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Artigo de periodico
Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism (2018)
Nascimento, M.; Piran, R.; Da Costa, R. M.; Giordani, M. A.; Carneiro, Fernando Silva; Aguiar, D. H.; Dias, M. C.; Sugizaki, M. M.; Luvizotto, R. A.; Nascimento, A. F.; Bomfim, G. F.
Source: Life Sciences. Unidade: FMRP
Subjects: AORTA, FÍGADO, PRESSÃO SANGUÍNEA, ENDOTÉLIO VASCULAR, DOENÇAS VASCULARES, RATOS WISTAR
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ABNT
NASCIMENTO, M. et al. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, v. 212, p. 168-175, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.09.051. Acesso em: 04 nov. 2024.
APA
Nascimento, M., Piran, R., Da Costa, R. M., Giordani, M. A., Carneiro, F. S., Aguiar, D. H., et al. (2018). Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, 212, 168-175. doi:10.1016/j.lfs.2018.09.051
NLM
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Vancouver
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Artigo de periodico
O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway (2018)
Miguez, Jéssica S. G.; Dela Justina, Vanessa; Bressan, Alecsander F. M.; Marchi, Patrícia G. F.; França, Adenilda C. Honorio; Carneiro, Fernando Silva; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes; Giachini, Fernanda R.; Lima, Victor V.
Source: Life Sciences. Unidade: FMRP
Subjects: CITOCINAS, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
MIGUEZ, Jéssica S. G. et al. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, v. 209, p. 78-84, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.07.058. Acesso em: 04 nov. 2024.
APA
Miguez, J. S. G., Dela Justina, V., Bressan, A. F. M., Marchi, P. G. F., França, A. C. H., Carneiro, F. S., et al. (2018). O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, 209, 78-84. doi:10.1016/j.lfs.2018.07.058
NLM
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Vancouver
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Artigo de periodico
O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels (2018)
Dela Justina, Vanessa; Passos Junior, Rinaldo R. dos; Bressan, Alecsander F.; Passaglia, Rita de Cássia Aleixo Tostes; Carneiro, Fernando Silva; Soares, Thaigra S.; Volpato, Gustavo T.; Lima, Victor Vitorino; Martin, Sebastian San; Giachini, Fernanda R.
Source: Life Sciences. Unidade: FMRP
Subjects: GRAVIDEZ, HIPERGLICEMIA, TROFOBLASTO, CÉLULAS ENDOTELIAIS, RATOS
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ABNT
DELA JUSTINA, Vanessa et al. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, v. 205, p. 18-25, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.05.013. Acesso em: 04 nov. 2024.
APA
Dela Justina, V., Passos Junior, R. R. dos, Bressan, A. F., Passaglia, R. de C. A. T., Carneiro, F. S., Soares, T. S., et al. (2018). O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, 205, 18-25. doi:10.1016/j.lfs.2018.05.013
NLM
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Vancouver
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Artigo de periodico
Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats (2018)
Riberio, Diogo Farias; Cella, Paola Sanchez; Silva, Lilian Eslaine Costa Mendes da; Jordão Júnior, Alceu Afonso; Deminice, Rafael
Source: Life Sciences. Unidade: FMRP
Subjects: EXERCÍCIO FÍSICO, RATOS, PESOS E MEDIDAS CORPORAIS, FISIOLOGIA ANIMAL
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ABNT
RIBERIO, Diogo Farias et al. Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats. Life Sciences, v. 196, p. 63-68, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.01.003. Acesso em: 04 nov. 2024.
APA
Riberio, D. F., Cella, P. S., Silva, L. E. C. M. da, Jordão Júnior, A. A., & Deminice, R. (2018). Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats. Life Sciences, 196, 63-68. doi:10.1016/j.lfs.2018.01.003
NLM
Riberio DF, Cella PS, Silva LECM da, Jordão Júnior AA, Deminice R. Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats [Internet]. Life Sciences. 2018 ; 196 63-68.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.01.003
Vancouver
Riberio DF, Cella PS, Silva LECM da, Jordão Júnior AA, Deminice R. Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats [Internet]. Life Sciences. 2018 ; 196 63-68.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.01.003
Artigo de periodico
Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids (2017)
Diniz, Mariana C.; Olivon, Vania C.; Tavares, Lívia D.; Simplicio, Janaina A.; Gonzaga, Natália A.; Souza, Daniele G. de; Bendhack, Lusiane Maria; Tirapelli, Carlos Renato ; Bonaventura, Daniella
Source: Life Sciences. Unidades: FCFRP, EERP
Subjects: NITRATOS, AORTA, VASODILATAÇÃO, OXIGÊNIO
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ABNT
DINIZ, Mariana C. et al. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, v. 176, p. 26-34, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.03.016. Acesso em: 04 nov. 2024.
