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Artigo de periodico
DDAH1 and DDAH2 polymorphisms associate with asymmetrical dimethylarginine plasma levels in erectile dysfunction patients but not in healthy controls (2019)
Brites-Anselmi, Guilhermo; Azevedo, Ana Maria Milanez; Miyazaki, Anderson Heiji Lima ; Pinheiro, Lucas Cezar ; Coeli-Lacchini, Fernanda Borchers; Andrade, Murilo Ferreira de; Molina, Carlos Augusto Fernandes ; Tucci Júnior, Silvio; Hirsch, Emilio; Tanus-Santos, José Eduardo ; Lacchini, Riccardo
Fonte: Nitric Oxide. Unidades: EERP, FMRP
Assuntos: ÓXIDO NÍTRICO, DISFUNÇÃO ERÉTIL, POLIMORFISMO (GENÉTICA)
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ABNT
BRITES-ANSELMI, Guilhermo et al. DDAH1 and DDAH2 polymorphisms associate with asymmetrical dimethylarginine plasma levels in erectile dysfunction patients but not in healthy controls. Nitric Oxide, v. 92, n. 1, p. 11-17, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.08.001. Acesso em: 02 nov. 2024.
APA
Brites-Anselmi, G., Azevedo, A. M. M., Miyazaki, A. H. L., Pinheiro, L. C., Coeli-Lacchini, F. B., Andrade, M. F. de, et al. (2019). DDAH1 and DDAH2 polymorphisms associate with asymmetrical dimethylarginine plasma levels in erectile dysfunction patients but not in healthy controls. Nitric Oxide, 92( 1), 11-17. doi:10.1016/j.niox.2019.08.001
NLM
Brites-Anselmi G, Azevedo AMM, Miyazaki AHL, Pinheiro LC, Coeli-Lacchini FB, Andrade MF de, Molina CAF, Tucci Júnior S, Hirsch E, Tanus-Santos JE, Lacchini R. DDAH1 and DDAH2 polymorphisms associate with asymmetrical dimethylarginine plasma levels in erectile dysfunction patients but not in healthy controls [Internet]. Nitric Oxide. 2019 ; 92( 1): 11-17.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.niox.2019.08.001
Vancouver
Brites-Anselmi G, Azevedo AMM, Miyazaki AHL, Pinheiro LC, Coeli-Lacchini FB, Andrade MF de, Molina CAF, Tucci Júnior S, Hirsch E, Tanus-Santos JE, Lacchini R. DDAH1 and DDAH2 polymorphisms associate with asymmetrical dimethylarginine plasma levels in erectile dysfunction patients but not in healthy controls [Internet]. Nitric Oxide. 2019 ; 92( 1): 11-17.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.niox.2019.08.001
Artigo de periodico
Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder (2019)
Vale, Gabriel Tavares do; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Oliveira, Mariana G. de; Justo, Alberto F. O; Alexandre, Eduardo C.; Tanus-Santos, José Eduardo ; Antunes, Edson; Tirapelli, Carlos Renato
Fonte: Canadian Journal of Physiology and Pharmacology. Unidades: FMRP, EERP
Assuntos: ETANOL, ESTRESSE OXIDATIVO, BEXIGA
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ABNT
VALE, Gabriel Tavares do et al. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, v. 97, n. 12, p. 1103-1114, 2019Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2019-0143. Acesso em: 02 nov. 2024.
APA
Vale, G. T. do, Sousa, A. H. de, Gonzaga, N. de A., Oliveira, M. G. de, Justo, A. F. O., Alexandre, E. C., et al. (2019). Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, 97( 12), 1103-1114. doi:10.1139/cjpp-2019-0143
NLM
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Vancouver
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Fonte: European Journal of Pharmacology. Unidades: FMRP, EERP
Assuntos: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 02 nov. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms (2019)
Nakashima, Marcelo A.; Silva, Carla Beatriz Pereira da ; Gonzaga, Natália de Almeida; Simplicio, Janaina A.; Omoto, Ana Carolina Mieko ; Tirapelli, Luis Fernando; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Fonte: Cytokine. Unidades: FMRP, EERP
Assuntos: ESTRESSE OXIDATIVO, ETANOL, CITOTOXINAS, SEPSE, INTERLEUCINAS
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ABNT
NAKASHIMA, Marcelo A. et al. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, v. 121, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2019.154734. Acesso em: 02 nov. 2024.
