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Artigo de periodico
Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice (2022)
Castro, Paula A. T. S.; Barbosa, Germanna M.; Machanocker, Dafiner H.; Peres, Raphael S.; Cunha, Thiago Mattar ; Cunha, Jonathan E.; Oliveira, Francisco F. B.; Ramalho, Fernando Silva ; Russo, Thiago L.; Cunha, Fernando de Queiroz ; Salvini, Tania F.
Source: Plos one. Unidade: FMRP
Subjects: CRIOTERAPIA, INFLAMAÇÃO, ARTRITE, JOELHO
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ABNT
CASTRO, Paula A. T. S. et al. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice. Plos one, v. 17, n. 1, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0261667. Acesso em: 17 nov. 2024.
APA
Castro, P. A. T. S., Barbosa, G. M., Machanocker, D. H., Peres, R. S., Cunha, T. M., Cunha, J. E., et al. (2022). Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice. Plos one, 17( 1), 1-14. doi:10.1371/journal.pone.0261667
NLM
Castro PATS, Barbosa GM, Machanocker DH, Peres RS, Cunha TM, Cunha JE, Oliveira FFB, Ramalho FS, Russo TL, Cunha F de Q, Salvini TF. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice [Internet]. Plos one. 2022 ; 17( 1): 1-14.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0261667
Vancouver
Castro PATS, Barbosa GM, Machanocker DH, Peres RS, Cunha TM, Cunha JE, Oliveira FFB, Ramalho FS, Russo TL, Cunha F de Q, Salvini TF. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice [Internet]. Plos one. 2022 ; 17( 1): 1-14.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0261667
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 17 nov. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus (2018)
Ferreira, Raphael Gomes; Rodrigues, Lilian Cataldi ; Nascimento, Daniele Carvalho Bernardo; Kanashiro, Alexandre ; Melo, Paulo Henrique de; Borges, Vanessa de Fátima; Gozzi, Aline ; Prado, Douglas da Silva ; Borges, Marcos de Carvalho; Ramalho, Fernando Silva ; Stowell, Sean R.; Cummings, Richard D.; Dias-Baruffi, Marcelo ; Cunha, Fernando de Queiroz ; Alves-Filho, José Carlos Farias
Source: Journal of Infection. Unidades: FCFRP, FMRP
Subjects: SEPSE, NEUTRÓFILOS, INFLAMAÇÃO
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ABNT
FERREIRA, Raphael Gomes et al. Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus. Journal of Infection, v. 77, n. 5, p. 391-397, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jinf.2018.06.010. Acesso em: 17 nov. 2024.
APA
Ferreira, R. G., Rodrigues, L. C., Nascimento, D. C. B., Kanashiro, A., Melo, P. H. de, Borges, V. de F., et al. (2018). Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus. Journal of Infection, 77( 5), 391-397. doi:10.1016/j.jinf.2018.06.010
NLM
Ferreira RG, Rodrigues LC, Nascimento DCB, Kanashiro A, Melo PH de, Borges V de F, Gozzi A, Prado D da S, Borges M de C, Ramalho FS, Stowell SR, Cummings RD, Dias-Baruffi M, Cunha F de Q, Alves-Filho JCF. Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus [Internet]. Journal of Infection. 2018 ; 77( 5): 391-397.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.jinf.2018.06.010
Vancouver
Ferreira RG, Rodrigues LC, Nascimento DCB, Kanashiro A, Melo PH de, Borges V de F, Gozzi A, Prado D da S, Borges M de C, Ramalho FS, Stowell SR, Cummings RD, Dias-Baruffi M, Cunha F de Q, Alves-Filho JCF. Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus [Internet]. Journal of Infection. 2018 ; 77( 5): 391-397.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.jinf.2018.06.010
Artigo de periodico
Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells (2016)
Carregaro, Vanessa; Napimoga, Marcelo H; Peres, Raphael S; Benevides, Luciana; Sacramento, Laís Amorim; Pinto, Larissa G; Grespan, Renata; Cunha, Thiago Mattar; Silva, João Santana da; Cunha, Fernando de Queiroz
Source: Mediators of Inflammation. Unidade: FMRP
Subjects: ARTRITE, PROSTAGLANDINAS, FATORES DE TRANSCRIÇÃO, RECEPTORES, INTERLEUCINAS
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ABNT
CARREGARO, Vanessa et al. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, v. 2016, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/9626427. Acesso em: 17 nov. 2024.
