Neuron-associated macrophage proliferation in the sensory ganglia is associated with peripheral nerve injury-induced neuropathic pain involving CX3CR1 signaling (2023)
- Authors:
- Guimarães, Rafaela Mano

- Silva, Conceição Elidianne Aníbal
- Davoli-Ferreira, Marcela
- Gomes, Francisco Isaac Fernandes

- Mendes, Atlante Silva

- Cavallini, Maria Cláudia Magalhães

- Fonseca, Miriam M.
- Damasceno, Samara
- Andrade, Larissa Pinto de
- Colonna, Marco
- Rivat, Cyril
- Cunha, Fernando de Queiroz

- Alves Filho, José Carlos Farias

- Cunha, Thiago Mattar

- Guimarães, Rafaela Mano
- USP affiliated authors: CUNHA, FERNANDO DE QUEIROZ - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; CUNHA, THIAGO MATTAR - FMRP ; GUIMARÃES, RAFAELA MANO - FMRP ; SILVA, CONCEIÇÃO ELIDIANNE ANIBAL - FMRP ; GOMES, FRANCISCO ISAAC FERNANDES - FMRP ; MENDES, ATLANTE SILVA - FMRP ; CAVALLINI, MARIA CLÁUDIA MAGALHÃES - FMRP ; DAMASCENO, SAMARA - FMRP ; ANDRADE, LARISSA PINTO DE - FMRP
- Unidade: FMRP
- DOI: 10.7554/eLife.78515
- Subjects: PROLIFERAÇÃO CELULAR; GÂNGLIOS ESPINHAIS; MACRÓFAGOS; NERVOS PERIFÉRICOS
- Keywords: Cytokines; Immunology; Inflammation; Macrophages; Monocytes; Mouse; Nerve injury
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
GUIMARÃES, Rafaela Mano et al. Neuron-associated macrophage proliferation in the sensory ganglia is associated with peripheral nerve injury-induced neuropathic pain involving CX3CR1 signaling. eLife, v. 12, p. 1-26, 2023Tradução . . Disponível em: https://doi.org/10.7554/eLife.78515. Acesso em: 27 dez. 2025. -
APA
Guimarães, R. M., Silva, C. E. A., Davoli-Ferreira, M., Gomes, F. I. F., Mendes, A. S., Cavallini, M. C. M., et al. (2023). Neuron-associated macrophage proliferation in the sensory ganglia is associated with peripheral nerve injury-induced neuropathic pain involving CX3CR1 signaling. eLife, 12, 1-26. doi:10.7554/eLife.78515 -
NLM
Guimarães RM, Silva CEA, Davoli-Ferreira M, Gomes FIF, Mendes AS, Cavallini MCM, Fonseca MM, Damasceno S, Andrade LP de, Colonna M, Rivat C, Cunha F de Q, Alves Filho JCF, Cunha TM. Neuron-associated macrophage proliferation in the sensory ganglia is associated with peripheral nerve injury-induced neuropathic pain involving CX3CR1 signaling [Internet]. eLife. 2023 ; 12 1-26.[citado 2025 dez. 27 ] Available from: https://doi.org/10.7554/eLife.78515 -
Vancouver
Guimarães RM, Silva CEA, Davoli-Ferreira M, Gomes FIF, Mendes AS, Cavallini MCM, Fonseca MM, Damasceno S, Andrade LP de, Colonna M, Rivat C, Cunha F de Q, Alves Filho JCF, Cunha TM. Neuron-associated macrophage proliferation in the sensory ganglia is associated with peripheral nerve injury-induced neuropathic pain involving CX3CR1 signaling [Internet]. eLife. 2023 ; 12 1-26.[citado 2025 dez. 27 ] Available from: https://doi.org/10.7554/eLife.78515 - Regulatory T cells counteract neuropathic pain through inhibition of the Th1 response at the site of peripheral nerve injury
- IL-27 counteracts neuropathic pain development through induction of IL-10
- Skin-resident dendritic cells mediate postoperative pain via CCR4 on sensory neurons
- Sensory neuron–associated macrophages as novel modulators of neuropathic pain
- Peripheral nitric oxide signaling directly blocks inflammatory pain
- Toll-like receptor 9 activation in neutrophils impairs chemotaxis and reduces sepsis outcome
- Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice
- The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β
- Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice
- Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation
Informações sobre o DOI: 10.7554/eLife.78515 (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
| 003236059.pdf | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas