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Artigo de periodico
Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model (2019)
Pérez, Malena Martínez ; Martins, Larissa Marfori Sampaio; Dias, Murilo Solano; Pereira, Camila André; Leite, Jefferson Antonio; Gonçalves, Enrico Cerioni Spiropulos; Almeida, Paula Zaghetto de ; Freitas, Emanuelle Neivert de; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da ; Sartori, Daniela Carlos
Fonte: Immunology. Unidade: FMRP
Assuntos: DOENÇAS METABÓLICAS, INFLAMAÇÃO, OBESIDADE, IMUNOLOGIA, HOMEOSTASE, INTESTINOS
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PÉREZ, Malena Martínez et al. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, v. 156, n. 4, p. 339-355, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13028. Acesso em: 03 nov. 2024.
APA
Pérez, M. M., Martins, L. M. S., Dias, M. S., Pereira, C. A., Leite, J. A., Gonçalves, E. C. S., et al. (2019). Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, 156( 4), 339-355. doi:10.1111/imm.13028
NLM
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1111/imm.13028
Vancouver
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1111/imm.13028
Artigo de periodico
The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression (2018)
Amano, Mariane Tami ; Castoldi, Angela ; Andrade-Oliveira, Vinicius ; Latância, Marcela Teatin ; Terra, Fernanda Fernandes ; Correa-Costa, Matheus ; Breda, Cristiane Naffah Souza ; Felizardo, Raphael José Ferreira ; Pereira, Welbert Oliveira de ; Silva, Marina Burgos da ; Miyagi, Mariana Yasue Saito ; Aguiar, Cristhiane Favero ; Hiyane, Meire Ioshie ; Silva, João Santana da ; Moura, Ivan Cruz ; Câmara, Niels Olsen Saraiva
Fonte: International Immunopharmacology. Unidades: ICB, FMRP, FCF, BIOTECNOLOGIA
Assuntos: INSUFICIÊNCIA RENAL AGUDA, NEFROPATIAS, MACRÓFAGOS, IMUNOLOGIA
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AMANO, Mariane Tami et al. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, v. 64, p. 151-161, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.08.020. Acesso em: 03 nov. 2024.
APA
Amano, M. T., Castoldi, A., Andrade-Oliveira, V., Latância, M. T., Terra, F. F., Correa-Costa, M., et al. (2018). The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, 64, 151-161. doi:10.1016/j.intimp.2018.08.020
NLM
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Vancouver
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Artigo de periodico
T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses (2018)
Reynolds, C. J.; Suleyman, O. M.; Ortega-Prieto, A. M.; Skelton, J. K.; Bonnesoeur, P.; Blohm, A.; Carregaro, Vanessa ; Silva, João Santana da ; James, E. A.; Maillère, B.; Dorner, M.; Boyton, R. J.; Altmann, D. M.
Fonte: Scientific Reports. Unidade: FMRP
Assuntos: ZIKA VÍRUS, GENES DE RESPOSTA IMUNE, LINFÓCITOS, REAÇÃO CRUZADA
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REYNOLDS, C. J. et al. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses. Scientific Reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-18781-1. Acesso em: 03 nov. 2024.
APA
Reynolds, C. J., Suleyman, O. M., Ortega-Prieto, A. M., Skelton, J. K., Bonnesoeur, P., Blohm, A., et al. (2018). T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses. Scientific Reports, 8. doi:10.1038/s41598-017-18781-1
NLM
Reynolds CJ, Suleyman OM, Ortega-Prieto AM, Skelton JK, Bonnesoeur P, Blohm A, Carregaro V, Silva JS da, James EA, Maillère B, Dorner M, Boyton RJ, Altmann DM. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41598-017-18781-1
Vancouver
Reynolds CJ, Suleyman OM, Ortega-Prieto AM, Skelton JK, Bonnesoeur P, Blohm A, Carregaro V, Silva JS da, James EA, Maillère B, Dorner M, Boyton RJ, Altmann DM. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41598-017-18781-1
Artigo de periodico
The effects of physical therapy with neuromuscular electrical stimulation in patients with septic shock: study protocol for a randomized cross-over design (2018)
Lago, Alessandra Fabiane ; Oliveira, Anamaria Siriani de ; Souza, Hugo Celso Dutra de ; Silva, João Santana da ; Basile Filho, Aníbal; Gastaldi, Ada Clarice
Fonte: Medicine. Unidade: FMRP
Assuntos: BRONCOPATIAS, FIBROSE PULMONAR, EXERCÍCIO FÍSICO
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LAGO, Alessandra Fabiane et al. The effects of physical therapy with neuromuscular electrical stimulation in patients with septic shock: study protocol for a randomized cross-over design. Medicine, v. 97, n. 6, 2018Tradução . . Disponível em: https://doi.org/10.1097/md.0000000000009736. Acesso em: 03 nov. 2024.
APA
Lago, A. F., Oliveira, A. S. de, Souza, H. C. D. de, Silva, J. S. da, Basile Filho, A., & Gastaldi, A. C. (2018). The effects of physical therapy with neuromuscular electrical stimulation in patients with septic shock: study protocol for a randomized cross-over design. Medicine, 97( 6). doi:10.1097/md.0000000000009736
NLM
Lago AF, Oliveira AS de, Souza HCD de, Silva JS da, Basile Filho A, Gastaldi AC. The effects of physical therapy with neuromuscular electrical stimulation in patients with septic shock: study protocol for a randomized cross-over design [Internet]. Medicine. 2018 ; 97( 6):[citado 2024 nov. 03 ] Available from: https://doi.org/10.1097/md.0000000000009736
Vancouver
Lago AF, Oliveira AS de, Souza HCD de, Silva JS da, Basile Filho A, Gastaldi AC. The effects of physical therapy with neuromuscular electrical stimulation in patients with septic shock: study protocol for a randomized cross-over design [Internet]. Medicine. 2018 ; 97( 6):[citado 2024 nov. 03 ] Available from: https://doi.org/10.1097/md.0000000000009736
Artigo de periodico
Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence (2018)
Kannen, Vinicius; Sakita, Juliana Y.; Carneiro, Zumira A.; Bader, Michael; Alenina, Natalia; Teixeira, Regina R.; Oliveira, Enio C. de; Brunaldi, Mariângela O.; Gasparotto, Bianca; Sartori, Daniela Carlos; Fernandes, Cleverson R.; Silva, João Santana da; Andrade, Marcus V.; Silva Júnior, Wilson Araújo da; Uyemura, Sérgio Akira; Garcia, Sérgio Britto
Fonte: Digestive Diseases and Sciences. Unidades: FMRP, FCFRP
Assuntos: DOENÇA DE CHAGAS, NEURÔNIOS, DOENÇAS PARASITÁRIAS
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KANNEN, Vinicius et al. Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence. Digestive Diseases and Sciences, v. 63, n. 6, p. 1473-1484, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10620-018-5015-6. Acesso em: 03 nov. 2024.
APA
Kannen, V., Sakita, J. Y., Carneiro, Z. A., Bader, M., Alenina, N., Teixeira, R. R., et al. (2018). Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence. Digestive Diseases and Sciences, 63( 6), 1473-1484. doi:10.1007/s10620-018-5015-6
NLM
Kannen V, Sakita JY, Carneiro ZA, Bader M, Alenina N, Teixeira RR, Oliveira EC de, Brunaldi MO, Gasparotto B, Sartori DC, Fernandes CR, Silva JS da, Andrade MV, Silva Júnior WA da, Uyemura SA, Garcia SB. Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence [Internet]. Digestive Diseases and Sciences. 2018 ; 63( 6): 1473-1484.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1007/s10620-018-5015-6
Vancouver
Kannen V, Sakita JY, Carneiro ZA, Bader M, Alenina N, Teixeira RR, Oliveira EC de, Brunaldi MO, Gasparotto B, Sartori DC, Fernandes CR, Silva JS da, Andrade MV, Silva Júnior WA da, Uyemura SA, Garcia SB. Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence [Internet]. Digestive Diseases and Sciences. 2018 ; 63( 6): 1473-1484.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1007/s10620-018-5015-6
Artigo de periodico
The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing (2018)
Guimarães, Francielle Rodrigues; Sales-Campos, Helioswilton; Nardini, Viviani; Costa, Thiago Alvares da; Fonseca, Monique Thaís Costa; Rodrigues Júnior, Virmondes; Sorgi, Carlos Arterio; Silva, João Santana da; Chica, Javier Emílio Lazo; Faccioli, Lucia Helena; Cardoso, Cristina Ribeiro de Barros
Fonte: Clinical Immunology. Unidades: FCFRP, FMRP
Assuntos: LEUCOTRIENOS, INFLAMAÇÃO, PELE, BIOMARCADORES
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GUIMARÃES, Francielle Rodrigues et al. The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing. Clinical Immunology, v. 190, p. 74-83, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2017.08.022. Acesso em: 03 nov. 2024.
