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Artigo de periodico
Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats (2017)
Araujo, Priscila Xavier; Costa, Tiago Januário; Echem, Cinthya; Oliveira, Maria Aparecida de; Santos-Eichler, Rosangela Aparecida; Colli, Lucas Giglio; Jiménez-Altayó, Francesc; Vila, Elisabet; Akamine, Eliana Hiromi ; Dantas, Ana Paula; Ceravolo, Graziela Scalianti; Carvalho, Maria Helena Catelli de
Source: Journal of Pharmacology and Experimental Therapeutics. Unidade: ICB
Assunto: FARMACOLOGIA
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ABNT
ARAUJO, Priscila Xavier et al. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, v. 362, n. 1, p. 98-107, 2017Tradução . . Disponível em: https://doi.org/10.1124/jpet.117.240465. Acesso em: 01 nov. 2024.
APA
Araujo, P. X., Costa, T. J., Echem, C., Oliveira, M. A. de, Santos-Eichler, R. A., Colli, L. G., et al. (2017). Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, 362( 1), 98-107. doi:10.1124/jpet.117.240465
NLM
Araujo PX, Costa TJ, Echem C, Oliveira MA de, Santos-Eichler RA, Colli LG, Jiménez-Altayó F, Vila E, Akamine EH, Dantas AP, Ceravolo GS, Carvalho MHC de. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats [Internet]. Journal of Pharmacology and Experimental Therapeutics. 2017 ; 362( 1): 98-107.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1124/jpet.117.240465
Vancouver
Araujo PX, Costa TJ, Echem C, Oliveira MA de, Santos-Eichler RA, Colli LG, Jiménez-Altayó F, Vila E, Akamine EH, Dantas AP, Ceravolo GS, Carvalho MHC de. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats [Internet]. Journal of Pharmacology and Experimental Therapeutics. 2017 ; 362( 1): 98-107.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1124/jpet.117.240465
Artigo de periodico
Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products (2016)
Almeida, Paula Campi Locatelli de; Herrera, Bruno Schneider; Jesus, Flavia Neto de; Napolitano, Mauro; Teixeira, Simone Aparecida; Dantas, Aline Maia; Spolidorio, Luis Carlos; Akamine, Eliana Hiromi ; Mayer, Marcia Pinto Alves ; Carvalho, Maria Helena Catelli de; Costa, Soraia Katia Pereira ; Muscará, Marcelo Nicolás
Source: Archives of Oral Biology. Unidade: ICB
Subjects: PERIODONTITE, ENDOTÉLIO, AORTA, DOENÇAS CARDIOVASCULARES, ÓXIDO NÍTRICO, INFLAÇÃO
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ABNT
ALMEIDA, Paula Campi Locatelli de et al. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products. Archives of Oral Biology, v. 63, p. 66-74, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.archoralbio.2015.11.022. Acesso em: 01 nov. 2024.
APA
Almeida, P. C. L. de, Herrera, B. S., Jesus, F. N. de, Napolitano, M., Teixeira, S. A., Dantas, A. M., et al. (2016). Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products. Archives of Oral Biology, 63, 66-74. doi:10.1016/j.archoralbio.2015.11.022
NLM
Almeida PCL de, Herrera BS, Jesus FN de, Napolitano M, Teixeira SA, Dantas AM, Spolidorio LC, Akamine EH, Mayer MPA, Carvalho MHC de, Costa SKP, Muscará MN. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products [Internet]. Archives of Oral Biology. 2016 ; 63 66-74.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.archoralbio.2015.11.022
Vancouver
Almeida PCL de, Herrera BS, Jesus FN de, Napolitano M, Teixeira SA, Dantas AM, Spolidorio LC, Akamine EH, Mayer MPA, Carvalho MHC de, Costa SKP, Muscará MN. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products [Internet]. Archives of Oral Biology. 2016 ; 63 66-74.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.archoralbio.2015.11.022
Artigo de periodico
Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats (2015)
Bomfim, G. F.; Echem, Cinthya; Martins, C. B.; Costa, T. J.; Sartoretto, Simone Marcieli; Santos, R. A. dos; Oliveira, M. A.; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo; Webb, R. C.; Carvalho, Maria Helena Catelli de
Source: Life Sciences. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, INFLAMAÇÃO, HIPERTENSÃO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOMFIM, G. F. et al. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, v. 122, p. 1-7, 2015Tradução . . Disponível em: http://lattes.cnpq.br/5055165961832291. Acesso em: 01 nov. 2024.
APA
Bomfim, G. F., Echem, C., Martins, C. B., Costa, T. J., Sartoretto, S. M., Santos, R. A. dos, et al. (2015). Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, 122, 1-7. doi:10.1016/j.lfs.2014.12.001
NLM
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 nov. 01 ] Available from: http://lattes.cnpq.br/5055165961832291
Vancouver
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 nov. 01 ] Available from: http://lattes.cnpq.br/5055165961832291
Artigo de periodico
Leptin downregulates LPS-induced lung injury: role of corticosterone and insulin (2014)
Landgraf, Maristella A.; Silva, Reinaldo C.; Correa-Costa, Matheus; Hiyane, Meire Ioshino; Carvalho, Maria Helena Catelli de; Landgraf, Richardt G.; Câmara, Niels Olsen Saraiva
Source: Cellular Physiology and Biochemistry. Unidade: ICB
Subjects: FARMACOLOGIA, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LANDGRAF, Maristella A. et al. Leptin downregulates LPS-induced lung injury: role of corticosterone and insulin. Cellular Physiology and Biochemistry, v. 33, n. 3, p. 835-846, 2014Tradução . . Disponível em: https://doi.org/10.1159/000358656. Acesso em: 01 nov. 2024.
APA
Landgraf, M. A., Silva, R. C., Correa-Costa, M., Hiyane, M. I., Carvalho, M. H. C. de, Landgraf, R. G., & Câmara, N. O. S. (2014). Leptin downregulates LPS-induced lung injury: role of corticosterone and insulin. Cellular Physiology and Biochemistry, 33( 3), 835-846. doi:10.1159/000358656
NLM
Landgraf MA, Silva RC, Correa-Costa M, Hiyane MI, Carvalho MHC de, Landgraf RG, Câmara NOS. Leptin downregulates LPS-induced lung injury: role of corticosterone and insulin [Internet]. Cellular Physiology and Biochemistry. 2014 ; 33( 3): 835-846.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1159/000358656
Vancouver
Landgraf MA, Silva RC, Correa-Costa M, Hiyane MI, Carvalho MHC de, Landgraf RG, Câmara NOS. Leptin downregulates LPS-induced lung injury: role of corticosterone and insulin [Internet]. Cellular Physiology and Biochemistry. 2014 ; 33( 3): 835-846.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1159/000358656

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