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Artigo de periodico
Ethanol and cyclophosphamide induce similar nephrotoxic effects: possible role for Nox4 and superoxide (2021)
Sousa, Arthur Henrique de; Vale, Gabriel Tavares do ; Silva, Carla Beatriz Pereira da ; Awata, Wanessa Mayumi Carvalho; Pinheiro, Lucas Cezar ; Tirapelli, Carlos Renato
Source: Canadian Journal of Physiology and Pharmacology. Unidades: EERP, FMRP
Subjects: ANIMAIS, RATOS, ETANOL, SUPERÓXIDO DISMUTASE
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ABNT
SOUSA, Arthur Henrique de et al. Ethanol and cyclophosphamide induce similar nephrotoxic effects: possible role for Nox4 and superoxide. Canadian Journal of Physiology and Pharmacology, v. 99, n. 7, p. 744-751, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2020-0246. Acesso em: 18 nov. 2024.
APA
Sousa, A. H. de, Vale, G. T. do, Silva, C. B. P. da, Awata, W. M. C., Pinheiro, L. C., & Tirapelli, C. R. (2021). Ethanol and cyclophosphamide induce similar nephrotoxic effects: possible role for Nox4 and superoxide. Canadian Journal of Physiology and Pharmacology, 99( 7), 744-751. doi:10.1139/cjpp-2020-0246
NLM
Sousa AH de, Vale GT do, Silva CBP da, Awata WMC, Pinheiro LC, Tirapelli CR. Ethanol and cyclophosphamide induce similar nephrotoxic effects: possible role for Nox4 and superoxide [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 744-751.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/cjpp-2020-0246
Vancouver
Sousa AH de, Vale GT do, Silva CBP da, Awata WMC, Pinheiro LC, Tirapelli CR. Ethanol and cyclophosphamide induce similar nephrotoxic effects: possible role for Nox4 and superoxide [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 744-751.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/cjpp-2020-0246
Artigo de periodico
Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis (2021)
Valle, Gabriel Tavares do; Ricci, Sthefany Teodoro; Silva, Alessandra Oliveira; Tirapelli, Carlos Renato ; Ceron, Carla Speroni
Source: Canadian Journal of Physiology and Pharmacology. Unidade: EERP
Subjects: ANIMAIS, PERÓXIDO DE HIDROGÊNIO, RATOS, ESTRESSE OXIDATIVO, SEPSE
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ABNT
VALLE, Gabriel Tavares do et al. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis. Canadian Journal of Physiology and Pharmacology, v. 99, n. 7, p. 699-707, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2020-0564. Acesso em: 18 nov. 2024.
APA
Valle, G. T. do, Ricci, S. T., Silva, A. O., Tirapelli, C. R., & Ceron, C. S. (2021). Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis. Canadian Journal of Physiology and Pharmacology, 99( 7), 699-707. doi:10.1139/cjpp-2020-0564
NLM
Valle GT do, Ricci ST, Silva AO, Tirapelli CR, Ceron CS. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 699-707.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/cjpp-2020-0564
Vancouver
Valle GT do, Ricci ST, Silva AO, Tirapelli CR, Ceron CS. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 699-707.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/cjpp-2020-0564
Artigo de periodico
Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors (2020)
Vale, Gabriel Tavares do; Leoni, Drieli; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Uliana, Daniela L; La Gata, Davi C; Resstel, Leonardo Barbosa Moraes; Padovan, Cláudia Maria ; Tirapelli, Carlos Renato
Source: Stress. Unidades: FMRP, FFCLRP, EERP
Subjects: ESTRESSE, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
VALE, Gabriel Tavares do et al. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors. Stress, v. 23, n. 3, p. 328-337, 2020Tradução . . Disponível em: https://doi.org/10.1080/10253890.2019.1675627. Acesso em: 18 nov. 2024.
