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Artigo de periodico
The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex (2022)
Katayama, Pedro L.; Leirão, Isabela P.; Kanashiro, Alexandre; Luiz, João Paulo Mesquita ; Cunha, Fernando de Queiroz ; Navegantes, Luiz Carlos Carvalho ; Menani, Jose Vanderlei; Zoccal, Daniel Breseghello; Colombari, Debora Simoes de Almeida ; Colombari, Eduardo
Source: Brain, Behavior, and Immunity. Unidade: FMRP
Subjects: SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, ANTI-INFLAMATÓRIOS
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ABNT
KATAYAMA, Pedro L. et al. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, v. 102, p. 370-386, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2022.03.014. Acesso em: 09 nov. 2024.
APA
Katayama, P. L., Leirão, I. P., Kanashiro, A., Luiz, J. P. M., Cunha, F. de Q., Navegantes, L. C. C., et al. (2022). The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, 102, 370-386. doi:10.1016/j.bbi.2022.03.014
NLM
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Vancouver
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Artigo de periodico
Physiological sympathetic activation reduces systemic inflammation: role of baroreflex and chemoreflex (2021)
Brognara, Fernanda ; Castania, Jaci Airton; Kanashiro, Alexandre; Dias, Daniel Penteado Martins; Salgado, Hélio César
Source: Frontiers in Immunology. Unidades: FMRP, EERP
Subjects: SISTEMA NERVOSO AUTÔNOMO, PRESSORECEPTORES, PRESSÃO SANGUÍNEA, INFLAMAÇÃO, INTERLEUCINA 10, SISTEMA NERVOSO SIMPÁTICO
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ABNT
BROGNARA, Fernanda et al. Physiological sympathetic activation reduces systemic inflammation: role of baroreflex and chemoreflex. Frontiers in Immunology, v. 12, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.637845. Acesso em: 09 nov. 2024.
APA
Brognara, F., Castania, J. A., Kanashiro, A., Dias, D. P. M., & Salgado, H. C. (2021). Physiological sympathetic activation reduces systemic inflammation: role of baroreflex and chemoreflex. Frontiers in Immunology, 12, 1-12. doi:10.3389/fimmu.2021.637845
NLM
Brognara F, Castania JA, Kanashiro A, Dias DPM, Salgado HC. Physiological sympathetic activation reduces systemic inflammation: role of baroreflex and chemoreflex [Internet]. Frontiers in Immunology. 2021 ; 12 1-12.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2021.637845
Vancouver
Brognara F, Castania JA, Kanashiro A, Dias DPM, Salgado HC. Physiological sympathetic activation reduces systemic inflammation: role of baroreflex and chemoreflex [Internet]. Frontiers in Immunology. 2021 ; 12 1-12.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2021.637845
Artigo de periodico
Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats (2020)
Passaglia, Patrícia; Faim, Felipe de Lima; Batalhão, Marcelo Eduardo; Bendhack, Lusiane Maria ; Antunes-Rodrigues, José; Ulloa, Luis; Kanashiro, Alexandre; Cárnio, Evelin Capellari
Source: Brain, Behavior, and Immunity. Unidades: EERP, FCFRP, FMRP
Subjects: ANGIOTENSINAS, LIPOPOLISSACARÍDEOS, SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, HIPOTENSÃO
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ABNT
PASSAGLIA, Patrícia et al. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats. Brain, Behavior, and Immunity, v. 88, p. 606-618, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2020.04.059. Acesso em: 09 nov. 2024.
