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Artigo de periodico
Moderate exercise modulates tumor metabolism of triple-negative breast cancer (2020)
Vulczak, Anderson ; Souza, Anderson de Oliveira ; Ferrari, Gustavo Duarte ; Azzolini, Ana Elisa Caleiro Seixas ; Bisson, Gabriela Silva ; Alberici, Luciane Carla
Source: Cells. Unidades: FCFRP, EERP, FMRP
Subjects: MITOCÔNDRIAS, NEOPLASIAS MAMÁRIAS, ATIVIDADE FÍSICA, EXERCÍCIO FÍSICO, METABOLISMO
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VULCZAK, Anderson et al. Moderate exercise modulates tumor metabolism of triple-negative breast cancer. Cells, v. 9, n. 3, 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9030628. Acesso em: 17 nov. 2024.
APA
Vulczak, A., Souza, A. de O., Ferrari, G. D., Azzolini, A. E. C. S., Bisson, G. S., & Alberici, L. C. (2020). Moderate exercise modulates tumor metabolism of triple-negative breast cancer. Cells, 9( 3). doi:10.3390/cells9030628
NLM
Vulczak A, Souza A de O, Ferrari GD, Azzolini AECS, Bisson GS, Alberici LC. Moderate exercise modulates tumor metabolism of triple-negative breast cancer [Internet]. Cells. 2020 ; 9( 3):[citado 2024 nov. 17 ] Available from: https://doi.org/10.3390/cells9030628
Vancouver
Vulczak A, Souza A de O, Ferrari GD, Azzolini AECS, Bisson GS, Alberici LC. Moderate exercise modulates tumor metabolism of triple-negative breast cancer [Internet]. Cells. 2020 ; 9( 3):[citado 2024 nov. 17 ] Available from: https://doi.org/10.3390/cells9030628
Artigo de periodico
Altered global microRNA expression in hepatic stellate cells LX-2 by angiotensin-(1–7) and miRNA-1914-5p identification as regulator of pro-fibrogenic elements and lipid metabolism (2018)
Oliveira da Silva, Brenda de; Alberici, Luciane Carla; Ramos, Letícia Ferreira; Silva, Caio Mateus; Silveira, Marina Bonfogo da; Dechant, Carlos R. P.; Friedman, Scott L.; Sakane, Kumiko Koibuchi; Gonçalves, Letícia Rocha; Moraes, Karen C. M.
Source: The International Journal of Biochemistry & Cell Biology. Unidade: FCFRP
Subjects: ANGIOTENSINAS, METABOLISMO DE GORDURA, MICRORNAS, REGULAÇÃO GÊNICA, EXPRESSÃO GÊNICA
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OLIVEIRA DA SILVA, Brenda de et al. Altered global microRNA expression in hepatic stellate cells LX-2 by angiotensin-(1–7) and miRNA-1914-5p identification as regulator of pro-fibrogenic elements and lipid metabolism. The International Journal of Biochemistry & Cell Biology, v. 98, p. 137-155, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.biocel.2018.02.018. Acesso em: 17 nov. 2024.
APA
Oliveira da Silva, B. de, Alberici, L. C., Ramos, L. F., Silva, C. M., Silveira, M. B. da, Dechant, C. R. P., et al. (2018). Altered global microRNA expression in hepatic stellate cells LX-2 by angiotensin-(1–7) and miRNA-1914-5p identification as regulator of pro-fibrogenic elements and lipid metabolism. The International Journal of Biochemistry & Cell Biology, 98, 137-155. doi:10.1016/j.biocel.2018.02.018
NLM
Oliveira da Silva B de, Alberici LC, Ramos LF, Silva CM, Silveira MB da, Dechant CRP, Friedman SL, Sakane KK, Gonçalves LR, Moraes KCM. Altered global microRNA expression in hepatic stellate cells LX-2 by angiotensin-(1–7) and miRNA-1914-5p identification as regulator of pro-fibrogenic elements and lipid metabolism [Internet]. The International Journal of Biochemistry & Cell Biology. 2018 ; 98 137-155.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.biocel.2018.02.018
Vancouver
Oliveira da Silva B de, Alberici LC, Ramos LF, Silva CM, Silveira MB da, Dechant CRP, Friedman SL, Sakane KK, Gonçalves LR, Moraes KCM. Altered global microRNA expression in hepatic stellate cells LX-2 by angiotensin-(1–7) and miRNA-1914-5p identification as regulator of pro-fibrogenic elements and lipid metabolism [Internet]. The International Journal of Biochemistry & Cell Biology. 2018 ; 98 137-155.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.biocel.2018.02.018
Artigo de periodico
The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms (2017)
Prida, Javier Marín; Andreu, Gilberto L. Pardo; Rossignoli, Camila Pederiva; Durruthy, Michael González; Rodríguez, Estael Ochoa; Reyes, Yamila Verdecia; Acosta, Roberto Fernández; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Source: Toxicology in Vitro. Unidade: FCFRP
Subjects: NEOPLASIAS, CITOTOXICIDADE IMUNOLÓGICA, MITOCÔNDRIAS, FÁRMACOS
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PRIDA, Javier Marín et al. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, v. 42, p. 21-30, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2017.03.011. Acesso em: 17 nov. 2024.