APA
Guimarães, F. R., Sales-Campos, H., Nardini, V., Costa, T. A. da, Fonseca, M. T. C., Rodrigues Júnior, V., et al. (2018). The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing. Clinical Immunology, 190, 74-83. doi:10.1016/j.clim.2017.08.022
NLM
Guimarães FR, Sales-Campos H, Nardini V, Costa TA da, Fonseca MTC, Rodrigues Júnior V, Sorgi CA, Silva JS da, Chica JEL, Faccioli LH, Cardoso CR de B. The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing [Internet]. Clinical Immunology. 2018 ; 190 74-83.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.clim.2017.08.022
Vancouver
Guimarães FR, Sales-Campos H, Nardini V, Costa TA da, Fonseca MTC, Rodrigues Júnior V, Sorgi CA, Silva JS da, Chica JEL, Faccioli LH, Cardoso CR de B. The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing [Internet]. Clinical Immunology. 2018 ; 190 74-83.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.clim.2017.08.022
Artigo de periodico
Diabetes mellitus and sepsis: A challenging association (2017)
Trevelin, Silvia C.; Carlos, Daniela; Beretta, Matteo; Silva, João Santana da; Cunha, Fernando de Queiroz
Fonte: Shock. Unidade: FMRP
Assuntos: DIABETES MELLITUS, SEPTICEMIA, INSULINA, SEPSE, GLICOSE
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ABNT
TREVELIN, Silvia C. et al. Diabetes mellitus and sepsis: A challenging association. Shock, v. 43, n. 7, p. 276-287, 2017Tradução . . Disponível em: https://doi.org/10.1097/SHK.0000000000000778. Acesso em: 03 nov. 2024.
APA
Trevelin, S. C., Carlos, D., Beretta, M., Silva, J. S. da, & Cunha, F. de Q. (2017). Diabetes mellitus and sepsis: A challenging association. Shock, 43( 7), 276-287. doi:10.1097/SHK.0000000000000778
NLM
Trevelin SC, Carlos D, Beretta M, Silva JS da, Cunha F de Q. Diabetes mellitus and sepsis: A challenging association [Internet]. Shock. 2017 ; 43( 7): 276-287.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1097/SHK.0000000000000778
Vancouver
Trevelin SC, Carlos D, Beretta M, Silva JS da, Cunha F de Q. Diabetes mellitus and sepsis: A challenging association [Internet]. Shock. 2017 ; 43( 7): 276-287.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1097/SHK.0000000000000778
Artigo de periodico
Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis (2017)
Souza, Maria C.; Fonseca, Denise Morais da ; Kanashiro, Alexandre; Benevides, Luciana; Medina, Tiago S.; Dias, Murilo S.; Andrade, Warrison A.; Bonfá, Giuliano; Silva, Marcondes A. B.; Gozzi, Aline; Borges, Marcos de Carvalho; Gazzinelli, Ricardo T.; Alves Filho, José Carlos Farias; Cunha, Fernando Queiróz; Silva, João Santana da
Fonte: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Assuntos: MODELOS ANIMAIS DE DOENÇAS, INTERFERONS, ÓXIDO NÍTRICO, SEPSE, TOXOPLASMOSE
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SOUZA, Maria C. et al. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, v. 7, 2017Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2017.00116. Acesso em: 03 nov. 2024.
