Filtros : "Indexado na Base de Dados PubMed" "Diniz, Gabriela Placoná" Removidos: "Oliva, Glaucius" "2019" "Indexado na Base de Dados Embase" "Indexado na Base de Dados Biosis Previews" Limpar

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  • Source: Clinical Science. Unidade: ICB

    Subjects: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, ANATOMIA

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    • ABNT

      SENGER, Nathalia et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, v. 132, n. 11, p. 1117-1133, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20171606. Acesso em: 07 jul. 2024.
    • APA

      Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Barreto-Chaves, M. L. M. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
    • NLM

      Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1042/CS20171606
    • Vancouver

      Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1042/CS20171606
  • Source: Therapeutic Advances in Cardiovascular Disease. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      DINIZ, Gabriela Placoná et al. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, v. 10, n. 4, p. 192-202, 2016Tradução . . Disponível em: https://doi.org/10.1177/1753944715623228. Acesso em: 07 jul. 2024.
    • APA

      Diniz, G. P., Senger, N., Carneiro-Ramos, M. S., Santos, R. A. S., & Barreto-Chaves, M. L. M. (2016). Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, 10( 4), 192-202. doi:10.1177/1753944715623228
    • NLM

      Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1177/1753944715623228
    • Vancouver

      Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1177/1753944715623228
  • Source: Basic Research in Cardiology. Unidade: ICB

    Subjects: ANATOMIA, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, ANGIOTENSINA II

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    • ABNT

      DINIZ, Gabriela Placoná et al. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, v. 110, n. 5, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00395-015-0504-7. Acesso em: 07 jul. 2024.
    • APA

      Diniz, G. P., Lino, C. A., Guedes, E. C., Moreira, L. do N., & Barreto-Chaves, M. L. M. (2015). Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, 110( 5), 1-11. doi:10.1007/s00395-015-0504-7
    • NLM

      Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
    • Vancouver

      Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      TAKANO, Ana Paula Cremasco e DINIZ, Gabriela Placoná e BARRETO-CHAVES, Maria Luiza Morais. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, v. 376, n. 1-2, p. 43-50, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.05.024. Acesso em: 07 jul. 2024.
    • APA

      Takano, A. P. C., Diniz, G. P., & Barreto-Chaves, M. L. M. (2013). AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, 376( 1-2), 43-50. doi:10.1016/j.mce.2013.05.024
    • NLM

      Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
    • Vancouver

      Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      DINIZ, Gabriela Placoná e TAKANO, Ana Paula Cremasco e BARRETO-CHAVES, Maria Luiza Morais. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, v. 374, n. 1-2, p. 117-124, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.04.010. Acesso em: 07 jul. 2024.
    • APA

      Diniz, G. P., Takano, A. P. C., & Barreto-Chaves, M. L. M. (2013). miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, 374( 1-2), 117-124. doi:10.1016/j.mce.2013.04.010
    • NLM

      Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
    • Vancouver

      Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
  • Source: Journal of Molecular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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    • ABNT

      DINIZ, Gabriela Placoná et al. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression. Journal of Molecular Endocrinology, v. 49, n. 1, p. 11-20, 2012Tradução . . Disponível em: https://doi.org/10.1530/JME-11-0141. Acesso em: 07 jul. 2024.
    • APA

      Diniz, G. P., Takano, A. P. C., Brunetto-Ract, E. L., Silva, F. G. da, Nunes, M. T., & Barreto-Chaves, M. L. M. (2012). New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression. Journal of Molecular Endocrinology, 49( 1), 11-20. doi:10.1530/JME-11-0141
    • NLM

      Diniz GP, Takano APC, Brunetto-Ract EL, Silva FG da, Nunes MT, Barreto-Chaves MLM. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression [Internet]. Journal of Molecular Endocrinology. 2012 ; 49( 1): 11-20.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1530/JME-11-0141
    • Vancouver

