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Artigo de periodico
Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells (2016)
Carregaro, Vanessa; Napimoga, Marcelo H; Peres, Raphael S; Benevides, Luciana; Sacramento, Laís Amorim; Pinto, Larissa G; Grespan, Renata; Cunha, Thiago Mattar; Silva, João Santana da; Cunha, Fernando de Queiroz
Fonte: Mediators of Inflammation. Unidade: FMRP
Assuntos: ARTRITE, PROSTAGLANDINAS, FATORES DE TRANSCRIÇÃO, RECEPTORES, INTERLEUCINAS
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ABNT
CARREGARO, Vanessa et al. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, v. 2016, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/9626427. Acesso em: 30 jun. 2024.
APA
Carregaro, V., Napimoga, M. H., Peres, R. S., Benevides, L., Sacramento, L. A., Pinto, L. G., et al. (2016). Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, 2016. doi:10.1155/2016/9626427
NLM
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2016/9626427
Vancouver
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2016/9626427
Artigo de periodico
Blockage of eosinopoiesis by IL-17A is prevented by cytokine and lipid mediators of allergic inflammation (2015)
Xavier-Elsas, Pedro; Luca, Bianca de; Queto, Túlio; Vieira, Bruno Marques; Masid-de-Brito, Daniela; Dahab, Elizabeth Chen; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Gaspar-Elsas, Maria Ignez
Fonte: Mediators of Inflammation. Unidade: FMRP
Assuntos: MEDULA ÓSSEA, NEUTRÓFILOS, CITOCINAS, LIPÍDEOS, INTERLEUCINAS
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ABNT
XAVIER-ELSAS, Pedro et al. Blockage of eosinopoiesis by IL-17A is prevented by cytokine and lipid mediators of allergic inflammation. Mediators of Inflammation, v. 2015, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/968932. Acesso em: 30 jun. 2024.
APA
Xavier-Elsas, P., Luca, B. de, Queto, T., Vieira, B. M., Masid-de-Brito, D., Dahab, E. C., et al. (2015). Blockage of eosinopoiesis by IL-17A is prevented by cytokine and lipid mediators of allergic inflammation. Mediators of Inflammation, 2015. doi:10.1155/2015/968932
NLM
Xavier-Elsas P, Luca B de, Queto T, Vieira BM, Masid-de-Brito D, Dahab EC, Alves Filho JCF, Cunha F de Q, Gaspar-Elsas MI. Blockage of eosinopoiesis by IL-17A is prevented by cytokine and lipid mediators of allergic inflammation [Internet]. Mediators of Inflammation. 2015 ; 2015[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2015/968932
Vancouver
Xavier-Elsas P, Luca B de, Queto T, Vieira BM, Masid-de-Brito D, Dahab EC, Alves Filho JCF, Cunha F de Q, Gaspar-Elsas MI. Blockage of eosinopoiesis by IL-17A is prevented by cytokine and lipid mediators of allergic inflammation [Internet]. Mediators of Inflammation. 2015 ; 2015[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2015/968932
Artigo de periodico
The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice (2014)
Canavaci, Adriana Monte Cassiano; Sorgi, Carlos Artério; Martins, Vicente de Paulo; Morais, Fabiana Rosseto; Sousa, Érika V. G. de; Trindade, Bruno Caetano; Cunha, Fernando de Queiróz; Rossi, Marcos Antonio; Aronoff, David M.; Faccioli, Lúcia Helena; Nomizo, Auro
Fonte: Mediators of Inflammation. Unidades: FCFRP, FMRP
Assuntos: TRYPANOSOMA CRUZI, INTERLEUCINAS, ANTIPARASITÁRIOS (FARMACOLOGIA)
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ABNT
CANAVACI, Adriana Monte Cassiano et al. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice. Mediators of Inflammation, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/893634. Acesso em: 30 jun. 2024.
APA
Canavaci, A. M. C., Sorgi, C. A., Martins, V. de P., Morais, F. R., Sousa, É. V. G. de, Trindade, B. C., et al. (2014). The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice. Mediators of Inflammation, 2014. doi:10.1155/2014/893634
NLM
Canavaci AMC, Sorgi CA, Martins V de P, Morais FR, Sousa ÉVG de, Trindade BC, Cunha F de Q, Rossi MA, Aronoff DM, Faccioli LH, Nomizo A. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2014/893634
Vancouver
Canavaci AMC, Sorgi CA, Martins V de P, Morais FR, Sousa ÉVG de, Trindade BC, Cunha F de Q, Rossi MA, Aronoff DM, Faccioli LH, Nomizo A. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2014/893634
Artigo de periodico
Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages (2014)
Saia, Rafael Simone; Mestriner, Fabiola Leslie Antunes Cardoso; Bertozi, Giuliana; Cunha, Fernando de Queiróz; Cárnio, Evelin Capellari
Fonte: Mediators of Inflammation. Unidades: FMRP, EERP
Assuntos: HORMÔNIOS DO TRATO GASTROINTESTINAL, MACRÓFAGOS, ÓXIDO NÍTRICO
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ABNT
SAIA, Rafael Simone et al. Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages. Mediators of Inflammation, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/896029. Acesso em: 30 jun. 2024.