APA
Passaglia, P., Faim, F. de L., Batalhão, M. E., Bendhack, L. M., Antunes-Rodrigues, J., Ulloa, L., et al. (2020). Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats. Brain, Behavior, and Immunity, 88, 606-618. doi:10.1016/j.bbi.2020.04.059
NLM
Passaglia P, Faim F de L, Batalhão ME, Bendhack LM, Antunes-Rodrigues J, Ulloa L, Kanashiro A, Cárnio EC. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats [Internet]. Brain, Behavior, and Immunity. 2020 ; 88 606-618.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbi.2020.04.059
Vancouver
Passaglia P, Faim F de L, Batalhão ME, Bendhack LM, Antunes-Rodrigues J, Ulloa L, Kanashiro A, Cárnio EC. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats [Internet]. Brain, Behavior, and Immunity. 2020 ; 88 606-618.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbi.2020.04.059
Artigo de periodico
Time course of hemodynamic responses to different doses of lipopolysaccharide in unanesthetized male rats (2019)
Brognara, Fernanda ; Castania, Jaci Airton; Dias, Daniel Penteado Martins; Kanashiro, Alexandre ; Salgado, Hélio César
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: HEMODINÂMICA, POLISSACARÍDEOS, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA
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ABNT
BROGNARA, Fernanda et al. Time course of hemodynamic responses to different doses of lipopolysaccharide in unanesthetized male rats. Frontiers in Physiology, v. 10, p. art. 771 [8] , 2019Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.00771. Acesso em: 09 nov. 2024.
APA
Brognara, F., Castania, J. A., Dias, D. P. M., Kanashiro, A., & Salgado, H. C. (2019). Time course of hemodynamic responses to different doses of lipopolysaccharide in unanesthetized male rats. Frontiers in Physiology, 10, art. 771 [8] . doi:10.3389/fphys.2019.00771
NLM
Brognara F, Castania JA, Dias DPM, Kanashiro A, Salgado HC. Time course of hemodynamic responses to different doses of lipopolysaccharide in unanesthetized male rats [Internet]. Frontiers in Physiology. 2019 ; 10 art. 771 [8] .[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fphys.2019.00771
Vancouver
Brognara F, Castania JA, Dias DPM, Kanashiro A, Salgado HC. Time course of hemodynamic responses to different doses of lipopolysaccharide in unanesthetized male rats [Internet]. Frontiers in Physiology. 2019 ; 10 art. 771 [8] .[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fphys.2019.00771
Artigo de periodico
The atypical chemokine receptor ACKR2 is protective against sepsis (2018)
Castanheira, Fernanda Vargas e Silva; Borges, Vanessa de Fátima; Sônego, Fabiane; Kanashiro, Alexandre ; Donate, Paula Barbim; Melo, Paulo Henrique de; Pallas, Kenneth; Russo, Remo C.; Amaral, Flávio A.; Teixeira, Mauro M.; Ramalho, Fernando Silva ; Cunha, Thiago Mattar ; Liew, Foo Y.; Alves Filho, José Carlos Farias ; Graham, Gerard J.; Cunha, Fernando de Queiroz
Source: SHOCK. Unidade: FMRP
Subjects: SEPSE, QUIMIOTERAPIA
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ABNT
CASTANHEIRA, Fernanda Vargas e Silva et al. The atypical chemokine receptor ACKR2 is protective against sepsis. SHOCK, v. 49, n. 6, p. 682-689, 2018Tradução . . Disponível em: https://doi.org/10.1097/shk.0000000000000969. Acesso em: 09 nov. 2024.
APA
Castanheira, F. V. e S., Borges, V. de F., Sônego, F., Kanashiro, A., Donate, P. B., Melo, P. H. de, et al. (2018). The atypical chemokine receptor ACKR2 is protective against sepsis. SHOCK, 49( 6), 682-689. doi:10.1097/shk.0000000000000969
NLM
Castanheira FV e S, Borges V de F, Sônego F, Kanashiro A, Donate PB, Melo PH de, Pallas K, Russo RC, Amaral FA, Teixeira MM, Ramalho FS, Cunha TM, Liew FY, Alves Filho JCF, Graham GJ, Cunha F de Q. The atypical chemokine receptor ACKR2 is protective against sepsis [Internet]. SHOCK. 2018 ; 49( 6): 682-689.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/shk.0000000000000969
Vancouver
Castanheira FV e S, Borges V de F, Sônego F, Kanashiro A, Donate PB, Melo PH de, Pallas K, Russo RC, Amaral FA, Teixeira MM, Ramalho FS, Cunha TM, Liew FY, Alves Filho JCF, Graham GJ, Cunha F de Q. The atypical chemokine receptor ACKR2 is protective against sepsis [Internet]. SHOCK. 2018 ; 49( 6): 682-689.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/shk.0000000000000969
Artigo de periodico
Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation (2018)
Kanashiro, Alexandre; Franchin, Marcelo; Bassi, Gabriel Shimizu; Santana, Dênis Augusto Reis; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Ulloa, Luis; Rodrigues, Gerson Jonathan
Source: Fundamental and Clinical Pharmacology. Unidade: FMRP
Subjects: ARTRITE, RECEPTORES ANDROGÊNICOS, SISTEMA NERVOSO PARASSIMPÁTICO, NEUTRÓFILOS
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ABNT
KANASHIRO, Alexandre et al. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, v. 32, n. 2, p. 155-162, 2018Tradução . . Disponível em: https://doi.org/10.1111/fcp.12338. Acesso em: 09 nov. 2024.
