ReP USP - Resultado da busca

Artigo de periodico
Ethanol: striking the cardiovascular system by harming the gut microbiota (2021)
Silva, Carla Brigagão Pacheco da ; Oliveira, Jefferson Elias ; McCarthy, Cameron; Wenceslau, Camilla F.; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology : Heart and Circulatory Physiology. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, ÁLCOOL, DOENÇAS CARDIOVASCULARES, PREBIÓTICOS, PROBIÓTICOS
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ABNT
SILVA, Carla Brigagão Pacheco da et al. Ethanol: striking the cardiovascular system by harming the gut microbiota. American Journal of Physiology : Heart and Circulatory Physiology, v. 321, n. 2, p. H275-H291, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00225.2021. Acesso em: 13 nov. 2024.
APA
Silva, C. B. P. da, Oliveira, J. E., McCarthy, C., Wenceslau, C. F., Carlos, D., & Tostes, R. de C. A. (2021). Ethanol: striking the cardiovascular system by harming the gut microbiota. American Journal of Physiology : Heart and Circulatory Physiology, 321( 2), H275-H291. doi:10.1152/ajpheart.00225.2021
NLM
Silva CBP da, Oliveira JE, McCarthy C, Wenceslau CF, Carlos D, Tostes R de CA. Ethanol: striking the cardiovascular system by harming the gut microbiota [Internet]. American Journal of Physiology : Heart and Circulatory Physiology. 2021 ; 321( 2): H275-H291.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpheart.00225.2021
Vancouver
Silva CBP da, Oliveira JE, McCarthy C, Wenceslau CF, Carlos D, Tostes R de CA. Ethanol: striking the cardiovascular system by harming the gut microbiota [Internet]. American Journal of Physiology : Heart and Circulatory Physiology. 2021 ; 321( 2): H275-H291.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpheart.00225.2021
Parte de monografia/livro
Modulación de la dinámica arterial (2019)
Ballejo, Gustavo ; Tostes, Rita de Cássia Aleixo ; Salgado, Hélio César
Source: Biomecánica arterial: bases fisiológicas y aplicaciones clínicas. Unidade: FMRP
Subjects: FISIOLOGIA, SISTEMA CARDIOVASCULAR, MÚSCULO LISO VASCULAR
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ABNT
BALLEJO, Gustavo e TOSTES, Rita de Cássia Aleixo e SALGADO, Hélio César. Modulación de la dinámica arterial. Biomecánica arterial: bases fisiológicas y aplicaciones clínicas. Tradução . Ciudad Autónoma de Buenos Aires: Fundación Universitaria Dr. René Favaloro, 2019. . . Acesso em: 13 nov. 2024.
APA
Ballejo, G., Tostes, R. de C. A., & Salgado, H. C. (2019). Modulación de la dinámica arterial. In Biomecánica arterial: bases fisiológicas y aplicaciones clínicas. Ciudad Autónoma de Buenos Aires: Fundación Universitaria Dr. René Favaloro.
NLM
Ballejo G, Tostes R de CA, Salgado HC. Modulación de la dinámica arterial. In: Biomecánica arterial: bases fisiológicas y aplicaciones clínicas. Ciudad Autónoma de Buenos Aires: Fundación Universitaria Dr. René Favaloro; 2019. [citado 2024 nov. 13 ]
Vancouver
Ballejo G, Tostes R de CA, Salgado HC. Modulación de la dinámica arterial. In: Biomecánica arterial: bases fisiológicas y aplicaciones clínicas. Ciudad Autónoma de Buenos Aires: Fundación Universitaria Dr. René Favaloro; 2019. [citado 2024 nov. 13 ]
Artigo de periodico
Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity (2018)
Costa, Rafael M.; Neves, Karla B.; Passaglia, Rita de Cássia Aleixo Tostes; Lobato, Núbia S.
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: TECIDO ADIPOSO, OBESIDADE, SISTEMA CARDIOVASCULAR
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ABNT
COSTA, Rafael M. et al. Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity. Frontiers in Physiology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fphys.2018.00253. Acesso em: 13 nov. 2024.
