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Artigo de periodico
COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis (2023)
Barreto, Ester A.; Cruz, Amanda S.; Veras, Flávio Protásio ; Martins, Ronaldo Bragança ; Bernardelli, Rafaella S.; Paiva, Isadora Marques ; Lima, Thais Melquiades de; Singh, Youvika ; Guimaraes, Raphael C.; Damasceno, Samara; Pereira, Nayara; Alves, João Manoel; Gonçalves, Tiago T.; Forato, Julia; Muraro, Stéfanie P.; Souza, Gabriela F.; Batah, Sabrina Setembre ; Proença-Modena, José L.; Mori, Marcelo A.; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Cunha, Thiago Mattar ; Nakaya, Helder Takashi Imoto ; Fabro, Alexandre Todorovic ; Oliveira, Renê Donizeti Ribeiro de; Arruda, Eurico ; Rea, Rosângela; Réa Neto, Álvaro; Silva, Miguel M Fernandes da; Leiria, Luiz Osório Silveira
Fonte: Proceedings of the National Academy of Sciences. Unidades: FMRP, FCF
Assuntos: GLICOSE, COVID-19, GLICOGENOSE, HIPERGLICEMIA, ESTUDOS RETROSPECTIVOS
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ABNT
BARRETO, Ester A. et al. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, v. 120, n. 21, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2217119120. Acesso em: 23 jul. 2024.
APA
Barreto, E. A., Cruz, A. S., Veras, F. P., Martins, R. B., Bernardelli, R. S., Paiva, I. M., et al. (2023). COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, 120( 21), 1-10. doi:10.1073/pnas.2217119120
NLM
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1073/pnas.2217119120
Vancouver
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1073/pnas.2217119120
Artigo de periodico
Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction (2022)
Silva, Josiane Fernandes da; Bolsoni, Juliana A.; Costa, Rafael Menezes da ; Alves, Juliano Vilela ; Bressan, Alecsander Fabrício Moreira ; Silva, Luiz Eduardo Virgilio da ; Costa, Tiago Januário da ; Oliveira, Antonio E. R.; Manzato, Carla Pavan; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Nakaya, Helder Takashi Imoto ; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo
Fonte: British Journal of Pharmacology. Unidades: FMRP, FCF
Assuntos: GORDURAS NA DIETA, CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, OBESIDADE, HIDROCARBONETOS, VASODILATAÇÃO
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SILVA, Josiane Fernandes da et al. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, v. 179, n. 12, p. 2938-2952, 2022Tradução . . Disponível em: https://doi.org/10.1111/bph.15789. Acesso em: 23 jul. 2024.
APA
Silva, J. F. da, Bolsoni, J. A., Costa, R. M. da, Alves, J. V., Bressan, A. F. M., Silva, L. E. V. da, et al. (2022). Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, 179( 12), 2938-2952. doi:10.1111/bph.15789
NLM
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1111/bph.15789
Vancouver
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1111/bph.15789
Artigo de periodico
Morphological, cellular, and molecular basis of brain infection in COVID-19 patients (2022)
Crunfli, Fernanda ; Carregari, Victor Corasolla ; Veras, Flávio Protásio; Silva, Lucas S.; Nogueira, Mateus Henrique ; Antunes, André Saraiva Leão Marcelo ; Vendramini, Pedro Henrique; Valença, Aline Gazzola Fragnani; Teles, Caroline Brandão; Munhoz, Carolina Demarchi ; Nakaya, Helder Takashi Imoto ; Melo, Bruno Marcel; Almeida, Glaucia Maria de; Paiva, Isadora Marques; Silva, Bruna Manuella Souza; Guimarães, Rafaela Mano; Mendes, Niele Dias; Batah, Sabrina Setembre ; Siyuan, Li; Tavares, Lucas Alves ; Viana, Rosa Maria Mendes; Alves Filho, José Carlos Farias ; Arruda Neto, Eurico de; Podolsky-Gondim, Guilherme Gozzoli ; Santos, Marcelo Volpon ; Neder, Luciano; Louzada Júnior, Paulo; Oliveira, Renê Donizeti Ribeiro de; Cunha, Fernando de Queiroz ; Fabro, Alexandre Todorovic; Sebollela, Adriano Silva ; Cunha, Thiago Mattar
Fonte: Proceedings of the National Academy of Sciences. Unidades: ICB, FCF, FCFRP, FMRP
Assuntos: FARMACOLOGIA, COVID 19
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ABNT
CRUNFLI, Fernanda et al. Morphological, cellular, and molecular basis of brain infection in COVID-19 patients. Proceedings of the National Academy of Sciences, v. 119, n. 35, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1073/pnas.2200960119. Acesso em: 23 jul. 2024.
