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Artigo de periodico
Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model (2021)
Villaça, Catharina de Barros Pimentel ; Paula, Carolina Cavalcante de ; Oliveira, Caroline Cruz de ; Vilas-Boas, Eloisa Aparecida ; Silva, Junia Carolina dos Santos ; Oliveira, Sérgio Ferreira de; Abdulkader, Fernando Rodrigues de Moraes ; Ferreira, Sandra Mara; Ortis, Fernanda
Source: Experimental Physiology. Unidade: ICB
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, EXERCÍCIO FÍSICO, DIABETES MELLITUS, MODELOS ANIMAIS DE DOENÇAS, ILHOTAS DE LANGERHANS, INFLAMAÇÃO, CAMUNDONGOS, ANTINEOPLÁSICOS, ÍNSULA DO PÂNCREAS ANIMAL, GLUCAGON
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ABNT
VILLAÇA, Catharina de Barros Pimentel et al. Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model. Experimental Physiology, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1113/EP088872. Acesso em: 12 nov. 2024.
APA
Villaça, C. de B. P., Paula, C. C. de, Oliveira, C. C. de, Vilas-Boas, E. A., Silva, J. C. dos S., Oliveira, S. F. de, et al. (2021). Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model. Experimental Physiology, 1-16. doi:10.1113/EP088872
NLM
Villaça C de BP, Paula CC de, Oliveira CC de, Vilas-Boas EA, Silva JC dos S, Oliveira SF de, Abdulkader FR de M, Ferreira SM, Ortis F. Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model [Internet]. Experimental Physiology. 2021 ; 1-16.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1113/EP088872
Vancouver
Villaça C de BP, Paula CC de, Oliveira CC de, Vilas-Boas EA, Silva JC dos S, Oliveira SF de, Abdulkader FR de M, Ferreira SM, Ortis F. Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model [Internet]. Experimental Physiology. 2021 ; 1-16.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1113/EP088872
Artigo de periodico
Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88 (2017)
Castoldi, Angela; Oliveira, Vinícius Alves de; Aguiar, Cristhiane Fávero; Amano, Mariane Tami; Lee, Jennifer; Miyagi, Marcelli Terumi; Latância, Marcela Teatin; Braga, Tárcio Teodoro; Silva, Marina Burgos da; Ignacio, Aline; Lima, Joanna Darck Carola Correia; Loures, Flavio Vieira; Albuquerque, José Antônio Tavares de; Macêdo, Marina Barguil; Almeida, Rafael Ribeiro; Gaiarsa, Jonas W.; Martinez, Luis Alberto Luevano; Belchior, Thiago; Hiyane, Meire Ioshie; Brown, Gordon D; Mori, Marcelo A.; Hoffmann, Christian ; Seelaender, Marília Cerqueira Leite; Festuccia, William Tadeu Lara ; Vieira, Pedro Manoel Moraes; Câmara, Niels Olsen Saraiva
Source: Cell Reports. Unidades: ICB, FCF
Subjects: OBESIDADE, INSULINA
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CASTOLDI, Angela et al. Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88. Cell Reports, v. 19, p. 2272-2288, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2017.05.059. Acesso em: 12 nov. 2024.
