ReP USP - Resultado da busca

Artigo de periodico
Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis (2017)
Tiburcio, Taynã Cristina; Willebrords, Joost; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Nogueira, Marina Sayuri; Yanguas, Sara Crespo; Maes, Michaël; Silva, Elisangela dos Anjos; Dagli, Maria Lúcia Zaidan ; Castro, Inar Alves de ; Oliveira, Cláudia Pinto Marques Souza de; Vinken, Mathieu; Cogliati, Bruno
Source: Clinical and Experimental Pharmacology and Physiology. Unidades: FMVZ, FCF, FM
Subjects: CONEXINAS, HEPATOPATIAS, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TIBURCIO, Taynã Cristina et al. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis. Clinical and Experimental Pharmacology and Physiology, v. 44, n. 2, p. 197-206, 2017Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.12701. Acesso em: 05 nov. 2024.
APA
Tiburcio, T. C., Willebrords, J., Silva, T. C. da, Pereira, I. V. A., Nogueira, M. S., Yanguas, S. C., et al. (2017). Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis. Clinical and Experimental Pharmacology and Physiology, 44( 2), 197-206. doi:10.1111/1440-1681.12701
NLM
Tiburcio TC, Willebrords J, Silva TC da, Pereira IVA, Nogueira MS, Yanguas SC, Maes M, Silva E dos A, Dagli MLZ, Castro IA de, Oliveira CPMS de, Vinken M, Cogliati B. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis [Internet]. Clinical and Experimental Pharmacology and Physiology. 2017 ; 44( 2): 197-206.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/1440-1681.12701
Vancouver
Tiburcio TC, Willebrords J, Silva TC da, Pereira IVA, Nogueira MS, Yanguas SC, Maes M, Silva E dos A, Dagli MLZ, Castro IA de, Oliveira CPMS de, Vinken M, Cogliati B. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis [Internet]. Clinical and Experimental Pharmacology and Physiology. 2017 ; 44( 2): 197-206.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/1440-1681.12701
Artigo de periodico
Connexin32: a mediator of acetaminophen-induced liver injury? (2016)
Maes, Michaël; McGill, Mitchell R; Silva, Tereza Cristina da; Lebofsky, Margitta; Araújo, Cintia Maria Monteiro de; Tiburcio, Taynã; Pereira, Isabel Veloso Alves; Willebrords, Joost; Yanguas, Sara Crespo; Farhood, Anwar; Dagli, Maria Lúcia Zaidan ; Jaeschke, Hartmut; Cogliati, Bruno ; Vinken, Mathieu
Source: Toxicology Mechanisms and Methods. Unidade: FMVZ
Subjects: CONEXINAS, HEPATOPATIAS, TOXICIDADE EM ANIMAL, ANALGÉSICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAES, Michaël et al. Connexin32: a mediator of acetaminophen-induced liver injury?. Toxicology Mechanisms and Methods, v. 26, n. 2, p. 88-96, 2016Tradução . . Disponível em: https://doi.org/10.3109/15376516.2015.1103000. Acesso em: 05 nov. 2024.
APA
Maes, M., McGill, M. R., Silva, T. C. da, Lebofsky, M., Araújo, C. M. M. de, Tiburcio, T., et al. (2016). Connexin32: a mediator of acetaminophen-induced liver injury? Toxicology Mechanisms and Methods, 26( 2), 88-96. doi:10.3109/15376516.2015.1103000
NLM
Maes M, McGill MR, Silva TC da, Lebofsky M, Araújo CMM de, Tiburcio T, Pereira IVA, Willebrords J, Yanguas SC, Farhood A, Dagli MLZ, Jaeschke H, Cogliati B, Vinken M. Connexin32: a mediator of acetaminophen-induced liver injury? [Internet]. Toxicology Mechanisms and Methods. 2016 ; 26( 2): 88-96.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3109/15376516.2015.1103000