APA
Vale, G. T. do, Leoni, D., Sousa, A. H. de, Gonzaga, N. de A., Uliana, D. L., La Gata, D. C., et al. (2020). Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors. Stress, 23( 3), 328-337. doi:10.1080/10253890.2019.1675627
NLM
Vale GT do, Leoni D, Sousa AH de, Gonzaga N de A, Uliana DL, La Gata DC, Resstel LBM, Padovan CM, Tirapelli CR. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors [Internet]. Stress. 2020 ; 23( 3): 328-337.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/10253890.2019.1675627
Vancouver
Vale GT do, Leoni D, Sousa AH de, Gonzaga N de A, Uliana DL, La Gata DC, Resstel LBM, Padovan CM, Tirapelli CR. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors [Internet]. Stress. 2020 ; 23( 3): 328-337.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/10253890.2019.1675627
Artigo de periodico
Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection (2020)
Malacco, Nathalia Luisa Sousa de Oliveira; Souza, Jessica Amanda Marques; Martins, Flavia Rayssa Braga; Rachid, Milene Alvarenga; Simplicio, Janaina Aparecida; Tirapelli, Carlos Renato ; Sabino, Adriano de Paula; Queiroz-Junior, Celso Martins; Goes, Grazielle Ribeiro; Vieira, Leda Quercia; Souza, Danielle Glória; Pinho, Vanessa; Teixeira, Mauro Martins; Soriani, Frederico Marianetti
Source: eLife. Unidade: EERP
Subjects: ASPERGILLUS, ETANOL, CONSUMO, IMUNOLOGIA, DOENÇAS INFECCIOSAS, INFLAMAÇÃO, MICROBIOLOGIA, NEUTRÓFILOS
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ABNT
MALACCO, Nathalia Luisa Sousa de Oliveira et al. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection. eLife, v. 9, 2020Tradução . . Disponível em: https://doi.org/10.7554/eLife.58855. Acesso em: 18 nov. 2024.
APA
Malacco, N. L. S. de O., Souza, J. A. M., Martins, F. R. B., Rachid, M. A., Simplicio, J. A., Tirapelli, C. R., et al. (2020). Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection. eLife, 9. doi:10.7554/eLife.58855
NLM
Malacco NLS de O, Souza JAM, Martins FRB, Rachid MA, Simplicio JA, Tirapelli CR, Sabino A de P, Queiroz-Junior CM, Goes GR, Vieira LQ, Souza DG, Pinho V, Teixeira MM, Soriani FM. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection [Internet]. eLife. 2020 ; 9[citado 2024 nov. 18 ] Available from: https://doi.org/10.7554/eLife.58855
Vancouver
Malacco NLS de O, Souza JAM, Martins FRB, Rachid MA, Simplicio JA, Tirapelli CR, Sabino A de P, Queiroz-Junior CM, Goes GR, Vieira LQ, Souza DG, Pinho V, Teixeira MM, Soriani FM. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection [Internet]. eLife. 2020 ; 9[citado 2024 nov. 18 ] Available from: https://doi.org/10.7554/eLife.58855
Artigo de periodico
Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder (2019)
Vale, Gabriel Tavares do; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Oliveira, Mariana G. de; Justo, Alberto F. O; Alexandre, Eduardo C.; Tanus-Santos, José Eduardo ; Antunes, Edson; Tirapelli, Carlos Renato
Source: Canadian Journal of Physiology and Pharmacology. Unidades: FMRP, EERP
Subjects: ETANOL, ESTRESSE OXIDATIVO, BEXIGA
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ABNT
VALE, Gabriel Tavares do et al. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, v. 97, n. 12, p. 1103-1114, 2019Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2019-0143. Acesso em: 18 nov. 2024.
