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Artigo de periodico
Role of dystrophin in acute Trypanosoma cruzi infection (2014)
Malvestio, Lygia M; Celes, Mara R. N; Milanezi, Cristiane Maria; Silva, João Santana da; Jelicks, Linda A; Tanowitz, Hebert B; Rossi, Marcos Antonio; Prado, Cibele Maria
Source: Microbes and Infection. Unidade: FMRP
Subjects: DOENÇA DE CHAGAS, INFLAMAÇÃO (METABOLISMO), TRYPANOSOMA CRUZI, MIÓCITOS DE MÚSCULO LISO
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ABNT
MALVESTIO, Lygia M et al. Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 9, p. 768-777, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.07.010. Acesso em: 17 nov. 2024.
APA
Malvestio, L. M., Celes, M. R. N., Milanezi, C. M., Silva, J. S. da, Jelicks, L. A., Tanowitz, H. B., et al. (2014). Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, 16( 9), 768-777. doi:10.1016/j.micinf.2014.07.010
NLM
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Vancouver
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Artigo de periodico
Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms (2011)
Oliveira, Marcela S.; Floriano, Elaine Medeiros; Mazin, Suleimy Cristina; Martinez, Edson Zangiacomi; Vicente, Walter Villela de Andrade; Peres, Luiz Cesar; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Source: Cardiovascular Pathology. Unidade: FMRP
Subjects: ISQUEMIA MIOCÁRDICA, CARDIOPATIAS CONGÊNITAS, ESTRESSE OXIDATIVO
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ABNT
OLIVEIRA, Marcela S. et al. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms. Cardiovascular Pathology, v. 20, n. 1, p. e43-e52, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.carpath.2010.01.012. Acesso em: 17 nov. 2024.
APA
Oliveira, M. S., Floriano, E. M., Mazin, S. C., Martinez, E. Z., Vicente, W. V. de A., Peres, L. C., et al. (2011). Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms. Cardiovascular Pathology, 20( 1), e43-e52. doi:10.1016/j.carpath.2010.01.012
NLM
Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, Vicente WV de A, Peres LC, Rossi MA, Ramos SG. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms [Internet]. Cardiovascular Pathology. 2011 ;20( 1): e43-e52.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.carpath.2010.01.012
Vancouver
Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, Vicente WV de A, Peres LC, Rossi MA, Ramos SG. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms [Internet]. Cardiovascular Pathology. 2011 ;20( 1): e43-e52.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.carpath.2010.01.012
Artigo de periodico
Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle (2010)
Baruffi, Marcelo Dias; Stowell, Sean R.; Song, Shuh-Chyung; Arthur, Connie M.; Cho, Moonjae; Montes, Marlise Bonetti Agostinho; Rossi, Marcos Antonio; James, Judith A.; McEver, Rodger P.; Rodrigues, Lílian Cataldi; Cummings, Richard D.
Source: Glycobiology. Unidades: FCFRP, ICB, FMRP
Subjects: LEUCÓCITOS, MÚSCULOS, ANTICORPOS MONOCLONAIS
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ABNT
BARUFFI, Marcelo Dias et al. Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle. Glycobiology, v. 20, n. 5, p. 507-520, 2010Tradução . . Disponível em: https://doi.org/10.1093/glycob/cwp203. Acesso em: 17 nov. 2024.
APA
Baruffi, M. D., Stowell, S. R., Song, S. -C., Arthur, C. M., Cho, M., Montes, M. B. A., et al. (2010). Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle. Glycobiology, 20( 5), 507-520. doi:10.1093/glycob/cwp203
NLM
Baruffi MD, Stowell SR, Song S-C, Arthur CM, Cho M, Montes MBA, Rossi MA, James JA, McEver RP, Rodrigues LC, Cummings RD. Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle [Internet]. Glycobiology. 2010 ; 20( 5): 507-520.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1093/glycob/cwp203
Vancouver
Baruffi MD, Stowell SR, Song S-C, Arthur CM, Cho M, Montes MBA, Rossi MA, James JA, McEver RP, Rodrigues LC, Cummings RD. Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle [Internet]. Glycobiology. 2010 ; 20( 5): 507-520.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1093/glycob/cwp203
Artigo de periodico
Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling (2010)
Castro, Michele Mazzaron de; Rizzi, Elen; Prado, Cibele Maria; Rossi, Marcos Antonio; Santos, José Eduardo Tanus dos; Gerlach, Raquel Fernanda
Source: Matrix Biology. Unidades: FMRP, FORP
Subjects: HIPERTENSÃO, METALOPROTEINASES
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ABNT
CASTRO, Michele Mazzaron de et al. Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling. Matrix Biology, v. 29, n. 3, p. 194-201, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.matbio.2009.11.005. Acesso em: 17 nov. 2024.