APA
Castoldi, A., Oliveira, V. A. de, Aguiar, C. F., Amano, M. T., Lee, J., Miyagi, M. T., et al. (2017). Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88. Cell Reports, 19, 2272-2288. doi:10.1016/j.celrep.2017.05.059
NLM
Castoldi A, Oliveira VA de, Aguiar CF, Amano MT, Lee J, Miyagi MT, Latância MT, Braga TT, Silva MB da, Ignacio A, Lima JDCC, Loures FV, Albuquerque JAT de, Macêdo MB, Almeida RR, Gaiarsa JW, Martinez LAL, Belchior T, Hiyane MI, Brown GD, Mori MA, Hoffmann C, Seelaender MCL, Festuccia WTL, Vieira PMM, Câmara NOS. Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88 [Internet]. Cell Reports. 2017 ; 19 2272-2288.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.celrep.2017.05.059
Vancouver
Castoldi A, Oliveira VA de, Aguiar CF, Amano MT, Lee J, Miyagi MT, Latância MT, Braga TT, Silva MB da, Ignacio A, Lima JDCC, Loures FV, Albuquerque JAT de, Macêdo MB, Almeida RR, Gaiarsa JW, Martinez LAL, Belchior T, Hiyane MI, Brown GD, Mori MA, Hoffmann C, Seelaender MCL, Festuccia WTL, Vieira PMM, Câmara NOS. Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88 [Internet]. Cell Reports. 2017 ; 19 2272-2288.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.celrep.2017.05.059
Artigo de periodico
Metabolic impact of light phase-restricted fructose consumption is linked to changes in hypothalamic AMPK phosphorylation and melatonina production in rats (2017)
Faria, Juliana de Almeida; Araújo, Thiago Matos F. de; Razolli, Daniela S.; Ignácio-Souza, Letícia Martins; Souza, Dailson Nogueira; Bordin, Silvana ; Anhê, Gabriel Forato
Source: Nutrients. Unidade: ICB
Assunto: FISIOLOGIA
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FARIA, Juliana de Almeida et al. Metabolic impact of light phase-restricted fructose consumption is linked to changes in hypothalamic AMPK phosphorylation and melatonina production in rats. Nutrients, v. 9, n. 4, p. 1-18, 2017Tradução . . Disponível em: https://doi.org/10.3390/nu9040332. Acesso em: 12 nov. 2024.
APA
Faria, J. de A., Araújo, T. M. F. de, Razolli, D. S., Ignácio-Souza, L. M., Souza, D. N., Bordin, S., & Anhê, G. F. (2017). Metabolic impact of light phase-restricted fructose consumption is linked to changes in hypothalamic AMPK phosphorylation and melatonina production in rats. Nutrients, 9( 4), 1-18. doi:10.3390/nu9040332
NLM
Faria J de A, Araújo TMF de, Razolli DS, Ignácio-Souza LM, Souza DN, Bordin S, Anhê GF. Metabolic impact of light phase-restricted fructose consumption is linked to changes in hypothalamic AMPK phosphorylation and melatonina production in rats [Internet]. Nutrients. 2017 ; 9( 4): 1-18.[citado 2024 nov. 12 ] Available from: https://doi.org/10.3390/nu9040332
Vancouver
Faria J de A, Araújo TMF de, Razolli DS, Ignácio-Souza LM, Souza DN, Bordin S, Anhê GF. Metabolic impact of light phase-restricted fructose consumption is linked to changes in hypothalamic AMPK phosphorylation and melatonina production in rats [Internet]. Nutrients. 2017 ; 9( 4): 1-18.[citado 2024 nov. 12 ] Available from: https://doi.org/10.3390/nu9040332
Artigo de periodico
Inflammatory state of periaortic adipose tissue in mice under obesogenic dietary regimens (2016)
Masi, Laureane Nunes; Crisma, Amanda Rabello; Martins, Amanda Roque; Amaral, Cátia Lira do; Torres, Rosângela Pavan; Mancini-Filho, Jorge ; Hirabara, Sandro Massao; Curi, Rui
Source: Journal of Nutrition & Intermediary Metabolism. Unidades: FCF, ICB
Subjects: INFLAMAÇÃO, ENDOTÉLIO, CITOCINAS
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MASI, Laureane Nunes et al. Inflammatory state of periaortic adipose tissue in mice under obesogenic dietary regimens. Journal of Nutrition & Intermediary Metabolism, v. 6, p. 1-7, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jnim.2016.04.009. Acesso em: 12 nov. 2024.
