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Artigo de periodico
A genetic cluster of patients with variant xeroderma pigmentosum with two diferent founder mutations (2017)
Munford, Veridiana; Castro, L. P.; Souto, R.; Lerner, Leticia Koch; Vilar, Juliana Brandstetterb; Quayle, Carolina de Barros Reis; Asif, Huma; Schuch, André Passaglia; de Souza, T. A.; Ienne, S.; Alves, F. I. A.; Moura, L. M. S.; Galante, P. A. F.; Camargo, A. A.; Liboredo, R.; Pena, S. D. J.; Sarasin, A.; Chaibub, S. C.; Menck, Carlos Frederico Martins
Fonte: British Journal of Dermatology. Unidade: ICB
Assuntos: MICROBIOLOGIA, MUTAÇÃO GENÉTICA, FENÓTIPOS, GENES, PIGMENTOS BIOLÓGICOS, GENÉTICA MICROBIANA
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MUNFORD, Veridiana et al. A genetic cluster of patients with variant xeroderma pigmentosum with two diferent founder mutations. British Journal of Dermatology, v. 176, n. 5, p. 1270-1278, 2017Tradução . . Disponível em: https://doi.org/10.1111/bjd.15084. Acesso em: 09 ago. 2024.
APA
Munford, V., Castro, L. P., Souto, R., Lerner, L. K., Vilar, J. B., Quayle, C. de B. R., et al. (2017). A genetic cluster of patients with variant xeroderma pigmentosum with two diferent founder mutations. British Journal of Dermatology, 176( 5), 1270-1278. doi:10.1111/bjd.15084
NLM
Munford V, Castro LP, Souto R, Lerner LK, Vilar JB, Quayle C de BR, Asif H, Schuch AP, de Souza TA, Ienne S, Alves FIA, Moura LMS, Galante PAF, Camargo AA, Liboredo R, Pena SDJ, Sarasin A, Chaibub SC, Menck CFM. A genetic cluster of patients with variant xeroderma pigmentosum with two diferent founder mutations [Internet]. British Journal of Dermatology. 2017 ; 176( 5): 1270-1278.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1111/bjd.15084
Vancouver
Munford V, Castro LP, Souto R, Lerner LK, Vilar JB, Quayle C de BR, Asif H, Schuch AP, de Souza TA, Ienne S, Alves FIA, Moura LMS, Galante PAF, Camargo AA, Liboredo R, Pena SDJ, Sarasin A, Chaibub SC, Menck CFM. A genetic cluster of patients with variant xeroderma pigmentosum with two diferent founder mutations [Internet]. British Journal of Dermatology. 2017 ; 176( 5): 1270-1278.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1111/bjd.15084
Artigo de periodico
LMNB1 mutation causes cerebellar involvement and a genome instability defect (2017)
Pedroso, José Luiz; Munford, Veridiana; Bastos, André Uchimura; Castro, Ligia Pereira de; Marussi, Victor Hugo Rocha; Silva, Gisele Sampaio; Arita, Juliana Harumi; Menck, Carlos Frederico Martins ; Barsottini, Orlando G.
Fonte: Journal of the Neurological Sciences. Unidade: ICB
Assuntos: MICROBIOLOGIA, MUTAÇÃO GENÉTICA, GENOMAS, ATAXIA, CEREBELO
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PEDROSO, José Luiz et al. LMNB1 mutation causes cerebellar involvement and a genome instability defect. Journal of the Neurological Sciences, v. 379, p. 249-252, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jns.2017.06.027. Acesso em: 09 ago. 2024.