APA
Costa, R. M., Neves, K. B., Passaglia, R. de C. A. T., & Lobato, N. S. (2018). Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity. Frontiers in Physiology, 9. doi:10.3389/fphys.2018.00253
NLM
Costa RM, Neves KB, Passaglia R de CAT, Lobato NS. Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity [Internet]. Frontiers in Physiology. 2018 ; 9[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fphys.2018.00253
Vancouver
Costa RM, Neves KB, Passaglia R de CAT, Lobato NS. Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity [Internet]. Frontiers in Physiology. 2018 ; 9[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fphys.2018.00253
Trabalho de evento-resumo periodico
Testosterone downregulates the Nrf2 system and promotes vascular dysfunction in HFD fed mice (2017)
Costa, Rafael Menezes; Lopes, Rheure Alves; Mestriner, Fabiola; Tostes, Rita de Cássia Aleixo
Source: FASEB Journal. Conference titles: Experimental Biology. Unidade: FMRP
Subjects: TESTOSTERONA, SISTEMA CARDIOVASCULAR, BIOLOGIA EXPERIMENTAL
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ABNT
COSTA, Rafael Menezes et al. Testosterone downregulates the Nrf2 system and promotes vascular dysfunction in HFD fed mice. FASEB Journal. Bethesda: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 13 nov. 2024. , 2017
APA
Costa, R. M., Lopes, R. A., Mestriner, F., & Tostes, R. de C. A. (2017). Testosterone downregulates the Nrf2 system and promotes vascular dysfunction in HFD fed mice. FASEB Journal. Bethesda: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Costa RM, Lopes RA, Mestriner F, Tostes R de CA. Testosterone downregulates the Nrf2 system and promotes vascular dysfunction in HFD fed mice. FASEB Journal. 2017 ; 31( 1):[citado 2024 nov. 13 ]
Vancouver
Costa RM, Lopes RA, Mestriner F, Tostes R de CA. Testosterone downregulates the Nrf2 system and promotes vascular dysfunction in HFD fed mice. FASEB Journal. 2017 ; 31( 1):[citado 2024 nov. 13 ]
Artigo de periodico
Reactive oxygen species: players in the cardiovascular effects of testosterone (2016)
Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Carvalho, Maria Helena Catelli de; Reckelhoff, Jane F.
Source: American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. Unidades: FMRP, ICB
Subjects: DOENÇAS CARDIOVASCULARES, SISTEMA CARDIOVASCULAR, TERAPIA DA REPOSIÇÃO HORMONAL, ESTRESSE OXIDATIVO, TESTOSTERONA
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ABNT
TOSTES, Rita de Cássia Aleixo et al. Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, v. 310, n. 1, p. R1-R14, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00392.2014. Acesso em: 13 nov. 2024.
APA
Tostes, R. de C. A., Carneiro, F. S., Carvalho, M. H. C. de, & Reckelhoff, J. F. (2016). Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, 310( 1), R1-R14. doi:10.1152/ajpregu.00392.2014
NLM
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Vancouver
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Trabalho de evento-resumo
Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation (2014)
Pereira, Camila A.; Ferreira, Nathane S.; Mestriner, Fabiola Leslie Antunes Cardoso; Resstel, Leonardo Barbosa Moraes; Carneiro, Fernando Silva; Tostes, Rita de Cassia Aleixo
Source: Final Program. Conference titles: High Blood Pressure Research - Scientific Sessions. Unidade: FMRP
Subjects: ANTIDEPRESSIVOS, SISTEMA CARDIOVASCULAR, ARTÉRIAS (ALTERAÇÃO)
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ABNT
PEREIRA, Camila A. et al. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. 2014, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2014. . Acesso em: 13 nov. 2024.
