Network analysis reveals that the tumor suppressor lncRNA GAS5 acts as a double-edged sword in response to DNA damage in gastric cancer (2022)
- Authors:
- USP affiliated authors: HASHIMOTO, RONALDO FUMIO - IME ; GUPTA, SHANTANU - IME
- Unidade: IME
- DOI: 10.1038/s41598-022-21492-x
- Subjects: FUNÇÕES BOOLEANAS; DNA; RNA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Scientific Reports
- ISSN: 2045-2322
- Volume/Número/Paginação/Ano: v. 12, artigo n. 18312, p. 1-10, 2022
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
GUPTA, Shantanu et al. Network analysis reveals that the tumor suppressor lncRNA GAS5 acts as a double-edged sword in response to DNA damage in gastric cancer. Scientific Reports, v. 12, n. artigo 18312, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41598-022-21492-x. Acesso em: 25 nov. 2025. -
APA
Gupta, S., Panda, P. K., Luo, W., Hashimoto, R. F., & Ahuja, R. (2022). Network analysis reveals that the tumor suppressor lncRNA GAS5 acts as a double-edged sword in response to DNA damage in gastric cancer. Scientific Reports, 12( artigo 18312), 1-10. doi:10.1038/s41598-022-21492-x -
NLM
Gupta S, Panda PK, Luo W, Hashimoto RF, Ahuja R. Network analysis reveals that the tumor suppressor lncRNA GAS5 acts as a double-edged sword in response to DNA damage in gastric cancer [Internet]. Scientific Reports. 2022 ; 12( artigo 18312): 1-10.[citado 2025 nov. 25 ] Available from: https://doi.org/10.1038/s41598-022-21492-x -
Vancouver
Gupta S, Panda PK, Luo W, Hashimoto RF, Ahuja R. Network analysis reveals that the tumor suppressor lncRNA GAS5 acts as a double-edged sword in response to DNA damage in gastric cancer [Internet]. Scientific Reports. 2022 ; 12( artigo 18312): 1-10.[citado 2025 nov. 25 ] Available from: https://doi.org/10.1038/s41598-022-21492-x - DNA damage-induced ferroptosis: a boolean model regulating p53 and non-coding RNAs in drug resistance
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- Targeting NSCLC drug resistance: systems biology insights into the MALAT1/miR-145-5p axis and Wip1 in regulating ferroptosis and apoptosis
- Dynamical analysis of a Boolean network model of the oncogene role of lncRNA ANRIL and lncRNA UFC1 in non-dmall vell lung cancer
- Dynamical modeling of miR‑34a, miR‑449a, and miR‑16 reveals numerous DDR signaling pathways regulating senescence, autophagy, and apoptosis in HeLa cells
- A Boolean model of the oncogene role of FAM111B in lung adenocarcinoma
- Quadra-stable dynamics of p53 and PTEN in the DNA damage response
- The lncRNA DLX6-AS1/miR-16-5p axis regulates autophagy and apoptosis in non-small cell lung cancer: a Boolean model of cell death
- A dynamic Boolean network reveals that the BMI1 and MALAT1 axis is associated with drug resistance by limiting miR-145-5p in non-small cell lung cancer
Informações sobre o DOI: 10.1038/s41598-022-21492-x (Fonte: oaDOI API)
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