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Artigo de periodico
Metagenomics-enabled reverse-genetics assembly and characterization of myotis bat morbillivirus (2023)
Ikegame, Satoshi ; Carmichael, Jillian C. ; Wells, Heather ; O’Brien, Robert L. Furler ; Acklin, Joshua A. ; Chiu, Hsin-Ping ; Oguntuyo, Kasopefoluwa Y. ; Cox, Robert M. ; Patel, Aum R. ; Kowdle, Shreyas ; Stevens, Christian S. ; Eckley, Miles ; Shijun Zhan ; Lim, Jean K.; Veit, Ethan C. ; Evans, Matthew J.; Hashiguchi, Takao ; Schountz, Tony ; Epstein, Jonathan H. ; Plemper, Richard K. ; Daszak, Peter ; Anthony, Simon J. ; Lee, Benhur Lee ; Durigon, Edison Luiz
Source: Nature Microbiology. Unidade: ICB
Subjects: MICROBIOLOGIA, GENÔMICA, INIBIDORES DE ENZIMAS, MORCEGOS, MICROSCOPIA ELETRÔNICA, VIGILÂNCIA EPIDEMIOLÓGICA, CÉLULAS EPITELIAIS, MACRÓFAGOS, INTERFERONS
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IKEGAME, Satoshi et al. Metagenomics-enabled reverse-genetics assembly and characterization of myotis bat morbillivirus. Nature Microbiology, p. 1-31, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41564-023-01380-4. Acesso em: 06 nov. 2024.
APA
Ikegame, S., Carmichael, J. C., Wells, H., O’Brien, R. L. F., Acklin, J. A., Chiu, H. -P., et al. (2023). Metagenomics-enabled reverse-genetics assembly and characterization of myotis bat morbillivirus. Nature Microbiology, 1-31. doi:10.1038/s41564-023-01380-4
NLM
Ikegame S, Carmichael JC, Wells H, O’Brien RLF, Acklin JA, Chiu H-P, Oguntuyo KY, Cox RM, Patel AR, Kowdle S, Stevens CS, Eckley M, Shijun Zhan, Lim JK, Veit EC, Evans MJ, Hashiguchi T, Schountz T, Epstein JH, Plemper RK, Daszak P, Anthony SJ, Lee BL, Durigon EL. Metagenomics-enabled reverse-genetics assembly and characterization of myotis bat morbillivirus [Internet]. Nature Microbiology. 2023 ; 1-31.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41564-023-01380-4
Vancouver
Ikegame S, Carmichael JC, Wells H, O’Brien RLF, Acklin JA, Chiu H-P, Oguntuyo KY, Cox RM, Patel AR, Kowdle S, Stevens CS, Eckley M, Shijun Zhan, Lim JK, Veit EC, Evans MJ, Hashiguchi T, Schountz T, Epstein JH, Plemper RK, Daszak P, Anthony SJ, Lee BL, Durigon EL. Metagenomics-enabled reverse-genetics assembly and characterization of myotis bat morbillivirus [Internet]. Nature Microbiology. 2023 ; 1-31.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41564-023-01380-4
Artigo de periodico
Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer (2021)
Staquicini, Fernanda I. ; Hajitou, Amin ; Driessen, Wouter HP ; Proneth, Bettina ; Vila, Marina Cardó ; Staquicini, Daniela I.; Markosian, Christopher ; Hoh, Maria ; Cortez, Mauro ; Nehra, Anupama Hooda ; Jaloudi, Mohammed; Silva, Israel T. ; Buttura, Jaqueline; Nunes, Diana; Dias Neto, Emmanuel ; Eckhardt, Bedrich ; Ramírez, Javier Ruiz ; Dogra, Prashant ; Wang, Zhihui; Cristini, Vittorio ; Trepel, Martin; Anderson, Robin ; Sidman, Richard L.; Gelovani, Juri G. ; Cristofanilli, Massimo ; Hortobagy, Gabriel; Bhujwalla, Zaver M. ; Burley, Stephen ; Arap, Wadih ; Pasqualini, Renata
Source: eLife. Unidade: ICB
Subjects: PARASITOLOGIA, QUIMIOTERÁPICOS, ANTINEOPLÁSICOS, NEOPLASIAS MAMÁRIAS, CÉLULAS CULTIVADAS DE TUMOR, MACRÓFAGOS, VITAMINA D, CAMUNDONGOS, CITOCINAS, PEPTÍDEOS, FÁRMACOS IMUNOSSUPRESSORES
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STAQUICINI, Fernanda I. et al. Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer. eLife, v. 10, p. 1-24, 2021Tradução . . Disponível em: https://doi.org/10.7554/eLife.65145. Acesso em: 06 nov. 2024.