APA
Nakashima, M. A., Silva, C. B. P. da, Gonzaga, N. de A., Simplicio, J. A., Omoto, A. C. M., Tirapelli, L. F., et al. (2019). Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, 121. doi:10.1016/j.cyto.2019.154734
NLM
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Vancouver
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Artigo de periodico
Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects (2019)
Rizzi, Elen; Amaral, Jefferson Henrich; Guimarães, Danielle Aparecida; Conde-Tella, Sandra de Oliveira; Pinheiro, Lucas Cézar ; Gerlach, Raquel Fernanda; Castro, Michele Mazzaron de; Tanus-Santos, José Eduardo
Fonte: Free Radical Biology and Medicine. Unidades: FMRP, EERP, FORP
Assuntos: NITRITOS, ESTRESSE OXIDATIVO, HIPERTENSÃO
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ABNT
RIZZI, Elen et al. Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects. Free Radical Biology and Medicine, v. 130, p. 234-243, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.11.002. Acesso em: 02 nov. 2024.
APA
Rizzi, E., Amaral, J. H., Guimarães, D. A., Conde-Tella, S. de O., Pinheiro, L. C., Gerlach, R. F., et al. (2019). Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects. Free Radical Biology and Medicine, 130, 234-243. doi:10.1016/j.freeradbiomed.2018.11.002
NLM
Rizzi E, Amaral JH, Guimarães DA, Conde-Tella S de O, Pinheiro LC, Gerlach RF, Castro MM de, Tanus-Santos JE. Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects [Internet]. Free Radical Biology and Medicine. 2019 ; 130 234-243.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.11.002
Vancouver
Rizzi E, Amaral JH, Guimarães DA, Conde-Tella S de O, Pinheiro LC, Gerlach RF, Castro MM de, Tanus-Santos JE. Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects [Internet]. Free Radical Biology and Medicine. 2019 ; 130 234-243.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.11.002
Artigo de periodico
Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway (2019)
Amaral, Jefferson Henrich; Rizzi, Elen S.; Alves-Lopes, Rhéure; Pinheiro, Lucas Cézar ; Tostes, Rita de Cássia Aleixo ; Tanus-Santos, José Eduardo
Fonte: Free Radical Biology and Medicine. Unidades: EERP, FMRP
Assuntos: ANTIOXIDANTES, NITRITOS, HIPERTENSÃO
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ABNT
AMARAL, Jefferson Henrich et al. Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway. Free Radical Biology and Medicine, v. 141, p. 261-268, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2019.06.028. Acesso em: 02 nov. 2024.
APA
Amaral, J. H., Rizzi, E. S., Alves-Lopes, R., Pinheiro, L. C., Tostes, R. de C. A., & Tanus-Santos, J. E. (2019). Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway. Free Radical Biology and Medicine, 141, 261-268. doi:10.1016/j.freeradbiomed.2019.06.028
NLM
Amaral JH, Rizzi ES, Alves-Lopes R, Pinheiro LC, Tostes R de CA, Tanus-Santos JE. Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway [Internet]. Free Radical Biology and Medicine. 2019 ; 141 261-268.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.06.028
Vancouver
Amaral JH, Rizzi ES, Alves-Lopes R, Pinheiro LC, Tostes R de CA, Tanus-Santos JE. Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway [Internet]. Free Radical Biology and Medicine. 2019 ; 141 261-268.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.06.028
Artigo de periodico
Sodium nitrite improves hypertension-induced myocardial dysfunction by mechanisms involving cardiac S-nitrosylation (2019)
Neto-Neves, Evandro M.; Pinheiro, Lucas Cézar ; Nogueira, Renato Corrêa; Portella, Rafael L.; Batista, Rose Inês Matos ; Tanus-Santos, José Eduardo
Fonte: Journal of Molecular and Cellular Cardiology. Unidades: EERP, FMRP
Assunto: NITRITOS
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ABNT
NETO-NEVES, Evandro M. et al. Sodium nitrite improves hypertension-induced myocardial dysfunction by mechanisms involving cardiac S-nitrosylation. Journal of Molecular and Cellular Cardiology, v. 134, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.yjmcc.2019.06.012. Acesso em: 02 nov. 2024.
