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Trabalho de evento-resumo periodico
Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis (2014)
Olivon, Vânia Cláudia; Ferreira, Raphael G.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENZIMAS, SEPSE, PRESSÃO SANGUÍNEA
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ABNT
OLIVON, Vânia Cláudia et al. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8. Acesso em: 12 jun. 2024. , 2014
APA
Olivon, V. C., Ferreira, R. G., Mestriner, F. L. A. C., Alves Filho, J. C. F., Cunha, F. de Q., & Passaglia, R. de C. A. T. (2014). Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
NLM
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
Vancouver
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
Trabalho de evento-resumo periodico
Downregulation of vascular factor-erythroid 2-related factor-2 and associated antioxidant enzymes in hypertension (2014)
Lopes, Rheure A.; Neves, Karla B.; Montezano, Augusto; Passaglia, Rita de Cassia Aleixo Tostes; Touyz, Rhian
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: HIPERTENSÃO, ANGIOTENSINA II, ESTRESSE OXIDATIVO
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ABNT
LOPES, Rheure A. et al. Downregulation of vascular factor-erythroid 2-related factor-2 and associated antioxidant enzymes in hypertension. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A067.abstract?sid=55a5cbeb-11f8-41eb-96b5-1ce5c09bae04. Acesso em: 12 jun. 2024. , 2014
APA
Lopes, R. A., Neves, K. B., Montezano, A., Passaglia, R. de C. A. T., & Touyz, R. (2014). Downregulation of vascular factor-erythroid 2-related factor-2 and associated antioxidant enzymes in hypertension. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A067.abstract?sid=55a5cbeb-11f8-41eb-96b5-1ce5c09bae04
NLM
Lopes RA, Neves KB, Montezano A, Passaglia R de CAT, Touyz R. Downregulation of vascular factor-erythroid 2-related factor-2 and associated antioxidant enzymes in hypertension [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A067.abstract?sid=55a5cbeb-11f8-41eb-96b5-1ce5c09bae04
Vancouver
Lopes RA, Neves KB, Montezano A, Passaglia R de CAT, Touyz R. Downregulation of vascular factor-erythroid 2-related factor-2 and associated antioxidant enzymes in hypertension [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A067.abstract?sid=55a5cbeb-11f8-41eb-96b5-1ce5c09bae04
Trabalho de evento-resumo periodico
Castration impairs rat internal pudendal artery reactivity and structure (2014)
Lopes, Rheure A.; Neves, Karla B.; Silva, Marcondes A.; Olivon, Vânia Cláudia; Ruginsk, Silvia; Ramalho, Leandra Naíra Zambelli; Rodrigues, José Antunes; Passaglia, Rita de Cassia Aleixo Tostes; Carneiro, Fernando Silva
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ARTÉRIAS, CASTRAÇÃO ANIMAL
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ABNT
LOPES, Rheure A. et al. Castration impairs rat internal pudendal artery reactivity and structure. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A353.abstract?sid=26a4ece4-6e9d-4308-afef-b01e4d258004. Acesso em: 12 jun. 2024. , 2014
APA
Lopes, R. A., Neves, K. B., Silva, M. A., Olivon, V. C., Ruginsk, S., Ramalho, L. N. Z., et al. (2014). Castration impairs rat internal pudendal artery reactivity and structure. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A353.abstract?sid=26a4ece4-6e9d-4308-afef-b01e4d258004
NLM
Lopes RA, Neves KB, Silva MA, Olivon VC, Ruginsk S, Ramalho LNZ, Rodrigues JA, Passaglia R de CAT, Carneiro FS. Castration impairs rat internal pudendal artery reactivity and structure [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A353.abstract?sid=26a4ece4-6e9d-4308-afef-b01e4d258004
Vancouver
Lopes RA, Neves KB, Silva MA, Olivon VC, Ruginsk S, Ramalho LNZ, Rodrigues JA, Passaglia R de CAT, Carneiro FS. Castration impairs rat internal pudendal artery reactivity and structure [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A353.abstract?sid=26a4ece4-6e9d-4308-afef-b01e4d258004
Trabalho de evento-resumo periodico