      Diniz GP, Takano APC, Brunetto-Ract EL, Silva FG da, Nunes MT, Barreto-Chaves MLM. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression [Internet]. Journal of Molecular Endocrinology. 2012 ; 49( 1): 11-20.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1530/JME-11-0141
  • Source: Autonomic Neuroscience: Basic and Clinical. Unidade: FM

    Subjects: HEMODINÂMICA (ALTERAÇÃO), SISTEMA NERVOSO AUTÔNOMO, HIPERTENSÃO, MODELOS ANIMAIS, HIPERTENSÃO PULMONAR, DENERVAÇÃO

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    • ABNT

      FLUES, K. et al. Cardiac and pulmonary arterial remodeling after sinoaortic denervation in normotensive rats. Autonomic Neuroscience: Basic and Clinical, v. 166, n. Ja, p. 47-53, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2011.10.005. Acesso em: 07 jul. 2024.
    • APA

      Flues, K., Moraes-Silva, I. C., Mostarda, C., Souza, P. R. M., Diniz, G. P., Moreira, E. D., et al. (2012). Cardiac and pulmonary arterial remodeling after sinoaortic denervation in normotensive rats. Autonomic Neuroscience: Basic and Clinical, 166( Ja), 47-53. doi:10.1016/j.autneu.2011.10.005
    • NLM

      Flues K, Moraes-Silva IC, Mostarda C, Souza PRM, Diniz GP, Moreira ED, Piratello AC, Barreto-Chaves MLM, De Angelis K, Salemi VMC, Irigoyen MC, Caldini EG. Cardiac and pulmonary arterial remodeling after sinoaortic denervation in normotensive rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2012 ; 166( Ja): 47-53.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.autneu.2011.10.005
    • Vancouver

      Flues K, Moraes-Silva IC, Mostarda C, Souza PRM, Diniz GP, Moreira ED, Piratello AC, Barreto-Chaves MLM, De Angelis K, Salemi VMC, Irigoyen MC, Caldini EG. Cardiac and pulmonary arterial remodeling after sinoaortic denervation in normotensive rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2012 ; 166( Ja): 47-53.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.autneu.2011.10.005
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      ARAUJO, A. S. R. et al. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, v. 333, n. 1, p. 78-84, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.12.018. Acesso em: 07 jul. 2024.
    • APA

      Araujo, A. S. R., Diniz, G. P., Seibel, F. E. R., Branchini, G., Ribeiro, M. F. M., Brum, I. S., et al. (2011). Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, 333( 1), 78-84. doi:10.1016/j.mce.2010.12.018
    • NLM

      Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
    • Vancouver

      Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
  • Source: Vascular Pharmacology. Unidade: ICB

    Assunto: ANATOMIA

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    • ABNT

      BARRETO-CHAVES, Maria Luiza Morais et al. The crosstalk between thyroid hormones and the Renin–Angiotensin System. Vascular Pharmacology, v. 52, n. 3-4, p. 166-170, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2009.10.009. Acesso em: 07 jul. 2024.
    • APA

      Barreto-Chaves, M. L. M., Carrillo-Sepúlveda, M. A., Carneiro-Ramos, M. S., Gomes, D. A., & Diniz, G. P. (2010). The crosstalk between thyroid hormones and the Renin–Angiotensin System. Vascular Pharmacology, 52( 3-4), 166-170. doi:10.1016/j.vph.2009.10.009
    • NLM

      Barreto-Chaves MLM, Carrillo-Sepúlveda MA, Carneiro-Ramos MS, Gomes DA, Diniz GP. The crosstalk between thyroid hormones and the Renin–Angiotensin System [Internet]. Vascular Pharmacology. 2010 ; 52( 3-4): 166-170.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.vph.2009.10.009
    • Vancouver

      Barreto-Chaves MLM, Carrillo-Sepúlveda MA, Carneiro-Ramos MS, Gomes DA, Diniz GP. The crosstalk between thyroid hormones and the Renin–Angiotensin System [Internet]. Vascular Pharmacology. 2010 ; 52( 3-4): 166-170.[citado 2024 jul. 07 ] Available from: https://doi.org/10.1016/j.vph.2009.10.009

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