ReP USP - Resultado da busca

Artigo de periodico
Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis (2022)
Marinovic, Marcelo Paradiso; Sousa Filho, Celso Pereira Batista de ; Batista, F. A. H; Avelino, T. M; Cogliati, Bruno ; Figueira, A. C. M; Otton, Rosemari ; Rodrigues, Alice Cristina
Source: Journal of Nutritional Biochemistry. Unidades: FMVZ, ICB
Subjects: CHÁ, ADIPONECTINA, FÍGADO GORDUROSO, OBESIDADE, ANTIOXIDANTES, INSULINA, EXTRATOS (FORMAS FARMACÊUTICAS), FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARINOVIC, Marcelo Paradiso et al. Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis. Journal of Nutritional Biochemistry, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2022.108957. Acesso em: 18 nov. 2024.
APA
Marinovic, M. P., Sousa Filho, C. P. B. de, Batista, F. A. H., Avelino, T. M., Cogliati, B., Figueira, A. C. M., et al. (2022). Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis. Journal of Nutritional Biochemistry. doi:10.1016/j.jnutbio.2022.108957
NLM
Marinovic MP, Sousa Filho CPB de, Batista FAH, Avelino TM, Cogliati B, Figueira ACM, Otton R, Rodrigues AC. Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis [Internet]. Journal of Nutritional Biochemistry. 2022 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jnutbio.2022.108957
Vancouver
Marinovic MP, Sousa Filho CPB de, Batista FAH, Avelino TM, Cogliati B, Figueira ACM, Otton R, Rodrigues AC. Green tea extract increases adiponectin and PPAR α levels to improve hepatic steatosis [Internet]. Journal of Nutritional Biochemistry. 2022 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jnutbio.2022.108957
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Source: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Subjects: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 18 nov. 2024.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Artigo de periodico
Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization (2019)
Santos, Nilton Barreto dos ; Franco, Roseane Durante; Camarini, Rosana ; Munhoz, Carolina Demarchi ; Eichler, Rosangela Aparecida dos Santos ; Gewehr, Mayara Calegaro Ferrari ; Reckziegel, Patricia ; Dale, Camila Squarzoni ; Silva, Victoria R. O. da; Borges, Vanessa de Fátima; Lima, Braulio Henrique Freire ; Cunha, Fernando de Queiroz ; Visniauskas, Bruna ; Chagas, Jair Ribeiro ; Tufik, Sergio; Peres, Fernanda Fiel; Abilio, Vanessa Costhek ; Florio, Jorge Camilo ; Iwai, Leo Kei ; Rioli, Vanessa ; Presoto, Benedito C.; Guimarães, Alessander O. ; Pesquero, João Bosco ; Bader, Michael ; Castro, Leandro Mantovani de ; Ferro, Emer Suavinho
Source: Biomolecules. Unidades: ICB, FMVZ, FMRP
Subjects: ENZIMAS, NEUROPEPTÍDEOS, DEGENERAÇÃO NEURAL, INFLAMAÇÃO, ANATOMIA, FARMACOLOGIA, OLIGOPEPTÍDEOS, CAMUNDONGOS, FENÓTIPOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Nilton Barreto dos et al. Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization. Biomolecules, v. 9, n. 8, p. 1-38, 2019Tradução . . Disponível em: https://doi.org/10.3390/biom9080382. Acesso em: 18 nov. 2024.
APA
Santos, N. B. dos, Franco, R. D., Camarini, R., Munhoz, C. D., Eichler, R. A. dos S., Gewehr, M. C. F., et al. (2019). Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization. Biomolecules, 9( 8), 1-38. doi:10.3390/biom9080382
NLM
Santos NB dos, Franco RD, Camarini R, Munhoz CD, Eichler RA dos S, Gewehr MCF, Reckziegel P, Dale CS, Silva VRO da, Borges V de F, Lima BHF, Cunha F de Q, Visniauskas B, Chagas JR, Tufik S, Peres FF, Abilio VC, Florio JC, Iwai LK, Rioli V, Presoto BC, Guimarães AO, Pesquero JB, Bader M, Castro LM de, Ferro ES. Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization [Internet]. Biomolecules. 2019 ; 9( 8): 1-38.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/biom9080382
Vancouver
Santos NB dos, Franco RD, Camarini R, Munhoz CD, Eichler RA dos S, Gewehr MCF, Reckziegel P, Dale CS, Silva VRO da, Borges V de F, Lima BHF, Cunha F de Q, Visniauskas B, Chagas JR, Tufik S, Peres FF, Abilio VC, Florio JC, Iwai LK, Rioli V, Presoto BC, Guimarães AO, Pesquero JB, Bader M, Castro LM de, Ferro ES. Thimet oligopeptidase (EC 3.4.24.15) key functions suggested by knockout mice phenotype characterization [Internet]. Biomolecules. 2019 ; 9( 8): 1-38.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/biom9080382
Artigo de periodico
Insulin Substrate Receptor (IRS) proteins in normal and malignant hematopoiesis (2018)
Machado Neto, João Agostinho ; Fenerich, Bruna Alves; Alves, Ana Paula Nunes Rodrigues; Fernandes, Jaqueline Cristina; Scopim-Ribeiro, Renata; Coelho-Silva, Juan Luiz ; Traina, Fabiola
Source: Clinics. Unidades: ICB, FMRP
Subjects: RECEPTORES DE INSULINA, LEUCEMIA, HEMATOPOESE, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACHADO NETO, João Agostinho et al. Insulin Substrate Receptor (IRS) proteins in normal and malignant hematopoiesis. Clinics, v. 73, n. Suppl 1, 2018Tradução . . Disponível em: https://doi.org/10.6061/clinics/2018/e566s. Acesso em: 18 nov. 2024.
APA
Machado Neto, J. A., Fenerich, B. A., Alves, A. P. N. R., Fernandes, J. C., Scopim-Ribeiro, R., Coelho-Silva, J. L., & Traina, F. (2018). Insulin Substrate Receptor (IRS) proteins in normal and malignant hematopoiesis. Clinics, 73( Suppl 1). doi:10.6061/clinics/2018/e566s
NLM
Machado Neto JA, Fenerich BA, Alves APNR, Fernandes JC, Scopim-Ribeiro R, Coelho-Silva JL, Traina F. Insulin Substrate Receptor (IRS) proteins in normal and malignant hematopoiesis [Internet]. Clinics. 2018 ; 73( Suppl 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.6061/clinics/2018/e566s
Vancouver
Machado Neto JA, Fenerich BA, Alves APNR, Fernandes JC, Scopim-Ribeiro R, Coelho-Silva JL, Traina F. Insulin Substrate Receptor (IRS) proteins in normal and malignant hematopoiesis [Internet]. Clinics. 2018 ; 73( Suppl 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.6061/clinics/2018/e566s
Artigo de periodico
Chemical profiling of two congeneric sea mat corals along the Brazilian coast: adaptive and functional patterns (2018)
Costa-Lotufo, Letícia Veras ; Carnevale Neto, Fausto ; Trindade-Silva, Amaro Emiliano ; Silva, Ricardo Roberto da; Silva, Genivaldo Gueiros Z. ; Wilke, Diego Veras ; Pinto, Francisco das Chagas Lima ; Sahm, Bianca Del Bianco ; Jimenez, P. C. ; Silva, Jacqueline Nakau Mendonça Galiote; Lotufo, Tito Monteiro da Cruz ; Pessoa, Otilia D. L. ; Lopes, Norberto Peporine
Source: Chemical Communications. Unidades: ICB, FCFRP, IO, IQSC
Subjects: ANTHOZOA, ESPECTROMETRIA DE MASSAS, METABOLÔMICA, ESTRUTURA MOLECULAR (QUÍMICA TEÓRICA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA-LOTUFO, Letícia Veras et al. Chemical profiling of two congeneric sea mat corals along the Brazilian coast: adaptive and functional patterns. Chemical Communications, v. 54, n. 16, p. 1952-1955, 2018Tradução . . Disponível em: https://doi.org/10.1039/c7cc08411k. Acesso em: 18 nov. 2024.