The baroreflex modulates the anti-inflammatory response caused by intravenous lipopolysaccharide (lps) (2014)
Brognara, Fernanda; Becari, Christiane; Castania, Jaci Airton; Dias, Daniel Penteado Martins; Tezini, Geisa Cristina Singolani Vaccari; Lopes, Alexandre; Cunha, Thiago Mattar; Fazan Júnior, Rubens; Salgado, Hélio Cesar
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: FISIOLOGIA DO SISTEMA NERVOSO, ESTIMULAÇÃO ELÉTRICA
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ABNT
BROGNARA, Fernanda et al. The baroreflex modulates the anti-inflammatory response caused by intravenous lipopolysaccharide (lps). Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A458.abstract?sid=3f2fea4b-240d-43ea-992d-cd1f51fe85bc. Acesso em: 12 jun. 2024. , 2014
APA
Brognara, F., Becari, C., Castania, J. A., Dias, D. P. M., Tezini, G. C. S. V., Lopes, A., et al. (2014). The baroreflex modulates the anti-inflammatory response caused by intravenous lipopolysaccharide (lps). Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A458.abstract?sid=3f2fea4b-240d-43ea-992d-cd1f51fe85bc
NLM
Brognara F, Becari C, Castania JA, Dias DPM, Tezini GCSV, Lopes A, Cunha TM, Fazan Júnior R, Salgado HC. The baroreflex modulates the anti-inflammatory response caused by intravenous lipopolysaccharide (lps) [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A458.abstract?sid=3f2fea4b-240d-43ea-992d-cd1f51fe85bc
Vancouver
Brognara F, Becari C, Castania JA, Dias DPM, Tezini GCSV, Lopes A, Cunha TM, Fazan Júnior R, Salgado HC. The baroreflex modulates the anti-inflammatory response caused by intravenous lipopolysaccharide (lps) [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A458.abstract?sid=3f2fea4b-240d-43ea-992d-cd1f51fe85bc
Trabalho de evento-resumo periodico
G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction (2014)
Ferreira, Nathanne dos Santos; Cau, Stefany B.; Manzato, Carla P.; Silva, Marcondes A.; Carneiro, Fernando Silva; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENDOTÉLIO VASCULAR, DIABETES MELLITUS
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ABNT
FERREIRA, Nathanne dos Santos et al. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366. Acesso em: 12 jun. 2024. , 2014
APA
Ferreira, N. dos S., Cau, S. B., Manzato, C. P., Silva, M. A., Carneiro, F. S., & Passaglia, R. de C. A. T. (2014). G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366
NLM
Ferreira N dos S, Cau SB, Manzato CP, Silva MA, Carneiro FS, Passaglia R de CAT. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366
Vancouver
Ferreira N dos S, Cau SB, Manzato CP, Silva MA, Carneiro FS, Passaglia R de CAT. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366
Trabalho de evento-resumo periodico
Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation (2014)
Pereira, Camila A.; Ferreira, Nathanne dos Santos; Mestriner, Fabiola Leslie Antunes Cardoso; Resstel, Leonardo Barbosa Moraes; Carneiro, Fernando Silva; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ANTIDEPRESSIVOS, SISTEMA CARDIOVASCULAR, ARTÉRIAS (ALTERAÇÃO)
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ABNT
PEREIRA, Camila A. et al. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d. Acesso em: 12 jun. 2024. , 2014
APA
Pereira, C. A., Ferreira, N. dos S., Mestriner, F. L. A. C., Resstel, L. B. M., Carneiro, F. S., & Passaglia, R. de C. A. T. (2014). Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d
NLM
Pereira CA, Ferreira N dos S, Mestriner FLAC, Resstel LBM, Carneiro FS, Passaglia R de CAT. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d
Vancouver
Pereira CA, Ferreira N dos S, Mestriner FLAC, Resstel LBM, Carneiro FS, Passaglia R de CAT. Chronic treatment with fluoxetine increases relaxation of rat resistance mesenteric arteries via Atp-sensitive potassium channels activation [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A339.abstract?sid=ceb883c1-c438-4e11-82fc-f6e6ca97987d
Trabalho de evento-resumo periodico
Carotid chemoreceptor activation blunts the hypotensive response caused by the electrical stimulation of the carotid sinus in conscious rats (2014)