APA
Pedroso, J. L., Munford, V., Bastos, A. U., Castro, L. P. de, Marussi, V. H. R., Silva, G. S., et al. (2017). LMNB1 mutation causes cerebellar involvement and a genome instability defect. Journal of the Neurological Sciences, 379, 249-252. doi:10.1016/j.jns.2017.06.027
NLM
Pedroso JL, Munford V, Bastos AU, Castro LP de, Marussi VHR, Silva GS, Arita JH, Menck CFM, Barsottini OG. LMNB1 mutation causes cerebellar involvement and a genome instability defect [Internet]. Journal of the Neurological Sciences. 2017 ; 379 249-252.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.jns.2017.06.027
Vancouver
Pedroso JL, Munford V, Bastos AU, Castro LP de, Marussi VHR, Silva GS, Arita JH, Menck CFM, Barsottini OG. LMNB1 mutation causes cerebellar involvement and a genome instability defect [Internet]. Journal of the Neurological Sciences. 2017 ; 379 249-252.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.jns.2017.06.027
Artigo de periodico
Sunlight damage to cellular DNA: focus on oxidatively generated lesions (2017)
Schuch, André Passaglia; Moreno, Natália Cestari; Schuch, Natielen Jacques; Menck, Carlos Frederico Martins ; Garcia, Camila Carrião Machado
Fonte: Free Radical Biology and Medicine. Unidade: ICB
Assunto: MICROBIOLOGIA
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SCHUCH, André Passaglia et al. Sunlight damage to cellular DNA: focus on oxidatively generated lesions. Free Radical Biology and Medicine, v. 107, p. 110-124, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2017.01.029. Acesso em: 09 ago. 2024.
APA
Schuch, A. P., Moreno, N. C., Schuch, N. J., Menck, C. F. M., & Garcia, C. C. M. (2017). Sunlight damage to cellular DNA: focus on oxidatively generated lesions. Free Radical Biology and Medicine, 107, 110-124. doi:10.1016/j.freeradbiomed.2017.01.029
NLM
Schuch AP, Moreno NC, Schuch NJ, Menck CFM, Garcia CCM. Sunlight damage to cellular DNA: focus on oxidatively generated lesions [Internet]. Free Radical Biology and Medicine. 2017 ; 107 110-124.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2017.01.029
Vancouver
Schuch AP, Moreno NC, Schuch NJ, Menck CFM, Garcia CCM. Sunlight damage to cellular DNA: focus on oxidatively generated lesions [Internet]. Free Radical Biology and Medicine. 2017 ; 107 110-124.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2017.01.029
Artigo de periodico
Molecular characterization of Caulobacter crescentus mutator strains (2017)
Martins-Pinheiro, Marinalva; Oliveira, Alice R.; Valencia, Alexy Orozco; Fernández-Silva, Frank Sydnei; Silva, Larissa Gomes da; Lopes-Kulishev, Carina Oliveira; Italiani, Valeria Cristina da Silva; Marques, Marilis do Valle ; Menck, Carlos Frederico Martins ; Galhardo, Rodrigo da Silva
Fonte: Gene. Unidade: ICB
Assuntos: MICROBIOLOGIA, GENÉTICA BACTERIANA, MUTAÇÃO GENÉTICA, BACTÉRIAS GRAM-NEGATIVAS, SEQUENCIAMENTO GENÉTICO, MUTAGÊNESE
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MARTINS-PINHEIRO, Marinalva et al. Molecular characterization of Caulobacter crescentus mutator strains. Gene, v. 626, p. 251-257, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.gene.2017.05.038. Acesso em: 09 ago. 2024.