APA
Souza, M. C., Fonseca, D. M. da, Kanashiro, A., Benevides, L., Medina, T. S., Dias, M. S., et al. (2017). Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, 7. doi:10.3389/fcimb.2017.00116
NLM
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Vancouver
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Artigo de periodico
Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection (2017)
Sacramento, Laís A.; Costa, Jéssica L. da; Lima, Mikhael H. F. de; Sampaio, Pedro A.; Almeida, Roque P.; Cunha, Fernando Queiróz; Silva, João Santana da; Carregaro, Vanessa
Fonte: Frontiers in Microbiology. Unidade: FMRP
Assuntos: LEISHMANIA INFANTUM, CÉLULAS DENDRÍTICAS, LEISHMANIOSE VISCERAL, NEUTRÓFILOS
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ABNT
SACRAMENTO, Laís A. et al. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.00262. Acesso em: 03 nov. 2024.
APA
Sacramento, L. A., Costa, J. L. da, Lima, M. H. F. de, Sampaio, P. A., Almeida, R. P., Cunha, F. Q., et al. (2017). Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, 8. doi:10.3389/fmicb.2017.00262
NLM
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Vancouver
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Artigo de periodico
Distinct roles of Th17 and Th1 cells in inflammatory responses associated with the presentation of paucibacillary leprosy and leprosy reactions (2017)
Santos, M. B.; Oliveira, D. T. de; Cazzaniga, R. A.; Varjão, C. S.; Santos, P. L. dos; Santos, M. L. B.; Correia, C. B.; Faria, D. R.; Simon, M. do V.; Silva, João Santana da
Fonte: Scandinavian Journal of Immunology. Unidade: FMRP
Assuntos: HANSENÍASE, DOENÇAS INFECCIOSAS, INFLAMAÇÃO, INTERLEUCINAS, MICROSCOPIA CONFOCAL, CITOMETRIA DE FLUXO
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ABNT
SANTOS, M. B. et al. Distinct roles of Th17 and Th1 cells in inflammatory responses associated with the presentation of paucibacillary leprosy and leprosy reactions. Scandinavian Journal of Immunology, v. 86, n. 1, p. 40-49, 2017Tradução . . Disponível em: https://doi.org/10.1111/sji.12558. Acesso em: 03 nov. 2024.
APA
Santos, M. B., Oliveira, D. T. de, Cazzaniga, R. A., Varjão, C. S., Santos, P. L. dos, Santos, M. L. B., et al. (2017). Distinct roles of Th17 and Th1 cells in inflammatory responses associated with the presentation of paucibacillary leprosy and leprosy reactions. Scandinavian Journal of Immunology, 86( 1), 40-49. doi:10.1111/sji.12558
NLM
Santos MB, Oliveira DT de, Cazzaniga RA, Varjão CS, Santos PL dos, Santos MLB, Correia CB, Faria DR, Simon M do V, Silva JS da. Distinct roles of Th17 and Th1 cells in inflammatory responses associated with the presentation of paucibacillary leprosy and leprosy reactions [Internet]. Scandinavian Journal of Immunology. 2017 ; 86( 1): 40-49.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1111/sji.12558
Vancouver
Santos MB, Oliveira DT de, Cazzaniga RA, Varjão CS, Santos PL dos, Santos MLB, Correia CB, Faria DR, Simon M do V, Silva JS da. Distinct roles of Th17 and Th1 cells in inflammatory responses associated with the presentation of paucibacillary leprosy and leprosy reactions [Internet]. Scandinavian Journal of Immunology. 2017 ; 86( 1): 40-49.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1111/sji.12558
Artigo de periodico
Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis (2017)
Dias-Teixeira, Karina Luiza; Calegari-Silva, Teresa C.; Medina, Jorge M.; Vivarini, Áislan C.; Cavalcanti, Átila; Teteo, Nataly; Santana, Alynne Karen M.; Real, Fernando; Gomes, Ciro M.; Pereira, Renata Meirelles Santos; Fasel, Nicolas; Silva, João Santana da; Aktas, Bertal H.; Lopes, Ulisses G.