Katayama, Pedro Lourenço; Castania, Jaci Airton; Fazan Júnior, Rubens; Salgado, Hélio Cesar
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ARTÉRIAS CARÓTIDAS, FISIOLOGIA DO SISTEMA NERVOSO
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ABNT
KATAYAMA, Pedro Lourenço et al. Carotid chemoreceptor activation blunts the hypotensive response caused by the electrical stimulation of the carotid sinus in conscious rats. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A032.abstract?sid=acb4fb67-ff0c-4aa8-8613-94f91ce8512f. Acesso em: 12 jun. 2024. , 2014
APA
Katayama, P. L., Castania, J. A., Fazan Júnior, R., & Salgado, H. C. (2014). Carotid chemoreceptor activation blunts the hypotensive response caused by the electrical stimulation of the carotid sinus in conscious rats. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A032.abstract?sid=acb4fb67-ff0c-4aa8-8613-94f91ce8512f
NLM
Katayama PL, Castania JA, Fazan Júnior R, Salgado HC. Carotid chemoreceptor activation blunts the hypotensive response caused by the electrical stimulation of the carotid sinus in conscious rats [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A032.abstract?sid=acb4fb67-ff0c-4aa8-8613-94f91ce8512f
Vancouver
Katayama PL, Castania JA, Fazan Júnior R, Salgado HC. Carotid chemoreceptor activation blunts the hypotensive response caused by the electrical stimulation of the carotid sinus in conscious rats [Internet]. Hypertension. 2014 ; 64[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A032.abstract?sid=acb4fb67-ff0c-4aa8-8613-94f91ce8512f
Artigo de periodico
Specific respiratory neuron types have increased excitability that drive presympathetic neurones in neurogenic hypertension (2014)
Moraes, Davi José de Almeida; Machado, Benedito Honório; Paton, Julian F. R.
Source: Hypertension. Unidade: FMRP
Subjects: POTENCIAIS DE AÇÃO (FISIOLOGIA), HIPERTENSÃO (FISIOPATOLOGIA), NERVO FRÊNICO, SISTEMA NERVOSO SIMPÁTICO
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ABNT
MORAES, Davi José de Almeida e MACHADO, Benedito Honório e PATON, Julian F. R. Specific respiratory neuron types have increased excitability that drive presympathetic neurones in neurogenic hypertension. Hypertension, v. 63, n. 6, p. 1309-1318, 2014Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.113.02283. Acesso em: 12 jun. 2024.
APA
Moraes, D. J. de A., Machado, B. H., & Paton, J. F. R. (2014). Specific respiratory neuron types have increased excitability that drive presympathetic neurones in neurogenic hypertension. Hypertension, 63( 6), 1309-1318. doi:10.1161/HYPERTENSIONAHA.113.02283
NLM
Moraes DJ de A, Machado BH, Paton JFR. Specific respiratory neuron types have increased excitability that drive presympathetic neurones in neurogenic hypertension [Internet]. Hypertension. 2014 ; 63( 6): 1309-1318.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.113.02283
Vancouver
Moraes DJ de A, Machado BH, Paton JFR. Specific respiratory neuron types have increased excitability that drive presympathetic neurones in neurogenic hypertension [Internet]. Hypertension. 2014 ; 63( 6): 1309-1318.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.113.02283
Trabalho de evento-resumo periodico
Blood Pressure Variability: An Independent Marker to Morbimortality (2013)
Paulini, Janaina; Nishi, Fernanda; Jorge, Luciana; Pires, Romulo; Krieger, Eduardo Moacir; Irigoyen, Maria Claudia
Source: Hypertension. Conference titles: American Heart Association's High Blood Pressure Research. Unidade: FM
Subjects: HIPERTENSÃO (FISIOPATOLOGIA), INDICADORES DE MORBI-MORTALIDADE, PRESSÃO SANGUÍNEA
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ABNT
PAULINI, Janaina et al. Blood Pressure Variability: An Independent Marker to Morbimortality. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 12 jun. 2024. , 2013
APA
Paulini, J., Nishi, F., Jorge, L., Pires, R., Krieger, E. M., & Irigoyen, M. C. (2013). Blood Pressure Variability: An Independent Marker to Morbimortality. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo.
