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Artigo de periodico
Advanced glycation end products-induced insulin resistance involves repression of skeletal muscle GLUT4 expression (2018)
Pinto Júnior, Danilo Antônio Corrêa; Silva, Karolline Santana da; Michalani, Maria Luiza Estimo; Yonamine, Caio Yogi; Esteves, João Victor Del Conti; Fabre, Nelly Takashima; Thieme, Karina ; Catanozi, Sérgio; Okamato, Maristela Mitiko; Seraphim, Patricia Monteiro; Giannela, Maria Lucia Cardillo Correa; Passarelli, Marisa; Machado, Ubiratan Fabres
Source: Scientific reports. Unidades: FM, ICB
Subjects: MÚSCULOS, RETÍCULO ENDOPLASMÁTICO, ALBUMINAS, RATOS, INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINTO JÚNIOR, Danilo Antônio Corrêa et al. Advanced glycation end products-induced insulin resistance involves repression of skeletal muscle GLUT4 expression. Scientific reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-26482-6. Acesso em: 21 jun. 2024.
APA
Pinto Júnior, D. A. C., Silva, K. S. da, Michalani, M. L. E., Yonamine, C. Y., Esteves, J. V. D. C., Fabre, N. T., et al. (2018). Advanced glycation end products-induced insulin resistance involves repression of skeletal muscle GLUT4 expression. Scientific reports, 8. doi:10.1038/s41598-018-26482-6
NLM
Pinto Júnior DAC, Silva KS da, Michalani MLE, Yonamine CY, Esteves JVDC, Fabre NT, Thieme K, Catanozi S, Okamato MM, Seraphim PM, Giannela MLCC, Passarelli M, Machado UF. Advanced glycation end products-induced insulin resistance involves repression of skeletal muscle GLUT4 expression [Internet]. Scientific reports. 2018 ; 8[citado 2024 jun. 21 ] Available from: https://doi.org/10.1038/s41598-018-26482-6
Vancouver
Pinto Júnior DAC, Silva KS da, Michalani MLE, Yonamine CY, Esteves JVDC, Fabre NT, Thieme K, Catanozi S, Okamato MM, Seraphim PM, Giannela MLCC, Passarelli M, Machado UF. Advanced glycation end products-induced insulin resistance involves repression of skeletal muscle GLUT4 expression [Internet]. Scientific reports. 2018 ; 8[citado 2024 jun. 21 ] Available from: https://doi.org/10.1038/s41598-018-26482-6
Artigo de periodico
AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages (2018)
Iborra, Rodrigo Tallada; Lima, Adriana Machado Saldiba de; Okuda, Ligia Shimabukuro; Pinto, Paula Ramos; Nakandakare, Edna Regina; Machado, Ubiratan Fabres ; Giannella, Maria Lúcia Cardillo Côrrea; Pickford, Russell; Woods, Tom; Brimble, Margaret Anne; Rye, Kerry-Anne; Lu, Rui; Yokoyama, Shinji; Passarelli, Marisa
Source: Journal of diabetes and its complications. Unidades: FM, ICB
Subjects: DIABETES MELLITUS, ALBUMINAS, EXPRESSÃO GÊNICA, ARTERIOSCLEROSE, MACRÓFAGOS, LISOSSOMOS
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ABNT
IBORRA, Rodrigo Tallada et al. AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages. Journal of diabetes and its complications, v. 32, n. 1, p. 1-10, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2017.09.012. Acesso em: 21 jun. 2024.
APA
Iborra, R. T., Lima, A. M. S. de, Okuda, L. S., Pinto, P. R., Nakandakare, E. R., Machado, U. F., et al. (2018). AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages. Journal of diabetes and its complications, 32( 1), 1-10. doi:10.1016/j.jdiacomp.2017.09.012
NLM
Iborra RT, Lima AMS de, Okuda LS, Pinto PR, Nakandakare ER, Machado UF, Giannella MLCC, Pickford R, Woods T, Brimble MA, Rye K-A, Lu R, Yokoyama S, Passarelli M. AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages [Internet]. Journal of diabetes and its complications. 2018 ; 32( 1): 1-10.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1016/j.jdiacomp.2017.09.012
Vancouver
Iborra RT, Lima AMS de, Okuda LS, Pinto PR, Nakandakare ER, Machado UF, Giannella MLCC, Pickford R, Woods T, Brimble MA, Rye K-A, Lu R, Yokoyama S, Passarelli M. AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages [Internet]. Journal of diabetes and its complications. 2018 ; 32( 1): 1-10.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1016/j.jdiacomp.2017.09.012
Artigo de periodico
Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products (2017)
Fabre, Nelly T.; Thieme, Karina ; Silva, Karolline S.; Catanozi, Sergio; Cavaleiro, Ana Mercedes; Pinto jr, Danilo A. C.; Maristela M. Okamoto,; Morais, Mychel Raony P. T.; Falquetto, Bárbara ; Zorn, Telma M.; Machado, Ubiratan Fabres ; Passarelli, Marisa; Correa-Giannella, Maria Lúcia
Source: Molecular and Cellular Endocrinology. Unidades: FM, ICB
Subjects: FÍGADO, DOENÇA CRÔNICA, INSULINA (RESISTÊNCIA), RATOS
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ABNT
FABRE, Nelly T. et al. Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products. Molecular and Cellular Endocrinology, v. 447, p. 116-124, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.02.035. Acesso em: 21 jun. 2024.