APA
Diniz, M. C., Olivon, V. C., Tavares, L. D., Simplicio, J. A., Gonzaga, N. A., Souza, D. G. de, et al. (2017). Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, 176, 26-34. doi:10.1016/j.lfs.2017.03.016
NLM
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Vancouver
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Artigo de periodico
Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases (2016)
Gohar, Eman Y; Giachini, Fernanda R; Pollock, David M; Tostes, Rita de Cássia Aleixo
Source: Life Sciences. Unidade: FMRP
Subjects: DOENÇAS CARDIOVASCULARES, NEFROPATIAS, ENDOTELINAS, DIFERENÇAS SEXUAIS (PSICOGÊNESE), CROMOSSOMOS SEXUAIS
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ABNT
GOHAR, Eman Y et al. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life Sciences, v. 159, p. 20-29, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.02.093. Acesso em: 04 nov. 2024.
APA
Gohar, E. Y., Giachini, F. R., Pollock, D. M., & Tostes, R. de C. A. (2016). Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life Sciences, 159, 20-29. doi:10.1016/j.lfs.2016.02.093
NLM
Gohar EY, Giachini FR, Pollock DM, Tostes R de CA. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases [Internet]. Life Sciences. 2016 ; 159 20-29.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.093
Vancouver
Gohar EY, Giachini FR, Pollock DM, Tostes R de CA. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases [Internet]. Life Sciences. 2016 ; 159 20-29.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.093
Artigo de periodico
Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery (2016)
Rodrigues, Fernanda Luciano; Lopes, Rhéure Alves M.; Fais, Rafael Sobrano; Oliveira, Lucas de; Prado, Cibele M.; Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva
Source: Life Sciences. Unidade: FMRP
Subjects: DISFUNÇÃO ERÉTIL, PÊNIS, INSUFICIÊNCIA CARDÍACA, ARTÉRIAS, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
RODRIGUES, Fernanda Luciano et al. Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery. Life Sciences, v. 145, p. 9-18, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.12.005. Acesso em: 04 nov. 2024.
APA
Rodrigues, F. L., Lopes, R. A. M., Fais, R. S., Oliveira, L. de, Prado, C. M., Tostes, R. de C. A., & Carneiro, F. S. (2016). Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery. Life Sciences, 145, 9-18. doi:10.1016/j.lfs.2015.12.005
NLM
Rodrigues FL, Lopes RAM, Fais RS, Oliveira L de, Prado CM, Tostes R de CA, Carneiro FS. Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery [Internet]. Life Sciences. 2016 ; 145 9-18.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.005
Vancouver
Rodrigues FL, Lopes RAM, Fais RS, Oliveira L de, Prado CM, Tostes R de CA, Carneiro FS. Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery [Internet]. Life Sciences. 2016 ; 145 9-18.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.005
Artigo de periodico
Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice (2016)
Lima, Victor V; Zemse, Saiprasad M; Chiao, Chin-Wei; Bomfim, Gisele F; Tostes, Rita de Cássia Aleixo; Webb, R. Clinton; Giachini, Fernanda R
Source: Life Sciences. Unidade: FMRP
Subjects: HIPERTENSÃO, INFLAMAÇÃO, INTERLEUCINA-10, ANGIOTENSINA II, AORTA
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ABNT
LIMA, Victor V et al. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice. Life Sciences, v. 145, p. 137-143, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.12.009. Acesso em: 04 nov. 2024.
APA
Lima, V. V., Zemse, S. M., Chiao, C. -W., Bomfim, G. F., Tostes, R. de C. A., Webb, R. C., & Giachini, F. R. (2016). Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice. Life Sciences, 145, 137-143. doi:10.1016/j.lfs.2015.12.009
NLM
Lima VV, Zemse SM, Chiao C-W, Bomfim GF, Tostes R de CA, Webb RC, Giachini FR. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice [Internet]. Life Sciences. 2016 ; 145 137-143.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.009
Vancouver
Lima VV, Zemse SM, Chiao C-W, Bomfim GF, Tostes R de CA, Webb RC, Giachini FR. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice [Internet]. Life Sciences. 2016 ; 145 137-143.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.009
Artigo de periodico
Protective effect of treatment with thiamine or benfotiamine on liver oxidative damage in rat model of acute ethanol intoxication (2016)
Portari, Guilherme Vannucchi; Ovidio, Paula Payão; Deminice, Rafael; Jordão, Alceu Afonso
Source: Life Sciences. Unidade: FMRP
Subjects: INTOXICAÇÃO, ÁLCOOL, HEPATOPATIAS, ESTRESSE OXIDATIVO, MODELOS ANIMAIS DE DOENÇAS
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ABNT
PORTARI, Guilherme Vannucchi et al. Protective effect of treatment with thiamine or benfotiamine on liver oxidative damage in rat model of acute ethanol intoxication. Life Sciences, v. 162, p. 21-24, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.08.017. Acesso em: 04 nov. 2024.
