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  • Source: Frontiers in Bioscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      NEWSHOLME, Philip et al. Amino acids and diabetes: implications for endocrine, metabolic and immune function. Frontiers in Bioscience, v. 16, n. 1, p. 315-339, 2011Tradução . . Disponível em: https://doi.org/10.2741/3690. Acesso em: 19 nov. 2024.
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      Newsholme, P., Abdulkader, F. R. de M., Rebelato, E., Romanatto, T., Pinheiro, C. H. da J., Vitzel, K. F., et al. (2011). Amino acids and diabetes: implications for endocrine, metabolic and immune function. Frontiers in Bioscience, 16( 1), 315-339. doi:10.2741/3690
    • NLM

      Newsholme P, Abdulkader FR de M, Rebelato E, Romanatto T, Pinheiro CH da J, Vitzel KF, Silva EP, Bazotte RB, Araújo Filho JP de, Curi R, Gorjão R, Pithon-Curi TC. Amino acids and diabetes: implications for endocrine, metabolic and immune function [Internet]. Frontiers in Bioscience. 2011 ; 16( 1): 315-339.[citado 2024 nov. 19 ] Available from: https://doi.org/10.2741/3690
    • Vancouver

      Newsholme P, Abdulkader FR de M, Rebelato E, Romanatto T, Pinheiro CH da J, Vitzel KF, Silva EP, Bazotte RB, Araújo Filho JP de, Curi R, Gorjão R, Pithon-Curi TC. Amino acids and diabetes: implications for endocrine, metabolic and immune function [Internet]. Frontiers in Bioscience. 2011 ; 16( 1): 315-339.[citado 2024 nov. 19 ] Available from: https://doi.org/10.2741/3690
  • Source: Acta Physiologica. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEON, E. B. de et al. Neuromuscular electrical stimulation improves GLUT-4 and morphological characteristics of skeletal muscle in rats with heart failure. Acta Physiologica, v. 201, n. 2, p. 265-273, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1748-1716.2010.02176.x. Acesso em: 19 nov. 2024.
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      Leon, E. B. de, Bortoluzzi, A., Rucatti, A., Nunes, R. B., Saur, L., Rodrigues, M., et al. (2011). Neuromuscular electrical stimulation improves GLUT-4 and morphological characteristics of skeletal muscle in rats with heart failure. Acta Physiologica, 201( 2), 265-273. doi:10.1111/j.1748-1716.2010.02176.x
    • NLM

      Leon EB de, Bortoluzzi A, Rucatti A, Nunes RB, Saur L, Rodrigues M, Oliveira U, Wagner ABTA, Xavier LL, Machado UF, Schaan BD, Dall'Ago P. Neuromuscular electrical stimulation improves GLUT-4 and morphological characteristics of skeletal muscle in rats with heart failure [Internet]. Acta Physiologica. 2011 ; 201( 2): 265-273.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/j.1748-1716.2010.02176.x
    • Vancouver

      Leon EB de, Bortoluzzi A, Rucatti A, Nunes RB, Saur L, Rodrigues M, Oliveira U, Wagner ABTA, Xavier LL, Machado UF, Schaan BD, Dall'Ago P. Neuromuscular electrical stimulation improves GLUT-4 and morphological characteristics of skeletal muscle in rats with heart failure [Internet]. Acta Physiologica. 2011 ; 201( 2): 265-273.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/j.1748-1716.2010.02176.x
  • Source: Pancreas. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      VALLE, Maíra Mello Rezende et al. Alterations of NADPH oxidase activity in rat pancreatic islets induced by a high-fat diet. Pancreas, v. 40, n. 3, p. 390-395, 2011Tradução . . Acesso em: 19 nov. 2024.
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      Valle, M. M. R., Graciano, M. F. R., Oliveira, E. R. L. de, Camporez, J. P. G., Akamine, E. H., Carvalho, C. R. de O., et al. (2011). Alterations of NADPH oxidase activity in rat pancreatic islets induced by a high-fat diet. Pancreas, 40( 3), 390-395.
    • NLM

      Valle MMR, Graciano MFR, Oliveira ERL de, Camporez JPG, Akamine EH, Carvalho CR de O, Curi R, Carpinelli AR. Alterations of NADPH oxidase activity in rat pancreatic islets induced by a high-fat diet. Pancreas. 2011 ; 40( 3): 390-395.[citado 2024 nov. 19 ]
    • Vancouver