APA
Neto-Neves, E. M., Pinheiro, L. C., Nogueira, R. C., Portella, R. L., Batista, R. I. M., & Tanus-Santos, J. E. (2019). Sodium nitrite improves hypertension-induced myocardial dysfunction by mechanisms involving cardiac S-nitrosylation. Journal of Molecular and Cellular Cardiology, 134. doi:10.1016/j.yjmcc.2019.06.012
NLM
Neto-Neves EM, Pinheiro LC, Nogueira RC, Portella RL, Batista RIM, Tanus-Santos JE. Sodium nitrite improves hypertension-induced myocardial dysfunction by mechanisms involving cardiac S-nitrosylation [Internet]. Journal of Molecular and Cellular Cardiology. 2019 ; 134[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.yjmcc.2019.06.012
Vancouver
Neto-Neves EM, Pinheiro LC, Nogueira RC, Portella RL, Batista RIM, Tanus-Santos JE. Sodium nitrite improves hypertension-induced myocardial dysfunction by mechanisms involving cardiac S-nitrosylation [Internet]. Journal of Molecular and Cellular Cardiology. 2019 ; 134[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.yjmcc.2019.06.012
Artigo de periodico
Mechanisms impairing blood pressure responses to nitrite and nitrate (2019)
Oliveira-Paula, Gustavo Henrique; Pinheiro, Lucas Cézar ; Tanus-Santos, José Eduardo
Fonte: Nitric Oxide. Unidades: FMRP, EERP
Assuntos: HIPERTENSÃO, ÓXIDO NÍTRICO, NITRATOS, NITRITOS
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ABNT
OLIVEIRA-PAULA, Gustavo Henrique e PINHEIRO, Lucas Cézar e TANUS-SANTOS, José Eduardo. Mechanisms impairing blood pressure responses to nitrite and nitrate. Nitric Oxide, v. 85, p. 35-43, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.01.015. Acesso em: 02 nov. 2024.
APA
Oliveira-Paula, G. H., Pinheiro, L. C., & Tanus-Santos, J. E. (2019). Mechanisms impairing blood pressure responses to nitrite and nitrate. Nitric Oxide, 85, 35-43. doi:10.1016/j.niox.2019.01.015
NLM
Oliveira-Paula GH, Pinheiro LC, Tanus-Santos JE. Mechanisms impairing blood pressure responses to nitrite and nitrate [Internet]. Nitric Oxide. 2019 ; 85 35-43.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.niox.2019.01.015
Vancouver
Oliveira-Paula GH, Pinheiro LC, Tanus-Santos JE. Mechanisms impairing blood pressure responses to nitrite and nitrate [Internet]. Nitric Oxide. 2019 ; 85 35-43.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.niox.2019.01.015
Artigo de periodico
Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin (2019)
Gonzaga, Natália de Almeida; Awata, Wanessa Mayumi Carvalho; Tanus-Santos, José Eduardo ; Padovan, Júlio C.; Tirapelli, Carlos Renato
Fonte: European Journal of Pharmacology. Unidades: FMRP, EERP
Assuntos: AORTA, ETANOL (REMOÇÃO)
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ABNT
GONZAGA, Natália de Almeida et al. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin. European Journal of Pharmacology, v. 847, p. 103-112, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.01.055. Acesso em: 02 nov. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Tanus-Santos, J. E., Padovan, J. C., & Tirapelli, C. R. (2019). Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin. European Journal of Pharmacology, 847, 103-112. doi:10.1016/j.ejphar.2019.01.055
NLM
Gonzaga N de A, Awata WMC, Tanus-Santos JE, Padovan JC, Tirapelli CR. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin [Internet]. European Journal of Pharmacology. 2019 ; 847 103-112.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2019.01.055
Vancouver
Gonzaga N de A, Awata WMC, Tanus-Santos JE, Padovan JC, Tirapelli CR. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin [Internet]. European Journal of Pharmacology. 2019 ; 847 103-112.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2019.01.055
Artigo de periodico
Sodium nitrite attenuates MMP-9 production by endothelial cells and may explain similar effects of atorvastatin (2016)
Meschiari, Cesar A; Pinheiro, Lucas C; Guimaraes, Danielle A; Gerlach, Raquel Fernanda; Tanus-Santos, José Eduardo
Fonte: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidades: FORP, FMRP
Assuntos: ÓXIDO NÍTRICO, CELULAS ENDOTELIAIS, METALOPROTEINASES, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
MESCHIARI, Cesar A et al. Sodium nitrite attenuates MMP-9 production by endothelial cells and may explain similar effects of atorvastatin. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 389, n. 2, p. 223-231, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00210-015-1192-4. Acesso em: 02 nov. 2024.
