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Artigo de periodico
High frequency of WNT-activated medulloblastomas with CTNNB1 wild type suggests a higher proportion of hereditary cases in a Latin-Iberian population (2023)
Moreno, Daniel Antunes; Bonatelli, Murilo; Antoniazzi, Augusto Perazzolo; Paula, Flávia Escremim de; Leal, Leticia Ferro; Garcia, Felipe Antônio de Oliveira; Paula, André Escremim de; Teixeira, Gustavo Ramos; Santana, Iara Viana Vidigal; Saggioro, Fabiano Pinto ; Neder, Luciano ; Valera, Elvis Terci ; Scrideli, Carlos Alberto ; Stavale, João; Malheiros, Suzana Maria Fleury; Lima, Matheus; Hajj, Glaucia Noeli Maroso; Garcia-Rivello, Hernan; Christiansen, Silvia; Nunes, Susana; Gil-da-Costa, Maria João; Pinheiro, Jorge; Martins, Flavia Delgado; Junior, Carlos Almeida; Mançano, Bruna Minniti; Reis, Rui Manuel
Source: Frontiers in Oncology. Unidade: FMRP
Subjects: NEOPLASIAS CEREBRAIS, MEDULOBLASTOMA, HEREDITARIEDADE, MUTAÇÃO GENÉTICA
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ABNT
MORENO, Daniel Antunes et al. High frequency of WNT-activated medulloblastomas with CTNNB1 wild type suggests a higher proportion of hereditary cases in a Latin-Iberian population. Frontiers in Oncology, v. 13, 2023Tradução . . Disponível em: https://doi.org/10.3389/fonc.2023.1237170. Acesso em: 18 nov. 2024.
APA
Moreno, D. A., Bonatelli, M., Antoniazzi, A. P., Paula, F. E. de, Leal, L. F., Garcia, F. A. de O., et al. (2023). High frequency of WNT-activated medulloblastomas with CTNNB1 wild type suggests a higher proportion of hereditary cases in a Latin-Iberian population. Frontiers in Oncology, 13. doi:10.3389/fonc.2023.1237170
NLM
Moreno DA, Bonatelli M, Antoniazzi AP, Paula FE de, Leal LF, Garcia FA de O, Paula AE de, Teixeira GR, Santana IVV, Saggioro FP, Neder L, Valera ET, Scrideli CA, Stavale J, Malheiros SMF, Lima M, Hajj GNM, Garcia-Rivello H, Christiansen S, Nunes S, Gil-da-Costa MJ, Pinheiro J, Martins FD, Junior CA, Mançano BM, Reis RM. High frequency of WNT-activated medulloblastomas with CTNNB1 wild type suggests a higher proportion of hereditary cases in a Latin-Iberian population [Internet]. Frontiers in Oncology. 2023 ;13[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fonc.2023.1237170
Vancouver
Moreno DA, Bonatelli M, Antoniazzi AP, Paula FE de, Leal LF, Garcia FA de O, Paula AE de, Teixeira GR, Santana IVV, Saggioro FP, Neder L, Valera ET, Scrideli CA, Stavale J, Malheiros SMF, Lima M, Hajj GNM, Garcia-Rivello H, Christiansen S, Nunes S, Gil-da-Costa MJ, Pinheiro J, Martins FD, Junior CA, Mançano BM, Reis RM. High frequency of WNT-activated medulloblastomas with CTNNB1 wild type suggests a higher proportion of hereditary cases in a Latin-Iberian population [Internet]. Frontiers in Oncology. 2023 ;13[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fonc.2023.1237170
Artigo de periodico
Identification of HDAC4 as a potential therapeutic target and prognostic biomarker for ZFTA-fused ependymomas (2023)
Sousa, Graziella Ribeiro de ; Salomão, Karina B.; Nagano, Luís Fernando Peinado ; Riemondy, Kent A.; Chagas, Pablo Shimaoka ; Veronez, Luciana Chain ; Saggioro, Fabiano Pinto ; Marie, Suely Kazue Nagahashi ; Yunes, José A.; Cardinalli, Izilda Aparecida; Brandalise, Silvia Regina ; Queiroz, Rosane Gomes de Paula; Scrideli, Carlos Alberto ; Donson, Andrew M.; Foreman, Nicholas K.; Tone, Luiz Gonzaga ; Valera, Elvis Terci
Source: Cancer Gene Therapy. Unidades: FMRP, FM, FCFRP
Subjects: NEOPLASIAS CEREBRAIS, PERFILAÇÃO DA EXPRESSÃO GÊNICA, PROGNÓSTICO, FATORES DE TRANSCRIÇÃO, BIOMARCADORES
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ABNT
SOUSA, Graziella Ribeiro de et al. Identification of HDAC4 as a potential therapeutic target and prognostic biomarker for ZFTA-fused ependymomas. Cancer Gene Therapy, v. 30, p. 1105-1113, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41417-023-00616-z. Acesso em: 18 nov. 2024.
APA
Sousa, G. R. de, Salomão, K. B., Nagano, L. F. P., Riemondy, K. A., Chagas, P. S., Veronez, L. C., et al. (2023). Identification of HDAC4 as a potential therapeutic target and prognostic biomarker for ZFTA-fused ependymomas. Cancer Gene Therapy, 30, 1105-1113. doi:10.1038/s41417-023-00616-z
NLM
Sousa GR de, Salomão KB, Nagano LFP, Riemondy KA, Chagas PS, Veronez LC, Saggioro FP, Marie SKN, Yunes JA, Cardinalli IA, Brandalise SR, Queiroz RG de P, Scrideli CA, Donson AM, Foreman NK, Tone LG, Valera ET. Identification of HDAC4 as a potential therapeutic target and prognostic biomarker for ZFTA-fused ependymomas [Internet]. Cancer Gene Therapy. 2023 ; 30 1105-1113.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41417-023-00616-z
Vancouver
Sousa GR de, Salomão KB, Nagano LFP, Riemondy KA, Chagas PS, Veronez LC, Saggioro FP, Marie SKN, Yunes JA, Cardinalli IA, Brandalise SR, Queiroz RG de P, Scrideli CA, Donson AM, Foreman NK, Tone LG, Valera ET. Identification of HDAC4 as a potential therapeutic target and prognostic biomarker for ZFTA-fused ependymomas [Internet]. Cancer Gene Therapy. 2023 ; 30 1105-1113.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41417-023-00616-z
Artigo de periodico
Clinical Prognostic Implications of Wnt Hub Genes Expression in Medulloblastoma (2023)
Silva, Andréa Martins da; Baroni, Mirella ; Salomão, Karina Bezerra ; Chagas, Pablo Ferreira das ; Silva, Ricardo Bonfim; Geron, Lenisa; Cruzeiro, Gustavo Alencastro Veiga ; Silva Junior, Wilson Araújo da ; Correa, Carolina Alves Pereira ; Carlotti Júnior, Carlos Gilberto ; Queiroz, Rosane Gomes de Paula; Marie, Suely Kazue Nagahashi ; Brandalise, Silvia Regina ; Yunes, Jose Andres ; Scrideli, Carlos Alberto ; Valera, Elvis Terci ; Tone, Luiz Gonzaga
Source: CELLULAR AND MOLECULAR NEUROBIOLOGY. Unidades: FMRP, FM
Subjects: MEDULOBLASTOMA, BIOMARCADORES, CRIANÇAS, NEOPLASIAS CEREBRAIS
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ABNT
SILVA, Andréa Martins da et al. Clinical Prognostic Implications of Wnt Hub Genes Expression in Medulloblastoma. CELLULAR AND MOLECULAR NEUROBIOLOGY, v. 43, n. 2, p. 813-826, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/57968. Acesso em: 18 nov. 2024.
APA
Silva, A. M. da, Baroni, M., Salomão, K. B., Chagas, P. F. das, Silva, R. B., Geron, L., et al. (2023). Clinical Prognostic Implications of Wnt Hub Genes Expression in Medulloblastoma. CELLULAR AND MOLECULAR NEUROBIOLOGY, 43( 2), 813-826. doi:10.1007/s10571-022-01217-4
NLM
Silva AM da, Baroni M, Salomão KB, Chagas PF das, Silva RB, Geron L, Cruzeiro GAV, Silva Junior WA da, Correa CAP, Carlotti Júnior CG, Queiroz RG de P, Marie SKN, Brandalise SR, Yunes JA, Scrideli CA, Valera ET, Tone LG. Clinical Prognostic Implications of Wnt Hub Genes Expression in Medulloblastoma [Internet]. CELLULAR AND MOLECULAR NEUROBIOLOGY. 2023 ; 43( 2): 813-826.[citado 2024 nov. 18 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57968
Vancouver
Silva AM da, Baroni M, Salomão KB, Chagas PF das, Silva RB, Geron L, Cruzeiro GAV, Silva Junior WA da, Correa CAP, Carlotti Júnior CG, Queiroz RG de P, Marie SKN, Brandalise SR, Yunes JA, Scrideli CA, Valera ET, Tone LG. Clinical Prognostic Implications of Wnt Hub Genes Expression in Medulloblastoma [Internet]. CELLULAR AND MOLECULAR NEUROBIOLOGY. 2023 ; 43( 2): 813-826.[citado 2024 nov. 18 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57968
Artigo de periodico
The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth (2021)
Teixeira, Silvia Aparecida; Viapiano, Mariano S.; Andrade, Augusto F.; Nandhu, Mohan S.; Pezuk, Julia Alejandra; Bidinotto, Lucas T.; Sant'Ana, Veridiana Kiill Suazo de; Serafini, Luciano Neder; Carlotti Júnior, Carlos Gilberto ; Becker, Aline P.; Tone, Luiz Gonzaga ; Scrideli, Carlos Alberto
Source: Molecular Neurobiology. Unidade: FMRP
Subjects: ANIDRASE CARBÔNICA, METABOLISMO CELULAR, NEOPLASIAS CEREBRAIS, FARMACOTERAPIA, LINHAGEM CELULAR, CICLO CELULAR, SULFONAMIDAS
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ABNT
TEIXEIRA, Silvia Aparecida et al. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, v. 58, n. 9, p. 4520-4534, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02437-3. Acesso em: 18 nov. 2024.
APA
Teixeira, S. A., Viapiano, M. S., Andrade, A. F., Nandhu, M. S., Pezuk, J. A., Bidinotto, L. T., et al. (2021). The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, 58( 9), 4520-4534. doi:10.1007/s12035-021-02437-3
NLM
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Vancouver
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Artigo de periodico
Evaluating H3F3A K27M and G34R/V somatic mutations in a cohort of pediatric brain tumors of different and rare histologies (2020)
Oliveira, Vinicius Fernandes; Sousa, Graziella Ribeiro de ; Santos, Antonio Carlos dos ; Saggioro, Fabiano Pinto ; Machado, Hélio Rubens ; Oliveira, Ricardo Santos de ; Tone, Luiz Gonzaga ; Valera, Elvis Terci
Source: Child's Nervous System. Unidades: FMRP, SVOI
Subjects: NEOPLASIAS CEREBRAIS, EPIGÊNESE GENÉTICA, GLIOMA
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ABNT
OLIVEIRA, Vinicius Fernandes et al. Evaluating H3F3A K27M and G34R/V somatic mutations in a cohort of pediatric brain tumors of different and rare histologies. Child's Nervous System, v. 37, n. 2, p. 375-382, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00381-020-04852-8. Acesso em: 18 nov. 2024.