NLM
Paulini J, Nishi F, Jorge L, Pires R, Krieger EM, Irigoyen MC. Blood Pressure Variability: An Independent Marker to Morbimortality. Hypertension. 2013 ; 62( suppl. 3): 195 res. P-392.[citado 2024 jun. 12 ]
Vancouver
Paulini J, Nishi F, Jorge L, Pires R, Krieger EM, Irigoyen MC. Blood Pressure Variability: An Independent Marker to Morbimortality. Hypertension. 2013 ; 62( suppl. 3): 195 res. P-392.[citado 2024 jun. 12 ]
Artigo de periodico
Angiotensin II-independent angiotensin-(1-7) formation in rat hippocampus: involvement of thimet oligopeptidase (2013)
Pereira, Marilia G. A. G.; Souza, Laura L.; Becari, Christiane; Duarte, Diego A.; Camacho, Fabio R. B.; Oliveira, José Antonio Cortes de; Gomes, Marcelo Damario; Oliveira, Eduardo Brandt de; Salgado, Maria Cristina de Oliveira; Garcia-Cairasco, Norberto; Costa Neto, Cláudio Miguel
Source: Hypertension. Unidade: FMRP
Subjects: EPILEPSIA, ANGIOTENSINAS, SISTEMA RENINA-ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Marilia G. A. G. et al. Angiotensin II-independent angiotensin-(1-7) formation in rat hippocampus: involvement of thimet oligopeptidase. Hypertension, v. 62, n. 5, p. 879-885, 2013Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.113.01613. Acesso em: 12 jun. 2024.
APA
Pereira, M. G. A. G., Souza, L. L., Becari, C., Duarte, D. A., Camacho, F. R. B., Oliveira, J. A. C. de, et al. (2013). Angiotensin II-independent angiotensin-(1-7) formation in rat hippocampus: involvement of thimet oligopeptidase. Hypertension, 62( 5), 879-885. doi:10.1161/HYPERTENSIONAHA.113.01613
NLM
Pereira MGAG, Souza LL, Becari C, Duarte DA, Camacho FRB, Oliveira JAC de, Gomes MD, Oliveira EB de, Salgado MC de O, Garcia-Cairasco N, Costa Neto CM. Angiotensin II-independent angiotensin-(1-7) formation in rat hippocampus: involvement of thimet oligopeptidase [Internet]. Hypertension. 2013 ; 62( 5): 879-885.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.113.01613
Vancouver
Pereira MGAG, Souza LL, Becari C, Duarte DA, Camacho FRB, Oliveira JAC de, Gomes MD, Oliveira EB de, Salgado MC de O, Garcia-Cairasco N, Costa Neto CM. Angiotensin II-independent angiotensin-(1-7) formation in rat hippocampus: involvement of thimet oligopeptidase [Internet]. Hypertension. 2013 ; 62( 5): 879-885.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.113.01613
Artigo de periodico
P2Y1 receptors expressed by C1 neurons determine peripheral chemoreceptor modulation of breathing, sympathetic activity, and blood pressure (2013)
Wenker, Ian C.; Roberto Sobrinho, Cleyton; Takakura, Ana Carolina Thomaz; Mulkey, Daniel K.; Moreira, Thiago dos Santos
Source: Hypertension. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WENKER, Ian C. et al. P2Y1 receptors expressed by C1 neurons determine peripheral chemoreceptor modulation of breathing, sympathetic activity, and blood pressure. Hypertension, v. 62, n. 2, p. 263-273, 2013Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.113.01487. Acesso em: 12 jun. 2024.