APA
Fabre, N. T., Thieme, K., Silva, K. S., Catanozi, S., Cavaleiro, A. M., Pinto jr, D. A. C., et al. (2017). Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products. Molecular and Cellular Endocrinology, 447, 116-124. doi:10.1016/j.mce.2017.02.035
NLM
Fabre NT, Thieme K, Silva KS, Catanozi S, Cavaleiro AM, Pinto jr DAC, Maristela M. Okamoto, Morais MRPT, Falquetto B, Zorn TM, Machado UF, Passarelli M, Correa-Giannella ML. Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products [Internet]. Molecular and Cellular Endocrinology. 2017 ; 447 116-124.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1016/j.mce.2017.02.035
Vancouver
Fabre NT, Thieme K, Silva KS, Catanozi S, Cavaleiro AM, Pinto jr DAC, Maristela M. Okamoto, Morais MRPT, Falquetto B, Zorn TM, Machado UF, Passarelli M, Correa-Giannella ML. Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products [Internet]. Molecular and Cellular Endocrinology. 2017 ; 447 116-124.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1016/j.mce.2017.02.035
Artigo de periodico
Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes (2016)
Mohammedi, Kamel; Patente, Thiago A.; Bellili-Muñoz, Naima; Driss, Fathi; Le Nagard, Hervé; Fumeron, Frédéric; Roussel, Ronan; Hadjadj, Samy; Corrêa-Giannella, Maria Lúcia; Marre, Michel; Velho, Gilberto
Source: Metabolism Clinical and Experimental. Unidade: FM
Subjects: ENZIMAS (METABOLISMO), ESTRESSE OXIDATIVO, DIABETES MELLITUS (COMPLICAÇÕES), NEFROPATIAS (ETIOLOGIA), PEROXIDASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOHAMMEDI, Kamel et al. Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes. Metabolism Clinical and Experimental, v. 65, n. 2, p. 12-19, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2015.10.004. Acesso em: 21 jun. 2024.
APA
Mohammedi, K., Patente, T. A., Bellili-Muñoz, N., Driss, F., Le Nagard, H., Fumeron, F., et al. (2016). Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes. Metabolism Clinical and Experimental, 65( 2), 12-19. doi:10.1016/j.metabol.2015.10.004
NLM
Mohammedi K, Patente TA, Bellili-Muñoz N, Driss F, Le Nagard H, Fumeron F, Roussel R, Hadjadj S, Corrêa-Giannella ML, Marre M, Velho G. Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes [Internet]. Metabolism Clinical and Experimental. 2016 ; 65( 2): 12-19.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1016/j.metabol.2015.10.004
Vancouver
Mohammedi K, Patente TA, Bellili-Muñoz N, Driss F, Le Nagard H, Fumeron F, Roussel R, Hadjadj S, Corrêa-Giannella ML, Marre M, Velho G. Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes [Internet]. Metabolism Clinical and Experimental. 2016 ; 65( 2): 12-19.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1016/j.metabol.2015.10.004
Artigo de periodico
N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats (2016)
Thieme, Karina ; Silva, Karolline Santana da; Fabre, Nelly Takashima; Catazoni, Sergio; Monteiro, Maria Beatriz; Bezerra, Daniele Pereira Santos; Pessoa, Juliana Martins da Costa; Souza, Maria Oliveira de ; Machado, Ubiratan Fabres ; Passarelli, Marisa; Giannella, Maria Lucia Gardillo Correa
Source: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Subjects: ESTRESSE OXIDATIVO, NEFROPATIAS, RIM, RATOS
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ABNT
THIEME, Karina et al. N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats. Cellular Physiology and Biochemistry, v. 40, n. 3-4, p. 608-620, 2016Tradução . . Disponível em: https://doi.org/10.1159/000452574. Acesso em: 21 jun. 2024.
