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Trabalho de evento-resumo
Addition of histone deacetylase inhibitor in transgenic human cell culture inhibits the secretion of lysosomal enzyme (2021)
Figueiredo, Lílian Louise Souza ; Coelho, Ana Carolina; Wiezel, Cláudia Emília Vieira; Tirapelli, Daniela Pretti da Cunha; D'almeida, Vânia; Abraham, Kuruvilla Joseph ; Siciliano, Velia; Weiss, Ron; Gerson, Stanton; Fontes, Aparecida Maria
Source: Resumos. Conference titles: Congresso Brasileiro de Genética = Brazilian Congress of Genetics. Unidades: FMRP, ICMC
Subjects: CULTURA DE CÉLULAS, ENZIMAS, DOENÇAS GENÉTICAS, BIOLOGIA SINTÉTICA
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ABNT
FIGUEIREDO, Lílian Louise Souza et al. Addition of histone deacetylase inhibitor in transgenic human cell culture inhibits the secretion of lysosomal enzyme. 2021, Anais.. [Ribeirão Preto]: Sociedade Brasileira de Genética - SBG, 2021. Disponível em: https://www.sbg.org.br/anais. Acesso em: 14 nov. 2024.
APA
Figueiredo, L. L. S., Coelho, A. C., Wiezel, C. E. V., Tirapelli, D. P. da C., D'almeida, V., Abraham, K. J., et al. (2021). Addition of histone deacetylase inhibitor in transgenic human cell culture inhibits the secretion of lysosomal enzyme. In Resumos. [Ribeirão Preto]: Sociedade Brasileira de Genética - SBG. Recuperado de https://www.sbg.org.br/anais
NLM
Figueiredo LLS, Coelho AC, Wiezel CEV, Tirapelli DP da C, D'almeida V, Abraham KJ, Siciliano V, Weiss R, Gerson S, Fontes AM. Addition of histone deacetylase inhibitor in transgenic human cell culture inhibits the secretion of lysosomal enzyme [Internet]. Resumos. 2021 ;[citado 2024 nov. 14 ] Available from: https://www.sbg.org.br/anais
Vancouver
Figueiredo LLS, Coelho AC, Wiezel CEV, Tirapelli DP da C, D'almeida V, Abraham KJ, Siciliano V, Weiss R, Gerson S, Fontes AM. Addition of histone deacetylase inhibitor in transgenic human cell culture inhibits the secretion of lysosomal enzyme [Internet]. Resumos. 2021 ;[citado 2024 nov. 14 ] Available from: https://www.sbg.org.br/anais
Artigo de periodico
Prognostic significance of genome-wide DNA methylation profiles within the randomized, phase 3, EORTC CATNON trial on non-1p/19q deleted anaplastic glioma (2021)
Tesileanu, C. Mircea S.; Van den Bent, Martin; Sanson, Marc; Wick, Wolfgang; Brandes, Alba; Clement, Paul M.; Erridge, Sara C.; Vogelbaum, Michael A.; Nowak, Anna; Baurain, Jean; Mason, Warren P.; Wheeler, Helen; Chinot, Olivier L.; Gill, Sanjeev; Griffin, Matthew; Rogers, Leland; Taal, Walter; Rudà, Roberta; Weller, Michael; McBain, Catherine; Linde, Myra E. van; Sabedot, Thais S.; Hoogstrate, Youri; Deimling, Andreas von; Heer, Iris de; IJcken, Wilfred F. J. van; Brouwer, Rutger W. W.; Aldape, Kenneth; Jenkins, Robert B.; Dubbink, Hendrikus; Kros, Johan M.; Wesseling, Pieter; Cheung, Kin Jip; Golfinopoulos, Vassilis; Baumert, Brigitta; Gorlia, Thierry; Noushmehr, Houtan; French, Pim
Source: Neuro-Oncology. Unidade: FMRP
Subjects: NEOPLASIAS CEREBRAIS, METILAÇÃO DE DNA, GLIOMA, MUTAÇÃO, ESTUDOS PROSPECTIVOS
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TESILEANU, C. Mircea S. et al. Prognostic significance of genome-wide DNA methylation profiles within the randomized, phase 3, EORTC CATNON trial on non-1p/19q deleted anaplastic glioma. Neuro-Oncology, v. 23, n. 9, p. 1547-1559, 2021Tradução . . Disponível em: https://doi.org/10.1093/neuonc/noab088. Acesso em: 14 nov. 2024.