APA
Kanashiro, A., Franchin, M., Bassi, G. S., Santana, D. A. R., Cunha, T. M., Cunha, F. de Q., et al. (2018). Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, 32( 2), 155-162. doi:10.1111/fcp.12338
NLM
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1111/fcp.12338
Vancouver
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1111/fcp.12338
Artigo de periodico
Baroreflex stimulation attenuates central but not peripheral inflammation in conscious endotoxemic rats (2018)
Brognara, Fernanda; Castania, Jaci Airton; Dias, Daniel P. M.; Lopes, Alexandre H.; Fazan Júnior, Rubens; Kanashiro, Alexandre; Ulloa, Luis; Salgado, Hélio César
Source: Brain Research. Unidade: FMRP
Subjects: AORTA, BARORREFLEXO, HIPOTÁLAMO, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, NEUROIMUNOMODULAÇÃO, PRESSÃO SANGUÍNEA
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ABNT
BROGNARA, Fernanda et al. Baroreflex stimulation attenuates central but not peripheral inflammation in conscious endotoxemic rats. Brain Research, v. 1682, p. 54-60, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2018.01.003. Acesso em: 09 nov. 2024.
APA
Brognara, F., Castania, J. A., Dias, D. P. M., Lopes, A. H., Fazan Júnior, R., Kanashiro, A., et al. (2018). Baroreflex stimulation attenuates central but not peripheral inflammation in conscious endotoxemic rats. Brain Research, 1682, 54-60. doi:10.1016/j.brainres.2018.01.003
NLM
Brognara F, Castania JA, Dias DPM, Lopes AH, Fazan Júnior R, Kanashiro A, Ulloa L, Salgado HC. Baroreflex stimulation attenuates central but not peripheral inflammation in conscious endotoxemic rats [Internet]. Brain Research. 2018 ; 1682 54-60.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.brainres.2018.01.003
Vancouver
Brognara F, Castania JA, Dias DPM, Lopes AH, Fazan Júnior R, Kanashiro A, Ulloa L, Salgado HC. Baroreflex stimulation attenuates central but not peripheral inflammation in conscious endotoxemic rats [Internet]. Brain Research. 2018 ; 1682 54-60.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.brainres.2018.01.003
Artigo de periodico
The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 (2018)
Sônego, Fabiane; Castanheira, Fernanda V. S.; Horta, Catarina V.; Kanashiro, Alexandre; Czaikoski, Paula G.; Zamboni, Dario Simões; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: PNEUMONIA, NEUTRÓFILOS, QUIMIOCINAS, BACTÉRIAS GRAM-NEGATIVAS
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ABNT
SÔNEGO, Fabiane et al. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88. Inflammation Research, v. 67, n. 5, p. 435-443, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-018-1135-x. Acesso em: 09 nov. 2024.