APA
Masi, L. N., Crisma, A. R., Martins, A. R., Amaral, C. L. do, Torres, R. P., Mancini-Filho, J., et al. (2016). Inflammatory state of periaortic adipose tissue in mice under obesogenic dietary regimens. Journal of Nutrition & Intermediary Metabolism, 6, 1-7. doi:10.1016/j.jnim.2016.04.009
NLM
Masi LN, Crisma AR, Martins AR, Amaral CL do, Torres RP, Mancini-Filho J, Hirabara SM, Curi R. Inflammatory state of periaortic adipose tissue in mice under obesogenic dietary regimens [Internet]. Journal of Nutrition & Intermediary Metabolism. 2016 ; 6 1-7.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.jnim.2016.04.009
Vancouver
Masi LN, Crisma AR, Martins AR, Amaral CL do, Torres RP, Mancini-Filho J, Hirabara SM, Curi R. Inflammatory state of periaortic adipose tissue in mice under obesogenic dietary regimens [Internet]. Journal of Nutrition & Intermediary Metabolism. 2016 ; 6 1-7.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.jnim.2016.04.009
Artigo de periodico
Effect of topical insulin on second-degree burns in diabetic rats (2016)
Azevedo, F.; Pessoa, A.; Moreira, G.; Santos, Marinilce Fagundes dos ; Liberti, E.; Araujo, E.; Carvalho, Carla Roberta de Oliveira ; Saad, M.; Lima, M. H.
Source: Biological Research for Nursing. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
AZEVEDO, F. et al. Effect of topical insulin on second-degree burns in diabetic rats. Biological Research for Nursing, v. 18, n. 2, p. 181-192, 2016Tradução . . Disponível em: https://doi.org/10.1177/1099800415592175. Acesso em: 12 nov. 2024.
APA
Azevedo, F., Pessoa, A., Moreira, G., Santos, M. F. dos, Liberti, E., Araujo, E., et al. (2016). Effect of topical insulin on second-degree burns in diabetic rats. Biological Research for Nursing, 18( 2), 181-192. doi:10.1177/1099800415592175
NLM
Azevedo F, Pessoa A, Moreira G, Santos MF dos, Liberti E, Araujo E, Carvalho CR de O, Saad M, Lima MH. Effect of topical insulin on second-degree burns in diabetic rats [Internet]. Biological Research for Nursing. 2016 ; 18( 2): 181-192.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1177/1099800415592175
Vancouver
Azevedo F, Pessoa A, Moreira G, Santos MF dos, Liberti E, Araujo E, Carvalho CR de O, Saad M, Lima MH. Effect of topical insulin on second-degree burns in diabetic rats [Internet]. Biological Research for Nursing. 2016 ; 18( 2): 181-192.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1177/1099800415592175
Artigo de periodico
Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues (2016)
Victório, Jamaira A.; Fontes, Milene T.; Rossoni, Luciana Venturini ; Davel, Ana P.
Source: Frontiers in Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
VICTÓRIO, Jamaira A. et al. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, v. 7, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.3389/fphys.2016.00295. Acesso em: 12 nov. 2024.
APA
Victório, J. A., Fontes, M. T., Rossoni, L. V., & Davel, A. P. (2016). Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, 7, 1-10. doi:10.3389/fphys.2016.00295
NLM
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Vancouver
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Artigo de periodico
Gut bacteria products prevent AKI induced by ischemia-reperfusion (2015)
Andrade-Oliveira, Vinicius; Amano, Mariane Tami; Correa-Costa, Matheus; Castoldi, Angela; Felizardo, Raphael José Ferreira; Almeida, Danilo Candido de; Bassi, Enio José; Moraes-Vieira, Pedro Manoel; Hiyane, Meire Ioshie; Rodas, Andrea Cecilia Dorion; Peron, Jean Pierre Schatzmann ; Aguiar, Cristhiane Favero de; Reis, Marlene Antonia dos; Ribeiro, Willian R.; Valduga, Claudete Justina; Curi, Rui; Vinolo, Marco Aurelio Ramirez; Ferreira, Caroline Marcantonio; Câmara, Niels Olsen Saraiva
Source: Journal of the American Society of Nephrology. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, ISQUEMIA, INFLAMAÇÃO
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ABNT
ANDRADE-OLIVEIRA, Vinicius et al. Gut bacteria products prevent AKI induced by ischemia-reperfusion. Journal of the American Society of Nephrology, v. 26, n. 8, p. 1877-1888, 2015Tradução . . Disponível em: https://doi.org/10.1681/ASN.2014030288. Acesso em: 12 nov. 2024.