APA
Oliveira, V. F., Sousa, G. R. de, Santos, A. C. dos, Saggioro, F. P., Machado, H. R., Oliveira, R. S. de, et al. (2020). Evaluating H3F3A K27M and G34R/V somatic mutations in a cohort of pediatric brain tumors of different and rare histologies. Child's Nervous System, 37( 2), 375-382. doi:10.1007/s00381-020-04852-8
NLM
Oliveira VF, Sousa GR de, Santos AC dos, Saggioro FP, Machado HR, Oliveira RS de, Tone LG, Valera ET. Evaluating H3F3A K27M and G34R/V somatic mutations in a cohort of pediatric brain tumors of different and rare histologies [Internet]. Child's Nervous System. 2020 ; 37( 2): 375-382.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00381-020-04852-8
Vancouver
Oliveira VF, Sousa GR de, Santos AC dos, Saggioro FP, Machado HR, Oliveira RS de, Tone LG, Valera ET. Evaluating H3F3A K27M and G34R/V somatic mutations in a cohort of pediatric brain tumors of different and rare histologies [Internet]. Child's Nervous System. 2020 ; 37( 2): 375-382.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00381-020-04852-8
Artigo de periodico-carta/editorial
Perinatal complex low- and high-grade glial tumor harboring a novel GIGYF2-ALK fusion [Carta] (2020)
Valera, Elvis Terci ; Neder, Luciano ; Queiroz, Rosane Gomes de Paula; Santos, Antonio Carlos dos ; Sousa, Graziella Ribeiro de ; Oliveira, Ricardo Santos de ; Santos, Marcelo Volpon ; Machado, Hélio Rubens ; Tone, Luiz Gonzaga
Source: Pediatric Blood & Cancer. Unidade: FMRP
Subjects: NEOPLASIAS CEREBRAIS, FETO, NEONATOLOGIA
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ABNT
VALERA, Elvis Terci et al. Perinatal complex low- and high-grade glial tumor harboring a novel GIGYF2-ALK fusion [Carta]. Pediatric Blood & Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1002/pbc.28015. Acesso em: 18 nov. 2024. , 2020
APA
Valera, E. T., Neder, L., Queiroz, R. G. de P., Santos, A. C. dos, Sousa, G. R. de, Oliveira, R. S. de, et al. (2020). Perinatal complex low- and high-grade glial tumor harboring a novel GIGYF2-ALK fusion [Carta]. Pediatric Blood & Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1002/pbc.28015
NLM
Valera ET, Neder L, Queiroz RG de P, Santos AC dos, Sousa GR de, Oliveira RS de, Santos MV, Machado HR, Tone LG. Perinatal complex low- and high-grade glial tumor harboring a novel GIGYF2-ALK fusion [Carta] [Internet]. Pediatric Blood & Cancer. 2020 ; 67( 1): 101-103.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/pbc.28015
Vancouver
Valera ET, Neder L, Queiroz RG de P, Santos AC dos, Sousa GR de, Oliveira RS de, Santos MV, Machado HR, Tone LG. Perinatal complex low- and high-grade glial tumor harboring a novel GIGYF2-ALK fusion [Carta] [Internet]. Pediatric Blood & Cancer. 2020 ; 67( 1): 101-103.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/pbc.28015
Artigo de periodico
The TP53 p.R337H mutation is uncommon in a Brazilian cohort of pediatric patients diagnosed with ependymoma (2020)
Magalhães, Taciani de Almeida ; Borges, Kleiton Silva ; Sousa, Graziella Ribeiro de ; Brandalise, Silvia Regina; Seidinger, Ana Luiza; Scrideli, Carlos Alberto ; Shinjo, Sueli Mieko Oba ; Yunes, Jose Andres ; Tone, Luiz Gonzaga
Source: Neurological Sciences. Unidades: FMRP, FM
Subjects: DOENÇAS CEREBRAIS, PROTEÍNAS DO TECIDO NERVOSO, NEOPLASIAS CEREBRAIS
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ABNT
MAGALHÃES, Taciani de Almeida et al. The TP53 p.R337H mutation is uncommon in a Brazilian cohort of pediatric patients diagnosed with ependymoma. Neurological Sciences, v. 41, n. 3, p. 691-694, 2020Tradução . . Disponível em: https://doi.org/10.1007/s10072-019-04112-x. Acesso em: 18 nov. 2024.
APA
Magalhães, T. de A., Borges, K. S., Sousa, G. R. de, Brandalise, S. R., Seidinger, A. L., Scrideli, C. A., et al. (2020). The TP53 p.R337H mutation is uncommon in a Brazilian cohort of pediatric patients diagnosed with ependymoma. Neurological Sciences, 41( 3), 691-694. doi:10.1007/s10072-019-04112-x
NLM
Magalhães T de A, Borges KS, Sousa GR de, Brandalise SR, Seidinger AL, Scrideli CA, Shinjo SMO, Yunes JA, Tone LG. The TP53 p.R337H mutation is uncommon in a Brazilian cohort of pediatric patients diagnosed with ependymoma [Internet]. Neurological Sciences. 2020 ; 41( 3): 691-694.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10072-019-04112-x
Vancouver
Magalhães T de A, Borges KS, Sousa GR de, Brandalise SR, Seidinger AL, Scrideli CA, Shinjo SMO, Yunes JA, Tone LG. The TP53 p.R337H mutation is uncommon in a Brazilian cohort of pediatric patients diagnosed with ependymoma [Internet]. Neurological Sciences. 2020 ; 41( 3): 691-694.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10072-019-04112-x
Artigo de periodico
Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course (2020)
Wefers, Annika K.; Stichel, Damian; Schrimpf, Daniel; Coras, Roland; Pages, Mélanie; Tauziède-Espariat, Arnault; Varlet, Pascale; Schwarz, Daniel; Oliveira, Ricardo Santos de ; Queiroz, Rosane Gomes de Paula; Valera, Elvis Terci
Source: Acta Neuropathologica. Unidade: FMRP
Subjects: NEOPLASIAS CEREBRAIS, GLIOMA, GENES, FUSÃO CELULAR, METILAÇÃO DE DNA, ONCOGENES, PROTEÍNAS PROTO-ONCOGÊNICAS
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ABNT
WEFERS, Annika K. et al. Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course. Acta Neuropathologica, v. 139, n. 1, p. 193-209, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00401-019-02078-w. Acesso em: 18 nov. 2024.
