Hepatoblastomas exhibit marked NNMT downregulation driven by promoter DNA hypermethylation (2020)
- Authors:
- Rivas, Maria Prates
- Aguiar, Talita Ferreira Marques
- Maschietto, Mariana
- Lemes, Renan B
- Caires-Junior, Luiz Carlos
- Goulart, Ernesto
- Telles-Silva, Kayque Alves
- Novak, Estela
- Cristofani, Lilian Maria
- Odone, Vicente
- Cypriano, Monica
- Toledo, Silvia Regina Caminada de
- Carraro, Dirce Maria
- Escobar, Melissa Quintero
- Lee, Hana
- Johnston, Michael
- Costa, Cecilia Maria Lima da
- Cunha, Isabela Werneck da
- Tasic, Ljubica
- Pearson, Peter L
- Rosenberg, Carla
- Timchenko, Nikolai
- Krepischi, Ana Cristina Victorino
- USP affiliated authors: NOVAK, ESTELA MARIA - FM ; CRISTOFANI, LILIAN MARIA - FM ; ODONE FILHO, VICENTE - FM ; PEARSON, PETER LEES - IB ; ROSENBERG, CARLA - IB ; KREPISCHI, ANA CRISTINA VICTORINO - IB ; RIVAS, MARIA PRATES - IB ; AGUIAR, TALITA FERREIRA MARQUES - IB ; LEMES, RENAN BARBOSA - IB ; CAIRES JÚNIOR, LUIZ CARLOS DE - IB ; GUIMARÃES, ERNESTO DA SILVEIRA GOULART - IB ; SILVA, KAYQUE ALVES TELLES - IB
- Unidades: FM; IB
- DOI: 10.1177/1010428320977124
- Subjects: METILAÇÃO DE DNA; NEOPLASIAS HEPÁTICAS; METABOLÔMICA; EPIGÊNESE GENÉTICA; CARCINOGÊNESE
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Tumor Biology
- ISSN: 1423-0380
- Volume/Número/Paginação/Ano: on-line, Dec. 2020
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc
-
ABNT
RIVAS, Maria Prates et al. Hepatoblastomas exhibit marked NNMT downregulation driven by promoter DNA hypermethylation. Tumor Biology, 2020Tradução . . Disponível em: https://doi.org/10.1177/1010428320977124. Acesso em: 29 dez. 2025. -
APA
Rivas, M. P., Aguiar, T. F. M., Maschietto, M., Lemes, R. B., Caires-Junior, L. C., Goulart, E., et al. (2020). Hepatoblastomas exhibit marked NNMT downregulation driven by promoter DNA hypermethylation. Tumor Biology. doi:10.1177/1010428320977124 -
NLM
Rivas MP, Aguiar TFM, Maschietto M, Lemes RB, Caires-Junior LC, Goulart E, Telles-Silva KA, Novak E, Cristofani LM, Odone V, Cypriano M, Toledo SRC de, Carraro DM, Escobar MQ, Lee H, Johnston M, Costa CML da, Cunha IW da, Tasic L, Pearson PL, Rosenberg C, Timchenko N, Krepischi ACV. Hepatoblastomas exhibit marked NNMT downregulation driven by promoter DNA hypermethylation [Internet]. Tumor Biology. 2020 ;[citado 2025 dez. 29 ] Available from: https://doi.org/10.1177/1010428320977124 -
Vancouver
Rivas MP, Aguiar TFM, Maschietto M, Lemes RB, Caires-Junior LC, Goulart E, Telles-Silva KA, Novak E, Cristofani LM, Odone V, Cypriano M, Toledo SRC de, Carraro DM, Escobar MQ, Lee H, Johnston M, Costa CML da, Cunha IW da, Tasic L, Pearson PL, Rosenberg C, Timchenko N, Krepischi ACV. Hepatoblastomas exhibit marked NNMT downregulation driven by promoter DNA hypermethylation [Internet]. Tumor Biology. 2020 ;[citado 2025 dez. 29 ] Available from: https://doi.org/10.1177/1010428320977124 - TET Upregulation Leads to 5-Hydroxymethylation Enrichment in Hepatoblastoma
- DNA methylation as a key epigenetic player for hepatoblastoma characterization
- Copy number alterations in hepatoblastoma: literature review and a brazilian cohort analysis highlight new biological pathways
- Applied hepatic bioengineering: modeling the human liver using organoid and liver-on-a-chip technologies
- Germline copy number variations and cancer predisposition
- Acute promyelocytic leukemia in childhood and adolescence: treatment results of a modified AIDA protocol at a Brazilian center
- Lymphovascular invasion and histologic grade are associated with specific genomic profiles in invasive carcinomas of the breast
- Cytogenetically visible inversions are formed by multiple molecular mechanisms
- LINE-1 hypermethylation in peripheral blood of cutaneous melanoma patients is associated with metastasis
- Rare germline copy number variations in hereditary cutaneous melanoma
Informações sobre o DOI: 10.1177/1010428320977124 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
