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Artigo de periodico
PARP‑1 inhibition sensitizes temozolomide‑treated glioblastoma cell lines and decreases drug resistance independent of MGMT activity and PTEN proficiency (2020)
Montaldi, Ana P.; Lima, Sarah Caroline Gomes de ; Godoy, Paulo Roberto D'Auria Vieira; Xavier, Danilo Jordão; Sakamoto-Hojo, Elza Tiemi
Source: Oncology Reports. Unidades: FFCLRP, FMRP
Subjects: ANTINEOPLÁSICOS, FARMACOLOGIA, APOPTOSE, NEOPLASIAS CEREBRAIS, PATOLOGIA, GLIOMA, METABOLISMO, PROTEÍNAS
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ABNT
MONTALDI, Ana P. et al. PARP‑1 inhibition sensitizes temozolomide‑treated glioblastoma cell lines and decreases drug resistance independent of MGMT activity and PTEN proficiency. Oncology Reports, v. 44, n. 5, p. 2275-2287, 2020Tradução . . Disponível em: https://doi.org/10.3892/or.2020.7756. Acesso em: 13 jun. 2024.
APA
Montaldi, A. P., Lima, S. C. G. de, Godoy, P. R. D. 'A. V., Xavier, D. J., & Sakamoto-Hojo, E. T. (2020). PARP‑1 inhibition sensitizes temozolomide‑treated glioblastoma cell lines and decreases drug resistance independent of MGMT activity and PTEN proficiency. Oncology Reports, 44( 5), 2275-2287. doi:10.3892/or.2020.7756
NLM
Montaldi AP, Lima SCG de, Godoy PRD'AV, Xavier DJ, Sakamoto-Hojo ET. PARP‑1 inhibition sensitizes temozolomide‑treated glioblastoma cell lines and decreases drug resistance independent of MGMT activity and PTEN proficiency [Internet]. Oncology Reports. 2020 ; 44( 5): 2275-2287.[citado 2024 jun. 13 ] Available from: https://doi.org/10.3892/or.2020.7756
Vancouver
Montaldi AP, Lima SCG de, Godoy PRD'AV, Xavier DJ, Sakamoto-Hojo ET. PARP‑1 inhibition sensitizes temozolomide‑treated glioblastoma cell lines and decreases drug resistance independent of MGMT activity and PTEN proficiency [Internet]. Oncology Reports. 2020 ; 44( 5): 2275-2287.[citado 2024 jun. 13 ] Available from: https://doi.org/10.3892/or.2020.7756
Artigo de periodico
HEB silencing induces anti-proliferative effects on U87MG cells cultured as neurospheres and monolayers (2016)
Godoy, Paulo R. D. V.; Montaldi, Ana Paula L.; Sakamoto-Hojo, Elza Tiemi
Source: Molecular Medicine Reports. Unidade: FFCLRP
Subjects: ASTROCITOMA, FATORES DE TRANSCRIÇÃO, CÉLULAS-TRONCO, CITOMETRIA DE FLUXO, IMUNOHISTOQUÍMICA
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ABNT
GODOY, Paulo R. D. V. e MONTALDI, Ana Paula L. e SAKAMOTO-HOJO, Elza Tiemi. HEB silencing induces anti-proliferative effects on U87MG cells cultured as neurospheres and monolayers. Molecular Medicine Reports, v. 14, p. 5253-5260, 2016Tradução . . Disponível em: https://doi.org/10.3892/mmr.2016.5877. Acesso em: 13 jun. 2024.