      Valle MMR, Graciano MFR, Oliveira ERL de, Camporez JPG, Akamine EH, Carvalho CR de O, Curi R, Carpinelli AR. Alterations of NADPH oxidase activity in rat pancreatic islets induced by a high-fat diet. Pancreas. 2011 ; 40( 3): 390-395.[citado 2024 nov. 19 ]
  • Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BROMATI, Carla Rodrigues et al. UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 300, n. 1, p. R92-R100, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00169.2010. Acesso em: 19 nov. 2024.
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      Bromati, C. R., Santos, C. de L., Yamanaka, T. S., Nogueira, T. C. de A., Leonelli, M., Caperuto, L. C., et al. (2011). UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 300( 1), R92-R100. doi:10.1152/ajpregu.00169.2010
    • NLM

      Bromati CR, Santos C de L, Yamanaka TS, Nogueira TC de A, Leonelli M, Caperuto LC, Gorjão R, Leite AR, Anhê GF, Bordin S. UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 1): R92-R100.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00169.2010
    • Vancouver

      Bromati CR, Santos C de L, Yamanaka TS, Nogueira TC de A, Leonelli M, Caperuto LC, Gorjão R, Leite AR, Anhê GF, Bordin S. UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 1): R92-R100.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00169.2010
  • Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      TAKAKURA, Ana Carolina et al. Ventrolateral medulla mechanisms involved in cardiorespiratory responses to central chemoreceptor activation in rats. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 300, n. 2, p. R501-R510, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00220.2010. Acesso em: 19 nov. 2024.
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      Takakura, A. C., Colombari, E., Menani, J. V., & Moreira, T. dos S. (2011). Ventrolateral medulla mechanisms involved in cardiorespiratory responses to central chemoreceptor activation in rats. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 300( 2), R501-R510. doi:10.1152/ajpregu.00220.2010
    • NLM

      Takakura AC, Colombari E, Menani JV, Moreira T dos S. Ventrolateral medulla mechanisms involved in cardiorespiratory responses to central chemoreceptor activation in rats [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 2): R501-R510.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00220.2010
    • Vancouver

      Takakura AC, Colombari E, Menani JV, Moreira T dos S. Ventrolateral medulla mechanisms involved in cardiorespiratory responses to central chemoreceptor activation in rats [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 2): R501-R510.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00220.2010
  • Source: Nutrition. Unidade: ICB

    Assunto: FISIOLOGIA

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      WAYHS, Mônica Lisboa Chang et al. Transepithelial transport of glucose and mRNA of glucose transportes in the small intestine of rats with iron-deficiency anemia. Nutrition, v. 27, n. 3, p. 111-115, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.nut.2010.07.002. Acesso em: 19 nov. 2024.
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      Wayhs, M. L. C., Morais, M. B. de, Machado, U. F., Nassar, S. M., Fagundes Neto, U., & Amâncio, O. M. S. (2011). Transepithelial transport of glucose and mRNA of glucose transportes in the small intestine of rats with iron-deficiency anemia. Nutrition, 27( 3), 111-115. doi:10.1016/j.nut.2010.07.002
    • NLM

      Wayhs MLC, Morais MB de, Machado UF, Nassar SM, Fagundes Neto U, Amâncio OMS. Transepithelial transport of glucose and mRNA of glucose transportes in the small intestine of rats with iron-deficiency anemia [Internet]. Nutrition. 2011 ; 27( 3): 111-115.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.nut.2010.07.002
    • Vancouver

      Wayhs MLC, Morais MB de, Machado UF, Nassar SM, Fagundes Neto U, Amâncio OMS. Transepithelial transport of glucose and mRNA of glucose transportes in the small intestine of rats with iron-deficiency anemia [Internet]. Nutrition. 2011 ; 27( 3): 111-115.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.nut.2010.07.002
  • Source: Journal of Physiology. Unidades: ICB, FMRP

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 19 nov. 2024.
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      Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
    • NLM

