Filtros : "Indexado na Base de dados Medline" "Britto, Luiz Roberto Giorgetti de" Removido: "COMPORTAMENTO ANIMAL" Limpar

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  • Source: International Immunopharmacology. Unidade: ICB

    Subjects: FISIOLOGIA, IMUNOLOGIA

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    • ABNT

      FERNANDES, Patricia Dias et al. Production of nitric oxide by airways neutrophils in the initial phase of murine asthma. International Immunopharmacology, v. 7, n. 1, p. 96-102, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2006.09.004. Acesso em: 01 jun. 2024.
    • APA

      Fernandes, P. D., Landgraf, R. G., Britto, L. R. G. de, & Jancar, S. (2007). Production of nitric oxide by airways neutrophils in the initial phase of murine asthma. International Immunopharmacology, 7( 1), 96-102. doi:10.1016/j.intimp.2006.09.004
    • NLM

      Fernandes PD, Landgraf RG, Britto LRG de, Jancar S. Production of nitric oxide by airways neutrophils in the initial phase of murine asthma [Internet]. International Immunopharmacology. 2007 ; 7( 1): 96-102.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.intimp.2006.09.004
    • Vancouver

      Fernandes PD, Landgraf RG, Britto LRG de, Jancar S. Production of nitric oxide by airways neutrophils in the initial phase of murine asthma [Internet]. International Immunopharmacology. 2007 ; 7( 1): 96-102.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.intimp.2006.09.004
  • Source: Neuroscience. Unidades: ICB, IQ

    Subjects: FISIOLOGIA, BIOQUÍMICA, CARCINOMA

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      RESENDE, Rodrigo Ribeiro et al. P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation. Neuroscience, v. 146, n. 3, p. 1169-1181, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2007.02.041. Acesso em: 01 jun. 2024.
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      Resende, R. R., Majumder, P., Gomes, K. N., Britto, L. R. G. de, & Ulrich, H. (2007). P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation. Neuroscience, 146( 3), 1169-1181. doi:10.1016/j.neuroscience.2007.02.041
    • NLM

      Resende RR, Majumder P, Gomes KN, Britto LRG de, Ulrich H. P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation [Internet]. Neuroscience. 2007 ; 146( 3): 1169-1181.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2007.02.041
    • Vancouver

      Resende RR, Majumder P, Gomes KN, Britto LRG de, Ulrich H. P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation [Internet]. Neuroscience. 2007 ; 146( 3): 1169-1181.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2007.02.041
  • Source: FEBS Letters. Unidade: ICB

    Subjects: FISIOLOGIA, HISTOLOGIA

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      MEDINA, Mayrin Correa et al. Dehydroepiandrosterone increases b-cell mass and improves the glucose-induced insulin secretion by pancreatic islets from aged rats. FEBS Letters, v. 580, p. 285-290, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.febslet.2005.12.014. Acesso em: 01 jun. 2024.
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      Medina, M. C., Souza, L. C., Caperuto, L. C., Anhê, G. F., Amanso, A. M., Teixeira, V. P. A., et al. (2006). Dehydroepiandrosterone increases b-cell mass and improves the glucose-induced insulin secretion by pancreatic islets from aged rats. FEBS Letters, 580, 285-290. doi:10.1016/j.febslet.2005.12.014
    • NLM

      Medina MC, Souza LC, Caperuto LC, Anhê GF, Amanso AM, Teixeira VPA, Bordin S, Carpinelli AR, Britto LRG de, Barbieri RL, Borella MI, Carvalho CR de O. Dehydroepiandrosterone increases b-cell mass and improves the glucose-induced insulin secretion by pancreatic islets from aged rats [Internet]. FEBS Letters. 2006 ; 580 285-290.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.febslet.2005.12.014
    • Vancouver

      Medina MC, Souza LC, Caperuto LC, Anhê GF, Amanso AM, Teixeira VPA, Bordin S, Carpinelli AR, Britto LRG de, Barbieri RL, Borella MI, Carvalho CR de O. Dehydroepiandrosterone increases b-cell mass and improves the glucose-induced insulin secretion by pancreatic islets from aged rats [Internet]. FEBS Letters. 2006 ; 580 285-290.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.febslet.2005.12.014
  • Source: Inflammation Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      ANJOS-VALOTTA, E. A. et al. Inhibition of tumor necrosis factor-'alfa'-induced intercellular adhesion molecule-1 expression in diabetic rats: role of insulin. Inflammation Research, v. 55, n. 1, p. 16-22, 2006Tradução . . Disponível em: https://doi.org/10.1007/s00011-005-0003-7. Acesso em: 01 jun. 2024.
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      Anjos-Valotta, E. A., Martins, J. O., Oliveira, M. A. de, Casolari, D. A., Britto, L. R. G. de, Tostes, R. de C. A., et al. (2006). Inhibition of tumor necrosis factor-'alfa'-induced intercellular adhesion molecule-1 expression in diabetic rats: role of insulin. Inflammation Research, 55( 1), 16-22. doi:10.1007/s00011-005-0003-7
    • NLM

      Anjos-Valotta EA, Martins JO, Oliveira MA de, Casolari DA, Britto LRG de, Tostes R de CA, Fortes ZB, Sannomiya P. Inhibition of tumor necrosis factor-'alfa'-induced intercellular adhesion molecule-1 expression in diabetic rats: role of insulin [Internet]. Inflammation Research. 2006 ; 55( 1): 16-22.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1007/s00011-005-0003-7
    • Vancouver

      Anjos-Valotta EA, Martins JO, Oliveira MA de, Casolari DA, Britto LRG de, Tostes R de CA, Fortes ZB, Sannomiya P. Inhibition of tumor necrosis factor-'alfa'-induced intercellular adhesion molecule-1 expression in diabetic rats: role of insulin [Internet]. Inflammation Research. 2006 ; 55( 1): 16-22.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1007/s00011-005-0003-7

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