Vancouver
Maes M, McGill MR, Silva TC da, Lebofsky M, Araújo CMM de, Tiburcio T, Pereira IVA, Willebrords J, Yanguas SC, Farhood A, Dagli MLZ, Jaeschke H, Cogliati B, Vinken M. Connexin32: a mediator of acetaminophen-induced liver injury? [Internet]. Toxicology Mechanisms and Methods. 2016 ; 26( 2): 88-96.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3109/15376516.2015.1103000
Trabalho de evento-resumo periodico
Thioacetamide-induced chronic hepatotoxicity in BALB/c mice: a fibrosis model (2008)
Cogliati, Bruno ; Lima, Mirela Aline Real de; Aloia, Thiago Pinheiro Arrais; Silva, Tereza Cristina da; Matsuzaki, Patrícia; Agreste, Fernanda Rodrigues; Sanches, Daniel Soares; Chaible, Lucas Martins; Hernandez-Blazquez, Francisco Javier ; Dagli, Maria Lúcia Zaidan
Source: Toxicologic Pathology. Conference titles: Society of Toxicologic Pathology's. Annual Symposium Poster Presentations. Unidade: FMVZ
Subjects: DOENÇAS DO SISTEMA DIGESTÓRIO EM ANIMAL, FÍGADO, HEPATOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COGLIATI, Bruno et al. Thioacetamide-induced chronic hepatotoxicity in BALB/c mice: a fibrosis model. Toxicologic Pathology. Lawrence: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. Disponível em: http://tpx.sagepub.com/cgi/content/full/36/1/149. Acesso em: 05 nov. 2024. , 2008
APA
Cogliati, B., Lima, M. A. R. de, Aloia, T. P. A., Silva, T. C. da, Matsuzaki, P., Agreste, F. R., et al. (2008). Thioacetamide-induced chronic hepatotoxicity in BALB/c mice: a fibrosis model. Toxicologic Pathology. Lawrence: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. Recuperado de http://tpx.sagepub.com/cgi/content/full/36/1/149
NLM
Cogliati B, Lima MAR de, Aloia TPA, Silva TC da, Matsuzaki P, Agreste FR, Sanches DS, Chaible LM, Hernandez-Blazquez FJ, Dagli MLZ. Thioacetamide-induced chronic hepatotoxicity in BALB/c mice: a fibrosis model [Internet]. Toxicologic Pathology. 2008 ; 36( 1): 169 Res. P60.[citado 2024 nov. 05 ] Available from: http://tpx.sagepub.com/cgi/content/full/36/1/149
Vancouver
Cogliati B, Lima MAR de, Aloia TPA, Silva TC da, Matsuzaki P, Agreste FR, Sanches DS, Chaible LM, Hernandez-Blazquez FJ, Dagli MLZ. Thioacetamide-induced chronic hepatotoxicity in BALB/c mice: a fibrosis model [Internet]. Toxicologic Pathology. 2008 ; 36( 1): 169 Res. P60.[citado 2024 nov. 05 ] Available from: http://tpx.sagepub.com/cgi/content/full/36/1/149
Trabalho de evento-resumo periodico
Neoplasias hepáticas primárias em cães: Estudo retrospectivo (1999-2007) (2007)
Cogliati, Bruno ; Aloia, T. P. A.; Bosch, R. V.; Sanches, D. S.; Alves, V. A. F.; Mello, E. S.; Dagli, Maria Lúcia Zaidan ; Hernandez-Blazquez, Francisco Javier ; Real Lima, M. A.; Matsuzaki, P.
Source: Jornal brasileiro de patologia e medicina laboratorial. Conference titles: Congresso Brasileiro de Patologia. Unidades: FM, FMVZ
Subjects: NEOPLASIAS HEPÁTICAS (CLASSIFICAÇÃO), HEPATOPATIAS, CÃES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COGLIATI, Bruno et al. Neoplasias hepáticas primárias em cães: Estudo retrospectivo (1999-2007). Jornal brasileiro de patologia e medicina laboratorial. Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 05 nov. 2024. , 2007
APA