Fonte: Scientific Reports. Unidade: FMRP
Assuntos: LEISHMANIA, MACRÓFAGOS, RETÍCULO ENDOPLASMÁTICO
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ABNT
DIAS-TEIXEIRA, Karina Luiza et al. Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis. Scientific Reports, v. 7, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-17252-x. Acesso em: 03 nov. 2024.
APA
Dias-Teixeira, K. L., Calegari-Silva, T. C., Medina, J. M., Vivarini, Á. C., Cavalcanti, Á., Teteo, N., et al. (2017). Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis. Scientific Reports, 7( 1). doi:10.1038/s41598-017-17252-x
NLM
Dias-Teixeira KL, Calegari-Silva TC, Medina JM, Vivarini ÁC, Cavalcanti Á, Teteo N, Santana AKM, Real F, Gomes CM, Pereira RMS, Fasel N, Silva JS da, Aktas BH, Lopes UG. Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41598-017-17252-x
Vancouver
Dias-Teixeira KL, Calegari-Silva TC, Medina JM, Vivarini ÁC, Cavalcanti Á, Teteo N, Santana AKM, Real F, Gomes CM, Pereira RMS, Fasel N, Silva JS da, Aktas BH, Lopes UG. Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41598-017-17252-x
Artigo de periodico
Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity (2017)
Hartmann, Ana Paula; Carvalho, Marcelo Rodrigues de; Bernardes, Lilian Sibelle Campos; Moraes, Milena Hoehr de; Melo, Eduardo Borges de; Lopes, Carla Duque; Steindel, Mario; Silva, João Santana da; Carvalho, Ivone
Fonte: European Journal of Medicinal Chemistry. Unidades: FCFRP, FMRP
Assuntos: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI, INIBIDORES DE ENZIMAS
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ABNT
HARTMANN, Ana Paula et al. Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity. European Journal of Medicinal Chemistry, v. 140, p. 187-199, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejmech.2017.08.064. Acesso em: 03 nov. 2024.
APA
Hartmann, A. P., Carvalho, M. R. de, Bernardes, L. S. C., Moraes, M. H. de, Melo, E. B. de, Lopes, C. D., et al. (2017). Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity. European Journal of Medicinal Chemistry, 140, 187-199. doi:10.1016/j.ejmech.2017.08.064
NLM
Hartmann AP, Carvalho MR de, Bernardes LSC, Moraes MH de, Melo EB de, Lopes CD, Steindel M, Silva JS da, Carvalho I. Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity [Internet]. European Journal of Medicinal Chemistry. 2017 ; 140 187-199.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.ejmech.2017.08.064
Vancouver
Hartmann AP, Carvalho MR de, Bernardes LSC, Moraes MH de, Melo EB de, Lopes CD, Steindel M, Silva JS da, Carvalho I. Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity [Internet]. European Journal of Medicinal Chemistry. 2017 ; 140 187-199.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.ejmech.2017.08.064
Artigo de periodico
Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells (2016)
Carregaro, Vanessa; Napimoga, Marcelo H; Peres, Raphael S; Benevides, Luciana; Sacramento, Laís Amorim; Pinto, Larissa G; Grespan, Renata; Cunha, Thiago Mattar; Silva, João Santana da; Cunha, Fernando de Queiroz
Fonte: Mediators of Inflammation. Unidade: FMRP
Assuntos: ARTRITE, PROSTAGLANDINAS, FATORES DE TRANSCRIÇÃO, RECEPTORES, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARREGARO, Vanessa et al. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, v. 2016, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/9626427. Acesso em: 03 nov. 2024.