APA
Carregaro, V., Napimoga, M. H., Peres, R. S., Benevides, L., Sacramento, L. A., Pinto, L. G., et al. (2016). Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, 2016. doi:10.1155/2016/9626427
NLM
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 nov. 17 ] Available from: https://doi.org/10.1155/2016/9626427
Vancouver
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 nov. 17 ] Available from: https://doi.org/10.1155/2016/9626427
Artigo de periodico
A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli (2012)
Soares, Denis de Melo; Figueiredo, Maria Jose; Martins, Juliano Manvailer; Machado, Renes de Resende; Sorgi, Carlos Arterio; Faccioli, Lucia Helena; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Souza, Glória Emília Petto de
Source: Medical Microbiology and Immunology. Unidades: FCFRP, FMRP
Subjects: ESCHERICHIA COLI, FEBRE, CITOCINAS, INFECÇÕES BACTERIANAS
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ABNT
SOARES, Denis de Melo et al. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli. Medical Microbiology and Immunology, v. 201, n. 1, p. 47-60, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00430-011-0204-3. Acesso em: 17 nov. 2024.
APA
Soares, D. de M., Figueiredo, M. J., Martins, J. M., Machado, R. de R., Sorgi, C. A., Faccioli, L. H., et al. (2012). A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli. Medical Microbiology and Immunology, 201( 1), 47-60. doi:10.1007/s00430-011-0204-3
NLM
Soares D de M, Figueiredo MJ, Martins JM, Machado R de R, Sorgi CA, Faccioli LH, Alves-Filho JC, Cunha F de Q, Souza GEP de. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli [Internet]. Medical Microbiology and Immunology. 2012 ; 201( 1): 47-60.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s00430-011-0204-3
Vancouver
Soares D de M, Figueiredo MJ, Martins JM, Machado R de R, Sorgi CA, Faccioli LH, Alves-Filho JC, Cunha F de Q, Souza GEP de. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli [Internet]. Medical Microbiology and Immunology. 2012 ; 201( 1): 47-60.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s00430-011-0204-3
Artigo de periodico
Inhibition of iNOS induces antidepressant-like effects in mice: pharmacological and genetic evidence (2012)
Montezuma, Karina; Biojone, Caroline; Lisboa, Sabrina Francesca; Cunha, Fernando de Queiroz; Guimarães, Francisco Silveira; Joca, Sâmia Regiane Lourenço
Source: Neuropharmacology. Unidades: FMRP, FCFRP
Subjects: ÓXIDO NÍTRICO, DEPRESSÃO, ESTRESSE, ANTIDEPRESSIVOS
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ABNT
MONTEZUMA, Karina et al. Inhibition of iNOS induces antidepressant-like effects in mice: pharmacological and genetic evidence. Neuropharmacology, v. 62, n. 1, p. 485-491, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2011.09.004. Acesso em: 17 nov. 2024.
APA
Montezuma, K., Biojone, C., Lisboa, S. F., Cunha, F. de Q., Guimarães, F. S., & Joca, S. R. L. (2012). Inhibition of iNOS induces antidepressant-like effects in mice: pharmacological and genetic evidence. Neuropharmacology, 62( 1), 485-491. doi:10.1016/j.neuropharm.2011.09.004
NLM
Montezuma K, Biojone C, Lisboa SF, Cunha F de Q, Guimarães FS, Joca SRL. Inhibition of iNOS induces antidepressant-like effects in mice: pharmacological and genetic evidence [Internet]. Neuropharmacology. 2012 ; 62( 1): 485-491.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.neuropharm.2011.09.004
Vancouver
Montezuma K, Biojone C, Lisboa SF, Cunha F de Q, Guimarães FS, Joca SRL. Inhibition of iNOS induces antidepressant-like effects in mice: pharmacological and genetic evidence [Internet]. Neuropharmacology. 2012 ; 62( 1): 485-491.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.neuropharm.2011.09.004
Artigo de periodico
Protective effect of RC-3095, an antagonist of the gastrin-releasing peptide receptor, in experimental arthritis (2011)
Oliveira, P. G.; Grespan, Renata; Pinto, Larissa Garcia; Meurer, L.; Brenol, João Carlos Tavares; Roesler, Rafael; Schwartsmann, Gilberto; Cunha, Fernando de Queiroz; Xavier, R. M.