APA
Staquicini, F. I., Hajitou, A., Driessen, W. H. P., Proneth, B., Vila, M. C., Staquicini, D. I., et al. (2021). Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer. eLife, 10, 1-24. doi:10.7554/eLife.65145
NLM
Staquicini FI, Hajitou A, Driessen WHP, Proneth B, Vila MC, Staquicini DI, Markosian C, Hoh M, Cortez M, Nehra AH, Jaloudi M, Silva IT, Buttura J, Nunes D, Dias Neto E, Eckhardt B, Ramírez JR, Dogra P, Wang Z, Cristini V, Trepel M, Anderson R, Sidman RL, Gelovani JG, Cristofanilli M, Hortobagy G, Bhujwalla ZM, Burley S, Arap W, Pasqualini R. Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer [Internet]. eLife. 2021 ; 10 1-24.[citado 2024 nov. 06 ] Available from: https://doi.org/10.7554/eLife.65145
Vancouver
Staquicini FI, Hajitou A, Driessen WHP, Proneth B, Vila MC, Staquicini DI, Markosian C, Hoh M, Cortez M, Nehra AH, Jaloudi M, Silva IT, Buttura J, Nunes D, Dias Neto E, Eckhardt B, Ramírez JR, Dogra P, Wang Z, Cristini V, Trepel M, Anderson R, Sidman RL, Gelovani JG, Cristofanilli M, Hortobagy G, Bhujwalla ZM, Burley S, Arap W, Pasqualini R. Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer [Internet]. eLife. 2021 ; 10 1-24.[citado 2024 nov. 06 ] Available from: https://doi.org/10.7554/eLife.65145
Artigo de periodico
STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection (2021)
Gomes, Marco Tulio R.; Guimarães, Erika S.; Marinho, Fabio V.; Macedo, Isabella; Aguiar, Eric R. G. R.; Barber, Glen N.; Moraes-Vieira, Pedro M. M.; Alves Filho, José Carlos Farias ; Oliveira, Sergio C.
Source: PLOS Pathogens. Unidade: FMRP
Subjects: MACRÓFAGOS, INFLAMAÇÃO, INFECÇÕES RESPIRATÓRIAS, BRUCELOSE, MITOCÔNDRIAS, REAÇÃO EM CADEIA POR POLIMERASE
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GOMES, Marco Tulio R. et al. STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection. PLOS Pathogens, 2021Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1009597. Acesso em: 06 nov. 2024.
APA
Gomes, M. T. R., Guimarães, E. S., Marinho, F. V., Macedo, I., Aguiar, E. R. G. R., Barber, G. N., et al. (2021). STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection. PLOS Pathogens. doi:10.1371/journal.ppat.1009597
NLM
Gomes MTR, Guimarães ES, Marinho FV, Macedo I, Aguiar ERGR, Barber GN, Moraes-Vieira PMM, Alves Filho JCF, Oliveira SC. STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection [Internet]. PLOS Pathogens. 2021 ;[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.ppat.1009597
Vancouver
Gomes MTR, Guimarães ES, Marinho FV, Macedo I, Aguiar ERGR, Barber GN, Moraes-Vieira PMM, Alves Filho JCF, Oliveira SC. STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection [Internet]. PLOS Pathogens. 2021 ;[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.ppat.1009597
Artigo de periodico
Bexarotene enhances macrophage erythrophagocytosis and hematoma clearance in experimental intracerebral hemorrhage (2020)
Chang, Che-Feng; Massey, Jordan; Osherov, Artem; Costa, Luís Henrique Angenendt da ; Sansing, Lauren H.
