Filtros : "ICB-BMA" "Inglaterra" Removidos: "Fisiologia" "FMRP-RPP" "Colômbia" "Reuniao Anual da Sociedade Brasileira de Quimica" Limpar

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  • Fonte: Heliyon. Unidade: ICB

    Assuntos: ANATOMIA, NEURÔNIOS, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, CÉREBRO, MAPEAMENTO CEREBRAL, HIPOTÁLAMO, CÓRTEX CEREBRAL, AXÔNIOS

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    • ABNT

      BORTOLOCI, João Guilherme Tassoni e MOTTA, Simone Cristina. Failure of AAV retrograde tracer transduction in hypothalamic projections to the periaqueductal gray matter. Heliyon, v. 8, n. 8, p. 6 , 2022Tradução . . Disponível em: https://doi.org/10.1016/j.heliyon.2022.e10243. Acesso em: 18 nov. 2024.
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      Bortoloci, J. G. T., & Motta, S. C. (2022). Failure of AAV retrograde tracer transduction in hypothalamic projections to the periaqueductal gray matter. Heliyon, 8( 8), 6 . doi:10.1016/j.heliyon.2022.e10243
    • NLM

      Bortoloci JGT, Motta SC. Failure of AAV retrograde tracer transduction in hypothalamic projections to the periaqueductal gray matter [Internet]. Heliyon. 2022 ; 8( 8): 6 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.heliyon.2022.e10243
    • Vancouver

      Bortoloci JGT, Motta SC. Failure of AAV retrograde tracer transduction in hypothalamic projections to the periaqueductal gray matter [Internet]. Heliyon. 2022 ; 8( 8): 6 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.heliyon.2022.e10243
  • Fonte: Cardiovascular Research. Unidade: ICB

    Assuntos: ANATOMIA, INFLAMAÇÃO, ENZIMAS, DOENÇAS CARDIOVASCULARES, SISTEMA CARDIOVASCULAR

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    • ABNT

      RIOS, Francisco J. et al. Chanzyme TRPM7 protects against cardiovascular inflammation and fibrosis. Cardiovascular Research, p. 1-15, 2019Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvz164. Acesso em: 18 nov. 2024.
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      Rios, F. J., Zou, Z. -G., Harvey, A. P., Harvey, K. Y., Nosalski, R., Anyfanti, P., et al. (2019). Chanzyme TRPM7 protects against cardiovascular inflammation and fibrosis. Cardiovascular Research, 1-15. doi:10.1093/cvr/cvz164
    • NLM

      Rios FJ, Zou Z-G, Harvey AP, Harvey KY, Nosalski R, Anyfanti P, Camargo L de L, Lacchini S, Ryazanov AG, Ryazanova L, McGrath S, Guzik TJ, Goodyear CS, Montezano AC, Touyz RM. Chanzyme TRPM7 protects against cardiovascular inflammation and fibrosis [Internet]. Cardiovascular Research. 2019 ; 1-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1093/cvr/cvz164
    • Vancouver

      Rios FJ, Zou Z-G, Harvey AP, Harvey KY, Nosalski R, Anyfanti P, Camargo L de L, Lacchini S, Ryazanov AG, Ryazanova L, McGrath S, Guzik TJ, Goodyear CS, Montezano AC, Touyz RM. Chanzyme TRPM7 protects against cardiovascular inflammation and fibrosis [Internet]. Cardiovascular Research. 2019 ; 1-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1093/cvr/cvz164
  • Fonte: British Journal of Nutrition. Unidade: ICB

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      MARÇAL, Anderson Carlos et al. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, v. 109, n. 12, p. 2154-2165, 2013Tradução . . Disponível em: https://doi.org/10.1017/S0007114512004576. Acesso em: 18 nov. 2024.
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      Marçal, A. C., Camporez, J. P. G., Lima-Salgado, T. M., Cintra, D. E., Akamine, E. H., Ribeiro, L. M., et al. (2013). Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, 109( 12), 2154-2165. doi:10.1017/S0007114512004576
    • NLM

      Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1017/S0007114512004576
    • Vancouver

      Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1017/S0007114512004576
  • Fonte: Neuroscience. Unidades: ICB, FMRP

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      DONATO JUNIOR, José et al. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition. Neuroscience, v. 241, p. 67-79, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.03.013. Acesso em: 18 nov. 2024.
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      Donato Junior, J., Lee. C,, Ratra, D. V., Franci, C. R., Canteras, N. S., & Elias, C. F. (2013). Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition. Neuroscience, 241, 67-79. doi:10.1016/j.neuroscience.2013.03.013
    • NLM

      Donato Junior J, Lee. C, Ratra DV, Franci CR, Canteras NS, Elias CF. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition [Internet]. Neuroscience. 2013 ; 241 67-79.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.03.013
    • Vancouver

      Donato Junior J, Lee. C, Ratra DV, Franci CR, Canteras NS, Elias CF. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition [Internet]. Neuroscience. 2013 ; 241 67-79.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.03.013
  • Fonte: Journal of the American Heart Association. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      DISATNIK, Marie-Hélène et al. Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction. Journal of the American Heart Association, v. 2, n. 5, p. 1-14, 2013Tradução . . Disponível em: https://doi.org/10.1161/JAHA.113.000461. Acesso em: 18 nov. 2024.
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      Disatnik, M. -H., Ferreira, J. C. B., Campos, J. C., Gomes, K. M. S., Dourado, P. M. M., Qi, X., & Mochly-Rosen, D. (2013). Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction. Journal of the American Heart Association, 2( 5), 1-14. doi:10.1161/JAHA.113.000461
    • NLM

      Disatnik M-H, Ferreira JCB, Campos JC, Gomes KMS, Dourado PMM, Qi X, Mochly-Rosen D. Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction [Internet]. Journal of the American Heart Association. 2013 ; 2( 5): 1-14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1161/JAHA.113.000461
    • Vancouver

      Disatnik M-H, Ferreira JCB, Campos JC, Gomes KMS, Dourado PMM, Qi X, Mochly-Rosen D. Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction [Internet]. Journal of the American Heart Association. 2013 ; 2( 5): 1-14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1161/JAHA.113.000461
  • Fonte: Food and Chemical Toxicology. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      CAMPOS, Juliane Cruz e GOMES, Kátia Maria Sampaio e FERREIRA, Julio Cesar Batista. Impact of exercise training on redox signaling in cardiovascular diseases. Food and Chemical Toxicology, v. 61, p. 107-119, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.fct.2013.08.035. Acesso em: 18 nov. 2024.
    • APA

      Campos, J. C., Gomes, K. M. S., & Ferreira, J. C. B. (2013). Impact of exercise training on redox signaling in cardiovascular diseases. Food and Chemical Toxicology, 61, 107-119. doi:10.1016/j.fct.2013.08.035
    • NLM

      Campos JC, Gomes KMS, Ferreira JCB. Impact of exercise training on redox signaling in cardiovascular diseases [Internet]. Food and Chemical Toxicology. 2013 ; 61107-119.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.fct.2013.08.035
    • Vancouver

      Campos JC, Gomes KMS, Ferreira JCB. Impact of exercise training on redox signaling in cardiovascular diseases [Internet]. Food and Chemical Toxicology. 2013 ; 61107-119.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.fct.2013.08.035
  • Fonte: Lasers in Medical Science. Unidades: ICB, EACH

    Assunto: ANATOMIA

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    • ABNT

      SILVA, Lucila Hernandes da et al. GaAs 904-nm laser irradiation improves myofiber mass recovery during regeneration of skeletal muscle previously damaged by crotoxin. Lasers in Medical Science, v. 27, n. 5, p. 993-1000, 2012Tradução . . Disponível em: https://doi.org/10.1007/s10103-011-1031-x. Acesso em: 18 nov. 2024.
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      Silva, L. H. da, Silva, M. T. da, Gutierrez, R. M., Conte, T. C., Toledo, C. A., Aoki, M. S., et al. (2012). GaAs 904-nm laser irradiation improves myofiber mass recovery during regeneration of skeletal muscle previously damaged by crotoxin. Lasers in Medical Science, 27( 5), 993-1000. doi:10.1007/s10103-011-1031-x
    • NLM

      Silva LH da, Silva MT da, Gutierrez RM, Conte TC, Toledo CA, Aoki MS, Liebano RE, Miyabara EH. GaAs 904-nm laser irradiation improves myofiber mass recovery during regeneration of skeletal muscle previously damaged by crotoxin [Internet]. Lasers in Medical Science. 2012 ; 27( 5): 993-1000.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10103-011-1031-x
    • Vancouver

      Silva LH da, Silva MT da, Gutierrez RM, Conte TC, Toledo CA, Aoki MS, Liebano RE, Miyabara EH. GaAs 904-nm laser irradiation improves myofiber mass recovery during regeneration of skeletal muscle previously damaged by crotoxin [Internet]. Lasers in Medical Science. 2012 ; 27( 5): 993-1000.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10103-011-1031-x
  • Fonte: Journal of Molecular Endocrinology. Unidade: ICB

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      DINIZ, Gabriela Placoná et al. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression. Journal of Molecular Endocrinology, v. 49, n. 1, p. 11-20, 2012Tradução . . Disponível em: https://doi.org/10.1530/JME-11-0141. Acesso em: 18 nov. 2024.
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      Diniz, G. P., Takano, A. P. C., Brunetto-Ract, E. L., Silva, F. G. da, Nunes, M. T., & Barreto-Chaves, M. L. M. (2012). New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression. Journal of Molecular Endocrinology, 49( 1), 11-20. doi:10.1530/JME-11-0141
    • NLM

      Diniz GP, Takano APC, Brunetto-Ract EL, Silva FG da, Nunes MT, Barreto-Chaves MLM. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression [Internet]. Journal of Molecular Endocrinology. 2012 ; 49( 1): 11-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1530/JME-11-0141
    • Vancouver

      Diniz GP, Takano APC, Brunetto-Ract EL, Silva FG da, Nunes MT, Barreto-Chaves MLM. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression [Internet]. Journal of Molecular Endocrinology. 2012 ; 49( 1): 11-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1530/JME-11-0141
  • Fonte: NMR in Biomedicine. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      JOSAN, Sonal et al. In vivo measurement of aldehyde dehydrogenase-2 activity in rat liver ethanol model using dynamic MRSI of hyperpolarized [1-13C]pyruvate. NMR in Biomedicine, v. 6, 2012Tradução . . Disponível em: https://doi.org/10.1002/nbm.2897. Acesso em: 18 nov. 2024.
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      Josan, S., Xu, T., Yen, Y. -F., Hurd, R., Ferreira, J. C. B., Chen, C. -H., et al. (2012). In vivo measurement of aldehyde dehydrogenase-2 activity in rat liver ethanol model using dynamic MRSI of hyperpolarized [1-13C]pyruvate. NMR in Biomedicine, 6. doi:10.1002/nbm.2897
    • NLM

      Josan S, Xu T, Yen Y-F, Hurd R, Ferreira JCB, Chen C-H, Mochly-Rosen D, Pfefferbaum A, Mayer D, Spielman D. In vivo measurement of aldehyde dehydrogenase-2 activity in rat liver ethanol model using dynamic MRSI of hyperpolarized [1-13C]pyruvate [Internet]. NMR in Biomedicine. 2012 ; 6[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/nbm.2897
    • Vancouver