APA
Sônego, F., Castanheira, F. V. S., Horta, C. V., Kanashiro, A., Czaikoski, P. G., Zamboni, D. S., et al. (2018). The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88. Inflammation Research, 67( 5), 435-443. doi:10.1007/s00011-018-1135-x
NLM
Sônego F, Castanheira FVS, Horta CV, Kanashiro A, Czaikoski PG, Zamboni DS, Alves Filho JCF, Cunha F de Q. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 [Internet]. Inflammation Research. 2018 ; 67( 5): 435-443.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00011-018-1135-x
Vancouver
Sônego F, Castanheira FVS, Horta CV, Kanashiro A, Czaikoski PG, Zamboni DS, Alves Filho JCF, Cunha F de Q. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 [Internet]. Inflammation Research. 2018 ; 67( 5): 435-443.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00011-018-1135-x
Artigo de periodico
Modulation of experimental arthritis by vagal sensory and central brain stimulation (2017)
Bassi, Gabriel Shimizu; Dias, Daniel Penteado Martins; Franchin, Marcelo; Talbot, Jhimmy; Reis, Daniel Gustavo; Menezes, Gustavo Batista; Castania, Jaci Airton; Garcia-Cairasco, Norberto; Resstel, Leonardo Barbosa Moraes; Salgado, Hélio César; Cunha, Fernando de Queiroz; Cunha, Thiago Mattar; Ulloa, Luis; Kanashiro, Alexandre
Source: Brain, Behavior, and Immunity. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, ANTAGONISTAS, SISTEMA NERVOSO SIMPÁTICO, ESTIMULAÇÃO ELÉTRICA, NERVO VAGO
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ABNT
BASSI, Gabriel Shimizu et al. Modulation of experimental arthritis by vagal sensory and central brain stimulation. Brain, Behavior, and Immunity, v. 64, p. 330-343, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2017.04.003. Acesso em: 09 nov. 2024.
APA
Bassi, G. S., Dias, D. P. M., Franchin, M., Talbot, J., Reis, D. G., Menezes, G. B., et al. (2017). Modulation of experimental arthritis by vagal sensory and central brain stimulation. Brain, Behavior, and Immunity, 64, 330-343. doi:10.1016/j.bbi.2017.04.003
NLM
Bassi GS, Dias DPM, Franchin M, Talbot J, Reis DG, Menezes GB, Castania JA, Garcia-Cairasco N, Resstel LBM, Salgado HC, Cunha F de Q, Cunha TM, Ulloa L, Kanashiro A. Modulation of experimental arthritis by vagal sensory and central brain stimulation [Internet]. Brain, Behavior, and Immunity. 2017 ; 64 330-343.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbi.2017.04.003
Vancouver
Bassi GS, Dias DPM, Franchin M, Talbot J, Reis DG, Menezes GB, Castania JA, Garcia-Cairasco N, Resstel LBM, Salgado HC, Cunha F de Q, Cunha TM, Ulloa L, Kanashiro A. Modulation of experimental arthritis by vagal sensory and central brain stimulation [Internet]. Brain, Behavior, and Immunity. 2017 ; 64 330-343.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbi.2017.04.003
Artigo de periodico
Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis (2017)
Souza, Maria C.; Fonseca, Denise Morais da ; Kanashiro, Alexandre; Benevides, Luciana; Medina, Tiago S.; Dias, Murilo S.; Andrade, Warrison A.; Bonfá, Giuliano; Silva, Marcondes A. B.; Gozzi, Aline; Borges, Marcos de Carvalho; Gazzinelli, Ricardo T.; Alves Filho, José Carlos Farias; Cunha, Fernando Queiróz; Silva, João Santana da
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: MODELOS ANIMAIS DE DOENÇAS, INTERFERONS, ÓXIDO NÍTRICO, SEPSE, TOXOPLASMOSE
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ABNT
SOUZA, Maria C. et al. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, v. 7, 2017Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2017.00116. Acesso em: 09 nov. 2024.
APA
Souza, M. C., Fonseca, D. M. da, Kanashiro, A., Benevides, L., Medina, T. S., Dias, M. S., et al. (2017). Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, 7. doi:10.3389/fcimb.2017.00116
NLM
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Vancouver
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Artigo de periodico
Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats (2017)
Santos-Junior, Nilton Nascimento; Costa, Luís Henrique Angenendt; Catalão, Carlos Henrique Rocha; Kanashiro, Alexandre; Sharshar, Tarek; Rocha, Maria José Alves da
Source: Pituitary. Unidade: FORP
Subjects: CITOCINAS, SEPSE, SISTEMA NERVOSO CENTRAL, VASOPRESSINAS, OCITOCINA
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ABNT
SANTOS-JUNIOR, Nilton Nascimento et al. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, v. 20, n. 5, p. 515-521, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11102-017-0812-z. Acesso em: 09 nov. 2024.