Source: Stroke. Unidade: FORP
Subjects: ANIMAIS, CÉREBRO, MODELOS ANIMAIS, ERITRÓCITOS, HEMATOMA, MACRÓFAGOS
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CHANG, Che-Feng et al. Bexarotene enhances macrophage erythrophagocytosis and hematoma clearance in experimental intracerebral hemorrhage. Stroke, v. 51, n. 2, p. 612-618, 2020Tradução . . Disponível em: https://doi.org/10.1161/STROKEAHA.119.027037. Acesso em: 06 nov. 2024.
APA
Chang, C. -F., Massey, J., Osherov, A., Costa, L. H. A. da, & Sansing, L. H. (2020). Bexarotene enhances macrophage erythrophagocytosis and hematoma clearance in experimental intracerebral hemorrhage. Stroke, 51( 2), 612-618. doi:10.1161/STROKEAHA.119.027037
NLM
Chang C-F, Massey J, Osherov A, Costa LHA da, Sansing LH. Bexarotene enhances macrophage erythrophagocytosis and hematoma clearance in experimental intracerebral hemorrhage [Internet]. Stroke. 2020 ; 51( 2): 612-618.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1161/STROKEAHA.119.027037
Vancouver
Chang C-F, Massey J, Osherov A, Costa LHA da, Sansing LH. Bexarotene enhances macrophage erythrophagocytosis and hematoma clearance in experimental intracerebral hemorrhage [Internet]. Stroke. 2020 ; 51( 2): 612-618.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1161/STROKEAHA.119.027037
Artigo de periodico
Clearance of Pneumococcal colonization in infants is delayed through altered macrophage trafficking (2015)
Siegel, Steven J.; Tamashiro, Edwin ; Weiser, Jeffrey N.
Source: PLOS Pathogens. Unidade: FMRP
Subjects: MACRÓFAGOS, BEBÊS, NASOFARINGE, QUIMIOCINAS, ANTIBIÓTICOS, INFLAMAÇÃO, STREPTOCOCCUS PNEUMONIAE
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SIEGEL, Steven J. e TAMASHIRO, Edwin e WEISER, Jeffrey N. Clearance of Pneumococcal colonization in infants is delayed through altered macrophage trafficking. PLOS Pathogens, v. 11, n. 6, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1005004. Acesso em: 06 nov. 2024.
APA
Siegel, S. J., Tamashiro, E., & Weiser, J. N. (2015). Clearance of Pneumococcal colonization in infants is delayed through altered macrophage trafficking. PLOS Pathogens, 11( 6), 1-17. doi:10.1371/journal.ppat.1005004
NLM
Siegel SJ, Tamashiro E, Weiser JN. Clearance of Pneumococcal colonization in infants is delayed through altered macrophage trafficking [Internet]. PLOS Pathogens. 2015 ; 11( 6): 1-17.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.ppat.1005004
Vancouver
Siegel SJ, Tamashiro E, Weiser JN. Clearance of Pneumococcal colonization in infants is delayed through altered macrophage trafficking [Internet]. PLOS Pathogens. 2015 ; 11( 6): 1-17.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.ppat.1005004
Artigo de periodico
The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis (2014)
Secatto, Adriana; Soares, Elyara Maria; Locachevic, Gisele Aparecida ; Assis, Patricia Aparecida de; Paula-Silva, Francisco Wanderley Garcia de ; Serezani, Carlos Henrique Cardoso; Medeiros, Alexandra Ivo de ; Faccioli, Lucia Helena
Source: PLOS ONE. Unidades: FORP, ICB, FMRP, FCFRP
Subjects: HISTOPLASMA, MACRÓFAGOS, FAGOCITOSE, NEUTRÓFILOS, FERMENTOS, FUNGOS, ENZIMAS, LEUCOTRIENOS
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SECATTO, Adriana et al. The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis. PLOS ONE, v. 9, n. 1, p. 1-9, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0085083. Acesso em: 06 nov. 2024.