APA
Crunfli, F., Carregari, V. C., Veras, F. P., Silva, L. S., Nogueira, M. H., Antunes, A. S. L. M., et al. (2022). Morphological, cellular, and molecular basis of brain infection in COVID-19 patients. Proceedings of the National Academy of Sciences, 119( 35), 1-12. doi:10.1073/pnas.2200960119
NLM
Crunfli F, Carregari VC, Veras FP, Silva LS, Nogueira MH, Antunes ASLM, Vendramini PH, Valença AGF, Teles CB, Munhoz CD, Nakaya HTI, Melo BM, Almeida GM de, Paiva IM, Silva BMS, Guimarães RM, Mendes ND, Batah SS, Siyuan L, Tavares LA, Viana RMM, Alves Filho JCF, Arruda Neto E de, Podolsky-Gondim GG, Santos MV, Neder L, Louzada Júnior P, Oliveira RDR de, Cunha F de Q, Fabro AT, Sebollela AS, Cunha TM. Morphological, cellular, and molecular basis of brain infection in COVID-19 patients [Internet]. Proceedings of the National Academy of Sciences. 2022 ; 119( 35): 1-12.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1073/pnas.2200960119
Vancouver
Crunfli F, Carregari VC, Veras FP, Silva LS, Nogueira MH, Antunes ASLM, Vendramini PH, Valença AGF, Teles CB, Munhoz CD, Nakaya HTI, Melo BM, Almeida GM de, Paiva IM, Silva BMS, Guimarães RM, Mendes ND, Batah SS, Siyuan L, Tavares LA, Viana RMM, Alves Filho JCF, Arruda Neto E de, Podolsky-Gondim GG, Santos MV, Neder L, Louzada Júnior P, Oliveira RDR de, Cunha F de Q, Fabro AT, Sebollela AS, Cunha TM. Morphological, cellular, and molecular basis of brain infection in COVID-19 patients [Internet]. Proceedings of the National Academy of Sciences. 2022 ; 119( 35): 1-12.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1073/pnas.2200960119
Artigo de periodico
Efferocytosis of SARS-CoV-2-infected dying cells impairs macrophage anti-inflammatory functions and clearance of apoptotic cells (2022)
Salina, Ana Carolina Guerta ; Santos, Douglas dos; Rodrigues, Tamara Silva; Rocha, Marlon Fortes; Freitas Filho, Edismauro Garcia ; Terrel, Daniel Leonardo Alzamora ; Castro, Ícaro Maia Santos ; Silva, Thais Fernanda de Campo Fraga da ; Lima, Mikhael Haruo Fernandes de; Nascimento, Daniele Carvalho Bernardo; Silva, Camila M.; Toller-kawahisa, Juliana Escher; Becerra, Amanda de Matos; Oliveira, Samuel; Caetité, Diego ; Almeida, Leticia de; Ishimoto, Adriene Yumi; Lima, Thais Melquiades de; Martins, Ronaldo Bragança ; Veras, Flávio Protásio ; Amaral, Natália Brasil do; Giannini, Marcela Cavichioli; Bonjorno, Letícia Pastorelli; Lopes, Maria Isabel Fernandes; Benatti, Maíra Nilson; Batah, Sabrina Setembre ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Martins, Maria Auxiliadora ; Assad, Rodrigo Luppino; Almeida, Sérgio Couto Luna de; Oliveira, Fabíola Reis de; Arruda Neto, Eurico de; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Bonato, Vânia Luiza Deperon ; Cunha, Fernando de Queiroz ; Fabro, Alexandre Todorovic ; Nakaya, Helder Takashi Imoto ; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Cunha, Larissa Dias da
Fonte: eLife. Unidades: FMRP, FCF, BIOINFORMÁTICA
Assuntos: COVID-19, INFLAMAÇÃO, MACRÓFAGOS, CÉLULAS
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ABNT