APA
Vale, G. T. do, Sousa, A. H. de, Gonzaga, N. de A., Oliveira, M. G. de, Justo, A. F. O., Alexandre, E. C., et al. (2019). Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, 97( 12), 1103-1114. doi:10.1139/cjpp-2019-0143
NLM
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Vancouver
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 18 nov. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms (2019)
Nakashima, Marcelo A.; Silva, Carla Beatriz Pereira da ; Gonzaga, Natália de Almeida; Simplicio, Janaina A.; Omoto, Ana Carolina Mieko ; Tirapelli, Luis Fernando; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Source: Cytokine. Unidades: FMRP, EERP
Subjects: ESTRESSE OXIDATIVO, ETANOL, CITOTOXINAS, SEPSE, INTERLEUCINAS
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ABNT
NAKASHIMA, Marcelo A. et al. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, v. 121, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2019.154734. Acesso em: 18 nov. 2024.
APA
Nakashima, M. A., Silva, C. B. P. da, Gonzaga, N. de A., Simplicio, J. A., Omoto, A. C. M., Tirapelli, L. F., et al. (2019). Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, 121. doi:10.1016/j.cyto.2019.154734
NLM
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Vancouver
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Artigo de periodico
Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin (2019)
Gonzaga, Natália de Almeida; Awata, Wanessa Mayumi Carvalho; Tanus-Santos, José Eduardo ; Padovan, Júlio C.; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: AORTA, ETANOL (REMOÇÃO)
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ABNT
GONZAGA, Natália de Almeida et al. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin. European Journal of Pharmacology, v. 847, p. 103-112, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.01.055. Acesso em: 18 nov. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Tanus-Santos, J. E., Padovan, J. C., & Tirapelli, C. R. (2019). Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin. European Journal of Pharmacology, 847, 103-112. doi:10.1016/j.ejphar.2019.01.055
NLM
Gonzaga N de A, Awata WMC, Tanus-Santos JE, Padovan JC, Tirapelli CR. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin [Internet]. European Journal of Pharmacology. 2019 ; 847 103-112.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2019.01.055
Vancouver
Gonzaga N de A, Awata WMC, Tanus-Santos JE, Padovan JC, Tirapelli CR. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin [Internet]. European Journal of Pharmacology. 2019 ; 847 103-112.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2019.01.055
Artigo de periodico
Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS (2018)
Ceron, Carla S.; Vale, Gabriel T. do; Simplicio, Janaina A.; Ricci, Sthefany T.; Martinis, Bruno Spinosa de; Freitas, Andressa de; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FFCLRP, EERP
Subjects: ESTRESSE OXIDATIVO, ETANOL, CITOCINAS, SEPSE, INTERLEUCINAS
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ABNT
CERON, Carla S. et al. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, v. 825, p. 39-47, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2018.02.008. Acesso em: 18 nov. 2024.
APA
Ceron, C. S., Vale, G. T. do, Simplicio, J. A., Ricci, S. T., Martinis, B. S. de, Freitas, A. de, & Tirapelli, C. R. (2018). Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, 825, 39-47. doi:10.1016/j.ejphar.2018.02.008
NLM
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008
Vancouver
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008
Artigo de periodico
Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide (2018)
Gonzaga, Natália de Almeida; Awata, Wanessa M. C.; Vale, Gabriel Tavares do; Marchi, Kátia Colombo; Muniz, Jaqueline Jóice ; Santos, José Eduardo Tanus dos ; Tirapelli, Carlos Renato
Source: Vascular Pharmacology. Unidades: FMRP, EERP
Subjects: ETANOL, PERÓXIDO DE HIDROGÊNIO, TECIDO ADIPOSO, CATALASE, ENDOTÉLIO VASCULAR
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ABNT
GONZAGA, Natália de Almeida et al. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide. Vascular Pharmacology, v. 111, p. 44-53, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2018.08.010. Acesso em: 18 nov. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Vale, G. T. do, Marchi, K. C., Muniz, J. J., Santos, J. E. T. dos, & Tirapelli, C. R. (2018). Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide. Vascular Pharmacology, 111, 44-53. doi:10.1016/j.vph.2018.08.010
NLM
Gonzaga N de A, Awata WMC, Vale GT do, Marchi KC, Muniz JJ, Santos JET dos, Tirapelli CR. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide [Internet]. Vascular Pharmacology. 2018 ; 111 44-53.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.vph.2018.08.010
Vancouver
Gonzaga N de A, Awata WMC, Vale GT do, Marchi KC, Muniz JJ, Santos JET dos, Tirapelli CR. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide [Internet]. Vascular Pharmacology. 2018 ; 111 44-53.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.vph.2018.08.010
Artigo de periodico
Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers (2018)
Pinto, Carina Guidi; Marchi, Kátia Colombo; Arizza, Ailton Amarante; Leite, Ana Paula Silveira; Tirapelli, Carlos Renato ; Matheus, Selma Maria Michelin
Source: Acta Histochemica. Unidades: EERP, FMRP
Subjects: FIBRAS MUSCULARES, RATOS, ABUSO DE ÁLCOOL
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ABNT
PINTO, Carina Guidi et al. Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers. Acta Histochemica, v. 120, n. 8, p. 789-796, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.acthis.2018.09.004. Acesso em: 18 nov. 2024.
