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Artigo de periodico
Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway (2018)
Senger, Nathalia ; Melo, Marcos Barrouin; Diniz, Gabriela Placoná ; Santos, Maria José Campagnole dos ; Santos, Robson Augusto Souza dos; Barreto-Chaves, Maria Luiza Morais
Source: Clinical Science. Unidade: ICB
Subjects: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SENGER, Nathalia et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, v. 132, n. 11, p. 1117-1133, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20171606. Acesso em: 05 jul. 2024.
APA
Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Barreto-Chaves, M. L. M. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
NLM
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1042/CS20171606
Vancouver
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1042/CS20171606
Artigo de periodico
MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone (2017)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Moreno, Camila Rodrigues; Senger, Nathalia; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Cellular Physiology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, RNA DE TRANSFERÊNCIA, SISTEMA RENINA-ANGIOTENSINA, RATOS WISTAR, BLOQUEIO CARDÍACO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, v. 232, n. 12, p. 3360-3368, 2017Tradução . . Disponível em: https://doi.org/10.1002/jcp.25781. Acesso em: 05 jul. 2024.
APA
Diniz, G. P., Lino, C. A., Moreno, C. R., Senger, N., & Barreto-Chaves, M. L. M. (2017). MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, 232( 12), 3360-3368. doi:10.1002/jcp.25781
NLM
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/jcp.25781
Vancouver
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/jcp.25781
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 05 jul. 2024.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1042/CS20171368
Artigo de periodico
Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy (2016)
Diniz, Gabriela Placoná ; Senger, Nathalia; Carneiro-Ramos, Marcela S.; Santos, Robson A. S.; Barreto-Chaves, Maria Luiza Morais
Source: Therapeutic Advances in Cardiovascular Disease. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, v. 10, n. 4, p. 192-202, 2016Tradução . . Disponível em: https://doi.org/10.1177/1753944715623228. Acesso em: 05 jul. 2024.
APA
Diniz, G. P., Senger, N., Carneiro-Ramos, M. S., Santos, R. A. S., & Barreto-Chaves, M. L. M. (2016). Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, 10( 4), 192-202. doi:10.1177/1753944715623228
NLM
Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1177/1753944715623228
Vancouver
Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1177/1753944715623228
Artigo de periodico
Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor (2015)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Guedes, Elaine Castilho; Moreira, Luana do Nascimento; Barreto-Chaves, Maria Luiza Morais
Source: Basic Research in Cardiology. Unidade: ICB
Subjects: ANATOMIA, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, ANGIOTENSINA II
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, v. 110, n. 5, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00395-015-0504-7. Acesso em: 05 jul. 2024.
APA
Diniz, G. P., Lino, C. A., Guedes, E. C., Moreira, L. do N., & Barreto-Chaves, M. L. M. (2015). Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, 110( 5), 1-11. doi:10.1007/s00395-015-0504-7
NLM
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Vancouver
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Artigo de periodico
AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth (2013)
Takano, Ana Paula Cremasco ; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAKANO, Ana Paula Cremasco e DINIZ, Gabriela Placoná e BARRETO-CHAVES, Maria Luiza Morais. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, v. 376, n. 1-2, p. 43-50, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.05.024. Acesso em: 05 jul. 2024.
APA
Takano, A. P. C., Diniz, G. P., & Barreto-Chaves, M. L. M. (2013). AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, 376( 1-2), 43-50. doi:10.1016/j.mce.2013.05.024
NLM
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Vancouver
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Artigo de periodico
miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation (2013)
Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná e TAKANO, Ana Paula Cremasco e BARRETO-CHAVES, Maria Luiza Morais. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, v. 374, n. 1-2, p. 117-124, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.04.010. Acesso em: 05 jul. 2024.
APA
Diniz, G. P., Takano, A. P. C., & Barreto-Chaves, M. L. M. (2013). miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, 374( 1-2), 117-124. doi:10.1016/j.mce.2013.04.010
NLM
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Vancouver
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010

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