APA
Portari, G. V., Ovidio, P. P., Deminice, R., & Jordão, A. A. (2016). Protective effect of treatment with thiamine or benfotiamine on liver oxidative damage in rat model of acute ethanol intoxication. Life Sciences, 162, 21-24. doi:10.1016/j.lfs.2016.08.017
NLM
Portari GV, Ovidio PP, Deminice R, Jordão AA. Protective effect of treatment with thiamine or benfotiamine on liver oxidative damage in rat model of acute ethanol intoxication [Internet]. Life Sciences. 2016 ; 162 21-24.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.08.017
Vancouver
Portari GV, Ovidio PP, Deminice R, Jordão AA. Protective effect of treatment with thiamine or benfotiamine on liver oxidative damage in rat model of acute ethanol intoxication [Internet]. Life Sciences. 2016 ; 162 21-24.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.08.017
Artigo de periodico
Cardiovascular responses elicited by continuous versus intermittent electrical stimulation of the aortic depressor nerve in conscious rats (2016)
Brognara, Fernanda; Dias, Daniel P. Martins; Castania, Jaci Airton; Fazan Júnior, Rubens; Lewis, Stephen J.; Salgado, Hélio César
Source: Life Sciences. Unidade: FMRP
Subjects: AORTA, PRESSÃO SANGUÍNEA, ESTIMULAÇÃO ELÉTRICA, FREQUÊNCIA CARDÍACA
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ABNT
BROGNARA, Fernanda et al. Cardiovascular responses elicited by continuous versus intermittent electrical stimulation of the aortic depressor nerve in conscious rats. Life Sciences, v. 148, p. 99-105, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.02.033. Acesso em: 04 nov. 2024.
APA
Brognara, F., Dias, D. P. M., Castania, J. A., Fazan Júnior, R., Lewis, S. J., & Salgado, H. C. (2016). Cardiovascular responses elicited by continuous versus intermittent electrical stimulation of the aortic depressor nerve in conscious rats. Life Sciences, 148, 99-105. doi:10.1016/j.lfs.2016.02.033
NLM
Brognara F, Dias DPM, Castania JA, Fazan Júnior R, Lewis SJ, Salgado HC. Cardiovascular responses elicited by continuous versus intermittent electrical stimulation of the aortic depressor nerve in conscious rats [Internet]. Life Sciences. 2016 ; 148 99-105.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.033
Vancouver
Brognara F, Dias DPM, Castania JA, Fazan Júnior R, Lewis SJ, Salgado HC. Cardiovascular responses elicited by continuous versus intermittent electrical stimulation of the aortic depressor nerve in conscious rats [Internet]. Life Sciences. 2016 ; 148 99-105.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.033
Artigo de periodico
Noradrenaline microinjected into the dorsal periaqueductal gray matter causes anxiolytic-like effects in rats tested in the elevated T-maze (2016)
Estrada, Viviane Batista; Matsubara, Natália Kimie; Gomes, Marcus Vinicius; Corrêa, Fernando Morgan de Aguiar; Pelosi, Gislaine Garcia
Source: Life Sciences. Unidade: FMRP
Subjects: ANSIOLÍTICOS, ANSIEDADE, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, NORADRENALINA
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ABNT
ESTRADA, Viviane Batista et al. Noradrenaline microinjected into the dorsal periaqueductal gray matter causes anxiolytic-like effects in rats tested in the elevated T-maze. Life Sciences, v. 152, p. 94-98, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.03.011. Acesso em: 04 nov. 2024.