APA
Andrade-Oliveira, V., Amano, M. T., Correa-Costa, M., Castoldi, A., Felizardo, R. J. F., Almeida, D. C. de, et al. (2015). Gut bacteria products prevent AKI induced by ischemia-reperfusion. Journal of the American Society of Nephrology, 26( 8), 1877-1888. doi:10.1681/ASN.2014030288
NLM
Andrade-Oliveira V, Amano MT, Correa-Costa M, Castoldi A, Felizardo RJF, Almeida DC de, Bassi EJ, Moraes-Vieira PM, Hiyane MI, Rodas ACD, Peron JPS, Aguiar CF de, Reis MA dos, Ribeiro WR, Valduga CJ, Curi R, Vinolo MAR, Ferreira CM, Câmara NOS. Gut bacteria products prevent AKI induced by ischemia-reperfusion [Internet]. Journal of the American Society of Nephrology. 2015 ; 26( 8): 1877-1888.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1681/ASN.2014030288
Vancouver
Andrade-Oliveira V, Amano MT, Correa-Costa M, Castoldi A, Felizardo RJF, Almeida DC de, Bassi EJ, Moraes-Vieira PM, Hiyane MI, Rodas ACD, Peron JPS, Aguiar CF de, Reis MA dos, Ribeiro WR, Valduga CJ, Curi R, Vinolo MAR, Ferreira CM, Câmara NOS. Gut bacteria products prevent AKI induced by ischemia-reperfusion [Internet]. Journal of the American Society of Nephrology. 2015 ; 26( 8): 1877-1888.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1681/ASN.2014030288
Parte de monografia/livro
Funções biológicas dos ácidos agraxos (2015)
Gorjão, Renata (Organizador); Abdulkader, Fernando Rodrigues de Moraes ; Hirabara, Sandro Massao (Organizador); Sato, Fábio Takeo (colab); Vinolo, Marco Aurélio Ramirez (Organizador)
Source: Ácidos graxos na saúde e na doença: influência da genética, nutrição, exercício físico e esporte. Unidade: ICB
Assunto: ÁCIDOS GRAXOS
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ABNT
ABDULKADER, Fernando Rodrigues de Moraes. Funções biológicas dos ácidos agraxos. Ácidos graxos na saúde e na doença: influência da genética, nutrição, exercício físico e esporte. Tradução . Curitiba: Editora CRV, 2015. . . Acesso em: 12 nov. 2024.
APA
Abdulkader, F. R. de M. (2015). Funções biológicas dos ácidos agraxos. In Ácidos graxos na saúde e na doença: influência da genética, nutrição, exercício físico e esporte. Curitiba: Editora CRV.
NLM
Abdulkader FR de M. Funções biológicas dos ácidos agraxos. In: Ácidos graxos na saúde e na doença: influência da genética, nutrição, exercício físico e esporte. Curitiba: Editora CRV; 2015. [citado 2024 nov. 12 ]
Vancouver
Abdulkader FR de M. Funções biológicas dos ácidos agraxos. In: Ácidos graxos na saúde e na doença: influência da genética, nutrição, exercício físico e esporte. Curitiba: Editora CRV; 2015. [citado 2024 nov. 12 ]
Artigo de periodico
Production of vegetable oil blends and structured lipids and their effect on wound healing (2015)
Ract, Juliana Neves Rodrigues ; Soares, Fabiana Andréia Schafer de Martini; Rodrigues, Hosana Gomes; Bortolon, José Ricardo; Murata, Gilson Masahiro; Gonçalves, Maria Inês de Almeida; Hatanaka, Elaine; Curi, Rui; Gioielli, Luiz Antonio
Source: Brazilian Journal of Pharmaceutical Sciences. Unidades: FCF, ICB
Subjects: LIPÍDEOS, CICATRIZAÇÃO
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ABNT
RACT, Juliana Neves Rodrigues et al. Production of vegetable oil blends and structured lipids and their effect on wound healing. Brazilian Journal of Pharmaceutical Sciences, v. 51, n. 2, p. 415-427, 2015Tradução . . Disponível em: https://doi.org/10.1590/s1984-82502015000200019. Acesso em: 12 nov. 2024.
