COVID-19 pandemic and dysbiosis: can the ivermectin hysteria lead to an increase of autoimmune neuroinflammatory diseases? (2020)
- Authors:
- USP affiliated authors: PERON, JEAN PIERRE SCHATZMANN - ICB ; NAKAYA, HELDER TAKASHI IMOTO - FCF
- Unidades: ICB; FCF
- DOI: 10.1615/CritRevImmunol.v40.i6
- Subjects: COVID-19; ANTIPARASITÁRIOS; IMUNOLOGIA; HISTERIA; DOENÇAS DO SISTEMA NERVOSO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Critical Reviews™ in Immunology
- ISSN: 1040-8401
- Volume/Número/Paginação/Ano: v. 46, n. 6, p. 537–542, 2020
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão submetida (Pré-print)
- Acessar versão aberta:
-
ABNT
PERON, Jean Pierre Schatzmann et al. COVID-19 pandemic and dysbiosis: can the ivermectin hysteria lead to an increase of autoimmune neuroinflammatory diseases?. Critical Reviews™ in Immunology, v. 46, n. 6, p. 537–542, 2020Tradução . . Disponível em: https://doi.org/10.1615/CritRevImmunol.v40.i6. Acesso em: 31 mar. 2026. -
APA
Peron, J. P. S., Nakaya, H. T. I., Schlindwein, M. A. M., & Gonçalves, M. V. M. (2020). COVID-19 pandemic and dysbiosis: can the ivermectin hysteria lead to an increase of autoimmune neuroinflammatory diseases? Critical Reviews™ in Immunology, 46( 6), 537–542. doi:10.1615/CritRevImmunol.v40.i6 -
NLM
Peron JPS, Nakaya HTI, Schlindwein MAM, Gonçalves MVM. COVID-19 pandemic and dysbiosis: can the ivermectin hysteria lead to an increase of autoimmune neuroinflammatory diseases? [Internet]. Critical Reviews™ in Immunology. 2020 ; 46( 6): 537–542.[citado 2026 mar. 31 ] Available from: https://doi.org/10.1615/CritRevImmunol.v40.i6 -
Vancouver
Peron JPS, Nakaya HTI, Schlindwein MAM, Gonçalves MVM. COVID-19 pandemic and dysbiosis: can the ivermectin hysteria lead to an increase of autoimmune neuroinflammatory diseases? [Internet]. Critical Reviews™ in Immunology. 2020 ; 46( 6): 537–542.[citado 2026 mar. 31 ] Available from: https://doi.org/10.1615/CritRevImmunol.v40.i6 - Susceptibility of the elderly to sars-cov-2 infection: ace-2 overexpression, shedding, and antibody-dependent enhancement (ade)
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