APA
Thieme, K., Silva, K. S. da, Fabre, N. T., Catazoni, S., Monteiro, M. B., Bezerra, D. P. S., et al. (2016). N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats. Cellular Physiology and Biochemistry, 40( 3-4), 608-620. doi:10.1159/000452574
NLM
Thieme K, Silva KS da, Fabre NT, Catazoni S, Monteiro MB, Bezerra DPS, Pessoa JM da C, Souza MO de, Machado UF, Passarelli M, Giannella MLGC. N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats [Internet]. Cellular Physiology and Biochemistry. 2016 ; 40( 3-4): 608-620.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1159/000452574
Vancouver
Thieme K, Silva KS da, Fabre NT, Catazoni S, Monteiro MB, Bezerra DPS, Pessoa JM da C, Souza MO de, Machado UF, Passarelli M, Giannella MLGC. N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats [Internet]. Cellular Physiology and Biochemistry. 2016 ; 40( 3-4): 608-620.[citado 2024 jun. 21 ] Available from: https://doi.org/10.1159/000452574
Artigo de periodico-carta/editorial
SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial] (2013)
Corrêa-Giannella, Maria Lucia; Machado, Ubiratan Fabres
Source: Pharmacogenomics. Unidades: FM, ICB
Subjects: INSULINA (RESISTÊNCIA), DIABETES MELLITUS, FARMACOLOGIA CLÍNICA, GENES
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ABNT
CORRÊA-GIANNELLA, Maria Lucia e MACHADO, Ubiratan Fabres. SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial]. Pharmacogenomics. London: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://www.futuremedicine.com/doi/pdf/10.2217/pgs.13.45. Acesso em: 21 jun. 2024. , 2013
APA
Corrêa-Giannella, M. L., & Machado, U. F. (2013). SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial]. Pharmacogenomics. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.2217/pgs.10.93
NLM
Corrêa-Giannella ML, Machado UF. SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial] [Internet]. Pharmacogenomics. 2013 ; 14( 8): 847-850.[citado 2024 jun. 21 ] Available from: http://www.futuremedicine.com/doi/pdf/10.2217/pgs.13.45
Vancouver
Corrêa-Giannella ML, Machado UF. SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial] [Internet]. Pharmacogenomics. 2013 ; 14( 8): 847-850.[citado 2024 jun. 21 ] Available from: http://www.futuremedicine.com/doi/pdf/10.2217/pgs.13.45
Artigo de periodico
Glutathione Ethyl Ester Supplementation during Pancreatic Islet Isolation Improves Viability and Transplant Outcomes in a Murine Marginal Islet Mass Model (2013)
Amaral, Alexandre S. Raposo do; Pawlick, Rena L.; Rodrigues, Erika; Costal, Flavia; Pepper, Andrew; Galvão, Flávio H. Ferreira; Correa-Giannella, Maria Lucia; Shapiro, A. M.James
Source: PLOS ONE. Unidade: FM
Subjects: GLUTATIONA TRANSFERASE, EXPERIMENTOS ANIMAIS, TERAPIAS COMPLEMENTARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMARAL, Alexandre S. Raposo do et al. Glutathione Ethyl Ester Supplementation during Pancreatic Islet Isolation Improves Viability and Transplant Outcomes in a Murine Marginal Islet Mass Model. PLOS ONE, v. 8, n. 2, p. 8 , 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0055288. Acesso em: 21 jun. 2024.
APA
Amaral, A. S. R. do, Pawlick, R. L., Rodrigues, E., Costal, F., Pepper, A., Galvão, F. H. F., et al. (2013). Glutathione Ethyl Ester Supplementation during Pancreatic Islet Isolation Improves Viability and Transplant Outcomes in a Murine Marginal Islet Mass Model. PLOS ONE, 8( 2), 8 . doi:10.1371/journal.pone.0055288
NLM
Amaral ASR do, Pawlick RL, Rodrigues E, Costal F, Pepper A, Galvão FHF, Correa-Giannella ML, Shapiro AMJ. Glutathione Ethyl Ester Supplementation during Pancreatic Islet Isolation Improves Viability and Transplant Outcomes in a Murine Marginal Islet Mass Model [Internet]. PLOS ONE. 2013 ; 8( 2): 8 .[citado 2024 jun. 21 ] Available from: https://doi.org/10.1371/journal.pone.0055288
Vancouver
Amaral ASR do, Pawlick RL, Rodrigues E, Costal F, Pepper A, Galvão FHF, Correa-Giannella ML, Shapiro AMJ. Glutathione Ethyl Ester Supplementation during Pancreatic Islet Isolation Improves Viability and Transplant Outcomes in a Murine Marginal Islet Mass Model [Internet]. PLOS ONE. 2013 ; 8( 2): 8 .[citado 2024 jun. 21 ] Available from: https://doi.org/10.1371/journal.pone.0055288

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