APA
Ract, J. N. R., Soares, F. A. S. de M., Rodrigues, H. G., Bortolon, J. R., Murata, G. M., Gonçalves, M. I. de A., et al. (2015). Production of vegetable oil blends and structured lipids and their effect on wound healing. Brazilian Journal of Pharmaceutical Sciences, 51( 2), 415-427. doi:10.1590/s1984-82502015000200019
NLM
Ract JNR, Soares FAS de M, Rodrigues HG, Bortolon JR, Murata GM, Gonçalves MI de A, Hatanaka E, Curi R, Gioielli LA. Production of vegetable oil blends and structured lipids and their effect on wound healing [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2015 ; 51( 2): 415-427.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1590/s1984-82502015000200019
Vancouver
Ract JNR, Soares FAS de M, Rodrigues HG, Bortolon JR, Murata GM, Gonçalves MI de A, Hatanaka E, Curi R, Gioielli LA. Production of vegetable oil blends and structured lipids and their effect on wound healing [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2015 ; 51( 2): 415-427.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1590/s1984-82502015000200019
Artigo de periodico
Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway (2015)
Davel, Ana P.; Victorio, Jamaira A.; Delbin, Maria Andréia; Fukuda, Livia Emy; Rossoni, Luciana Venturini
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ENDOTÉLIO, RATOS, ARTÉRIA PULMONAR, ADRENÉRGICOS
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ABNT
DAVEL, Ana P. et al. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, v. 125, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.01.018. Acesso em: 12 nov. 2024.
APA
Davel, A. P., Victorio, J. A., Delbin, M. A., Fukuda, L. E., & Rossoni, L. V. (2015). Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, 125, 49-56. doi:10.1016/j.lfs.2015.01.018
NLM
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Vancouver
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Artigo de periodico
Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling (2014)
Couto, Gisele Kruger; Davel, Ana P; Brum, Patricia Chakur; Rossoni, Luciana Venturini
Source: Experimental physiology. Unidades: ICB, EEFE
Subjects: FISIOLOGIA, SISTEMA CARDIOVASCULAR
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ABNT
COUTO, Gisele Kruger et al. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, v. 99, n. 10, p. 1427–1438, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2014.079236. Acesso em: 12 nov. 2024.
APA
Couto, G. K., Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, 99( 10), 1427–1438. doi:10.1113/expphysiol.2014.079236
NLM
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
Vancouver
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
Artigo de periodico
An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages (2014)
Moreira, Vanessa; Lomonte, Bruno; Vinolo, Marco Aurélio Ramirez; Curi, Rui; Gutiérrez, José María; Teixeira, Catarina
Source: Mediators of Inflammation. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MOREIRA, Vanessa et al. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/105879. Acesso em: 12 nov. 2024.
APA
Moreira, V., Lomonte, B., Vinolo, M. A. R., Curi, R., Gutiérrez, J. M., & Teixeira, C. (2014). An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, 2014, 1-10. doi:10.1155/2014/105879
NLM
Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1155/2014/105879
Vancouver
Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1155/2014/105879
Artigo de periodico
Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway (2014)
Davel, Ana P; Brum, Patricia Chakur; Rossoni, Luciana Venturini
Source: PLOS ONE. Unidades: EEFE, ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇAS CARDIOVASCULARES
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ABNT
DAVEL, Ana P e BRUM, Patricia Chakur e ROSSONI, Luciana Venturini. Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway. PLOS ONE, v. 9, n. 3, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0091877. Acesso em: 12 nov. 2024.