APA
Secatto, A., Soares, E. M., Locachevic, G. A., Assis, P. A. de, Paula-Silva, F. W. G. de, Serezani, C. H. C., et al. (2014). The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis. PLOS ONE, 9( 1), 1-9. doi:10.1371/journal.pone.0085083
NLM
Secatto A, Soares EM, Locachevic GA, Assis PA de, Paula-Silva FWG de, Serezani CHC, Medeiros AI de, Faccioli LH. The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis [Internet]. PLOS ONE. 2014 ; 9( 1): 1-9.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.pone.0085083
Vancouver
Secatto A, Soares EM, Locachevic GA, Assis PA de, Paula-Silva FWG de, Serezani CHC, Medeiros AI de, Faccioli LH. The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis [Internet]. PLOS ONE. 2014 ; 9( 1): 1-9.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.pone.0085083
Artigo de periodico
Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation (2013)
Carneiro, Alan Brito; Iaciura, Bruna Maria Ferreira; Nohara, Lilian Lie; Lopes, Carla Duque ; Veas, Esteban Mauricio Cordero; Mariano, Vania Sammartino; Bozza, Patricia Torres; Lopes, Ulisses Gazos; Atella, Georgia Correa; Almeida, Igor Correia ; Silva-Neto, Mário Alberto Cardoso
Source: PLOS ONE. Unidade: FMRP
Subjects: MACRÓFAGOS, ÓXIDO NÍTRICO, TRANSFECÇÃO, FATORES DE TRANSCRIÇÃO, FOSFOLIPÍDEOS
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CARNEIRO, Alan Brito et al. Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation. PLOS ONE, v. 8, n. 9, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076233. Acesso em: 06 nov. 2024.
APA
Carneiro, A. B., Iaciura, B. M. F., Nohara, L. L., Lopes, C. D., Veas, E. M. C., Mariano, V. S., et al. (2013). Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation. PLOS ONE, 8( 9), 1-9. doi:10.1371/journal.pone.0076233
NLM
Carneiro AB, Iaciura BMF, Nohara LL, Lopes CD, Veas EMC, Mariano VS, Bozza PT, Lopes UG, Atella GC, Almeida IC, Silva-Neto MAC. Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation [Internet]. PLOS ONE. 2013 ; 8( 9): 1-9.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.pone.0076233
Vancouver
Carneiro AB, Iaciura BMF, Nohara LL, Lopes CD, Veas EMC, Mariano VS, Bozza PT, Lopes UG, Atella GC, Almeida IC, Silva-Neto MAC. Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation [Internet]. PLOS ONE. 2013 ; 8( 9): 1-9.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.pone.0076233
Artigo de periodico
Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila (2008)
Shin, Sunny; Case, Christopher L.; Archer, Kristina A.; Nogueira, Catarina V.; Kobayashi, Koichi S.; Flavell, Richard A.; Roy, Craig R.; Zamboni, Dario Simões
Source: PLOS Pathogens. Unidade: FMRP
Subjects: LEGIONELLA PNEUMOPHILA, MACRÓFAGOS, CITOCINAS, SECREÇÃO, TRANSCRIÇÃO GÊNICA, SISTEMA IMUNE, VIRULÊNCIA
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SHIN, Sunny et al. Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila. PLOS Pathogens, v. 4, n. 11, p. 1-13, 2008Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1000220. Acesso em: 06 nov. 2024.
APA
Shin, S., Case, C. L., Archer, K. A., Nogueira, C. V., Kobayashi, K. S., Flavell, R. A., et al. (2008). Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila. PLOS Pathogens, 4( 11), 1-13. doi:10.1371/journal.ppat.1000220
NLM
Shin S, Case CL, Archer KA, Nogueira CV, Kobayashi KS, Flavell RA, Roy CR, Zamboni DS. Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila [Internet]. PLOS Pathogens. 2008 ; 4( 11): 1-13.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.ppat.1000220
Vancouver
Shin S, Case CL, Archer KA, Nogueira CV, Kobayashi KS, Flavell RA, Roy CR, Zamboni DS. Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila [Internet]. PLOS Pathogens. 2008 ; 4( 11): 1-13.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1371/journal.ppat.1000220

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