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  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SOUZA, Arnaldo H. de et al. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, v. 439, p. 354-362, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.09.022. Acesso em: 10 jul. 2024.
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      Souza, A. H. de, Santos, L. R. B., Roma, L. P., Bensellam, M., Carpinelli, A. R., & Jonas, J. -C. (2017). NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, 439, 354-362. doi:10.1016/j.mce.2016.09.022
    • NLM

      Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
    • Vancouver

      Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
  • Source: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      FERREIRA-NETO, H. C. et al. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation. Neuroscience, v. 349, p. 253-263, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.02.05. Acesso em: 10 jul. 2024.
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      Ferreira-Neto, H. C., Ribeiro, I. M. R., Moreira, T. dos S., Yao, S. T., & Antunes, V. R. (2017). Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation. Neuroscience, 349, 253-263. doi:10.1016/j.neuroscience.2017.02.05
    • NLM

      Ferreira-Neto HC, Ribeiro IMR, Moreira T dos S, Yao ST, Antunes VR. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation [Internet]. Neuroscience. 2017 ; 349 253-263.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.05
    • Vancouver

      Ferreira-Neto HC, Ribeiro IMR, Moreira T dos S, Yao ST, Antunes VR. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation [Internet]. Neuroscience. 2017 ; 349 253-263.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.05
  • Source: Acta Physiologica. Unidade: ICB

    Assunto: FISIOLOGIA

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      TEIXEIRA, S. da Silva et al. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice. Acta Physiologica, v. 220, n. 2, p. 238-250, 2017Tradução . . Disponível em: https://doi.org/10.1111/apha.12821. Acesso em: 10 jul. 2024.
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      Teixeira, S. da S., Filgueira, C., Sieglaff, D. H., Benod , C., Villagomez, R., Minze, L. J., et al. (2017). 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice. Acta Physiologica, 220( 2), 238-250. doi:10.1111/apha.12821
    • NLM

      Teixeira S da S, Filgueira C, Sieglaff DH, Benod C, Villagomez R, Minze LJ, Zhang A, Webb P, Nunes MT. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice [Internet]. Acta Physiologica. 2017 ; 220( 2): 238-250.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1111/apha.12821
    • Vancouver

      Teixeira S da S, Filgueira C, Sieglaff DH, Benod C, Villagomez R, Minze LJ, Zhang A, Webb P, Nunes MT. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice [Internet]. Acta Physiologica. 2017 ; 220( 2): 238-250.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1111/apha.12821
  • Source: Journal of Proteomics. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      CAFÉ-MENDES, C. C. et al. Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients. Journal of Proteomics, v. 151, p. 97-105, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2016.05.025. Acesso em: 10 jul. 2024.
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      Café-Mendes, C. C., Ferro, E. S., Torrão, A. da S., Crunfli, F., Rioli, V., Schmitt, A., et al. (2017). Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients. Journal of Proteomics, 151, 97-105. doi:10.1016/j.jprot.2016.05.025
    • NLM

      Café-Mendes CC, Ferro ES, Torrão A da S, Crunfli F, Rioli V, Schmitt A, Falkai P, Britto LRG de, Turck CW, Martins-de-Souza D. Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients [Internet]. Journal of Proteomics. 2017 ; 151 97-105.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.jprot.2016.05.025
    • Vancouver

      Café-Mendes CC, Ferro ES, Torrão A da S, Crunfli F, Rioli V, Schmitt A, Falkai P, Britto LRG de, Turck CW, Martins-de-Souza D. Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients [Internet]. Journal of Proteomics. 2017 ; 151 97-105.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.jprot.2016.05.025
  • Source: Neurotoxicity Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RAVELLI, Katherine Garcia et al. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, v. 31, n. 3, p. 327-333, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12640-016-9684-7. Acesso em: 10 jul. 2024.
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      Ravelli, K. G., Rosário, B. dos A., Camarini, R., Hernandes, M. S., & Britto, L. R. G. de. (2017). Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, 31( 3), 327-333. doi:10.1007/s12640-016-9684-7
    • NLM

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
    • Vancouver

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
  • Source: Molecular and Cellular Endocrinology. Unidades: FMRP, ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      SILVEIRA, Marina Augusto et al. STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, v. 448, p. 55-65, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.03.024. Acesso em: 10 jul. 2024.
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      Silveira, M. A., Furigo, I. C., Zampieri, T. T., Bohlen, T. M., Paula, D. G. de, Franci, C. R., et al. (2017). STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, 448, 55-65. doi:10.1016/j.mce.2017.03.024
    • NLM

      Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
    • Vancouver

      Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
  • Source: Journal of Physiology. Unidades: ICB, EEFE

    Subjects: INSUFICIÊNCIA CARDÍACA, SISTEMA NERVOSO PARASSIMPÁTICO, SISTEMA NERVOSO AUTÔNOMO, TREINAMENTO FÍSICO, FISIOLOGIA

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      ICHIGE, Marcelo Hiro Akiyoshi et al. Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure. Journal of Physiology, v. no 2016, n. 21, p. 6241-6254, 2016Tradução . . Disponível em: https://doi.org/10.1113/JP272730. Acesso em: 10 jul. 2024.
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      Ichige, M. H. A., Santos, C. R. dos, Jordão, C. P., Ceroni, A., Negrão, C. E., & Michelini, L. C. (2016). Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure. Journal of Physiology, no 2016( 21), 6241-6254. doi:10.1113/JP272730
    • NLM

      Ichige MHA, Santos CR dos, Jordão CP, Ceroni A, Negrão CE, Michelini LC. Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure [Internet]. Journal of Physiology. 2016 ; no 2016( 21): 6241-6254.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/JP272730
    • Vancouver

      Ichige MHA, Santos CR dos, Jordão CP, Ceroni A, Negrão CE, Michelini LC. Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure [Internet]. Journal of Physiology. 2016 ; no 2016( 21): 6241-6254.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/JP272730
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline et al. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, v. 426, p. 73-90, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.02.006. Acesso em: 10 jul. 2024.
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      Serrano-Nascimento, C., Nicola, J. P., Teixeira, S. da S., Poyares, L. L., Lellis-Santos, C., Bordin, S., et al. (2016). Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, 426, 73-90. doi:10.1016/j.mce.2016.02.006
    • NLM

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
    • Vancouver

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
  • Source: Journal of Comparative Neurology. Unidade: ICB

    Subjects: SISTEMA NERVOSO AUTÔNOMO, FISIOLOGIA

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      GOKOZAN, Hamza Numan et al. Area postrema undergoes dynamic postnatal changes in mice and humans. Journal of Comparative Neurology, v. 524, n. 6, p. 1259-1269, 2016Tradução . . Disponível em: https://doi.org/10.1002/cne.23903. Acesso em: 10 jul. 2024.
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      Gokozan, H. N., Baig, F., Corcoran, S., Catacutan, F. P., Gygli, P. E., Takakura, A. C., et al. (2016). Area postrema undergoes dynamic postnatal changes in mice and humans. Journal of Comparative Neurology, 524( 6), 1259-1269. doi:10.1002/cne.23903
    • NLM

      Gokozan HN, Baig F, Corcoran S, Catacutan FP, Gygli PE, Takakura AC, Moreira T dos S, Czeisler C, Otero JJ. Area postrema undergoes dynamic postnatal changes in mice and humans [Internet]. Journal of Comparative Neurology. 2016 ; 524( 6): 1259-1269.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1002/cne.23903
    • Vancouver

      Gokozan HN, Baig F, Corcoran S, Catacutan FP, Gygli PE, Takakura AC, Moreira T dos S, Czeisler C, Otero JJ. Area postrema undergoes dynamic postnatal changes in mice and humans [Internet]. Journal of Comparative Neurology. 2016 ; 524( 6): 1259-1269.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1002/cne.23903
  • Source: Life Sciences. Unidade: ICB

    Subjects: FISIOLOGIA, ENDOTÉLIO, RATOS, ARTÉRIA PULMONAR, ADRENÉRGICOS

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      DAVEL, Ana P. et al. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, v. 125, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.01.018. Acesso em: 10 jul. 2024.
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      Davel, A. P., Victorio, J. A., Delbin, M. A., Fukuda, L. E., & Rossoni, L. V. (2015). Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, 125, 49-56. doi:10.1016/j.lfs.2015.01.018
    • NLM

      Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
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      Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
  • Source: Journal of Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      ROBERTO SOBRINHO, Cleyton et al. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, v. 15, p. 1309-1323, 2014Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2013.268490. Acesso em: 10 jul. 2024.
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      Roberto Sobrinho, C., Wenker, I. C., Poss, E. M., Takakura, A. C., Moreira, T. dos S., & Mulkey, D. K. (2014). Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, 15, 1309-1323. doi:10.1113/jphysiol.2013.268490
    • NLM

      Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
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      Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
  • Source: Mediators of Inflammation. Unidade: ICB

    Assunto: FISIOLOGIA

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      MOREIRA, Vanessa et al. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/105879. Acesso em: 10 jul. 2024.
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      Moreira, V., Lomonte, B., Vinolo, M. A. R., Curi, R., Gutiérrez, J. M., & Teixeira, C. (2014). An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, 2014, 1-10. doi:10.1155/2014/105879
    • NLM

      Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1155/2014/105879
    • Vancouver

      Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1155/2014/105879
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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      MORAES, Paulo Alexandre et al. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, v. 114, n. 1, p. 36-44, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.07.040. Acesso em: 10 jul. 2024.
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      Moraes, P. A., Yonamine, C. Y., Pinto Junior, D. C., Esteves, J. V. D. C., Machado, U. F., & Mori, R. C. (2014). Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, 114( 1), 36-44. doi:10.1016/j.lfs.2014.07.040
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      Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
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      Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
  • Source: Mediators of Inflammation. Unidade: ICB

    Subjects: FISIOLOGIA, HISTOLOGIA

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      ROSA NETO, José Cesar e LIRA, Fábio Santos e FESTUCCIA, William Tadeu Lara. Immunometabolism: molecular mechanisms, diseases, and therapies. Mediators of Inflammation. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1155/2014/585708. Acesso em: 10 jul. 2024. , 2014
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      Rosa Neto, J. C., Lira, F. S., & Festuccia, W. T. L. (2014). Immunometabolism: molecular mechanisms, diseases, and therapies. Mediators of Inflammation. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1155/2014/585708
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      Rosa Neto JC, Lira FS, Festuccia WTL. Immunometabolism: molecular mechanisms, diseases, and therapies [Internet]. Mediators of Inflammation. 2014 ; 2014 1-2.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1155/2014/585708
    • Vancouver

      Rosa Neto JC, Lira FS, Festuccia WTL. Immunometabolism: molecular mechanisms, diseases, and therapies [Internet]. Mediators of Inflammation. 2014 ; 2014 1-2.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1155/2014/585708
  • Source: Experimental Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      TAKAKURA, Ana Carolina et al. Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats. Experimental Physiology, v. 99, n. 3, p. 571-585, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.076752. Acesso em: 10 jul. 2024.
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      Takakura, A. C., Falquetto, B., Cruz, J. C., Colombari, E., & Moreira, T. dos S. (2014). Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats. Experimental Physiology, 99( 3), 571-585. doi:10.1113/expphysiol.2013.076752
    • NLM

      Takakura AC, Falquetto B, Cruz JC, Colombari E, Moreira T dos S. Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats [Internet]. Experimental Physiology. 2014 ; 99( 3): 571-585.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/expphysiol.2013.076752
    • Vancouver

      Takakura AC, Falquetto B, Cruz JC, Colombari E, Moreira T dos S. Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats [Internet]. Experimental Physiology. 2014 ; 99( 3): 571-585.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/expphysiol.2013.076752
  • Source: Metabolism: Clinical and Experimental. Unidade: ICB

    Subjects: FISIOLOGIA, INSULINA, RNA, EXPRESSÃO GÊNICA

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      ZANQUETTA, Melissa Moreira et al. Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding. Metabolism: Clinical and Experimental, v. 63, n. 3, p. 328-334, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2013.11.001. Acesso em: 10 jul. 2024.
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      Zanquetta, M. M., Alves-Wagner, A. B., Mori, R. C., Campello, R. S., & Machado, U. F. (2014). Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding. Metabolism: Clinical and Experimental, 63( 3), 328-334. doi:10.1016/j.metabol.2013.11.001
    • NLM

      Zanquetta MM, Alves-Wagner AB, Mori RC, Campello RS, Machado UF. Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding [Internet]. Metabolism: Clinical and Experimental. 2014 ; 63( 3): 328-334.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.metabol.2013.11.001
    • Vancouver