APA
Martins-Pinheiro, M., Oliveira, A. R., Valencia, A. O., Fernández-Silva, F. S., Silva, L. G. da, Lopes-Kulishev, C. O., et al. (2017). Molecular characterization of Caulobacter crescentus mutator strains. Gene, 626, 251-257. doi:10.1016/j.gene.2017.05.038
NLM
Martins-Pinheiro M, Oliveira AR, Valencia AO, Fernández-Silva FS, Silva LG da, Lopes-Kulishev CO, Italiani VC da S, Marques M do V, Menck CFM, Galhardo R da S. Molecular characterization of Caulobacter crescentus mutator strains [Internet]. Gene. 2017 ; 626 251-257.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.gene.2017.05.038
Vancouver
Martins-Pinheiro M, Oliveira AR, Valencia AO, Fernández-Silva FS, Silva LG da, Lopes-Kulishev CO, Italiani VC da S, Marques M do V, Menck CFM, Galhardo R da S. Molecular characterization of Caulobacter crescentus mutator strains [Internet]. Gene. 2017 ; 626 251-257.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.gene.2017.05.038
Artigo de periodico
Autophagy roles in the modulation of DNA repair pathways (2017)
Gomes, Luciana Rodrigues; Menck, Carlos Frederico Martins ; Leandro, Giovana da Silva
Fonte: International Journal of Molecular Sciences. Unidade: ICB
Assuntos: MICROBIOLOGIA, REPARAÇÃO DE DNA, CÉLULAS MORTAS, DNA RECOMBINANTE, NUCLEOTÍDEOS, HOMEOSTASE
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ABNT
GOMES, Luciana Rodrigues e MENCK, Carlos Frederico Martins e LEANDRO, Giovana da Silva. Autophagy roles in the modulation of DNA repair pathways. International Journal of Molecular Sciences, v. 18, n. 11, p. 1-21, 2017Tradução . . Disponível em: https://doi.org/10.3390/ijms18112351. Acesso em: 09 ago. 2024.
APA
Gomes, L. R., Menck, C. F. M., & Leandro, G. da S. (2017). Autophagy roles in the modulation of DNA repair pathways. International Journal of Molecular Sciences, 18( 11), 1-21. doi:10.3390/ijms18112351
NLM
Gomes LR, Menck CFM, Leandro G da S. Autophagy roles in the modulation of DNA repair pathways [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 11): 1-21.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3390/ijms18112351
Vancouver
Gomes LR, Menck CFM, Leandro G da S. Autophagy roles in the modulation of DNA repair pathways [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 11): 1-21.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3390/ijms18112351
Artigo de periodico
Major roles for pyrimidine dimers, nucleotide excision repair, and ATR in the alternative splicing response to UV irradiation (2017)
Muñoz, Manuel J.; Moreno, Nicolás Nieto; Giono, Luciana E.; Botto, Adrián E. Cambindo; Dujardin, Gwendal; Bastianello, Giulia; Lavore, Stefania; Torres-Méndez, Antonio; Menck, Carlos Frederico Martins ; Blencowe, Benjamin J.; Irimia, Manuel; Foiani, Marco; Kornblihtt, Alberto R.
Fonte: Cell Reports. Unidade: ICB
Assuntos: MICROBIOLOGIA, NUCLEOTÍDEOS, BENZENO, RADIAÇÃO ULTRAVIOLETA, RNA POLIMERASES, DANO AO DNA
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ABNT
MUÑOZ, Manuel J. et al. Major roles for pyrimidine dimers, nucleotide excision repair, and ATR in the alternative splicing response to UV irradiation. Cell Reports, v. 18, n. 12, p. 2868-2879, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2017.02.066. Acesso em: 09 ago. 2024.
