Impaired gene expression due to iodine excess in the development and differentiation of eEndoderm and thyroid is associated with epigenetic changes (2020)
- Authors:
- USP affiliated authors: NUNES, MARIA TEREZA - ICB ; Nascimento, Caroline Serrano -
- Unidade: ICB
- DOI: 10.1089/thy.2018.0658
- Subjects: FISIOLOGIA; EPIGÊNESE GENÉTICA; EXPRESSÃO GÊNICA; CÉLULAS-TRONCO; ENDODERMA; DIFERENCIAÇÃO CELULAR; IODO; GLÂNDULA TIREOIDE; RATOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: New Rochelle
- Date published: 2020
- Source:
- Status:
- Nenhuma versão em acesso aberto identificada
-
ABNT
NASCIMENTO, Caroline Serrano et al. Impaired gene expression due to iodine excess in the development and differentiation of eEndoderm and thyroid is associated with epigenetic changes. Thyroid, v. 30, n. 4, p. 1-12, 2020Tradução . . Disponível em: https://doi.org/10.1089/thy.2018.0658. Acesso em: 14 abr. 2026. -
APA
Nascimento, C. S., Bernal, J. M., Ribeiro, R. da R., Nunes, M. T., & Santisteban, P. (2020). Impaired gene expression due to iodine excess in the development and differentiation of eEndoderm and thyroid is associated with epigenetic changes. Thyroid, 30( 4), 1-12. doi:10.1089/thy.2018.0658 -
NLM
Nascimento CS, Bernal JM, Ribeiro R da R, Nunes MT, Santisteban P. Impaired gene expression due to iodine excess in the development and differentiation of eEndoderm and thyroid is associated with epigenetic changes [Internet]. Thyroid. 2020 ; 30( 4): 1-12.[citado 2026 abr. 14 ] Available from: https://doi.org/10.1089/thy.2018.0658 -
Vancouver
Nascimento CS, Bernal JM, Ribeiro R da R, Nunes MT, Santisteban P. Impaired gene expression due to iodine excess in the development and differentiation of eEndoderm and thyroid is associated with epigenetic changes [Internet]. Thyroid. 2020 ; 30( 4): 1-12.[citado 2026 abr. 14 ] Available from: https://doi.org/10.1089/thy.2018.0658 - Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood
- Iodide excess reduces the activity of sodium-iodide symporter (nis) promoter: the role of PAX8 and P65 transcription factors on the inhibitory effects of this oligoelementrats
- Papel dos hormonios tireoideanos (ht) na expressao genica da mioglobina (mb) do tecido muscular esqueletico de ratos
- Possivel participacao da 5' monodesiodase do tipo ii na regulacao da expressao genica do hormonio de crescimento (gh) por hormonio tireoideano
- Effect of neonatal hyperthyroidism on GH gene expression reprogramming and physiological repercussions in rat adulthood
- T3 rapidly modulates TSHβ mRNA stability and translational rate in the pituitary of hypothyroid rats
- Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1
- Arginine induces GH gene expression by activating NOS/NO signaling in rat isolated hemi-pituitaries
- Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring
- Thyroid hormone treatment decreases hepatic glucose production and renal reabsorption of glucose in alloxan-induced diabetic Wistar rats
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