APA
Wenker, I. C., Roberto Sobrinho, C., Takakura, A. C. T., Mulkey, D. K., & Moreira, T. dos S. (2013). P2Y1 receptors expressed by C1 neurons determine peripheral chemoreceptor modulation of breathing, sympathetic activity, and blood pressure. Hypertension, 62( 2), 263-273. doi:10.1161/HYPERTENSIONAHA.113.01487
NLM
Wenker IC, Roberto Sobrinho C, Takakura ACT, Mulkey DK, Moreira T dos S. P2Y1 receptors expressed by C1 neurons determine peripheral chemoreceptor modulation of breathing, sympathetic activity, and blood pressure [Internet]. Hypertension. 2013 ; 62( 2): 263-273.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.113.01487
Vancouver
Wenker IC, Roberto Sobrinho C, Takakura ACT, Mulkey DK, Moreira T dos S. P2Y1 receptors expressed by C1 neurons determine peripheral chemoreceptor modulation of breathing, sympathetic activity, and blood pressure [Internet]. Hypertension. 2013 ; 62( 2): 263-273.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.113.01487
Artigo de periodico
Medullary respiratory network drives sympathetic overactivity and hypertension in rats submitted to chronic intermittent hypoxia (2012)
Moraes, Davi J. A.; Zoccal, Daniel B.; Machado, Benedito Honório
Source: Hypertension. Unidade: FMRP
Subjects: SISTEMA RESPIRATÓRIO, HIPERTENSÃO, RATOS (EXPERIMENTOS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAES, Davi J. A. e ZOCCAL, Daniel B. e MACHADO, Benedito Honório. Medullary respiratory network drives sympathetic overactivity and hypertension in rats submitted to chronic intermittent hypoxia. Hypertension, v. 60, n. 6, p. 1374-1380, 2012Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.111.189332. Acesso em: 12 jun. 2024.
APA
Moraes, D. J. A., Zoccal, D. B., & Machado, B. H. (2012). Medullary respiratory network drives sympathetic overactivity and hypertension in rats submitted to chronic intermittent hypoxia. Hypertension, 60( 6), 1374-1380. doi:10.1161/HYPERTENSIONAHA.111.189332
NLM
Moraes DJA, Zoccal DB, Machado BH. Medullary respiratory network drives sympathetic overactivity and hypertension in rats submitted to chronic intermittent hypoxia [Internet]. Hypertension. 2012 ; 60( 6): 1374-1380.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.189332
Vancouver
Moraes DJA, Zoccal DB, Machado BH. Medullary respiratory network drives sympathetic overactivity and hypertension in rats submitted to chronic intermittent hypoxia [Internet]. Hypertension. 2012 ; 60( 6): 1374-1380.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.189332
Artigo de periodico
Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways (2012)
Chignalia, Andreia Zago; Schuldt, Elke Z.; Camargo, Lívia de Lucca; Montezano, Augusto C.; Callera, Gláucia E.; Laurindo, Francisco R.; Lopes, Lucia Rossetti ; Avellar, Maria Cristina W.; Carvalho, Maria Helena Catelli; Fortes, Zuleica Bruno; Touyz, Rhian M.; Passaglia, Rita de Cássia Aleixo Tostes
Source: Hypertension. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIGNALIA, Andreia Zago et al. Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways. Hypertension, v. 59, n. 6, p. 1263-1271, 2012Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.111.180620. Acesso em: 12 jun. 2024.