Source: Arthritis & Rheumatism. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS (EFEITOS), ARTRITE
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ABNT
OLIVEIRA, P. G. et al. Protective effect of RC-3095, an antagonist of the gastrin-releasing peptide receptor, in experimental arthritis. Arthritis & Rheumatism, v. 63, n. 10, p. 2956-2965, 2011Tradução . . Disponível em: https://doi.org/10.1002/art.30486. Acesso em: 17 nov. 2024.
APA
Oliveira, P. G., Grespan, R., Pinto, L. G., Meurer, L., Brenol, J. C. T., Roesler, R., et al. (2011). Protective effect of RC-3095, an antagonist of the gastrin-releasing peptide receptor, in experimental arthritis. Arthritis & Rheumatism, 63( 10), 2956-2965. doi:10.1002/art.30486
NLM
Oliveira PG, Grespan R, Pinto LG, Meurer L, Brenol JCT, Roesler R, Schwartsmann G, Cunha F de Q, Xavier RM. Protective effect of RC-3095, an antagonist of the gastrin-releasing peptide receptor, in experimental arthritis [Internet]. Arthritis & Rheumatism. 2011 ; 63( 10): 2956-2965.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1002/art.30486
Vancouver
Oliveira PG, Grespan R, Pinto LG, Meurer L, Brenol JCT, Roesler R, Schwartsmann G, Cunha F de Q, Xavier RM. Protective effect of RC-3095, an antagonist of the gastrin-releasing peptide receptor, in experimental arthritis [Internet]. Arthritis & Rheumatism. 2011 ; 63( 10): 2956-2965.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1002/art.30486
Artigo de periodico
Collagen induced arthritis (CIA) in mice features regulatory transcriptional network connecting major histocompatibility complex (MHC H2) with autoantigen genes in the thymus (2011)
Donate, Paula Barbim; Fornari, Thaís Arouca; Junta, Cristina Moraes; Magalhães, Danielle Aparecida Rosa de; Macedo, Cláudia; Cunha, Thiago Mattar; Nguyen, Catherine; Cunha, Fernando de Queiroz; Passos, Geraldo Aleixo Silva
Source: Immunobiology. Unidades: FMRP, FORP
Subjects: EXPRESSÃO GÊNICA, ARTRITE
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ABNT
DONATE, Paula Barbim et al. Collagen induced arthritis (CIA) in mice features regulatory transcriptional network connecting major histocompatibility complex (MHC H2) with autoantigen genes in the thymus. Immunobiology, v. 216, n. 5, p. 591-603, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2010.09.007. Acesso em: 17 nov. 2024.
APA
Donate, P. B., Fornari, T. A., Junta, C. M., Magalhães, D. A. R. de, Macedo, C., Cunha, T. M., et al. (2011). Collagen induced arthritis (CIA) in mice features regulatory transcriptional network connecting major histocompatibility complex (MHC H2) with autoantigen genes in the thymus. Immunobiology, 216( 5), 591-603. doi:10.1016/j.imbio.2010.09.007
NLM
Donate PB, Fornari TA, Junta CM, Magalhães DAR de, Macedo C, Cunha TM, Nguyen C, Cunha F de Q, Passos GAS. Collagen induced arthritis (CIA) in mice features regulatory transcriptional network connecting major histocompatibility complex (MHC H2) with autoantigen genes in the thymus [Internet]. Immunobiology. 2011 ; 216( 5): 591-603.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.imbio.2010.09.007
Vancouver
Donate PB, Fornari TA, Junta CM, Magalhães DAR de, Macedo C, Cunha TM, Nguyen C, Cunha F de Q, Passos GAS. Collagen induced arthritis (CIA) in mice features regulatory transcriptional network connecting major histocompatibility complex (MHC H2) with autoantigen genes in the thymus [Internet]. Immunobiology. 2011 ; 216( 5): 591-603.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.imbio.2010.09.007
Artigo de periodico
Inhibition of hydrogen sulphide formation reduces cisplatin-induced renal damage (2011)
Francescato, Heloísa Della Coletta; Cunha, Fernando de Queiroz; Costa, Roberto Silva; Barbosa Júnior, Fernando; Boim, Mirian Aparecida; Arnoni, Carine Prisco; Silva, Cleonice Giovanni Alves da; Coimbra, Terezila Machado
Source: Nephrology, Dialysis, Transplantation. Unidades: FMRP, FCFRP
Subjects: INSUFICIÊNCIA RENAL CRÔNICA, INFLAMAÇÃO
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ABNT
FRANCESCATO, Heloísa Della Coletta et al. Inhibition of hydrogen sulphide formation reduces cisplatin-induced renal damage. Nephrology, Dialysis, Transplantation, v. 26, n. 2, p. 479-488, 2011Tradução . . Disponível em: https://doi.org/10.1093/ndt/gfq447. Acesso em: 17 nov. 2024.