APA
Tesileanu, C. M. S., Van den Bent, M., Sanson, M., Wick, W., Brandes, A., Clement, P. M., et al. (2021). Prognostic significance of genome-wide DNA methylation profiles within the randomized, phase 3, EORTC CATNON trial on non-1p/19q deleted anaplastic glioma. Neuro-Oncology, 23( 9), 1547-1559. doi:10.1093/neuonc/noab088
NLM
Tesileanu CMS, Van den Bent M, Sanson M, Wick W, Brandes A, Clement PM, Erridge SC, Vogelbaum MA, Nowak A, Baurain J, Mason WP, Wheeler H, Chinot OL, Gill S, Griffin M, Rogers L, Taal W, Rudà R, Weller M, McBain C, Linde ME van, Sabedot TS, Hoogstrate Y, Deimling A von, Heer I de, IJcken WFJ van, Brouwer RWW, Aldape K, Jenkins RB, Dubbink H, Kros JM, Wesseling P, Cheung KJ, Golfinopoulos V, Baumert B, Gorlia T, Noushmehr H, French P. Prognostic significance of genome-wide DNA methylation profiles within the randomized, phase 3, EORTC CATNON trial on non-1p/19q deleted anaplastic glioma [Internet]. Neuro-Oncology. 2021 ; 23( 9): 1547-1559.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/noab088
Vancouver
Tesileanu CMS, Van den Bent M, Sanson M, Wick W, Brandes A, Clement PM, Erridge SC, Vogelbaum MA, Nowak A, Baurain J, Mason WP, Wheeler H, Chinot OL, Gill S, Griffin M, Rogers L, Taal W, Rudà R, Weller M, McBain C, Linde ME van, Sabedot TS, Hoogstrate Y, Deimling A von, Heer I de, IJcken WFJ van, Brouwer RWW, Aldape K, Jenkins RB, Dubbink H, Kros JM, Wesseling P, Cheung KJ, Golfinopoulos V, Baumert B, Gorlia T, Noushmehr H, French P. Prognostic significance of genome-wide DNA methylation profiles within the randomized, phase 3, EORTC CATNON trial on non-1p/19q deleted anaplastic glioma [Internet]. Neuro-Oncology. 2021 ; 23( 9): 1547-1559.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/noab088
Trabalho de evento-resumo periodico
Addition of histone deacetylase inhibitor in human cell cultures generated by gene therapy and synthetic biology approaches inhibits the secretion of lysosomal enzyme (2021)
Figueiredo, Lílian L. S.; Wiezel, Cláudia Emília Vieira ; Tirapelli, Daniela Pretti da Cunha ; D'Almeida, Vania; Siciliano, Velia; Weiss, Ron; Abraham, Kuruvilla Joseph ; Gerson, Stanton L.; Fontes, Aparecida Maria
Source: Molecular Therapy. Conference titles: ASGCT Annual Meeting. Unidades: FMRP, ICMC
Subjects: CULTURA DE CÉLULAS, ENZIMAS, DOENÇAS GENÉTICAS, BIOLOGIA SINTÉTICA
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ABNT
FIGUEIREDO, Lílian L. S. et al. Addition of histone deacetylase inhibitor in human cell cultures generated by gene therapy and synthetic biology approaches inhibits the secretion of lysosomal enzyme. Molecular Therapy. Cambridge: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 14 nov. 2024. , 2021
APA
Figueiredo, L. L. S., Wiezel, C. E. V., Tirapelli, D. P. da C., D'Almeida, V., Siciliano, V., Weiss, R., et al. (2021). Addition of histone deacetylase inhibitor in human cell cultures generated by gene therapy and synthetic biology approaches inhibits the secretion of lysosomal enzyme. Molecular Therapy. Cambridge: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Figueiredo LLS, Wiezel CEV, Tirapelli DP da C, D'Almeida V, Siciliano V, Weiss R, Abraham KJ, Gerson SL, Fontes AM. Addition of histone deacetylase inhibitor in human cell cultures generated by gene therapy and synthetic biology approaches inhibits the secretion of lysosomal enzyme. Molecular Therapy. 2021 ; 29( 4): 251.[citado 2024 nov. 14 ]
Vancouver
Figueiredo LLS, Wiezel CEV, Tirapelli DP da C, D'Almeida V, Siciliano V, Weiss R, Abraham KJ, Gerson SL, Fontes AM. Addition of histone deacetylase inhibitor in human cell cultures generated by gene therapy and synthetic biology approaches inhibits the secretion of lysosomal enzyme. Molecular Therapy. 2021 ; 29( 4): 251.[citado 2024 nov. 14 ]
Artigo de periodico
Loss of H3K27me3 in meningiomas (2021)
Nassir, Farshad; Wang, Justin Z.; Singh, Olivia; Karimi, Shirin; Dalcourt, Tatyana; Ijad, Nazanin; Pirouzmand, Neda; Ng, Ho-Keung; Saladino, Andrea; Noushmehr, Houtan
Source: Neuro-Oncology. Unidade: FMRP
Subjects: NEOPLASIAS MENÍNGEAS, MENINGIOMA, RECIDIVA LOCAL DE NEOPLASIA, REPRODUTIBILIDADE DE RESULTADOS
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ABNT
NASSIR, Farshad et al. Loss of H3K27me3 in meningiomas. Neuro-Oncology, v. 23, n. 8, p. 1282-1291, 2021Tradução . . Disponível em: https://doi.org/10.1093/neuonc/noab036. Acesso em: 14 nov. 2024.
