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Artigo de periodico
Acute autonomic effects of rose oxide on cardiovascular parameters of Wistar and spontaneously hypertensive rats (2021)
Santos, Rômulo B. dos; Oliveira, Lucas V. de C.; Sena, Emerson P.; Sousa, Damião P. de; Maia Filho, Antônio L. M.; Soriano, Renato Nery; Lopes, Luciano da S.; Branco, Luiz Guilherme de Siqueira ; Oliveira, Aldeidia P. de; Salgado, Hélio César ; Sabino, João Paulo J.
Source: Life Sciences. Unidades: FORP, FMRP
Subjects: HIPERTENSÃO, SISTEMA NERVOSO AUTÔNOMO, ANTI-INFLAMATÓRIOS NÃO ESTEROIDES, BARORREFLEXO, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA
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ABNT
SANTOS, Rômulo B. dos et al. Acute autonomic effects of rose oxide on cardiovascular parameters of Wistar and spontaneously hypertensive rats. Life Sciences, v. 287, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.120107. Acesso em: 14 nov. 2024.
APA
Santos, R. B. dos, Oliveira, L. V. de C., Sena, E. P., Sousa, D. P. de, Maia Filho, A. L. M., Soriano, R. N., et al. (2021). Acute autonomic effects of rose oxide on cardiovascular parameters of Wistar and spontaneously hypertensive rats. Life Sciences, 287, 1-12. doi:10.1016/j.lfs.2021.120107
NLM
Santos RB dos, Oliveira LV de C, Sena EP, Sousa DP de, Maia Filho ALM, Soriano RN, Lopes L da S, Branco LG de S, Oliveira AP de, Salgado HC, Sabino JPJ. Acute autonomic effects of rose oxide on cardiovascular parameters of Wistar and spontaneously hypertensive rats [Internet]. Life Sciences. 2021 ; 287 1-12.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2021.120107
Vancouver
Santos RB dos, Oliveira LV de C, Sena EP, Sousa DP de, Maia Filho ALM, Soriano RN, Lopes L da S, Branco LG de S, Oliveira AP de, Salgado HC, Sabino JPJ. Acute autonomic effects of rose oxide on cardiovascular parameters of Wistar and spontaneously hypertensive rats [Internet]. Life Sciences. 2021 ; 287 1-12.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2021.120107
Artigo de periodico
Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism (2018)
Nascimento, M.; Piran, R.; Da Costa, R. M.; Giordani, M. A.; Carneiro, Fernando Silva; Aguiar, D. H.; Dias, M. C.; Sugizaki, M. M.; Luvizotto, R. A.; Nascimento, A. F.; Bomfim, G. F.
Source: Life Sciences. Unidade: FMRP
Subjects: AORTA, FÍGADO, PRESSÃO SANGUÍNEA, ENDOTÉLIO VASCULAR, DOENÇAS VASCULARES, RATOS WISTAR
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ABNT
NASCIMENTO, M. et al. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, v. 212, p. 168-175, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.09.051. Acesso em: 14 nov. 2024.
APA
Nascimento, M., Piran, R., Da Costa, R. M., Giordani, M. A., Carneiro, F. S., Aguiar, D. H., et al. (2018). Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, 212, 168-175. doi:10.1016/j.lfs.2018.09.051
NLM
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Vancouver
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Artigo de periodico
O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway (2018)
Miguez, Jéssica S. G.; Dela Justina, Vanessa; Bressan, Alecsander F. M.; Marchi, Patrícia G. F.; França, Adenilda C. Honorio; Carneiro, Fernando Silva; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes; Giachini, Fernanda R.; Lima, Victor V.
Source: Life Sciences. Unidade: FMRP
Subjects: CITOCINAS, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
MIGUEZ, Jéssica S. G. et al. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, v. 209, p. 78-84, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.07.058. Acesso em: 14 nov. 2024.
APA
Miguez, J. S. G., Dela Justina, V., Bressan, A. F. M., Marchi, P. G. F., França, A. C. H., Carneiro, F. S., et al. (2018). O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, 209, 78-84. doi:10.1016/j.lfs.2018.07.058
NLM
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Vancouver
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Artigo de periodico
O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels (2018)
Dela Justina, Vanessa; Passos Junior, Rinaldo R. dos; Bressan, Alecsander F.; Passaglia, Rita de Cássia Aleixo Tostes; Carneiro, Fernando Silva; Soares, Thaigra S.; Volpato, Gustavo T.; Lima, Victor Vitorino; Martin, Sebastian San; Giachini, Fernanda R.