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
    • Vancouver

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
  • Source: International Journal of Developmental Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEONELLI, Mauro e MARTINS, Daniel de Oliveira e BRITTO, Luiz Roberto Giorgetti de. TRPV1 receptors modulate retinal development. International Journal of Developmental Neuroscience, v. 29, n. 4, p. 405-413, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.ijdevneu.2011.03.002. Acesso em: 19 nov. 2024.
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      Leonelli, M., Martins, D. de O., & Britto, L. R. G. de. (2011). TRPV1 receptors modulate retinal development. International Journal of Developmental Neuroscience, 29( 4), 405-413. doi:10.1016/j.ijdevneu.2011.03.002
    • NLM

      Leonelli M, Martins D de O, Britto LRG de. TRPV1 receptors modulate retinal development [Internet]. International Journal of Developmental Neuroscience. 2011 ; 29( 4): 405-413.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ijdevneu.2011.03.002
    • Vancouver

      Leonelli M, Martins D de O, Britto LRG de. TRPV1 receptors modulate retinal development [Internet]. International Journal of Developmental Neuroscience. 2011 ; 29( 4): 405-413.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ijdevneu.2011.03.002
  • Source: Growth Hormone & IGF Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      ROMERO, Frederico Gerlinger et al. Chronic supplementation of beta-hydroxy-beta methylbutyrate (HMβ) increases the activity of the GH/IGF-I axis and induces hyperinsulinemia in rats. Growth Hormone & IGF Research, v. 21, n. 2, p. 57-62, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.ghir.2010.12.006. Acesso em: 19 nov. 2024.
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      Romero, F. G., Ferreira, L. G., Giannocco, G., & Nunes, M. T. (2011). Chronic supplementation of beta-hydroxy-beta methylbutyrate (HMβ) increases the activity of the GH/IGF-I axis and induces hyperinsulinemia in rats. Growth Hormone & IGF Research, 21( 2), 57-62. doi:10.1016/j.ghir.2010.12.006
    • NLM

      Romero FG, Ferreira LG, Giannocco G, Nunes MT. Chronic supplementation of beta-hydroxy-beta methylbutyrate (HMβ) increases the activity of the GH/IGF-I axis and induces hyperinsulinemia in rats [Internet]. Growth Hormone & IGF Research. 2011 ; 21( 2): 57-62.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ghir.2010.12.006
    • Vancouver

      Romero FG, Ferreira LG, Giannocco G, Nunes MT. Chronic supplementation of beta-hydroxy-beta methylbutyrate (HMβ) increases the activity of the GH/IGF-I axis and induces hyperinsulinemia in rats [Internet]. Growth Hormone & IGF Research. 2011 ; 21( 2): 57-62.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ghir.2010.12.006
  • Source: The Journal of Membrane Biology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SILVA, Olívia Beloto da e MACHADO, Ubiratan Fabres e SOUZA, Maria Oliveira de. Glucose-induced regulation of NHEs activity and SGLTs expression involves the PKA signaling pathway. The Journal of Membrane Biology, v. 239, n. 3, p. 157-165, 2011Tradução . . Disponível em: https://doi.org/10.1007/s00232-010-9334-6. Acesso em: 19 nov. 2024.
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      Silva, O. B. da, Machado, U. F., & Souza, M. O. de. (2011). Glucose-induced regulation of NHEs activity and SGLTs expression involves the PKA signaling pathway. The Journal of Membrane Biology, 239( 3), 157-165. doi:10.1007/s00232-010-9334-6
    • NLM

      Silva OB da, Machado UF, Souza MO de. Glucose-induced regulation of NHEs activity and SGLTs expression involves the PKA signaling pathway [Internet]. The Journal of Membrane Biology. 2011 ; 239( 3): 157-165.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00232-010-9334-6
    • Vancouver

      Silva OB da, Machado UF, Souza MO de. Glucose-induced regulation of NHEs activity and SGLTs expression involves the PKA signaling pathway [Internet]. The Journal of Membrane Biology. 2011 ; 239( 3): 157-165.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00232-010-9334-6
  • Source: Chronobiology International. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      GARCIA, Rodrigo Antonio Peliciari et al. Expression of circadian clock and melatonin receptors within cultured rat cardiomyocytes. Chronobiology International, v. 28, n. 1, p. 21-30, 2011Tradução . . Disponível em: https://doi.org/10.3109/07420528.2010.525675. Acesso em: 19 nov. 2024.
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      Garcia, R. A. P., Zanquetta, M. M., Silva, J. A. da, Gomes, D. A., Barreto-Chaves, M. L. M., & Cipolla-Neto, J. (2011). Expression of circadian clock and melatonin receptors within cultured rat cardiomyocytes. Chronobiology International, 28( 1), 21-30. doi:10.3109/07420528.2010.525675
    • NLM

