Filtros : "Indexado no Index Medicus" "BENDHACK, LUSIANE MARIA" Removidos: "Nutrição em Saúde Pública" "Cunha, Fernando de Queiroz" "LOTUFO, PAULO ANDRADE" Limpar

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  • Source: Autonomic Neuroscience: Basic and Clinical. Unidades: FCFRP, FMRP

    Subjects: ANÓXIA (PATOLOGIA), PRESSÃO SANGUÍNEA, REGULAÇÃO GÊNICA, ÓXIDO NÍTRICO, MODELOS ANIMAIS DE DOENÇAS

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    • ABNT

      PAJOLLA, Gisela P. et al. Immunoreactivity for neuronal NOS and fluorescent indication of NO formation in the NTS of juvenile rats submitted to chronic intermittent hypoxia. Autonomic Neuroscience: Basic and Clinical, v. 148, n. 1-2, p. 55-62, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2009.03.003. Acesso em: 02 jul. 2024.
    • APA

      Pajolla, G. P., Accorsi Mendonça, D., Lunardi, C. N., Bendhack, L. M., Machado, B. H., & Llewellyn Smith, I. J. (2009). Immunoreactivity for neuronal NOS and fluorescent indication of NO formation in the NTS of juvenile rats submitted to chronic intermittent hypoxia. Autonomic Neuroscience: Basic and Clinical, 148( 1-2), 55-62. doi:10.1016/j.autneu.2009.03.003
    • NLM

      Pajolla GP, Accorsi Mendonça D, Lunardi CN, Bendhack LM, Machado BH, Llewellyn Smith IJ. Immunoreactivity for neuronal NOS and fluorescent indication of NO formation in the NTS of juvenile rats submitted to chronic intermittent hypoxia [Internet]. Autonomic Neuroscience: Basic and Clinical. 2009 ; 148( 1-2): 55-62.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.autneu.2009.03.003
    • Vancouver

      Pajolla GP, Accorsi Mendonça D, Lunardi CN, Bendhack LM, Machado BH, Llewellyn Smith IJ. Immunoreactivity for neuronal NOS and fluorescent indication of NO formation in the NTS of juvenile rats submitted to chronic intermittent hypoxia [Internet]. Autonomic Neuroscience: Basic and Clinical. 2009 ; 148( 1-2): 55-62.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.autneu.2009.03.003
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: FCFRP

    Subjects: VASODILATAÇÃO, ÓXIDO NÍTRICO, RUTÊNIO

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    • ABNT

      LUNARDI, C. N. e SILVA, R. S. e BENDHACK, Lusiane Maria. New nitric oxide donors based on ruthenium complexes. Brazilian Journal of Medical and Biological Research, v. 42, n. 1, p. 87-93, 2009Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2009000100013. Acesso em: 02 jul. 2024.
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      Lunardi, C. N., Silva, R. S., & Bendhack, L. M. (2009). New nitric oxide donors based on ruthenium complexes. Brazilian Journal of Medical and Biological Research, 42( 1), 87-93. doi:10.1590/s0100-879x2009000100013
    • NLM

      Lunardi CN, Silva RS, Bendhack LM. New nitric oxide donors based on ruthenium complexes [Internet]. Brazilian Journal of Medical and Biological Research. 2009 ; 42( 1): 87-93.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/s0100-879x2009000100013
    • Vancouver

      Lunardi CN, Silva RS, Bendhack LM. New nitric oxide donors based on ruthenium complexes [Internet]. Brazilian Journal of Medical and Biological Research. 2009 ; 42( 1): 87-93.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/s0100-879x2009000100013
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Subjects: VASODILATAÇÃO, ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS

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      OLIVEIRA, Ana P. S. et al. Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells. Vascular Pharmacology, v. 50, n. 5-6, p. 153-159, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2008.11.010. Acesso em: 02 jul. 2024.
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      Oliveira, A. P. S., Lunardi, C. N., Rodrigues, G. J., & Bendhack, L. M. (2009). Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells. Vascular Pharmacology, 50( 5-6), 153-159. doi:10.1016/j.vph.2008.11.010
    • NLM

      Oliveira APS, Lunardi CN, Rodrigues GJ, Bendhack LM. Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells [Internet]. Vascular Pharmacology. 2009 ; 50( 5-6): 153-159.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.vph.2008.11.010
    • Vancouver

      Oliveira APS, Lunardi CN, Rodrigues GJ, Bendhack LM. Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells [Internet]. Vascular Pharmacology. 2009 ; 50( 5-6): 153-159.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.vph.2008.11.010
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Subjects: ENDOTÉLIO, AORTA, RETÍCULO SARCOPLÁSMICO