Source: Life Sciences. Unidade: FMRP
Subjects: GRAVIDEZ, HIPERGLICEMIA, TROFOBLASTO, CÉLULAS ENDOTELIAIS, RATOS
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ABNT
DELA JUSTINA, Vanessa et al. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, v. 205, p. 18-25, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.05.013. Acesso em: 14 nov. 2024.
APA
Dela Justina, V., Passos Junior, R. R. dos, Bressan, A. F., Passaglia, R. de C. A. T., Carneiro, F. S., Soares, T. S., et al. (2018). O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, 205, 18-25. doi:10.1016/j.lfs.2018.05.013
NLM
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Vancouver
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Artigo de periodico
Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats (2018)
Riberio, Diogo Farias; Cella, Paola Sanchez; Silva, Lilian Eslaine Costa Mendes da; Jordão Júnior, Alceu Afonso; Deminice, Rafael
Source: Life Sciences. Unidade: FMRP
Subjects: EXERCÍCIO FÍSICO, RATOS, PESOS E MEDIDAS CORPORAIS, FISIOLOGIA ANIMAL
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ABNT
RIBERIO, Diogo Farias et al. Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats. Life Sciences, v. 196, p. 63-68, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.01.003. Acesso em: 14 nov. 2024.
APA
Riberio, D. F., Cella, P. S., Silva, L. E. C. M. da, Jordão Júnior, A. A., & Deminice, R. (2018). Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats. Life Sciences, 196, 63-68. doi:10.1016/j.lfs.2018.01.003
NLM
Riberio DF, Cella PS, Silva LECM da, Jordão Júnior AA, Deminice R. Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats [Internet]. Life Sciences. 2018 ; 196 63-68.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.01.003
Vancouver
Riberio DF, Cella PS, Silva LECM da, Jordão Júnior AA, Deminice R. Acute exercise alters homocysteine plasma concentration in an intensity-dependent manner due increased methyl flux in liver of rats [Internet]. Life Sciences. 2018 ; 196 63-68.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.01.003
Artigo de periodico
Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids (2017)
Diniz, Mariana C.; Olivon, Vania C.; Tavares, Lívia D.; Simplicio, Janaina A.; Gonzaga, Natália A.; Souza, Daniele G. de; Bendhack, Lusiane Maria; Tirapelli, Carlos Renato ; Bonaventura, Daniella
Source: Life Sciences. Unidades: FCFRP, EERP
Subjects: NITRATOS, AORTA, VASODILATAÇÃO, OXIGÊNIO
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ABNT
DINIZ, Mariana C. et al. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, v. 176, p. 26-34, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.03.016. Acesso em: 14 nov. 2024.
APA
Diniz, M. C., Olivon, V. C., Tavares, L. D., Simplicio, J. A., Gonzaga, N. A., Souza, D. G. de, et al. (2017). Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, 176, 26-34. doi:10.1016/j.lfs.2017.03.016
NLM
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Vancouver
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Artigo de periodico
Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles (2016)
Pereira, Bruno C.; Rocha, Alisson L. da; Pinto, Ana P.; Pauli, José R.; Souza, Claudio T. de; Cintra, Dennys E.; Ropelle, Eduardo R.; Freitas, Ellen Cristini de ; Zagatto, Alessandro M.; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidade: EEFERP
Subjects: EXERCÍCIO FÍSICO, ESTRESSE, MÚSCULO ESQUELÉTICO, ANIMAIS DE LABORATÓRIO
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ABNT
PEREIRA, Bruno C. et al. Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles. Life Sciences, v. 145, p. 144-151, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.12.037. Acesso em: 14 nov. 2024.