APA
Wefers, A. K., Stichel, D., Schrimpf, D., Coras, R., Pages, M., Tauziède-Espariat, A., et al. (2020). Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course. Acta Neuropathologica, 139( 1), 193-209. doi:10.1007/s00401-019-02078-w
NLM
Wefers AK, Stichel D, Schrimpf D, Coras R, Pages M, Tauziède-Espariat A, Varlet P, Schwarz D, Oliveira RS de, Queiroz RG de P, Valera ET. Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course [Internet]. Acta Neuropathologica. 2020 ; 139( 1): 193-209.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00401-019-02078-w
Vancouver
Wefers AK, Stichel D, Schrimpf D, Coras R, Pages M, Tauziède-Espariat A, Varlet P, Schwarz D, Oliveira RS de, Queiroz RG de P, Valera ET. Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course [Internet]. Acta Neuropathologica. 2020 ; 139( 1): 193-209.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00401-019-02078-w
Artigo de periodico
High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of miR-10b-5p and miR-630 in the tumor aggressiveness (2020)
Darrigo Júnior, Luiz Guilherme; Baroni, Mirella; Lira, Régia Caroline Peixoto; Teixeira, Silvia Aparecida ; Fedatto, Paola Fernanda; Silveira, Vanessa da Silva ; Sant'Ana, Veridiana Kiill Suazo de; Veronez, Luciana Chain; Panepucci, Rodrigo Alexandre; Antonio, David Santos Marco; Yunes, Jose Andres; Brandalise, Silvia Regina; Aguiar, Simone dos Santos; Neder, Luciano; Oliveira, Ricardo Santos de; Machado, Hélio Rubens; Carlotti Júnior, Carlos Gilberto ; Tone, Luiz Gonzaga ; Valera, Elvis Terci; Scrideli, Carlos Alberto
Source: Molecular Biology Reports. Unidade: FMRP
Subjects: MICRORNAS, NEOPLASIAS CEREBRAIS
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ABNT
DARRIGO JÚNIOR, Luiz Guilherme et al. High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of miR-10b-5p and miR-630 in the tumor aggressiveness. Molecular Biology Reports, v. 47, n. 9, p. 6949-6959, 2020Tradução . . Disponível em: https://doi.org/10.1007/s11033-020-05754-3. Acesso em: 18 nov. 2024.
APA
Darrigo Júnior, L. G., Baroni, M., Lira, R. C. P., Teixeira, S. A., Fedatto, P. F., Silveira, V. da S., et al. (2020). High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of miR-10b-5p and miR-630 in the tumor aggressiveness. Molecular Biology Reports, 47( 9), 6949-6959. doi:10.1007/s11033-020-05754-3
NLM
Darrigo Júnior LG, Baroni M, Lira RCP, Teixeira SA, Fedatto PF, Silveira V da S, Sant'Ana VKS de, Veronez LC, Panepucci RA, Antonio DSM, Yunes JA, Brandalise SR, Aguiar S dos S, Neder L, Oliveira RS de, Machado HR, Carlotti Júnior CG, Tone LG, Valera ET, Scrideli CA. High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of miR-10b-5p and miR-630 in the tumor aggressiveness [Internet]. Molecular Biology Reports. 2020 ; 47( 9): 6949-6959.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s11033-020-05754-3
Vancouver
Darrigo Júnior LG, Baroni M, Lira RCP, Teixeira SA, Fedatto PF, Silveira V da S, Sant'Ana VKS de, Veronez LC, Panepucci RA, Antonio DSM, Yunes JA, Brandalise SR, Aguiar S dos S, Neder L, Oliveira RS de, Machado HR, Carlotti Júnior CG, Tone LG, Valera ET, Scrideli CA. High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of miR-10b-5p and miR-630 in the tumor aggressiveness [Internet]. Molecular Biology Reports. 2020 ; 47( 9): 6949-6959.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s11033-020-05754-3
Trabalho de evento-resumo
The interplay between musashi and notch pathways in pediatric medulloblastomas (2019)
Chagas, Pablo Ferreira das ; Bomfim-Silva, Ricardo; Cruzeiro, Gustavo Alencastro Veiga ; Veronez, Luciana Chain ; Nagano, Luis Fernando ; Borges, Kleiton Silva ; Salomão, Karina Bezerra ; Queiroz, Rosane Gomes de Paula; Brandalise, Silvia Regina; Scrideli, Carlos Alberto ; Tone, Luiz Gonzaga ; Valera, Elvis Terci
Source: Resumos. Conference titles: Brazilian Congress of Genetics. Unidade: FMRP
Subjects: MEDULOBLASTOMA, NEOPLASIAS CEREBRAIS, EXPRESSÃO GÊNICA
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ABNT
CHAGAS, Pablo Ferreira das et al. The interplay between musashi and notch pathways in pediatric medulloblastomas. 2019, Anais.. Águas de Lindóia: Sociedade Brasileira de Genética, 2019. Disponível em: https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf. Acesso em: 18 nov. 2024.
APA
Chagas, P. F. das, Bomfim-Silva, R., Cruzeiro, G. A. V., Veronez, L. C., Nagano, L. F., Borges, K. S., et al. (2019). The interplay between musashi and notch pathways in pediatric medulloblastomas. In Resumos. Águas de Lindóia: Sociedade Brasileira de Genética. Recuperado de https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf
NLM
Chagas PF das, Bomfim-Silva R, Cruzeiro GAV, Veronez LC, Nagano LF, Borges KS, Salomão KB, Queiroz RG de P, Brandalise SR, Scrideli CA, Tone LG, Valera ET. The interplay between musashi and notch pathways in pediatric medulloblastomas [Internet]. Resumos. 2019 ;[citado 2024 nov. 18 ] Available from: https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf
Vancouver
Chagas PF das, Bomfim-Silva R, Cruzeiro GAV, Veronez LC, Nagano LF, Borges KS, Salomão KB, Queiroz RG de P, Brandalise SR, Scrideli CA, Tone LG, Valera ET. The interplay between musashi and notch pathways in pediatric medulloblastomas [Internet]. Resumos. 2019 ;[citado 2024 nov. 18 ] Available from: https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf
Trabalho de evento-resumo periodico
High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of MIR-10B and MIR-630 in the tumor aggressiveness (2019)
Darrigo Júnior, Luiz Guilherme ; Barroni, M.; Lira, Régia Caroline Peixoto ; Fedatto, Paola Fernanda ; Silveira, Vanessa da Silva ; Suazo, Veridiana Kill; Panepucci, Rodrigo Alexandre ; Antonio, D. S. Marco; Valera, Elvis Terci ; Aguiar, S. D. S.; Yunes, Jose Andres ; Brandalise, Silvia Regina; Serafini, Luciano Neder ; Oliveira, R. D. S.; Machado, Hélio Rubens; Carlotti Júnior, Carlos Gilberto ; Tone, Luiz Gonzaga ; Scrideli, Carlos Alberto
Source: Pediatric Blood & Cancer. Conference titles: Congress of the International Society of Paediatric Oncology - SIOP. Unidade: FMRP
Subjects: MICRORNAS, NEOPLASIAS CEREBRAIS
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ABNT
DARRIGO JÚNIOR, Luiz Guilherme et al. High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of MIR-10B and MIR-630 in the tumor aggressiveness. Pediatric Blood & Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1002/pbc.27989. Acesso em: 18 nov. 2024. , 2019
APA
Darrigo Júnior, L. G., Barroni, M., Lira, R. C. P., Fedatto, P. F., Silveira, V. da S., Suazo, V. K., et al. (2019). High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of MIR-10B and MIR-630 in the tumor aggressiveness. Pediatric Blood & Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1002/pbc.27989
NLM
Darrigo Júnior LG, Barroni M, Lira RCP, Fedatto PF, Silveira V da S, Suazo VK, Panepucci RA, Antonio DSM, Valera ET, Aguiar SDS, Yunes JA, Brandalise SR, Serafini LN, Oliveira RDS, Machado HR, Carlotti Júnior CG, Tone LG, Scrideli CA. High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of MIR-10B and MIR-630 in the tumor aggressiveness [Internet]. Pediatric Blood & Cancer. 2019 ; 66 S143-S144.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/pbc.27989
Vancouver
Darrigo Júnior LG, Barroni M, Lira RCP, Fedatto PF, Silveira V da S, Suazo VK, Panepucci RA, Antonio DSM, Valera ET, Aguiar SDS, Yunes JA, Brandalise SR, Serafini LN, Oliveira RDS, Machado HR, Carlotti Júnior CG, Tone LG, Scrideli CA. High-throughput microRNA profile in adult and pediatric primary glioblastomas: the role of MIR-10B and MIR-630 in the tumor aggressiveness [Internet]. Pediatric Blood & Cancer. 2019 ; 66 S143-S144.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/pbc.27989
Artigo de periodico
Impact of the canonical Wnt pathway activation on the pathogenesis and prognosis of adamantinomatous craniopharyngiomas (2018)
Jucá, Carlos Eduardo Barros; Colli, Leandro Machado; Martins, Clarissa Silva; Campanini, Marina Lanciotti; Paixão, Beatriz; Jucá, Renata Viana; Saggioro, Fabiano Pinto; Oliveira, Ricardo Santos de; Moreira, Ayrton Custódio; Machado, Hélio Rubens; Neder, Luciano; Antonini, Sonir Roberto Rauber; Castro, Margaret de
Source: Hormone and Metabolic Research. Unidades: FMRP, SVOI
Subjects: NEOPLASIAS CEREBRAIS, GLÂNDULA PITUITÁRIA, PROGNÓSTICO
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ABNT
JUCÁ, Carlos Eduardo Barros et al. Impact of the canonical Wnt pathway activation on the pathogenesis and prognosis of adamantinomatous craniopharyngiomas. Hormone and Metabolic Research, v. 50, n. 7, p. 575-581, 2018Tradução . . Disponível em: https://doi.org/10.1055/a-0593-5956. Acesso em: 18 nov. 2024.