APA
Muñoz, M. J., Moreno, N. N., Giono, L. E., Botto, A. E. C., Dujardin, G., Bastianello, G., et al. (2017). Major roles for pyrimidine dimers, nucleotide excision repair, and ATR in the alternative splicing response to UV irradiation. Cell Reports, 18( 12), 2868-2879. doi:10.1016/j.celrep.2017.02.066
NLM
Muñoz MJ, Moreno NN, Giono LE, Botto AEC, Dujardin G, Bastianello G, Lavore S, Torres-Méndez A, Menck CFM, Blencowe BJ, Irimia M, Foiani M, Kornblihtt AR. Major roles for pyrimidine dimers, nucleotide excision repair, and ATR in the alternative splicing response to UV irradiation [Internet]. Cell Reports. 2017 ; 18( 12): 2868-2879.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.celrep.2017.02.066
Vancouver
Muñoz MJ, Moreno NN, Giono LE, Botto AEC, Dujardin G, Bastianello G, Lavore S, Torres-Méndez A, Menck CFM, Blencowe BJ, Irimia M, Foiani M, Kornblihtt AR. Major roles for pyrimidine dimers, nucleotide excision repair, and ATR in the alternative splicing response to UV irradiation [Internet]. Cell Reports. 2017 ; 18( 12): 2868-2879.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.celrep.2017.02.066
Artigo de periodico
Chaperone-mediated autophagy prevents cellular transformationby regulating MYC proteasomal degradation (2017)
Gomes, Luciana Rodrigues; Menck, Carlos Frederico Martins ; Cuervo, Ana Maria
Fonte: Autophagy. Unidade: ICB
Assuntos: MICROBIOLOGIA, LISOSSOMOS, CÉLULAS CULTIVADAS DE TUMOR, ONCOGENES, GENÉTICA MOLECULAR, METABOLISMO DE PROTEÍNA, PROTEÍNAS
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GOMES, Luciana Rodrigues e MENCK, Carlos Frederico Martins e CUERVO, Ana Maria. Chaperone-mediated autophagy prevents cellular transformationby regulating MYC proteasomal degradation. Autophagy, v. 13, n. 5, p. 928-940, 2017Tradução . . Disponível em: https://doi.org/10.1080/15548627.2017.1293767. Acesso em: 09 ago. 2024.
APA
Gomes, L. R., Menck, C. F. M., & Cuervo, A. M. (2017). Chaperone-mediated autophagy prevents cellular transformationby regulating MYC proteasomal degradation. Autophagy, 13( 5), 928-940. doi:10.1080/15548627.2017.1293767
NLM
Gomes LR, Menck CFM, Cuervo AM. Chaperone-mediated autophagy prevents cellular transformationby regulating MYC proteasomal degradation [Internet]. Autophagy. 2017 ; 13( 5): 928-940.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1080/15548627.2017.1293767
Vancouver
Gomes LR, Menck CFM, Cuervo AM. Chaperone-mediated autophagy prevents cellular transformationby regulating MYC proteasomal degradation [Internet]. Autophagy. 2017 ; 13( 5): 928-940.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1080/15548627.2017.1293767
Artigo de periodico
Sunlight damage to cellular DNA: focus on oxidatively generated lesions (2017)
Schuch, André Passaglia; Moreno, Natália Cestari; Schuch, Natielen Jacques; Menck, Carlos Frederico Martins ; Garcia, Camila Carrião Machado
Fonte: Free Radical Biology and Medicine. Unidade: ICB
Assuntos: MICROBIOLOGIA, DANO AO DNA, RADIAÇÃO ULTRAVIOLETA, OXIDAÇÃO, MUTAGÊNESE, UVA, CARCINOGÊNESE
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ABNT
SCHUCH, André Passaglia et al. Sunlight damage to cellular DNA: focus on oxidatively generated lesions. Free Radical Biology and Medicine, v. 107, p. 110-124, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2017.01.029. Acesso em: 09 ago. 2024.
APA
Schuch, A. P., Moreno, N. C., Schuch, N. J., Menck, C. F. M., & Garcia, C. C. M. (2017). Sunlight damage to cellular DNA: focus on oxidatively generated lesions. Free Radical Biology and Medicine, 107, 110-124. doi:10.1016/j.freeradbiomed.2017.01.029
NLM
Schuch AP, Moreno NC, Schuch NJ, Menck CFM, Garcia CCM. Sunlight damage to cellular DNA: focus on oxidatively generated lesions [Internet]. Free Radical Biology and Medicine. 2017 ; 107 110-124.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2017.01.029
Vancouver
Schuch AP, Moreno NC, Schuch NJ, Menck CFM, Garcia CCM. Sunlight damage to cellular DNA: focus on oxidatively generated lesions [Internet]. Free Radical Biology and Medicine. 2017 ; 107 110-124.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2017.01.029
Artigo de periodico
Evolutionary and functional relationships of the dha regulon by genomic context analysis (2016)
Martins-Pinheiro, Marinalva; Lima, Wanessa C.; Asif, Huma; Oller, Cláudio A.; Menck, Carlos Frederico Martins
Fonte: PLOS ONE. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
MARTINS-PINHEIRO, Marinalva et al. Evolutionary and functional relationships of the dha regulon by genomic context analysis. PLOS ONE, v. 11, n. 3, p. 1-13, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0150772. Acesso em: 09 ago. 2024.