APA
Prida, J. M., Andreu, G. L. P., Rossignoli, C. P., Durruthy, M. G., Rodríguez, E. O., Reyes, Y. V., et al. (2017). The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, 42, 21-30. doi:10.1016/j.tiv.2017.03.011
NLM
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Vancouver
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Artigo de periodico
Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle (2017)
Teodoro, Bruno G.; Sampaio, Igor H.; Bomfim, Lucas H. M.; Queiroz, André L.; Silveira, Leonardo R.; Souza, Anderson O.; Fernandes, Anna M. A. P.; Eberlin, Marcos N.; Huang, Tai-Yu; Zheng, Donghai; Neufer, P. Darrell; Cortright, Ronald N.; Alberici, Luciane Carla
Source: The Journal of Physiology. Unidade: FCFRP
Subjects: MÚSCULO ESQUELÉTICO, METABOLISMO DE GORDURA, MITOCÔNDRIAS, ÁCIDOS GRAXOS
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TEODORO, Bruno G. et al. Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle. The Journal of Physiology, v. 595, n. 3, p. 677-693, 2017Tradução . . Disponível em: https://doi.org/10.1113/jp272962. Acesso em: 17 nov. 2024.
APA
Teodoro, B. G., Sampaio, I. H., Bomfim, L. H. M., Queiroz, A. L., Silveira, L. R., Souza, A. O., et al. (2017). Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle. The Journal of Physiology, 595( 3), 677-693. doi:10.1113/jp272962
NLM
Teodoro BG, Sampaio IH, Bomfim LHM, Queiroz AL, Silveira LR, Souza AO, Fernandes AMAP, Eberlin MN, Huang T-Y, Zheng D, Neufer PD, Cortright RN, Alberici LC. Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle [Internet]. The Journal of Physiology. 2017 ; 595( 3): 677-693.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1113/jp272962
Vancouver
Teodoro BG, Sampaio IH, Bomfim LHM, Queiroz AL, Silveira LR, Souza AO, Fernandes AMAP, Eberlin MN, Huang T-Y, Zheng D, Neufer PD, Cortright RN, Alberici LC. Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle [Internet]. The Journal of Physiology. 2017 ; 595( 3): 677-693.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1113/jp272962
Artigo de periodico
Protective action of Omega-3 on paraquat intoxication in Drosophila melanogaster (2017)
Souza, Anderson de Oliveira; Couto-Lima, Carlos Antônio; Machado, Maiaro Cabral Rosa; Espreafico, Enilza Maria; Ramos, Ricardo Guelerman Pinheiro; Alberici, Luciane Carla
Source: Journal of Toxicology and Environmental Health, Part A. Unidades: FMRP, FCFRP
Subjects: DROSOPHILA, TOXICOLOGIA, ÁCIDOS GRAXOS, SUPLEMENTOS DIETÉTICOS, HERBICIDAS
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SOUZA, Anderson de Oliveira et al. Protective action of Omega-3 on paraquat intoxication in Drosophila melanogaster. Journal of Toxicology and Environmental Health, Part A, v. 80, n. 19-21, p. 1050-1063, 2017Tradução . . Disponível em: https://doi.org/10.1080/15287394.2017.1357345. Acesso em: 17 nov. 2024.
APA
Souza, A. de O., Couto-Lima, C. A., Machado, M. C. R., Espreafico, E. M., Ramos, R. G. P., & Alberici, L. C. (2017). Protective action of Omega-3 on paraquat intoxication in Drosophila melanogaster. Journal of Toxicology and Environmental Health, Part A, 80( 19-21), 1050-1063. doi:10.1080/15287394.2017.1357345
NLM
Souza A de O, Couto-Lima CA, Machado MCR, Espreafico EM, Ramos RGP, Alberici LC. Protective action of Omega-3 on paraquat intoxication in Drosophila melanogaster [Internet]. Journal of Toxicology and Environmental Health, Part A. 2017 ; 80( 19-21): 1050-1063.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1080/15287394.2017.1357345
Vancouver
Souza A de O, Couto-Lima CA, Machado MCR, Espreafico EM, Ramos RGP, Alberici LC. Protective action of Omega-3 on paraquat intoxication in Drosophila melanogaster [Internet]. Journal of Toxicology and Environmental Health, Part A. 2017 ; 80( 19-21): 1050-1063.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1080/15287394.2017.1357345
Artigo de periodico
QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload (2017)
Durruthy, Michael González; Castro, Micheli; Nunes, Silvana Manske; Lima, Juliane Ventura; Alberici, Luciane Carla; Naal, Zeki; Sawazaki, David T. Atique; Curti, Carlos; Ruas, Caroline Pires; Gelesky, Marcos A.; Roy, Kunal; González-Díaz, Humberto; Monserrat, José M.
Source: Carbon. Unidade: FCFRP
Subjects: NANOTUBOS DE CARBONO, MITOCÔNDRIAS, CURVAS DE DOSE-RESPOSTA, TEORIA DE PERTURBAÇÃO PARA OPERADORES LINEARES
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DURRUTHY, Michael González et al. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, v. 115, p. 312-330, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.carbon.2017.01.002. Acesso em: 17 nov. 2024.
APA
Durruthy, M. G., Castro, M., Nunes, S. M., Lima, J. V., Alberici, L. C., Naal, Z., et al. (2017). QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, 115, 312-330. doi:10.1016/j.carbon.2017.01.002
NLM
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Vancouver
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Artigo de periodico
Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes (2017)
Dechandt, Carlos R. P.; Zuccolotto-dos-Reis, Felippe H.; Teodoro, Bruno G.; Fernandes, Anna Maria A. P.; Eberlin, Marcos N.; Kettelhut, Isis do Carmo; Curti, Carlos; Alberici, Luciane Carla
Source: Journal of Bioenergetics and Biomembranes. Unidades: FMRP, FCFRP
Subjects: CÉLULAS CULTIVADAS, MITOCÔNDRIAS, METABOLISMO DE GORDURA, FÍGADO GORDUROSO
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DECHANDT, Carlos R. P. et al. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, v. 49, n. 5, p. 399-411, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10863-017-9725-9. Acesso em: 17 nov. 2024.
APA
Dechandt, C. R. P., Zuccolotto-dos-Reis, F. H., Teodoro, B. G., Fernandes, A. M. A. P., Eberlin, M. N., Kettelhut, I. do C., et al. (2017). Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, 49( 5), 399-411. doi:10.1007/s10863-017-9725-9
NLM
Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
Vancouver
Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
Artigo de periodico
SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line (2017)
Sobral, Lays M.; Coletta, Ricardo D.; Alberici, Luciane Carla; Curti, Carlos; Leopoldino, Andréia Machado
Source: The FEBS Journal. Unidade: FCFRP
Subjects: TOXICOLOGIA, CÉLULAS, METABOLISMO, MITOCÔNDRIAS, RESPIRAÇÃO
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SOBRAL, Lays M. et al. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, v. 284, n. 17, p. 2774-2785, 2017Tradução . . Disponível em: https://doi.org/10.1111/febs.14148. Acesso em: 17 nov. 2024.
APA
Sobral, L. M., Coletta, R. D., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2017). SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, 284( 17), 2774-2785. doi:10.1111/febs.14148
NLM
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/febs.14148
Vancouver
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/febs.14148
Artigo de periodico
Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants (2017)
González-Durruthy, Michael; Alberici, Luciane C.; Curti, Carlos; Naal, Zeki; Sawazaki, David Tatsuo A; Vázquez-Naya, José M.; González-Díaz, Humberto; Munteanu, Cristian R.
Source: Journal of Chemical Information and Modeling. Unidade: FCFRP
Subjects: ENTROPIA, REGRESSÃO LINEAR, MITOCÔNDRIAS, NANOTUBOS DE CARBONO, BIOMEDICINA, OXIGÊNIO
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GONZÁLEZ-DURRUTHY, Michael et al. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, v. 57, n. 5, p. 1029-1044, 2017Tradução . . Disponível em: https://doi.org/10.1021/acs.jcim.6b00458. Acesso em: 17 nov. 2024.