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      ROCHA, Matheus L. e BENDHACK, Lusiane Maria. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology, v. 50, n. 3-4, p. 98-103, 2009Tradução . . Acesso em: 02 jul. 2024.
    • APA

      Rocha, M. L., & Bendhack, L. M. (2009). Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology, 50( 3-4), 98-103.
    • NLM

      Rocha ML, Bendhack LM. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology. 2009 ; 50( 3-4): 98-103.[citado 2024 jul. 02 ]
    • Vancouver

      Rocha ML, Bendhack LM. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology. 2009 ; 50( 3-4): 98-103.[citado 2024 jul. 02 ]
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, ENDOTÉLIO VASCULAR, VASODILATAÇÃO

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      BONAVENTURA, Daniella et al. A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta. Nitric Oxide, v. 18, n. 4, p. 287-295, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2008.02.004. Acesso em: 02 jul. 2024.
    • APA

      Bonaventura, D., Lunardi, C. N., Rodrigues, G. J., Neto, M. A., & Bendhack, L. M. (2008). A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta. Nitric Oxide, 18( 4), 287-295. doi:10.1016/j.niox.2008.02.004
    • NLM

      Bonaventura D, Lunardi CN, Rodrigues GJ, Neto MA, Bendhack LM. A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta [Internet]. Nitric Oxide. 2008 ; 18( 4): 287-295.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2008.02.004
    • Vancouver

      Bonaventura D, Lunardi CN, Rodrigues GJ, Neto MA, Bendhack LM. A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta [Internet]. Nitric Oxide. 2008 ; 18( 4): 287-295.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2008.02.004
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, NEFROPATIAS, VITAMINA C

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      RODRIGUES, G. J. et al. Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats. Nitric Oxide, v. 18, n. 3, p. 176-183, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2007.12.002. Acesso em: 02 jul. 2024.
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      Rodrigues, G. J., Lunardi, C. N., Lima, R. G., Santos, C. X., Laurindo, F. R. M., Silva, R. S. da, & Bendhack, L. M. (2008). Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats. Nitric Oxide, 18( 3), 176-183. doi:10.1016/j.niox.2007.12.002
    • NLM

      Rodrigues GJ, Lunardi CN, Lima RG, Santos CX, Laurindo FRM, Silva RS da, Bendhack LM. Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats [Internet]. Nitric Oxide. 2008 ; 18( 3): 176-183.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2007.12.002
    • Vancouver

      Rodrigues GJ, Lunardi CN, Lima RG, Santos CX, Laurindo FRM, Silva RS da, Bendhack LM. Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats [Internet]. Nitric Oxide. 2008 ; 18( 3): 176-183.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2007.12.002
  • Source: Cardiovascular Research. Unidades: ICB, FCFRP

    Subjects: PRESSÃO SANGUÍNEA (VARIABILIDADE), DENERVAÇÃO

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      ROCHA, Matheus L. et al. Blood pressure variability increases connexin expression in the vascular smooth muscle of rats. Cardiovascular Research, v. 80, n. 1, p. 123-130, 2008Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvn178. Acesso em: 02 jul. 2024.
    • APA

      Rocha, M. L., Kihara, A. H., Davel, A. P., Britto, L. R. G. de, Rossoni, L. V., & Bendhack, L. M. (2008). Blood pressure variability increases connexin expression in the vascular smooth muscle of rats. Cardiovascular Research, 80( 1), 123-130. doi:10.1093/cvr/cvn178
    • NLM

      Rocha ML, Kihara AH, Davel AP, Britto LRG de, Rossoni LV, Bendhack LM. Blood pressure variability increases connexin expression in the vascular smooth muscle of rats [Internet]. Cardiovascular Research. 2008 ; 80( 1): 123-130.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1093/cvr/cvn178
    • Vancouver

      Rocha ML, Kihara AH, Davel AP, Britto LRG de, Rossoni LV, Bendhack LM. Blood pressure variability increases connexin expression in the vascular smooth muscle of rats [Internet]. Cardiovascular Research. 2008 ; 80( 1): 123-130.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1093/cvr/cvn178
  • Source: Basic and Clinical Pharmacology and Toxicology. Unidade: FCFRP