APA
Francescato, H. D. C., Cunha, F. de Q., Costa, R. S., Barbosa Júnior, F., Boim, M. A., Arnoni, C. P., et al. (2011). Inhibition of hydrogen sulphide formation reduces cisplatin-induced renal damage. Nephrology, Dialysis, Transplantation, 26( 2), 479-488. doi:10.1093/ndt/gfq447
NLM
Francescato HDC, Cunha F de Q, Costa RS, Barbosa Júnior F, Boim MA, Arnoni CP, Silva CGA da, Coimbra TM. Inhibition of hydrogen sulphide formation reduces cisplatin-induced renal damage [Internet]. Nephrology, Dialysis, Transplantation. 2011 ; 26( 2): 479-488.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1093/ndt/gfq447
Vancouver
Francescato HDC, Cunha F de Q, Costa RS, Barbosa Júnior F, Boim MA, Arnoni CP, Silva CGA da, Coimbra TM. Inhibition of hydrogen sulphide formation reduces cisplatin-induced renal damage [Internet]. Nephrology, Dialysis, Transplantation. 2011 ; 26( 2): 479-488.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1093/ndt/gfq447
Artigo de periodico
Role of pratelet-activating factor in the pathogenesis of 5-fluorouracil-induced intestinal mucositis in mice (2011)
Soares, Pedro Marcos G.; Lima Junior, Roberto César P.; Mota, José Maurício S. C.; Justino, Priscilla F. C.; Brito, Gerly Anne C.; Ribeiro, Ronaldo de Albuquerque; Cunha, Fernando de Queiroz; Souza, Marcellus Henrique Loiola Ponte de
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: CITOCINAS, SISTEMA GASTROINTESTINAL
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ABNT
SOARES, Pedro Marcos G. et al. Role of pratelet-activating factor in the pathogenesis of 5-fluorouracil-induced intestinal mucositis in mice. Cancer Chemotherapy and Pharmacology, v. 68, n. 3, p. 713-720, 2011Tradução . . Disponível em: https://doi.org/10.1007/s00280-010-1540-5. Acesso em: 17 nov. 2024.
APA
Soares, P. M. G., Lima Junior, R. C. P., Mota, J. M. S. C., Justino, P. F. C., Brito, G. A. C., Ribeiro, R. de A., et al. (2011). Role of pratelet-activating factor in the pathogenesis of 5-fluorouracil-induced intestinal mucositis in mice. Cancer Chemotherapy and Pharmacology, 68( 3), 713-720. doi:10.1007/s00280-010-1540-5
NLM
Soares PMG, Lima Junior RCP, Mota JMSC, Justino PFC, Brito GAC, Ribeiro R de A, Cunha F de Q, Souza MHLP de. Role of pratelet-activating factor in the pathogenesis of 5-fluorouracil-induced intestinal mucositis in mice [Internet]. Cancer Chemotherapy and Pharmacology. 2011 ; 68( 3): 713-720.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s00280-010-1540-5
Vancouver
Soares PMG, Lima Junior RCP, Mota JMSC, Justino PFC, Brito GAC, Ribeiro R de A, Cunha F de Q, Souza MHLP de. Role of pratelet-activating factor in the pathogenesis of 5-fluorouracil-induced intestinal mucositis in mice [Internet]. Cancer Chemotherapy and Pharmacology. 2011 ; 68( 3): 713-720.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s00280-010-1540-5
Artigo de periodico
Agglutinin isolated from the red marine alga Hypnea cervicornis J. Agardh reduces inflammatory hypernociception: involvement of nitric oxide (2010)
Figueiredo, Jozi Godoy; Bitencourt, Flávio da Silveira; Cunha, Thiago Mattar; Luz, Patrícia Bastos; Nascimento, Kyria S.; Mota, Mário Rogério Lima; Sampaio, Alexandre Holanda; Cavada, Benildo Sousa; Cunha, Fernando de Queiroz; Alencar, Nylane Maria Nunes de
Source: Pharmacology, Biochemistry and Behavior. Unidade: FMRP
Subjects: CITOCINAS, INFLAMAÇÃO, PEROXIDASE, ÓXIDO NÍTRICO
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ABNT
FIGUEIREDO, Jozi Godoy et al. Agglutinin isolated from the red marine alga Hypnea cervicornis J. Agardh reduces inflammatory hypernociception: involvement of nitric oxide. Pharmacology, Biochemistry and Behavior, v. 96, n. 4, p. 271-377, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2010.06.008. Acesso em: 17 nov. 2024.