APA
Meschiari, C. A., Pinheiro, L. C., Guimaraes, D. A., Gerlach, R. F., & Tanus-Santos, J. E. (2016). Sodium nitrite attenuates MMP-9 production by endothelial cells and may explain similar effects of atorvastatin. Naunyn-Schmiedeberg's Archives of Pharmacology, 389( 2), 223-231. doi:10.1007/s00210-015-1192-4
NLM
Meschiari CA, Pinheiro LC, Guimaraes DA, Gerlach RF, Tanus-Santos JE. Sodium nitrite attenuates MMP-9 production by endothelial cells and may explain similar effects of atorvastatin [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 2): 223-231.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00210-015-1192-4
Vancouver
Meschiari CA, Pinheiro LC, Guimaraes DA, Gerlach RF, Tanus-Santos JE. Sodium nitrite attenuates MMP-9 production by endothelial cells and may explain similar effects of atorvastatin [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 2): 223-231.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00210-015-1192-4
Artigo de periodico
Homocysteine and nitrite levels are modulated by MTHFR 677C>T polymorphism in obese women treated with simvastatin (2014)
Villela, Maria P; Andrade, Vanessa L; Eccard, Bryelle; Jordão Júnior, Alceu Afonso; Sertório, Jonas T; Tanus-Santos, José Eduardo; Silva, Ieda F. O; Silveira, Josianne N; Sandrim, Valéria C
Fonte: Clinical and Experimental Pharmacology and Physiology. Unidade: FMRP
Assuntos: OBESIDADE, MULHERES, FÁRMACOS (SISTEMA CARDIOVASCULAR) (EFEITOS BIOLÓGICOS)
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ABNT
VILLELA, Maria P et al. Homocysteine and nitrite levels are modulated by MTHFR 677C>T polymorphism in obese women treated with simvastatin. Clinical and Experimental Pharmacology and Physiology, v. 41, n. 10, p. 744-747, 2014Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.12284. Acesso em: 02 nov. 2024.
APA
Villela, M. P., Andrade, V. L., Eccard, B., Jordão Júnior, A. A., Sertório, J. T., Tanus-Santos, J. E., et al. (2014). Homocysteine and nitrite levels are modulated by MTHFR 677C>T polymorphism in obese women treated with simvastatin. Clinical and Experimental Pharmacology and Physiology, 41( 10), 744-747. doi:10.1111/1440-1681.12284
NLM
Villela MP, Andrade VL, Eccard B, Jordão Júnior AA, Sertório JT, Tanus-Santos JE, Silva IFO, Silveira JN, Sandrim VC. Homocysteine and nitrite levels are modulated by MTHFR 677C>T polymorphism in obese women treated with simvastatin [Internet]. Clinical and Experimental Pharmacology and Physiology. 2014 ; 41( 10): 744-747.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/1440-1681.12284
Vancouver
Villela MP, Andrade VL, Eccard B, Jordão Júnior AA, Sertório JT, Tanus-Santos JE, Silva IFO, Silveira JN, Sandrim VC. Homocysteine and nitrite levels are modulated by MTHFR 677C>T polymorphism in obese women treated with simvastatin [Internet]. Clinical and Experimental Pharmacology and Physiology. 2014 ; 41( 10): 744-747.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/1440-1681.12284
Artigo de periodico
Inducible nitric oxide synthase as a possible target in hypertension (2014)
Oliveira-Paula, Gustavo H; Lacchini, Riccardo ; Tanus-Santos, José Eduardo
Fonte: Current Drug Targets. Unidades: EERP, FMRP
Assuntos: FÁRMACOS (SISTEMA CARDIOVASCULAR), MODELOS ANIMAIS DE DOENÇAS, HIPERTENSÃO, ÓXIDO NÍTRICO, EXPRESSÃO GÊNICA
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ABNT
OLIVEIRA-PAULA, Gustavo H e LACCHINI, Riccardo e TANUS-SANTOS, José Eduardo. Inducible nitric oxide synthase as a possible target in hypertension. Current Drug Targets, v. 15, n. 2, p. 164-174, 2014Tradução . . Disponível em: https://doi.org/10.2174/13894501113146660227. Acesso em: 02 nov. 2024.