APA
Nassir, F., Wang, J. Z., Singh, O., Karimi, S., Dalcourt, T., Ijad, N., et al. (2021). Loss of H3K27me3 in meningiomas. Neuro-Oncology, 23( 8), 1282-1291. doi:10.1093/neuonc/noab036
NLM
Nassir F, Wang JZ, Singh O, Karimi S, Dalcourt T, Ijad N, Pirouzmand N, Ng H-K, Saladino A, Noushmehr H. Loss of H3K27me3 in meningiomas [Internet]. Neuro-Oncology. 2021 ; 23( 8): 1282-1291.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/noab036
Vancouver
Nassir F, Wang JZ, Singh O, Karimi S, Dalcourt T, Ijad N, Pirouzmand N, Ng H-K, Saladino A, Noushmehr H. Loss of H3K27me3 in meningiomas [Internet]. Neuro-Oncology. 2021 ; 23( 8): 1282-1291.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/noab036
Artigo de periodico
Interpreting pathways to discover cancer driver genes with Moonlight (2020)
Colaprico, Antonio; Olsen, Catharina; Bailey, Matthew H.; Odom, Gabriel J.; Terkelsen, Thilde; Silva, Tiago Chedraoui ; Olsen, André V.; Cantini, Laura; Zinovyev, Andrei; Barillot, Emmanuel; Noushmehr, Houtan ; Bertoli, Gloria; Castiglioni, Isabella; Cava, Claudia; Bontempi, Gianluca; Chen, Xi Steven; Papaleo, Elena
Source: Nature Communications. Unidade: FMRP
Subjects: BIOINFORMÁTICA, METILAÇÃO, DNA, GENES, NEOPLASIAS, ONCOGENES
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ABNT
COLAPRICO, Antonio et al. Interpreting pathways to discover cancer driver genes with Moonlight. Nature Communications, v. 311, p. 1-17, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41467-019-13803-0. Acesso em: 14 nov. 2024.
APA
Colaprico, A., Olsen, C., Bailey, M. H., Odom, G. J., Terkelsen, T., Silva, T. C., et al. (2020). Interpreting pathways to discover cancer driver genes with Moonlight. Nature Communications, 311, 1-17. doi:10.1038/s41467-019-13803-0
NLM
Colaprico A, Olsen C, Bailey MH, Odom GJ, Terkelsen T, Silva TC, Olsen AV, Cantini L, Zinovyev A, Barillot E, Noushmehr H, Bertoli G, Castiglioni I, Cava C, Bontempi G, Chen XS, Papaleo E. Interpreting pathways to discover cancer driver genes with Moonlight [Internet]. Nature Communications. 2020 ; 311 1-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1038/s41467-019-13803-0
Vancouver
Colaprico A, Olsen C, Bailey MH, Odom GJ, Terkelsen T, Silva TC, Olsen AV, Cantini L, Zinovyev A, Barillot E, Noushmehr H, Bertoli G, Castiglioni I, Cava C, Bontempi G, Chen XS, Papaleo E. Interpreting pathways to discover cancer driver genes with Moonlight [Internet]. Nature Communications. 2020 ; 311 1-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1038/s41467-019-13803-0
Trabalho de evento-resumo periodico
Prognostic significance of epigenetic subtypes and CPGS associated with progression to G-CIMP low in the eortc randomized phase III intergroup catnon (2020)
Tesileanu, C. Mircea S.; Van den Bent, Martin; Sabedot, Thais; Sanson, Marc; Brandes, Alba; Wick, Wolfgang; Clement, Paul; Erridge, Sarah; Vogelbaum, Michael; Nowak, Anna; Baurain, Jean; Mason, Warren; Wheeler, Helen; Weller, Michael; Heer, Iris de; Dubbink, Hendrikus; Kros, Johan M; Aldape, Kenneth; Wesseling, Pieter; Golfinopoulos, Vassilis; Gorlia, Thierry; Baumert, Brigitta; Noushmehr, Houtan ; French, Pim
Source: Neuro-Oncology. Conference titles: Annual Scientific Meeting and Education Day of the Society for Neuro-Oncology. Unidade: FMRP
Subjects: EPIGÊNESE GENÉTICA, GLIOMA, NEOPLASIAS CEREBRAIS, METILAÇÃO DE DNA, RADIOTERAPIA
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ABNT
TESILEANU, C. Mircea S. et al. Prognostic significance of epigenetic subtypes and CPGS associated with progression to G-CIMP low in the eortc randomized phase III intergroup catnon. Neuro-Oncology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1093/neuonc/noaa215.693. Acesso em: 14 nov. 2024. , 2020
APA
Tesileanu, C. M. S., Van den Bent, M., Sabedot, T., Sanson, M., Brandes, A., Wick, W., et al. (2020). Prognostic significance of epigenetic subtypes and CPGS associated with progression to G-CIMP low in the eortc randomized phase III intergroup catnon. Neuro-Oncology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1093/neuonc/noaa215.693
NLM
Tesileanu CMS, Van den Bent M, Sabedot T, Sanson M, Brandes A, Wick W, Clement P, Erridge S, Vogelbaum M, Nowak A, Baurain J, Mason W, Wheeler H, Weller M, Heer I de, Dubbink H, Kros JM, Aldape K, Wesseling P, Golfinopoulos V, Gorlia T, Baumert B, Noushmehr H, French P. Prognostic significance of epigenetic subtypes and CPGS associated with progression to G-CIMP low in the eortc randomized phase III intergroup catnon [Internet]. Neuro-Oncology. 2020 ; 22( suppl. 2): ii166.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/noaa215.693
Vancouver
Tesileanu CMS, Van den Bent M, Sabedot T, Sanson M, Brandes A, Wick W, Clement P, Erridge S, Vogelbaum M, Nowak A, Baurain J, Mason W, Wheeler H, Weller M, Heer I de, Dubbink H, Kros JM, Aldape K, Wesseling P, Golfinopoulos V, Gorlia T, Baumert B, Noushmehr H, French P. Prognostic significance of epigenetic subtypes and CPGS associated with progression to G-CIMP low in the eortc randomized phase III intergroup catnon [Internet]. Neuro-Oncology. 2020 ; 22( suppl. 2): ii166.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/noaa215.693
Artigo de periodico
RGBM: regularized gradient boosting machines for identification of the transcriptional regulators of discrete glioma subtypes (2018)
Mall, Raghvendra; Cerulo, Luigi; Garofano, Luciano; Frattini, Veronique; Kunji, Khalid; Bensmail, Halima; Sabedot, Thais Sarraf; Noushmehr, Houtan; Lasorella, Anna; Iavarone, Antonio; Ceccarelli, Michele
Source: Nucleic Acids Research. Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, NEOPLASIAS CEREBRAIS
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ABNT
MALL, Raghvendra et al. RGBM: regularized gradient boosting machines for identification of the transcriptional regulators of discrete glioma subtypes. Nucleic Acids Research, v. 46, n. 7, 2018Tradução . . Disponível em: https://doi.org/10.1093/nar/gky015. Acesso em: 14 nov. 2024.
APA
Mall, R., Cerulo, L., Garofano, L., Frattini, V., Kunji, K., Bensmail, H., et al. (2018). RGBM: regularized gradient boosting machines for identification of the transcriptional regulators of discrete glioma subtypes. Nucleic Acids Research, 46( 7). doi:10.1093/nar/gky015
NLM
Mall R, Cerulo L, Garofano L, Frattini V, Kunji K, Bensmail H, Sabedot TS, Noushmehr H, Lasorella A, Iavarone A, Ceccarelli M. RGBM: regularized gradient boosting machines for identification of the transcriptional regulators of discrete glioma subtypes [Internet]. Nucleic Acids Research. 2018 ; 46( 7):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/nar/gky015
Vancouver
Mall R, Cerulo L, Garofano L, Frattini V, Kunji K, Bensmail H, Sabedot TS, Noushmehr H, Lasorella A, Iavarone A, Ceccarelli M. RGBM: regularized gradient boosting machines for identification of the transcriptional regulators of discrete glioma subtypes [Internet]. Nucleic Acids Research. 2018 ; 46( 7):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/nar/gky015
Artigo de periodico
SpidermiR: an R/Bioconductor package for integrative analysis with miRNA data (2017)
Cava, Claudia; Colaprico, Antonio; Bertoli, Gloria; Graudenzi, Gloria; Silva, Tiago C.; Olsen, Catharina; Noushmehr, Houtan; Bontempi, Gianluca; Mauri, Giancarlo; Castiglioni, Isabella
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: NEOPLASIAS MAMÁRIAS, MICRORNAS, PROTEÍNAS
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ABNT
CAVA, Claudia et al. SpidermiR: an R/Bioconductor package for integrative analysis with miRNA data. International Journal of Molecular Sciences, v. 18, n. 2, 2017Tradução . . Disponível em: https://doi.org/10.3390/ijms18020274. Acesso em: 14 nov. 2024.