    Subjects: AORTA, PRESSÃO SANGUÍNEA, FARMACOLOGIA

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      ROCHA, Matheus L. e BENDHACK, Lusiane Maria. Aortas isolated from sinoaortic-denervated rats exhibit rhythmic contractions that are regulated by pharmacologically distinct calcium sources. Basic and Clinical Pharmacology and Toxicology, v. 102, n. 4, p. 352-359, 2008Tradução . . Acesso em: 02 jul. 2024.
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      Rocha, M. L., & Bendhack, L. M. (2008). Aortas isolated from sinoaortic-denervated rats exhibit rhythmic contractions that are regulated by pharmacologically distinct calcium sources. Basic and Clinical Pharmacology and Toxicology, 102( 4), 352-359.
    • NLM

      Rocha ML, Bendhack LM. Aortas isolated from sinoaortic-denervated rats exhibit rhythmic contractions that are regulated by pharmacologically distinct calcium sources. Basic and Clinical Pharmacology and Toxicology. 2008 ; 102( 4): 352-359.[citado 2024 jul. 02 ]
    • Vancouver

      Rocha ML, Bendhack LM. Aortas isolated from sinoaortic-denervated rats exhibit rhythmic contractions that are regulated by pharmacologically distinct calcium sources. Basic and Clinical Pharmacology and Toxicology. 2008 ; 102( 4): 352-359.[citado 2024 jul. 02 ]
  • Source: Atherosclerosis. Unidades: FCFRP, FORP, FMRP

    Subjects: HIPERTENSÃO, NEFROPATIAS, PRESSÃO SANGUÍNEA, DOENÇAS VASCULARES

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      CASTRO, Michele M. et al. Metalloproteinase inhibition ameliorates hypertension and prevents vascular dysfunction and remodeling in renovascular hypertensive rats. Atherosclerosis, v. 198, n. 2, p. 320-331, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.atherosclerosis.2007.10.011. Acesso em: 02 jul. 2024.
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      Castro, M. M., Rizzi, E., Figueiredo-Lopes, L., Fernandes, K., Bendhack, L. M., Pitol, D. L., et al. (2008). Metalloproteinase inhibition ameliorates hypertension and prevents vascular dysfunction and remodeling in renovascular hypertensive rats. Atherosclerosis, 198( 2), 320-331. doi:10.1016/j.atherosclerosis.2007.10.011
    • NLM

      Castro MM, Rizzi E, Figueiredo-Lopes L, Fernandes K, Bendhack LM, Pitol DL, Gerlach RF, Tanus-Santos JE. Metalloproteinase inhibition ameliorates hypertension and prevents vascular dysfunction and remodeling in renovascular hypertensive rats [Internet]. Atherosclerosis. 2008 ; 198( 2): 320-331.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2007.10.011
    • Vancouver

      Castro MM, Rizzi E, Figueiredo-Lopes L, Fernandes K, Bendhack LM, Pitol DL, Gerlach RF, Tanus-Santos JE. Metalloproteinase inhibition ameliorates hypertension and prevents vascular dysfunction and remodeling in renovascular hypertensive rats [Internet]. Atherosclerosis. 2008 ; 198( 2): 320-331.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2007.10.011
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Subjects: VASODILATAÇÃO, RUTÊNIO, ÓXIDO NÍTRICO

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      LUNARDI, Claure N. et al. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology, v. 47, n. 2-3, p. 139-144, 2007Tradução . . Acesso em: 02 jul. 2024.
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      Lunardi, C. N., Vercesi, J. A., Silva, R. S. da, & Bendhack, L. M. (2007). Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology, 47( 2-3), 139-144.
    • NLM

      Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology. 2007 ; 47( 2-3): 139-144.[citado 2024 jul. 02 ]
    • Vancouver

      Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology. 2007 ; 47( 2-3): 139-144.[citado 2024 jul. 02 ]
  • Source: European Journal of Pharmacology. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, VASODILATAÇÃO, FARMACOLOGIA EXPERIMENTAL

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      DIAS, Katy Lísias Gondim et al. Mechanisms involved in the vasodilator effect induced by diosgenin in rat superior mesenteric artery. European Journal of Pharmacology, v. 574, p. 172-178, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2007.07.017. Acesso em: 02 jul. 2024.
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      Dias, K. L. G., Correia, N. de A., Pereira, K. K. G., Barbosa Filho, J. M., Cavalcante, K. V. M., Araújo, I. G. A., et al. (2007). Mechanisms involved in the vasodilator effect induced by diosgenin in rat superior mesenteric artery. European Journal of Pharmacology, 574, 172-178. doi:10.1016/j.ejphar.2007.07.017
    • NLM