APA
Figueiredo, J. G., Bitencourt, F. da S., Cunha, T. M., Luz, P. B., Nascimento, K. S., Mota, M. R. L., et al. (2010). Agglutinin isolated from the red marine alga Hypnea cervicornis J. Agardh reduces inflammatory hypernociception: involvement of nitric oxide. Pharmacology, Biochemistry and Behavior, 96( 4), 271-377. doi:10.1016/j.pbb.2010.06.008
NLM
Figueiredo JG, Bitencourt F da S, Cunha TM, Luz PB, Nascimento KS, Mota MRL, Sampaio AH, Cavada BS, Cunha F de Q, Alencar NMN de. Agglutinin isolated from the red marine alga Hypnea cervicornis J. Agardh reduces inflammatory hypernociception: involvement of nitric oxide [Internet]. Pharmacology, Biochemistry and Behavior. 2010 ; 96( 4): 271-377.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.pbb.2010.06.008
Vancouver
Figueiredo JG, Bitencourt F da S, Cunha TM, Luz PB, Nascimento KS, Mota MRL, Sampaio AH, Cavada BS, Cunha F de Q, Alencar NMN de. Agglutinin isolated from the red marine alga Hypnea cervicornis J. Agardh reduces inflammatory hypernociception: involvement of nitric oxide [Internet]. Pharmacology, Biochemistry and Behavior. 2010 ; 96( 4): 271-377.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.pbb.2010.06.008
Artigo de periodico
Routine abdominal drains after Roux-en-Y gastric bypass: a prospective evaluation of the inflammatory response (2010)
Salgado Júnior, Wilson; Cunha, Fernando de Queiroz; Santos, José Sebastião dos; Nonino-Borges, Carla Barbosa; Sankarankutty, Ajith Kumar; Castro e Silva Júnior, Orlando de; Ceneviva, Reginaldo
Source: Surgery for Obesity and Related Diseases. Unidade: FMRP
Subjects: INFLAMAÇÃO, CIRURGIA BARIÁTRICA, COMPLICAÇÕES PÓS-OPERATÓRIAS
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ABNT
SALGADO JÚNIOR, Wilson et al. Routine abdominal drains after Roux-en-Y gastric bypass: a prospective evaluation of the inflammatory response. Surgery for Obesity and Related Diseases, v. 6, n. 6, p. 648-652, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.soard.2009.09.017. Acesso em: 17 nov. 2024.
APA
Salgado Júnior, W., Cunha, F. de Q., Santos, J. S. dos, Nonino-Borges, C. B., Sankarankutty, A. K., Castro e Silva Júnior, O. de, & Ceneviva, R. (2010). Routine abdominal drains after Roux-en-Y gastric bypass: a prospective evaluation of the inflammatory response. Surgery for Obesity and Related Diseases, 6( 6), 648-652. doi:10.1016/j.soard.2009.09.017
NLM
Salgado Júnior W, Cunha F de Q, Santos JS dos, Nonino-Borges CB, Sankarankutty AK, Castro e Silva Júnior O de, Ceneviva R. Routine abdominal drains after Roux-en-Y gastric bypass: a prospective evaluation of the inflammatory response [Internet]. Surgery for Obesity and Related Diseases. 2010 ; 6( 6): 648-652.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.soard.2009.09.017
Vancouver
Salgado Júnior W, Cunha F de Q, Santos JS dos, Nonino-Borges CB, Sankarankutty AK, Castro e Silva Júnior O de, Ceneviva R. Routine abdominal drains after Roux-en-Y gastric bypass: a prospective evaluation of the inflammatory response [Internet]. Surgery for Obesity and Related Diseases. 2010 ; 6( 6): 648-652.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.soard.2009.09.017
Artigo de periodico
Gastroprotective effect of heme-oxygenase 1/biliverdin/CO pathway in ethanol-induced gastric damage in mice (2010)
Gomes, Antoniella Souza; Gadelha, Gemima G.; Lima, Samara J.; Garcia, Joyce A.; Medeiros, Jand Venes Rolim; Havt, Alexandre; Lima, Aldo A.; Ribeiro, Ronaldo de Albuquerque; Brito, Gerly Anne C.; Cunha, Fernando de Queiroz; Souza, Marcellus Henrique Loiola Ponte de
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: MUCOSA GÁSTRICA, ESTÔMAGO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOMES, Antoniella Souza et al. Gastroprotective effect of heme-oxygenase 1/biliverdin/CO pathway in ethanol-induced gastric damage in mice. European Journal of Pharmacology, v. 642, n. 1-3, p. 140-145, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2010.05.023. Acesso em: 17 nov. 2024.