APA
Martins-Pinheiro, M., Lima, W. C., Asif, H., Oller, C. A., & Menck, C. F. M. (2016). Evolutionary and functional relationships of the dha regulon by genomic context analysis. PLOS ONE, 11( 3), 1-13. doi:10.1371/journal.pone.0150772
NLM
Martins-Pinheiro M, Lima WC, Asif H, Oller CA, Menck CFM. Evolutionary and functional relationships of the dha regulon by genomic context analysis [Internet]. PLOS ONE. 2016 ; 11( 3): 1-13.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0150772
Vancouver
Martins-Pinheiro M, Lima WC, Asif H, Oller CA, Menck CFM. Evolutionary and functional relationships of the dha regulon by genomic context analysis [Internet]. PLOS ONE. 2016 ; 11( 3): 1-13.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0150772
Artigo de periodico
Microenvironment and autophagy cross-talk: implications in cancer therapy (2016)
Gomes, Luciana R.; Vessoni, Alexandre T.; Menck, Carlos Frederico Martins
Fonte: Pharmacological Research. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
GOMES, Luciana R. e VESSONI, Alexandre T. e MENCK, Carlos Frederico Martins. Microenvironment and autophagy cross-talk: implications in cancer therapy. Pharmacological Research, v. 107, p. 300-307, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.03.031. Acesso em: 09 ago. 2024.
APA
Gomes, L. R., Vessoni, A. T., & Menck, C. F. M. (2016). Microenvironment and autophagy cross-talk: implications in cancer therapy. Pharmacological Research, 107, 300-307. doi:10.1016/j.phrs.2016.03.031
NLM
Gomes LR, Vessoni AT, Menck CFM. Microenvironment and autophagy cross-talk: implications in cancer therapy [Internet]. Pharmacological Research. 2016 ; 107 300-307.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.phrs.2016.03.031
Vancouver
Gomes LR, Vessoni AT, Menck CFM. Microenvironment and autophagy cross-talk: implications in cancer therapy [Internet]. Pharmacological Research. 2016 ; 107 300-307.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.phrs.2016.03.031
Artigo de periodico
DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes (2015)
Andrade-Lima, Leonardo Carmo de; Veloso, Artur; Paulsen, Michelle T.; Menck, Carlos Frederico Martins ; Ljungman, Mats
Fonte: Nucleic Acids Research. Unidade: ICB
Assuntos: MICROBIOLOGIA, DNA, RNA, GENES, RNA POLIMERASES
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ABNT
ANDRADE-LIMA, Leonardo Carmo de et al. DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes. Nucleic Acids Research, v. 43, n. 5, p. 2744-2756, 2015Tradução . . Disponível em: https://doi.org/10.1093/nar/gkv148. Acesso em: 09 ago. 2024.
APA
Andrade-Lima, L. C. de, Veloso, A., Paulsen, M. T., Menck, C. F. M., & Ljungman, M. (2015). DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes. Nucleic Acids Research, 43( 5), 2744-2756. doi:10.1093/nar/gkv148
NLM
Andrade-Lima LC de, Veloso A, Paulsen MT, Menck CFM, Ljungman M. DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes [Internet]. Nucleic Acids Research. 2015 ; 43( 5): 2744-2756.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1093/nar/gkv148
Vancouver
Andrade-Lima LC de, Veloso A, Paulsen MT, Menck CFM, Ljungman M. DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes [Internet]. Nucleic Acids Research. 2015 ; 43( 5): 2744-2756.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1093/nar/gkv148
Artigo de periodico
Mutation in PNKP presenting initially as axonal Charcot-Marie-Tooth disease (2015)
Pedroso, José Luiz; Rocha, Clarissa R. R.; Macedo-Souza, Lucia I.; Mario, Vitor de; Marques Junior, Wilson; Barsottini, Orlando G. P.; Oliveira, Acary S. Bulle; Menck, Carlos Frederico Martins ; Kok, Fernando
Fonte: Neurology: Genetics. Unidades: ICB, FM
Assunto: MICROBIOLOGIA
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ABNT
PEDROSO, José Luiz et al. Mutation in PNKP presenting initially as axonal Charcot-Marie-Tooth disease. Neurology: Genetics, v. 1, n. 4, p. 1-3, 2015Tradução . . Disponível em: https://doi.org/10.1212/NXG.0000000000000030. Acesso em: 09 ago. 2024.