APA
Pereira, C. A., Ferreira, N. S., Mestriner, F. L. A. C., Resstel, L. B. M., Carneiro, F. S., & Tostes, R. de C. A. (2014). Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. In Final Program. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Pereira CA, Ferreira NS, Mestriner FLAC, Resstel LBM, Carneiro FS, Tostes R de CA. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Final Program. 2014 ;[citado 2024 nov. 13 ]
Vancouver
Pereira CA, Ferreira NS, Mestriner FLAC, Resstel LBM, Carneiro FS, Tostes R de CA. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Final Program. 2014 ;[citado 2024 nov. 13 ]
Trabalho de evento-resumo periodico
Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation (2014)
Pereira, Camila A.; Ferreira, Nathanne dos Santos; Mestriner, Fabiola Leslie Antunes Cardoso; Resstel, Leonardo Barbosa Moraes; Carneiro, Fernando Silva; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ANTIDEPRESSIVOS, SISTEMA CARDIOVASCULAR, ARTÉRIAS (ALTERAÇÃO)
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ABNT
PEREIRA, Camila A. et al. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d. Acesso em: 13 nov. 2024. , 2014
APA
Pereira, C. A., Ferreira, N. dos S., Mestriner, F. L. A. C., Resstel, L. B. M., Carneiro, F. S., & Passaglia, R. de C. A. T. (2014). Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d
NLM
Pereira CA, Ferreira N dos S, Mestriner FLAC, Resstel LBM, Carneiro FS, Passaglia R de CAT. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation [Internet]. Hypertension. 2014 ; 64[citado 2024 nov. 13 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d
Vancouver
Pereira CA, Ferreira N dos S, Mestriner FLAC, Resstel LBM, Carneiro FS, Passaglia R de CAT. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation [Internet]. Hypertension. 2014 ; 64[citado 2024 nov. 13 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d
Trabalho de evento-resumo
Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension (2012)
Cau, Stefany B.; Silva, Marcondes A.; Coimbra, Terezila Machado; Alves Filho, José Carlos; Zamboni, Dario Simões; Tostes, Rita de Cassia Aleixo
Source: Final Program. Conference titles: High Blood Pressure Research Scientific Sessions (HBPR). Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, HIPERTENSÃO, ANGIOTENSINA II
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ABNT
CAU, Stefany B. et al. Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. 2012, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 13 nov. 2024.
APA
Cau, S. B., Silva, M. A., Coimbra, T. M., Alves Filho, J. C., Zamboni, D. S., & Tostes, R. de C. A. (2012). Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. In Final Program. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Cau SB, Silva MA, Coimbra TM, Alves Filho JC, Zamboni DS, Tostes R de CA. Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. Final Program. 2012 ;[citado 2024 nov. 13 ]
Vancouver
Cau SB, Silva MA, Coimbra TM, Alves Filho JC, Zamboni DS, Tostes R de CA. Caspase-1 activation contributes to angiotensin-II-induced vascular dysfunction and hypertension. Final Program. 2012 ;[citado 2024 nov. 13 ]
Artigo de periodico
S-nitrosylation inhibits protein kinase C-mediated contraction in mouse aorta (2011)
Choi, Hyehun; Passaglia, Rita de Cassia Aleixo Tostes; Webb, R. Clinton
Source: Journal of Cardiovascular Pharmacology. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, PROTEÍNAS QUINASES
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ABNT
CHOI, Hyehun e PASSAGLIA, Rita de Cassia Aleixo Tostes e WEBB, R. Clinton. S-nitrosylation inhibits protein kinase C-mediated contraction in mouse aorta. Journal of Cardiovascular Pharmacology, v. 57, n. 1, p. 65-71, 2011Tradução . . Acesso em: 13 nov. 2024.
APA
Choi, H., Passaglia, R. de C. A. T., & Webb, R. C. (2011). S-nitrosylation inhibits protein kinase C-mediated contraction in mouse aorta. Journal of Cardiovascular Pharmacology, 57( 1), 65-71.
NLM
Choi H, Passaglia R de CAT, Webb RC. S-nitrosylation inhibits protein kinase C-mediated contraction in mouse aorta. Journal of Cardiovascular Pharmacology. 2011 ; 57( 1): 65-71.[citado 2024 nov. 13 ]
Vancouver
Choi H, Passaglia R de CAT, Webb RC. S-nitrosylation inhibits protein kinase C-mediated contraction in mouse aorta. Journal of Cardiovascular Pharmacology. 2011 ; 57( 1): 65-71.[citado 2024 nov. 13 ]
Artigo de periodico
O-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature (2011)
Lima, Victor Vitorino; Giachini, Fernanda Regina; Hardy, David M.; Webb, Robert Clinton; Passaglia, Rita de Cassia Aleixo Tostes
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, ENDOTELINAS
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ABNT
LIMA, Victor Vitorino et al. O-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 300, n. 2, p. R236-R250, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00230.2010. Acesso em: 13 nov. 2024.