APA
Castro, M. M. de, Rizzi, E., Prado, C. M., Rossi, M. A., Santos, J. E. T. dos, & Gerlach, R. F. (2010). Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling. Matrix Biology, 29( 3), 194-201. doi:10.1016/j.matbio.2009.11.005
NLM
Castro MM de, Rizzi E, Prado CM, Rossi MA, Santos JET dos, Gerlach RF. Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling [Internet]. Matrix Biology. 2010 ; 29( 3): 194-201.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.matbio.2009.11.005
Vancouver
Castro MM de, Rizzi E, Prado CM, Rossi MA, Santos JET dos, Gerlach RF. Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling [Internet]. Matrix Biology. 2010 ; 29( 3): 194-201.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.matbio.2009.11.005
Artigo de periodico
Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis (2010)
Celes, Mara Rúbia Nunes; Malvestio, Lygia M.; Torres-Dueñas, Diego; Blefari, Valdecir; Campos, Érica Carolina; Ramos, Simone Gusmão; Prado, Cibele Maria; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Source: Laboratory Investigation. Unidade: FMRP
Subjects: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
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ABNT
CELES, Mara Rúbia Nunes et al. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, v. 90, n. 4, p. 531-542, 2010Tradução . . Disponível em: https://doi.org/10.1186/cc9157. Acesso em: 17 nov. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Torres-Dueñas, D., Blefari, V., Campos, É. C., Ramos, S. G., et al. (2010). Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, 90( 4), 531-542. doi:10.1186/cc9157
NLM
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1186/cc9157
Vancouver
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1186/cc9157
Artigo de periodico
Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress (2009)
Faria, Gisele; Cardoso, Cristina Ribeiro Barros; Larson, Roy Edward; Silva, João Santana da; Rossi, Marcos Antonio
Source: Toxicology and Applied Pharmacology. Unidades: FORP, FMRP
Subjects: APOPTOSE, FIBROBLASTOS, RETÍCULO ENDOPLASMÁTICO
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ABNT
FARIA, Gisele et al. Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress. Toxicology and Applied Pharmacology, v. 234, n. 2, p. 256-265, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2008.10.012. Acesso em: 17 nov. 2024.
APA
Faria, G., Cardoso, C. R. B., Larson, R. E., Silva, J. S. da, & Rossi, M. A. (2009). Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress. Toxicology and Applied Pharmacology, 234( 2), 256-265. doi:10.1016/j.taap.2008.10.012
NLM
Faria G, Cardoso CRB, Larson RE, Silva JS da, Rossi MA. Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress [Internet]. Toxicology and Applied Pharmacology. 2009 ; 234( 2): 256-265.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.taap.2008.10.012
Vancouver
Faria G, Cardoso CRB, Larson RE, Silva JS da, Rossi MA. Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress [Internet]. Toxicology and Applied Pharmacology. 2009 ; 234( 2): 256-265.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.taap.2008.10.012
Artigo de periodico
Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? (2009)
Beneli, Cristina Tonin; Barbosa, Priscila de Freitas; Floriano, Elaine Medeiros; Abreu, Mônica Azevedo de; Ramalho, Fernando Silva; Elias Júnior, Jorge; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Source: International Journal of Experimental Pathology. Unidade: FMRP
Subjects: ARTERIOSCLEROSE, ESTENOSE DA VALVA AÓRTICA, FIBRINÓLISE
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ABNT
BENELI, Cristina Tonin et al. Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation?. International Journal of Experimental Pathology, v. 90, n. 3, p. 295-310, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2613.2008.00630.x. Acesso em: 17 nov. 2024.
APA
Beneli, C. T., Barbosa, P. de F., Floriano, E. M., Abreu, M. A. de, Ramalho, F. S., Elias Júnior, J., et al. (2009). Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? International Journal of Experimental Pathology, 90( 3), 295-310. doi:10.1111/j.1365-2613.2008.00630.x
NLM
Beneli CT, Barbosa P de F, Floriano EM, Abreu MA de, Ramalho FS, Elias Júnior J, Rossi MA, Ramos SG. Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? [Internet]. International Journal of Experimental Pathology. 2009 ; 90( 3): 295-310.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/j.1365-2613.2008.00630.x
Vancouver
Beneli CT, Barbosa P de F, Floriano EM, Abreu MA de, Ramalho FS, Elias Júnior J, Rossi MA, Ramos SG. Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? [Internet]. International Journal of Experimental Pathology. 2009 ; 90( 3): 295-310.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/j.1365-2613.2008.00630.x
Artigo de periodico
B cells are involved in the modulation of pathogenic gut immune response in food-allergic enteropathy (2008)
Cardoso, Cristina Ribeiro; Provinciatto, Pauline Rossetti; Godoi, Dannielle Fernandes; Vieira, Thalita Silva; Ferreira, Beatriz Rossetti ; Teixeira, Gerlinde; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: Clinical and Experimental Immunology. Unidades: FMRP, EERP
Subjects: ENTEROPATIAS INFLAMATÓRIAS, HIPERSENSIBILIDADE ALIMENTAR (ANÁLISE)
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ABNT
CARDOSO, Cristina Ribeiro et al. B cells are involved in the modulation of pathogenic gut immune response in food-allergic enteropathy. Clinical and Experimental Immunology, v. 154, n. 2, p. 153-161, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2249.2008.03748.x. Acesso em: 17 nov. 2024.
APA
Cardoso, C. R., Provinciatto, P. R., Godoi, D. F., Vieira, T. S., Ferreira, B. R., Teixeira, G., et al. (2008). B cells are involved in the modulation of pathogenic gut immune response in food-allergic enteropathy. Clinical and Experimental Immunology, 154( 2), 153-161. doi:10.1111/j.1365-2249.2008.03748.x
NLM
Cardoso CR, Provinciatto PR, Godoi DF, Vieira TS, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. B cells are involved in the modulation of pathogenic gut immune response in food-allergic enteropathy [Internet]. Clinical and Experimental Immunology. 2008 ; 154( 2): 153-161.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/j.1365-2249.2008.03748.x
Vancouver
Cardoso CR, Provinciatto PR, Godoi DF, Vieira TS, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. B cells are involved in the modulation of pathogenic gut immune response in food-allergic enteropathy [Internet]. Clinical and Experimental Immunology. 2008 ; 154( 2): 153-161.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/j.1365-2249.2008.03748.x

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