APA
Cava, C., Colaprico, A., Bertoli, G., Graudenzi, G., Silva, T. C., Olsen, C., et al. (2017). SpidermiR: an R/Bioconductor package for integrative analysis with miRNA data. International Journal of Molecular Sciences, 18( 2). doi:10.3390/ijms18020274
NLM
Cava C, Colaprico A, Bertoli G, Graudenzi G, Silva TC, Olsen C, Noushmehr H, Bontempi G, Mauri G, Castiglioni I. SpidermiR: an R/Bioconductor package for integrative analysis with miRNA data [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 2):[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/ijms18020274
Vancouver
Cava C, Colaprico A, Bertoli G, Graudenzi G, Silva TC, Olsen C, Noushmehr H, Bontempi G, Mauri G, Castiglioni I. SpidermiR: an R/Bioconductor package for integrative analysis with miRNA data [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 2):[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/ijms18020274
Artigo de periodico
Molecular profiling reveals biologically discrete subsets and pathways of progression in diffuse glioma (2016)
Ceccarelli, Michele; Barthel, Floris P.; Malta, Tathiane M.; Sabedot, Thais S.; Salama, Sofie R.; Murray, Bradley A.; Morozova, Olena; Newton, Yulia; Radenbaugh, Amie; Pagnotta, Stefano M.; Anjum, Samreen; Wang, Jiguang; Manyam, Ganiraju; Zoppoli, Pietro; Ling, Shiyun; Rao, Arjun A.; Grifford, Mia; Cherniack, Andrew D.; Zhang, Hailei; Poisson, Laila; Carlotti Júnior, Carlos Gilberto; Tirapelli, Daniela Pretti da Cunha; Rao, Arvind; Mikkelsen, Tom; Lau, Ching C.; Yung, W.K. Alfred; Rabadan, Raul; Huse, Jason; Brat, Daniel J.; Lehman, Norman L.; Barnholtz-Sloan, Jill S.; Zheng, Siyuan; Hess, Kenneth; Rao, Ganesh; Meyerson, Matthew; Beroukhim, Rameen; Cooper, Lee; Akbani, Rehan; Wrensch, Margaret; Haussler, David; Aldape, Kenneth D.; Laird, Peter W.; Gutmann, David H.; Noushmehr, Houtan; Iavarone, Antonio; Verhaak, Roel G.W.
Source: Cell. Unidade: FMRP
Subjects: GLIOMA, NEOPLASIAS CEREBRAIS, PROLIFERAÇÃO CELULAR, METILAÇÃO DE DNA, MUTAÇÃO GENÉTICA
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CECCARELLI, Michele et al. Molecular profiling reveals biologically discrete subsets and pathways of progression in diffuse glioma. Cell, v. 164, p. 550-563, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.cell.2015.12.028. Acesso em: 14 nov. 2024.
APA
Ceccarelli, M., Barthel, F. P., Malta, T. M., Sabedot, T. S., Salama, S. R., Murray, B. A., et al. (2016). Molecular profiling reveals biologically discrete subsets and pathways of progression in diffuse glioma. Cell, 164, 550-563. doi:10.1016/j.cell.2015.12.028
NLM
Ceccarelli M, Barthel FP, Malta TM, Sabedot TS, Salama SR, Murray BA, Morozova O, Newton Y, Radenbaugh A, Pagnotta SM, Anjum S, Wang J, Manyam G, Zoppoli P, Ling S, Rao AA, Grifford M, Cherniack AD, Zhang H, Poisson L, Carlotti Júnior CG, Tirapelli DP da C, Rao A, Mikkelsen T, Lau CC, Yung WKA, Rabadan R, Huse J, Brat DJ, Lehman NL, Barnholtz-Sloan JS, Zheng S, Hess K, Rao G, Meyerson M, Beroukhim R, Cooper L, Akbani R, Wrensch M, Haussler D, Aldape KD, Laird PW, Gutmann DH, Noushmehr H, Iavarone A, Verhaak RGW. Molecular profiling reveals biologically discrete subsets and pathways of progression in diffuse glioma [Internet]. Cell. 2016 ; 164 550-563.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cell.2015.12.028
