv.2020.105069
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MINI, Camila Alessandra et al. Immortalized equivalent human epidermis as a platform to evaluation hair dyes toxicity: efficiency comparison between 3D and monolayer culture. Chemico-Biological Interactions, v. 330, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2020.109227. Acesso em: 09 out. 2024.
APA
Mini, C. A., Dorta, D. J., Maria-Engler, S. S., & Oliveira, D. P. de. (2020). Immortalized equivalent human epidermis as a platform to evaluation hair dyes toxicity: efficiency comparison between 3D and monolayer culture. Chemico-Biological Interactions, 330, 1-9. doi:10.1016/j.cbi.2020.109227
NLM
Mini CA, Dorta DJ, Maria-Engler SS, Oliveira DP de. Immortalized equivalent human epidermis as a platform to evaluation hair dyes toxicity: efficiency comparison between 3D and monolayer culture [Internet]. Chemico-Biological Interactions. 2020 ; 330 1-9.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.cbi.2020.109227
Vancouver
Mini CA, Dorta DJ, Maria-Engler SS, Oliveira DP de. Immortalized equivalent human epidermis as a platform to evaluation hair dyes toxicity: efficiency comparison between 3D and monolayer culture [Internet]. Chemico-Biological Interactions. 2020 ; 330 1-9.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.cbi.2020.109227
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LEO, Thais Canassa de et al. Engineering of galectin-3 for glycan-binding optical imaging. Biochemical and Biophysical Research Communication, v. 521, n. 3, p. 674-680, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2019.10.161. Acesso em: 09 out. 2024.
APA
Leo, T. C. de, Santos, S. N. dos, Andrade, C. D. C., Ricci, E., Turato, W. M., Lopes, N. P., et al. (2020). Engineering of galectin-3 for glycan-binding optical imaging. Biochemical and Biophysical Research Communication, 521( 3), 674-680. doi:10.1016/j.bbrc.2019.10.161
NLM
Leo TC de, Santos SN dos, Andrade CDC, Ricci E, Turato WM, Lopes NP, Oliveira RS, Bernardes ES, Dias-Baruffi M. Engineering of galectin-3 for glycan-binding optical imaging [Internet]. Biochemical and Biophysical Research Communication. 2020 ; 521( 3): 674-680.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.bbrc.2019.10.161
Vancouver
Leo TC de, Santos SN dos, Andrade CDC, Ricci E, Turato WM, Lopes NP, Oliveira RS, Bernardes ES, Dias-Baruffi M. Engineering of galectin-3 for glycan-binding optical imaging [Internet]. Biochemical and Biophysical Research Communication. 2020 ; 521( 3): 674-680.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.bbrc.2019.10.161
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Brazilian Journal of Pharmaceutical Sciences. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Universidade de São Paulo - USP, Faculdade de Ciências Farmacêuticas - FCF. . Acesso em: 09 out. 2024. , 2019
APA
Brazilian Journal of Pharmaceutical Sciences. (2019). Brazilian Journal of Pharmaceutical Sciences. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Universidade de São Paulo - USP, Faculdade de Ciências Farmacêuticas - FCF.
NLM
Brazilian Journal of Pharmaceutical Sciences. Brazilian Journal of Pharmaceutical Sciences. 2019 ;[citado 2024 out. 09 ]
Vancouver
Brazilian Journal of Pharmaceutical Sciences. Brazilian Journal of Pharmaceutical Sciences. 2019 ;[citado 2024 out. 09 ]
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DONADIO, Janaina Lombello Santos et al. Genetic variants in selenoprotein genes modulate biomarkers of selenium status in response to Brazil nut supplementation (the SU.BRA.NUT study). Clinical Nutrition, v. 38, n. 2, p. 539-548, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.clnu.2018.03.011. Acesso em: 09 out. 2024.
APA
Donadio, J. L. S., Rogero, M. M., Guerra-Shinohara, E. M., Barbosa Júnior, F., Desmarchelier, C., Borel, P., et al. (2019). Genetic variants in selenoprotein genes modulate biomarkers of selenium status in response to Brazil nut supplementation (the SU.BRA.NUT study). Clinical Nutrition, 38( 2), 539-548. doi:10.1016/j.clnu.2018.03.011
NLM
Donadio JLS, Rogero MM, Guerra-Shinohara EM, Barbosa Júnior F, Desmarchelier C, Borel P, Sneddon AA, Hesketh JE, Cozzolino SMF. Genetic variants in selenoprotein genes modulate biomarkers of selenium status in response to Brazil nut supplementation (the SU.BRA.NUT study) [Internet]. Clinical Nutrition. 2019 ; 38( 2): 539-548.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.clnu.2018.03.011
Vancouver
Donadio JLS, Rogero MM, Guerra-Shinohara EM, Barbosa Júnior F, Desmarchelier C, Borel P, Sneddon AA, Hesketh JE, Cozzolino SMF. Genetic variants in selenoprotein genes modulate biomarkers of selenium status in response to Brazil nut supplementation (the SU.BRA.NUT study) [Internet]. Clinical Nutrition. 2019 ; 38( 2): 539-548.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.clnu.2018.03.011
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ZANONI, Thalita Boldrin et al. Allergens of permanent hair dyes induces epidermal damage, skin barrier loss and IL-1 α increase in epidermal in vitro model. Food and Chemical Toxicology, v. 112, p. 265-272, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.fct.2017.12.033. Acesso em: 09 out. 2024.