APA
Oliveira-Paula, G. H., Lacchini, R., & Tanus-Santos, J. E. (2014). Inducible nitric oxide synthase as a possible target in hypertension. Current Drug Targets, 15( 2), 164-174. doi:10.2174/13894501113146660227
NLM
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. Inducible nitric oxide synthase as a possible target in hypertension [Internet]. Current Drug Targets. 2014 ; 15( 2): 164-174.[citado 2024 nov. 02 ] Available from: https://doi.org/10.2174/13894501113146660227
Vancouver
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. Inducible nitric oxide synthase as a possible target in hypertension [Internet]. Current Drug Targets. 2014 ; 15( 2): 164-174.[citado 2024 nov. 02 ] Available from: https://doi.org/10.2174/13894501113146660227
Artigo de periodico
Relationship between adiponectin and nitrite in healthy and preeclampsia pregnancies (2013)
Eleuterio, Nibia Mariana; Palei, Ana C. T.; Machado, Jackeline S. Rangel; Tanus-Santos, José Eduardo; Cavalli, Ricardo de Carvalho; Sandrim, Valeria C.
Fonte: Clinica Chimica Acta. Unidade: FMRP
Assuntos: PRÉ-ECLÂMPSIA, ÓXIDO NÍTRICO, ADIPONECTINA
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ABNT
ELEUTERIO, Nibia Mariana et al. Relationship between adiponectin and nitrite in healthy and preeclampsia pregnancies. Clinica Chimica Acta, v. 423, p. 112-115, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.cca.2013.04.027. Acesso em: 02 nov. 2024.
APA
Eleuterio, N. M., Palei, A. C. T., Machado, J. S. R., Tanus-Santos, J. E., Cavalli, R. de C., & Sandrim, V. C. (2013). Relationship between adiponectin and nitrite in healthy and preeclampsia pregnancies. Clinica Chimica Acta, 423, 112-115. doi:10.1016/j.cca.2013.04.027
NLM
Eleuterio NM, Palei ACT, Machado JSR, Tanus-Santos JE, Cavalli R de C, Sandrim VC. Relationship between adiponectin and nitrite in healthy and preeclampsia pregnancies [Internet]. Clinica Chimica Acta. 2013 ; 423 112-115.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cca.2013.04.027
Vancouver
Eleuterio NM, Palei ACT, Machado JSR, Tanus-Santos JE, Cavalli R de C, Sandrim VC. Relationship between adiponectin and nitrite in healthy and preeclampsia pregnancies [Internet]. Clinica Chimica Acta. 2013 ; 423 112-115.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cca.2013.04.027
Artigo de periodico
Circulating interleukin-6 and high-sensitivity C-reactive protein decrease after periodontal therapy in otherwise healthy subjects (2009)
Marcaccini, Andrea M.; Meschiari, César A.; Sorgi, Carlos Artério; Saraiva, Maria da Conceição Pereira; Souza, Ana Maria de; Faccioli, Lúcia Helena; Tanus-Santos, José Eduardo; Novaes Júnior, Arthur Belém; Gerlach, Raquel Fernanda
Fonte: Journal of Periodontology. Unidades: FORP, FMRP, FCFRP
Assuntos: CITOQUÍMICA, INFLAMAÇÃO, PERIODONTITE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARCACCINI, Andrea M. et al. Circulating interleukin-6 and high-sensitivity C-reactive protein decrease after periodontal therapy in otherwise healthy subjects. Journal of Periodontology, v. 80, n. 4, p. 594-602, 2009Tradução . . Acesso em: 02 nov. 2024.