APA
Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway. PLOS ONE, 9( 3). doi:10.1371/journal.pone.0091877
NLM
Davel AP, Brum PC, Rossoni LV. Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway [Internet]. PLOS ONE. 2014 ; 9( 3):[citado 2024 nov. 12 ] Available from: https://doi.org/10.1371/journal.pone.0091877
Vancouver
Davel AP, Brum PC, Rossoni LV. Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway [Internet]. PLOS ONE. 2014 ; 9( 3):[citado 2024 nov. 12 ] Available from: https://doi.org/10.1371/journal.pone.0091877
Artigo de periodico
Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats (2014)
Rodrigues, Sandra C.; Pantaleão, Lucas Carminatti; Nogueira, Tatiane Cristina de Araujo; Gomes, Patrícia R.; Albuquerque, Gabriela G.; Nachbar, Renato Tadeu; Torres-Leal, Francisco Leonardo; Caperuto, Luciana Chagas; Lellis-Santos, Camilo; Anhê, Gabriel F0rato; Bordin, Silvana
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. Unidade: ICB
Subjects: FISIOLOGIA, METABOLISMO ANIMAL, PROTEINAS QUINASES, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Sandra C. et al. Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, v. 307, n. 9, p. R1146-R1156, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00513.2013. Acesso em: 12 nov. 2024.
APA
Rodrigues, S. C., Pantaleão, L. C., Nogueira, T. C. de A., Gomes, P. R., Albuquerque, G. G., Nachbar, R. T., et al. (2014). Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 307( 9), R1146-R1156. doi:10.1152/ajpregu.00513.2013
NLM
Rodrigues SC, Pantaleão LC, Nogueira TC de A, Gomes PR, Albuquerque GG, Nachbar RT, Torres-Leal FL, Caperuto LC, Lellis-Santos C, Anhê GF0rato, Bordin S. Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2014 ; 307( 9): R1146-R1156.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpregu.00513.2013
Vancouver
Rodrigues SC, Pantaleão LC, Nogueira TC de A, Gomes PR, Albuquerque GG, Nachbar RT, Torres-Leal FL, Caperuto LC, Lellis-Santos C, Anhê GF0rato, Bordin S. Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2014 ; 307( 9): R1146-R1156.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpregu.00513.2013
Artigo de periodico
Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle (2014)
Teodoro, Bruno G.; Baraldi, Flavia G; Sampaio, Igor H.; Bomfim, Lucas H. M.; Queiroz, André L.; Passos, Madla A.; Carneiro, Everardo M.; Alberici, Luciane Carla; Gomis, Ramon; Amaral, Fernanda G.; Cipolla-Neto, Jose; Araújo, Michel B.; Lima, Tanes; Uyemura, Sérgio Akira; Silveira, Leonardo dos Reis; Vieira, Elaine
Source: Journal of Pineal Research. Unidades: ICB, FCFRP, EEFERP
Assunto: FISIOLOGIA
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ABNT
TEODORO, Bruno G. et al. Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle. Journal of Pineal Research, v. 57, n. 2, p. 155-167, 2014Tradução . . Disponível em: https://doi.org/10.1111/jpi.12157. Acesso em: 12 nov. 2024.