APA
Pedroso, J. L., Rocha, C. R. R., Macedo-Souza, L. I., Mario, V. de, Marques Junior, W., Barsottini, O. G. P., et al. (2015). Mutation in PNKP presenting initially as axonal Charcot-Marie-Tooth disease. Neurology: Genetics, 1( 4), 1-3. doi:10.1212/NXG.0000000000000030
NLM
Pedroso JL, Rocha CRR, Macedo-Souza LI, Mario V de, Marques Junior W, Barsottini OGP, Oliveira ASB, Menck CFM, Kok F. Mutation in PNKP presenting initially as axonal Charcot-Marie-Tooth disease [Internet]. Neurology: Genetics. 2015 ; 1( 4): 1-3.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1212/NXG.0000000000000030
Vancouver
Pedroso JL, Rocha CRR, Macedo-Souza LI, Mario V de, Marques Junior W, Barsottini OGP, Oliveira ASB, Menck CFM, Kok F. Mutation in PNKP presenting initially as axonal Charcot-Marie-Tooth disease [Internet]. Neurology: Genetics. 2015 ; 1( 4): 1-3.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1212/NXG.0000000000000030
Artigo de periodico
ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways (2015)
Andrade-Lima, Leonardo Carmo de; Andrade, Luciana N.; Menck, Carlos Frederico Martins
Fonte: Journal of Cell Science. Unidade: ICB
Assuntos: MICROBIOLOGIA, APOPTOSE
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ABNT
ANDRADE-LIMA, Leonardo Carmo de e ANDRADE, Luciana N. e MENCK, Carlos Frederico Martins. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways. Journal of Cell Science, v. 28, n. 1, p. 150-159, 2015Tradução . . Disponível em: https://doi.org/10.1242/jcs.161596. Acesso em: 09 ago. 2024.
APA
Andrade-Lima, L. C. de, Andrade, L. N., & Menck, C. F. M. (2015). ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways. Journal of Cell Science, 28( 1), 150-159. doi:10.1242/jcs.161596
NLM
Andrade-Lima LC de, Andrade LN, Menck CFM. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways [Internet]. Journal of Cell Science. 2015 ; 28( 1): 150-159.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1242/jcs.161596
Vancouver
Andrade-Lima LC de, Andrade LN, Menck CFM. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways [Internet]. Journal of Cell Science. 2015 ; 28( 1): 150-159.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1242/jcs.161596
Artigo de periodico
Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome (2014)
Quinet, Annabel; Vessoni, Alexandre Teixeira; Rocha, Clarissa R. R.; Gottifredi, Vanessa; Biard, Denis; Sarasin, Alain; Menck, Carlos Frederico Martins ; Stary, Anne
Fonte: DNA Repair. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
QUINET, Annabel et al. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, v. 14, p. 27-38, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2013.12.005. Acesso em: 09 ago. 2024.
APA
Quinet, A., Vessoni, A. T., Rocha, C. R. R., Gottifredi, V., Biard, D., Sarasin, A., et al. (2014). Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, 14, 27-38. doi:10.1016/j.dnarep.2013.12.005
NLM
Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
Vancouver
Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
Artigo de periodico
DNA repair diseases: what do they tell us about cancer and aging? (2014)
Menck, Carlos Frederico Martins ; Munford, Veridiana
Fonte: Genetics and Molecular Biology. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
MENCK, Carlos Frederico Martins e MUNFORD, Veridiana. DNA repair diseases: what do they tell us about cancer and aging?. Genetics and Molecular Biology, v. 37, n. 1 (Suppl), p. 220-233, 2014Tradução . . Disponível em: https://doi.org/10.1590/s1415-47572014000200008. Acesso em: 09 ago. 2024.
