Source: Clinics. Conference titles: Simpósio "Avanços em Pesquisas Médicas dos Laboratórios de Investigação Médica do Hospital das Clínicas da FMUSP". Unidade: FM
Subjects: APOPTOSE, PÂNCREAS (FISIOPATOLOGIA), EXPRESSÃO GÊNICA, FIBROBLASTOS, ANTIOXIDANTES (APLICAÇÕES TERAPÊUTICAS)
ABNT
COSTA, F S L et al. Dual effect of advanced glycation end products (AGES) in pancreatic isler apoptosis and the protective role of benfotiamine and mitoq. Clinics. São Paulo: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 17 ago. 2024. , 2010APA
Costa, F. S. L., Oliveira, E. R., Amaral, A. S. R., Lima, A. M. S., Passarelli, M., Giannella-Neto, D., & Giannella, M. L. C. C. (2010). Dual effect of advanced glycation end products (AGES) in pancreatic isler apoptosis and the protective role of benfotiamine and mitoq. Clinics. São Paulo: Faculdade de Medicina, Universidade de São Paulo.NLM
Costa FSL, Oliveira ER, Amaral ASR, Lima AMS, Passarelli M, Giannella-Neto D, Giannella MLCC. Dual effect of advanced glycation end products (AGES) in pancreatic isler apoptosis and the protective role of benfotiamine and mitoq. Clinics. 2010 ; 65 S38.[citado 2024 ago. 17 ]Vancouver
Costa FSL, Oliveira ER, Amaral ASR, Lima AMS, Passarelli M, Giannella-Neto D, Giannella MLCC. Dual effect of advanced glycation end products (AGES) in pancreatic isler apoptosis and the protective role of benfotiamine and mitoq. Clinics. 2010 ; 65 S38.[citado 2024 ago. 17 ]