Lentiviral gene therapy rescues p47phox chronic granulomatous disease and the ability to fight Salmonella infection in mice (2020)
- Authors:
- Autor USP: CONDINO NETO, ANTONIO - ICB
- Unidade: ICB
- DOI: 10.1038/s41434-020-0164-6
- Subjects: IMUNOLOGIA; SALMONELOSE ANIMAL; CAMUNDONGOS; GRANULOMA; INFECÇÕES BACTERIANAS GRAM-NEGATIVAS; IMUNODEFICIÊNCIA DE VARIÁVEL COMUM; INFLAMAÇÃO; MUTAÇÃO GENÉTICA; ATIVAÇÃO ENZIMÁTICA; ENZIMAS; CÉLULAS-TRONCO; GENÉTICA ANIMAL
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Gene Therapy
- ISSN: 1476-5462
- Volume/Número/Paginação/Ano: v. 27, p. 459–469, 2020
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
SCHEJTMAN, Andrea et al. Lentiviral gene therapy rescues p47phox chronic granulomatous disease and the ability to fight Salmonella infection in mice. Gene Therapy, v. 27, p. 459–469, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41434-020-0164-6. Acesso em: 27 jan. 2026. -
APA
Schejtman, A., Aragão Filho, W. C., Clare, S., Zinicola, M., Weisser, M., Burns, S. O., et al. (2020). Lentiviral gene therapy rescues p47phox chronic granulomatous disease and the ability to fight Salmonella infection in mice. Gene Therapy, 27, 459–469. doi:10.1038/s41434-020-0164-6 -
NLM
Schejtman A, Aragão Filho WC, Clare S, Zinicola M, Weisser M, Burns SO, Booth C, Gaspar HB, Thomas DC, Condino Neto A, Thrasher AJ, Santilli G. Lentiviral gene therapy rescues p47phox chronic granulomatous disease and the ability to fight Salmonella infection in mice [Internet]. Gene Therapy. 2020 ; 27 459–469.[citado 2026 jan. 27 ] Available from: https://doi.org/10.1038/s41434-020-0164-6 -
Vancouver
Schejtman A, Aragão Filho WC, Clare S, Zinicola M, Weisser M, Burns SO, Booth C, Gaspar HB, Thomas DC, Condino Neto A, Thrasher AJ, Santilli G. Lentiviral gene therapy rescues p47phox chronic granulomatous disease and the ability to fight Salmonella infection in mice [Internet]. Gene Therapy. 2020 ; 27 459–469.[citado 2026 jan. 27 ] Available from: https://doi.org/10.1038/s41434-020-0164-6 - Neutrophils and monocytes from patients with CD40L deficiency present a defective respiratory burst response to several stimuli and this defect can be reversed by exogenous IFN-Gamma
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Informações sobre o DOI: 10.1038/s41434-020-0164-6 (Fonte: oaDOI API)
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