APA
Pinto, C. G., Marchi, K. C., Arizza, A. A., Leite, A. P. S., Tirapelli, C. R., & Matheus, S. M. M. (2018). Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers. Acta Histochemica, 120( 8), 789-796. doi:10.1016/j.acthis.2018.09.004
NLM
Pinto CG, Marchi KC, Arizza AA, Leite APS, Tirapelli CR, Matheus SMM. Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers [Internet]. Acta Histochemica. 2018 ; 120( 8): 789-796.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.acthis.2018.09.004
Vancouver
Pinto CG, Marchi KC, Arizza AA, Leite APS, Tirapelli CR, Matheus SMM. Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers [Internet]. Acta Histochemica. 2018 ; 120( 8): 789-796.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.acthis.2018.09.004
Artigo de periodico
Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection (2018)
Colato, Rafaela Pravato; Muniz, Vânia Brazão; Gabriel Tavares do Vale; Fabricia Helena Santello; Pedro Alexandre Sampaio; Tirapelli, Carlos Renato ; Bisson, Gabriela Silva ; Prado Junior, José Clóvis do
Source: Cytokine. Unidades: EERP, FCFRP
Subjects: ESTRESSE OXIDATIVO, ENVELHECIMENTO, CITOCINAS, TIMO (SISTEMAS SANGUÍNEO E IMUNE), TRYPANOSOMA CRUZI
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ABNT
COLATO, Rafaela Pravato et al. Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection. Cytokine, v. 111, p. 88-96, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2018.08.004. Acesso em: 18 nov. 2024.
APA
Colato, R. P., Muniz, V. B., Gabriel Tavares do Vale,, Fabricia Helena Santello,, Pedro Alexandre Sampaio,, Tirapelli, C. R., et al. (2018). Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection. Cytokine, 111, 88-96. doi:10.1016/j.cyto.2018.08.004
NLM
Colato RP, Muniz VB, Gabriel Tavares do Vale, Fabricia Helena Santello, Pedro Alexandre Sampaio, Tirapelli CR, Bisson GS, Prado Junior JC do. Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection [Internet]. Cytokine. 2018 ; 111 88-96.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cyto.2018.08.004
Vancouver
Colato RP, Muniz VB, Gabriel Tavares do Vale, Fabricia Helena Santello, Pedro Alexandre Sampaio, Tirapelli CR, Bisson GS, Prado Junior JC do. Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection [Internet]. Cytokine. 2018 ; 111 88-96.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cyto.2018.08.004
Artigo de periodico
Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection (2017)
Brazão, Vânia; Santello, Fabrícia H.; Colato, Rafaela P.; Mazotti, Tamires T.; Tazinafo, Lucas F.; Toldo, Miriam Paula Alonso; Vale, Gabriel T. do; Tirapelli, Carlos Renato ; Prado Júnior, José Clóvis do
Source: Journal of Pineal Research. Unidades: EERP, FCFRP
Subjects: TRYPANOSOMA CRUZI, MELATONINA, DOENÇA DE CHAGAS, ESTRESSE OXIDATIVO
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ABNT
BRAZÃO, Vânia et al. Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection. Journal of Pineal Research, v. 63, 2017Tradução . . Disponível em: https://doi.org/10.1111/jpi.12409. Acesso em: 18 nov. 2024.