      Josan S, Xu T, Yen Y-F, Hurd R, Ferreira JCB, Chen C-H, Mochly-Rosen D, Pfefferbaum A, Mayer D, Spielman D. In vivo measurement of aldehyde dehydrogenase-2 activity in rat liver ethanol model using dynamic MRSI of hyperpolarized [1-13C]pyruvate [Internet]. NMR in Biomedicine. 2012 ; 6[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/nbm.2897
  • Fonte: Experimental Physiology. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      SCHENKEL, Paulo Cavalheiro et al. Time course of hydrogen peroxide–thioredoxin balance and its influence on the intracellular signalling in myocardial infarction. Experimental Physiology, v. 97, n. 6, p. 741-749, 2012Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2012.064832. Acesso em: 18 nov. 2024.
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      Schenkel, P. C., Tavares, A. M. V., Fernandes, R. O., Diniz, G. P., Ludke, A. R. L., Ribeiro, M. F. M., et al. (2012). Time course of hydrogen peroxide–thioredoxin balance and its influence on the intracellular signalling in myocardial infarction. Experimental Physiology, 97( 6), 741-749. doi:10.1113/expphysiol.2012.064832
    • NLM

      Schenkel PC, Tavares AMV, Fernandes RO, Diniz GP, Ludke ARL, Ribeiro MFM, Araujo AS da R, Barreto-Chaves MLM, Belló-Klein A. Time course of hydrogen peroxide–thioredoxin balance and its influence on the intracellular signalling in myocardial infarction [Internet]. Experimental Physiology. 2012 ; 97( 6): 741-749.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1113/expphysiol.2012.064832
    • Vancouver

      Schenkel PC, Tavares AMV, Fernandes RO, Diniz GP, Ludke ARL, Ribeiro MFM, Araujo AS da R, Barreto-Chaves MLM, Belló-Klein A. Time course of hydrogen peroxide–thioredoxin balance and its influence on the intracellular signalling in myocardial infarction [Internet]. Experimental Physiology. 2012 ; 97( 6): 741-749.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1113/expphysiol.2012.064832
  • Fonte: Molecular Pain. Unidade: ICB

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      SANTOS, Fabio Martinez dos et al. Neural mobilization reverses behavioral and cellular changes that characterize neuropathic pain in rats. Molecular Pain, v. 8, p. 1-9, 2012Tradução . . Disponível em: https://doi.org/10.1186/1744-8069-8-57. Acesso em: 18 nov. 2024.
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      Santos, F. M. dos, Silva, J. T., Giardine, A. C., Rocha, P. de A., Achermann, A. P. P., Alves, A. da S., et al. (2012). Neural mobilization reverses behavioral and cellular changes that characterize neuropathic pain in rats. Molecular Pain, 8, 1-9. doi:10.1186/1744-8069-8-57
    • NLM

      Santos FM dos, Silva JT, Giardine AC, Rocha P de A, Achermann APP, Alves A da S, Britto LRG de, Chacur M. Neural mobilization reverses behavioral and cellular changes that characterize neuropathic pain in rats [Internet]. Molecular Pain. 2012 ; 8 1-9.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/1744-8069-8-57
    • Vancouver

      Santos FM dos, Silva JT, Giardine AC, Rocha P de A, Achermann APP, Alves A da S, Britto LRG de, Chacur M. Neural mobilization reverses behavioral and cellular changes that characterize neuropathic pain in rats [Internet]. Molecular Pain. 2012 ; 8 1-9.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/1744-8069-8-57
  • Fonte: Behavioural Pharmacology. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      GUTIERREZ, Vanessa Pacciari et al. The peripheral L-arginine–nitric oxide–cyclic GMP pathway and ATP-sensitive K+ channels are involved in the antinociceptive effect of crotalphine on neuropathic pain in rats. Behavioural Pharmacology, v. 23, n. 1, p. 14-24, 2012Tradução . . Disponível em: https://doi.org/10.1097/FBP.0b013e32834eafbc. Acesso em: 18 nov. 2024.
    • APA

      Gutierrez, V. P., Zambelli, V. O., Picolo, G., Chacur, M., Sampaio, S. C., Brigatte, P., et al. (2012). The peripheral L-arginine–nitric oxide–cyclic GMP pathway and ATP-sensitive K+ channels are involved in the antinociceptive effect of crotalphine on neuropathic pain in rats. Behavioural Pharmacology, 23( 1), 14-24. doi:10.1097/FBP.0b013e32834eafbc
    • NLM