SALINA, Ana Carolina Guerta et al. Efferocytosis of SARS-CoV-2-infected dying cells impairs macrophage anti-inflammatory functions and clearance of apoptotic cells. eLife, v. 11, p. 1-24, 2022Tradução . . Disponível em: https://doi.org/10.7554/eLife.74443. Acesso em: 23 jul. 2024.
APA
Salina, A. C. G., Santos, D. dos, Rodrigues, T. S., Rocha, M. F., Freitas Filho, E. G., Terrel, D. L. A., et al. (2022). Efferocytosis of SARS-CoV-2-infected dying cells impairs macrophage anti-inflammatory functions and clearance of apoptotic cells. eLife, 11, 1-24. doi:10.7554/eLife.74443
NLM
Salina ACG, Santos D dos, Rodrigues TS, Rocha MF, Freitas Filho EG, Terrel DLA, Castro ÍMS, Silva TF de CF da, Lima MHF de, Nascimento DCB, Silva CM, Toller-kawahisa JE, Becerra A de M, Oliveira S, Caetité D, Almeida L de, Ishimoto AY, Lima TM de, Martins RB, Veras FP, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Benatti MN, Batah SS, Santana R de C, Vilar FC, Martins MA, Assad RL, Almeida SCL de, Oliveira FR de, Arruda Neto E de, Cunha TM, Alves Filho JCF, Bonato VLD, Cunha F de Q, Fabro AT, Nakaya HTI, Zamboni DS, Louzada Júnior P, Oliveira RDR de, Cunha LD da. Efferocytosis of SARS-CoV-2-infected dying cells impairs macrophage anti-inflammatory functions and clearance of apoptotic cells [Internet]. eLife. 2022 ; 11 1-24.[citado 2024 jul. 23 ] Available from: https://doi.org/10.7554/eLife.74443
Vancouver
Salina ACG, Santos D dos, Rodrigues TS, Rocha MF, Freitas Filho EG, Terrel DLA, Castro ÍMS, Silva TF de CF da, Lima MHF de, Nascimento DCB, Silva CM, Toller-kawahisa JE, Becerra A de M, Oliveira S, Caetité D, Almeida L de, Ishimoto AY, Lima TM de, Martins RB, Veras FP, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Benatti MN, Batah SS, Santana R de C, Vilar FC, Martins MA, Assad RL, Almeida SCL de, Oliveira FR de, Arruda Neto E de, Cunha TM, Alves Filho JCF, Bonato VLD, Cunha F de Q, Fabro AT, Nakaya HTI, Zamboni DS, Louzada Júnior P, Oliveira RDR de, Cunha LD da. Efferocytosis of SARS-CoV-2-infected dying cells impairs macrophage anti-inflammatory functions and clearance of apoptotic cells [Internet]. eLife. 2022 ; 11 1-24.[citado 2024 jul. 23 ] Available from: https://doi.org/10.7554/eLife.74443
Artigo de periodico
Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota (2022)
Arruda, José Alcides Almeida de ; Corrêa, Jôice Dias ; Singh, Youvika ; Oliveira, Sicília Rezende ; Machado, Caio Cavalcante ; Schneider, Ayda Henriques ; Medeiros, Julliane Dutra ; Fernandes, Gabriel R; Macari, Soraia ; Barrioni, Breno Rocha ; Santos, Mariana de Souza; Rodrigues, Letícia Fernanda Duffles ; Nakaya, Helder Takashi Imoto ; Fukada, Sandra Yasuyo ; Graves, Dana T ; Cunha, Fernando Queiroz ; Silva, Tarcília Aparecida
Fonte: Anaerobe. Unidades: FMRP, FCFRP, FCF, FORP
Assuntos: ARTRITE REUMATOIDE, FARMACOTERAPIA, METOTREXATO
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ABNT
ARRUDA, José Alcides Almeida de et al. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota. Anaerobe, v. 75, p. 1-11 art. 102577, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.anaerobe.2022.102577. Acesso em: 23 jul. 2024.