Cogliati, B., Aloia, T. P. A., Bosch, R. V., Sanches, D. S., Alves, V. A. F., Mello, E. S., et al. (2007). Neoplasias hepáticas primárias em cães: Estudo retrospectivo (1999-2007). Jornal brasileiro de patologia e medicina laboratorial. Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo.
NLM
Cogliati B, Aloia TPA, Bosch RV, Sanches DS, Alves VAF, Mello ES, Dagli MLZ, Hernandez-Blazquez FJ, Real Lima MA, Matsuzaki P. Neoplasias hepáticas primárias em cães: Estudo retrospectivo (1999-2007). Jornal brasileiro de patologia e medicina laboratorial. 2007 ; 43( 5): 137.[citado 2024 nov. 05 ]
Vancouver
Cogliati B, Aloia TPA, Bosch RV, Sanches DS, Alves VAF, Mello ES, Dagli MLZ, Hernandez-Blazquez FJ, Real Lima MA, Matsuzaki P. Neoplasias hepáticas primárias em cães: Estudo retrospectivo (1999-2007). Jornal brasileiro de patologia e medicina laboratorial. 2007 ; 43( 5): 137.[citado 2024 nov. 05 ]
Texto na web
Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase (2005)
Ong, Thomas Prates ; Heidor, Renato ; Conti, Aline de; Dagli, Maria Lúcia Zaidan ; Moreno, Fernando Salvador
Source: Carcinogenesis. Unidades: FMVZ, FCF
Subjects: CARCINOGÊNESE ANIMAL, HEPATOPATIAS, QUÍMICA ORGÂNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ONG, Thomas Prates et al. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. Oxford: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. . Acesso em: 05 nov. 2024. , 2005
APA
Ong, T. P., Heidor, R., Conti, A. de, Dagli, M. L. Z., & Moreno, F. S. (2005). Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. Oxford: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo.
NLM
Ong TP, Heidor R, Conti A de, Dagli MLZ, Moreno FS. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. 2005 ; 1-43.[citado 2024 nov. 05 ]
Vancouver
Ong TP, Heidor R, Conti A de, Dagli MLZ, Moreno FS. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. 2005 ; 1-43.[citado 2024 nov. 05 ]
Artigo de periodico
Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis (1998)
Dagli, Maria Lúcia Zaidan ; Guerra, José Luiz; Sinhorini, Idercio Luiz; Wu, Ton-Sen; Rizzi, Maria Beatriz de Souza Lima; Penteado, Marilene de Vuono Camargo; Moreno, Fernando Salvador
Source: Pathology. Unidades: FMVZ, FCF
Subjects: CARCINOGÊNESE ANIMAL, CAROTENOIDES, HEPATOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAGLI, Maria Lúcia Zaidan et al. Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis. Pathology, v. 30, n. 3, p. 259-266, 1998Tradução . . Disponível em: https://doi.org/10.1080/00313029800169416. Acesso em: 05 nov. 2024.
APA
Dagli, M. L. Z., Guerra, J. L., Sinhorini, I. L., Wu, T. -S., Rizzi, M. B. de S. L., Penteado, M. de V. C., & Moreno, F. S. (1998). Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis. Pathology, 30( 3), 259-266. doi:10.1080/00313029800169416
NLM
Dagli MLZ, Guerra JL, Sinhorini IL, Wu T-S, Rizzi MB de SL, Penteado M de VC, Moreno FS. Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis [Internet]. Pathology. 1998 ; 30( 3): 259-266.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1080/00313029800169416
Vancouver
Dagli MLZ, Guerra JL, Sinhorini IL, Wu T-S, Rizzi MB de SL, Penteado M de VC, Moreno FS. Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis [Internet]. Pathology. 1998 ; 30( 3): 259-266.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1080/00313029800169416

USP Schools

ReP

Filtros

Authors

USP affiliated authors

Date published

Subjects

Source title

Indexado em

Non normalized affiliation