      Gutierrez VP, Zambelli VO, Picolo G, Chacur M, Sampaio SC, Brigatte P, Konno K, Cury Y. The peripheral L-arginine–nitric oxide–cyclic GMP pathway and ATP-sensitive K+ channels are involved in the antinociceptive effect of crotalphine on neuropathic pain in rats [Internet]. Behavioural Pharmacology. 2012 ; 23( 1): 14-24.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1097/FBP.0b013e32834eafbc
    • Vancouver

      Gutierrez VP, Zambelli VO, Picolo G, Chacur M, Sampaio SC, Brigatte P, Konno K, Cury Y. The peripheral L-arginine–nitric oxide–cyclic GMP pathway and ATP-sensitive K+ channels are involved in the antinociceptive effect of crotalphine on neuropathic pain in rats [Internet]. Behavioural Pharmacology. 2012 ; 23( 1): 14-24.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1097/FBP.0b013e32834eafbc
  • Fonte: Nature Reviews Neuroscience. Unidade: ICB

    Assunto: ANATOMIA

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      GROSS, Cornelius T. e CANTERAS, Newton Sabino. The many paths to fear. Nature Reviews Neuroscience, v. 13, n. 9, p. 651-658, 2012Tradução . . Disponível em: https://doi.org/10.1038/nrn3301. Acesso em: 18 nov. 2024.
    • APA

      Gross, C. T., & Canteras, N. S. (2012). The many paths to fear. Nature Reviews Neuroscience, 13( 9), 651-658. doi:10.1038/nrn3301
    • NLM

      Gross CT, Canteras NS. The many paths to fear [Internet]. Nature Reviews Neuroscience. 2012 ; 13( 9): 651-658.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/nrn3301
    • Vancouver

      Gross CT, Canteras NS. The many paths to fear [Internet]. Nature Reviews Neuroscience. 2012 ; 13( 9): 651-658.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/nrn3301
  • Fonte: Neuroscience. Unidade: ICB

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      MARTINEZ, R. C. et al. Amygdalar roles during exposure to a live predator and to a predator-associated context. Neuroscience, v. 172, n. 13, p. 314-328, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2010.10.033. Acesso em: 18 nov. 2024.
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      Martinez, R. C., Carvalho-Netto, E. F., Ribeiro-Barbosa, É. R., Baldo, M. V. C., & Canteras, N. S. (2011). Amygdalar roles during exposure to a live predator and to a predator-associated context. Neuroscience, 172( 13), 314-328. doi:10.1016/j.neuroscience.2010.10.033
    • NLM

      Martinez RC, Carvalho-Netto EF, Ribeiro-Barbosa ÉR, Baldo MVC, Canteras NS. Amygdalar roles during exposure to a live predator and to a predator-associated context [Internet]. Neuroscience. 2011 ; 172( 13): 314-328.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2010.10.033
    • Vancouver

      Martinez RC, Carvalho-Netto EF, Ribeiro-Barbosa ÉR, Baldo MVC, Canteras NS. Amygdalar roles during exposure to a live predator and to a predator-associated context [Internet]. Neuroscience. 2011 ; 172( 13): 314-328.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2010.10.033
  • Fonte: Micron. Unidades: ICB, FORP

    Assunto: ANATOMIA

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      CIENA, Adriano Polican et al. Histochemical and ultrastructural changes of sternomastoid muscle in aged wistar rats. Micron, v. 42, n. 8, p. 871-876, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.micron.2011.06.003. Acesso em: 18 nov. 2024.
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      Ciena, A. P., Almeida, S. R. Y. de, Alves, P. H. de M., Bolina-Matos, R. de S., Dias, F. J., Issa, J. P. M., et al. (2011). Histochemical and ultrastructural changes of sternomastoid muscle in aged wistar rats. Micron, 42( 8), 871-876. doi:10.1016/j.micron.2011.06.003
    • NLM