APA
Lima, V. V., Giachini, F. R., Hardy, D. M., Webb, R. C., & Passaglia, R. de C. A. T. (2011). O-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 300( 2), R236-R250. doi:10.1152/ajpregu.00230.2010
NLM
Lima VV, Giachini FR, Hardy DM, Webb RC, Passaglia R de CAT. O-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 2): R236-R250.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpregu.00230.2010
Vancouver
Lima VV, Giachini FR, Hardy DM, Webb RC, Passaglia R de CAT. O-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 2): R236-R250.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpregu.00230.2010
Artigo de periodico
Transient receptor potential melastatin 7 (TRPM7) cation channels, magnesium and the vascular system in hypertension (2011)
Yogi, Álvaro; Callera, Gláucia Elena; Antunes, Tayze T.; Passaglia, Rita de Cassia Aleixo Tostes; Touyz, Rhian M.
Source: Circulation Journal. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, HIPERTENSÃO, MAGNÉSIO
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ABNT
YOGI, Álvaro et al. Transient receptor potential melastatin 7 (TRPM7) cation channels, magnesium and the vascular system in hypertension. Circulation Journal, v. 75, n. 2, p. 237-245, 2011Tradução . . Acesso em: 13 nov. 2024.
APA
Yogi, Á., Callera, G. E., Antunes, T. T., Passaglia, R. de C. A. T., & Touyz, R. M. (2011). Transient receptor potential melastatin 7 (TRPM7) cation channels, magnesium and the vascular system in hypertension. Circulation Journal, 75( 2), 237-245.
NLM
Yogi Á, Callera GE, Antunes TT, Passaglia R de CAT, Touyz RM. Transient receptor potential melastatin 7 (TRPM7) cation channels, magnesium and the vascular system in hypertension. Circulation Journal. 2011 ; 75( 2): 237-245.[citado 2024 nov. 13 ]
Vancouver
Yogi Á, Callera GE, Antunes TT, Passaglia R de CAT, Touyz RM. Transient receptor potential melastatin 7 (TRPM7) cation channels, magnesium and the vascular system in hypertension. Circulation Journal. 2011 ; 75( 2): 237-245.[citado 2024 nov. 13 ]
Artigo de periodico
Receptor and nonreceptor tyrosine kinases in vascular biology of hypertension (2010)
Yogi, Álvaro; O'Connor, Sarah E.; Callera, Glaucia E.; Tostes, Rita de Cássia Aleixo; Touyz, Rhian M.
Source: Current Opinion in Nephrology & Hipertension. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, HIPERTENSÃO, RECEPTORES
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ABNT
YOGI, Álvaro et al. Receptor and nonreceptor tyrosine kinases in vascular biology of hypertension. Current Opinion in Nephrology & Hipertension, v. 19, p. 169-176, 2010Tradução . . Disponível em: https://doi.org/10.1097/mnh.0b013e3283361c24. Acesso em: 13 nov. 2024.
APA
Yogi, Á., O'Connor, S. E., Callera, G. E., Tostes, R. de C. A., & Touyz, R. M. (2010). Receptor and nonreceptor tyrosine kinases in vascular biology of hypertension. Current Opinion in Nephrology & Hipertension, 19, 169-176. doi:10.1097/mnh.0b013e3283361c24
NLM
Yogi Á, O'Connor SE, Callera GE, Tostes R de CA, Touyz RM. Receptor and nonreceptor tyrosine kinases in vascular biology of hypertension [Internet]. Current Opinion in Nephrology & Hipertension. 2010 ; 19 169-176.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/mnh.0b013e3283361c24
Vancouver
Yogi Á, O'Connor SE, Callera GE, Tostes R de CA, Touyz RM. Receptor and nonreceptor tyrosine kinases in vascular biology of hypertension [Internet]. Current Opinion in Nephrology & Hipertension. 2010 ; 19 169-176.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/mnh.0b013e3283361c24

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