APA
Godoy, P. R. D. V., Montaldi, A. P. L., & Sakamoto-Hojo, E. T. (2016). HEB silencing induces anti-proliferative effects on U87MG cells cultured as neurospheres and monolayers. Molecular Medicine Reports, 14, 5253-5260. doi:10.3892/mmr.2016.5877
NLM
Godoy PRDV, Montaldi APL, Sakamoto-Hojo ET. HEB silencing induces anti-proliferative effects on U87MG cells cultured as neurospheres and monolayers [Internet]. Molecular Medicine Reports. 2016 ; 14 5253-5260.[citado 2024 jun. 13 ] Available from: https://doi.org/10.3892/mmr.2016.5877
Vancouver
Godoy PRDV, Montaldi APL, Sakamoto-Hojo ET. HEB silencing induces anti-proliferative effects on U87MG cells cultured as neurospheres and monolayers [Internet]. Molecular Medicine Reports. 2016 ; 14 5253-5260.[citado 2024 jun. 13 ] Available from: https://doi.org/10.3892/mmr.2016.5877
Trabalho de evento-resumo periodico
Alterations in gene expression, cell cycle control and apoptosis induced by ionizing radiation in fibroblast cell lines (2002)
Sakamoto-Hojo, Elza Tiemi; Mello, Stephano S; Cardoso, Renato S; Savoldi Barbosa, Marcela; Passos, Geraldo Aleixo Silva
Source: International Journal of Molecular Medicine. Conference titles: World Congress on Advances in Oncology. Unidades: FFCLRP, FORP
Subjects: DOENÇAS HEMATOLÓGICAS (TRATAMENTO), CROMOSSOMOS (ALTERAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SAKAMOTO-HOJO, Elza Tiemi et al. Alterations in gene expression, cell cycle control and apoptosis induced by ionizing radiation in fibroblast cell lines. International Journal of Molecular Medicine. Athens: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 13 jun. 2024. , 2002
APA
Sakamoto-Hojo, E. T., Mello, S. S., Cardoso, R. S., Savoldi Barbosa, M., & Passos, G. A. S. (2002). Alterations in gene expression, cell cycle control and apoptosis induced by ionizing radiation in fibroblast cell lines. International Journal of Molecular Medicine. Athens: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo.
NLM
Sakamoto-Hojo ET, Mello SS, Cardoso RS, Savoldi Barbosa M, Passos GAS. Alterations in gene expression, cell cycle control and apoptosis induced by ionizing radiation in fibroblast cell lines. International Journal of Molecular Medicine. 2002 ; 10 S58.[citado 2024 jun. 13 ]
Vancouver
Sakamoto-Hojo ET, Mello SS, Cardoso RS, Savoldi Barbosa M, Passos GAS. Alterations in gene expression, cell cycle control and apoptosis induced by ionizing radiation in fibroblast cell lines. International Journal of Molecular Medicine. 2002 ; 10 S58.[citado 2024 jun. 13 ]
Trabalho de evento-resumo periodico
Transformed AT (ataxia telangiectasia) fibroblasts are capable to form TCR (T-cell receptor) 'beta'/'gama' rearrangements under control of P53 gene (2002)
D'Arce, Luciana P G; Mello, Stephano S; Fachin, Ana L; Bassi, Carmen L; Passos, Geraldo Aleixo Silva; Sakamoto-Hojo, Elza Tiemi
Source: International Journal of Molecular Medicine. Conference titles: World Congress on Advances in Oncology. Unidades: FFCLRP, FORP
Subjects: DOENÇAS HEMATOLÓGICAS (TRATAMENTO), CROMOSSOMOS (ALTERAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
D'ARCE, Luciana P G et al. Transformed AT (ataxia telangiectasia) fibroblasts are capable to form TCR (T-cell receptor) 'beta'/'gama' rearrangements under control of P53 gene. International Journal of Molecular Medicine. Athens: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 13 jun. 2024. , 2002
APA
D'Arce, L. P. G., Mello, S. S., Fachin, A. L., Bassi, C. L., Passos, G. A. S., & Sakamoto-Hojo, E. T. (2002). Transformed AT (ataxia telangiectasia) fibroblasts are capable to form TCR (T-cell receptor) 'beta'/'gama' rearrangements under control of P53 gene. International Journal of Molecular Medicine. Athens: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo.