APA
Estrada, V. B., Matsubara, N. K., Gomes, M. V., Corrêa, F. M. de A., & Pelosi, G. G. (2016). Noradrenaline microinjected into the dorsal periaqueductal gray matter causes anxiolytic-like effects in rats tested in the elevated T-maze. Life Sciences, 152, 94-98. doi:10.1016/j.lfs.2016.03.011
NLM
Estrada VB, Matsubara NK, Gomes MV, Corrêa FM de A, Pelosi GG. Noradrenaline microinjected into the dorsal periaqueductal gray matter causes anxiolytic-like effects in rats tested in the elevated T-maze [Internet]. Life Sciences. 2016 ; 152 94-98.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.03.011
Vancouver
Estrada VB, Matsubara NK, Gomes MV, Corrêa FM de A, Pelosi GG. Noradrenaline microinjected into the dorsal periaqueductal gray matter causes anxiolytic-like effects in rats tested in the elevated T-maze [Internet]. Life Sciences. 2016 ; 152 94-98.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.03.011
Artigo de periodico
Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles (2016)
Pereira, Bruno C.; Rocha, Alisson L. da; Pinto, Ana P.; Pauli, José R.; Souza, Claudio T. de; Cintra, Dennys E.; Ropelle, Eduardo R.; Freitas, Ellen Cristini de ; Zagatto, Alessandro M.; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidade: EEFERP
Subjects: EXERCÍCIO FÍSICO, ESTRESSE, MÚSCULO ESQUELÉTICO, ANIMAIS DE LABORATÓRIO
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ABNT
PEREIRA, Bruno C. et al. Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles. Life Sciences, v. 145, p. 144-151, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.12.037. Acesso em: 04 nov. 2024.
APA
Pereira, B. C., Rocha, A. L. da, Pinto, A. P., Pauli, J. R., Souza, C. T. de, Cintra, D. E., et al. (2016). Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles. Life Sciences, 145, 144-151. doi:10.1016/j.lfs.2015.12.037
NLM
Pereira BC, Rocha AL da, Pinto AP, Pauli JR, Souza CT de, Cintra DE, Ropelle ER, Freitas EC de, Zagatto AM, Silva ASR da. Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles [Internet]. Life Sciences. 2016 ; 145 144-151.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.037
Vancouver
Pereira BC, Rocha AL da, Pinto AP, Pauli JR, Souza CT de, Cintra DE, Ropelle ER, Freitas EC de, Zagatto AM, Silva ASR da. Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles [Internet]. Life Sciences. 2016 ; 145 144-151.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.037
Artigo de periodico
Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet (2015)
Oharomari, Leandro Kansuke; Garcia, Nádia Fagundes; Freitas, Ellen Cristini de ; Jordão Júnior, Alceu Afonso; Ovidio, Paula Payão; Maia, Aline Rosa; Davel, Ana Paula; Moraes, Camila de
Source: Life Sciences. Unidades: EEFERP, FMRP
Subjects: EXERCÍCIO FÍSICO, ENDOTÉLIO, OBESIDADE, ESTRESSE OXIDATIVO, DIETA, SISTEMA CARDIOVASCULAR
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ABNT
OHAROMARI, Leandro Kansuke et al. Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet. Life Sciences, v. 139, p. 91-96, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.08.015. Acesso em: 04 nov. 2024.
APA
Oharomari, L. K., Garcia, N. F., Freitas, E. C. de, Jordão Júnior, A. A., Ovidio, P. P., Maia, A. R., et al. (2015). Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet. Life Sciences, 139, 91-96. doi:10.1016/j.lfs.2015.08.015
NLM
Oharomari LK, Garcia NF, Freitas EC de, Jordão Júnior AA, Ovidio PP, Maia AR, Davel AP, Moraes C de. Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet [Internet]. Life Sciences. 2015 ; 139 91-96.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.08.015
Vancouver
Oharomari LK, Garcia NF, Freitas EC de, Jordão Júnior AA, Ovidio PP, Maia AR, Davel AP, Moraes C de. Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet [Internet]. Life Sciences. 2015 ; 139 91-96.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.08.015
Artigo de periodico
Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet (2014)
Deminice, Rafael; Rosa, Flávia Troncon; Silva, Lilian Eslaine Costa Mendes da; Jordão Júnior, Alceu Afonso
Source: Life Sciences. Unidade: FMRP
Subjects: SUPLEMENTOS DIETÉTICOS, DEFICIÊNCIA DE PROTEÍNA (PATOLOGIA), EXPRESSÃO GÊNICA (EFEITOS DE DROGAS), FÍGADO (EFEITOS DE DROGAS)
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ABNT
DEMINICE, Rafael et al. Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet. Life Sciences, v. 105, n. 1-2, p. 43-47, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.04.015. Acesso em: 04 nov. 2024.
APA
Deminice, R., Rosa, F. T., Silva, L. E. C. M. da, & Jordão Júnior, A. A. (2014). Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet. Life Sciences, 105( 1-2), 43-47. doi:10.1016/j.lfs.2014.04.015
NLM
Deminice R, Rosa FT, Silva LECM da, Jordão Júnior AA. Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet [Internet]. Life Sciences. 2014 ; 105( 1-2): 43-47.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2014.04.015
Vancouver
Deminice R, Rosa FT, Silva LECM da, Jordão Júnior AA. Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet [Internet]. Life Sciences. 2014 ; 105( 1-2): 43-47.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2014.04.015

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