APA
Brazão, V., Santello, F. H., Colato, R. P., Mazotti, T. T., Tazinafo, L. F., Toldo, M. P. A., et al. (2017). Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection. Journal of Pineal Research, 63. doi:10.1111/jpi.12409
NLM
Brazão V, Santello FH, Colato RP, Mazotti TT, Tazinafo LF, Toldo MPA, Vale GT do, Tirapelli CR, Prado Júnior JC do. Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection [Internet]. Journal of Pineal Research. 2017 ; 63[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/jpi.12409
Vancouver
Brazão V, Santello FH, Colato RP, Mazotti TT, Tazinafo LF, Toldo MPA, Vale GT do, Tirapelli CR, Prado Júnior JC do. Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection [Internet]. Journal of Pineal Research. 2017 ; 63[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/jpi.12409
Artigo de periodico
Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids (2017)
Diniz, Mariana C.; Olivon, Vania C.; Tavares, Lívia D.; Simplicio, Janaina A.; Gonzaga, Natália A.; Souza, Daniele G. de; Bendhack, Lusiane Maria; Tirapelli, Carlos Renato ; Bonaventura, Daniella
Source: Life Sciences. Unidades: FCFRP, EERP
Subjects: NITRATOS, AORTA, VASODILATAÇÃO, OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Mariana C. et al. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, v. 176, p. 26-34, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.03.016. Acesso em: 18 nov. 2024.
APA
Diniz, M. C., Olivon, V. C., Tavares, L. D., Simplicio, J. A., Gonzaga, N. A., Souza, D. G. de, et al. (2017). Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, 176, 26-34. doi:10.1016/j.lfs.2017.03.016
NLM
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Vancouver
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Artigo de periodico
Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species (2017)
Côco, Hariane; Pernomian, Larissa; Pereira, Priscila C.; Gomes, Mayara Santos; Marchi, Katia C.; Lopes, Alexandre H.; Cunha, Thiago Mattar; Tirapelli, Carlos Renato ; Oliveira, Ana Maria de
Source: Journal of Pharmacy and Pharmacology. Unidades: FCFRP, FMRP, EERP
Subjects: ANGIOTENSINA II, ARTÉRIAS CARÓTIDAS, ENDOTÉLIO VASCULAR, ESTRESSE PSICOLÓGICO, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CÔCO, Hariane et al. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species. Journal of Pharmacy and Pharmacology, v. 69, n. 1, p. 52-65, 2017Tradução . . Disponível em: https://doi.org/10.1111/jphp.12659. Acesso em: 18 nov. 2024.
APA
Côco, H., Pernomian, L., Pereira, P. C., Gomes, M. S., Marchi, K. C., Lopes, A. H., et al. (2017). Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species. Journal of Pharmacy and Pharmacology, 69( 1), 52-65. doi:10.1111/jphp.12659
NLM
Côco H, Pernomian L, Pereira PC, Gomes MS, Marchi KC, Lopes AH, Cunha TM, Tirapelli CR, Oliveira AM de. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species [Internet]. Journal of Pharmacy and Pharmacology. 2017 ; 69( 1): 52-65.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/jphp.12659
Vancouver
Côco H, Pernomian L, Pereira PC, Gomes MS, Marchi KC, Lopes AH, Cunha TM, Tirapelli CR, Oliveira AM de. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species [Internet]. Journal of Pharmacy and Pharmacology. 2017 ; 69( 1): 52-65.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/jphp.12659
Artigo de periodico
Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats (2017)
Hott, Sara C.; Gomes, Felipe V.; Uliana, Daniela L.; Vale, Gabriel T.; Tirapelli, Carlos Renato ; Resstel, Leonardo Barbosa Moraes
Source: Neuropharmacology. Unidades: EERP, FMRP
Subjects: ÓXIDO NÍTRICO, SISTEMA CARDIOVASCULAR, GLUTAMATOS, MEDO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HOTT, Sara C. et al. Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats. Neuropharmacology, v. 112, n. Pt. A, p. 135-143, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2016.05.022. Acesso em: 18 nov. 2024.