APA
Zanoni, T. B., Pedrosa, T. do N., Catarino, C. M., Spiekstra, S. W., Oliveira, D. P. de, Hartog, G. J. M. D., et al. (2018). Allergens of permanent hair dyes induces epidermal damage, skin barrier loss and IL-1 α increase in epidermal in vitro model. Food and Chemical Toxicology, 112, 265-272. doi:10.1016/j.fct.2017.12.033
NLM
Zanoni TB, Pedrosa T do N, Catarino CM, Spiekstra SW, Oliveira DP de, Hartog GJMD, Bast A, Hagemann G, Gibbs S, Barros SB de M, Maria-Engler SS. Allergens of permanent hair dyes induces epidermal damage, skin barrier loss and IL-1 α increase in epidermal in vitro model [Internet]. Food and Chemical Toxicology. 2018 ; 112 265-272.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.fct.2017.12.033
Vancouver
Zanoni TB, Pedrosa T do N, Catarino CM, Spiekstra SW, Oliveira DP de, Hartog GJMD, Bast A, Hagemann G, Gibbs S, Barros SB de M, Maria-Engler SS. Allergens of permanent hair dyes induces epidermal damage, skin barrier loss and IL-1 α increase in epidermal in vitro model [Internet]. Food and Chemical Toxicology. 2018 ; 112 265-272.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.fct.2017.12.033
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
Brazilian Journal of Pharmaceutical Sciences. . São Paulo: Universidade de São Paulo - USP, Faculdade de Ciências Farmacêuticas - FCF. . Acesso em: 09 out. 2024. , 2018
APA
Brazilian Journal of Pharmaceutical Sciences. (2018). Brazilian Journal of Pharmaceutical Sciences. São Paulo: Universidade de São Paulo - USP, Faculdade de Ciências Farmacêuticas - FCF.
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SOUZA, Patrícia Reis de et al. Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis. Immunobiology, v. 223, n. 10, p. 577-585, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2018.07.003. Acesso em: 09 out. 2024.
APA
Souza, P. R. de, Guimarães, F. R., Sales-Campos, H., Bonfá, G., Nardini, V., Chica, J. E. L., et al. (2018). Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis. Immunobiology, 223( 10), 577-585. doi:10.1016/j.imbio.2018.07.003
NLM
Souza PR de, Guimarães FR, Sales-Campos H, Bonfá G, Nardini V, Chica JEL, Turato WM, Silva JS da, Zamboni DS, Cardoso CR de B. Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis [Internet]. Immunobiology. 2018 ; 223( 10): 577-585.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.imbio.2018.07.003
Vancouver
Souza PR de, Guimarães FR, Sales-Campos H, Bonfá G, Nardini V, Chica JEL, Turato WM, Silva JS da, Zamboni DS, Cardoso CR de B. Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis [Internet]. Immunobiology. 2018 ; 223( 10): 577-585.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.imbio.2018.07.003
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TALARICO, Juliana Nery de Souza et al. Association between heavy metal exposure and poor working memory and possible mediation effect of antioxidant defenses during aging. Science of the Total Environmental, v. 575, p. 750-757, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.scitotenv.2016.09.121. Acesso em: 09 out. 2024.