Vancouver
Ceccarelli M, Barthel FP, Malta TM, Sabedot TS, Salama SR, Murray BA, Morozova O, Newton Y, Radenbaugh A, Pagnotta SM, Anjum S, Wang J, Manyam G, Zoppoli P, Ling S, Rao AA, Grifford M, Cherniack AD, Zhang H, Poisson L, Carlotti Júnior CG, Tirapelli DP da C, Rao A, Mikkelsen T, Lau CC, Yung WKA, Rabadan R, Huse J, Brat DJ, Lehman NL, Barnholtz-Sloan JS, Zheng S, Hess K, Rao G, Meyerson M, Beroukhim R, Cooper L, Akbani R, Wrensch M, Haussler D, Aldape KD, Laird PW, Gutmann DH, Noushmehr H, Iavarone A, Verhaak RGW. Molecular profiling reveals biologically discrete subsets and pathways of progression in diffuse glioma [Internet]. Cell. 2016 ; 164 550-563.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cell.2015.12.028
Artigo de periodico
TCGAbiolinks: an R/Bioconductor package for integrative analysis of TCGA data (2016)
Colaprico, Antonio; Silva, Tiago C.; Olsen, Catharina; Garofano, Luciano; Cava, Claudia; Garolini, Davide; Sabedot, Thais S.; Malta, Tathiane M.; Pagnotta, Stefano; Castiglioni, Isabella; Ceccarelli, Michele; Bontempi, Gianluca; Noushmer, Houtan
Source: Nucleic Acids Research. Unidade: FMRP
Subjects: BIOMARCADORES, METILAÇÃO DE DNA, MINERAÇÃO DE DADOS, GENOMAS, NEOPLASIAS
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ABNT
COLAPRICO, Antonio et al. TCGAbiolinks: an R/Bioconductor package for integrative analysis of TCGA data. Nucleic Acids Research, v. 44, n. 8, 2016Tradução . . Disponível em: https://doi.org/10.1093/nar/gkv1507. Acesso em: 14 nov. 2024.
APA
Colaprico, A., Silva, T. C., Olsen, C., Garofano, L., Cava, C., Garolini, D., et al. (2016). TCGAbiolinks: an R/Bioconductor package for integrative analysis of TCGA data. Nucleic Acids Research, 44( 8). doi:10.1093/nar/gkv1507
NLM
Colaprico A, Silva TC, Olsen C, Garofano L, Cava C, Garolini D, Sabedot TS, Malta TM, Pagnotta S, Castiglioni I, Ceccarelli M, Bontempi G, Noushmer H. TCGAbiolinks: an R/Bioconductor package for integrative analysis of TCGA data [Internet]. Nucleic Acids Research. 2016 ; 44( 8):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/nar/gkv1507
Vancouver
Colaprico A, Silva TC, Olsen C, Garofano L, Cava C, Garolini D, Sabedot TS, Malta TM, Pagnotta S, Castiglioni I, Ceccarelli M, Bontempi G, Noushmer H. TCGAbiolinks: an R/Bioconductor package for integrative analysis of TCGA data [Internet]. Nucleic Acids Research. 2016 ; 44( 8):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/nar/gkv1507
Artigo de periodico
TCGA workflow: analyze cancer genomics and epigenomics data using bioconductor packages [version 2; referees: 1 approved, 2 approved with reservations] (2016)
Silva, Tiago C.; Colaprico, Antonio; Olsen, Catharina; D'Angelo, Fulvio; Bontempi, Gianluca; Ceccarelli, Michele; Noushmehr, Houtan
Source: F1000 Research. Unidade: FMRP
Subjects: BIOINFORMÁTICA, NEOPLASIAS, EPIGÊNESE GENÉTICA, GENÔMICA
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ABNT
SILVA, Tiago C. et al. TCGA workflow: analyze cancer genomics and epigenomics data using bioconductor packages [version 2; referees: 1 approved, 2 approved with reservations]. F1000 Research, v. 5, 2016Tradução . . Disponível em: https://doi.org/10.12688/f1000research.8923.2. Acesso em: 14 nov. 2024.