APA
Teodoro, B. G., Baraldi, F. G., Sampaio, I. H., Bomfim, L. H. M., Queiroz, A. L., Passos, M. A., et al. (2014). Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle. Journal of Pineal Research, 57( 2), 155-167. doi:10.1111/jpi.12157
NLM
Teodoro BG, Baraldi FG, Sampaio IH, Bomfim LHM, Queiroz AL, Passos MA, Carneiro EM, Alberici LC, Gomis R, Amaral FG, Cipolla-Neto J, Araújo MB, Lima T, Uyemura SA, Silveira L dos R, Vieira E. Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle [Internet]. Journal of Pineal Research. 2014 ; 57( 2): 155-167.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/jpi.12157
Vancouver
Teodoro BG, Baraldi FG, Sampaio IH, Bomfim LHM, Queiroz AL, Passos MA, Carneiro EM, Alberici LC, Gomis R, Amaral FG, Cipolla-Neto J, Araújo MB, Lima T, Uyemura SA, Silveira L dos R, Vieira E. Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle [Internet]. Journal of Pineal Research. 2014 ; 57( 2): 155-167.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/jpi.12157
Artigo de periodico
Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats (2014)
Davel, Ana Paula; Couto, Gisele Kruger; Wenceslau, Camila Ferreira; Peres, Emilia Cristina; Xavier, Fabiano Elias; Rossoni, Luciana Venturini
Source: Hormone Molecular Biology and Clinical Investigation. Unidades: ICB, HU
Subjects: FISIOLOGIA, BIOFÍSICA, ENDOTÉLIO VASCULAR, RATOS, AORTA DE ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAVEL, Ana Paula et al. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats. Hormone Molecular Biology and Clinical Investigation, v. 18, n. 2, p. 113-122, 2014Tradução . . Disponível em: https://doi.org/10.1515/hmbci-2013-0058. Acesso em: 12 nov. 2024.
APA
Davel, A. P., Couto, G. K., Wenceslau, C. F., Peres, E. C., Xavier, F. E., & Rossoni, L. V. (2014). Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats. Hormone Molecular Biology and Clinical Investigation, 18( 2), 113-122. doi:10.1515/hmbci-2013-0058
NLM
Davel AP, Couto GK, Wenceslau CF, Peres EC, Xavier FE, Rossoni LV. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats [Internet]. Hormone Molecular Biology and Clinical Investigation. 2014 ; 18( 2): 113-122.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1515/hmbci-2013-0058
Vancouver
Davel AP, Couto GK, Wenceslau CF, Peres EC, Xavier FE, Rossoni LV. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats [Internet]. Hormone Molecular Biology and Clinical Investigation. 2014 ; 18( 2): 113-122.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1515/hmbci-2013-0058
Artigo de periodico
Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation (2014)
Gomes, Patrícia R.; Graciano, Maria Fernanda; Pantaleão, Lucas Carminatti; Rennó, André L.; Rodrigues, Sandra Campos; Velloso, Lício Augusto; Latorraca, Márcia Q.; Carpinelli, Angelo Rafael ; Anhê, Gabriel F.; Bordin, Silvana
Source: American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. Unidade: ICB
Subjects: FISIOLOGIA, GLICOSE, ESTERÓIDES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOMES, Patrícia R. et al. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology, v. 306, n. 1, p. E109-E120, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00364.2013. Acesso em: 12 nov. 2024.
APA
Gomes, P. R., Graciano, M. F., Pantaleão, L. C., Rennó, A. L., Rodrigues, S. C., Velloso, L. A., et al. (2014). Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology, 306( 1), E109-E120. doi:10.1152/ajpendo.00364.2013
NLM
Gomes PR, Graciano MF, Pantaleão LC, Rennó AL, Rodrigues SC, Velloso LA, Latorraca MQ, Carpinelli AR, Anhê GF, Bordin S. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation [Internet]. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. 2014 ; 306( 1): E109-E120.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpendo.00364.2013
Vancouver
Gomes PR, Graciano MF, Pantaleão LC, Rennó AL, Rodrigues SC, Velloso LA, Latorraca MQ, Carpinelli AR, Anhê GF, Bordin S. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation [Internet]. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. 2014 ; 306( 1): E109-E120.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpendo.00364.2013
Artigo de periodico
Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats (2013)
Faria, Juliana A.; Kinote, Andrezza; Ignacio-Souza, Letícia M.; Araújo, Thiago M. de; Razolli, Daniela S.; Doneda, Diego L.; Paschoal, Lívia B.; Lellis-Santos, Camilo; Bertolini, Gisele L.; Velloso, Lício A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: American Journal of Physiology, Endocrinology and Methabolism. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FARIA, Juliana A. et al. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, v. 305, n. 2, p. E230-E242, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00094.2013. Acesso em: 12 nov. 2024.