      Garcia RAP, Zanquetta MM, Silva JA da, Gomes DA, Barreto-Chaves MLM, Cipolla-Neto J. Expression of circadian clock and melatonin receptors within cultured rat cardiomyocytes [Internet]. Chronobiology International. 2011 ; 28( 1): 21-30.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3109/07420528.2010.525675
    • Vancouver

      Garcia RAP, Zanquetta MM, Silva JA da, Gomes DA, Barreto-Chaves MLM, Cipolla-Neto J. Expression of circadian clock and melatonin receptors within cultured rat cardiomyocytes [Internet]. Chronobiology International. 2011 ; 28( 1): 21-30.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3109/07420528.2010.525675
  • Source: Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      NOGUEIRA, Tatiane Cristina de Araújo et al. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response. Endocrinology, v. 152, n. 4, p. 1253-1263, 2011Tradução . . Disponível em: https://doi.org/10.1210/en.2010-1088. Acesso em: 19 nov. 2024.
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      Nogueira, T. C. de A., Santos, C. de L., Jesus, D. S. de, Taneda, M., Rodrigues, S. C., Amaral, F. G. do, et al. (2011). Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response. Endocrinology, 152( 4), 1253-1263. doi:10.1210/en.2010-1088
    • NLM

      Nogueira TC de A, Santos C de L, Jesus DS de, Taneda M, Rodrigues SC, Amaral FG do, Lopes AMS, Cipolla Neto J, Bordin S, Anhê GF. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response [Internet]. Endocrinology. 2011 ; 152( 4): 1253-1263.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1210/en.2010-1088
    • Vancouver

      Nogueira TC de A, Santos C de L, Jesus DS de, Taneda M, Rodrigues SC, Amaral FG do, Lopes AMS, Cipolla Neto J, Bordin S, Anhê GF. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response [Internet]. Endocrinology. 2011 ; 152( 4): 1253-1263.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1210/en.2010-1088
  • Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BURGI, Katia et al. Tyrosine hydroxylase immunoreactivity as indicator of sympathetic activity: simultaneous evaluation in different tissues of hypertensive rats. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 300, n. 2, p. R264-R271, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00687.2009. Acesso em: 19 nov. 2024.
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      Burgi, K., Cavalleri, M. T., Alves, A. da S., Britto, L. R. G. de, Antunes, V. R., & Michelini, L. C. (2011). Tyrosine hydroxylase immunoreactivity as indicator of sympathetic activity: simultaneous evaluation in different tissues of hypertensive rats. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 300( 2), R264-R271. doi:10.1152/ajpregu.00687.2009
    • NLM

      Burgi K, Cavalleri MT, Alves A da S, Britto LRG de, Antunes VR, Michelini LC. Tyrosine hydroxylase immunoreactivity as indicator of sympathetic activity: simultaneous evaluation in different tissues of hypertensive rats [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 2): R264-R271.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00687.2009
    • Vancouver

      Burgi K, Cavalleri MT, Alves A da S, Britto LRG de, Antunes VR, Michelini LC. Tyrosine hydroxylase immunoreactivity as indicator of sympathetic activity: simultaneous evaluation in different tissues of hypertensive rats [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 2): R264-R271.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00687.2009
  • Source: Cell Biochemistry Biophysics. Unidade: ICB

    Assunto: FISIOLOGIA

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      SALGADO, Thais Martins de Lima et al. Molecular mechanisms by which saturated fatty acids modulate TNF-α expression in mouse macrophage lineage. Cell Biochemistry Biophysics, v. 59, n. 2, p. 89-97, 2011Tradução . . Disponível em: https://doi.org/10.1210/en.2010-1088. Acesso em: 19 nov. 2024.
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      Salgado, T. M. de L., Loureiro, T. C. A., Nascimento, C. S. do, Nunes, M. T., & Curi, R. (2011). Molecular mechanisms by which saturated fatty acids modulate TNF-α expression in mouse macrophage lineage. Cell Biochemistry Biophysics, 59( 2), 89-97. doi:10.1210/en.2010-1088
    • NLM

      Salgado TM de L, Loureiro TCA, Nascimento CS do, Nunes MT, Curi R. Molecular mechanisms by which saturated fatty acids modulate TNF-α expression in mouse macrophage lineage [Internet]. Cell Biochemistry Biophysics. 2011 ; 59( 2): 89-97.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1210/en.2010-1088
    • Vancouver

      Salgado TM de L, Loureiro TCA, Nascimento CS do, Nunes MT, Curi R. Molecular mechanisms by which saturated fatty acids modulate TNF-α expression in mouse macrophage lineage [Internet]. Cell Biochemistry Biophysics. 2011 ; 59( 2): 89-97.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1210/en.2010-1088
  • Source: Cell Biochemistry and Function. Unidade: ICB

    Assunto: FISIOLOGIA

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      POLETTO, Ana Cláudia et al. Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue. Cell Biochemistry and Function, v. 28, n. 2, p. 114-121, 2010Tradução . . Disponível em: https://doi.org/10.1002/cbf.1628. Acesso em: 19 nov. 2024.
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      Poletto, A. C., Anhê, G. F., Eichler, P., Takahashi, H., Furuya, D. T., Okamoto, M. M., et al. (2010). Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue. Cell Biochemistry and Function, 28( 2), 114-121. doi:10.1002/cbf.1628
    • NLM

      Poletto AC, Anhê GF, Eichler P, Takahashi H, Furuya DT, Okamoto MM, Curi R, Machado UF. Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue [Internet]. Cell Biochemistry and Function. 2010 ; 28( 2): 114-121.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1002/cbf.1628
    • Vancouver

      Poletto AC, Anhê GF, Eichler P, Takahashi H, Furuya DT, Okamoto MM, Curi R, Machado UF. Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue [Internet]. Cell Biochemistry and Function. 2010 ; 28( 2): 114-121.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1002/cbf.1628
  • Source: Biomaterials. Unidade: ICB

    Assunto: FISIOLOGIA

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      VALOIS, Caroline R. A. et al. The effect of DMSA-functionalized magnetic nanoparticles on transendothelial migration of monocytes in the murine lung via a b2 integrin-dependent pathway. Biomaterials, v. 31, n. 2, p. 366-374, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.biomaterials.2009.09.053. Acesso em: 19 nov. 2024.
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      Valois, C. R. A., Braz, J. M., Nunes, E. S., Vinolo, M. A. R., Lima, E. C. D., Curi, R., et al. (2010). The effect of DMSA-functionalized magnetic nanoparticles on transendothelial migration of monocytes in the murine lung via a b2 integrin-dependent pathway. Biomaterials, 31( 2), 366-374. doi:10.1016/j.biomaterials.2009.09.053
    • NLM

      Valois CRA, Braz JM, Nunes ES, Vinolo MAR, Lima ECD, Curi R, Kuebler WM, Azevedo RB. The effect of DMSA-functionalized magnetic nanoparticles on transendothelial migration of monocytes in the murine lung via a b2 integrin-dependent pathway [Internet]. Biomaterials. 2010 ; 31( 2): 366-374.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.biomaterials.2009.09.053
    • Vancouver

      Valois CRA, Braz JM, Nunes ES, Vinolo MAR, Lima ECD, Curi R, Kuebler WM, Azevedo RB. The effect of DMSA-functionalized magnetic nanoparticles on transendothelial migration of monocytes in the murine lung via a b2 integrin-dependent pathway [Internet]. Biomaterials. 2010 ; 31( 2): 366-374.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.biomaterials.2009.09.053
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      GOULART DA SILVA, F. et al. T3 acutely increases GH mRNA translation rate and GH secretion in hypothyroid rats. Molecular and Cellular Endocrinology, v. 317, n. 1/2, p. 1-7, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2009.12.005. Acesso em: 19 nov. 2024.
    • APA