      Zanquetta MM, Alves-Wagner AB, Mori RC, Campello RS, Machado UF. Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding [Internet]. Metabolism: Clinical and Experimental. 2014 ; 63( 3): 328-334.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.metabol.2013.11.001
  • Source: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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      WENCESLAU, Camilla F. e ROSSONI, Luciana Venturini. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, v. 32, n. 3, p. 542-555, 2014Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000000059. Acesso em: 10 jul. 2024.
    • APA

      Wenceslau, C. F., & Rossoni, L. V. (2014). Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, 32( 3), 542-555. doi:10.1097/HJH.0000000000000059
    • NLM

      Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
    • Vancouver

      Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
  • Source: BioMed Research International. Unidade: ICB

    Assunto: FISIOLOGIA

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      AMARAL, Cátia Lira do et al. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil. BioMed Research International, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/675021. Acesso em: 10 jul. 2024.
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      Amaral, C. L. do, Milagro, F. I., Curi, R., & Martínez, J. A. (2014). DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil. BioMed Research International, 2014, 1-10. doi:10.1155/2014/675021
    • NLM

      Amaral CL do, Milagro FI, Curi R, Martínez JA. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil [Internet]. BioMed Research International. 2014 ; 2014 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1155/2014/675021
    • Vancouver

      Amaral CL do, Milagro FI, Curi R, Martínez JA. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil [Internet]. BioMed Research International. 2014 ; 2014 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1155/2014/675021
  • Source: Journal of Comparative Neurology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SEGO, Chemutai et al. Lateral habenula and the rostromedial tegmental nucleus innervate neurochemically distinct subdivisions of the dorsal raphe nucleus in the rat. Journal of Comparative Neurology, v. 522, n. 7, p. 1454-1484, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0098614. Acesso em: 10 jul. 2024.
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      Sego, C., Gonçalves, L., Lima, L., Furigo, I. C., Donato Junior, J., & Metzger, M. A. (2014). Lateral habenula and the rostromedial tegmental nucleus innervate neurochemically distinct subdivisions of the dorsal raphe nucleus in the rat. Journal of Comparative Neurology, 522( 7), 1454-1484. doi:10.1371/journal.pone.0098614
    • NLM

      Sego C, Gonçalves L, Lima L, Furigo IC, Donato Junior J, Metzger MA. Lateral habenula and the rostromedial tegmental nucleus innervate neurochemically distinct subdivisions of the dorsal raphe nucleus in the rat [Internet]. Journal of Comparative Neurology. 2014 ; 522( 7): 1454-1484.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1371/journal.pone.0098614
    • Vancouver

      Sego C, Gonçalves L, Lima L, Furigo IC, Donato Junior J, Metzger MA. Lateral habenula and the rostromedial tegmental nucleus innervate neurochemically distinct subdivisions of the dorsal raphe nucleus in the rat [Internet]. Journal of Comparative Neurology. 2014 ; 522( 7): 1454-1484.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1371/journal.pone.0098614
  • Source: Acta Physiologica. Unidade: ICB

    Assunto: FISIOLOGIA

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      YONAMINE, Caio Yogi et al. Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats. Acta Physiologica, v. 212, n. 1, p. 62-74, 2014Tradução . . Disponível em: https://doi.org/10.1111/apha.12336. Acesso em: 10 jul. 2024.
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      Yonamine, C. Y., Teixeira, S. S., Campello, R. S., Gerlinger-Romero, F., Rodrigues Jr, C. F., Guimarães-Ferreira, L., et al. (2014). Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats. Acta Physiologica, 212( 1), 62-74. doi:10.1111/apha.12336
    • NLM

      Yonamine CY, Teixeira SS, Campello RS, Gerlinger-Romero F, Rodrigues Jr CF, Guimarães-Ferreira L, Machado UF, Nunes MT. Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats [Internet]. Acta Physiologica. 2014 ; 212( 1): 62-74.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1111/apha.12336
    • Vancouver

      Yonamine CY, Teixeira SS, Campello RS, Gerlinger-Romero F, Rodrigues Jr CF, Guimarães-Ferreira L, Machado UF, Nunes MT. Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats [Internet]. Acta Physiologica. 2014 ; 212( 1): 62-74.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1111/apha.12336

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