APA
González-Durruthy, M., Alberici, L. C., Curti, C., Naal, Z., Sawazaki, D. T. A., Vázquez-Naya, J. M., et al. (2017). Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, 57( 5), 1029-1044. doi:10.1021/acs.jcim.6b00458
NLM
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Vancouver
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Artigo de periodico
Direct visualization of neurotransmitters in rat brain slices by desorption electrospray ionization mass spectrometry imaging (DESI - MS) (2016)
Fernandes, Anna Maria A. P.; Vendramini, Pedro H.; Galaverna, Renan; Schwab, Nicolas V.; Alberici, Luciane Carla; Augusti, Rodinei; Castilho, Roger F.; Eberlin, Marcos N.
Source: Journal of The American Society for Mass Spectrometry. Unidade: FCFRP
Subjects: ESPECTROMETRIA DE MASSAS, NEUROTRANSMISSORES, CÉREBRO
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FERNANDES, Anna Maria A. P. et al. Direct visualization of neurotransmitters in rat brain slices by desorption electrospray ionization mass spectrometry imaging (DESI - MS). Journal of The American Society for Mass Spectrometry, v. 27, n. 12, p. 1944-1951, 2016Tradução . . Disponível em: https://doi.org/10.1007/s13361-016-1475-0. Acesso em: 17 nov. 2024.
APA
Fernandes, A. M. A. P., Vendramini, P. H., Galaverna, R., Schwab, N. V., Alberici, L. C., Augusti, R., et al. (2016). Direct visualization of neurotransmitters in rat brain slices by desorption electrospray ionization mass spectrometry imaging (DESI - MS). Journal of The American Society for Mass Spectrometry, 27( 12), 1944-1951. doi:10.1007/s13361-016-1475-0
NLM
Fernandes AMAP, Vendramini PH, Galaverna R, Schwab NV, Alberici LC, Augusti R, Castilho RF, Eberlin MN. Direct visualization of neurotransmitters in rat brain slices by desorption electrospray ionization mass spectrometry imaging (DESI - MS) [Internet]. Journal of The American Society for Mass Spectrometry. 2016 ; 27( 12): 1944-1951.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s13361-016-1475-0
Vancouver
Fernandes AMAP, Vendramini PH, Galaverna R, Schwab NV, Alberici LC, Augusti R, Castilho RF, Eberlin MN. Direct visualization of neurotransmitters in rat brain slices by desorption electrospray ionization mass spectrometry imaging (DESI - MS) [Internet]. Journal of The American Society for Mass Spectrometry. 2016 ; 27( 12): 1944-1951.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s13361-016-1475-0
Artigo de periodico
The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling (2015)
Andreu, Gilberto L. Pardo; Reis, Felippe H. Z; Dalalio, Felipe M; Figueredo, Yanier Nuñez; Rubio, Osmany Cuesta; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Source: Chemico-Biological Interactions. Unidade: FCFRP
Subjects: PRÓPOLIS, MITOCÔNDRIAS, TESTES DE TOXICIDADE, TOXICOLOGIA
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ABNT
ANDREU, Gilberto L. Pardo et al. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, v. 228, p. 28-34, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.01.010. Acesso em: 17 nov. 2024.
APA
Andreu, G. L. P., Reis, F. H. Z., Dalalio, F. M., Figueredo, Y. N., Rubio, O. C., Uyemura, S. A., et al. (2015). The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, 228, 28-34. doi:10.1016/j.cbi.2015.01.010
NLM
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Vancouver
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Artigo de periodico
Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria (2012)
Rodrigues, Fernando P.; Pestana, Cezar R.; Polizello, Ana Cristina Morseli; Pardo-Andreu, Gilberto L.; Uyemura, Sérgio Akira; Santos, Antônio Cardozo dos; Alberici, Luciane Carla; Silva, Roberto Santana da; Curti, Carlos
Source: Nitric Oxide. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, RUTÊNIO, ÓXIDO NÍTRICO
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ABNT
RODRIGUES, Fernando P. et al. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria. Nitric Oxide, v. 26, n. 3, p. 174-181, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2012.02.001. Acesso em: 17 nov. 2024.