      Goulart da Silva, F., Giannocco, G., Luchessi, A. D., Curi, R., & Nunes, M. T. (2010). T3 acutely increases GH mRNA translation rate and GH secretion in hypothyroid rats. Molecular and Cellular Endocrinology, 317( 1/2), 1-7. doi:10.1016/j.mce.2009.12.005
    • NLM

      Goulart da Silva F, Giannocco G, Luchessi AD, Curi R, Nunes MT. T3 acutely increases GH mRNA translation rate and GH secretion in hypothyroid rats [Internet]. Molecular and Cellular Endocrinology. 2010 ; 317( 1/2): 1-7.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.mce.2009.12.005
    • Vancouver

      Goulart da Silva F, Giannocco G, Luchessi AD, Curi R, Nunes MT. T3 acutely increases GH mRNA translation rate and GH secretion in hypothyroid rats [Internet]. Molecular and Cellular Endocrinology. 2010 ; 317( 1/2): 1-7.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.mce.2009.12.005
  • Source: Metabolism Clinical and Experimental. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      MORARI, Joseane et al. The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental, v. 59, n. 2, p. 215-223, 2010Tradução . . Acesso em: 19 nov. 2024.
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      Morari, J., Torsoni, A. S., Anhê, G. F., Roman, E. A., Cintra, D. E., Ward, L. S., et al. (2010). The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental, 59( 2), 215-223.
    • NLM

      Morari J, Torsoni AS, Anhê GF, Roman EA, Cintra DE, Ward LS, Bordin S, Velloso LA. The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental. 2010 ; 59( 2): 215-223.[citado 2024 nov. 19 ]
    • Vancouver

      Morari J, Torsoni AS, Anhê GF, Roman EA, Cintra DE, Ward LS, Bordin S, Velloso LA. The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental. 2010 ; 59( 2): 215-223.[citado 2024 nov. 19 ]
  • Source: Diabetologia. Unidade: ICB

    Assunto: FISIOLOGIA

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      NEWSHOLME, P. et al. Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell. Diabetologia, v. 52, p. 2489-2498, 2009Tradução . . Disponível em: https://doi.org/10.1007/s00125-009-1536-z. Acesso em: 19 nov. 2024.
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      Newsholme, P., Morgan, D., Rebelato, E., Oliveira-Emilio, H. C., Araujo Filho, J. P. de, Curi, R., & Carpinelli, A. R. (2009). Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell. Diabetologia, 52, 2489-2498. doi:10.1007/s00125-009-1536-z
    • NLM

      Newsholme P, Morgan D, Rebelato E, Oliveira-Emilio HC, Araujo Filho JP de, Curi R, Carpinelli AR. Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell [Internet]. Diabetologia. 2009 ; 52 2489-2498.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00125-009-1536-z
    • Vancouver

      Newsholme P, Morgan D, Rebelato E, Oliveira-Emilio HC, Araujo Filho JP de, Curi R, Carpinelli AR. Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell [Internet]. Diabetologia. 2009 ; 52 2489-2498.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00125-009-1536-z
  • Source: Muscle and Nerve. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

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      WAGNER, Ana Barbara Teixeira Alves et al. 'beta'-adrenergic activity preserves Glut4 protein in glycolytic fibers in fasting. Muscle and Nerve, v. 40, n. 5, p. 847-854, 2009Tradução . . Acesso em: 19 nov. 2024.
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      Wagner, A. B. T. A., Freitas, H. S. de, Souza, P. B. de, Seraphim, P. M., Mori, R. C. T., & Machado, U. F. (2009). 'beta'-adrenergic activity preserves Glut4 protein in glycolytic fibers in fasting. Muscle and Nerve, 40( 5), 847-854.
    • NLM

      Wagner ABTA, Freitas HS de, Souza PB de, Seraphim PM, Mori RCT, Machado UF. 'beta'-adrenergic activity preserves Glut4 protein in glycolytic fibers in fasting. Muscle and Nerve. 2009 ; 40( 5): 847-854.[citado 2024 nov. 19 ]
    • Vancouver

      Wagner ABTA, Freitas HS de, Souza PB de, Seraphim PM, Mori RCT, Machado UF. 'beta'-adrenergic activity preserves Glut4 protein in glycolytic fibers in fasting. Muscle and Nerve. 2009 ; 40( 5): 847-854.[citado 2024 nov. 19 ]

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