APA
Gomes, A. S., Gadelha, G. G., Lima, S. J., Garcia, J. A., Medeiros, J. V. R., Havt, A., et al. (2010). Gastroprotective effect of heme-oxygenase 1/biliverdin/CO pathway in ethanol-induced gastric damage in mice. European Journal of Pharmacology, 642( 1-3), 140-145. doi:10.1016/j.ejphar.2010.05.023
NLM
Gomes AS, Gadelha GG, Lima SJ, Garcia JA, Medeiros JVR, Havt A, Lima AA, Ribeiro R de A, Brito GAC, Cunha F de Q, Souza MHLP de. Gastroprotective effect of heme-oxygenase 1/biliverdin/CO pathway in ethanol-induced gastric damage in mice [Internet]. European Journal of Pharmacology. 2010 ; 642( 1-3): 140-145.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2010.05.023
Vancouver
Gomes AS, Gadelha GG, Lima SJ, Garcia JA, Medeiros JVR, Havt A, Lima AA, Ribeiro R de A, Brito GAC, Cunha F de Q, Souza MHLP de. Gastroprotective effect of heme-oxygenase 1/biliverdin/CO pathway in ethanol-induced gastric damage in mice [Internet]. European Journal of Pharmacology. 2010 ; 642( 1-3): 140-145.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2010.05.023
Artigo de periodico
Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis (2010)
Celes, Mara Rúbia Nunes; Malvestio, Lygia M.; Torres-Dueñas, Diego; Blefari, Valdecir; Campos, Érica Carolina; Ramos, Simone Gusmão; Prado, Cibele Maria; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Source: Laboratory Investigation. Unidade: FMRP
Subjects: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, Mara Rúbia Nunes et al. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, v. 90, n. 4, p. 531-542, 2010Tradução . . Disponível em: https://doi.org/10.1186/cc9157. Acesso em: 17 nov. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Torres-Dueñas, D., Blefari, V., Campos, É. C., Ramos, S. G., et al. (2010). Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, 90( 4), 531-542. doi:10.1186/cc9157
NLM
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1186/cc9157
Vancouver
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1186/cc9157
Artigo de periodico
An anti-inflammatory lectin from Luetzelburgia auriculata seeds inhibits adhesion and rolling of leukocytes and modulates histamine and PGE2 action in acute inflammation models (2010)
Alencar, Nylane Maria Nunes de; Oliveira, Raquel Sombra Basílio de; Figueiredo, Jozi Godoy; Cavalcante, Italo José Mesquita; Matos, Mayara Patrícia Viana de; Cunha, Fernando de Queiroz; Nunes, J. V. S.; Bomfim, Liezelotte Rezende; Ramos, Márcio Viana
Source: Inflammation Reserch. Unidade: FMRP
Subjects: LEUCÓCITOS, ANTI-INFLAMATÓRIOS NÃO ESTEROIDES, LECTINAS DE PLANTAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALENCAR, Nylane Maria Nunes de et al. An anti-inflammatory lectin from Luetzelburgia auriculata seeds inhibits adhesion and rolling of leukocytes and modulates histamine and PGE2 action in acute inflammation models. Inflammation Reserch, v. 59, n. 4, p. 245-254, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00011-009-0092-9. Acesso em: 17 nov. 2024.