APA
Talarico, J. N. de S., Marcourakis, T., Barbosa Júnior, F., Barros, S. B. de M., Rivelli, D. P., Pompéia, S., et al. (2017). Association between heavy metal exposure and poor working memory and possible mediation effect of antioxidant defenses during aging. Science of the Total Environmental, 575, 750-757. doi:10.1016/j.scitotenv.2016.09.121
NLM
Talarico JN de S, Marcourakis T, Barbosa Júnior F, Barros SB de M, Rivelli DP, Pompéia S, Caramelli P, Plusquellec P, Lupien SJ, Catucci RF, Alves AR, Suchecki D. Association between heavy metal exposure and poor working memory and possible mediation effect of antioxidant defenses during aging [Internet]. Science of the Total Environmental. 2017 ; 575 750-757.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.scitotenv.2016.09.121
Vancouver
Talarico JN de S, Marcourakis T, Barbosa Júnior F, Barros SB de M, Rivelli DP, Pompéia S, Caramelli P, Plusquellec P, Lupien SJ, Catucci RF, Alves AR, Suchecki D. Association between heavy metal exposure and poor working memory and possible mediation effect of antioxidant defenses during aging [Internet]. Science of the Total Environmental. 2017 ; 575 750-757.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.scitotenv.2016.09.121
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TALARICO, Juliana Nery de Souza et al. Lead exposure is related to hypercortisolemic profiles and allostatic load in Brazilian older adults. Environmental Research, v. 154, p. 261–268, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.envres.2017.01.012. Acesso em: 09 out. 2024.
APA
Talarico, J. N. de S., Suchecki, D., Juster, R. P., Plusquellec, P., Barbosa Júnior, F., Bunscheit, V., et al. (2017). Lead exposure is related to hypercortisolemic profiles and allostatic load in Brazilian older adults. Environmental Research, 154, 261–268. doi:10.1016/j.envres.2017.01.012
NLM
Talarico JN de S, Suchecki D, Juster RP, Plusquellec P, Barbosa Júnior F, Bunscheit V, Marcourakis T, Matos TM de, Lupien SJ. Lead exposure is related to hypercortisolemic profiles and allostatic load in Brazilian older adults [Internet]. Environmental Research. 2017 ; 154 261–268.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.envres.2017.01.012
Vancouver
Talarico JN de S, Suchecki D, Juster RP, Plusquellec P, Barbosa Júnior F, Bunscheit V, Marcourakis T, Matos TM de, Lupien SJ. Lead exposure is related to hypercortisolemic profiles and allostatic load in Brazilian older adults [Internet]. Environmental Research. 2017 ; 154 261–268.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.envres.2017.01.012
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TURATO, Walter Miguel et al. The impact of intestinal resection on the immune function of short bowel syndrome patients. Human Immunology, v. 77, p. 1202-1208, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.humimm.2016.07.237. Acesso em: 09 out. 2024.
APA
Turato, W. M., Sales-Campos, H., Braga, C. B. M., Cunha, S. F. de C. da, Silvah, J. H., Silva, J. S. da, et al. (2016). The impact of intestinal resection on the immune function of short bowel syndrome patients. Human Immunology, 77, 1202-1208. doi:10.1016/j.humimm.2016.07.237
NLM
Turato WM, Sales-Campos H, Braga CBM, Cunha SF de C da, Silvah JH, Silva JS da, Marchini JS, Cardoso CR de B. The impact of intestinal resection on the immune function of short bowel syndrome patients [Internet]. Human Immunology. 2016 ; 77 1202-1208.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.humimm.2016.07.237
Vancouver
Turato WM, Sales-Campos H, Braga CBM, Cunha SF de C da, Silvah JH, Silva JS da, Marchini JS, Cardoso CR de B. The impact of intestinal resection on the immune function of short bowel syndrome patients [Internet]. Human Immunology. 2016 ; 77 1202-1208.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.humimm.2016.07.237
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FRONZA, Marcio et al. Hyaluronidase decreases neutrophils infiltration to the inflammatory site. Inflammation Research, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00011-016-0935-0. Acesso em: 09 out. 2024.
APA
Fronza, M., Muhr, C., Silveira, D. S. C. da, Sorgi, C. A., Rodrigues, S. F. de P., Farsky, S. H. P., et al. (2016). Hyaluronidase decreases neutrophils infiltration to the inflammatory site. Inflammation Research, 1-10. doi:10.1007/s00011-016-0935-0
NLM
Fronza M, Muhr C, Silveira DSC da, Sorgi CA, Rodrigues SF de P, Farsky SHP, Silva FWG de P e, Merfort I, Faccioli LH. Hyaluronidase decreases neutrophils infiltration to the inflammatory site [Internet]. Inflammation Research. 2016 ; 1-10.[citado 2024 out. 09 ] Available from: https://doi.org/10.1007/s00011-016-0935-0
Vancouver
Fronza M, Muhr C, Silveira DSC da, Sorgi CA, Rodrigues SF de P, Farsky SHP, Silva FWG de P e, Merfort I, Faccioli LH. Hyaluronidase decreases neutrophils infiltration to the inflammatory site [Internet]. Inflammation Research. 2016 ; 1-10.[citado 2024 out. 09 ] Available from: https://doi.org/10.1007/s00011-016-0935-0