APA
Santos-Junior, N. N., Costa, L. H. A., Catalão, C. H. R., Kanashiro, A., Sharshar, T., & Rocha, M. J. A. da. (2017). Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, 20( 5), 515-521. doi:10.1007/s11102-017-0812-z
NLM
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Vancouver
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Artigo de periodico
Carotid sinus nerve electrical stimulation in conscious rats attenuates systemic inflammation via chemoreceptor activation (2017)
Santos-Almeida, Fernanda Machado; Domingos-Souza, Gean; Meschiari, César A.; Fávaro, Laura Campos; Becari, Christiane; Castania, Jaci Airton; Lopes, Alexandre; Cunha, Thiago Mattar; Moraes, Davi José de Almeida; Cunha, Fernando de Queiroz; Ulloa, Luis; Kanashiro, Alexandre; Tezini, Geisa C. S. V.; Salgado, Hélio César
Source: Scientific Reports. Unidade: FMRP
Subjects: SISTEMA NERVOSO AUTÔNOMO, ESTIMULAÇÃO ELÉTRICA, INFLAMAÇÃO
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ABNT
SANTOS-ALMEIDA, Fernanda Machado et al. Carotid sinus nerve electrical stimulation in conscious rats attenuates systemic inflammation via chemoreceptor activation. Scientific Reports, v. 7, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-06703-0. Acesso em: 09 nov. 2024.
APA
Santos-Almeida, F. M., Domingos-Souza, G., Meschiari, C. A., Fávaro, L. C., Becari, C., Castania, J. A., et al. (2017). Carotid sinus nerve electrical stimulation in conscious rats attenuates systemic inflammation via chemoreceptor activation. Scientific Reports, 7( 1). doi:10.1038/s41598-017-06703-0
NLM
Santos-Almeida FM, Domingos-Souza G, Meschiari CA, Fávaro LC, Becari C, Castania JA, Lopes A, Cunha TM, Moraes DJ de A, Cunha F de Q, Ulloa L, Kanashiro A, Tezini GCSV, Salgado HC. Carotid sinus nerve electrical stimulation in conscious rats attenuates systemic inflammation via chemoreceptor activation [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/s41598-017-06703-0
Vancouver
Santos-Almeida FM, Domingos-Souza G, Meschiari CA, Fávaro LC, Becari C, Castania JA, Lopes A, Cunha TM, Moraes DJ de A, Cunha F de Q, Ulloa L, Kanashiro A, Tezini GCSV, Salgado HC. Carotid sinus nerve electrical stimulation in conscious rats attenuates systemic inflammation via chemoreceptor activation [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/s41598-017-06703-0
Artigo de periodico
Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis (2016)
Bassi, Gabriel S; Malvar, David do C; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Kanashiro, Alexandre
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: ARTRITE, GABA, NEUROIMUNOMODULAÇÃO, NEUTRÓFILOS, SISTEMA NERVOSO SIMPÁTICO
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ABNT
BASSI, Gabriel S et al. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 389, n. 8, p. 851-861, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00210-016-1248-0. Acesso em: 09 nov. 2024.
APA
Bassi, G. S., Malvar, D. do C., Cunha, T. M., Cunha, F. de Q., & Kanashiro, A. (2016). Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis. Naunyn-Schmiedeberg's Archives of Pharmacology, 389( 8), 851-861. doi:10.1007/s00210-016-1248-0
NLM
Bassi GS, Malvar D do C, Cunha TM, Cunha F de Q, Kanashiro A. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 8): 851-861.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00210-016-1248-0
Vancouver
Bassi GS, Malvar D do C, Cunha TM, Cunha F de Q, Kanashiro A. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 8): 851-861.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00210-016-1248-0
Artigo de periodico
Paradoxical roles of the neutrophil in sepsis: protective and deleterious (2016)
Sônego, Fabiane; Castanheira, Fernanda Vargas e Silva; Ferreira, Raphael Gomes; Kanashiro, Alexandre; Leite, Caio Abner Vitorino Gonçalves; Nascimento, Daniele Carvalho; Colón, David Fernando; Borges, Vanessa de Fátima; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: SEPSE, NEUTRÓFILOS, RECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SÔNEGO, Fabiane et al. Paradoxical roles of the neutrophil in sepsis: protective and deleterious. Frontiers in Immunology, v. 7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00155. Acesso em: 09 nov. 2024.