APA
Costa-Lotufo, L. V., Carnevale Neto, F., Trindade-Silva, A. E., Silva, R. R. da, Silva, G. G. Z., Wilke, D. V., et al. (2018). Chemical profiling of two congeneric sea mat corals along the Brazilian coast: adaptive and functional patterns. Chemical Communications, 54( 16), 1952-1955. doi:10.1039/c7cc08411k
NLM
Costa-Lotufo LV, Carnevale Neto F, Trindade-Silva AE, Silva RR da, Silva GGZ, Wilke DV, Pinto F das CL, Sahm BDB, Jimenez PC, Silva JNMG, Lotufo TM da C, Pessoa ODL, Lopes NP. Chemical profiling of two congeneric sea mat corals along the Brazilian coast: adaptive and functional patterns [Internet]. Chemical Communications. 2018 ; 54( 16): 1952-1955.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1039/c7cc08411k
Vancouver
Costa-Lotufo LV, Carnevale Neto F, Trindade-Silva AE, Silva RR da, Silva GGZ, Wilke DV, Pinto F das CL, Sahm BDB, Jimenez PC, Silva JNMG, Lotufo TM da C, Pessoa ODL, Lopes NP. Chemical profiling of two congeneric sea mat corals along the Brazilian coast: adaptive and functional patterns [Internet]. Chemical Communications. 2018 ; 54( 16): 1952-1955.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1039/c7cc08411k
Artigo de periodico
RNAi mediated IL-6 in vitro knockdown in psoriasis skin model with topical siRNA delivery system based on liquid crystalline phase (2016)
Depieri, Lívia Vieira; Borgheti-Cardoso, Lívia Neves; Campos, Patrícia Mazureki; Otaguiri, Katia Kaori; Vicentini, Fabiana Testa Moura de Carvalho; Lopes, Luciana Biagini ; Fonseca, Maria José Vieira; Bentley, Maria Vitória Lopes Badra
Source: European Journal of Pharmaceutics and Biopharmaceutics. Unidades: ICB, FCFRP
Subjects: PSORÍASE, RNA, MEDICAMENTO TÓPICO, NANOPARTÍCULAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DEPIERI, Lívia Vieira et al. RNAi mediated IL-6 in vitro knockdown in psoriasis skin model with topical siRNA delivery system based on liquid crystalline phase. European Journal of Pharmaceutics and Biopharmaceutics, v. 105, p. 50-58, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejpb.2016.05.012. Acesso em: 18 nov. 2024.
APA
Depieri, L. V., Borgheti-Cardoso, L. N., Campos, P. M., Otaguiri, K. K., Vicentini, F. T. M. de C., Lopes, L. B., et al. (2016). RNAi mediated IL-6 in vitro knockdown in psoriasis skin model with topical siRNA delivery system based on liquid crystalline phase. European Journal of Pharmaceutics and Biopharmaceutics, 105, 50-58. doi:10.1016/j.ejpb.2016.05.012
NLM
Depieri LV, Borgheti-Cardoso LN, Campos PM, Otaguiri KK, Vicentini FTM de C, Lopes LB, Fonseca MJV, Bentley MVLB. RNAi mediated IL-6 in vitro knockdown in psoriasis skin model with topical siRNA delivery system based on liquid crystalline phase [Internet]. European Journal of Pharmaceutics and Biopharmaceutics. 2016 ; 105 50-58.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejpb.2016.05.012
Vancouver
Depieri LV, Borgheti-Cardoso LN, Campos PM, Otaguiri KK, Vicentini FTM de C, Lopes LB, Fonseca MJV, Bentley MVLB. RNAi mediated IL-6 in vitro knockdown in psoriasis skin model with topical siRNA delivery system based on liquid crystalline phase [Internet]. European Journal of Pharmaceutics and Biopharmaceutics. 2016 ; 105 50-58.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejpb.2016.05.012
Artigo de periodico
Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring (2015)
Kirsten, Thiago Berti; Chaves-Kirsten, Gabriela Pena; Bernardes, Suene; Scavone, Cristoforo ; Sarkis, Jorge Eduardo de Souza; Bernardi, Maria Martha; Felicio, Luciano Freitas
Source: Plos One. Unidades: ICB, FMVZ
Subjects: AUTISMO, GRAVIDEZ, LIPOPOLISSACARÍDEOS, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KIRSTEN, Thiago Berti et al. Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring. Plos One, v. 10, n. 7, p. 1-18, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0134565. Acesso em: 18 nov. 2024.