APA
Marcaccini, A. M., Meschiari, C. A., Sorgi, C. A., Saraiva, M. da C. P., Souza, A. M. de, Faccioli, L. H., et al. (2009). Circulating interleukin-6 and high-sensitivity C-reactive protein decrease after periodontal therapy in otherwise healthy subjects. Journal of Periodontology, 80( 4), 594-602.
NLM
Marcaccini AM, Meschiari CA, Sorgi CA, Saraiva M da CP, Souza AM de, Faccioli LH, Tanus-Santos JE, Novaes Júnior AB, Gerlach RF. Circulating interleukin-6 and high-sensitivity C-reactive protein decrease after periodontal therapy in otherwise healthy subjects. Journal of Periodontology. 2009 ; 80( 4): 594-602.[citado 2024 nov. 02 ]
Vancouver
Marcaccini AM, Meschiari CA, Sorgi CA, Saraiva M da CP, Souza AM de, Faccioli LH, Tanus-Santos JE, Novaes Júnior AB, Gerlach RF. Circulating interleukin-6 and high-sensitivity C-reactive protein decrease after periodontal therapy in otherwise healthy subjects. Journal of Periodontology. 2009 ; 80( 4): 594-602.[citado 2024 nov. 02 ]
Artigo de periodico
Mercury exposure increases circulating net matrix metalloproteinase (MMP)-2 and MMP-9 activities (2009)
Jacob-Ferreira, Anna L. B.; Passos, Carlos J. S.; Jordão, Alceu A.; Fillion, Myriam; Mergler, Donna; Lemire, Mélanie; Gerlach, Raquel Fernanda; Barbosa Júnior, Fernando; Tanus-Santos, José Eduardo
Fonte: Basic and Clinical Pharmacology and Toxicology. Unidades: FORP, FCFRP, FMRP
Assuntos: METALOPROTEINASES, ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES, MERCÚRIO (ELEMENTO QUÍMICO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JACOB-FERREIRA, Anna L. B. et al. Mercury exposure increases circulating net matrix metalloproteinase (MMP)-2 and MMP-9 activities. Basic and Clinical Pharmacology and Toxicology, v. 105, n. 4, p. 281-288, 2009Tradução . . Acesso em: 02 nov. 2024.
APA
Jacob-Ferreira, A. L. B., Passos, C. J. S., Jordão, A. A., Fillion, M., Mergler, D., Lemire, M., et al. (2009). Mercury exposure increases circulating net matrix metalloproteinase (MMP)-2 and MMP-9 activities. Basic and Clinical Pharmacology and Toxicology, 105( 4), 281-288.
NLM
Jacob-Ferreira ALB, Passos CJS, Jordão AA, Fillion M, Mergler D, Lemire M, Gerlach RF, Barbosa Júnior F, Tanus-Santos JE. Mercury exposure increases circulating net matrix metalloproteinase (MMP)-2 and MMP-9 activities. Basic and Clinical Pharmacology and Toxicology. 2009 ; 105( 4): 281-288.[citado 2024 nov. 02 ]
Vancouver
Jacob-Ferreira ALB, Passos CJS, Jordão AA, Fillion M, Mergler D, Lemire M, Gerlach RF, Barbosa Júnior F, Tanus-Santos JE. Mercury exposure increases circulating net matrix metalloproteinase (MMP)-2 and MMP-9 activities. Basic and Clinical Pharmacology and Toxicology. 2009 ; 105( 4): 281-288.[citado 2024 nov. 02 ]
Artigo de periodico
Lead in saliva from lead-exposed and unexposed children (2009)
Almeida, Glauce Regina Costa de; Freitas, Clarice Umbelino; Barbosa Júnior, Fernando; Tanus-Santos, José Eduardo; Gerlach, Raquel Fernanda
Fonte: Science of the Total Environment. Unidades: FORP, FMRP, FCFRP
Assuntos: DENTE DECÍDUO, SALIVA, CRIANÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Glauce Regina Costa de et al. Lead in saliva from lead-exposed and unexposed children. Science of the Total Environment, v. 407, n. 5, p. 1547-1550, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.scitotenv.2008.10.058. Acesso em: 02 nov. 2024.