APA
Sônego, F., Castanheira, F. V. e S., Ferreira, R. G., Kanashiro, A., Leite, C. A. V. G., Nascimento, D. C., et al. (2016). Paradoxical roles of the neutrophil in sepsis: protective and deleterious. Frontiers in Immunology, 7. doi:10.3389/fimmu.2016.00155
NLM
Sônego F, Castanheira FV e S, Ferreira RG, Kanashiro A, Leite CAVG, Nascimento DC, Colón DF, Borges V de F, Alves Filho JCF, Cunha F de Q. Paradoxical roles of the neutrophil in sepsis: protective and deleterious [Internet]. Frontiers in Immunology. 2016 ;7[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00155
Vancouver
Sônego F, Castanheira FV e S, Ferreira RG, Kanashiro A, Leite CAVG, Nascimento DC, Colón DF, Borges V de F, Alves Filho JCF, Cunha F de Q. Paradoxical roles of the neutrophil in sepsis: protective and deleterious [Internet]. Frontiers in Immunology. 2016 ;7[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00155
Artigo de periodico
Cytokines, but not corticotropin-releasing factor and endothelin-1, participate centrally in the febrile response in zymosan-induced arthritis in rats (2015)
Kanashiro, Alexandre; Figueiredo, Maria J; Malvar, David do C; Souza, Glória Emília Petto de
Source: Brain Research. Unidade: FCFRP
Subjects: CITOCINAS, ENDOTELINAS, FEBRE, PROSTAGLANDINAS, ARTRITE REUMATOIDE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KANASHIRO, Alexandre et al. Cytokines, but not corticotropin-releasing factor and endothelin-1, participate centrally in the febrile response in zymosan-induced arthritis in rats. Brain Research, v. 1610, p. 12-19, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2015.03.036. Acesso em: 09 nov. 2024.
APA
Kanashiro, A., Figueiredo, M. J., Malvar, D. do C., & Souza, G. E. P. de. (2015). Cytokines, but not corticotropin-releasing factor and endothelin-1, participate centrally in the febrile response in zymosan-induced arthritis in rats. Brain Research, 1610, 12-19. doi:10.1016/j.brainres.2015.03.036
NLM
Kanashiro A, Figueiredo MJ, Malvar D do C, Souza GEP de. Cytokines, but not corticotropin-releasing factor and endothelin-1, participate centrally in the febrile response in zymosan-induced arthritis in rats [Internet]. Brain Research. 2015 ; 1610 12-19.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.brainres.2015.03.036
Vancouver
Kanashiro A, Figueiredo MJ, Malvar D do C, Souza GEP de. Cytokines, but not corticotropin-releasing factor and endothelin-1, participate centrally in the febrile response in zymosan-induced arthritis in rats [Internet]. Brain Research. 2015 ; 1610 12-19.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.brainres.2015.03.036
Artigo de periodico
NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis (2013)
Figueiredo, Jozi; Ferreira, Ana Elisa; Silva, Rangel Leal; Ulloa, Luís; Grieco, Paolo; Cunha, Thiago Mattar; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Kanashiro, Alexandre
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: NEUTRÓFILOS (EFEITOS DE DROGAS), RECEPTORES ADRENÉRGICOS, NEUROIMUNOMODULAÇÃO, PERITONITE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FIGUEIREDO, Jozi et al. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 386, n. 4, p. 311-318, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00210-013-0834-7. Acesso em: 09 nov. 2024.