APA
Chignalia, A. Z., Schuldt, E. Z., Camargo, L. de L., Montezano, A. C., Callera, G. E., Laurindo, F. R., et al. (2012). Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways. Hypertension, 59( 6), 1263-1271. doi:10.1161/HYPERTENSIONAHA.111.180620
NLM
Chignalia AZ, Schuldt EZ, Camargo L de L, Montezano AC, Callera GE, Laurindo FR, Lopes LR, Avellar MCW, Carvalho MHC, Fortes ZB, Touyz RM, Passaglia R de CAT. Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways [Internet]. Hypertension. 2012 ; 59( 6): 1263-1271.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.180620
Vancouver
Chignalia AZ, Schuldt EZ, Camargo L de L, Montezano AC, Callera GE, Laurindo FR, Lopes LR, Avellar MCW, Carvalho MHC, Fortes ZB, Touyz RM, Passaglia R de CAT. Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways [Internet]. Hypertension. 2012 ; 59( 6): 1263-1271.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.180620
Artigo de periodico
Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients (2012)
Alves, M. J. N. N.; Santos, Marcelo Rodrigues dos; Nobre, Thais Simoes; Martinez, D. G.; Pereira Barretto, Antonio Carlos; Brum, Patrícia Chakur ; Rondon, Maria Urbana Pinto Brandão ; Middlekauff, Holly R.; Negrão, Carlos Eduardo
Source: Hypertension. Unidades: FM, EEFE
Subjects: INSUFICIÊNCIA CARDÍACA, SISTEMA NERVOSO SIMPÁTICO, VASODILATAÇÃO, HIPERTENSÃO, ENDOTÉLIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALVES, M. J. N. N. et al. Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients. Hypertension, v. 60, n. 3, p. 669-676, 2012Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.112.195776. Acesso em: 12 jun. 2024.
APA
Alves, M. J. N. N., Santos, M. R. dos, Nobre, T. S., Martinez, D. G., Pereira Barretto, A. C., Brum, P. C., et al. (2012). Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients. Hypertension, 60( 3), 669-676. doi:10.1161/HYPERTENSIONAHA.112.195776
NLM
Alves MJNN, Santos MR dos, Nobre TS, Martinez DG, Pereira Barretto AC, Brum PC, Rondon MUPB, Middlekauff HR, Negrão CE. Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients [Internet]. Hypertension. 2012 ; 60( 3): 669-676.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.112.195776
Vancouver
Alves MJNN, Santos MR dos, Nobre TS, Martinez DG, Pereira Barretto AC, Brum PC, Rondon MUPB, Middlekauff HR, Negrão CE. Mechanisms of blunted muscle vasodilation during peripheral chemoreceptor stimulation in heart failure patients [Internet]. Hypertension. 2012 ; 60( 3): 669-676.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.112.195776
Artigo de periodico
Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7) (2011)
Fernandes, Tiago; Hashimoto, Nara Y.; Magalhaes, Flavio C.; Fernandes, Fernanda B.; Casarini, Dulce E.; Carmona, Adriana K.; Krieger, Jose E.; Phillips, M. Ian; Oliveira, E. M.
Source: Hypertension. Unidades: FM, EEFE
Subjects: TREINAMENTO AERÓBICO, VENTRÍCULO CARDÍACO, SISTEMA RENINA-ANGIOTENSINA (ENZIMIOLOGIA), MIOCARDIOPATIA HIPERTRÓFICA (ENZIMIOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERNANDES, Tiago et al. Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7). Hypertension, v. 58, n. 2, p. 182-189, 2011Tradução . . Disponível em: http://hyper.ahajournals.org/content/early/2011/06/27/HYPERTENSIONAHA.110.168252.full.pdf+html. Acesso em: 12 jun. 2024.
APA
Fernandes, T., Hashimoto, N. Y., Magalhaes, F. C., Fernandes, F. B., Casarini, D. E., Carmona, A. K., et al. (2011). Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7). Hypertension, 58( 2), 182-189. doi:10.1161/HYPERTENSIONAHA.110.168252
NLM
Fernandes T, Hashimoto NY, Magalhaes FC, Fernandes FB, Casarini DE, Carmona AK, Krieger JE, Phillips MI, Oliveira EM. Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7) [Internet]. Hypertension. 2011 ; 58( 2): 182-189.[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/early/2011/06/27/HYPERTENSIONAHA.110.168252.full.pdf+html
Vancouver
Fernandes T, Hashimoto NY, Magalhaes FC, Fernandes FB, Casarini DE, Carmona AK, Krieger JE, Phillips MI, Oliveira EM. Aerobic Exercise Training-Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7) [Internet]. Hypertension. 2011 ; 58( 2): 182-189.[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/early/2011/06/27/HYPERTENSIONAHA.110.168252.full.pdf+html
Artigo de periodico
Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients (2011)
Martinez, Daniel G.; Nicolau, José C.; Lage, Rony Lopes; Toschi-Dias, Edgar; de Matos, Luciana D. N. J.; Alves, Maria Janieire N. N.; Trombetta, Ivani C.; Dias da Silva, Valdo J.; Middlekauff, Holly R.; Negrão, C. E.; Rondon, M. U.
Source: Hypertension. Unidades: FM, EEFE
Subjects: INFARTO DO MIÓCARDIO, TREINAMNETO FÍSICO, SEGUIMENTOS, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINEZ, Daniel G. et al. Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients. Hypertension, v. 58, n. 6, p. 1049-1056, 2011Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.111.176644. Acesso em: 12 jun. 2024.
APA
Martinez, D. G., Nicolau, J. C., Lage, R. L., Toschi-Dias, E., de Matos, L. D. N. J., Alves, M. J. N. N., et al. (2011). Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients. Hypertension, 58( 6), 1049-1056. doi:10.1161/HYPERTENSIONAHA.111.176644
NLM
Martinez DG, Nicolau JC, Lage RL, Toschi-Dias E, de Matos LDNJ, Alves MJNN, Trombetta IC, Dias da Silva VJ, Middlekauff HR, Negrão CE, Rondon MU. Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients [Internet]. Hypertension. 2011 ; 58( 6): 1049-1056.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.176644
Vancouver
Martinez DG, Nicolau JC, Lage RL, Toschi-Dias E, de Matos LDNJ, Alves MJNN, Trombetta IC, Dias da Silva VJ, Middlekauff HR, Negrão CE, Rondon MU. Effects of Long-Term Exercise Training on Autonomic Control in Myocardial Infarction Patients [Internet]. Hypertension. 2011 ; 58( 6): 1049-1056.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.176644
Artigo de periodico-carta/editorial
Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] (2011)
Fanelli, Camilla; Zatz, Roberto
Source: Hypertension. Unidade: FM
Subjects: ESTRESSE OXIDATIVO, SISTEMA RENINA-ANGIOTENSINA, HIPERTENSÃO (FISIOPATOLOGIA), EDITORIAL (COMENTÁRIOS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FANELLI, Camilla e ZATZ, Roberto. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html. Acesso em: 12 jun. 2024. , 2011
APA
Fanelli, C., & Zatz, R. (2011). Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. doi:10.1161/HYPERTENSIONAHA.110.167775
NLM
Fanelli C, Zatz R. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] [Internet]. Hypertension. 2011 ; 57( 3): 373-374.[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html
Vancouver
Fanelli C, Zatz R. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] [Internet]. Hypertension. 2011 ; 57( 3): 373-374.[citado 2024 jun. 12 ] Available from: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html
Artigo de periodico
Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 (2011)
Giachini, Fernanda R.; Lima, Victor V.; Carneiro, Fernando S.; Passaglia, Rita de Cassia Aleixo Tostes; Webb, R. Clinton
Source: Hypertension. Unidade: FMRP
Subjects: ANGIOTENSINA II, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GIACHINI, Fernanda R. et al. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, v. 57, n. 3, p. 655-663, 2011Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327. Acesso em: 12 jun. 2024.