APA
Almeida, G. R. C. de, Freitas, C. U., Barbosa Júnior, F., Tanus-Santos, J. E., & Gerlach, R. F. (2009). Lead in saliva from lead-exposed and unexposed children. Science of the Total Environment, 407( 5), 1547-1550. doi:10.1016/j.scitotenv.2008.10.058
NLM
Almeida GRC de, Freitas CU, Barbosa Júnior F, Tanus-Santos JE, Gerlach RF. Lead in saliva from lead-exposed and unexposed children [Internet]. Science of the Total Environment. 2009 ; 407( 5): 1547-1550.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.scitotenv.2008.10.058
Vancouver
Almeida GRC de, Freitas CU, Barbosa Júnior F, Tanus-Santos JE, Gerlach RF. Lead in saliva from lead-exposed and unexposed children [Internet]. Science of the Total Environment. 2009 ; 407( 5): 1547-1550.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.scitotenv.2008.10.058
Artigo de periodico
Isoflavone genistein inhibits the angiotensin-converting enzyme and alters the vascular responses to angiotensin I and bradykinin (2009)
Montenegro, Marcelo Freitas; Pessa, Lisandra R.; Santos, José Eduardo Tanus dos
Fonte: European Journal of Pharmacology. Unidade: FMRP
Assuntos: ANGIOTENSINA II, SISTEMA CARDIOVASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MONTENEGRO, Marcelo Freitas e PESSA, Lisandra R. e SANTOS, José Eduardo Tanus dos. Isoflavone genistein inhibits the angiotensin-converting enzyme and alters the vascular responses to angiotensin I and bradykinin. European Journal of Pharmacology, v. 607, n. 1/3, p. 173-177, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2009.02.015. Acesso em: 02 nov. 2024.
APA
Montenegro, M. F., Pessa, L. R., & Santos, J. E. T. dos. (2009). Isoflavone genistein inhibits the angiotensin-converting enzyme and alters the vascular responses to angiotensin I and bradykinin. European Journal of Pharmacology, 607( 1/3), 173-177. doi:10.1016/j.ejphar.2009.02.015
NLM
Montenegro MF, Pessa LR, Santos JET dos. Isoflavone genistein inhibits the angiotensin-converting enzyme and alters the vascular responses to angiotensin I and bradykinin [Internet]. European Journal of Pharmacology. 2009 ; 607( 1/3): 173-177.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2009.02.015
Vancouver
Montenegro MF, Pessa LR, Santos JET dos. Isoflavone genistein inhibits the angiotensin-converting enzyme and alters the vascular responses to angiotensin I and bradykinin [Internet]. European Journal of Pharmacology. 2009 ; 607( 1/3): 173-177.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2009.02.015
Artigo de periodico
A plateau detected in lead accumulation in subsurface deciduous enamel from individuals exposed to lead may be useful to identify children and regions exposed to higher levels of lead (2008)
Almeida, Glauce Regina Costa de; Guerra, Carolina de Souza; Tanus-Santos, José Eduardo; Barbosa Júnior, Fernando; Gerlach, Raquel Fernanda
Fonte: Environmental Research. Unidades: FORP, FCFRP, FMRP
Assuntos: ATENÇÃO À SAÚDE, CONTAMINAÇÃO, CHUMBO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Glauce Regina Costa de et al. A plateau detected in lead accumulation in subsurface deciduous enamel from individuals exposed to lead may be useful to identify children and regions exposed to higher levels of lead. Environmental Research, v. 107, n. 2, p. 264-270, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.envres.2008.01.002. Acesso em: 02 nov. 2024.