APA
Jucá, C. E. B., Colli, L. M., Martins, C. S., Campanini, M. L., Paixão, B., Jucá, R. V., et al. (2018). Impact of the canonical Wnt pathway activation on the pathogenesis and prognosis of adamantinomatous craniopharyngiomas. Hormone and Metabolic Research, 50( 7), 575-581. doi:10.1055/a-0593-5956
NLM
Jucá CEB, Colli LM, Martins CS, Campanini ML, Paixão B, Jucá RV, Saggioro FP, Oliveira RS de, Moreira AC, Machado HR, Neder L, Antonini SRR, Castro M de. Impact of the canonical Wnt pathway activation on the pathogenesis and prognosis of adamantinomatous craniopharyngiomas [Internet]. Hormone and Metabolic Research. 2018 ; 50( 7): 575-581.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1055/a-0593-5956
Vancouver
Jucá CEB, Colli LM, Martins CS, Campanini ML, Paixão B, Jucá RV, Saggioro FP, Oliveira RS de, Moreira AC, Machado HR, Neder L, Antonini SRR, Castro M de. Impact of the canonical Wnt pathway activation on the pathogenesis and prognosis of adamantinomatous craniopharyngiomas [Internet]. Hormone and Metabolic Research. 2018 ; 50( 7): 575-581.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1055/a-0593-5956
Trabalho de evento-resumo periodico
Silencig of GDF15 gene and response to radiotherapy and the treatment with temozolomide in glioblastoma cell lines (2015)
Baroni, M.; Fedatto, P.; Andrade, A.; Suazo, V.; Queiróz, R.; Tone, Luiz Gonzaga; Scrideli, Carlos Alberto
Source: Pediatric Blood and Cancer. Conference titles: Congress of the International Society of Paediatric Oncology. Unidade: FMRP
Subjects: NEOPLASIAS CEREBRAIS, RADIOTERAPIA, QUIMIOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARONI, M. et al. Silencig of GDF15 gene and response to radiotherapy and the treatment with temozolomide in glioblastoma cell lines. Pediatric Blood and Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2015
APA
Baroni, M., Fedatto, P., Andrade, A., Suazo, V., Queiróz, R., Tone, L. G., & Scrideli, C. A. (2015). Silencig of GDF15 gene and response to radiotherapy and the treatment with temozolomide in glioblastoma cell lines. Pediatric Blood and Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Baroni M, Fedatto P, Andrade A, Suazo V, Queiróz R, Tone LG, Scrideli CA. Silencig of GDF15 gene and response to radiotherapy and the treatment with temozolomide in glioblastoma cell lines. Pediatric Blood and Cancer. 2015 ; 62 S283.[citado 2024 nov. 18 ]
Vancouver
Baroni M, Fedatto P, Andrade A, Suazo V, Queiróz R, Tone LG, Scrideli CA. Silencig of GDF15 gene and response to radiotherapy and the treatment with temozolomide in glioblastoma cell lines. Pediatric Blood and Cancer. 2015 ; 62 S283.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Differential microRNAs expression profile in primary glioblastomas of children and adults, pilocytic astrocytomas and non-neoplastic white matter: identification of pathways involved in tumorigenesis (2015)
Darrigo Júnior, L. G.; Fedatto, P.; Silveira, Vanessa da Silva; Antonio, D. Marco; Valera, E.; Panepucci, R.; Goes, A.; Carlotti Júnior, Carlos Gilberto; Oliveira, R.; Neder, Luciano; Aguiar, S.; Yunes, J.; Brandalise, S.; Tone, Luiz Gonzaga; Scrideli, Carlos Alberto
Source: Pediatric Blood and Cancer. Conference titles: Congress of the International Society of Paediatric Oncology. Unidade: FMRP
Subjects: RNA, PEDIATRIA, NEOPLASIAS CEREBRAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DARRIGO JÚNIOR, L. G. et al. Differential microRNAs expression profile in primary glioblastomas of children and adults, pilocytic astrocytomas and non-neoplastic white matter: identification of pathways involved in tumorigenesis. Pediatric Blood and Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2015
APA
Darrigo Júnior, L. G., Fedatto, P., Silveira, V. da S., Antonio, D. M., Valera, E., Panepucci, R., et al. (2015). Differential microRNAs expression profile in primary glioblastomas of children and adults, pilocytic astrocytomas and non-neoplastic white matter: identification of pathways involved in tumorigenesis. Pediatric Blood and Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Darrigo Júnior LG, Fedatto P, Silveira V da S, Antonio DM, Valera E, Panepucci R, Goes A, Carlotti Júnior CG, Oliveira R, Neder L, Aguiar S, Yunes J, Brandalise S, Tone LG, Scrideli CA. Differential microRNAs expression profile in primary glioblastomas of children and adults, pilocytic astrocytomas and non-neoplastic white matter: identification of pathways involved in tumorigenesis. Pediatric Blood and Cancer. 2015 ; 62 S183.[citado 2024 nov. 18 ]
Vancouver
Darrigo Júnior LG, Fedatto P, Silveira V da S, Antonio DM, Valera E, Panepucci R, Goes A, Carlotti Júnior CG, Oliveira R, Neder L, Aguiar S, Yunes J, Brandalise S, Tone LG, Scrideli CA. Differential microRNAs expression profile in primary glioblastomas of children and adults, pilocytic astrocytomas and non-neoplastic white matter: identification of pathways involved in tumorigenesis. Pediatric Blood and Cancer. 2015 ; 62 S183.[citado 2024 nov. 18 ]
Artigo de periodico
Antitumour activity of AMG 900 alone or in combination with histone deacetylase inhibitor SaHa on medulloblastoma cell lines (2015)
Geron, Lenisa; Borges, Kleiton Silva; Andrade, Augusto Faria; Suazo, Veridiana Kill; Scrideli, Carlos Alberto; Tone, Luiz Gonzaga
Source: Neurological Research. Unidade: FMRP
Subjects: MEDULOBLASTOMA, FARMACOLOGIA MÉDICA, NEOPLASIAS CEREBRAIS, INIBIÇÃO
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ABNT
GERON, Lenisa et al. Antitumour activity of AMG 900 alone or in combination with histone deacetylase inhibitor SaHa on medulloblastoma cell lines. Neurological Research, v. 37, n. 8, p. 703-711, 2015Tradução . . Disponível em: https://doi.org/10.1179/1743132815Y.0000000048. Acesso em: 18 nov. 2024.
