IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation (2021)
- Authors:
- Autor USP: GAZZINELLI, RICARDO TOSTES - FMRP
- Unidade: FMRP
- DOI: 10.4049/jimmunol.2001194
- Subjects: DIFERENCIAÇÃO CELULAR; CÉLULAS CULTIVADAS; INFLAMAÇÃO; INTERFERONS; RECEPTORES; LIPOPOLISSACARÍDEOS; MONÓCITOS; CULTURA DE CÉLULAS; TOXOPLASMOSE
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 206, n. 7, p. 1642-1652, 2021
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
AMANCIO, Alice Muglia et al. IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation. Journal of Immunology, v. 206, n. 7, p. 1642-1652, 2021Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.2001194. Acesso em: 11 fev. 2026. -
APA
Amancio, A. M., Mittereder, L., Carletti, A., Tosh, K. W., Green, D., Antonelli, L. R., et al. (2021). IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation. Journal of Immunology, 206( 7), 1642-1652. doi:10.4049/jimmunol.2001194 -
NLM
Amancio AM, Mittereder L, Carletti A, Tosh KW, Green D, Antonelli LR, Gazzinelli RT, Sher A, Jankovic D. IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation [Internet]. Journal of Immunology. 2021 ; 206( 7): 1642-1652.[citado 2026 fev. 11 ] Available from: https://doi.org/10.4049/jimmunol.2001194 -
Vancouver
Amancio AM, Mittereder L, Carletti A, Tosh KW, Green D, Antonelli LR, Gazzinelli RT, Sher A, Jankovic D. IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation [Internet]. Journal of Immunology. 2021 ; 206( 7): 1642-1652.[citado 2026 fev. 11 ] Available from: https://doi.org/10.4049/jimmunol.2001194 - Rapamycin improves the response of effector and memory CD8+ T cells induced by immunization with ASP2 of Trypanosoma cruzi
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Informações sobre o DOI: 10.4049/jimmunol.2001194 (Fonte: oaDOI API)
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