APA
Carregaro, V., Napimoga, M. H., Peres, R. S., Benevides, L., Sacramento, L. A., Pinto, L. G., et al. (2016). Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, 2016. doi:10.1155/2016/9626427
NLM
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 nov. 03 ] Available from: https://doi.org/10.1155/2016/9626427
Vancouver
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 nov. 03 ] Available from: https://doi.org/10.1155/2016/9626427
Artigo de periodico
Histopathological correlates of global and segmental left ventricular systolic dysfunction in experimental chronic chagas cardiomyopathy (2016)
Oliveira, Luciano Fonseca Lemos de; Romano, Minna Moreira Dias; Carvalho, Eduardo Elias Vieira de; Cabeza, Jorge Mejia; Salgado, Hélio César; Fazan Júnior, Rubens; Costa, Renata Sesti; Silva, João Santana da; Higuchi, Maria de Lourdes; Maciel, Benedito Carlos; Cunha Neto, Edécio; Marin Neto, José Antonio; Simões, Marcus Vinicius
Fonte: Journal of the American Heart Association. Unidades: FMRP, FM
Assuntos: INSUFICIÊNCIA CARDÍACA, DOENÇA DE CHAGAS, ECOCARDIOGRAFIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Luciano Fonseca Lemos de et al. Histopathological correlates of global and segmental left ventricular systolic dysfunction in experimental chronic chagas cardiomyopathy. Journal of the American Heart Association, v. 5, n. 1, p. e002786-1 - e002786-11, 2016Tradução . . Disponível em: https://doi.org/10.1161/jaha.115.002786. Acesso em: 03 nov. 2024.
APA
Oliveira, L. F. L. de, Romano, M. M. D., Carvalho, E. E. V. de, Cabeza, J. M., Salgado, H. C., Fazan Júnior, R., et al. (2016). Histopathological correlates of global and segmental left ventricular systolic dysfunction in experimental chronic chagas cardiomyopathy. Journal of the American Heart Association, 5( 1), e002786-1 - e002786-11. doi:10.1161/jaha.115.002786
NLM
Oliveira LFL de, Romano MMD, Carvalho EEV de, Cabeza JM, Salgado HC, Fazan Júnior R, Costa RS, Silva JS da, Higuchi M de L, Maciel BC, Cunha Neto E, Marin Neto JA, Simões MV. Histopathological correlates of global and segmental left ventricular systolic dysfunction in experimental chronic chagas cardiomyopathy [Internet]. Journal of the American Heart Association. 2016 ; 5( 1): e002786-1 - e002786-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1161/jaha.115.002786
Vancouver
Oliveira LFL de, Romano MMD, Carvalho EEV de, Cabeza JM, Salgado HC, Fazan Júnior R, Costa RS, Silva JS da, Higuchi M de L, Maciel BC, Cunha Neto E, Marin Neto JA, Simões MV. Histopathological correlates of global and segmental left ventricular systolic dysfunction in experimental chronic chagas cardiomyopathy [Internet]. Journal of the American Heart Association. 2016 ; 5( 1): e002786-1 - e002786-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1161/jaha.115.002786
Artigo de periodico
Unveiling the role of the integrated endoplasmic reticulum stress response in leishmania infection: future perspectives (2016)
Dias-Teixeira, K. L.; Pereira, R. M.; Silva, João Santana da; Fasel, N.; Aktas, B. H.; Lopes, U. G.
Fonte: Frontiers in Immunology. Unidade: FMRP
Assuntos: LEISHMANIA, RETÍCULO ENDOPLASMÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DIAS-TEIXEIRA, K. L. et al. Unveiling the role of the integrated endoplasmic reticulum stress response in leishmania infection: future perspectives. Frontiers in Immunology, v. 7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00283. Acesso em: 03 nov. 2024.
APA
Dias-Teixeira, K. L., Pereira, R. M., Silva, J. S. da, Fasel, N., Aktas, B. H., & Lopes, U. G. (2016). Unveiling the role of the integrated endoplasmic reticulum stress response in leishmania infection: future perspectives. Frontiers in Immunology, 7. doi:10.3389/fimmu.2016.00283
NLM
Dias-Teixeira KL, Pereira RM, Silva JS da, Fasel N, Aktas BH, Lopes UG. Unveiling the role of the integrated endoplasmic reticulum stress response in leishmania infection: future perspectives [Internet]. Frontiers in Immunology. 2016 ;7[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2016.00283
Vancouver
Dias-Teixeira KL, Pereira RM, Silva JS da, Fasel N, Aktas BH, Lopes UG. Unveiling the role of the integrated endoplasmic reticulum stress response in leishmania infection: future perspectives [Internet]. Frontiers in Immunology. 2016 ;7[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2016.00283
Artigo de periodico
Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs (2016)
Costa, Guilherme de Paula; Lopes, Laís Roquete; Silva, Maria Cláudia da; Horta, Aline Luciano; Pontes, Washington Martins; Milanezi, Cristiane Maria; Guedes, Paulo Marcos da Mata; Lima, Wanderson Geraldo de; Schulz, Richard; Silva, João Santana da; Talvani, Andre
Fonte: Mediators of Inflammation. Unidade: FMRP
Assuntos: TRYPANOSOMA CRUZI, QUIMIOCINAS, RECEPTORES, SISTEMA CARDIOVASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Guilherme de Paula et al. Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs. Mediators of Inflammation, v. 2016, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/3694714. Acesso em: 03 nov. 2024.
