Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome (2024)
- Authors:
- Cesaro, Aline Cristina Parletta
- Cerri, Gabriela Cavazza
- Gasparini, Claudia Ribeiro Borba
- Panico, Karine
- Vieira Júnior, Denival Nascimento
- Rodrigues, Larissa Maria Zacarias
- Senger, Nathalia
- Silva, Amanda de Almeida
- Fevereiro, Marina Reingruber
- Diniz, Gabriela Placoná
- Irigoyen, Maria Cláudia Costa
- Chaves, Maria Luiza Morais Barreto de
- Cesaro, Aline Cristina Parletta
- USP affiliated authors: FEVEREIRO, MARINA REINGRUBER - ICB ; DINIZ, GABRIELA PLACONÁ - ICB ; IRIGOYEN, MARIA CLAUDIA COSTA - FM ; CHAVES, MARIA LUIZA DE MORAIS BARRETO DE - ICB ; CESARO, ALINE CRISTINA PARLETTA - ICB ; CERRI, GABRIELA CAVAZZA - ICB ; PANICO, KARINE - ICB ; VIEIRA JÚNIOR, DENIVAL NASCIMENTO - ICB ; RODRIGUES, LARISSA MARIA ZACARIAS - ICB ; SENGER, NATHALIA - ICB ; SILVA, AMANDA DE ALMEIDA - FM
- Unidades: ICB; FM
- DOI: 10.1007/s00424-024-02965-6
- Subjects: ANATOMIA; HIPERTROFIA VENTRICULAR ESQUERDA; INSUFICIÊNCIA CARDÍACA ANIMAL; CARDIOMEGALIA ANIMAL; HIPERTIREOIDISMO; HORMÔNIOS TIREOIDIANOS; FREQUÊNCIA CARDÍACA; CAMUNDONGOS; MODELOS ANIMAIS DE DOENÇAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Heidelberg
- Date published: 2024
- Source:
- Título: Pflügers Archiv - European Journal of Physiology
- ISSN: 1432-2013
- Volume/Número/Paginação/Ano: v. 476, p. 1065-1075, 2024
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CESARO, Aline Cristina Parletta et al. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome. Pflügers Archiv - European Journal of Physiology, v. 476, p. 1065-1075, 2024Tradução . . Disponível em: https://doi.org/10.1007/s00424-024-02965-6. Acesso em: 02 out. 2024. -
APA
Cesaro, A. C. P., Cerri, G. C., Gasparini, C. R. B., Panico, K., Vieira Júnior, D. N., Rodrigues, L. M. Z., et al. (2024). Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome. Pflügers Archiv - European Journal of Physiology, 476, 1065-1075. doi:10.1007/s00424-024-02965-6 -
NLM
Cesaro ACP, Cerri GC, Gasparini CRB, Panico K, Vieira Júnior DN, Rodrigues LMZ, Senger N, Silva A de A, Fevereiro MR, Diniz GP, Irigoyen MCC, Chaves MLMB de. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome [Internet]. Pflügers Archiv - European Journal of Physiology. 2024 ; 476 1065-1075.[citado 2024 out. 02 ] Available from: https://doi.org/10.1007/s00424-024-02965-6 -
Vancouver
Cesaro ACP, Cerri GC, Gasparini CRB, Panico K, Vieira Júnior DN, Rodrigues LMZ, Senger N, Silva A de A, Fevereiro MR, Diniz GP, Irigoyen MCC, Chaves MLMB de. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome [Internet]. Pflügers Archiv - European Journal of Physiology. 2024 ; 476 1065-1075.[citado 2024 out. 02 ] Available from: https://doi.org/10.1007/s00424-024-02965-6 - Impact of hyperthyroidism on cardiac hypertrophy
- Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway
- Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB
- AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth
- MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone
- miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation
- Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy
- Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor
- Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy
- Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction
Informações sobre o DOI: 10.1007/s00424-024-02965-6 (Fonte: oaDOI API)
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