APA
Pereira, B. C., Rocha, A. L. da, Pinto, A. P., Pauli, J. R., Souza, C. T. de, Cintra, D. E., et al. (2016). Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles. Life Sciences, 145, 144-151. doi:10.1016/j.lfs.2015.12.037
NLM
Pereira BC, Rocha AL da, Pinto AP, Pauli JR, Souza CT de, Cintra DE, Ropelle ER, Freitas EC de, Zagatto AM, Silva ASR da. Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles [Internet]. Life Sciences. 2016 ; 145 144-151.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.037
Vancouver
Pereira BC, Rocha AL da, Pinto AP, Pauli JR, Souza CT de, Cintra DE, Ropelle ER, Freitas EC de, Zagatto AM, Silva ASR da. Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles [Internet]. Life Sciences. 2016 ; 145 144-151.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.037
Artigo de periodico
Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet (2015)
Oharomari, Leandro Kansuke; Garcia, Nádia Fagundes; Freitas, Ellen Cristini de ; Jordão Júnior, Alceu Afonso; Ovidio, Paula Payão; Maia, Aline Rosa; Davel, Ana Paula; Moraes, Camila de
Source: Life Sciences. Unidades: EEFERP, FMRP
Subjects: EXERCÍCIO FÍSICO, ENDOTÉLIO, OBESIDADE, ESTRESSE OXIDATIVO, DIETA, SISTEMA CARDIOVASCULAR
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ABNT
OHAROMARI, Leandro Kansuke et al. Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet. Life Sciences, v. 139, p. 91-96, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.08.015. Acesso em: 14 nov. 2024.
APA
Oharomari, L. K., Garcia, N. F., Freitas, E. C. de, Jordão Júnior, A. A., Ovidio, P. P., Maia, A. R., et al. (2015). Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet. Life Sciences, 139, 91-96. doi:10.1016/j.lfs.2015.08.015
NLM
Oharomari LK, Garcia NF, Freitas EC de, Jordão Júnior AA, Ovidio PP, Maia AR, Davel AP, Moraes C de. Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet [Internet]. Life Sciences. 2015 ; 139 91-96.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2015.08.015
Vancouver
Oharomari LK, Garcia NF, Freitas EC de, Jordão Júnior AA, Ovidio PP, Maia AR, Davel AP, Moraes C de. Exercise training and taurine supplementation reduce oxidative stress and prevent endothelium dysfunction in rats fed a highly palatable diet [Internet]. Life Sciences. 2015 ; 139 91-96.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2015.08.015
Artigo de periodico
Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet (2014)
Deminice, Rafael; Rosa, Flávia Troncon; Silva, Lilian Eslaine Costa Mendes da; Jordão Júnior, Alceu Afonso
Source: Life Sciences. Unidade: FMRP
Subjects: SUPLEMENTOS DIETÉTICOS, DEFICIÊNCIA DE PROTEÍNA (PATOLOGIA), EXPRESSÃO GÊNICA (EFEITOS DE DROGAS), FÍGADO (EFEITOS DE DROGAS)
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ABNT
DEMINICE, Rafael et al. Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet. Life Sciences, v. 105, n. 1-2, p. 43-47, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.04.015. Acesso em: 14 nov. 2024.
APA
Deminice, R., Rosa, F. T., Silva, L. E. C. M. da, & Jordão Júnior, A. A. (2014). Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet. Life Sciences, 105( 1-2), 43-47. doi:10.1016/j.lfs.2014.04.015
NLM
Deminice R, Rosa FT, Silva LECM da, Jordão Júnior AA. Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet [Internet]. Life Sciences. 2014 ; 105( 1-2): 43-47.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2014.04.015
Vancouver
Deminice R, Rosa FT, Silva LECM da, Jordão Júnior AA. Taurine supplementation does not decrease homocysteine levels and liver injury induced by a choline-deficient diet [Internet]. Life Sciences. 2014 ; 105( 1-2): 43-47.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2014.04.015
Artigo de periodico
Functional and molecular effects of imidazoline receptor activation in heart failure (2011)
Stabile, Angelita Maria ; Aceros, Henry; Stockmeyer, Kim; Rahman, Abdel A. Abdel; Noiseux, Nicolas; Mukaddam-Daher, Suhayla
Source: Life Sciences. Unidade: EERP
Subjects: INSUFICIÊNCIA CARDÍACA, ECOCARDIOGRAFIA, APOPTOSE
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ABNT
STABILE, Angelita Maria et al. Functional and molecular effects of imidazoline receptor activation in heart failure. Life Sciences, v. 88, n. 11-12, p. 493-503, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2011.01.008. Acesso em: 14 nov. 2024.