APA
Saia, R. S., Mestriner, F. L. A. C., Bertozi, G., Cunha, F. de Q., & Cárnio, E. C. (2014). Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages. Mediators of Inflammation, 2014. doi:10.1155/2014/896029
NLM
Saia RS, Mestriner FLAC, Bertozi G, Cunha F de Q, Cárnio EC. Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2014/896029
Vancouver
Saia RS, Mestriner FLAC, Bertozi G, Cunha F de Q, Cárnio EC. Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/2014/896029
Artigo de periodico
Macrophage -derived neutrophil chemotactic factor is involved in the neutrophil recruitment inhibitory activity present in the supernatants of lp-stimulated macrophages (1996)
Tavares, M B M; Cunha, Fernando de Queiroz; Dias-Baruffi, M; Barreira, M C R; Ferreira, S. H.
Fonte: Mediators of Inflammation. Unidades: FMRP, FCFRP
Assunto: FARMACOLOGIA MOLECULAR
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ABNT
TAVARES, M B M et al. Macrophage -derived neutrophil chemotactic factor is involved in the neutrophil recruitment inhibitory activity present in the supernatants of lp-stimulated macrophages. Mediators of Inflammation, v. 5 , p. 116-20, 1996Tradução . . Disponível em: https://doi.org/10.1155/s0962935196000208. Acesso em: 30 jun. 2024.
APA
Tavares, M. B. M., Cunha, F. de Q., Dias-Baruffi, M., Barreira, M. C. R., & Ferreira, S. H. (1996). Macrophage -derived neutrophil chemotactic factor is involved in the neutrophil recruitment inhibitory activity present in the supernatants of lp-stimulated macrophages. Mediators of Inflammation, 5 , 116-20. doi:10.1155/s0962935196000208
NLM
Tavares MBM, Cunha F de Q, Dias-Baruffi M, Barreira MCR, Ferreira SH. Macrophage -derived neutrophil chemotactic factor is involved in the neutrophil recruitment inhibitory activity present in the supernatants of lp-stimulated macrophages [Internet]. Mediators of Inflammation. 1996 ;5 116-20.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s0962935196000208
Vancouver
Tavares MBM, Cunha F de Q, Dias-Baruffi M, Barreira MCR, Ferreira SH. Macrophage -derived neutrophil chemotactic factor is involved in the neutrophil recruitment inhibitory activity present in the supernatants of lp-stimulated macrophages [Internet]. Mediators of Inflammation. 1996 ;5 116-20.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s0962935196000208
Artigo de periodico
Isolation and partial chemical characterization of macrophage derived neutrophil chemotactive factor (1995)
Baruffi, M D; Barreira, M C R A; Cunha, Fernando de Queiroz; Ferreira, S. H.
Fonte: Mediators of Inflammation. Unidade: FMRP
Assuntos: IMUNOLOGIA (FARMACOLOGIA), IMUNOLOGIA
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ABNT
BARUFFI, M D et al. Isolation and partial chemical characterization of macrophage derived neutrophil chemotactive factor. Mediators of Inflammation, v. 4 , p. 257-62, 1995Tradução . . Disponível em: https://doi.org/10.1155/s096293519500041x. Acesso em: 30 jun. 2024.
APA
Baruffi, M. D., Barreira, M. C. R. A., Cunha, F. de Q., & Ferreira, S. H. (1995). Isolation and partial chemical characterization of macrophage derived neutrophil chemotactive factor. Mediators of Inflammation, 4 , 257-62. doi:10.1155/s096293519500041x
NLM
Baruffi MD, Barreira MCRA, Cunha F de Q, Ferreira SH. Isolation and partial chemical characterization of macrophage derived neutrophil chemotactive factor [Internet]. Mediators of Inflammation. 1995 ;4 257-62.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s096293519500041x
Vancouver
Baruffi MD, Barreira MCRA, Cunha F de Q, Ferreira SH. Isolation and partial chemical characterization of macrophage derived neutrophil chemotactive factor [Internet]. Mediators of Inflammation. 1995 ;4 257-62.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s096293519500041x
Artigo de periodico
Biological characterization of purified macrophage derived neutrophil chemotactic factor (1995)
Baruffi, M D; Barreira, M C R A; Cunha, Fernando de Queiroz; Ferreira, S. H.
