Production of Peroxymonocarbonate by steady-state micromolar H2O2 and activated macrophages in the presence of CO2/HCO3– evidenced by boronate probes (2024)
- Authors:
- USP affiliated authors: LINARES, EDLAINE - IQ ; TRUZZI, DANIELA RAMOS - IQ ; AUGUSTO, OHARA - IQ
- Unidade: IQ
- DOI: 10.1021/acs.chemrestox.4c00059
- Subjects: MACRÓFAGOS; CINÉTICA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2024
- Source:
- Título: Chemical Research in Toxicology
- ISSN: 0893-228X
- Volume/Número/Paginação/Ano: v. 37, n. 7, p. 1129-1138, 2024
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: cc-by
-
ABNT
LINARES, Edlaine et al. Production of Peroxymonocarbonate by steady-state micromolar H2O2 and activated macrophages in the presence of CO2/HCO3– evidenced by boronate probes. Chemical Research in Toxicology, v. 37, n. 7, p. 1129-1138, 2024Tradução . . Disponível em: https://dx.doi.org/10.1021/acs.chemrestox.4c00059. Acesso em: 26 dez. 2025. -
APA
Linares, E., Severino, D., Truzzi, D. R., Rios, N., Radi, R., & Augusto, O. (2024). Production of Peroxymonocarbonate by steady-state micromolar H2O2 and activated macrophages in the presence of CO2/HCO3– evidenced by boronate probes. Chemical Research in Toxicology, 37( 7), 1129-1138. doi:10.1021/acs.chemrestox.4c00059 -
NLM
Linares E, Severino D, Truzzi DR, Rios N, Radi R, Augusto O. Production of Peroxymonocarbonate by steady-state micromolar H2O2 and activated macrophages in the presence of CO2/HCO3– evidenced by boronate probes [Internet]. Chemical Research in Toxicology. 2024 ; 37( 7): 1129-1138.[citado 2025 dez. 26 ] Available from: https://dx.doi.org/10.1021/acs.chemrestox.4c00059 -
Vancouver
Linares E, Severino D, Truzzi DR, Rios N, Radi R, Augusto O. Production of Peroxymonocarbonate by steady-state micromolar H2O2 and activated macrophages in the presence of CO2/HCO3– evidenced by boronate probes [Internet]. Chemical Research in Toxicology. 2024 ; 37( 7): 1129-1138.[citado 2025 dez. 26 ] Available from: https://dx.doi.org/10.1021/acs.chemrestox.4c00059 - Electron paramagnetic resonance (EPR) for investigating relevant players of redox reactions: radicals, metalloproteins and transition metal ions
- The labile iron pool attenuates peroxynitrite-dependent damage and can no longer be considered solely a pro-oxidative cellular iron source
- Reaction of human hemoglobin and peroxynitrite: involvement of protein radicals on the reaction mechanism
- Dynamics of dinitrosyl iron complex (DNIC) formation with Low molecular weight thiols
- The peroxidatic thiol of peroxiredoxin 1 is nitrosated by nitrosoglutathione but coordinates to the dinitrosyl iron complex of glutathione
- EPR studies of radical production In vivo by lypopolysaccharide. Potential role of iron mobilized from iron-nitrosyl complexes
- Tempol inhibits macrophage leishmanicidal activity in infected mice
- Production of peroxymonocarbonate evidenced by boronate probes under steady-state micromolar H2O2 and in PMA-activated macrophages
- EPR studies of in vivo radical production by lipopolysaccharide: potential role of iron mobilized from iron-nitrosyl complexes
- Inhibition of in vivo leishmanicidal mechanisms by tempol: Nitric oxide down-regulation and oxidant scavenging
Informações sobre o DOI: 10.1021/acs.chemrestox.4c00059 (Fonte: oaDOI API)
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