      Ciena AP, Almeida SRY de, Alves PH de M, Bolina-Matos R de S, Dias FJ, Issa JPM, Iyomasa MM, Watanabe I-S. Histochemical and ultrastructural changes of sternomastoid muscle in aged wistar rats [Internet]. Micron. 2011 ; 42( 8): 871-876.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.micron.2011.06.003
    • Vancouver

      Ciena AP, Almeida SRY de, Alves PH de M, Bolina-Matos R de S, Dias FJ, Issa JPM, Iyomasa MM, Watanabe I-S. Histochemical and ultrastructural changes of sternomastoid muscle in aged wistar rats [Internet]. Micron. 2011 ; 42( 8): 871-876.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.micron.2011.06.003
  • Fonte: Current Opinion in Endocrinology, Diabetes and Obesity. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      BARRETO-CHAVES, Maria Luiza Morais e MONTEIRO, Priscilla de Souza e FÜRSTENAU, Cristina R. Acute actions of thyroid hormone on blood vessel biochemistry and physiology. Current Opinion in Endocrinology, Diabetes and Obesity, v. 18, n. 5, p. 300-303, 2011Tradução . . Disponível em: https://doi.org/10.1097/med.0b013e32834a785c. Acesso em: 18 nov. 2024.
    • APA

      Barreto-Chaves, M. L. M., Monteiro, P. de S., & Fürstenau, C. R. (2011). Acute actions of thyroid hormone on blood vessel biochemistry and physiology. Current Opinion in Endocrinology, Diabetes and Obesity, 18( 5), 300-303. doi:10.1097/med.0b013e32834a785c
    • NLM

      Barreto-Chaves MLM, Monteiro P de S, Fürstenau CR. Acute actions of thyroid hormone on blood vessel biochemistry and physiology [Internet]. Current Opinion in Endocrinology, Diabetes and Obesity. 2011 ; 18( 5): 300-303.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1097/med.0b013e32834a785c
    • Vancouver

      Barreto-Chaves MLM, Monteiro P de S, Fürstenau CR. Acute actions of thyroid hormone on blood vessel biochemistry and physiology [Internet]. Current Opinion in Endocrinology, Diabetes and Obesity. 2011 ; 18( 5): 300-303.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1097/med.0b013e32834a785c
  • Fonte: Life Sciences (1973). Unidade: ICB

    Assunto: ANATOMIA

    Acesso à fonteDOIComo citar
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      FÜRSTENAU, Cristina Ribas et al. L-NAME-treatment alters ectonucleotidase activities in kidney membranes of rats. Life Sciences (1973), v. 87, n. 9/10, p. 325-332, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2010.07.008. Acesso em: 18 nov. 2024.
    • APA

      Fürstenau, C. R., Ramos, D. B., Vuaden, F. C., Casali, E. A., Monteiro, P. de S., Trentin, D. da S., et al. (2010). L-NAME-treatment alters ectonucleotidase activities in kidney membranes of rats. Life Sciences (1973), 87( 9/10), 325-332. doi:10.1016/j.lfs.2010.07.008
    • NLM

      Fürstenau CR, Ramos DB, Vuaden FC, Casali EA, Monteiro P de S, Trentin D da S, Gossenheimer AN, Bogo MR, Bonan CD, Barreto-Chaves MLM, Sarkis JJF, Wofchuk ST. L-NAME-treatment alters ectonucleotidase activities in kidney membranes of rats [Internet]. Life Sciences (1973). 2010 ; 87( 9/10): 325-332.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.008
    • Vancouver

      Fürstenau CR, Ramos DB, Vuaden FC, Casali EA, Monteiro P de S, Trentin D da S, Gossenheimer AN, Bogo MR, Bonan CD, Barreto-Chaves MLM, Sarkis JJF, Wofchuk ST. L-NAME-treatment alters ectonucleotidase activities in kidney membranes of rats [Internet]. Life Sciences (1973). 2010 ; 87( 9/10): 325-332.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.008
  • Fonte: Clinical and Experimental Pharmacology and Physiology. Unidade: ICB