APA
Figueiredo, J., Ferreira, A. E., Silva, R. L., Ulloa, L., Grieco, P., Cunha, T. M., et al. (2013). NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis. Naunyn-Schmiedeberg's Archives of Pharmacology, 386( 4), 311-318. doi:10.1007/s00210-013-0834-7
NLM
Figueiredo J, Ferreira AE, Silva RL, Ulloa L, Grieco P, Cunha TM, Ferreira SH, Cunha F de Q, Kanashiro A. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 4): 311-318.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00210-013-0834-7
Vancouver
Figueiredo J, Ferreira AE, Silva RL, Ulloa L, Grieco P, Cunha TM, Ferreira SH, Cunha F de Q, Kanashiro A. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 4): 311-318.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00210-013-0834-7
Artigo de periodico
Quercetin restores plasma nitrite and nitroso species levels in renovascular hypertension (2010)
Montenegro, Marcelo F.; Neto-Neves, Evandro M.; Dias-Junior, Carlos A.; Ceron, Carla S.; Castro, Michele M.; Gomes, Valeria A.; Kanashiro, Alexandre; Tanus-Santos, José Eduardo
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: HIPERTENSÃO, ENDOTÉLIO VASCULAR, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MONTENEGRO, Marcelo F. et al. Quercetin restores plasma nitrite and nitroso species levels in renovascular hypertension. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 382, n. 4, p. 293-301, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00210-010-0546-1. Acesso em: 09 nov. 2024.
APA
Montenegro, M. F., Neto-Neves, E. M., Dias-Junior, C. A., Ceron, C. S., Castro, M. M., Gomes, V. A., et al. (2010). Quercetin restores plasma nitrite and nitroso species levels in renovascular hypertension. Naunyn-Schmiedeberg's Archives of Pharmacology, 382( 4), 293-301. doi:10.1007/s00210-010-0546-1
NLM
Montenegro MF, Neto-Neves EM, Dias-Junior CA, Ceron CS, Castro MM, Gomes VA, Kanashiro A, Tanus-Santos JE. Quercetin restores plasma nitrite and nitroso species levels in renovascular hypertension [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2010 ; 382( 4): 293-301.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00210-010-0546-1
Vancouver
Montenegro MF, Neto-Neves EM, Dias-Junior CA, Ceron CS, Castro MM, Gomes VA, Kanashiro A, Tanus-Santos JE. Quercetin restores plasma nitrite and nitroso species levels in renovascular hypertension [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2010 ; 382( 4): 293-301.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00210-010-0546-1
Artigo de periodico
Inflammatory mediators involved in the nociceptive and oedematogenic responses induced by Tityus serrulatus scorpion venom injected into rat paws (2008)
Pessini, Andréa Carla; Kanashiro, Alexandre; Malvar, David do C.; Machado, Renes R.; Soares, Denis M.; Figueiredo, Maria J.; Kalapothakis, Evanguedes; Souza, Glória Emília Petto de
Source: Toxicon. Unidade: FCFRP
Subjects: VENENOS DE ORIGEM ANIMAL, ESCORPIÕES, INFLAMAÇÃO, DOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PESSINI, Andréa Carla et al. Inflammatory mediators involved in the nociceptive and oedematogenic responses induced by Tityus serrulatus scorpion venom injected into rat paws. Toxicon, v. 52, n. 7, p. 729-736, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2008.08.017. Acesso em: 09 nov. 2024.
APA
Pessini, A. C., Kanashiro, A., Malvar, D. do C., Machado, R. R., Soares, D. M., Figueiredo, M. J., et al. (2008). Inflammatory mediators involved in the nociceptive and oedematogenic responses induced by Tityus serrulatus scorpion venom injected into rat paws. Toxicon, 52( 7), 729-736. doi:10.1016/j.toxicon.2008.08.017
NLM
Pessini AC, Kanashiro A, Malvar D do C, Machado RR, Soares DM, Figueiredo MJ, Kalapothakis E, Souza GEP de. Inflammatory mediators involved in the nociceptive and oedematogenic responses induced by Tityus serrulatus scorpion venom injected into rat paws [Internet]. Toxicon. 2008 ; 52( 7): 729-736.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.toxicon.2008.08.017
Vancouver
Pessini AC, Kanashiro A, Malvar D do C, Machado RR, Soares DM, Figueiredo MJ, Kalapothakis E, Souza GEP de. Inflammatory mediators involved in the nociceptive and oedematogenic responses induced by Tityus serrulatus scorpion venom injected into rat paws [Internet]. Toxicon. 2008 ; 52( 7): 729-736.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.toxicon.2008.08.017

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