APA
Costa, G. de P., Lopes, L. R., Silva, M. C. da, Horta, A. L., Pontes, W. M., Milanezi, C. M., et al. (2016). Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs. Mediators of Inflammation, 2016. doi:10.1155/2016/3694714
NLM
Costa G de P, Lopes LR, Silva MC da, Horta AL, Pontes WM, Milanezi CM, Guedes PM da M, Lima WG de, Schulz R, Silva JS da, Talvani A. Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 nov. 03 ] Available from: https://doi.org/10.1155/2016/3694714
Vancouver
Costa G de P, Lopes LR, Silva MC da, Horta AL, Pontes WM, Milanezi CM, Guedes PM da M, Lima WG de, Schulz R, Silva JS da, Talvani A. Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 nov. 03 ] Available from: https://doi.org/10.1155/2016/3694714
Artigo de periodico
Molecular characterization of trypanosoma cruzi Tc8.2 gene indicates two differential locations for the encoded protein in epimastigote and trypomastigote forms (2015)
Kian, Danielle; Lancheros, César Armando Contreras; Damiani, Igor Alexandre Campos; Fernandes, Tamiris Zanforlin Olmos; Pinge-Filho, Phileno; Santos, Márcia Regina Machado dos; Silveira, José Franco da; Nakamura, Celso Vataru; Silva, João Santana da; Yamada-Ogatta, Sueli Fumie; Yamauchi, Lucy Megumi
Fonte: Korean Journal of Parasitology. Unidade: FMRP
Assuntos: TRYPANOSOMA CRUZI, DOENÇAS DE CHAGAS, EXPRESSÃO GÊNICA, GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KIAN, Danielle et al. Molecular characterization of trypanosoma cruzi Tc8.2 gene indicates two differential locations for the encoded protein in epimastigote and trypomastigote forms. Korean Journal of Parasitology, v. 53, n. 4, p. 483-488, 2015Tradução . . Disponível em: https://doi.org/10.3347/kjp.2015.53.4.483. Acesso em: 03 nov. 2024.
APA
Kian, D., Lancheros, C. A. C., Damiani, I. A. C., Fernandes, T. Z. O., Pinge-Filho, P., Santos, M. R. M. dos, et al. (2015). Molecular characterization of trypanosoma cruzi Tc8.2 gene indicates two differential locations for the encoded protein in epimastigote and trypomastigote forms. Korean Journal of Parasitology, 53( 4), 483-488. doi:10.3347/kjp.2015.53.4.483
NLM
Kian D, Lancheros CAC, Damiani IAC, Fernandes TZO, Pinge-Filho P, Santos MRM dos, Silveira JF da, Nakamura CV, Silva JS da, Yamada-Ogatta SF, Yamauchi LM. Molecular characterization of trypanosoma cruzi Tc8.2 gene indicates two differential locations for the encoded protein in epimastigote and trypomastigote forms [Internet]. Korean Journal of Parasitology. 2015 ; 53( 4): 483-488.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3347/kjp.2015.53.4.483
Vancouver
Kian D, Lancheros CAC, Damiani IAC, Fernandes TZO, Pinge-Filho P, Santos MRM dos, Silveira JF da, Nakamura CV, Silva JS da, Yamada-Ogatta SF, Yamauchi LM. Molecular characterization of trypanosoma cruzi Tc8.2 gene indicates two differential locations for the encoded protein in epimastigote and trypomastigote forms [Internet]. Korean Journal of Parasitology. 2015 ; 53( 4): 483-488.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3347/kjp.2015.53.4.483
Artigo de periodico
Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection (2014)
Gutierrez, Fredy R. S; Pavanelli, Wander R; Medina, Tiago S; Silva, Grace K; Mariano, Flávia S; Guedes, Paulo M. M.; Mineo, Tiago W. P.; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Fonte: Microbes and Infection. Unidade: FMRP
Assuntos: TRYPANOSOMA CRUZI, OXIGENASE, LINFÓCITOS T
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUTIERREZ, Fredy R. S et al. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 1, p. 28-39, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2013.10.007. Acesso em: 03 nov. 2024.
