Tumor associated macrophages promoted fatal patient derived acute promyelocytic leukemia in vivo (2021)
- Authors:
- USP affiliated authors: REGO, EDUARDO MAGALHÃES - FM ; WEINHÄUSER, ISABEL - FMRP
- Unidades: FM; FMRP
- DOI: 10.1182/blood-2021-150699
- Subjects: MACRÓFAGOS; TUMOR CARCINOIDE; MARCADOR MOLECULAR
- Language: Inglês
- Imprenta:
- Source:
- Conference titles: ASH Annual Meeting and Exposition
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
WEINHÄUSER, Isabel et al. Tumor associated macrophages promoted fatal patient derived acute promyelocytic leukemia in vivo. Blood. Atlanta: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1182/blood-2021-150699. Acesso em: 27 dez. 2025. , 2021 -
APA
Weinhäuser, I., Martins, D. A. P., Hilberink, J. R., Almeida, L. Y. de, Silveira, D. R. A., Quek, L., et al. (2021). Tumor associated macrophages promoted fatal patient derived acute promyelocytic leukemia in vivo. Blood. Atlanta: Faculdade de Medicina, Universidade de São Paulo. doi:10.1182/blood-2021-150699 -
NLM
Weinhäuser I, Martins DAP, Hilberink JR, Almeida LY de, Silveira DRA, Quek L, Hogeling SM, Mota JM, Ammatuna E, Araujo ARL de, Huls GA, Schuringa JJ, Rego EM. Tumor associated macrophages promoted fatal patient derived acute promyelocytic leukemia in vivo [Internet]. Blood. 2021 ; 138 3472.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1182/blood-2021-150699 -
Vancouver
Weinhäuser I, Martins DAP, Hilberink JR, Almeida LY de, Silveira DRA, Quek L, Hogeling SM, Mota JM, Ammatuna E, Araujo ARL de, Huls GA, Schuringa JJ, Rego EM. Tumor associated macrophages promoted fatal patient derived acute promyelocytic leukemia in vivo [Internet]. Blood. 2021 ; 138 3472.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1182/blood-2021-150699 - An M2-polarized macrophage microenvironment drives leukemogenesis and poor prognosis in acute myeloid leukemia
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Informações sobre o DOI: 10.1182/blood-2021-150699 (Fonte: oaDOI API)
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