APA
Kirsten, T. B., Chaves-Kirsten, G. P., Bernardes, S., Scavone, C., Sarkis, J. E. de S., Bernardi, M. M., & Felicio, L. F. (2015). Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring. Plos One, 10( 7), 1-18. doi:10.1371/journal.pone.0134565
NLM
Kirsten TB, Chaves-Kirsten GP, Bernardes S, Scavone C, Sarkis JE de S, Bernardi MM, Felicio LF. Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring [Internet]. Plos One. 2015 ; 10( 7): 1-18.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0134565
Vancouver
Kirsten TB, Chaves-Kirsten GP, Bernardes S, Scavone C, Sarkis JE de S, Bernardi MM, Felicio LF. Lipopolysaccharide exposure induces maternal hypozincemia, and prenatal zinc treatment prevents autistic-like behaviors and disturbances in the striatal dopaminergic and mTOR systems of offspring [Internet]. Plos One. 2015 ; 10( 7): 1-18.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0134565
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidades: FMRP, FM, ICB
Subjects: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIGNALIA, Andreia Z et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, v. 129, n. 1, p. 39-48, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140548. Acesso em: 18 nov. 2024.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20140548
Artigo de periodico
Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs (2013)
Araújo, Karla Patrícia Cardoso; Bonuccelli, Gloria; Duarte, Caio Nogueira; Gaiad, Thaís Peixoto; Moreira, Dayson Friaça; Feder, David; Belizário, José Ernesto; Miglino, Maria Angélica; Lisanti, Michael P; Ambrósio, Carlos Eduardo
Source: Plos One. Unidades: FMVZ, FZEA, ICB
Subjects: ATROFIA MUSCULAR, CÃES, DISTROFIA MUSCULAR ANIMAL, GLICOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAÚJO, Karla Patrícia Cardoso et al. Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs. Plos One, v. 8, n. 4, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0061367. Acesso em: 18 nov. 2024.
APA
Araújo, K. P. C., Bonuccelli, G., Duarte, C. N., Gaiad, T. P., Moreira, D. F., Feder, D., et al. (2013). Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs. Plos One, 8( 4). doi:10.1371/journal.pone.0061367
NLM
Araújo KPC, Bonuccelli G, Duarte CN, Gaiad TP, Moreira DF, Feder D, Belizário JE, Miglino MA, Lisanti MP, Ambrósio CE. Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs [Internet]. Plos One. 2013 ; 8( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0061367
Vancouver
Araújo KPC, Bonuccelli G, Duarte CN, Gaiad TP, Moreira DF, Feder D, Belizário JE, Miglino MA, Lisanti MP, Ambrósio CE. Bortezomib (PS-341) treatment decreases inflammation and partially rescues the expression of the dystrophin- glycoprotein complex in GRMD dogs [Internet]. Plos One. 2013 ; 8( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0061367
Trabalho de evento-resumo periodico
Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle (2009)
Araújo, Karla Patrícia Cardoso; Moreira, Dayson Friaça; Gaiad, Thaís Peixoto; Miglino, Maria Angélica; Górniak, Silvana Lima ; Feder, D; Belizário, José Ernesto; Ambrósio, Carlos Eduardo
Source: Neuromuscular Disorders. Conference titles: International Congress of the World Muscle Society. Unidades: FMVZ, ICB, FZEA
Subjects: FARMACOLOGIA, CÃO GOLDEN RETRIEVER, DISTROFIA MUSCULAR ANIMAL, NEUROLOGIA VETERINÁRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAÚJO, Karla Patrícia Cardoso et al. Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle. Neuromuscular Disorders. Oxford: Elsevier Science. Disponível em: https://doi.org/10.1016/j.nmd.2009.06.218. Acesso em: 18 nov. 2024. , 2009
APA
Araújo, K. P. C., Moreira, D. F., Gaiad, T. P., Miglino, M. A., Górniak, S. L., Feder, D., et al. (2009). Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle. Neuromuscular Disorders. Oxford: Elsevier Science. doi:10.1016/j.nmd.2009.06.218
NLM
Araújo KPC, Moreira DF, Gaiad TP, Miglino MA, Górniak SL, Feder D, Belizário JE, Ambrósio CE. Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle [Internet]. Neuromuscular Disorders. 2009 ; 19( 8-9): 613.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.nmd.2009.06.218
Vancouver
Araújo KPC, Moreira DF, Gaiad TP, Miglino MA, Górniak SL, Feder D, Belizário JE, Ambrósio CE. Treatment with bortezomib (PS-341) of Golden Retriever Muscular Dystrophy (GRMD): analysis of proteasome inhibition and morphology of dystrophic skeletal muscle [Internet]. Neuromuscular Disorders. 2009 ; 19( 8-9): 613.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.nmd.2009.06.218
Artigo de periodico
Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin (2009)
Russo, Lilian Cristina; Goni, Camila Nascimento; Castro, Leandro Mantovani de; Asega, Amanda Francine; Camargo, Antonio Carlos Martins de; Trujillo, Cleber Augusto; Ulrich, Henning ; Glucksman, Marc J.; Scavone, Cristoforo ; Ferro, Emer Suavinho
Source: FEBS Journal. Unidades: ICB, IQ
Subjects: PROTEÍNAS QUINASES, CALMODULINA, OLIGOPEPTÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RUSSO, Lilian Cristina et al. Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin. FEBS Journal, v. 276, n. 16, p. 4358-4371, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1742-4658.2009.07144.x. Acesso em: 18 nov. 2024.
APA
Russo, L. C., Goni, C. N., Castro, L. M. de, Asega, A. F., Camargo, A. C. M. de, Trujillo, C. A., et al. (2009). Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin. FEBS Journal, 276( 16), 4358-4371. doi:10.1111/j.1742-4658.2009.07144.x
NLM
Russo LC, Goni CN, Castro LM de, Asega AF, Camargo ACM de, Trujillo CA, Ulrich H, Glucksman MJ, Scavone C, Ferro ES. Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin [Internet]. FEBS Journal. 2009 ; 276( 16): 4358-4371.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/j.1742-4658.2009.07144.x
Vancouver
Russo LC, Goni CN, Castro LM de, Asega AF, Camargo ACM de, Trujillo CA, Ulrich H, Glucksman MJ, Scavone C, Ferro ES. Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin [Internet]. FEBS Journal. 2009 ; 276( 16): 4358-4371.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/j.1742-4658.2009.07144.x
Artigo de periodico
Mechanisms involved in the reduced leukocyte migration in intrauterine undernourishment (2007)
Landgraf, Maristella de Almeida Vitta; Tostes, Rita de Cássia Aleixo; Borelli, Primavera ; Zorn, Telma Maria Tenório; Nigro, Dorothy; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Source: Nutrition. Unidades: ICB, FCF
Subjects: LEUCÓCITOS, NUTRIÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LANDGRAF, Maristella de Almeida Vitta et al. Mechanisms involved in the reduced leukocyte migration in intrauterine undernourishment. Nutrition, v. 23, n. 2, p. 145-156, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.nut.2006.08.020. Acesso em: 18 nov. 2024.
APA
Landgraf, M. de A. V., Tostes, R. de C. A., Borelli, P., Zorn, T. M. T., Nigro, D., Carvalho, M. H. C. de, & Fortes, Z. B. (2007). Mechanisms involved in the reduced leukocyte migration in intrauterine undernourishment. Nutrition, 23( 2), 145-156. doi:10.1016/j.nut.2006.08.020
NLM
Landgraf M de AV, Tostes R de CA, Borelli P, Zorn TMT, Nigro D, Carvalho MHC de, Fortes ZB. Mechanisms involved in the reduced leukocyte migration in intrauterine undernourishment [Internet]. Nutrition. 2007 ; 23( 2): 145-156.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.nut.2006.08.020
Vancouver
Landgraf M de AV, Tostes R de CA, Borelli P, Zorn TMT, Nigro D, Carvalho MHC de, Fortes ZB. Mechanisms involved in the reduced leukocyte migration in intrauterine undernourishment [Internet]. Nutrition. 2007 ; 23( 2): 145-156.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.nut.2006.08.020
Artigo de periodico
Stress-induced increment on total bronchoalveolar cell count in OVA-sensitized rats (2001)
Portela, Carlos de Paula; Gomes, Cristina de Oliveira Massoco-Salles ; Lima, Wothan Tavares de ; Palermo-Neto, João
Source: Physiology & Behavior. Unidades: ICB, FMVZ
Subjects: FARMACOLOGIA VETERINÁRIA, TOXICOLOGIA VETERINÁRIA, INFLAMAÇÃO, ALERGIA E IMUNOLOGIA, INTERAÇÃO SOCIAL (COMPORTAMENTO SOCIAL) (VETERINÁRIA), RATOS, ESTRESSE (INDUZIDO QUIMICAMENTE), ALBUMINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PORTELA, Carlos de Paula et al. Stress-induced increment on total bronchoalveolar cell count in OVA-sensitized rats. Physiology & Behavior, v. 72, n. 3, p. 415-420, 2001Tradução . . Disponível em: https://doi.org/10.1016/S0031-9384(00)00429-7. Acesso em: 18 nov. 2024.