APA
Hott, S. C., Gomes, F. V., Uliana, D. L., Vale, G. T., Tirapelli, C. R., & Resstel, L. B. M. (2017). Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats. Neuropharmacology, 112( Pt. A), 135-143. doi:10.1016/j.neuropharm.2016.05.022
NLM
Hott SC, Gomes FV, Uliana DL, Vale GT, Tirapelli CR, Resstel LBM. Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats [Internet]. Neuropharmacology. 2017 ; 112( Pt. A): 135-143.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuropharm.2016.05.022
Vancouver
Hott SC, Gomes FV, Uliana DL, Vale GT, Tirapelli CR, Resstel LBM. Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats [Internet]. Neuropharmacology. 2017 ; 112( Pt. A): 135-143.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuropharm.2016.05.022
Artigo de periodico
Pharmacological characterization of the mechanisms underlying the vascular effects of succinate (2016)
Leite, Letícia N.; Gonzaga, Natália A.; Simplicio, Janaina A.; Vale, Gabriel T. do; Carballido, José M.; Alves Filho, José Carlos Farias; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: AORTA TORÁCICA, ENDOTÉLIO VASCULAR, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Letícia N. et al. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, v. 789, p. 334-343, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2016.07.045. Acesso em: 18 nov. 2024.
APA
Leite, L. N., Gonzaga, N. A., Simplicio, J. A., Vale, G. T. do, Carballido, J. M., Alves Filho, J. C. F., & Tirapelli, C. R. (2016). Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, 789, 334-343. doi:10.1016/j.ejphar.2016.07.045
NLM
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
Vancouver
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
Artigo de periodico
Effect of whitening dentifrices: a double-blind randomized controlled trial (2016)
Pintado-Palomino, Karen; Vasconcelos, Cecília Vilela Matias; Silva, Roberto Juns da; Fressatti, Andre Luiz de Macedo; Motta, Bianka Jurca Gonçalves da; Pires-de-Souza, Fernanda de Carvalho Panzeri; Tirapelli, Carlos Renato
Source: Brazilian Oral Research. Unidade: FORP
Subjects: DENTIFRÍCIO, CLAREAMENTO DE DENTE, SENSIBILIDADE DA DENTINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINTADO-PALOMINO, Karen et al. Effect of whitening dentifrices: a double-blind randomized controlled trial. Brazilian Oral Research, v. 30, n. 1, 2016Tradução . . Disponível em: https://doi.org/10.1590/1807-3107bor-2016.vol30.0082. Acesso em: 18 nov. 2024.