APA
Arruda, J. A. A. de, Corrêa, J. D., Singh, Y., Oliveira, S. R., Machado, C. C., Schneider, A. H., et al. (2022). Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota. Anaerobe, 75, 1-11 art. 102577. doi:10.1016/j.anaerobe.2022.102577
NLM
Arruda JAA de, Corrêa JD, Singh Y, Oliveira SR, Machado CC, Schneider AH, Medeiros JD, Fernandes GR, Macari S, Barrioni BR, Santos M de S, Rodrigues LFD, Nakaya HTI, Fukada SY, Graves DT, Cunha FQ, Silva TA. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota [Internet]. Anaerobe. 2022 ; 75 1-11 art. 102577.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.anaerobe.2022.102577
Vancouver
Arruda JAA de, Corrêa JD, Singh Y, Oliveira SR, Machado CC, Schneider AH, Medeiros JD, Fernandes GR, Macari S, Barrioni BR, Santos M de S, Rodrigues LFD, Nakaya HTI, Fukada SY, Graves DT, Cunha FQ, Silva TA. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota [Internet]. Anaerobe. 2022 ; 75 1-11 art. 102577.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.anaerobe.2022.102577
Artigo de periodico
Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity (2021)
Nascimento, Daniele Carvalho ; Viacava, Paula Ramos; Ferreira, Raphael Gomes; Damaceno, Marina Alves; Piñeros, Annie Rocío Alvarez ; Melo, Paulo Henrique de; Donate, Paula Barbim ; Toller-Kawahisa, Juliana Escher; Zoppi, Daniel ; Veras, Flávio Protásio ; Peres, Raphael S. ; Silva, Luisa Menezes ; Caetité, Diego ; Oliveira, Antonio Edson Rocha; Castro, Icaro Maia Santos ; Kauffenstein, Gilles ; Nakaya, Helder Takashi Imoto ; Borges, Marcos de Carvalho ; Zamboni, Dario Simões ; Fonseca, Denise Morais da ; Paschoal, Jonas Augusto Rizzato ; Cunha, Thiago Mattar ; Quesniaux, Valerie ; Linden, Joel ; Cunha, Fernando Queiróz ; Ryffe, Bernhard; Alves-Filho, José Carlos
Fonte: Immunity. Unidades: FMRP, FCFRP, FCF, ICB, BIOINFORMÁTICA
Assuntos: ADENOSINA, FÁRMACOS IMUNOSSUPRESSORES, SEPSE, CAMUNDONGOS, INTERLEUCINA 10, MACRÓFAGOS
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ABNT
NASCIMENTO, Daniele Carvalho et al. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, v. 54, n. 9, p. 2024-2041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.immuni.2021.08.005. Acesso em: 23 jul. 2024.