APA
Portela, C. de P., Gomes, C. de O. M. -S., Lima, W. T. de, & Palermo-Neto, J. (2001). Stress-induced increment on total bronchoalveolar cell count in OVA-sensitized rats. Physiology & Behavior, 72( 3), 415-420. doi:10.1016/S0031-9384(00)00429-7
NLM
Portela C de P, Gomes C de OM-S, Lima WT de, Palermo-Neto J. Stress-induced increment on total bronchoalveolar cell count in OVA-sensitized rats [Internet]. Physiology & Behavior. 2001 ; 72( 3): 415-420.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/S0031-9384(00)00429-7
Vancouver
Portela C de P, Gomes C de OM-S, Lima WT de, Palermo-Neto J. Stress-induced increment on total bronchoalveolar cell count in OVA-sensitized rats [Internet]. Physiology & Behavior. 2001 ; 72( 3): 415-420.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/S0031-9384(00)00429-7
Artigo de periodico
Effects of haloperidol and GM1 ganglioside treatment on striatal D2 receptor binding and dopamine turnover (1998)
Vital, Maria Aparecida Barbato Frazão; Flório, Jorge Camilo; Frussa-Filho, Roberto; De Lucia, Roberto; Tufik, Sérgio; Palermo-Neto, João
Source: Life Sciences. Unidades: FMVZ, ICB
Subjects: GLICOLIPÍDEOS, RATOS, FARMACOLOGIA VETERINÁRIA, NEUROTRANSMISSORES, FÁRMACOS (SISTEMA NERVOSO CENTRAL)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VITAL, Maria Aparecida Barbato Frazão et al. Effects of haloperidol and GM1 ganglioside treatment on striatal D2 receptor binding and dopamine turnover. Life Sciences, v. 62, n. 13, p. 1161-1169, 1998Tradução . . Disponível em: https://doi.org/10.1016/S0024-3205(98)00042-3. Acesso em: 18 nov. 2024.
APA
Vital, M. A. B. F., Flório, J. C., Frussa-Filho, R., De Lucia, R., Tufik, S., & Palermo-Neto, J. (1998). Effects of haloperidol and GM1 ganglioside treatment on striatal D2 receptor binding and dopamine turnover. Life Sciences, 62( 13), 1161-1169. doi:10.1016/S0024-3205(98)00042-3
NLM
Vital MABF, Flório JC, Frussa-Filho R, De Lucia R, Tufik S, Palermo-Neto J. Effects of haloperidol and GM1 ganglioside treatment on striatal D2 receptor binding and dopamine turnover [Internet]. Life Sciences. 1998 ; 62( 13): 1161-1169.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/S0024-3205(98)00042-3
Vancouver
Vital MABF, Flório JC, Frussa-Filho R, De Lucia R, Tufik S, Palermo-Neto J. Effects of haloperidol and GM1 ganglioside treatment on striatal D2 receptor binding and dopamine turnover [Internet]. Life Sciences. 1998 ; 62( 13): 1161-1169.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/S0024-3205(98)00042-3

USP Schools

ReP

Filtros

USP Schools

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Funding Agencies

Indexado em

Non normalized affiliation