APA
Geron, L., Borges, K. S., Andrade, A. F., Suazo, V. K., Scrideli, C. A., & Tone, L. G. (2015). Antitumour activity of AMG 900 alone or in combination with histone deacetylase inhibitor SaHa on medulloblastoma cell lines. Neurological Research, 37( 8), 703-711. doi:10.1179/1743132815Y.0000000048
NLM
Geron L, Borges KS, Andrade AF, Suazo VK, Scrideli CA, Tone LG. Antitumour activity of AMG 900 alone or in combination with histone deacetylase inhibitor SaHa on medulloblastoma cell lines [Internet]. Neurological Research. 2015 ; 37( 8): 703-711.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1179/1743132815Y.0000000048
Vancouver
Geron L, Borges KS, Andrade AF, Suazo VK, Scrideli CA, Tone LG. Antitumour activity of AMG 900 alone or in combination with histone deacetylase inhibitor SaHa on medulloblastoma cell lines [Internet]. Neurological Research. 2015 ; 37( 8): 703-711.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1179/1743132815Y.0000000048
Artigo de periodico
Anticancer activity of 7-epiclusianone, a benzophenone from Garcinia brasiliensis, in glioblastoma (2015)
Sales, Leilane; Pezuk, Julia Alejandra; Borges, Kleiton Silva; Brassesco, María Sol; Scrideli, Carlos Alberto; Tone, Luiz Gonzaga; Santos, Marcelo Henrique dos; Ionta, Marisa; Oliveira, Jaqueline Carvalho de
Source: BMC Complementary and Alternative Medicine. Unidades: FMRP, FFCLRP
Subjects: NEOPLASIAS CEREBRAIS, ANTINEOPLÁSICOS, EXTRATOS (FORMAS FARMACÊUTICAS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SALES, Leilane et al. Anticancer activity of 7-epiclusianone, a benzophenone from Garcinia brasiliensis, in glioblastoma. BMC Complementary and Alternative Medicine, v. 15, 2015Tradução . . Disponível em: https://doi.org/10.1186/s12906-015-0911-1. Acesso em: 18 nov. 2024.
APA
Sales, L., Pezuk, J. A., Borges, K. S., Brassesco, M. S., Scrideli, C. A., Tone, L. G., et al. (2015). Anticancer activity of 7-epiclusianone, a benzophenone from Garcinia brasiliensis, in glioblastoma. BMC Complementary and Alternative Medicine, 15. doi:10.1186/s12906-015-0911-1
NLM
Sales L, Pezuk JA, Borges KS, Brassesco MS, Scrideli CA, Tone LG, Santos MH dos, Ionta M, Oliveira JC de. Anticancer activity of 7-epiclusianone, a benzophenone from Garcinia brasiliensis, in glioblastoma [Internet]. BMC Complementary and Alternative Medicine. 2015 ; 15[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12906-015-0911-1
Vancouver
Sales L, Pezuk JA, Borges KS, Brassesco MS, Scrideli CA, Tone LG, Santos MH dos, Ionta M, Oliveira JC de. Anticancer activity of 7-epiclusianone, a benzophenone from Garcinia brasiliensis, in glioblastoma [Internet]. BMC Complementary and Alternative Medicine. 2015 ; 15[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12906-015-0911-1
Trabalho de evento-resumo
Nanotechnology-based new strategies for treatment of glioblastoma and medulloblastoma (2015)
Paula, Leonardo Barcelos de; Primo, Fernando Lucas; Peres, N. T. A.; Andrade, A. F.; Canchaya, G. S.; Martinez-Rossi, Nilce Maria; Tone, Luiz Gonzaga; Faça, Vitor Marcel; Filho, A.; Tedesco, Antônio Cláudio
Source: Abstracts. Conference titles: International Congress of Pharmaceutical Sciences. Unidades: FMRP, FFCLRP
Subjects: NANOTECNOLOGIA, TERAPIA FOTODINÂMICA, NEOPLASIAS CEREBRAIS
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ABNT
PAULA, Leonardo Barcelos de et al. Nanotechnology-based new strategies for treatment of glioblastoma and medulloblastoma. 2015, Anais.. Ribeirão Preto: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2015. Disponível em: http://www.cifarp.com.br/abstracts2015/listaresumos.htm. Acesso em: 18 nov. 2024.