NLM
D'Arce LPG, Mello SS, Fachin AL, Bassi CL, Passos GAS, Sakamoto-Hojo ET. Transformed AT (ataxia telangiectasia) fibroblasts are capable to form TCR (T-cell receptor) 'beta'/'gama' rearrangements under control of P53 gene. International Journal of Molecular Medicine. 2002 ; 10 S58.[citado 2024 jun. 13 ]
Vancouver
D'Arce LPG, Mello SS, Fachin AL, Bassi CL, Passos GAS, Sakamoto-Hojo ET. Transformed AT (ataxia telangiectasia) fibroblasts are capable to form TCR (T-cell receptor) 'beta'/'gama' rearrangements under control of P53 gene. International Journal of Molecular Medicine. 2002 ; 10 S58.[citado 2024 jun. 13 ]
Trabalho de evento-resumo periodico
Analysis of TEL/AML1 fusions and alterations on chromossome 12 to evaluate the late effects of cancer therapy in leukemia (ALL) cured patients (2002)
Brassesco, M S; Camparoto, M L; Tone, Luiz Gonzaga; Sakamoto-Hojo, Elza Tiemi
Source: International Journal of Molecular Medicine. Conference titles: World Congress on Advances in Oncology. Unidades: FFCLRP, FMRP
Subjects: DOENÇAS HEMATOLÓGICAS (TRATAMENTO), CROMOSSOMOS (ALTERAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRASSESCO, M S et al. Analysis of TEL/AML1 fusions and alterations on chromossome 12 to evaluate the late effects of cancer therapy in leukemia (ALL) cured patients. International Journal of Molecular Medicine. Athens: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 13 jun. 2024. , 2002
APA
Brassesco, M. S., Camparoto, M. L., Tone, L. G., & Sakamoto-Hojo, E. T. (2002). Analysis of TEL/AML1 fusions and alterations on chromossome 12 to evaluate the late effects of cancer therapy in leukemia (ALL) cured patients. International Journal of Molecular Medicine. Athens: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo.
NLM
Brassesco MS, Camparoto ML, Tone LG, Sakamoto-Hojo ET. Analysis of TEL/AML1 fusions and alterations on chromossome 12 to evaluate the late effects of cancer therapy in leukemia (ALL) cured patients. International Journal of Molecular Medicine. 2002 ; 10 S58.[citado 2024 jun. 13 ]
Vancouver
Brassesco MS, Camparoto ML, Tone LG, Sakamoto-Hojo ET. Analysis of TEL/AML1 fusions and alterations on chromossome 12 to evaluate the late effects of cancer therapy in leukemia (ALL) cured patients. International Journal of Molecular Medicine. 2002 ; 10 S58.[citado 2024 jun. 13 ]
Trabalho de evento
Potentiation clastogenic action ellipticine by dna repair inhibitors caffeine and ara-c (1989)
Sakamoto-Hojo, Elza Tiemi; Takahashi, C S
Source: Annals. Conference titles: Annual Meeting of Eems on Environmental Mutagens - Carcinogens. Unidade: FFCLRP
Assunto: BIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SAKAMOTO-HOJO, Elza Tiemi e TAKAHASHI, C S. Potentiation clastogenic action ellipticine by dna repair inhibitors caffeine and ara-c. 1989, Anais.. Rhodes: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo, 1989. . Acesso em: 13 jun. 2024.
APA
Sakamoto-Hojo, E. T., & Takahashi, C. S. (1989). Potentiation clastogenic action ellipticine by dna repair inhibitors caffeine and ara-c. In Annals. Rhodes: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo.
NLM
Sakamoto-Hojo ET, Takahashi CS. Potentiation clastogenic action ellipticine by dna repair inhibitors caffeine and ara-c. Annals. 1989 ;[citado 2024 jun. 13 ]
Vancouver
Sakamoto-Hojo ET, Takahashi CS. Potentiation clastogenic action ellipticine by dna repair inhibitors caffeine and ara-c. Annals. 1989 ;[citado 2024 jun. 13 ]

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