      Dias KLG, Correia N de A, Pereira KKG, Barbosa Filho JM, Cavalcante KVM, Araújo IGA, Silva DF, Guedes DN, Anjos Neto M dos, Bendhack LM, Medeiros IA. Mechanisms involved in the vasodilator effect induced by diosgenin in rat superior mesenteric artery [Internet]. European Journal of Pharmacology. 2007 ; 574 172-178.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2007.07.017
    • Vancouver

      Dias KLG, Correia N de A, Pereira KKG, Barbosa Filho JM, Cavalcante KVM, Araújo IGA, Silva DF, Guedes DN, Anjos Neto M dos, Bendhack LM, Medeiros IA. Mechanisms involved in the vasodilator effect induced by diosgenin in rat superior mesenteric artery [Internet]. European Journal of Pharmacology. 2007 ; 574 172-178.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2007.07.017
  • Source: Biological and Pharmaceutical Bulletin. Unidade: FCFRP

    Subjects: AORTA, CANAIS IÔNICOS

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      ROCHA, Matheus Lavorenti e BENDHACK, Lusiane Maria. Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin, v. 30, n. 11, p. 2098-2104, 2007Tradução . . Acesso em: 02 jul. 2024.
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      Rocha, M. L., & Bendhack, L. M. (2007). Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin, 30( 11), 2098-2104.
    • NLM

      Rocha ML, Bendhack LM. Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin. 2007 ; 30( 11): 2098-2104.[citado 2024 jul. 02 ]
    • Vancouver

      Rocha ML, Bendhack LM. Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin. 2007 ; 30( 11): 2098-2104.[citado 2024 jul. 02 ]
  • Source: Medicina. Unidade: FCFRP

    Subjects: FARMACOLOGIA, RECEPTORES ADRENÉRGICOS, VASODILATAÇÃO, ENDOTÉLIO VASCULAR

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      NETO, Mário A. e RASCADO, Ricardo R. e BENDHACK, Lusiane Maria. Receptores 'beta' adrenérgicos no sistema cardiovascular. Medicina, v. 39, p. 3-12, 2006Tradução . . Acesso em: 02 jul. 2024.
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      Neto, M. A., Rascado, R. R., & Bendhack, L. M. (2006). Receptores 'beta' adrenérgicos no sistema cardiovascular. Medicina, 39, 3-12.
    • NLM

      Neto MA, Rascado RR, Bendhack LM. Receptores 'beta' adrenérgicos no sistema cardiovascular. Medicina. 2006 ; 39 3-12.[citado 2024 jul. 02 ]
    • Vancouver

      Neto MA, Rascado RR, Bendhack LM. Receptores 'beta' adrenérgicos no sistema cardiovascular. Medicina. 2006 ; 39 3-12.[citado 2024 jul. 02 ]
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, RUTÊNIO, FARMACOLOGIA

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      LUNARDI, Claure Nain et al. Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells. Nitric Oxide, v. 15, p. 252-258, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2006.02.001. Acesso em: 02 jul. 2024.
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      Lunardi, C. N., Cacciari, A. L., Silva, S. R., & Bendhack, L. M. (2006). Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells. Nitric Oxide, 15, 252-258. doi:10.1016/j.niox.2006.02.001
    • NLM

      Lunardi CN, Cacciari AL, Silva SR, Bendhack LM. Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells [Internet]. Nitric Oxide. 2006 ; 15 252-258.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2006.02.001
    • Vancouver

      Lunardi CN, Cacciari AL, Silva SR, Bendhack LM. Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells [Internet]. Nitric Oxide. 2006 ; 15 252-258.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2006.02.001
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, RUTÊNIO, VASODILATAÇÃO, FARMACOLOGIA

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      BONAVENTURA, Daniella et al. Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex. Nitric Oxide, v. 15, n. 4, p. 387-394, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2006.04.260. Acesso em: 02 jul. 2024.
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      Bonaventura, D., Oliveira, F. de S., Lunardi, C. N., Vercesi, J. A., Silva, R. S. da, & Bendhack, L. M. (2006). Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex. Nitric Oxide, 15( 4), 387-394. doi:10.1016/j.niox.2006.04.260
    • NLM

      Bonaventura D, Oliveira F de S, Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex [Internet]. Nitric Oxide. 2006 ; 15( 4): 387-394.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2006.04.260
    • Vancouver

      Bonaventura D, Oliveira F de S, Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex [Internet]. Nitric Oxide. 2006 ; 15( 4): 387-394.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.niox.2006.04.260
  • Source: Nitric Oxide. Unidades: FCFRP, FFCLRP