APA
Paula, L. B. de, Primo, F. L., Peres, N. T. A., Andrade, A. F., Canchaya, G. S., Martinez-Rossi, N. M., et al. (2015). Nanotechnology-based new strategies for treatment of glioblastoma and medulloblastoma. In Abstracts. Ribeirão Preto: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://www.cifarp.com.br/abstracts2015/listaresumos.htm
NLM
Paula LB de, Primo FL, Peres NTA, Andrade AF, Canchaya GS, Martinez-Rossi NM, Tone LG, Faça VM, Filho A, Tedesco AC. Nanotechnology-based new strategies for treatment of glioblastoma and medulloblastoma [Internet]. Abstracts. 2015 ;[citado 2024 nov. 18 ] Available from: http://www.cifarp.com.br/abstracts2015/listaresumos.htm
Vancouver
Paula LB de, Primo FL, Peres NTA, Andrade AF, Canchaya GS, Martinez-Rossi NM, Tone LG, Faça VM, Filho A, Tedesco AC. Nanotechnology-based new strategies for treatment of glioblastoma and medulloblastoma [Internet]. Abstracts. 2015 ;[citado 2024 nov. 18 ] Available from: http://www.cifarp.com.br/abstracts2015/listaresumos.htm
Trabalho de evento-resumo periodico
Polo-like kinase 1 as a potential target in pediatric medulloblastoma (2012)
Pezuk, Julia Alejandra; Brassesco, Maria Sol; Oliveira, Jaqueline Carvalho de; Morales, Andressa Gois; Queiroz, Rosane Gomes de Paula; Scrideli, Carlos Alberto; Valera, Elvis Terci; Tone, Luiz Gonzaga
Source: Pediatric Blood & Cancer. Conference titles: Congress of the International Society of Paediatric Oncology (SIOP). Unidade: FMRP
Subjects: CRIANÇAS, NEOPLASIAS CEREBRAIS, EXPRESSÃO GÊNICA, LINHAGEM CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEZUK, Julia Alejandra et al. Polo-like kinase 1 as a potential target in pediatric medulloblastoma. Pediatric Blood & Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2012
APA
Pezuk, J. A., Brassesco, M. S., Oliveira, J. C. de, Morales, A. G., Queiroz, R. G. de P., Scrideli, C. A., et al. (2012). Polo-like kinase 1 as a potential target in pediatric medulloblastoma. Pediatric Blood & Cancer. Hoboken: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Pezuk JA, Brassesco MS, Oliveira JC de, Morales AG, Queiroz RG de P, Scrideli CA, Valera ET, Tone LG. Polo-like kinase 1 as a potential target in pediatric medulloblastoma. Pediatric Blood & Cancer. 2012 ; 59( 6): 1092.[citado 2024 nov. 18 ]
Vancouver
Pezuk JA, Brassesco MS, Oliveira JC de, Morales AG, Queiroz RG de P, Scrideli CA, Valera ET, Tone LG. Polo-like kinase 1 as a potential target in pediatric medulloblastoma. Pediatric Blood & Cancer. 2012 ; 59( 6): 1092.[citado 2024 nov. 18 ]
Artigo de periodico
Pediatric meningiomas: a single-center experience with 15 consecutive cases and review of the literature (2012)
Santos, Marcelo Volpon; Furlanetti, Luciano; Valera, Elvis Terci; Brassesco, Maria Sol; Tone, Luiz Gonzaga; Oliveira, Ricardo Santos de
Source: Child's Nervous System. Unidade: FMRP
Subjects: MENINGIOMA, NEUROFIBROMATOSES, NEOPLASIAS CEREBRAIS, NEUROCIRURGIA, PEDIATRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Marcelo Volpon et al. Pediatric meningiomas: a single-center experience with 15 consecutive cases and review of the literature. Child's Nervous System, v. 28, n. 11, p. 1887-1896, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00381-012-1823-8. Acesso em: 18 nov. 2024.
APA
Santos, M. V., Furlanetti, L., Valera, E. T., Brassesco, M. S., Tone, L. G., & Oliveira, R. S. de. (2012). Pediatric meningiomas: a single-center experience with 15 consecutive cases and review of the literature. Child's Nervous System, 28( 11), 1887-1896. doi:10.1007/s00381-012-1823-8
NLM
Santos MV, Furlanetti L, Valera ET, Brassesco MS, Tone LG, Oliveira RS de. Pediatric meningiomas: a single-center experience with 15 consecutive cases and review of the literature [Internet]. Child's Nervous System. 2012 ; 28( 11): 1887-1896.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00381-012-1823-8
Vancouver
Santos MV, Furlanetti L, Valera ET, Brassesco MS, Tone LG, Oliveira RS de. Pediatric meningiomas: a single-center experience with 15 consecutive cases and review of the literature [Internet]. Child's Nervous System. 2012 ; 28( 11): 1887-1896.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00381-012-1823-8
Artigo de periodico
Inhibition of Aurora Kinases enhances chemosensitivity to temozolomide and causes radiosensitization in glioblastoma cells (2012)
Borges, Kleiton Silva; Gamero, Angel Mauricio Castro; Moreno, Daniel Antunes; Silveira, Vanessa da Silva; Brassesco, Maria Sol; Queiroz, Rosane Gomes de Paula; Oliveira, Harley Francisco de; Carlotti Júnior, Carlos Gilberto; Scrideli, Carlos Alberto; Tone, Luiz Gonzaga
Source: Journal of Cancer Research and Clinical Oncology. Unidade: FMRP
Subjects: NEOPLASIAS CEREBRAIS, PROTEÍNAS QUINASES, ANTINEOPLÁSICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Kleiton Silva et al. Inhibition of Aurora Kinases enhances chemosensitivity to temozolomide and causes radiosensitization in glioblastoma cells. Journal of Cancer Research and Clinical Oncology, v. 138, n. 3, p. 405-414, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00432-011-1111-0. Acesso em: 18 nov. 2024.
APA
Borges, K. S., Gamero, A. M. C., Moreno, D. A., Silveira, V. da S., Brassesco, M. S., Queiroz, R. G. de P., et al. (2012). Inhibition of Aurora Kinases enhances chemosensitivity to temozolomide and causes radiosensitization in glioblastoma cells. Journal of Cancer Research and Clinical Oncology, 138( 3), 405-414. doi:10.1007/s00432-011-1111-0
NLM
Borges KS, Gamero AMC, Moreno DA, Silveira V da S, Brassesco MS, Queiroz RG de P, Oliveira HF de, Carlotti Júnior CG, Scrideli CA, Tone LG. Inhibition of Aurora Kinases enhances chemosensitivity to temozolomide and causes radiosensitization in glioblastoma cells [Internet]. Journal of Cancer Research and Clinical Oncology. 2012 ; 138( 3): 405-414.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00432-011-1111-0
Vancouver
Borges KS, Gamero AMC, Moreno DA, Silveira V da S, Brassesco MS, Queiroz RG de P, Oliveira HF de, Carlotti Júnior CG, Scrideli CA, Tone LG. Inhibition of Aurora Kinases enhances chemosensitivity to temozolomide and causes radiosensitization in glioblastoma cells [Internet]. Journal of Cancer Research and Clinical Oncology. 2012 ; 138( 3): 405-414.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00432-011-1111-0

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