APA
Faria, J. A., Kinote, A., Ignacio-Souza, L. M., Araújo, T. M. de, Razolli, D. S., Doneda, D. L., et al. (2013). Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, 305( 2), E230-E242. doi:10.1152/ajpendo.00094.2013
NLM
Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
Vancouver
Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
Artigo de periodico
Protein malnutrition induces bone marrow mesenchymal stem cells commitment to adipogenic differentiation leading to hematopoietic failure (2013)
Cunha, Mayara Caldas Ramos; Lima, Fabiana da Silva ; Vinolo, Marco Aurélio Ramirez; Hastreiter, Araceli; Curi, Rui; Borelli, Primavera ; Fock, Ricardo Ambrósio
Source: PLOS ONE. Unidades: FCF, ICB, HU
Subjects: DESNUTRIÇÃO PROTEICO-ENERGÉTICA, INTERLEUCINA 3
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, Mayara Caldas Ramos et al. Protein malnutrition induces bone marrow mesenchymal stem cells commitment to adipogenic differentiation leading to hematopoietic failure. PLOS ONE, v. 8, n. 3, p. 1-12, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0058872. Acesso em: 12 nov. 2024.
APA
Cunha, M. C. R., Lima, F. da S., Vinolo, M. A. R., Hastreiter, A., Curi, R., Borelli, P., & Fock, R. A. (2013). Protein malnutrition induces bone marrow mesenchymal stem cells commitment to adipogenic differentiation leading to hematopoietic failure. PLOS ONE, 8( 3), 1-12. doi:10.1371/journal.pone.0058872
NLM
Cunha MCR, Lima F da S, Vinolo MAR, Hastreiter A, Curi R, Borelli P, Fock RA. Protein malnutrition induces bone marrow mesenchymal stem cells commitment to adipogenic differentiation leading to hematopoietic failure [Internet]. PLOS ONE. 2013 ; 8( 3): 1-12.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1371/journal.pone.0058872
Vancouver
Cunha MCR, Lima F da S, Vinolo MAR, Hastreiter A, Curi R, Borelli P, Fock RA. Protein malnutrition induces bone marrow mesenchymal stem cells commitment to adipogenic differentiation leading to hematopoietic failure [Internet]. PLOS ONE. 2013 ; 8( 3): 1-12.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1371/journal.pone.0058872
Artigo de periodico
Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro (2013)
Paschoal, Vivian Almeida; Vinolo, M. A. R.; Crisma, A. R.; Magdalon, Juliana; Curi, Rui
Source: Lipids. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PASCHOAL, Vivian Almeida et al. Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro. Lipids, v. 48, n. 2, p. 93-103, 2013Tradução . . Disponível em: https://doi.org/10.1007/s11745-012-3726-6. Acesso em: 12 nov. 2024.
APA
Paschoal, V. A., Vinolo, M. A. R., Crisma, A. R., Magdalon, J., & Curi, R. (2013). Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro. Lipids, 48( 2), 93-103. doi:10.1007/s11745-012-3726-6
NLM
Paschoal VA, Vinolo MAR, Crisma AR, Magdalon J, Curi R. Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro [Internet]. Lipids. 2013 ; 48( 2): 93-103.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s11745-012-3726-6
Vancouver
Paschoal VA, Vinolo MAR, Crisma AR, Magdalon J, Curi R. Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro [Internet]. Lipids. 2013 ; 48( 2): 93-103.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s11745-012-3726-6

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