APA
Nascimento, D. C., Viacava, P. R., Ferreira, R. G., Damaceno, M. A., Piñeros, A. R. A., Melo, P. H. de, et al. (2021). Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, 54( 9), 2024-2041. doi:10.1016/j.immuni.2021.08.005
NLM
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Vancouver
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Artigo de periodico
In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19 (2021)
Caten, Felipe Ten ; Dias, Patricia Gonzalez ; Castro, Icaro; Ogava, Rodrigo Luiz Tomio; Giddaluru, Jeevan ; Silva, Juan Carlo Santos e ; Martins, Felipe; Gonçalves, André Nicolau Aquime ; Martins, André Guilherme da Costa; Araújo, José Deney A ; Viegas, Ana Carolina Nakaya ; Cunha, Fernando de Queiroz ; Farsky, Sandra Helena Poliselli ; Bozza, Fernando A; Levin, Anna Sara Shafferman ; Pannaraj, Pia S ; Silva, Thushan I. de; Minoprio, Paola; Silva, Fabiano Pinheiro da; Andrade, Bruno B; Nakaya, Helder Takashi Imoto
Fonte: International Journal of Infectious Diseases. Unidades: FCF, FM, FMRP, BIOINFORMÁTICA
Assuntos: COVID-19, NEUTRÓFILOS, TRANSTORNOS DA COAGULAÇÃO SANGUÍNEA, IDADE, SEXO
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ABNT
CATEN, Felipe Ten et al. In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19. International Journal of Infectious Diseases, v. 105, p. 579–587, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.ijid.2021.03.016. Acesso em: 23 jul. 2024.
APA
Caten, F. T., Dias, P. G., Castro, I., Ogava, R. L. T., Giddaluru, J., Silva, J. C. S. e, et al. (2021). In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19. International Journal of Infectious Diseases, 105, 579–587. doi:10.1016/j.ijid.2021.03.016
NLM
Caten FT, Dias PG, Castro I, Ogava RLT, Giddaluru J, Silva JCS e, Martins F, Gonçalves ANA, Martins AG da C, Araújo JDA, Viegas ACN, Cunha F de Q, Farsky SHP, Bozza FA, Levin ASS, Pannaraj PS, Silva TI de, Minoprio P, Silva FP da, Andrade BB, Nakaya HTI. In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19 [Internet]. International Journal of Infectious Diseases. 2021 ; 105 579–587.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.ijid.2021.03.016
Vancouver
Caten FT, Dias PG, Castro I, Ogava RLT, Giddaluru J, Silva JCS e, Martins F, Gonçalves ANA, Martins AG da C, Araújo JDA, Viegas ACN, Cunha F de Q, Farsky SHP, Bozza FA, Levin ASS, Pannaraj PS, Silva TI de, Minoprio P, Silva FP da, Andrade BB, Nakaya HTI. In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19 [Internet]. International Journal of Infectious Diseases. 2021 ; 105 579–587.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.ijid.2021.03.016
Artigo de periodico
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis (2018)
Peres, Raphael Sanches; Donate, Paula Barbim; Talbot, Jhimmy; Cecílio, Nerry Tatiana; Lobo, Patricia Rolim Mendonça; Machado, Caio Cavalcante; Lima, Kalil William Alves de; Oliveira, Renê Donizete Ribeiro de; Carregaro, Vanessa; Nakaya, Helder Takashi Imoto ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Liew, Foo Yew; Louzada Júnior, Paulo; Cunha, Fernando Queiróz
Fonte: Journal of Autoimmunity. Unidades: FMRP, FCF
Assuntos: ARTRITE REUMATOIDE, METOTREXATO
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ABNT
PERES, Raphael Sanches et al. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, v. 90, p. 49-58, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jaut.2018.01.004. Acesso em: 23 jul. 2024.
APA
Peres, R. S., Donate, P. B., Talbot, J., Cecílio, N. T., Lobo, P. R. M., Machado, C. C., et al. (2018). TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, 90, 49-58. doi:10.1016/j.jaut.2018.01.004
NLM
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Vancouver
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004

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