APA
Silva, T. C., Colaprico, A., Olsen, C., D'Angelo, F., Bontempi, G., Ceccarelli, M., & Noushmehr, H. (2016). TCGA workflow: analyze cancer genomics and epigenomics data using bioconductor packages [version 2; referees: 1 approved, 2 approved with reservations]. F1000 Research, 5. doi:10.12688/f1000research.8923.2
NLM
Silva TC, Colaprico A, Olsen C, D'Angelo F, Bontempi G, Ceccarelli M, Noushmehr H. TCGA workflow: analyze cancer genomics and epigenomics data using bioconductor packages [version 2; referees: 1 approved, 2 approved with reservations] [Internet]. F1000 Research. 2016 ; 5[citado 2024 nov. 14 ] Available from: https://doi.org/10.12688/f1000research.8923.2
Vancouver
Silva TC, Colaprico A, Olsen C, D'Angelo F, Bontempi G, Ceccarelli M, Noushmehr H. TCGA workflow: analyze cancer genomics and epigenomics data using bioconductor packages [version 2; referees: 1 approved, 2 approved with reservations] [Internet]. F1000 Research. 2016 ; 5[citado 2024 nov. 14 ] Available from: https://doi.org/10.12688/f1000research.8923.2
Trabalho de evento-resumo periodico
Epigenetic alterations at intergenic regions associated with progression in a subset of IDH mutant gliomas (2016)
Sabedot, Thais Sarraf; Malta, Tathiane; Ceccarelli, Michele; Dihn, Huy; Berman, Benjamin; Noushmehr, Houtan
Source: Neuro-Oncology. Conference titles: Annual Scientific Meeting and Education Day of the Society for Neuro-Oncology. Unidade: FMRP
Subjects: EPIGÊNESE GENÉTICA, GLIOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SABEDOT, Thais Sarraf et al. Epigenetic alterations at intergenic regions associated with progression in a subset of IDH mutant gliomas. Neuro-Oncology. Oxford: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1093/neuonc/now212.339. Acesso em: 14 nov. 2024. , 2016
APA
Sabedot, T. S., Malta, T., Ceccarelli, M., Dihn, H., Berman, B., & Noushmehr, H. (2016). Epigenetic alterations at intergenic regions associated with progression in a subset of IDH mutant gliomas. Neuro-Oncology. Oxford: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1093/neuonc/now212.339
NLM
Sabedot TS, Malta T, Ceccarelli M, Dihn H, Berman B, Noushmehr H. Epigenetic alterations at intergenic regions associated with progression in a subset of IDH mutant gliomas [Internet]. Neuro-Oncology. 2016 ; 18( suppl_6): vi81.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/now212.339
Vancouver
Sabedot TS, Malta T, Ceccarelli M, Dihn H, Berman B, Noushmehr H. Epigenetic alterations at intergenic regions associated with progression in a subset of IDH mutant gliomas [Internet]. Neuro-Oncology. 2016 ; 18( suppl_6): vi81.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/now212.339
Trabalho de evento-resumo
TWIST2 is the cause of ablepharon macrostomia and barber say syndromes: reascertainment of two cases lost to follow-up as keys to solving these conditions (2015)
Markello, T.; Marchegiani, S.; Davis, T.; Tessadori, F.; van Haaften, G.; Brancati, F.; Pusey, B.; Wolfe, L.; Tifft, C.; Ferraz, Victor Evangelista de Faria; Roche, N.; Ramos, F.; Malicdan, M.; Boerkoel, C.; Gahl, W. A.; Vries, B. de; van Haelst, M.; Zenker, M.
Source: Poster Abstracts. Conference titles: Annual Meeting of the American Society of Human Genetics. Unidade: FMRP
Subjects: MACROSTOMIA, DOENÇAS GENÉTICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARKELLO, T. et al. TWIST2 is the cause of ablepharon macrostomia and barber say syndromes: reascertainment of two cases lost to follow-up as keys to solving these conditions. 2015, Anais.. Baltimore: ASHG, 2015. . Acesso em: 14 nov. 2024.
APA
Markello, T., Marchegiani, S., Davis, T., Tessadori, F., van Haaften, G., Brancati, F., et al. (2015). TWIST2 is the cause of ablepharon macrostomia and barber say syndromes: reascertainment of two cases lost to follow-up as keys to solving these conditions. In Poster Abstracts. Baltimore: ASHG.