APA
Alencar, N. M. N. de, Oliveira, R. S. B. de, Figueiredo, J. G., Cavalcante, I. J. M., Matos, M. P. V. de, Cunha, F. de Q., et al. (2010). An anti-inflammatory lectin from Luetzelburgia auriculata seeds inhibits adhesion and rolling of leukocytes and modulates histamine and PGE2 action in acute inflammation models. Inflammation Reserch, 59( 4), 245-254. doi:10.1007/s00011-009-0092-9
NLM
Alencar NMN de, Oliveira RSB de, Figueiredo JG, Cavalcante IJM, Matos MPV de, Cunha F de Q, Nunes JVS, Bomfim LR, Ramos MV. An anti-inflammatory lectin from Luetzelburgia auriculata seeds inhibits adhesion and rolling of leukocytes and modulates histamine and PGE2 action in acute inflammation models [Internet]. Inflammation Reserch. 2010 ; 59( 4): 245-254.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s00011-009-0092-9
Vancouver
Alencar NMN de, Oliveira RSB de, Figueiredo JG, Cavalcante IJM, Matos MPV de, Cunha F de Q, Nunes JVS, Bomfim LR, Ramos MV. An anti-inflammatory lectin from Luetzelburgia auriculata seeds inhibits adhesion and rolling of leukocytes and modulates histamine and PGE2 action in acute inflammation models [Internet]. Inflammation Reserch. 2010 ; 59( 4): 245-254.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s00011-009-0092-9
Artigo de periodico
Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis (2010)
Nascimento, Daniele C.; Alves-Filho, José Carlos; Sônego, Fabiane; Fukada, Sandra Yasuyo; Pereira, Marcelo S.; Benjamim, Cláudia; Zamboni, Dario Simões; Silva, João Santana da; Cunha, Fernando de Queiroz
Source: Critical Care Medicine. Unidade: FMRP
Subjects: SEPSE, FÁRMACOS IMUNOSSUPRESSORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Daniele C. et al. Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis. Critical Care Medicine, v. 38, n. 8, p. 1718-1725, 2010Tradução . . Disponível em: https://doi.org/10.1097/ccm.0b013e3181e78ad0. Acesso em: 17 nov. 2024.
APA
Nascimento, D. C., Alves-Filho, J. C., Sônego, F., Fukada, S. Y., Pereira, M. S., Benjamim, C., et al. (2010). Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis. Critical Care Medicine, 38( 8), 1718-1725. doi:10.1097/ccm.0b013e3181e78ad0
NLM
Nascimento DC, Alves-Filho JC, Sônego F, Fukada SY, Pereira MS, Benjamim C, Zamboni DS, Silva JS da, Cunha F de Q. Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis [Internet]. Critical Care Medicine. 2010 ; 38( 8): 1718-1725.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1097/ccm.0b013e3181e78ad0
Vancouver
Nascimento DC, Alves-Filho JC, Sônego F, Fukada SY, Pereira MS, Benjamim C, Zamboni DS, Silva JS da, Cunha F de Q. Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis [Internet]. Critical Care Medicine. 2010 ; 38( 8): 1718-1725.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1097/ccm.0b013e3181e78ad0
Artigo de periodico
Interleukin-17 and kidney allograft outcome (2009)
Crispim, Janaína Cristiana de Oliveira; Palomino, Gustavo Martelli; Rassi, Diane Meyre; Costa, Roberto Silva; Saber, L. T.; Cunha, Fernando de Queiroz; Donadi, Eduardo Antônio; Grespan, Renata
Source: Transplantation Proceedings. Unidade: FMRP
Subjects: TRANSPLANTE DE RIM, FISIOPATOLOGIA, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CRISPIM, Janaína Cristiana de Oliveira et al. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings, v. 41, n. 5, p. 1562-1564, 2009Tradução . . Acesso em: 17 nov. 2024.
APA
Crispim, J. C. de O., Palomino, G. M., Rassi, D. M., Costa, R. S., Saber, L. T., Cunha, F. de Q., et al. (2009). Interleukin-17 and kidney allograft outcome. Transplantation Proceedings, 41( 5), 1562-1564.