APA
Almeida, G. R. C. de, Guerra, C. de S., Tanus-Santos, J. E., Barbosa Júnior, F., & Gerlach, R. F. (2008). A plateau detected in lead accumulation in subsurface deciduous enamel from individuals exposed to lead may be useful to identify children and regions exposed to higher levels of lead. Environmental Research, 107( 2), 264-270. doi:10.1016/j.envres.2008.01.002
NLM
Almeida GRC de, Guerra C de S, Tanus-Santos JE, Barbosa Júnior F, Gerlach RF. A plateau detected in lead accumulation in subsurface deciduous enamel from individuals exposed to lead may be useful to identify children and regions exposed to higher levels of lead [Internet]. Environmental Research. 2008 ; 107( 2): 264-270.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.envres.2008.01.002
Vancouver
Almeida GRC de, Guerra C de S, Tanus-Santos JE, Barbosa Júnior F, Gerlach RF. A plateau detected in lead accumulation in subsurface deciduous enamel from individuals exposed to lead may be useful to identify children and regions exposed to higher levels of lead [Internet]. Environmental Research. 2008 ; 107( 2): 264-270.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.envres.2008.01.002
Artigo de periodico
Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats (2008)
Martinez, Marcio L. L.; Castro, Michele M.; Rizzi, Elen; Fernandes, Karla; Demacq, Caroline; Bendhack, Lusiane Maria; Gerlach, Raquel Fernanda; Tanus-Santos, José Eduardo
Fonte: European Journal of Pharmacology. Unidades: FCFRP, FORP, FMRP
Assuntos: HIPERTENSÃO, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINEZ, Marcio L. L. et al. Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats. European Journal of Pharmacology, v. 591, n. 1-3, p. 224-230, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.06.096. Acesso em: 02 nov. 2024.
APA
Martinez, M. L. L., Castro, M. M., Rizzi, E., Fernandes, K., Demacq, C., Bendhack, L. M., et al. (2008). Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats. European Journal of Pharmacology, 591( 1-3), 224-230. doi:10.1016/j.ejphar.2008.06.096
NLM
Martinez MLL, Castro MM, Rizzi E, Fernandes K, Demacq C, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats [Internet]. European Journal of Pharmacology. 2008 ; 591( 1-3): 224-230.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2008.06.096
Vancouver
Martinez MLL, Castro MM, Rizzi E, Fernandes K, Demacq C, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats [Internet]. European Journal of Pharmacology. 2008 ; 591( 1-3): 224-230.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2008.06.096
Artigo de periodico
Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia (2008)
Sandrim, Valéria C.; Palei, Ana C. T.; Metzger, Ingrid F.; Gomes, Valéria A.; Cavalli, Ricardo C.; Tanus-Santos, José Eduardo
Fonte: Hypertension. Unidade: FMRP
Assuntos: SAÚDE DA MULHER, ÓXIDO NÍTRICO, PRÉ-ECLÂMPSIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANDRIM, Valéria C. et al. Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension, v. 52, n. 2, p. 402-407, 2008Tradução . . Acesso em: 02 nov. 2024.
APA
Sandrim, V. C., Palei, A. C. T., Metzger, I. F., Gomes, V. A., Cavalli, R. C., & Tanus-Santos, J. E. (2008). Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension, 52( 2), 402-407.
NLM
Sandrim VC, Palei ACT, Metzger IF, Gomes VA, Cavalli RC, Tanus-Santos JE. Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension. 2008 ; 52( 2): 402-407.[citado 2024 nov. 02 ]
Vancouver
Sandrim VC, Palei ACT, Metzger IF, Gomes VA, Cavalli RC, Tanus-Santos JE. Nitric oxide formation is inversely related to serum levels of antiangiogenic factors soluble Fms-like tyrosine kinase-1 and soluble endogline in preeclampsia. Hypertension. 2008 ; 52( 2): 402-407.[citado 2024 nov. 02 ]

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