NLM
Markello T, Marchegiani S, Davis T, Tessadori F, van Haaften G, Brancati F, Pusey B, Wolfe L, Tifft C, Ferraz VE de F, Roche N, Ramos F, Malicdan M, Boerkoel C, Gahl WA, Vries B de, van Haelst M, Zenker M. TWIST2 is the cause of ablepharon macrostomia and barber say syndromes: reascertainment of two cases lost to follow-up as keys to solving these conditions. Poster Abstracts. 2015 ;[citado 2024 nov. 14 ]
Vancouver
Markello T, Marchegiani S, Davis T, Tessadori F, van Haaften G, Brancati F, Pusey B, Wolfe L, Tifft C, Ferraz VE de F, Roche N, Ramos F, Malicdan M, Boerkoel C, Gahl WA, Vries B de, van Haelst M, Zenker M. TWIST2 is the cause of ablepharon macrostomia and barber say syndromes: reascertainment of two cases lost to follow-up as keys to solving these conditions. Poster Abstracts. 2015 ;[citado 2024 nov. 14 ]
Trabalho de evento-resumo periodico
Epigenomic (DNA methylation and expression) signatures define subsets of both IDHmut and IDHwt glioma with distinct clinical outcomes (2015)
Malta, Tathiane; Sabedot, Thais; Ceccarelli, Michele; Barthel, Floris; Pagnotta, Stefano; Lavarone, Antonio; Verhaak, Roel; Noushmehr, Houtan
Source: Neuro-Oncology. Conference titles: Annual Scientific Meeting of the Society for Neuro-Oncology. Unidade: FMRP
Subjects: METILAÇÃO DE DNA, GLIOMA, DNA, GENOMAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MALTA, Tathiane et al. Epigenomic (DNA methylation and expression) signatures define subsets of both IDHmut and IDHwt glioma with distinct clinical outcomes. Neuro-Oncology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1093/neuonc/nov214.14. Acesso em: 14 nov. 2024. , 2015
APA
Malta, T., Sabedot, T., Ceccarelli, M., Barthel, F., Pagnotta, S., Lavarone, A., et al. (2015). Epigenomic (DNA methylation and expression) signatures define subsets of both IDHmut and IDHwt glioma with distinct clinical outcomes. Neuro-Oncology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1093/neuonc/nov214.14
NLM
Malta T, Sabedot T, Ceccarelli M, Barthel F, Pagnotta S, Lavarone A, Verhaak R, Noushmehr H. Epigenomic (DNA methylation and expression) signatures define subsets of both IDHmut and IDHwt glioma with distinct clinical outcomes [Internet]. Neuro-Oncology. 2015 ; 17[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/nov214.14
Vancouver
Malta T, Sabedot T, Ceccarelli M, Barthel F, Pagnotta S, Lavarone A, Verhaak R, Noushmehr H. Epigenomic (DNA methylation and expression) signatures define subsets of both IDHmut and IDHwt glioma with distinct clinical outcomes [Internet]. Neuro-Oncology. 2015 ; 17[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/nov214.14
Trabalho de evento-resumo periodico
A pan-glioma characterization of genomic, epigenomic and transcriptomic activities reveals novel relationships between histological subtypes and molecular signatures (2015)
Barthel, Floris P.; Ceccarelli, Michele; Malta, Tathiane M.; Sabedot, Thais S.; Salama, Sofie R.; Pagnotta, Stefano M.; Murray, Bradley A.; Morozova, Olena; Newton, Yulia; Brat, Daniel J.; Cherniack, Andrew D.; Zhang, Hailei; Poisson, Laila; Cooper, Lee; Rabadan, Raul; Laird, Peter W.; Gutmann, David H.; Noushmehr, Houtan; Lavarone, Antonio; Verhaak, G. W.
Source: Neuro-Oncology. Conference titles: Annual Scientific Meeting of the Society for Neuro-Oncology. Unidade: FMRP
Subjects: MUTAÇÃO GENÉTICA, GLIOMA, GENOMAS, EXPRESSÃO GÊNICA, METILAÇÃO DE DNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARTHEL, Floris P. et al. A pan-glioma characterization of genomic, epigenomic and transcriptomic activities reveals novel relationships between histological subtypes and molecular signatures. Neuro-Oncology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1093/neuonc/nov215.06. Acesso em: 14 nov. 2024. , 2015
APA
Barthel, F. P., Ceccarelli, M., Malta, T. M., Sabedot, T. S., Salama, S. R., Pagnotta, S. M., et al. (2015). A pan-glioma characterization of genomic, epigenomic and transcriptomic activities reveals novel relationships between histological subtypes and molecular signatures. Neuro-Oncology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1093/neuonc/nov215.06
NLM
Barthel FP, Ceccarelli M, Malta TM, Sabedot TS, Salama SR, Pagnotta SM, Murray BA, Morozova O, Newton Y, Brat DJ, Cherniack AD, Zhang H, Poisson L, Cooper L, Rabadan R, Laird PW, Gutmann DH, Noushmehr H, Lavarone A, Verhaak GW. A pan-glioma characterization of genomic, epigenomic and transcriptomic activities reveals novel relationships between histological subtypes and molecular signatures [Internet]. Neuro-Oncology. 2015 ; 17[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/nov215.06
Vancouver
Barthel FP, Ceccarelli M, Malta TM, Sabedot TS, Salama SR, Pagnotta SM, Murray BA, Morozova O, Newton Y, Brat DJ, Cherniack AD, Zhang H, Poisson L, Cooper L, Rabadan R, Laird PW, Gutmann DH, Noushmehr H, Lavarone A, Verhaak GW. A pan-glioma characterization of genomic, epigenomic and transcriptomic activities reveals novel relationships between histological subtypes and molecular signatures [Internet]. Neuro-Oncology. 2015 ; 17[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/neuonc/nov215.06

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