NLM
Crispim JC de O, Palomino GM, Rassi DM, Costa RS, Saber LT, Cunha F de Q, Donadi EA, Grespan R. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings. 2009 ; 41( 5): 1562-1564.[citado 2024 nov. 17 ]
Vancouver
Crispim JC de O, Palomino GM, Rassi DM, Costa RS, Saber LT, Cunha F de Q, Donadi EA, Grespan R. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings. 2009 ; 41( 5): 1562-1564.[citado 2024 nov. 17 ]
Artigo de periodico
The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron (2009)
Villarreal, Cristiane Flora; Sachs, Daniela; Funez, Mani Indiana; Parada, Carlos Amílcar; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: FARMACOLOGIA, DOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VILLARREAL, Cristiane Flora et al. The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron. Biochemical Pharmacology, v. 77, n. 5, p. 867-877, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2008.11.015. Acesso em: 17 nov. 2024.
APA
Villarreal, C. F., Sachs, D., Funez, M. I., Parada, C. A., Cunha, F. de Q., & Ferreira, S. H. (2009). The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron. Biochemical Pharmacology, 77( 5), 867-877. doi:10.1016/j.bcp.2008.11.015
NLM
Villarreal CF, Sachs D, Funez MI, Parada CA, Cunha F de Q, Ferreira SH. The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron [Internet]. Biochemical Pharmacology. 2009 ; 77( 5): 867-877.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.bcp.2008.11.015
Vancouver
Villarreal CF, Sachs D, Funez MI, Parada CA, Cunha F de Q, Ferreira SH. The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron [Internet]. Biochemical Pharmacology. 2009 ; 77( 5): 867-877.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.bcp.2008.11.015
Artigo de periodico
Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production (2009)
Lemos, Henrique de Paula; Grespan, Renata; Vieira, Sílvio Manfredo; Cunha, Thiago Mattar; Verri Júnior, Waldiceu Aparecido; Fernandes, Karla Simone S.; Souto, Fabrício Oliveira; Mclnnes, Iain B.; Ferreira, Sérgio Henrique; Liew, Foo Y.; Cunha, Fernando de Queiroz
Source: Proceedings of the Nacional Academy of Sciences. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, CITOCINAS, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEMOS, Henrique de Paula et al. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production. Proceedings of the Nacional Academy of Sciences, v. 106, n. 14, p. 5954-5959, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0812782106. Acesso em: 17 nov. 2024.
APA
Lemos, H. de P., Grespan, R., Vieira, S. M., Cunha, T. M., Verri Júnior, W. A., Fernandes, K. S. S., et al. (2009). Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production. Proceedings of the Nacional Academy of Sciences, 106( 14), 5954-5959. doi:10.1073/pnas.0812782106
NLM
Lemos H de P, Grespan R, Vieira SM, Cunha TM, Verri Júnior WA, Fernandes KSS, Souto FO, Mclnnes IB, Ferreira SH, Liew FY, Cunha F de Q. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production [Internet]. Proceedings of the Nacional Academy of Sciences. 2009 ; 106( 14): 5954-5959.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1073/pnas.0812782106
Vancouver
Lemos H de P, Grespan R, Vieira SM, Cunha TM, Verri Júnior WA, Fernandes KSS, Souto FO, Mclnnes IB, Ferreira SH, Liew FY, Cunha F de Q. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production [Internet]. Proceedings of the Nacional Academy of Sciences. 2009 ; 106( 14): 5954-5959.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1073/pnas.0812782106
Artigo de periodico
The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception (2009)
Sachs, Daniela; Villarreal, Cristiane Floral; Cunha, Fernando de Queiroz; Parada, Carlos Amílcar; Ferreira, Sérgio Henrique
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: PROTEÍNAS QUINASES, DOR, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SACHS, Daniela et al. The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology, v. 156, n. 5, p. 826-834, 2009Tradução . . Acesso em: 17 nov. 2024.
APA
Sachs, D., Villarreal, C. F., Cunha, F. de Q., Parada, C. A., & Ferreira, S. H. (2009). The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology, 156( 5), 826-834.
NLM
Sachs D, Villarreal CF, Cunha F de Q, Parada CA, Ferreira SH. The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology. 2009 ; 156( 5): 826-834.[citado 2024 nov. 17 ]
Vancouver
Sachs D, Villarreal CF, Cunha F de Q, Parada CA, Ferreira SH. The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology. 2009 ; 156( 5): 826-834.[citado 2024 nov. 17 ]

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