    Assunto: ANATOMIA

    Acesso à fonteDOIComo citar
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      MOSTARDA, C. et al. Autonomic impairment after myocardial infarction: role in cardiac remodeling and mortality. Clinical and Experimental Pharmacology and Physiology, v. 37, n. 4, 2010Tradução . . Disponível em: https://doi.org/10.1111/j.1440-1681.2009.05327.x. Acesso em: 18 nov. 2024.
    • APA

      Mostarda, C., Rodrigues, B., Vane, M., Moreira, E. D., Rosa, K. T., Moraes-Silva, I. C., et al. (2010). Autonomic impairment after myocardial infarction: role in cardiac remodeling and mortality. Clinical and Experimental Pharmacology and Physiology, 37( 4). doi:10.1111/j.1440-1681.2009.05327.x
    • NLM

      Mostarda C, Rodrigues B, Vane M, Moreira ED, Rosa KT, Moraes-Silva IC, Lacchini S, Casarini DE, De Angelis K, Irigoyen MC. Autonomic impairment after myocardial infarction: role in cardiac remodeling and mortality [Internet]. Clinical and Experimental Pharmacology and Physiology. 2010 ; 37( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/j.1440-1681.2009.05327.x
    • Vancouver

      Mostarda C, Rodrigues B, Vane M, Moreira ED, Rosa KT, Moraes-Silva IC, Lacchini S, Casarini DE, De Angelis K, Irigoyen MC. Autonomic impairment after myocardial infarction: role in cardiac remodeling and mortality [Internet]. Clinical and Experimental Pharmacology and Physiology. 2010 ; 37( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/j.1440-1681.2009.05327.x
  • Fonte: Micron. Unidade: ICB

    Assunto: ANATOMIA

    Acesso à fonteDOIComo citar
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      CARVALHO, Cláudio Antonio Ferraz de e BOLDRINI, Silvia de Campos e LIBERTI, Edson Aparecido. Estimating the length density of convoluted tubular systems – II: comparative analysis using five different methods. Micron, v. 41, n. 5, p. 439-443, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.micron.2010.02.007. Acesso em: 18 nov. 2024.
    • APA

      Carvalho, C. A. F. de, Boldrini, S. de C., & Liberti, E. A. (2010). Estimating the length density of convoluted tubular systems – II: comparative analysis using five different methods. Micron, 41( 5), 439-443. doi:10.1016/j.micron.2010.02.007
    • NLM

      Carvalho CAF de, Boldrini S de C, Liberti EA. Estimating the length density of convoluted tubular systems – II: comparative analysis using five different methods [Internet]. Micron. 2010 ; 41( 5): 439-443.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.micron.2010.02.007
    • Vancouver

      Carvalho CAF de, Boldrini S de C, Liberti EA. Estimating the length density of convoluted tubular systems – II: comparative analysis using five different methods [Internet]. Micron. 2010 ; 41( 5): 439-443.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.micron.2010.02.007
  • Fonte: European Journal of Pain Supplements. Nome do evento: International Congress on Neuropathic Pain. Unidade: ICB

    Assuntos: ANATOMIA, FISIOLOGIA

    Como citar
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      GIARDINE, A. e BRITTO, Luiz Roberto Giorgetti de e CHACUR, Marucia. Involvement of neuronal nitric oxide synthase in experimental neuropathic pain in rats. European Journal of Pain Supplements. Atenas: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2010
    • APA

      Giardine, A., Britto, L. R. G. de, & Chacur, M. (2010). Involvement of neuronal nitric oxide synthase in experimental neuropathic pain in rats. European Journal of Pain Supplements. Atenas: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Giardine A, Britto LRG de, Chacur M. Involvement of neuronal nitric oxide synthase in experimental neuropathic pain in rats. European Journal of Pain Supplements. 2010 ; 4( 1): 50 res. 169.[citado 2024 nov. 18 ]
    • Vancouver

      Giardine A, Britto LRG de, Chacur M. Involvement of neuronal nitric oxide synthase in experimental neuropathic pain in rats. European Journal of Pain Supplements. 2010 ; 4( 1): 50 res. 169.[citado 2024 nov. 18 ]

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