    Subjects: FARMACOLOGIA, CARDIOLOGIA

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      FEREZIN, Camila Z. et al. The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide, v. 13, p. 170-175, 2005Tradução . . Acesso em: 02 jul. 2024.
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      Ferezin, C. Z., Oliveira, F. S., Silva, R. S. da, Simioni, A. R., Tedesco, A. C., & Bendhack, L. M. (2005). The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide, 13, 170-175.
    • NLM

      Ferezin CZ, Oliveira FS, Silva RS da, Simioni AR, Tedesco AC, Bendhack LM. The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide. 2005 ; 13 170-175.[citado 2024 jul. 02 ]
    • Vancouver

      Ferezin CZ, Oliveira FS, Silva RS da, Simioni AR, Tedesco AC, Bendhack LM. The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide. 2005 ; 13 170-175.[citado 2024 jul. 02 ]
  • Source: European Journal of Pharmacology. Unidade: FCFRP

    Subjects: FARMACOLOGIA, CARDIOLOGIA

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      MOLIN, Josiane C. e SGUILLA, Fabiana S. e BENDHACK, Lusiane Maria. Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium. European Journal of Pharmacology, v. 522, p. 94-99, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2005.08.034. Acesso em: 02 jul. 2024.
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      Molin, J. C., Sguilla, F. S., & Bendhack, L. M. (2005). Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium. European Journal of Pharmacology, 522, 94-99. doi:10.1016/j.ejphar.2005.08.034
    • NLM

      Molin JC, Sguilla FS, Bendhack LM. Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium [Internet]. European Journal of Pharmacology. 2005 ; 522 94-99.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2005.08.034
    • Vancouver

      Molin JC, Sguilla FS, Bendhack LM. Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium [Internet]. European Journal of Pharmacology. 2005 ; 522 94-99.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2005.08.034
  • Source: Comparative Biochemistry and Physiology Part C. Unidades: FCFRP, FMRP

    Subjects: TOXICOLOGIA, FARMACOLOGIA CLÍNICA

    Acesso à fonteDOIHow to cite
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    • ABNT

      VASCONCELOS, Flávio et al. Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines. Comparative Biochemistry and Physiology Part C, v. 141, p. 85-92, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.cca.2005.05.012. Acesso em: 02 jul. 2024.
    • APA

      Vasconcelos, F., Lanchote, V. L., Bendhack, L. M., Giglio, J. R., Sampaio, S. V., & Arantes, E. C. (2005). Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines. Comparative Biochemistry and Physiology Part C, 141, 85-92. doi:10.1016/j.cca.2005.05.012
    • NLM

      Vasconcelos F, Lanchote VL, Bendhack LM, Giglio JR, Sampaio SV, Arantes EC. Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines [Internet]. Comparative Biochemistry and Physiology Part C. 2005 ; 141 85-92.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.cca.2005.05.012
    • Vancouver

      Vasconcelos F, Lanchote VL, Bendhack LM, Giglio JR, Sampaio SV, Arantes EC. Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines [Internet]. Comparative Biochemistry and Physiology Part C. 2005 ; 141 85-92.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.cca.2005.05.012
  • Source: Pharmacology. Unidade: FCFRP

    Assunto: FARMACOLOGIA

    How to cite
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    • ABNT

      GRIFONI, Samira de Campos e BENDHACK, Lusiane Maria. Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology, v. 70, p. 160-168, 2004Tradução . . Acesso em: 02 jul. 2024.
    • APA

      Grifoni, S. de C., & Bendhack, L. M. (2004). Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology, 70, 160-168.
    • NLM

      Grifoni S de C, Bendhack LM. Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology. 2004 ; 70 160-168.[citado 2024 jul. 02 ]
    • Vancouver

      Grifoni S de C, Bendhack LM. Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology. 2004 ; 70 160-168.[citado 2024 jul. 02 ]
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Subjects: FARMACOLOGIA, CARDIOLOGIA

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    • ABNT

      MOLIN, Josiane Cristina e BENDHACK, Lusiane Maria. Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms. Vascular Pharmacology, v. 42, p. 1-6, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2004.11.006. Acesso em: 02 jul. 2024.
    • APA

      Molin, J. C., & Bendhack, L. M. (2004). Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms. Vascular Pharmacology, 42, 1-6. doi:10.1016/j.vph.2004.11.006
    • NLM

      Molin JC, Bendhack LM. Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms [Internet]. Vascular Pharmacology. 2004 ; 42 1-6.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.vph.2004.11.006
    • Vancouver

      Molin JC, Bendhack LM. Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms [Internet]. Vascular Pharmacology. 2004 ; 42 1-6.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.vph.2004.11.006

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