APA
Menck, C. F. M., & Munford, V. (2014). DNA repair diseases: what do they tell us about cancer and aging? Genetics and Molecular Biology, 37( 1 (Suppl), 220-233. doi:10.1590/s1415-47572014000200008
NLM
Menck CFM, Munford V. DNA repair diseases: what do they tell us about cancer and aging? [Internet]. Genetics and Molecular Biology. 2014 ; 37( 1 (Suppl): 220-233.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/s1415-47572014000200008
Vancouver
Menck CFM, Munford V. DNA repair diseases: what do they tell us about cancer and aging? [Internet]. Genetics and Molecular Biology. 2014 ; 37( 1 (Suppl): 220-233.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/s1415-47572014000200008
Artigo de periodico
Glutathione depletion sensitizes cisplatin- and temozolomide-resistant glioma cells in vitro and in vivo (2014)
Rocha, C. R. R.; Garcia, C. C. M.; Vieira, D. B.; Quinet, A.; Andrade-Lima, L. C. de; Munford, Veridiana; Belizário, Jose Ernesto; Menck, Carlos Frederico Martins
Fonte: Cell Death and Diseases. Unidade: ICB
Assuntos: FARMACOLOGIA, MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROCHA, C. R. R. et al. Glutathione depletion sensitizes cisplatin- and temozolomide-resistant glioma cells in vitro and in vivo. Cell Death and Diseases, v. 5, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1038/cddis.2014.465. Acesso em: 09 ago. 2024.
APA
Rocha, C. R. R., Garcia, C. C. M., Vieira, D. B., Quinet, A., Andrade-Lima, L. C. de, Munford, V., et al. (2014). Glutathione depletion sensitizes cisplatin- and temozolomide-resistant glioma cells in vitro and in vivo. Cell Death and Diseases, 5, 1-10. doi:10.1038/cddis.2014.465
NLM
Rocha CRR, Garcia CCM, Vieira DB, Quinet A, Andrade-Lima LC de, Munford V, Belizário JE, Menck CFM. Glutathione depletion sensitizes cisplatin- and temozolomide-resistant glioma cells in vitro and in vivo [Internet]. Cell Death and Diseases. 2014 ; 5 1-10.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1038/cddis.2014.465
Vancouver
Rocha CRR, Garcia CCM, Vieira DB, Quinet A, Andrade-Lima LC de, Munford V, Belizário JE, Menck CFM. Glutathione depletion sensitizes cisplatin- and temozolomide-resistant glioma cells in vitro and in vivo [Internet]. Cell Death and Diseases. 2014 ; 5 1-10.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1038/cddis.2014.465
Artigo de periodico
Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells (2013)
Vernhes, Marioly; González-Pumariega, Maribel; Andrade, Luciana; Schuch, Andre Passaglia; Lima-Bessa, Keronninn Moreno de; Menck, Carlos Frederico Martins ; Sánchez-Lamar, Angel
Fonte: Pharmaceutical Biology. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VERNHES, Marioly et al. Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells. Pharmaceutical Biology, v. 51, n. 1, p. 1-7, 2013Tradução . . Disponível em: https://doi.org/10.3109/13880209.2012.695800. Acesso em: 09 ago. 2024.