APA
Pintado-Palomino, K., Vasconcelos, C. V. M., Silva, R. J. da, Fressatti, A. L. de M., Motta, B. J. G. da, Pires-de-Souza, F. de C. P., & Tirapelli, C. R. (2016). Effect of whitening dentifrices: a double-blind randomized controlled trial. Brazilian Oral Research, 30( 1). doi:10.1590/1807-3107bor-2016.vol30.0082
NLM
Pintado-Palomino K, Vasconcelos CVM, Silva RJ da, Fressatti AL de M, Motta BJG da, Pires-de-Souza F de CP, Tirapelli CR. Effect of whitening dentifrices: a double-blind randomized controlled trial [Internet]. Brazilian Oral Research. 2016 ; 30( 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1590/1807-3107bor-2016.vol30.0082
Vancouver
Pintado-Palomino K, Vasconcelos CVM, Silva RJ da, Fressatti AL de M, Motta BJG da, Pires-de-Souza F de CP, Tirapelli CR. Effect of whitening dentifrices: a double-blind randomized controlled trial [Internet]. Brazilian Oral Research. 2016 ; 30( 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1590/1807-3107bor-2016.vol30.0082
Artigo de periodico
Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries (2016)
Simplicio, Janaina A.; Hipólito, Ulisses Vilela; Vale, Gabriel Tavares do; Callera, Glaucia Elena; Pereira, Camila André; Touyz, Rhian M; Tostes, Rita de Cássia Aleixo; Tirapelli, Carlos Renato
Source: Arquivos Brasileiros de Cardiologia. Unidades: FMRP, EERP
Subjects: ETANOL, ESTRESSE OXIDATIVO, PROTEÍNAS, ENDOTÉLIO VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SIMPLICIO, Janaina A. et al. Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries. Arquivos Brasileiros de Cardiologia, v. 107, n. 5, p. 427-436, 2016Tradução . . Disponível em: https://doi.org/10.5935/abc.20160147. Acesso em: 18 nov. 2024.
APA
Simplicio, J. A., Hipólito, U. V., Vale, G. T. do, Callera, G. E., Pereira, C. A., Touyz, R. M., et al. (2016). Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries. Arquivos Brasileiros de Cardiologia, 107( 5), 427-436. doi:10.5935/abc.20160147
NLM
Simplicio JA, Hipólito UV, Vale GT do, Callera GE, Pereira CA, Touyz RM, Tostes R de CA, Tirapelli CR. Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries [Internet]. Arquivos Brasileiros de Cardiologia. 2016 ; 107( 5): 427-436.[citado 2024 nov. 18 ] Available from: https://doi.org/10.5935/abc.20160147
Vancouver
Simplicio JA, Hipólito UV, Vale GT do, Callera GE, Pereira CA, Touyz RM, Tostes R de CA, Tirapelli CR. Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries [Internet]. Arquivos Brasileiros de Cardiologia. 2016 ; 107( 5): 427-436.[citado 2024 nov. 18 ] Available from: https://doi.org/10.5935/abc.20160147
Artigo de periodico
Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR (2016)
Perassa, Ligia A.; Graton, Murilo E.; Potje, Simone R.; Troiano, Jéssica A.; Lima, Mariana S.; Vale, Gabriel T.; Pereira, Ariana A.F.; Nakamune, Ana Claúdia M.S.; Sumida, Doris H.; Tirapelli, Carlos Renato ; Bendhack, Lusiane Maria; Antoniali, Cristina
Source: Vascular Pharmacology. Unidades: EERP, FCFRP
Subjects: ENDOTÉLIO VASCULAR, ÓXIDO NÍTRICO, PRESSÃO SANGUÍNEA, COMPOSTOS ORGÂNICOS, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERASSA, Ligia A. et al. Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR. Vascular Pharmacology, v. 87, p. 38-48, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.06.005. Acesso em: 18 nov. 2024.
APA
Perassa, L. A., Graton, M. E., Potje, S. R., Troiano, J. A., Lima, M. S., Vale, G. T., et al. (2016). Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR. Vascular Pharmacology, 87, 38-48. doi:10.1016/j.vph.2016.06.005
NLM
Perassa LA, Graton ME, Potje SR, Troiano JA, Lima MS, Vale GT, Pereira AAF, Nakamune ACMS, Sumida DH, Tirapelli CR, Bendhack LM, Antoniali C. Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR [Internet]. Vascular Pharmacology. 2016 ; 87 38-48.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.vph.2016.06.005
Vancouver
Perassa LA, Graton ME, Potje SR, Troiano JA, Lima MS, Vale GT, Pereira AAF, Nakamune ACMS, Sumida DH, Tirapelli CR, Bendhack LM, Antoniali C. Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR [Internet]. Vascular Pharmacology. 2016 ; 87 38-48.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.vph.2016.06.005

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