APA
Rodrigues, F. P., Pestana, C. R., Polizello, A. C. M., Pardo-Andreu, G. L., Uyemura, S. A., Santos, A. C. dos, et al. (2012). Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria. Nitric Oxide, 26( 3), 174-181. doi:10.1016/j.niox.2012.02.001
NLM
Rodrigues FP, Pestana CR, Polizello ACM, Pardo-Andreu GL, Uyemura SA, Santos AC dos, Alberici LC, Silva RS da, Curti C. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria [Internet]. Nitric Oxide. 2012 ; 26( 3): 174-181.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.niox.2012.02.001
Vancouver
Rodrigues FP, Pestana CR, Polizello ACM, Pardo-Andreu GL, Uyemura SA, Santos AC dos, Alberici LC, Silva RS da, Curti C. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria [Internet]. Nitric Oxide. 2012 ; 26( 3): 174-181.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.niox.2012.02.001
Artigo de periodico
The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications (2011)
Pardo-Andreu, Gilberto L.; Nuñez-Figueredo, Yanier; Tudelli, Valeria G.; Cuesta-Rubio, Osmany; Rodrigues, Fernando P.; Pestana, Cezar R.; Uyemura, Sérgio Akira; Leopoldino, Andréia Machado; Alberici, Luciane Carla; Curti, Carlos
Source: Toxicology and Applied Pharmacology. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, ESTRESSE OXIDATIVO, TOXICOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PARDO-ANDREU, Gilberto L. et al. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications. Toxicology and Applied Pharmacology, v. 253, n. 3, p. 282-289, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2011.04.011. Acesso em: 17 nov. 2024.
APA
Pardo-Andreu, G. L., Nuñez-Figueredo, Y., Tudelli, V. G., Cuesta-Rubio, O., Rodrigues, F. P., Pestana, C. R., et al. (2011). The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications. Toxicology and Applied Pharmacology, 253( 3), 282-289. doi:10.1016/j.taap.2011.04.011
NLM
Pardo-Andreu GL, Nuñez-Figueredo Y, Tudelli VG, Cuesta-Rubio O, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications [Internet]. Toxicology and Applied Pharmacology. 2011 ; 253( 3): 282-289.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.taap.2011.04.011
Vancouver
Pardo-Andreu GL, Nuñez-Figueredo Y, Tudelli VG, Cuesta-Rubio O, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications [Internet]. Toxicology and Applied Pharmacology. 2011 ; 253( 3): 282-289.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.taap.2011.04.011
Artigo de periodico
Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis (2011)
Zecchin, Karina G.; Rossato, Franco A.; Raposo, Helena F.; Melo, Daniela R.; Alberici, Luciane Carla; Oliveira, Helena C. F.; Castilho, Roger F.; Coletta, Ricardo D.; Vercesi, Aníbal E.; Graner, Edgard
Source: Laboratory Investigation. Unidade: FCFRP
Subjects: APOPTOSE, MELANOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZECCHIN, Karina G. et al. Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis. Laboratory Investigation, v. 91, n. 2, p. 232-240, 2011Tradução . . Disponível em: https://doi.org/10.1038/labinvest.2010.157. Acesso em: 17 nov. 2024.
APA
Zecchin, K. G., Rossato, F. A., Raposo, H. F., Melo, D. R., Alberici, L. C., Oliveira, H. C. F., et al. (2011). Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis. Laboratory Investigation, 91( 2), 232-240. doi:10.1038/labinvest.2010.157
NLM
Zecchin KG, Rossato FA, Raposo HF, Melo DR, Alberici LC, Oliveira HCF, Castilho RF, Coletta RD, Vercesi AE, Graner E. Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis [Internet]. Laboratory Investigation. 2011 ; 91( 2): 232-240.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/labinvest.2010.157
Vancouver
Zecchin KG, Rossato FA, Raposo HF, Melo DR, Alberici LC, Oliveira HCF, Castilho RF, Coletta RD, Vercesi AE, Graner E. Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis [Internet]. Laboratory Investigation. 2011 ; 91( 2): 232-240.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/labinvest.2010.157
Artigo de periodico
Mitochondrial energy metabolism and redox responses to hypertriglyceridemia (2011)
Alberici, Luciane Carla; Vercesi, Anibal E.; Oliveira, Helena C. F.
Source: Journal of Bioenergetics and Biomembranes. Unidade: FCFRP
Subjects: HIPERTRIGLICERIDEMIA, MITOCÔNDRIAS, POTÁSSIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALBERICI, Luciane Carla e VERCESI, Anibal E. e OLIVEIRA, Helena C. F. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia. Journal of Bioenergetics and Biomembranes, v. 43, p. 19-23, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10863-011-9326-y. Acesso em: 17 nov. 2024.
APA
Alberici, L. C., Vercesi, A. E., & Oliveira, H. C. F. (2011). Mitochondrial energy metabolism and redox responses to hypertriglyceridemia. Journal of Bioenergetics and Biomembranes, 43, 19-23. doi:10.1007/s10863-011-9326-y
NLM
Alberici LC, Vercesi AE, Oliveira HCF. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43 19-23.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s10863-011-9326-y
Vancouver
Alberici LC, Vercesi AE, Oliveira HCF. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43 19-23.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s10863-011-9326-y

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