APA
Gutierrez, F. R. S., Pavanelli, W. R., Medina, T. S., Silva, G. K., Mariano, F. S., Guedes, P. M. M., et al. (2014). Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, 16( 1), 28-39. doi:10.1016/j.micinf.2013.10.007
NLM
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Vancouver
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Artigo de periodico
Role of dystrophin in acute Trypanosoma cruzi infection (2014)
Malvestio, Lygia M; Celes, Mara R. N; Milanezi, Cristiane Maria; Silva, João Santana da; Jelicks, Linda A; Tanowitz, Hebert B; Rossi, Marcos Antonio; Prado, Cibele Maria
Fonte: Microbes and Infection. Unidade: FMRP
Assuntos: DOENÇA DE CHAGAS, INFLAMAÇÃO (METABOLISMO), TRYPANOSOMA CRUZI, MIÓCITOS DE MÚSCULO LISO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MALVESTIO, Lygia M et al. Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 9, p. 768-777, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.07.010. Acesso em: 03 nov. 2024.
APA
Malvestio, L. M., Celes, M. R. N., Milanezi, C. M., Silva, J. S. da, Jelicks, L. A., Tanowitz, H. B., et al. (2014). Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, 16( 9), 768-777. doi:10.1016/j.micinf.2014.07.010
NLM
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Vancouver
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Artigo de periodico
Association of matrix metalloproteinase inducer (EMMPRIN) with the expression of matrix metalloproteinases-1, -2 and -9 during periapical lesion development (2014)
Sousa, Natália Guimarães Kalatzis; Cardoso, Cristina Ribeiro de Barros; Silva, João Santana da; Kuga, Milton Carlos; Tanomaru Filho, Mário; Faria, Gisele
Fonte: Archives of Oral Biology. Unidades: FCFRP, FMRP
Assuntos: ENDODONTIA, LESÕES PERIAPICAIS, METALOPROTEINASES, MATRIZ EXTRACELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUSA, Natália Guimarães Kalatzis et al. Association of matrix metalloproteinase inducer (EMMPRIN) with the expression of matrix metalloproteinases-1, -2 and -9 during periapical lesion development. Archives of Oral Biology, v. 59, n. 9, p. 944-953, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.archoralbio.2014.05.021. Acesso em: 03 nov. 2024.
APA
Sousa, N. G. K., Cardoso, C. R. de B., Silva, J. S. da, Kuga, M. C., Tanomaru Filho, M., & Faria, G. (2014). Association of matrix metalloproteinase inducer (EMMPRIN) with the expression of matrix metalloproteinases-1, -2 and -9 during periapical lesion development. Archives of Oral Biology, 59( 9), 944-953. doi:10.1016/j.archoralbio.2014.05.021
NLM
Sousa NGK, Cardoso CR de B, Silva JS da, Kuga MC, Tanomaru Filho M, Faria G. Association of matrix metalloproteinase inducer (EMMPRIN) with the expression of matrix metalloproteinases-1, -2 and -9 during periapical lesion development [Internet]. Archives of Oral Biology. 2014 ; 59( 9): 944-953.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.archoralbio.2014.05.021
Vancouver
Sousa NGK, Cardoso CR de B, Silva JS da, Kuga MC, Tanomaru Filho M, Faria G. Association of matrix metalloproteinase inducer (EMMPRIN) with the expression of matrix metalloproteinases-1, -2 and -9 during periapical lesion development [Internet]. Archives of Oral Biology. 2014 ; 59( 9): 944-953.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.archoralbio.2014.05.021

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