Fonte: Mediators of Inflammation. Unidade: FMRP
Assunto: IMUNOLOGIA (FARMACOLOGIA)
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ABNT
BARUFFI, M D et al. Biological characterization of purified macrophage derived neutrophil chemotactic factor. Mediators of Inflammation, v. 4 , p. 263-9, 1995Tradução . . Disponível em: https://doi.org/10.1155/s0962935195000421. Acesso em: 30 jun. 2024.
APA
Baruffi, M. D., Barreira, M. C. R. A., Cunha, F. de Q., & Ferreira, S. H. (1995). Biological characterization of purified macrophage derived neutrophil chemotactic factor. Mediators of Inflammation, 4 , 263-9. doi:10.1155/s0962935195000421
NLM
Baruffi MD, Barreira MCRA, Cunha F de Q, Ferreira SH. Biological characterization of purified macrophage derived neutrophil chemotactic factor [Internet]. Mediators of Inflammation. 1995 ;4 263-9.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s0962935195000421
Vancouver
Baruffi MD, Barreira MCRA, Cunha F de Q, Ferreira SH. Biological characterization of purified macrophage derived neutrophil chemotactic factor [Internet]. Mediators of Inflammation. 1995 ;4 263-9.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s0962935195000421
Artigo de periodico
In vivo monuclear phagocyte migration: paradoxical effect of adrenalectomy (1994)
Heluy Neto, N E; Cunha, Fernando de Queiroz; Ferreira, S. H.
Fonte: Mediators of Inflammation. Unidade: FMRP
Assuntos: FARMACOLOGIA, INFLAMAÇÃO (PATOLOGIA)
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ABNT
HELUY NETO, N E e CUNHA, Fernando de Queiroz e FERREIRA, S. H. In vivo monuclear phagocyte migration: paradoxical effect of adrenalectomy. Mediators of Inflammation, v. 3 , p. 52-79, 1994Tradução . . Acesso em: 30 jun. 2024.
APA
Heluy Neto, N. E., Cunha, F. de Q., & Ferreira, S. H. (1994). In vivo monuclear phagocyte migration: paradoxical effect of adrenalectomy. Mediators of Inflammation, 3 , 52-79.
NLM
Heluy Neto NE, Cunha F de Q, Ferreira SH. In vivo monuclear phagocyte migration: paradoxical effect of adrenalectomy. Mediators of Inflammation. 1994 ;3 52-79.[citado 2024 jun. 30 ]
Vancouver
Heluy Neto NE, Cunha F de Q, Ferreira SH. In vivo monuclear phagocyte migration: paradoxical effect of adrenalectomy. Mediators of Inflammation. 1994 ;3 52-79.[citado 2024 jun. 30 ]
Artigo de periodico
Neutrophil recruitment inhibitory factor: a possible candidate for a novel cytokine (1992)
Tamashiro, W M C; Tavares-Murta, B M; Cunha, Fernando de Queiroz; Barreira, M C R A; Nogueira, R M D; Ferreira, S. H.
Fonte: Mediators of Inflammation. Unidade: FMRP
Assunto: INFLAMAÇÃO (IMUNOLOGIA)
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ABNT
TAMASHIRO, W M C et al. Neutrophil recruitment inhibitory factor: a possible candidate for a novel cytokine. Mediators of Inflammation, v. 1 , p. 49-54, 1992Tradução . . Disponível em: https://doi.org/10.1155/s0962935192000103. Acesso em: 30 jun. 2024.
APA
Tamashiro, W. M. C., Tavares-Murta, B. M., Cunha, F. de Q., Barreira, M. C. R. A., Nogueira, R. M. D., & Ferreira, S. H. (1992). Neutrophil recruitment inhibitory factor: a possible candidate for a novel cytokine. Mediators of Inflammation, 1 , 49-54. doi:10.1155/s0962935192000103
NLM
Tamashiro WMC, Tavares-Murta BM, Cunha F de Q, Barreira MCRA, Nogueira RMD, Ferreira SH. Neutrophil recruitment inhibitory factor: a possible candidate for a novel cytokine [Internet]. Mediators of Inflammation. 1992 ;1 49-54.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s0962935192000103
Vancouver
Tamashiro WMC, Tavares-Murta BM, Cunha F de Q, Barreira MCRA, Nogueira RMD, Ferreira SH. Neutrophil recruitment inhibitory factor: a possible candidate for a novel cytokine [Internet]. Mediators of Inflammation. 1992 ;1 49-54.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1155/s0962935192000103

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