APA
Vernhes, M., González-Pumariega, M., Andrade, L., Schuch, A. P., Lima-Bessa, K. M. de, Menck, C. F. M., & Sánchez-Lamar, A. (2013). Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells. Pharmaceutical Biology, 51( 1), 1-7. doi:10.3109/13880209.2012.695800
NLM
Vernhes M, González-Pumariega M, Andrade L, Schuch AP, Lima-Bessa KM de, Menck CFM, Sánchez-Lamar A. Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells [Internet]. Pharmaceutical Biology. 2013 ; 51( 1): 1-7.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3109/13880209.2012.695800
Vancouver
Vernhes M, González-Pumariega M, Andrade L, Schuch AP, Lima-Bessa KM de, Menck CFM, Sánchez-Lamar A. Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells [Internet]. Pharmaceutical Biology. 2013 ; 51( 1): 1-7.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3109/13880209.2012.695800
Artigo de periodico
Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress (2013)
Soltys, Daniela Tathiana; Rocha, Clarissa R. R.; Lerner, Letícia K.; Souza, Tiago Antonio de; Munford, Veridiana; Cabral, Fernanda; Nardo, Tiziana; Stefanini, Miria; Sarasin, Alain; Cabral-Neto, Januário B.; Menck, Carlos Frederico Martins
Fonte: Human Mutation. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOLTYS, Daniela Tathiana et al. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, v. 34, n. 3, p. 481-489, 2013Tradução . . Disponível em: https://doi.org/10.1002/humu.22259. Acesso em: 09 ago. 2024.
APA
Soltys, D. T., Rocha, C. R. R., Lerner, L. K., Souza, T. A. de, Munford, V., Cabral, F., et al. (2013). Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, 34( 3), 481-489. doi:10.1002/humu.22259
NLM
Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1002/humu.22259
Vancouver
Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1002/humu.22259
Artigo de periodico
UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts (2013)
Ortolan, Tatiana Grohmann; Menck, Carlos Frederico Martins
Fonte: PLOS ONE. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ORTOLAN, Tatiana Grohmann e MENCK, Carlos Frederico Martins. UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts. PLOS ONE, v. 8, n. 10, p. 1-82013, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076936. Acesso em: 09 ago. 2024.
APA
Ortolan, T. G., & Menck, C. F. M. (2013). UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts. PLOS ONE, 8( 10), 1-82013. doi:10.1371/journal.pone.0076936
NLM
Ortolan TG, Menck CFM. UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts [Internet]. PLOS ONE. 2013 ; 8( 10): 1-82013.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0076936
Vancouver
Ortolan TG, Menck CFM. UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts [Internet]. PLOS ONE. 2013 ; 8( 10): 1-82013.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0076936
Artigo de periodico
The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation (2013)
Beckedorff, Felipe Cesar Ferrarezi; Ayupe, Ana Carolina; Crocci-Souza, Renan; Amaral, Murilo Sena; Nakaya, Helder Takashi Imoto ; Soltys, Daniela Tathiana; Menck, Carlos Frederico Martins ; Reis, Eduardo Moraes ; Verjovski-Almeida, Sergio
Fonte: PLOS Genetics. Unidades: IQ, ICB
Assuntos: PROLIFERAÇÃO CELULAR, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BECKEDORFF, Felipe Cesar Ferrarezi et al. The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation. PLOS Genetics, v. 9, n. 8, p. 1-15, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pgen.1003705. Acesso em: 09 ago. 2024.
APA
Beckedorff, F. C. F., Ayupe, A. C., Crocci-Souza, R., Amaral, M. S., Nakaya, H. T. I., Soltys, D. T., et al. (2013). The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation. PLOS Genetics, 9( 8), 1-15. doi:10.1371/journal.pgen.1003705
NLM
Beckedorff FCF, Ayupe AC, Crocci-Souza R, Amaral MS, Nakaya HTI, Soltys DT, Menck CFM, Reis EM, Verjovski-Almeida S. The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation [Internet]. PLOS Genetics. 2013 ; 9( 8): 1-15.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pgen.1003705
Vancouver
Beckedorff FCF, Ayupe AC, Crocci-Souza R, Amaral MS, Nakaya HTI, Soltys DT, Menck CFM, Reis EM, Verjovski-Almeida S. The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation [Internet]. PLOS Genetics. 2013 ; 9( 8): 1-15.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pgen.1003705

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