APA
Stabile, A. M., Aceros, H., Stockmeyer, K., Rahman, A. A. A., Noiseux, N., & Mukaddam-Daher, S. (2011). Functional and molecular effects of imidazoline receptor activation in heart failure. Life Sciences, 88( 11-12), 493-503. doi:10.1016/j.lfs.2011.01.008
NLM
Stabile AM, Aceros H, Stockmeyer K, Rahman AAA, Noiseux N, Mukaddam-Daher S. Functional and molecular effects of imidazoline receptor activation in heart failure [Internet]. Life Sciences. 2011 ; 88( 11-12): 493-503.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2011.01.008
Vancouver
Stabile AM, Aceros H, Stockmeyer K, Rahman AAA, Noiseux N, Mukaddam-Daher S. Functional and molecular effects of imidazoline receptor activation in heart failure [Internet]. Life Sciences. 2011 ; 88( 11-12): 493-503.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2011.01.008
Artigo de periodico
NO donors-relaxation in impaired in aorta from hypertensive rats due to a reduced involvement of K+ channels and sarcoplasmic reticulum 'Ca IND. 2+'_ATPase (2011)
Bonaventura, Daniella; Lima, Renata Galvão de; Silva, Roberto Santana da; Bendhack, Lusiane Maria
Source: Life Sciences. Unidade: FCFRP
Subjects: ÓXIDO NÍTRICO, HIPERTENSÃO
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ABNT
BONAVENTURA, Daniella et al. NO donors-relaxation in impaired in aorta from hypertensive rats due to a reduced involvement of K+ channels and sarcoplasmic reticulum 'Ca IND. 2+'_ATPase. Life Sciences, v. 89, n. 17-18, p. 595-602, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2011.07.022. Acesso em: 14 nov. 2024.
APA
Bonaventura, D., Lima, R. G. de, Silva, R. S. da, & Bendhack, L. M. (2011). NO donors-relaxation in impaired in aorta from hypertensive rats due to a reduced involvement of K+ channels and sarcoplasmic reticulum 'Ca IND. 2+'_ATPase. Life Sciences, 89( 17-18), 595-602. doi:10.1016/j.lfs.2011.07.022
NLM
Bonaventura D, Lima RG de, Silva RS da, Bendhack LM. NO donors-relaxation in impaired in aorta from hypertensive rats due to a reduced involvement of K+ channels and sarcoplasmic reticulum 'Ca IND. 2+'_ATPase [Internet]. Life Sciences. 2011 ; 89( 17-18): 595-602.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2011.07.022
Vancouver
Bonaventura D, Lima RG de, Silva RS da, Bendhack LM. NO donors-relaxation in impaired in aorta from hypertensive rats due to a reduced involvement of K+ channels and sarcoplasmic reticulum 'Ca IND. 2+'_ATPase [Internet]. Life Sciences. 2011 ; 89( 17-18): 595-602.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2011.07.022
Artigo de periodico
Altered expression of connexins in urethane-induced mouse lung adenomas (2006)
Avanzo, José Luis; Mesnil, Marc; Hernandez-Blazquez, Francisco Javier ; Silva, Tereza Cristina da; Fukumasu, Heidge; Mori, Claudia Madalena Cabrera ; Yamasaki, Hiroshi; Dagli, Maria Lúcia Zaidan
Source: Life Sciences. Unidade: FMVZ
Subjects: CARCINOGÊNESE ANIMAL, REAÇÃO EM CADEIA POR POLIMERASE, RATOS
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ABNT
AVANZO, José Luis et al. Altered expression of connexins in urethane-induced mouse lung adenomas. Life Sciences, v. No 2006, n. 23, p. 2202-2208, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2006.07.033. Acesso em: 14 nov. 2024.
APA
Avanzo, J. L., Mesnil, M., Hernandez-Blazquez, F. J., Silva, T. C. da, Fukumasu, H., Mori, C. M. C., et al. (2006). Altered expression of connexins in urethane-induced mouse lung adenomas. Life Sciences, No 2006( 23), 2202-2208. doi:10.1016/j.lfs.2006.07.033
NLM
Avanzo JL, Mesnil M, Hernandez-Blazquez FJ, Silva TC da, Fukumasu H, Mori CMC, Yamasaki H, Dagli MLZ. Altered expression of connexins in urethane-induced mouse lung adenomas [Internet]. Life Sciences. 2006 ; No 2006( 23): 2202-2208.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2006.07.033
Vancouver
Avanzo JL, Mesnil M, Hernandez-Blazquez FJ, Silva TC da, Fukumasu H, Mori CMC, Yamasaki H, Dagli MLZ. Altered expression of connexins in urethane-induced mouse lung adenomas [Internet]. Life Sciences. 2006 ; No 2006( 23): 2202-2208.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2006.07.033

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