APA
Giachini, F. R., Lima, V. V., Carneiro, F. S., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, 57( 3), 655-663. doi:10.1161/HYPERTENSIONAHA.110.164327
NLM
Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
Vancouver
Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
Artigo de periodico
Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension (2011)
Pedrosa, Rodrigo P.; Drager, Luciano F.; Gonzaga, Carolina C.; Sousa, Marcio G.; Paula, Lílian K. G. de; Amaro, Aline C. S.; Amodeo, Celso; Bortolotto, Luiz A.; Krieger, Eduardo M.; Bradley, T. Douglas,; Lorenzi-Filho, Geraldo
Source: Hypertension. Unidade: FM
Subjects: APNEIA DO SONO, HIPERTENSÃO (DIAGNÓSTICO), PREVALÊNCIA, OBESIDADE, ULTRASSONOGRAFIA (MÉTODOS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEDROSA, Rodrigo P. et al. Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension. Hypertension, v. 58, n. 5, p. 811-817, 2011Tradução . . Disponível em: https://doi.org/10.1161/\200BHYPERTENSIONAHA.111.179788. Acesso em: 12 jun. 2024.
APA
Pedrosa, R. P., Drager, L. F., Gonzaga, C. C., Sousa, M. G., Paula, L. K. G. de, Amaro, A. C. S., et al. (2011). Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension. Hypertension, 58( 5), 811-817. doi:10.1161/\200BHYPERTENSIONAHA.111.179788
NLM
Pedrosa RP, Drager LF, Gonzaga CC, Sousa MG, Paula LKG de, Amaro ACS, Amodeo C, Bortolotto LA, Krieger EM, Bradley TD, Lorenzi-Filho G. Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension [Internet]. Hypertension. 2011 ; 58( 5): 811-817.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/\200BHYPERTENSIONAHA.111.179788
Vancouver
Pedrosa RP, Drager LF, Gonzaga CC, Sousa MG, Paula LKG de, Amaro ACS, Amodeo C, Bortolotto LA, Krieger EM, Bradley TD, Lorenzi-Filho G. Obstructive Sleep Apnea The Most Common Secondary Cause of Hypertension Associated With Resistant Hypertension [Internet]. Hypertension. 2011 ; 58( 5): 811-817.[citado 2024 jun. 12 ] Available from: https://doi.org/10.1161/\200BHYPERTENSIONAHA.111.179788
Artigo de periodico
O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1 (2010)
Lima, Victor Vitorino; Giachini, Fernanda Regina Casa Grande; Carneiro, Fernando S.; Carneiro, Zidonia N.; Saleh, Mohamed A.; Polock, David M.; Ergul, Adviye; Webb, R. Clinton; Passaglia, Rita de Cassia Aleixo Tostes; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli
Source: Hypertension. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Victor Vitorino et al. O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension, v. 55, n. 1, p. 180-188, 2010Tradução . . Acesso em: 12 jun. 2024.
APA
Lima, V. V., Giachini, F. R. C. G., Carneiro, F. S., Carneiro, Z. N., Saleh, M. A., Polock, D. M., et al. (2010). O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension, 55( 1), 180-188.
NLM
Lima VV, Giachini FRCG, Carneiro FS, Carneiro ZN, Saleh MA, Polock DM, Ergul A, Webb RC, Passaglia R de CAT, Fortes ZB, Carvalho MHC. O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension. 2010 ; 55( 1): 180-188.[citado 2024 jun. 12 ]
Vancouver
Lima VV, Giachini FRCG, Carneiro FS, Carneiro ZN, Saleh MA, Polock DM, Ergul A, Webb RC, Passaglia R de CAT, Fortes ZB, Carvalho MHC. O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension. 2010 ; 55( 1): 180-188.[citado 2024 jun. 12 ]

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